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LINGO-1 promotes lysosomal degradation of amyloid-β protein precursor.

Pathobiology of aging & age related diseases

de Laat R, Meabon JS, Wiley JC, Hudson MP, Montine TJ, Bothwell M.
PMID: 25758563
Pathobiol Aging Age Relat Dis. 2015 Mar 09;5:25796. doi: 10.3402/pba.v5.25796. eCollection 2015.

Sequential proteolytic cleavages of amyloid-β protein precursor (AβPP) by β-secretase and γ-secretase generate amyloid β (Aβ) peptides, which are thought to contribute to Alzheimer's disease (AD). Much of this processing occurs in endosomes following endocytosis of AβPP from the...

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de Laat R, Meabon JS, Wiley JC, et al. LINGO-1 promotes lysosomal degradation of amyloid-β protein precursor. Pathobiol Aging Age Relat Dis. 2015;5:25796doi: 10.3402/pba.v5.25796.
de Laat, R., Meabon, J. S., Wiley, J. C., Hudson, M. P., Montine, T. J., & Bothwell, M. (2015). LINGO-1 promotes lysosomal degradation of amyloid-β protein precursor. Pathobiology of aging & age related diseases, 525796. https://doi.org/10.3402/pba.v5.25796
de Laat, Rian, et al. "LINGO-1 promotes lysosomal degradation of amyloid-β protein precursor." Pathobiology of aging & age related diseases vol. 5 (2015): 25796. doi: https://doi.org/10.3402/pba.v5.25796
de Laat R, Meabon JS, Wiley JC, Hudson MP, Montine TJ, Bothwell M. LINGO-1 promotes lysosomal degradation of amyloid-β protein precursor. Pathobiol Aging Age Relat Dis. 2015 Mar 09;5:25796. doi: 10.3402/pba.v5.25796. eCollection 2015. PMID: 25758563; PMCID: PMC4355507.
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Obesity and inflammation markers in relation to leukocyte telomere length in a cross-sectional study of persons with Barrett's esophagus.

BMC obesity

Hardikar S, Song X, Risques RA, Montine TJ, Duggan C, Blount PL, Reid BJ, Anderson GL, Kratz M, White E, Vaughan TL.
PMID: 26380096
BMC Obes. 2015 Sep 10;2:32. doi: 10.1186/s40608-015-0063-3. eCollection 2015.

BACKGROUND: Telomere shortening is associated with increasing age, male gender and lifestyle factors such as obesity and smoking. Inflammation has also been implicated in cellular senescence and may promote telomere shortening in chronic conditions such as obesity and diabetes....

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Hardikar S, Song X, Risques RA, et al. Obesity and inflammation markers in relation to leukocyte telomere length in a cross-sectional study of persons with Barrett's esophagus. BMC Obes. 2015;2:32doi: 10.1186/s40608-015-0063-3.
Hardikar, S., Song, X., Risques, R. A., Montine, T. J., Duggan, C., Blount, P. L., Reid, B. J., Anderson, G. L., Kratz, M., White, E., & Vaughan, T. L. (2015). Obesity and inflammation markers in relation to leukocyte telomere length in a cross-sectional study of persons with Barrett's esophagus. BMC obesity, 232. https://doi.org/10.1186/s40608-015-0063-3
Hardikar, Sheetal, et al. "Obesity and inflammation markers in relation to leukocyte telomere length in a cross-sectional study of persons with Barrett's esophagus." BMC obesity vol. 2 (2015): 32. doi: https://doi.org/10.1186/s40608-015-0063-3
Hardikar S, Song X, Risques RA, Montine TJ, Duggan C, Blount PL, Reid BJ, Anderson GL, Kratz M, White E, Vaughan TL. Obesity and inflammation markers in relation to leukocyte telomere length in a cross-sectional study of persons with Barrett's esophagus. BMC Obes. 2015 Sep 10;2:32. doi: 10.1186/s40608-015-0063-3. eCollection 2015. PMID: 26380096; PMCID: PMC4566310.
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Surface phases and photocatalytic activity correlation of Bi2O3/Bi2O4-x nanocomposite.

