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Experimental lupus is aggravated in mouse strains with impaired induction of neutrophil extracellular traps.

JCI insight

Kienhöfer D, Hahn J, Stoof J, Csepregi JZ, Reinwald C, Urbonaviciute V, Johnsson C, Maueröder C, Podolska MJ, Biermann MH, Leppkes M, Harrer T, Hultqvist M, Olofsson P, Munoz LE, Mocsai A, Herrmann M, Schett G, Holmdahl R, Hoffmann MH.
PMID: 28515366
JCI Insight. 2017 May 18;2(10). doi: 10.1172/jci.insight.92920. eCollection 2017 May 18.

Many effector mechanisms of neutrophils have been implicated in the pathogenesis of systemic lupus erythematosus (SLE). Neutrophil extracellular traps (NETs) have been assigned a particularly detrimental role. Here we investigated the functional impact of neutrophils and NETs on a...

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Kienhöfer D, Hahn J, Stoof J, et al. Experimental lupus is aggravated in mouse strains with impaired induction of neutrophil extracellular traps. JCI Insight. 2017;2(10)doi: 10.1172/jci.insight.92920.
Kienhöfer, D., Hahn, J., Stoof, J., Csepregi, J. Z., Reinwald, C., Urbonaviciute, V., Johnsson, C., Maueröder, C., Podolska, M. J., Biermann, M. H., Leppkes, M., Harrer, T., Hultqvist, M., Olofsson, P., Munoz, L. E., Mocsai, A., Herrmann, M., Schett, G., Holmdahl, R., & Hoffmann, M. H. (2017). Experimental lupus is aggravated in mouse strains with impaired induction of neutrophil extracellular traps. JCI insight, 2(10), . https://doi.org/10.1172/jci.insight.92920
Kienhöfer, Deborah, et al. "Experimental lupus is aggravated in mouse strains with impaired induction of neutrophil extracellular traps." JCI insight vol. 2,10 (2017). doi: https://doi.org/10.1172/jci.insight.92920
Kienhöfer D, Hahn J, Stoof J, Csepregi JZ, Reinwald C, Urbonaviciute V, Johnsson C, Maueröder C, Podolska MJ, Biermann MH, Leppkes M, Harrer T, Hultqvist M, Olofsson P, Munoz LE, Mocsai A, Herrmann M, Schett G, Holmdahl R, Hoffmann MH. Experimental lupus is aggravated in mouse strains with impaired induction of neutrophil extracellular traps. JCI Insight. 2017 May 18;2(10). doi: 10.1172/jci.insight.92920. eCollection 2017 May 18. PMID: 28515366; PMCID: PMC5436535.
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Molecular and Translational Classifications of DAMPs in Immunogenic Cell Death.

Frontiers in immunology

Garg AD, Galluzzi L, Apetoh L, Baert T, Birge RB, Bravo-San Pedro JM, Breckpot K, Brough D, Chaurio R, Cirone M, Coosemans A, Coulie PG, De Ruysscher D, Dini L, de Witte P, Dudek-Peric AM, Faggioni A, Fucikova J, Gaipl US, Golab J, Gougeon ML, Hamblin MR, Hemminki A, Herrmann M, Hodge JW, Kepp O, Kroemer G, Krysko DV, Land WG, Madeo F, Manfredi AA, Mattarollo SR, Maueroder C, Merendino N, Multhoff G, Pabst T, Ricci JE, Riganti C, Romano E, Rufo N, Smyth MJ, Sonnemann J, Spisek R, Stagg J, Vacchelli E, Vandenabeele P, Vandenberk L, Van den Eynde BJ, Van Gool S, Velotti F, Zitvogel L, Agostinis P.
PMID: 26635802
Front Immunol. 2015 Nov 20;6:588. doi: 10.3389/fimmu.2015.00588. eCollection 2015.

The immunogenicity of malignant cells has recently been acknowledged as a critical determinant of efficacy in cancer therapy. Thus, besides developing direct immunostimulatory regimens, including dendritic cell-based vaccines, checkpoint-blocking therapies, and adoptive T-cell transfer, researchers have started to focus...

