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Right-sided subcutaneous implantable cardioverter-defibrillator placement in a patient with dextrocardia, tetralogy of Fallot, and conduction disease.

HeartRhythm case reports

Ceresnak SR, Motonaga KS, Rogers IS, Viswanathan MN.
PMID: 28491545
HeartRhythm Case Rep. 2015 Jun 12;1(4):186-189. doi: 10.1016/j.hrcr.2015.02.001. eCollection 2015 Jul.

No abstract available.

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Ceresnak SR, Motonaga KS, Rogers IS, et al. Right-sided subcutaneous implantable cardioverter-defibrillator placement in a patient with dextrocardia, tetralogy of Fallot, and conduction disease. HeartRhythm Case Rep. 2015;1(4):186-189doi: 10.1016/j.hrcr.2015.02.001.
Ceresnak, S. R., Motonaga, K. S., Rogers, I. S., & Viswanathan, M. N. (2015). Right-sided subcutaneous implantable cardioverter-defibrillator placement in a patient with dextrocardia, tetralogy of Fallot, and conduction disease. HeartRhythm case reports, 1(4), 186-189. https://doi.org/10.1016/j.hrcr.2015.02.001
Ceresnak, Scott R, et al. "Right-sided subcutaneous implantable cardioverter-defibrillator placement in a patient with dextrocardia, tetralogy of Fallot, and conduction disease." HeartRhythm case reports vol. 1,4 (2015): 186-189. doi: https://doi.org/10.1016/j.hrcr.2015.02.001
Ceresnak SR, Motonaga KS, Rogers IS, Viswanathan MN. Right-sided subcutaneous implantable cardioverter-defibrillator placement in a patient with dextrocardia, tetralogy of Fallot, and conduction disease. HeartRhythm Case Rep. 2015 Jun 12;1(4):186-189. doi: 10.1016/j.hrcr.2015.02.001. eCollection 2015 Jul. PMID: 28491545; PMCID: PMC5419248.
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Computing handedness: quantized and superposed switch and dynamic memory of helical polysilylene.

Journal of the American Chemical Society

Fujiki M, Koe JR, Motonaga M, Nakashima H, Terao K, Teramoto A.
PMID: 11427048
J Am Chem Soc. 2001 Jul 04;123(26):6253-61. doi: 10.1021/ja0026509.

Two new conjugating helical polymers comprising a rodlike silicon backbone and enantiopure chiral pendants, poly[(R)-3,7-dimethyloctyl-(S)-3-methylpentylsilylene] (PS-1) and its diastereomeric poly[(S)-3,7-dimethyloctyl-(S)-3-methylpentylsilylene] (PS-2), were prepared. Molecular mechanics calculations of PS-1 and PS-2 model oligomers indicated a double well potential energy curve...

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Fujiki M, Koe JR, Motonaga M, et al. Computing handedness: quantized and superposed switch and dynamic memory of helical polysilylene. J Am Chem Soc. 2001;123(26):6253-61doi: 10.1021/ja0026509.
Fujiki, M., Koe, J. R., Motonaga, M., Nakashima, H., Terao, K., & Teramoto, A. (2001). Computing handedness: quantized and superposed switch and dynamic memory of helical polysilylene. Journal of the American Chemical Society, 123(26), 6253-61. https://doi.org/10.1021/ja0026509
Fujiki, M, et al. "Computing handedness: quantized and superposed switch and dynamic memory of helical polysilylene." Journal of the American Chemical Society vol. 123,26 (2001): 6253-61. doi: https://doi.org/10.1021/ja0026509
Fujiki M, Koe JR, Motonaga M, Nakashima H, Terao K, Teramoto A. Computing handedness: quantized and superposed switch and dynamic memory of helical polysilylene. J Am Chem Soc. 2001 Jul 04;123(26):6253-61. doi: 10.1021/ja0026509. PMID: 11427048.
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Solvent and temperature effects on the chiral aggregation of poly(alkylarylsilane)s bearing remote chiral groups.