Journal of the American Chemical Society

Hameed A, Montini T, Gombac V, Fornasiero P.
PMID: 18598025
J Am Chem Soc. 2008 Jul 30;130(30):9658-9. doi: 10.1021/ja803603y. Epub 2008 Jul 04.

UV-visible irradiation induces surface alteration of Bi2O3 leading to Bi2O3/Bi2O4-x nanocomposites with excellent photocatalytic activity.

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Hameed A, Montini T, Gombac V, et al. Surface phases and photocatalytic activity correlation of Bi2O3/Bi2O4-x nanocomposite. J Am Chem Soc. 2008;130(30):9658-9doi: 10.1021/ja803603y.
Hameed, A., Montini, T., Gombac, V., & Fornasiero, P. (2008). Surface phases and photocatalytic activity correlation of Bi2O3/Bi2O4-x nanocomposite. Journal of the American Chemical Society, 130(30), 9658-9. https://doi.org/10.1021/ja803603y
Hameed, Abdul, et al. "Surface phases and photocatalytic activity correlation of Bi2O3/Bi2O4-x nanocomposite." Journal of the American Chemical Society vol. 130,30 (2008): 9658-9. doi: https://doi.org/10.1021/ja803603y
Hameed A, Montini T, Gombac V, Fornasiero P. Surface phases and photocatalytic activity correlation of Bi2O3/Bi2O4-x nanocomposite. J Am Chem Soc. 2008 Jul 30;130(30):9658-9. doi: 10.1021/ja803603y. Epub 2008 Jul 04. PMID: 18598025.
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Pd-Dissolution through a mild and effective one-step reaction and its application for Pd-recovery from spent catalytic converters.

Chemical communications (Cambridge, England)

Serpe A, Bigoli F, Cabras MC, Fornasiero P, Graziani M, Mercuri ML, Montini T, Pilia L, Trogu EF, Deplano P.
PMID: 15719109
Chem Commun (Camb). 2005 Feb 28;(8):1040-2. doi: 10.1039/b415799k. Epub 2005 Jan 10.

We describe here an innovative method for Pd-dissolution using the reagent N,N'-dimethylperhydrodiazepine-2,3-dithione diiodine adduct which, being safe and powerful, is appealing for practical applications: remarkably almost quantitative Pd-recovery from model spent three-way catalysts has been obtained, showing that the...

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Serpe A, Bigoli F, Cabras MC, et al. Pd-Dissolution through a mild and effective one-step reaction and its application for Pd-recovery from spent catalytic converters. Chem Commun (Camb). 2005;(8):1040-2doi: 10.1039/b415799k.
Serpe, A., Bigoli, F., Cabras, M. C., Fornasiero, P., Graziani, M., Mercuri, M. L., Montini, T., Pilia, L., Trogu, E. F., & Deplano, P. (2005). Pd-Dissolution through a mild and effective one-step reaction and its application for Pd-recovery from spent catalytic converters. Chemical communications (Cambridge, England), (8), 1040-2. https://doi.org/10.1039/b415799k
Serpe, Angela, et al. "Pd-Dissolution through a mild and effective one-step reaction and its application for Pd-recovery from spent catalytic converters." Chemical communications (Cambridge, England) vol. ,8 (2005): 1040-2. doi: https://doi.org/10.1039/b415799k
Serpe A, Bigoli F, Cabras MC, Fornasiero P, Graziani M, Mercuri ML, Montini T, Pilia L, Trogu EF, Deplano P. Pd-Dissolution through a mild and effective one-step reaction and its application for Pd-recovery from spent catalytic converters. Chem Commun (Camb). 2005 Feb 28;(8):1040-2. doi: 10.1039/b415799k. Epub 2005 Jan 10. PMID: 15719109.
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Behaviour of ?-methylserotonin in rat brain synaptosomes.