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Garg AD, Galluzzi L, Apetoh L, et al. Molecular and Translational Classifications of DAMPs in Immunogenic Cell Death. Front Immunol. 2015;6:588doi: 10.3389/fimmu.2015.00588.
Garg, A. D., Galluzzi, L., Apetoh, L., Baert, T., Birge, R. B., Bravo-San Pedro, J. M., Breckpot, K., Brough, D., Chaurio, R., Cirone, M., Coosemans, A., Coulie, P. G., De Ruysscher, D., Dini, L., de Witte, P., Dudek-Peric, A. M., Faggioni, A., Fucikova, J., Gaipl, U. S., Golab, J., Gougeon, M. L., Hamblin, M. R., Hemminki, A., Herrmann, M., Hodge, J. W., Kepp, O., Kroemer, G., Krysko, D. V., Land, W. G., Madeo, F., Manfredi, A. A., Mattarollo, S. R., Maueroder, C., Merendino, N., Multhoff, G., Pabst, T., Ricci, J. E., Riganti, C., Romano, E., Rufo, N., Smyth, M. J., Sonnemann, J., Spisek, R., Stagg, J., Vacchelli, E., Vandenabeele, P., Vandenberk, L., Van den Eynde, B. J., Van Gool, S., Velotti, F., Zitvogel, L., & Agostinis, P. (2015). Molecular and Translational Classifications of DAMPs in Immunogenic Cell Death. Frontiers in immunology, 6588. https://doi.org/10.3389/fimmu.2015.00588
Garg, Abhishek D, et al. "Molecular and Translational Classifications of DAMPs in Immunogenic Cell Death." Frontiers in immunology vol. 6 (2015): 588. doi: https://doi.org/10.3389/fimmu.2015.00588
Garg AD, Galluzzi L, Apetoh L, Baert T, Birge RB, Bravo-San Pedro JM, Breckpot K, Brough D, Chaurio R, Cirone M, Coosemans A, Coulie PG, De Ruysscher D, Dini L, de Witte P, Dudek-Peric AM, Faggioni A, Fucikova J, Gaipl US, Golab J, Gougeon ML, Hamblin MR, Hemminki A, Herrmann M, Hodge JW, Kepp O, Kroemer G, Krysko DV, Land WG, Madeo F, Manfredi AA, Mattarollo SR, Maueroder C, Merendino N, Multhoff G, Pabst T, Ricci JE, Riganti C, Romano E, Rufo N, Smyth MJ, Sonnemann J, Spisek R, Stagg J, Vacchelli E, Vandenabeele P, Vandenberk L, Van den Eynde BJ, Van Gool S, Velotti F, Zitvogel L, Agostinis P. Molecular and Translational Classifications of DAMPs in Immunogenic Cell Death. Front Immunol. 2015 Nov 20;6:588. doi: 10.3389/fimmu.2015.00588. eCollection 2015. PMID: 26635802; PMCID: PMC4653610.
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[Suicides and Suicid Rates in Germany, Bavaria and Upper Frankonia].

Psychiatrische Praxis

Mauerer C, Wolfersdorf M, Keller F.
PMID: 13130368
Psychiatr Prax. 2003 May;30:176-178. doi: 10.1055/s-2003-39755.

The authors give an actual survey about suicides and suicide rates in Germany, Bavaria and Upper Frankonia. Their special interest are significant trends in the last years. These trends will be shown and shortly described.