Journal of the American Chemical Society

Nakashima H, Fujiki M, Koe JR, Motonaga M.
PMID: 11456817
J Am Chem Soc. 2001 Mar 07;123(9):1963-9. doi: 10.1021/ja000869h.

Novel switchable chiroptical characteristics of poly(alkylarylsilane) microaggregates, controllable by the choice of good/poor solvent ratio (solvent polarity), solvent addition order, and sample temperature are described. The formation of stable chiral aggregates depends critically on the polysilane structure and stereochemistry....

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Nakashima H, Fujiki M, Koe JR, et al. Solvent and temperature effects on the chiral aggregation of poly(alkylarylsilane)s bearing remote chiral groups. J Am Chem Soc. 2001;123(9):1963-9doi: 10.1021/ja000869h.
Nakashima, H., Fujiki, M., Koe, J. R., & Motonaga, M. (2001). Solvent and temperature effects on the chiral aggregation of poly(alkylarylsilane)s bearing remote chiral groups. Journal of the American Chemical Society, 123(9), 1963-9. https://doi.org/10.1021/ja000869h
Nakashima, H, et al. "Solvent and temperature effects on the chiral aggregation of poly(alkylarylsilane)s bearing remote chiral groups." Journal of the American Chemical Society vol. 123,9 (2001): 1963-9. doi: https://doi.org/10.1021/ja000869h
Nakashima H, Fujiki M, Koe JR, Motonaga M. Solvent and temperature effects on the chiral aggregation of poly(alkylarylsilane)s bearing remote chiral groups. J Am Chem Soc. 2001 Mar 07;123(9):1963-9. doi: 10.1021/ja000869h. PMID: 11456817.
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Combined treatment with DPP-4 inhibitor linagliptin and SGLT2 inhibitor empagliflozin attenuates neointima formation after vascular injury in diabetic mice.

Biochemistry and biophysics reports

Takahashi H, Nomiyama T, Terawaki Y, Horikawa T, Kawanami T, Hamaguchi Y, Tanaka T, Motonaga R, Fukuda T, Tanabe M, Yanase T.
PMID: 31032431
Biochem Biophys Rep. 2019 Apr 19;18:100640. doi: 10.1016/j.bbrep.2019.100640. eCollection 2019 Jul.

Incretin therapy has emerged as one of the most popular medications for type 2 diabetes. We have previously reported that the dipeptidyl peptidase-4 (DPP-4) inhibitor linagliptin attenuates neointima formation after vascular injury in non-diabetic mice. In the present study,...

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Takahashi H, Nomiyama T, Terawaki Y, et al. Combined treatment with DPP-4 inhibitor linagliptin and SGLT2 inhibitor empagliflozin attenuates neointima formation after vascular injury in diabetic mice. Biochem Biophys Rep. 2019;18:100640doi: 10.1016/j.bbrep.2019.100640.
Takahashi, H., Nomiyama, T., Terawaki, Y., Horikawa, T., Kawanami, T., Hamaguchi, Y., Tanaka, T., Motonaga, R., Fukuda, T., Tanabe, M., & Yanase, T. (2019). Combined treatment with DPP-4 inhibitor linagliptin and SGLT2 inhibitor empagliflozin attenuates neointima formation after vascular injury in diabetic mice. Biochemistry and biophysics reports, 18100640. https://doi.org/10.1016/j.bbrep.2019.100640
Takahashi, Hiroyuki, et al. "Combined treatment with DPP-4 inhibitor linagliptin and SGLT2 inhibitor empagliflozin attenuates neointima formation after vascular injury in diabetic mice." Biochemistry and biophysics reports vol. 18 (2019): 100640. doi: https://doi.org/10.1016/j.bbrep.2019.100640
Takahashi H, Nomiyama T, Terawaki Y, Horikawa T, Kawanami T, Hamaguchi Y, Tanaka T, Motonaga R, Fukuda T, Tanabe M, Yanase T. Combined treatment with DPP-4 inhibitor linagliptin and SGLT2 inhibitor empagliflozin attenuates neointima formation after vascular injury in diabetic mice. Biochem Biophys Rep. 2019 Apr 19;18:100640. doi: 10.1016/j.bbrep.2019.100640. eCollection 2019 Jul. PMID: 31032431; PMCID: PMC6477163.
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Mono- and Biallelic Protein-Truncating Variants in Alpha-Actinin 2 Cause Cardiomyopathy Through Distinct Mechanisms.