Neurochemistry international

Montine TJ, Sourkes TL.
PMID: 20504488
Neurochem Int. 1989;15(2):227-31. doi: 10.1016/0197-0186(89)90105-8.

Twenty-four h after administration of ?-methyl-dl-tryptophan (AMTP) to rats intrasynaptosomal ?-methylserotonin (AM5HT) levels were 5.5-fold greater than those of serotonin (5HT) in control animals, and were accompanied by a 90% reduction in 5HT concentrations. Behaviour of AM5HT in nerve...

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Montine TJ, Sourkes TL. Behaviour of ?-methylserotonin in rat brain synaptosomes. Neurochem Int. 1989;15(2):227-31doi: 10.1016/0197-0186(89)90105-8.
Montine, T. J., & Sourkes, T. L. (1989). Behaviour of ?-methylserotonin in rat brain synaptosomes. Neurochemistry international, 15(2), 227-31. https://doi.org/10.1016/0197-0186(89)90105-8
Montine, T J, and Sourkes, T L. "Behaviour of ?-methylserotonin in rat brain synaptosomes." Neurochemistry international vol. 15,2 (1989): 227-31. doi: https://doi.org/10.1016/0197-0186(89)90105-8
Montine TJ, Sourkes TL. Behaviour of ?-methylserotonin in rat brain synaptosomes. Neurochem Int. 1989;15(2):227-31. doi: 10.1016/0197-0186(89)90105-8. PMID: 20504488.
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Stress and the inflammatory process: a major cause of pancreatic cell death in type 2 diabetes.

Diabetes, metabolic syndrome and obesity : targets and therapy

Montane J, Cadavez L, Novials A.
PMID: 24520198
Diabetes Metab Syndr Obes. 2014 Feb 03;7:25-34. doi: 10.2147/DMSO.S37649. eCollection 2014.

Type 2 diabetes (T2D) is a complex metabolic disorder characterized by hyperglycemia in the context of insulin resistance, which precedes insulin deficiency as a result of β-cell failure. Accumulating evidence indicates that β-cell loss in T2D results as a...

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Montane J, Cadavez L, Novials A. Stress and the inflammatory process: a major cause of pancreatic cell death in type 2 diabetes. Diabetes Metab Syndr Obes. 2014;7:25-34doi: 10.2147/DMSO.S37649.
Montane, J., Cadavez, L., & Novials, A. (2014). Stress and the inflammatory process: a major cause of pancreatic cell death in type 2 diabetes. Diabetes, metabolic syndrome and obesity : targets and therapy, 725-34. https://doi.org/10.2147/DMSO.S37649
Montane, Joel, et al. "Stress and the inflammatory process: a major cause of pancreatic cell death in type 2 diabetes." Diabetes, metabolic syndrome and obesity : targets and therapy vol. 7 (2014): 25-34. doi: https://doi.org/10.2147/DMSO.S37649
Montane J, Cadavez L, Novials A. Stress and the inflammatory process: a major cause of pancreatic cell death in type 2 diabetes. Diabetes Metab Syndr Obes. 2014 Feb 03;7:25-34. doi: 10.2147/DMSO.S37649. eCollection 2014. PMID: 24520198; PMCID: PMC3917922.
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The contradictory effect of the methoxy-substituent in palladium-catalyzed ethylene/methyl acrylate cooligomerization.

Dalton transactions (Cambridge, England : 2003)

Rosar V, Meduri A, Montini T, Fornasiero P, Zangrando E, Milani B.
PMID: 29417113
Dalton Trans. 2018 Feb 20;47(8):2778-2790. doi: 10.1039/c7dt04465h.