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Mauerer C, Wolfersdorf M, Keller F. Suizidzahlen und -raten im Vergleich Deutschland, Bayern und Oberfranken. [Suicides and Suicid Rates in Germany, Bavaria and Upper Frankonia]. Psychiatr Prax. 2003;30:176-178doi: 10.1055/s-2003-39755.
Mauerer, C., Wolfersdorf, M., & Keller, F. (2003). Suizidzahlen und -raten im Vergleich Deutschland, Bayern und Oberfranken. [Suicides and Suicid Rates in Germany, Bavaria and Upper Frankonia]. Psychiatrische Praxis, 30176-178. https://doi.org/10.1055/s-2003-39755
Mauerer, Christian, et al. "Suizidzahlen und -raten im Vergleich Deutschland, Bayern und Oberfranken." [Suicides and Suicid Rates in Germany, Bavaria and Upper Frankonia]. Psychiatrische Praxis vol. 30 (2003): 176-178. doi: https://doi.org/10.1055/s-2003-39755
Mauerer C, Wolfersdorf M, Keller F. Suizidzahlen und -raten im Vergleich Deutschland, Bayern und Oberfranken. [Suicides and Suicid Rates in Germany, Bavaria and Upper Frankonia]. Psychiatr Prax. 2003 May;30:176-178. doi: 10.1055/s-2003-39755. German. PMID: 13130368.
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Tumor immunotherapy: lessons from autoimmunity.

Frontiers in immunology

Maueröder C, Munoz LE, Chaurio RA, Herrmann M, Schett G, Berens C.
PMID: 24860574
Front Immunol. 2014 May 13;5:212. doi: 10.3389/fimmu.2014.00212. eCollection 2014.

No abstract available.

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Maueröder C, Munoz LE, Chaurio RA, et al. Tumor immunotherapy: lessons from autoimmunity. Front Immunol. 2014;5:212doi: 10.3389/fimmu.2014.00212.
Maueröder, C., Munoz, L. E., Chaurio, R. A., Herrmann, M., Schett, G., & Berens, C. (2014). Tumor immunotherapy: lessons from autoimmunity. Frontiers in immunology, 5212. https://doi.org/10.3389/fimmu.2014.00212
Maueröder, Christian, et al. "Tumor immunotherapy: lessons from autoimmunity." Frontiers in immunology vol. 5 (2014): 212. doi: https://doi.org/10.3389/fimmu.2014.00212
Maueröder C, Munoz LE, Chaurio RA, Herrmann M, Schett G, Berens C. Tumor immunotherapy: lessons from autoimmunity. Front Immunol. 2014 May 13;5:212. doi: 10.3389/fimmu.2014.00212. eCollection 2014. PMID: 24860574; PMCID: PMC4026709.
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A20 deficiency in myeloid cells protects mice from diet-induced obesity and insulin resistance due to increased fatty acid metabolism.

Cell reports

Catrysse L, Maes B, Mehrotra P, Martens A, Hoste E, Martens L, Maueröder C, Remmerie A, Bujko A, Slowicka K, Sze M, Vikkula H, Ghesquière B, Scott CL, Saeys Y, van de Sluis B, Ravichandran K, Janssens S, van Loo G.
PMID: 34551300
Cell Rep. 2021 Sep 21;36(12):109748. doi: 10.1016/j.celrep.2021.109748.

Obesity-induced inflammation is a major driving force in the development of insulin resistance, type 2 diabetes (T2D), and related metabolic disorders. During obesity, macrophages accumulate in the visceral adipose tissue, creating a low-grade inflammatory environment. Nuclear factor κB (NF-κB)...

