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Showing 1 to 12 of 103 entries
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Exponential Attenuation of Through-Bond Transmission in a Polyene: Theory and Potential Realizations.

ACS nano

Tsuji Y, Movassagh R, Datta S, Hoffmann R.
PMID: 26390251
ACS Nano. 2015 Nov 24;9(11):11109-20. doi: 10.1021/acsnano.5b04615. Epub 2015 Sep 29.

An exponential falloff with separation of electron transfer and transport through molecular wires is observed and has attracted theoretical attention. In this study, the attenuation of transmission in linear and cyclic polyenes is related to bond alternation. The explicit...

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Tsuji Y, Movassagh R, Datta S, et al. Exponential Attenuation of Through-Bond Transmission in a Polyene: Theory and Potential Realizations. ACS Nano. 2015;9(11):11109-20doi: 10.1021/acsnano.5b04615.
Tsuji, Y., Movassagh, R., Datta, S., & Hoffmann, R. (2015). Exponential Attenuation of Through-Bond Transmission in a Polyene: Theory and Potential Realizations. ACS nano, 9(11), 11109-20. https://doi.org/10.1021/acsnano.5b04615
Tsuji, Yuta, et al. "Exponential Attenuation of Through-Bond Transmission in a Polyene: Theory and Potential Realizations." ACS nano vol. 9,11 (2015): 11109-20. doi: https://doi.org/10.1021/acsnano.5b04615
Tsuji Y, Movassagh R, Datta S, Hoffmann R. Exponential Attenuation of Through-Bond Transmission in a Polyene: Theory and Potential Realizations. ACS Nano. 2015 Nov 24;9(11):11109-20. doi: 10.1021/acsnano.5b04615. Epub 2015 Sep 29. PMID: 26390251.
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Electrophilic Carbonyl Activation: Competing Condensative Cyclizations of Tryptamine Derivatives.

Tetrahedron letters

Liu F, Movassaghi M.
PMID: 26120209
Tetrahedron Lett. 2015 Jun 03;56(23):2995-3000. doi: 10.1016/j.tetlet.2014.09.022.

A series of tryptamine derived bisindole substrates were subject to electrophilic activation of the functional grouping at their alpha-nitrogen in the form of iminium ions to enable cyclization onto the sterically hindered indole substructure. Our observations regarding divergent cyclization...

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Liu F, Movassaghi M. Electrophilic Carbonyl Activation: Competing Condensative Cyclizations of Tryptamine Derivatives. Tetrahedron Lett. 2015;56(23):2995-3000doi: 10.1016/j.tetlet.2014.09.022.
Liu, F., & Movassaghi, M. (2015). Electrophilic Carbonyl Activation: Competing Condensative Cyclizations of Tryptamine Derivatives. Tetrahedron letters, 56(23), 2995-3000. https://doi.org/10.1016/j.tetlet.2014.09.022
Liu, Fan, and Movassaghi, Mohammad. "Electrophilic Carbonyl Activation: Competing Condensative Cyclizations of Tryptamine Derivatives." Tetrahedron letters vol. 56,23 (2015): 2995-3000. doi: https://doi.org/10.1016/j.tetlet.2014.09.022
Liu F, Movassaghi M. Electrophilic Carbonyl Activation: Competing Condensative Cyclizations of Tryptamine Derivatives. Tetrahedron Lett. 2015 Jun 03;56(23):2995-3000. doi: 10.1016/j.tetlet.2014.09.022. PMID: 26120209; PMCID: PMC4480791.
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Genotyping of Clostridium perfringens isolated from healthy and diseased ostriches (Struthio camelus).

Iranian journal of microbiology

Razmyar J, Kalidari GA, Tolooe A, Rad M, Movassaghi AR.
PMID: 25954489
Iran J Microbiol. 2014 Feb;6(1):31-6.

BACKGROUND AND OBJECTIVES: Clostridium perfringens is more prevalent type of clostridia genus isolated from the intestinal tract of ostrich (Struthio camelus). Necrotic enteritis (NE) is a potentially fatal gastrointestinal (GI) disease of poultry and other avian species, which produces...

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Razmyar J, Kalidari GA, Tolooe A, et al. Genotyping of Clostridium perfringens isolated from healthy and diseased ostriches (Struthio camelus). Iran J Microbiol. 2014;6(1):31-6
Razmyar, J., Kalidari, G. A., Tolooe, A., Rad, M., & Movassaghi, A. R. (2014). Genotyping of Clostridium perfringens isolated from healthy and diseased ostriches (Struthio camelus). Iranian journal of microbiology, 6(1), 31-6.
Razmyar, Jamshid, et al. "Genotyping of Clostridium perfringens isolated from healthy and diseased ostriches (Struthio camelus)." Iranian journal of microbiology vol. 6,1 (2014): 31-6.
Razmyar J, Kalidari GA, Tolooe A, Rad M, Movassaghi AR. Genotyping of Clostridium perfringens isolated from healthy and diseased ostriches (Struthio camelus). Iran J Microbiol. 2014 Feb;6(1):31-6. PMID: 25954489; PMCID: PMC4419043.
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Total synthesis of all (-)-Agelastatin alkaloids.