Two new nonsymmetric bis(aryl-imino)acenaphthene ligands (Ar,Ar'-BIAN) and one symmetric Ar

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Rosar V, Meduri A, Montini T, et al. The contradictory effect of the methoxy-substituent in palladium-catalyzed ethylene/methyl acrylate cooligomerization. Dalton Trans. 2018;47(8):2778-2790doi: 10.1039/c7dt04465h.
Rosar, V., Meduri, A., Montini, T., Fornasiero, P., Zangrando, E., & Milani, B. (2018). The contradictory effect of the methoxy-substituent in palladium-catalyzed ethylene/methyl acrylate cooligomerization. Dalton transactions (Cambridge, England : 2003), 47(8), 2778-2790. https://doi.org/10.1039/c7dt04465h
Rosar, V, et al. "The contradictory effect of the methoxy-substituent in palladium-catalyzed ethylene/methyl acrylate cooligomerization." Dalton transactions (Cambridge, England : 2003) vol. 47,8 (2018): 2778-2790. doi: https://doi.org/10.1039/c7dt04465h
Rosar V, Meduri A, Montini T, Fornasiero P, Zangrando E, Milani B. The contradictory effect of the methoxy-substituent in palladium-catalyzed ethylene/methyl acrylate cooligomerization. Dalton Trans. 2018 Feb 20;47(8):2778-2790. doi: 10.1039/c7dt04465h. PMID: 29417113.
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Cross-Linked Carbon Nanotube Adsorbents for Water Treatment: Tuning the Sorption Capacity through Chemical Functionalization.

ACS applied materials & interfaces

Barrejón M, Syrgiannis Z, Burian M, Bosi S, Montini T, Fornasiero P, Amenitsch H, Prato M.
PMID: 30844229
ACS Appl Mater Interfaces. 2019 Apr 03;11(13):12920-12930. doi: 10.1021/acsami.8b20557. Epub 2019 Mar 20.

The development of carbon-based membrane adsorbent materials for water treatment has become a hot topic in recent years. Among them, carbon nanotubes (CNTs) are promising materials because of its large surface area, high aspect ratio, great chemical reactivity, and...

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Barrejón M, Syrgiannis Z, Burian M, et al. Cross-Linked Carbon Nanotube Adsorbents for Water Treatment: Tuning the Sorption Capacity through Chemical Functionalization. ACS Appl Mater Interfaces. 2019;11(13):12920-12930doi: 10.1021/acsami.8b20557.
Barrejón, M., Syrgiannis, Z., Burian, M., Bosi, S., Montini, T., Fornasiero, P., Amenitsch, H., & Prato, M. (2019). Cross-Linked Carbon Nanotube Adsorbents for Water Treatment: Tuning the Sorption Capacity through Chemical Functionalization. ACS applied materials & interfaces, 11(13), 12920-12930. https://doi.org/10.1021/acsami.8b20557
Barrejón, Myriam, et al. "Cross-Linked Carbon Nanotube Adsorbents for Water Treatment: Tuning the Sorption Capacity through Chemical Functionalization." ACS applied materials & interfaces vol. 11,13 (2019): 12920-12930. doi: https://doi.org/10.1021/acsami.8b20557
Barrejón M, Syrgiannis Z, Burian M, Bosi S, Montini T, Fornasiero P, Amenitsch H, Prato M. Cross-Linked Carbon Nanotube Adsorbents for Water Treatment: Tuning the Sorption Capacity through Chemical Functionalization. ACS Appl Mater Interfaces. 2019 Apr 03;11(13):12920-12930. doi: 10.1021/acsami.8b20557. Epub 2019 Mar 20. PMID: 30844229.
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Flow cytometric evaluation of crude synaptosome preparation as a way to study synaptic alteration in neurodegenerative diseases.

Neuromethods

Postupna NO, Latimer CS, Keene CD, Montine KS, Montine TJ, Darvas M.
PMID: 32139977
Neuromethods. 2018;141:297-310. doi: 10.1007/978-1-4939-8739-9_17. Epub 2018 Sep 22.

Neurodegenerative diseases, the most common among them Alzheimer's disease (AD) and Lewy body disease (LBD), are a group of progressive incurable illnesses. In both AD and LBD, abundant evidence points to the synapse as the critical and early focus...