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Catrysse L, Maes B, Mehrotra P, et al. A20 deficiency in myeloid cells protects mice from diet-induced obesity and insulin resistance due to increased fatty acid metabolism. Cell Rep. 2021;36(12):109748doi: 10.1016/j.celrep.2021.109748.
Catrysse, L., Maes, B., Mehrotra, P., Martens, A., Hoste, E., Martens, L., Maueröder, C., Remmerie, A., Bujko, A., Slowicka, K., Sze, M., Vikkula, H., Ghesquière, B., Scott, C. L., Saeys, Y., van de Sluis, B., Ravichandran, K., Janssens, S., & van Loo, G. (2021). A20 deficiency in myeloid cells protects mice from diet-induced obesity and insulin resistance due to increased fatty acid metabolism. Cell reports, 36(12), 109748. https://doi.org/10.1016/j.celrep.2021.109748
Catrysse, Leen, et al. "A20 deficiency in myeloid cells protects mice from diet-induced obesity and insulin resistance due to increased fatty acid metabolism." Cell reports vol. 36,12 (2021): 109748. doi: https://doi.org/10.1016/j.celrep.2021.109748
Catrysse L, Maes B, Mehrotra P, Martens A, Hoste E, Martens L, Maueröder C, Remmerie A, Bujko A, Slowicka K, Sze M, Vikkula H, Ghesquière B, Scott CL, Saeys Y, van de Sluis B, Ravichandran K, Janssens S, van Loo G. A20 deficiency in myeloid cells protects mice from diet-induced obesity and insulin resistance due to increased fatty acid metabolism. Cell Rep. 2021 Sep 21;36(12):109748. doi: 10.1016/j.celrep.2021.109748. PMID: 34551300.
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The progression of cell death affects the rejection of allogeneic tumors in immune-competent mice - implications for cancer therapy.

Frontiers in immunology

Chaurio RA, Muñoz LE, Maueröder C, Janko C, Harrer T, Fürnrohr BG, Niederweis M, Bilyy R, Schett G, Herrmann M, Berens C.
PMID: 25426116
Front Immunol. 2014 Nov 11;5:560. doi: 10.3389/fimmu.2014.00560. eCollection 2014.

Large amounts of dead and dying cells are produced during cancer therapy and allograft rejection. Depending on the death pathway and stimuli involved, dying cells exhibit diverse features, resulting in defined physiological consequences for the host. It is not...

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Chaurio RA, Muñoz LE, Maueröder C, et al. The progression of cell death affects the rejection of allogeneic tumors in immune-competent mice - implications for cancer therapy. Front Immunol. 2014;5:560doi: 10.3389/fimmu.2014.00560.
Chaurio, R. A., Muñoz, L. E., Maueröder, C., Janko, C., Harrer, T., Fürnrohr, B. G., Niederweis, M., Bilyy, R., Schett, G., Herrmann, M., & Berens, C. (2014). The progression of cell death affects the rejection of allogeneic tumors in immune-competent mice - implications for cancer therapy. Frontiers in immunology, 5560. https://doi.org/10.3389/fimmu.2014.00560
Chaurio, Ricardo A, et al. "The progression of cell death affects the rejection of allogeneic tumors in immune-competent mice - implications for cancer therapy." Frontiers in immunology vol. 5 (2014): 560. doi: https://doi.org/10.3389/fimmu.2014.00560
Chaurio RA, Muñoz LE, Maueröder C, Janko C, Harrer T, Fürnrohr BG, Niederweis M, Bilyy R, Schett G, Herrmann M, Berens C. The progression of cell death affects the rejection of allogeneic tumors in immune-competent mice - implications for cancer therapy. Front Immunol. 2014 Nov 11;5:560. doi: 10.3389/fimmu.2014.00560. eCollection 2014. PMID: 25426116; PMCID: PMC4227513.
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Ménage-à-Trois: The Ratio of Bicarbonate to CO.

Frontiers in immunology

Maueröder C, Mahajan A, Paulus S, Gößwein S, Hahn J, Kienhöfer D, Biermann MH, Tripal P, Friedrich RP, Munoz LE, Neurath MF, Becker C, Schett GA, Herrmann M, Leppkes M.
PMID: 28018350
Front Immunol. 2016 Dec 09;7:583. doi: 10.3389/fimmu.2016.00583. eCollection 2016.