Chemical science

Movassaghi M, Siegel DS, Han S.
PMID: 21218186
Chem Sci. 2010 Jan 01;1:561-566. doi: 10.1039/c0sc00351d.

The pyrrole-imidazole family of marine alkaloids, derived from linear clathrodin-like precursors, constitutes a diverse array of structurally complex natural products. The bioactive agelastatins are members of this family that possess a tetracyclic molecular framework incorporating C4-C8 and C7-N12 bond...

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Movassaghi M, Siegel DS, Han S. Total synthesis of all (-)-Agelastatin alkaloids. Chem Sci. 2010;1:561-566doi: 10.1039/c0sc00351d.
Movassaghi, M., Siegel, D. S., & Han, S. (2010). Total synthesis of all (-)-Agelastatin alkaloids. Chemical science, 1561-566. https://doi.org/10.1039/c0sc00351d
Movassaghi, Mohammad, et al. "Total synthesis of all (-)-Agelastatin alkaloids." Chemical science vol. 1 (2010): 561-566. doi: https://doi.org/10.1039/c0sc00351d
Movassaghi M, Siegel DS, Han S. Total synthesis of all (-)-Agelastatin alkaloids. Chem Sci. 2010 Jan 01;1:561-566. doi: 10.1039/c0sc00351d. PMID: 21218186; PMCID: PMC3016719.
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High-throughput sequencing identifies STAT3 as the DNA-associated factor for p53-NF-kappaB-complex-dependent gene expression in human heart failure.

Genome medicine

Choy MK, Movassagh M, Siggens L, Vujic A, Goddard M, Sánchez A, Perkins N, Figg N, Bennett M, Carroll J, Foo R.
PMID: 20546595
Genome Med. 2010 Jun 14;2(6):37. doi: 10.1186/gm158.

BACKGROUND: Genome-wide maps of DNA regulatory elements and their interaction with transcription factors may form a framework for understanding regulatory circuits and gene expression control in human disease, but how these networks, comprising transcription factors and DNA-binding proteins, form...

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Choy MK, Movassagh M, Siggens L, et al. High-throughput sequencing identifies STAT3 as the DNA-associated factor for p53-NF-kappaB-complex-dependent gene expression in human heart failure. Genome Med. 2010;2(6):37doi: 10.1186/gm158.
Choy, M. K., Movassagh, M., Siggens, L., Vujic, A., Goddard, M., Sánchez, A., Perkins, N., Figg, N., Bennett, M., Carroll, J., & Foo, R. (2010). High-throughput sequencing identifies STAT3 as the DNA-associated factor for p53-NF-kappaB-complex-dependent gene expression in human heart failure. Genome medicine, 2(6), 37. https://doi.org/10.1186/gm158
Choy, Mun-Kit, et al. "High-throughput sequencing identifies STAT3 as the DNA-associated factor for p53-NF-kappaB-complex-dependent gene expression in human heart failure." Genome medicine vol. 2,6 (2010): 37. doi: https://doi.org/10.1186/gm158
Choy MK, Movassagh M, Siggens L, Vujic A, Goddard M, Sánchez A, Perkins N, Figg N, Bennett M, Carroll J, Foo R. High-throughput sequencing identifies STAT3 as the DNA-associated factor for p53-NF-kappaB-complex-dependent gene expression in human heart failure. Genome Med. 2010 Jun 14;2(6):37. doi: 10.1186/gm158. PMID: 20546595; PMCID: PMC2905097.
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Criticality without frustration for quantum spin-1 chains.

Physical review letters

Bravyi S, Caha L, Movassagh R, Nagaj D, Shor PW.
PMID: 23215521
Phys Rev Lett. 2012 Nov 16;109(20):207202. doi: 10.1103/PhysRevLett.109.207202. Epub 2012 Nov 16.

Frustration-free (FF) spin chains have a property that their ground state minimizes all individual terms in the chain Hamiltonian. We ask how entangled the ground state of a FF quantum spin-s chain with nearest-neighbor interactions can be for small...

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Bravyi S, Caha L, Movassagh R, et al. Criticality without frustration for quantum spin-1 chains. Phys Rev Lett. 2012;109(20):207202doi: 10.1103/PhysRevLett.109.207202.
Bravyi, S., Caha, L., Movassagh, R., Nagaj, D., & Shor, P. W. (2012). Criticality without frustration for quantum spin-1 chains. Physical review letters, 109(20), 207202. https://doi.org/10.1103/PhysRevLett.109.207202
Bravyi, Sergey, et al. "Criticality without frustration for quantum spin-1 chains." Physical review letters vol. 109,20 (2012): 207202. doi: https://doi.org/10.1103/PhysRevLett.109.207202
Bravyi S, Caha L, Movassagh R, Nagaj D, Shor PW. Criticality without frustration for quantum spin-1 chains. Phys Rev Lett. 2012 Nov 16;109(20):207202. doi: 10.1103/PhysRevLett.109.207202. Epub 2012 Nov 16. PMID: 23215521.
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IgE induces proliferation in human airway smooth muscle cells: role of MAPK and STAT3 pathways.