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Postupna NO, Latimer CS, Keene CD, et al. Flow cytometric evaluation of crude synaptosome preparation as a way to study synaptic alteration in neurodegenerative diseases. Neuromethods. 2018;141:297-310doi: 10.1007/978-1-4939-8739-9_17.
Postupna, N. O., Latimer, C. S., Keene, C. D., Montine, K. S., Montine, T. J., & Darvas, M. (2018). Flow cytometric evaluation of crude synaptosome preparation as a way to study synaptic alteration in neurodegenerative diseases. Neuromethods, 141297-310. https://doi.org/10.1007/978-1-4939-8739-9_17
Postupna, Nadia O, et al. "Flow cytometric evaluation of crude synaptosome preparation as a way to study synaptic alteration in neurodegenerative diseases." Neuromethods vol. 141 (2018): 297-310. doi: https://doi.org/10.1007/978-1-4939-8739-9_17
Postupna NO, Latimer CS, Keene CD, Montine KS, Montine TJ, Darvas M. Flow cytometric evaluation of crude synaptosome preparation as a way to study synaptic alteration in neurodegenerative diseases. Neuromethods. 2018;141:297-310. doi: 10.1007/978-1-4939-8739-9_17. Epub 2018 Sep 22. PMID: 32139977; PMCID: PMC7058378.
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Genome-wide association study and functional validation implicates JADE1 in tauopathy.

Acta neuropathologica

Farrell K, Kim S, Han N, Iida MA, Gonzalez EM, Otero-Garcia M, Walker JM, Richardson TE, Renton AE, Andrews SJ, Fulton-Howard B, Humphrey J, Vialle RA, Bowles KR, de Paiva Lopes K, Whitney K, Dangoor DK, Walsh H, Marcora E, Hefti MM, Casella A, Sissoko CT, Kapoor M, Novikova G, Udine E, Wong G, Tang W, Bhangale T, Hunkapiller J, Ayalon G, Graham RR, Cherry JD, Cortes EP, Borukov VY, McKee AC, Stein TD, Vonsattel JP, Teich AF, Gearing M, Glass J, Troncoso JC, Frosch MP, Hyman BT, Dickson DW, Murray ME, Attems J, Flanagan ME, Mao Q, Mesulam MM, Weintraub S, Woltjer RL, Pham T, Kofler J, Schneider JA, Yu L, Purohit DP, Haroutunian V, Hof PR, Gandy S, Sano M, Beach TG, Poon W, Kawas CH, Corrada MM, Rissman RA, Metcalf J, Shuldberg S, Salehi B, Nelson PT, Trojanowski JQ, Lee EB, Wolk DA, McMillan CT, Keene CD, Latimer CS, Montine TJ, Kovacs GG, Lutz MI, Fischer P, Perrin RJ, Cairns NJ, Franklin EE, Cohen HT, Raj T, Cobos I, Frost B, Goate A, White Iii CL, Crary JF.
PMID: 34719765
Acta Neuropathol. 2022 Jan;143(1):33-53. doi: 10.1007/s00401-021-02379-z. Epub 2021 Nov 01.

Primary age-related tauopathy (PART) is a neurodegenerative pathology with features distinct from but also overlapping with Alzheimer disease (AD). While both exhibit Alzheimer-type temporal lobe neurofibrillary degeneration alongside amnestic cognitive impairment, PART develops independently of amyloid-β (Aβ) plaques. The...