Circulation. Genomic and precision medicine

Lindholm ME, Jimenez-Morales D, Zhu H, Seo K, Amar D, Zhao C, Raja A, Madhvani R, Abramowitz S, Espenel C, Sutton S, Caleshu C, Berry GJ, Motonaga KS, Dunn K, Platt J, Ashley EA, Wheeler MT.
PMID: 34802252
Circ Genom Precis Med. 2021 Nov 22;CIRCGEN121003419. doi: 10.1161/CIRCGEN.121.003419. Epub 2021 Nov 22.

BACKGROUND: ACTN2 (alpha-actinin 2) anchors actin within cardiac sarcomeres. The mechanisms linking METHODS: Patients harboring ACTN2 protein-truncating variants were identified using a custom mutation pipeline. In patient-derived iPSC-cardiomyocytes, we investigated transcriptional profiles using RNA sequencing, contractile properties using video-based...

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Lindholm ME, Jimenez-Morales D, Zhu H, et al. Mono- and Biallelic Protein-Truncating Variants in Alpha-Actinin 2 Cause Cardiomyopathy Through Distinct Mechanisms. Circ Genom Precis Med. 2021;CIRCGEN121003419doi: 10.1161/CIRCGEN.121.003419.
Lindholm, M. E., Jimenez-Morales, D., Zhu, H., Seo, K., Amar, D., Zhao, C., Raja, A., Madhvani, R., Abramowitz, S., Espenel, C., Sutton, S., Caleshu, C., Berry, G. J., Motonaga, K. S., Dunn, K., Platt, J., Ashley, E. A., & Wheeler, M. T. (2021). Mono- and Biallelic Protein-Truncating Variants in Alpha-Actinin 2 Cause Cardiomyopathy Through Distinct Mechanisms. Circulation. Genomic and precision medicine, CIRCGEN121003419. https://doi.org/10.1161/CIRCGEN.121.003419
Lindholm, Malene E, et al. "Mono- and Biallelic Protein-Truncating Variants in Alpha-Actinin 2 Cause Cardiomyopathy Through Distinct Mechanisms." Circulation. Genomic and precision medicine vol. (2021): CIRCGEN121003419. doi: https://doi.org/10.1161/CIRCGEN.121.003419
Lindholm ME, Jimenez-Morales D, Zhu H, Seo K, Amar D, Zhao C, Raja A, Madhvani R, Abramowitz S, Espenel C, Sutton S, Caleshu C, Berry GJ, Motonaga KS, Dunn K, Platt J, Ashley EA, Wheeler MT. Mono- and Biallelic Protein-Truncating Variants in Alpha-Actinin 2 Cause Cardiomyopathy Through Distinct Mechanisms. Circ Genom Precis Med. 2021 Nov 22;CIRCGEN121003419. doi: 10.1161/CIRCGEN.121.003419. Epub 2021 Nov 22. PMID: 34802252.
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A paediatric cardiology handbook for the digital age.

Cardiology in the young

Rochelson E, Clark BC, Motonaga KS.
PMID: 34353409
Cardiol Young. 2021 Aug 06;1-6. doi: 10.1017/S1047951121003061. Epub 2021 Aug 06.

INTRODUCTION: Paediatric cardiology fellows, tasked with studying a large and dynamic field, may benefit from a quick-access digital resource that reflects contemporary practice. The objective of this study was to evaluate the effectiveness of a paediatric cardiology handbook smartphone...