In this study, we identified and characterized the potential of a high ratio of bicarbonate to CO

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Maueröder C, Mahajan A, Paulus S, et al. Ménage-à-Trois: The Ratio of Bicarbonate to CO. Front Immunol. 2016;7:583doi: 10.3389/fimmu.2016.00583.
Maueröder, C., Mahajan, A., Paulus, S., Gößwein, S., Hahn, J., Kienhöfer, D., Biermann, M. H., Tripal, P., Friedrich, R. P., Munoz, L. E., Neurath, M. F., Becker, C., Schett, G. A., Herrmann, M., & Leppkes, M. (2016). Ménage-à-Trois: The Ratio of Bicarbonate to CO. Frontiers in immunology, 7583. https://doi.org/10.3389/fimmu.2016.00583
Maueröder, Christian, et al. "Ménage-à-Trois: The Ratio of Bicarbonate to CO." Frontiers in immunology vol. 7 (2016): 583. doi: https://doi.org/10.3389/fimmu.2016.00583
Maueröder C, Mahajan A, Paulus S, Gößwein S, Hahn J, Kienhöfer D, Biermann MH, Tripal P, Friedrich RP, Munoz LE, Neurath MF, Becker C, Schett GA, Herrmann M, Leppkes M. Ménage-à-Trois: The Ratio of Bicarbonate to CO. Front Immunol. 2016 Dec 09;7:583. doi: 10.3389/fimmu.2016.00583. eCollection 2016. PMID: 28018350; PMCID: PMC5145884.
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Inosine Released from Dying or Dead Cells Stimulates Cell Proliferation .

Frontiers in immunology

Chen J, Chaurio RA, Maueröder C, Derer A, Rauh M, Kost A, Liu Y, Mo X, Hueber A, Bilyy R, Herrmann M, Zhao Y, Muñoz LE.
PMID: 28496447
Front Immunol. 2017 Apr 27;8:504. doi: 10.3389/fimmu.2017.00504. eCollection 2017.

INTRODUCTION: Many antitumor therapies induce apoptotic cell death in order to cause tumor regression. Paradoxically, apoptotic cells are also known to promote wound healing, cell proliferation, and tumor cell repopulation in multicellular organisms. We aimed to characterize the nature...

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Chen J, Chaurio RA, Maueröder C, et al. Inosine Released from Dying or Dead Cells Stimulates Cell Proliferation . Front Immunol. 2017;8:504doi: 10.3389/fimmu.2017.00504.
Chen, J., Chaurio, R. A., Maueröder, C., Derer, A., Rauh, M., Kost, A., Liu, Y., Mo, X., Hueber, A., Bilyy, R., Herrmann, M., Zhao, Y., & Muñoz, L. E. (2017). Inosine Released from Dying or Dead Cells Stimulates Cell Proliferation . Frontiers in immunology, 8504. https://doi.org/10.3389/fimmu.2017.00504
Chen, Jin, et al. "Inosine Released from Dying or Dead Cells Stimulates Cell Proliferation ." Frontiers in immunology vol. 8 (2017): 504. doi: https://doi.org/10.3389/fimmu.2017.00504
Chen J, Chaurio RA, Maueröder C, Derer A, Rauh M, Kost A, Liu Y, Mo X, Hueber A, Bilyy R, Herrmann M, Zhao Y, Muñoz LE. Inosine Released from Dying or Dead Cells Stimulates Cell Proliferation . Front Immunol. 2017 Apr 27;8:504. doi: 10.3389/fimmu.2017.00504. eCollection 2017. PMID: 28496447; PMCID: PMC5406388.
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Oxidative Burst-Dependent NETosis Is Implicated in the Resolution of Necrosis-Associated Sterile Inflammation.

Frontiers in immunology

Biermann MH, Podolska MJ, Knopf J, Reinwald C, Weidner D, Maueröder C, Hahn J, Kienhöfer D, Barras A, Boukherroub R, Szunerits S, Bilyy R, Hoffmann M, Zhao Y, Schett G, Herrmann M, Munoz LE.
PMID: 27990145
Front Immunol. 2016 Dec 01;7:557. doi: 10.3389/fimmu.2016.00557. eCollection 2016.

Necrosis is associated with a profound inflammatory response. The regulation of necrosis-associated inflammation, particularly the mechanisms responsible for resolution of inflammation is incompletely characterized. Nanoparticles are known to induce plasma membrane damage and necrosis followed by sterile inflammation. We...