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Farrell K, Kim S, Han N, et al. Genome-wide association study and functional validation implicates JADE1 in tauopathy. Acta Neuropathol. 2021;143(1):33-53doi: 10.1007/s00401-021-02379-z.
Farrell, K., Kim, S., Han, N., Iida, M. A., Gonzalez, E. M., Otero-Garcia, M., Walker, J. M., Richardson, T. E., Renton, A. E., Andrews, S. J., Fulton-Howard, B., Humphrey, J., Vialle, R. A., Bowles, K. R., de Paiva Lopes, K., Whitney, K., Dangoor, D. K., Walsh, H., Marcora, E., Hefti, M. M., Casella, A., Sissoko, C. T., Kapoor, M., Novikova, G., Udine, E., Wong, G., Tang, W., Bhangale, T., Hunkapiller, J., Ayalon, G., Graham, R. R., Cherry, J. D., Cortes, E. P., Borukov, V. Y., McKee, A. C., Stein, T. D., Vonsattel, J. P., Teich, A. F., Gearing, M., Glass, J., Troncoso, J. C., Frosch, M. P., Hyman, B. T., Dickson, D. W., Murray, M. E., Attems, J., Flanagan, M. E., Mao, Q., Mesulam, M. M., Weintraub, S., Woltjer, R. L., Pham, T., Kofler, J., Schneider, J. A., Yu, L., Purohit, D. P., Haroutunian, V., Hof, P. R., Gandy, S., Sano, M., Beach, T. G., Poon, W., Kawas, C. H., Corrada, M. M., Rissman, R. A., Metcalf, J., Shuldberg, S., Salehi, B., Nelson, P. T., Trojanowski, J. Q., Lee, E. B., Wolk, D. A., McMillan, C. T., Keene, C. D., Latimer, C. S., Montine, T. J., Kovacs, G. G., Lutz, M. I., Fischer, P., Perrin, R. J., Cairns, N. J., Franklin, E. E., Cohen, H. T., Raj, T., Cobos, I., Frost, B., Goate, A., White Iii, C. L., & Crary, J. F. (2022). Genome-wide association study and functional validation implicates JADE1 in tauopathy. Acta neuropathologica, 143(1), 33-53. https://doi.org/10.1007/s00401-021-02379-z
Farrell, Kurt, et al. "Genome-wide association study and functional validation implicates JADE1 in tauopathy." Acta neuropathologica vol. 143,1 (2022): 33-53. doi: https://doi.org/10.1007/s00401-021-02379-z
Farrell K, Kim S, Han N, Iida MA, Gonzalez EM, Otero-Garcia M, Walker JM, Richardson TE, Renton AE, Andrews SJ, Fulton-Howard B, Humphrey J, Vialle RA, Bowles KR, de Paiva Lopes K, Whitney K, Dangoor DK, Walsh H, Marcora E, Hefti MM, Casella A, Sissoko CT, Kapoor M, Novikova G, Udine E, Wong G, Tang W, Bhangale T, Hunkapiller J, Ayalon G, Graham RR, Cherry JD, Cortes EP, Borukov VY, McKee AC, Stein TD, Vonsattel JP, Teich AF, Gearing M, Glass J, Troncoso JC, Frosch MP, Hyman BT, Dickson DW, Murray ME, Attems J, Flanagan ME, Mao Q, Mesulam MM, Weintraub S, Woltjer RL, Pham T, Kofler J, Schneider JA, Yu L, Purohit DP, Haroutunian V, Hof PR, Gandy S, Sano M, Beach TG, Poon W, Kawas CH, Corrada MM, Rissman RA, Metcalf J, Shuldberg S, Salehi B, Nelson PT, Trojanowski JQ, Lee EB, Wolk DA, McMillan CT, Keene CD, Latimer CS, Montine TJ, Kovacs GG, Lutz MI, Fischer P, Perrin RJ, Cairns NJ, Franklin EE, Cohen HT, Raj T, Cobos I, Frost B, Goate A, White Iii CL, Crary JF. Genome-wide association study and functional validation implicates JADE1 in tauopathy. Acta Neuropathol. 2022 Jan;143(1):33-53. doi: 10.1007/s00401-021-02379-z. Epub 2021 Nov 01. PMID: 34719765.
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Discovery of G2019S-Selective Leucine Rich Repeat Protein Kinase 2 inhibitors with in vivo efficacy.

European journal of medicinal chemistry

Leśniak RK, Nichols RJ, Schonemann M, Zhao J, Gajera CR, Fitch WL, Lam G, Nguyen KC, Smith M, Montine TJ.
PMID: 34992038
Eur J Med Chem. 2021 Dec 28;229:114080. doi: 10.1016/j.ejmech.2021.114080. Epub 2021 Dec 28.