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Rochelson E, Clark BC, Motonaga KS. A paediatric cardiology handbook for the digital age. Cardiol Young. 2021;1-6doi: 10.1017/S1047951121003061.
Rochelson, E., Clark, B. C., & Motonaga, K. S. (2021). A paediatric cardiology handbook for the digital age. Cardiology in the young, 1-6. https://doi.org/10.1017/S1047951121003061
Rochelson, Ellis, et al. "A paediatric cardiology handbook for the digital age." Cardiology in the young vol. (2021): 1-6. doi: https://doi.org/10.1017/S1047951121003061
Rochelson E, Clark BC, Motonaga KS. A paediatric cardiology handbook for the digital age. Cardiol Young. 2021 Aug 06;1-6. doi: 10.1017/S1047951121003061. Epub 2021 Aug 06. PMID: 34353409.
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Mono- and Biallelic Protein-Truncating Variants in Alpha-Actinin 2 Cause Cardiomyopathy Through Distinct Mechanisms.

Circulation. Genomic and precision medicine

Lindholm ME, Jimenez-Morales D, Zhu H, Seo K, Amar D, Zhao C, Raja A, Madhvani R, Abramowitz S, Espenel C, Sutton S, Caleshu C, Berry GJ, Motonaga KS, Dunn K, Platt J, Ashley EA, Wheeler MT.
PMID: 34802252
Circ Genom Precis Med. 2021 Dec;14(6):e003419. doi: 10.1161/CIRCGEN.121.003419. Epub 2021 Nov 22.

BACKGROUND: ACTN2 (alpha-actinin 2) anchors actin within cardiac sarcomeres. The mechanisms linking METHODS: Patients harboring ACTN2 protein-truncating variants were identified using a custom mutation pipeline. In patient-derived iPSC-cardiomyocytes, we investigated transcriptional profiles using RNA sequencing, contractile properties using video-based...

Cite
Lindholm ME, Jimenez-Morales D, Zhu H, et al. Mono- and Biallelic Protein-Truncating Variants in Alpha-Actinin 2 Cause Cardiomyopathy Through Distinct Mechanisms. Circ Genom Precis Med. 2021;14(6):e003419doi: 10.1161/CIRCGEN.121.003419.
Lindholm, M. E., Jimenez-Morales, D., Zhu, H., Seo, K., Amar, D., Zhao, C., Raja, A., Madhvani, R., Abramowitz, S., Espenel, C., Sutton, S., Caleshu, C., Berry, G. J., Motonaga, K. S., Dunn, K., Platt, J., Ashley, E. A., & Wheeler, M. T. (2021). Mono- and Biallelic Protein-Truncating Variants in Alpha-Actinin 2 Cause Cardiomyopathy Through Distinct Mechanisms. Circulation. Genomic and precision medicine, 14(6), e003419. https://doi.org/10.1161/CIRCGEN.121.003419
Lindholm, Malene E, et al. "Mono- and Biallelic Protein-Truncating Variants in Alpha-Actinin 2 Cause Cardiomyopathy Through Distinct Mechanisms." Circulation. Genomic and precision medicine vol. 14,6 (2021): e003419. doi: https://doi.org/10.1161/CIRCGEN.121.003419
Lindholm ME, Jimenez-Morales D, Zhu H, Seo K, Amar D, Zhao C, Raja A, Madhvani R, Abramowitz S, Espenel C, Sutton S, Caleshu C, Berry GJ, Motonaga KS, Dunn K, Platt J, Ashley EA, Wheeler MT. Mono- and Biallelic Protein-Truncating Variants in Alpha-Actinin 2 Cause Cardiomyopathy Through Distinct Mechanisms. Circ Genom Precis Med. 2021 Dec;14(6):e003419. doi: 10.1161/CIRCGEN.121.003419. Epub 2021 Nov 22. PMID: 34802252; PMCID: PMC8692448.
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Effects of an overnight high-carbohydrate meal on muscle glycogen after rapid weight loss in male collegiate wrestlers.

BMC sports science, medicine & rehabilitation

Kondo E, Shiose K, Osawa T, Motonaga K, Kamei A, Nakajima K, Sagayama H, Wada T, Nishiguchi S, Takahashi H.
PMID: 34416921
BMC Sports Sci Med Rehabil. 2021 Aug 20;13(1):96. doi: 10.1186/s13102-021-00325-w.