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Biermann MH, Podolska MJ, Knopf J, et al. Oxidative Burst-Dependent NETosis Is Implicated in the Resolution of Necrosis-Associated Sterile Inflammation. Front Immunol. 2016;7:557doi: 10.3389/fimmu.2016.00557.
Biermann, M. H., Podolska, M. J., Knopf, J., Reinwald, C., Weidner, D., Maueröder, C., Hahn, J., Kienhöfer, D., Barras, A., Boukherroub, R., Szunerits, S., Bilyy, R., Hoffmann, M., Zhao, Y., Schett, G., Herrmann, M., & Munoz, L. E. (2016). Oxidative Burst-Dependent NETosis Is Implicated in the Resolution of Necrosis-Associated Sterile Inflammation. Frontiers in immunology, 7557. https://doi.org/10.3389/fimmu.2016.00557
Biermann, Mona H C, et al. "Oxidative Burst-Dependent NETosis Is Implicated in the Resolution of Necrosis-Associated Sterile Inflammation." Frontiers in immunology vol. 7 (2016): 557. doi: https://doi.org/10.3389/fimmu.2016.00557
Biermann MH, Podolska MJ, Knopf J, Reinwald C, Weidner D, Maueröder C, Hahn J, Kienhöfer D, Barras A, Boukherroub R, Szunerits S, Bilyy R, Hoffmann M, Zhao Y, Schett G, Herrmann M, Munoz LE. Oxidative Burst-Dependent NETosis Is Implicated in the Resolution of Necrosis-Associated Sterile Inflammation. Front Immunol. 2016 Dec 01;7:557. doi: 10.3389/fimmu.2016.00557. eCollection 2016. PMID: 27990145; PMCID: PMC5131011.
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A20 deficiency in myeloid cells protects mice from diet-induced obesity and insulin resistance due to increased fatty acid metabolism.

Cell reports

Catrysse L, Maes B, Mehrotra P, Martens A, Hoste E, Martens L, Maueröder C, Remmerie A, Bujko A, Slowicka K, Sze M, Vikkula H, Ghesquière B, Scott CL, Saeys Y, van de Sluis B, Ravichandran K, Janssens S, van Loo G.
PMID: 34551300
Cell Rep. 2021 Sep 21;36(12):109748. doi: 10.1016/j.celrep.2021.109748.

Obesity-induced inflammation is a major driving force in the development of insulin resistance, type 2 diabetes (T2D), and related metabolic disorders. During obesity, macrophages accumulate in the visceral adipose tissue, creating a low-grade inflammatory environment. Nuclear factor κB (NF-κB)...

Cite
Catrysse L, Maes B, Mehrotra P, et al. A20 deficiency in myeloid cells protects mice from diet-induced obesity and insulin resistance due to increased fatty acid metabolism. Cell Rep. 2021;36(12):109748doi: 10.1016/j.celrep.2021.109748.
Catrysse, L., Maes, B., Mehrotra, P., Martens, A., Hoste, E., Martens, L., Maueröder, C., Remmerie, A., Bujko, A., Slowicka, K., Sze, M., Vikkula, H., Ghesquière, B., Scott, C. L., Saeys, Y., van de Sluis, B., Ravichandran, K., Janssens, S., & van Loo, G. (2021). A20 deficiency in myeloid cells protects mice from diet-induced obesity and insulin resistance due to increased fatty acid metabolism. Cell reports, 36(12), 109748. https://doi.org/10.1016/j.celrep.2021.109748
Catrysse, Leen, et al. "A20 deficiency in myeloid cells protects mice from diet-induced obesity and insulin resistance due to increased fatty acid metabolism." Cell reports vol. 36,12 (2021): 109748. doi: https://doi.org/10.1016/j.celrep.2021.109748
Catrysse L, Maes B, Mehrotra P, Martens A, Hoste E, Martens L, Maueröder C, Remmerie A, Bujko A, Slowicka K, Sze M, Vikkula H, Ghesquière B, Scott CL, Saeys Y, van de Sluis B, Ravichandran K, Janssens S, van Loo G. A20 deficiency in myeloid cells protects mice from diet-induced obesity and insulin resistance due to increased fatty acid metabolism. Cell Rep. 2021 Sep 21;36(12):109748. doi: 10.1016/j.celrep.2021.109748. PMID: 34551300.
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[Diagnostic Problemes Morbus Pick versus Depression].