Mutations in the Leucine Rich Repeat Protein Kinase 2 gene (LRRK2) are the most common genetic causes of Parkinson's Disease (PD). The G2019S mutation is the most common inherited LRRK2 mutation, occurs in the kinase domain, and results in...

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Leśniak RK, Nichols RJ, Schonemann M, et al. Discovery of G2019S-Selective Leucine Rich Repeat Protein Kinase 2 inhibitors with in vivo efficacy. Eur J Med Chem. 2021;229:114080doi: 10.1016/j.ejmech.2021.114080.
Leśniak, R. K., Nichols, R. J., Schonemann, M., Zhao, J., Gajera, C. R., Fitch, W. L., Lam, G., Nguyen, K. C., Smith, M., & Montine, T. J. (2021). Discovery of G2019S-Selective Leucine Rich Repeat Protein Kinase 2 inhibitors with in vivo efficacy. European journal of medicinal chemistry, 229114080. https://doi.org/10.1016/j.ejmech.2021.114080
Leśniak, Robert K, et al. "Discovery of G2019S-Selective Leucine Rich Repeat Protein Kinase 2 inhibitors with in vivo efficacy." European journal of medicinal chemistry vol. 229 (2021): 114080. doi: https://doi.org/10.1016/j.ejmech.2021.114080
Leśniak RK, Nichols RJ, Schonemann M, Zhao J, Gajera CR, Fitch WL, Lam G, Nguyen KC, Smith M, Montine TJ. Discovery of G2019S-Selective Leucine Rich Repeat Protein Kinase 2 inhibitors with in vivo efficacy. Eur J Med Chem. 2021 Dec 28;229:114080. doi: 10.1016/j.ejmech.2021.114080. Epub 2021 Dec 28. PMID: 34992038.
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Urinary metabolomic signatures as indicators of injury severity following traumatic brain injury: A pilot study.

IBRO neuroscience reports

Bykowski EA, Petersson JN, Dukelow S, Ho C, Debert CT, Montina T, Metz GAS.
PMID: 34786572
IBRO Neurosci Rep. 2021 Oct 27;11:200-206. doi: 10.1016/j.ibneur.2021.10.003. eCollection 2021 Dec.

BACKGROUND: Analysis of fluid metabolites has the potential to provide insight into the neuropathophysiology of injury in patients with traumatic brain injury (TBI).OBJECTIVE: Using a METHODS: Urine samples were collected within 7 days and at 6-months post-injury in male...

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Bykowski EA, Petersson JN, Dukelow S, et al. Urinary metabolomic signatures as indicators of injury severity following traumatic brain injury: A pilot study. IBRO Neurosci Rep. 2021;11:200-206doi: 10.1016/j.ibneur.2021.10.003.
Bykowski, E. A., Petersson, J. N., Dukelow, S., Ho, C., Debert, C. T., Montina, T., & Metz, G. A. S. (2021). Urinary metabolomic signatures as indicators of injury severity following traumatic brain injury: A pilot study. IBRO neuroscience reports, 11200-206. https://doi.org/10.1016/j.ibneur.2021.10.003
Bykowski, Elani A, et al. "Urinary metabolomic signatures as indicators of injury severity following traumatic brain injury: A pilot study." IBRO neuroscience reports vol. 11 (2021): 200-206. doi: https://doi.org/10.1016/j.ibneur.2021.10.003
Bykowski EA, Petersson JN, Dukelow S, Ho C, Debert CT, Montina T, Metz GAS. Urinary metabolomic signatures as indicators of injury severity following traumatic brain injury: A pilot study. IBRO Neurosci Rep. 2021 Oct 27;11:200-206. doi: 10.1016/j.ibneur.2021.10.003. eCollection 2021 Dec. PMID: 34786572; PMCID: PMC8578034.
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Showing 1 to 12 of 104 entries