BACKGROUND: Severe rapid weight loss (RWL) induces a decrease in muscle glycogen (mGly). Nevertheless, adequate carbohydrate intake after RWL has not been reported to optimize muscle glycogen following a weigh-in the evening until a wrestling tournament morning. The purpose...

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Kondo E, Shiose K, Osawa T, et al. Effects of an overnight high-carbohydrate meal on muscle glycogen after rapid weight loss in male collegiate wrestlers. BMC Sports Sci Med Rehabil. 2021;13(1):96doi: 10.1186/s13102-021-00325-w.
Kondo, E., Shiose, K., Osawa, T., Motonaga, K., Kamei, A., Nakajima, K., Sagayama, H., Wada, T., Nishiguchi, S., & Takahashi, H. (2021). Effects of an overnight high-carbohydrate meal on muscle glycogen after rapid weight loss in male collegiate wrestlers. BMC sports science, medicine & rehabilitation, 13(1), 96. https://doi.org/10.1186/s13102-021-00325-w
Kondo, Emi, et al. "Effects of an overnight high-carbohydrate meal on muscle glycogen after rapid weight loss in male collegiate wrestlers." BMC sports science, medicine & rehabilitation vol. 13,1 (2021): 96. doi: https://doi.org/10.1186/s13102-021-00325-w
Kondo E, Shiose K, Osawa T, Motonaga K, Kamei A, Nakajima K, Sagayama H, Wada T, Nishiguchi S, Takahashi H. Effects of an overnight high-carbohydrate meal on muscle glycogen after rapid weight loss in male collegiate wrestlers. BMC Sports Sci Med Rehabil. 2021 Aug 20;13(1):96. doi: 10.1186/s13102-021-00325-w. PMID: 34416921; PMCID: PMC8379859.
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Case Report of Primary Lung Abscesses Due to Hypervirulent .

Infection and drug resistance

Hirai J, Sakanashi D, Momose M, Koga T, Kinjo T, Haranaga S, Motonaga E, Fujita J.
PMID: 32606819
Infect Drug Resist. 2020 Jun 10;13:1691-1695. doi: 10.2147/IDR.S252251. eCollection 2020.

Hypervirulent

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Hirai J, Sakanashi D, Momose M, et al. Case Report of Primary Lung Abscesses Due to Hypervirulent . Infect Drug Resist. 2020;13:1691-1695doi: 10.2147/IDR.S252251.
Hirai, J., Sakanashi, D., Momose, M., Koga, T., Kinjo, T., Haranaga, S., Motonaga, E., & Fujita, J. (2020). Case Report of Primary Lung Abscesses Due to Hypervirulent . Infection and drug resistance, 131691-1695. https://doi.org/10.2147/IDR.S252251
Hirai, Jun, et al. "Case Report of Primary Lung Abscesses Due to Hypervirulent ." Infection and drug resistance vol. 13 (2020): 1691-1695. doi: https://doi.org/10.2147/IDR.S252251
Hirai J, Sakanashi D, Momose M, Koga T, Kinjo T, Haranaga S, Motonaga E, Fujita J. Case Report of Primary Lung Abscesses Due to Hypervirulent . Infect Drug Resist. 2020 Jun 10;13:1691-1695. doi: 10.2147/IDR.S252251. eCollection 2020. PMID: 32606819; PMCID: PMC7293903.
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2021 PACES Expert Consensus Statement on the Indications and Management of Cardiovascular Implantable Electronic Devices in Pediatric Patients: Executive Summary.

Heart rhythm

, Silka MJ, Shah MJ, Silva JNA, Balaji S, Beach CM, Benjamin MN, Berul CI, Cannon B, Cecchin F, Cohen MI, Dalal AS, Dechert BE, Foster A, Gebauer R, Gonzalez Corcia MC, Kannankeril PJ, Karpawich PP, Kim JJ, Krishna MR, Kubuš P, LaPage MJ, Mah DY, Malloy-Walton L, Miyazaki A, Motonaga KS, Niu MC, Olen M, Paul T, Rosenthal E, Saarel EV, Silvetti MS, Stephenson EA, Tan RB, Triedman J, Von Bergen NH, Wackel PL.
PMID: 34363987
Heart Rhythm. 2021 Nov;18(11):1925-1950. doi: 10.1016/j.hrthm.2021.07.051. Epub 2021 Jul 29.