Psychiatrische Praxis

Wolfersdorf M, Mauerer C, Schüler M, Purucker M, Groenewald FJ, Keller P.
PMID: 13130342
Psychiatr Prax. 2003 May;30:75-77. doi: 10.1055/s-2003-39740.

A case study based on the clinical picture and the short term course we describe diagnostic problems in a case of Morbus Pick.

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Wolfersdorf M, Mauerer C, Schüler M, et al. Differenzialdiagnostische Schwierigkeiten Morbus Pick versus Depression - Eine Kasuistik. [Diagnostic Problemes Morbus Pick versus Depression]. Psychiatr Prax. 2003;30:75-77doi: 10.1055/s-2003-39740.
Wolfersdorf, M., Mauerer, C., Schüler, M., Purucker, M., Groenewald, F. J., & Keller, P. (2003). Differenzialdiagnostische Schwierigkeiten Morbus Pick versus Depression - Eine Kasuistik. [Diagnostic Problemes Morbus Pick versus Depression]. Psychiatrische Praxis, 3075-77. https://doi.org/10.1055/s-2003-39740
Wolfersdorf, Manfred, et al. "Differenzialdiagnostische Schwierigkeiten Morbus Pick versus Depression - Eine Kasuistik." [Diagnostic Problemes Morbus Pick versus Depression]. Psychiatrische Praxis vol. 30 (2003): 75-77. doi: https://doi.org/10.1055/s-2003-39740
Wolfersdorf M, Mauerer C, Schüler M, Purucker M, Groenewald FJ, Keller P. Differenzialdiagnostische Schwierigkeiten Morbus Pick versus Depression - Eine Kasuistik. [Diagnostic Problemes Morbus Pick versus Depression]. Psychiatr Prax. 2003 May;30:75-77. doi: 10.1055/s-2003-39740. German. PMID: 13130342.
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Inflammatory etiopathogenesis of systemic lupus erythematosus: an update.

Journal of inflammation research

Podolska MJ, Biermann MH, Maueröder C, Hahn J, Herrmann M.
PMID: 26316795
J Inflamm Res. 2015 Aug 20;8:161-71. doi: 10.2147/JIR.S70325. eCollection 2015.

The immune system struggles every day between responding to foreign antigens and tolerating self-antigens to delicately maintain tissue homeostasis. If self-tolerance is broken, the development of autoimmunity can be the consequence, as it is in the case of the...

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Podolska MJ, Biermann MH, Maueröder C, et al. Inflammatory etiopathogenesis of systemic lupus erythematosus: an update. J Inflamm Res. 2015;8:161-71doi: 10.2147/JIR.S70325.
Podolska, M. J., Biermann, M. H., Maueröder, C., Hahn, J., & Herrmann, M. (2015). Inflammatory etiopathogenesis of systemic lupus erythematosus: an update. Journal of inflammation research, 8161-71. https://doi.org/10.2147/JIR.S70325
Podolska, Malgorzata J, et al. "Inflammatory etiopathogenesis of systemic lupus erythematosus: an update." Journal of inflammation research vol. 8 (2015): 161-71. doi: https://doi.org/10.2147/JIR.S70325
Podolska MJ, Biermann MH, Maueröder C, Hahn J, Herrmann M. Inflammatory etiopathogenesis of systemic lupus erythematosus: an update. J Inflamm Res. 2015 Aug 20;8:161-71. doi: 10.2147/JIR.S70325. eCollection 2015. PMID: 26316795; PMCID: PMC4548750.
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