No abstract available.

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, Silka MJ, Shah MJ, et al. 2021 PACES Expert Consensus Statement on the Indications and Management of Cardiovascular Implantable Electronic Devices in Pediatric Patients: Executive Summary. Heart Rhythm. 2021;18(11):1925-1950doi: 10.1016/j.hrthm.2021.07.051.
, Silka, M. J., Shah, M. J., Silva, J. N. A., Balaji, S., Beach, C. M., Benjamin, M. N., Berul, C. I., Cannon, B., Cecchin, F., Cohen, M. I., Dalal, A. S., Dechert, B. E., Foster, A., Gebauer, R., Gonzalez Corcia, M. C., Kannankeril, P. J., Karpawich, P. P., Kim, J. J., Krishna, M. R., Kubuš, P., LaPage, M. J., Mah, D. Y., Malloy-Walton, L., Miyazaki, A., Motonaga, K. S., Niu, M. C., Olen, M., Paul, T., Rosenthal, E., Saarel, E. V., Silvetti, M. S., Stephenson, E. A., Tan, R. B., Triedman, J., Von Bergen, N. H., & Wackel, P. L. (2021). 2021 PACES Expert Consensus Statement on the Indications and Management of Cardiovascular Implantable Electronic Devices in Pediatric Patients: Executive Summary. Heart rhythm, 18(11), 1925-1950. https://doi.org/10.1016/j.hrthm.2021.07.051
, et al. "2021 PACES Expert Consensus Statement on the Indications and Management of Cardiovascular Implantable Electronic Devices in Pediatric Patients: Executive Summary." Heart rhythm vol. 18,11 (2021): 1925-1950. doi: https://doi.org/10.1016/j.hrthm.2021.07.051
, Silka MJ, Shah MJ, Silva JNA, Balaji S, Beach CM, Benjamin MN, Berul CI, Cannon B, Cecchin F, Cohen MI, Dalal AS, Dechert BE, Foster A, Gebauer R, Gonzalez Corcia MC, Kannankeril PJ, Karpawich PP, Kim JJ, Krishna MR, Kubuš P, LaPage MJ, Mah DY, Malloy-Walton L, Miyazaki A, Motonaga KS, Niu MC, Olen M, Paul T, Rosenthal E, Saarel EV, Silvetti MS, Stephenson EA, Tan RB, Triedman J, Von Bergen NH, Wackel PL. 2021 PACES Expert Consensus Statement on the Indications and Management of Cardiovascular Implantable Electronic Devices in Pediatric Patients: Executive Summary. Heart Rhythm. 2021 Nov;18(11):1925-1950. doi: 10.1016/j.hrthm.2021.07.051. Epub 2021 Jul 29. PMID: 34363987.
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Mono- and Biallelic Protein-Truncating Variants in Alpha-Actinin 2 Cause Cardiomyopathy Through Distinct Mechanisms.

Circulation. Genomic and precision medicine

Lindholm ME, Jimenez-Morales D, Zhu H, Seo K, Amar D, Zhao C, Raja A, Madhvani R, Abramowitz S, Espenel C, Sutton S, Caleshu C, Berry GJ, Motonaga KS, Dunn K, Platt J, Ashley EA, Wheeler MT.
PMID: 34802252
Circ Genom Precis Med. 2021 Dec;14(6):e003419. doi: 10.1161/CIRCGEN.121.003419. Epub 2021 Nov 22.

BACKGROUND: ACTN2 (alpha-actinin 2) anchors actin within cardiac sarcomeres. The mechanisms linking METHODS: Patients harboring ACTN2 protein-truncating variants were identified using a custom mutation pipeline. In patient-derived iPSC-cardiomyocytes, we investigated transcriptional profiles using RNA sequencing, contractile properties using video-based...

Cite
Lindholm ME, Jimenez-Morales D, Zhu H, et al. Mono- and Biallelic Protein-Truncating Variants in Alpha-Actinin 2 Cause Cardiomyopathy Through Distinct Mechanisms. Circ Genom Precis Med. 2021;14(6):e003419doi: 10.1161/CIRCGEN.121.003419.
Lindholm, M. E., Jimenez-Morales, D., Zhu, H., Seo, K., Amar, D., Zhao, C., Raja, A., Madhvani, R., Abramowitz, S., Espenel, C., Sutton, S., Caleshu, C., Berry, G. J., Motonaga, K. S., Dunn, K., Platt, J., Ashley, E. A., & Wheeler, M. T. (2021). Mono- and Biallelic Protein-Truncating Variants in Alpha-Actinin 2 Cause Cardiomyopathy Through Distinct Mechanisms. Circulation. Genomic and precision medicine, 14(6), e003419. https://doi.org/10.1161/CIRCGEN.121.003419
Lindholm, Malene E, et al. "Mono- and Biallelic Protein-Truncating Variants in Alpha-Actinin 2 Cause Cardiomyopathy Through Distinct Mechanisms." Circulation. Genomic and precision medicine vol. 14,6 (2021): e003419. doi: https://doi.org/10.1161/CIRCGEN.121.003419
Lindholm ME, Jimenez-Morales D, Zhu H, Seo K, Amar D, Zhao C, Raja A, Madhvani R, Abramowitz S, Espenel C, Sutton S, Caleshu C, Berry GJ, Motonaga KS, Dunn K, Platt J, Ashley EA, Wheeler MT. Mono- and Biallelic Protein-Truncating Variants in Alpha-Actinin 2 Cause Cardiomyopathy Through Distinct Mechanisms. Circ Genom Precis Med. 2021 Dec;14(6):e003419. doi: 10.1161/CIRCGEN.121.003419. Epub 2021 Nov 22. PMID: 34802252.
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Hypophysitis induced by ipilimumab and nivolumab combination therapy for advanced renal cell carcinoma: A case report.

Urology case reports

Motonaga A, Nakanishi S, Tanaka K, Nishida S, Izumi K, Saito S.
PMID: 33868946
Urol Case Rep. 2021 Mar 26;38:101661. doi: 10.1016/j.eucr.2021.101661. eCollection 2021 Sep.

Ipilimumab and nivolumab combination therapy is effective against unresectable or metastatic renal cell carcinoma. However, it is associated with many immune-related adverse events, including hypophysitis that is difficult to diagnose early because of non-specific initial symptoms. Herein, we report...

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Motonaga A, Nakanishi S, Tanaka K, et al. Hypophysitis induced by ipilimumab and nivolumab combination therapy for advanced renal cell carcinoma: A case report. Urol Case Rep. 2021;38:101661doi: 10.1016/j.eucr.2021.101661.
Motonaga, A., Nakanishi, S., Tanaka, K., Nishida, S., Izumi, K., & Saito, S. (2021). Hypophysitis induced by ipilimumab and nivolumab combination therapy for advanced renal cell carcinoma: A case report. Urology case reports, 38101661. https://doi.org/10.1016/j.eucr.2021.101661
Motonaga, Aoi, et al. "Hypophysitis induced by ipilimumab and nivolumab combination therapy for advanced renal cell carcinoma: A case report." Urology case reports vol. 38 (2021): 101661. doi: https://doi.org/10.1016/j.eucr.2021.101661
Motonaga A, Nakanishi S, Tanaka K, Nishida S, Izumi K, Saito S. Hypophysitis induced by ipilimumab and nivolumab combination therapy for advanced renal cell carcinoma: A case report. Urol Case Rep. 2021 Mar 26;38:101661. doi: 10.1016/j.eucr.2021.101661. eCollection 2021 Sep. PMID: 33868946; PMCID: PMC8040277.
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Showing 1 to 12 of 34 entries