Allergy, asthma, and clinical immunology : official journal of the Canadian Society of Allergy and Clinical Immunology

Redhu NS, Shan L, Al-Subait D, Ashdown HL, Movassagh H, Lamkhioued B, Gounni AS.
PMID: 24499258
Allergy Asthma Clin Immunol. 2013 Oct 17;9(1):41. doi: 10.1186/1710-1492-9-41.

Airway remodeling is not specifically targeted by current asthma medications, partly owing to the lack of understanding of remodeling mechanisms, altogether posing great challenges in asthma treatment. Increased airway smooth muscle (ASM) mass due to hyperplasia/hypertrophy contributes significantly to...

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Redhu NS, Shan L, Al-Subait D, et al. IgE induces proliferation in human airway smooth muscle cells: role of MAPK and STAT3 pathways. Allergy Asthma Clin Immunol. 2013;9(1):41doi: 10.1186/1710-1492-9-41.
Redhu, N. S., Shan, L., Al-Subait, D., Ashdown, H. L., Movassagh, H., Lamkhioued, B., & Gounni, A. S. (2013). IgE induces proliferation in human airway smooth muscle cells: role of MAPK and STAT3 pathways. Allergy, asthma, and clinical immunology : official journal of the Canadian Society of Allergy and Clinical Immunology, 9(1), 41. https://doi.org/10.1186/1710-1492-9-41
Redhu, Naresh Singh, et al. "IgE induces proliferation in human airway smooth muscle cells: role of MAPK and STAT3 pathways." Allergy, asthma, and clinical immunology : official journal of the Canadian Society of Allergy and Clinical Immunology vol. 9,1 (2013): 41. doi: https://doi.org/10.1186/1710-1492-9-41
Redhu NS, Shan L, Al-Subait D, Ashdown HL, Movassagh H, Lamkhioued B, Gounni AS. IgE induces proliferation in human airway smooth muscle cells: role of MAPK and STAT3 pathways. Allergy Asthma Clin Immunol. 2013 Oct 17;9(1):41. doi: 10.1186/1710-1492-9-41. PMID: 24499258; PMCID: PMC3842672.
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Pentoxifylline Protects the Rat Liver Against Fibrosis and Apoptosis Induced by Acute Administration of 3,4-Methylenedioxymethamphetamine (MDMA or Ecstasy).

Iranian journal of basic medical sciences

Movassaghi S, Nadia Sharifi Z, Mohammadzadeh F, Soleimani M.
PMID: 24106597
Iran J Basic Med Sci. 2013 Aug;16(8):922-7.

OBJECTIVE(S): 3,4-Methylenedioxymethamphetamine (MDMA) is one of the most popular drugs of abuse in the world with hallucinogenic properties that has been shown to induce apoptosis in liver cells. The present study aimed to investigate the effects of pentoxifylline (PTX)...

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Movassaghi S, Nadia Sharifi Z, Mohammadzadeh F, et al. Pentoxifylline Protects the Rat Liver Against Fibrosis and Apoptosis Induced by Acute Administration of 3,4-Methylenedioxymethamphetamine (MDMA or Ecstasy). Iran J Basic Med Sci. 2013;16(8):922-7
Movassaghi, S., Nadia Sharifi, Z., Mohammadzadeh, F., & Soleimani, M. (2013). Pentoxifylline Protects the Rat Liver Against Fibrosis and Apoptosis Induced by Acute Administration of 3,4-Methylenedioxymethamphetamine (MDMA or Ecstasy). Iranian journal of basic medical sciences, 16(8), 922-7.
Movassaghi, Shabnam, et al. "Pentoxifylline Protects the Rat Liver Against Fibrosis and Apoptosis Induced by Acute Administration of 3,4-Methylenedioxymethamphetamine (MDMA or Ecstasy)." Iranian journal of basic medical sciences vol. 16,8 (2013): 922-7.
Movassaghi S, Nadia Sharifi Z, Mohammadzadeh F, Soleimani M. Pentoxifylline Protects the Rat Liver Against Fibrosis and Apoptosis Induced by Acute Administration of 3,4-Methylenedioxymethamphetamine (MDMA or Ecstasy). Iran J Basic Med Sci. 2013 Aug;16(8):922-7. PMID: 24106597; PMCID: PMC3786105.
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Effect of Pentoxifylline on Ischemia- induced Brain Damage and Spatial Memory Impairment in Rat.

Iranian journal of basic medical sciences

Movassaghi S, Nadia Sharifi Z, Soleimani M, Joghataii MT, Hashemi M, Shafaroodi H, Mehdizadeh M.
PMID: 23493977
Iran J Basic Med Sci. 2012 Sep;15(5):1083-90.

OBJECTIVES: The brief interruption of cerebral blood flow causes permanent brain damage and behavioral dysfunction. The hippocampus is highly vulnerable to ischemic insults, particularly the CA1 pyramidal cell layer. There is no effective pharmacological strategy for improving brain tissue...

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Movassaghi S, Nadia Sharifi Z, Soleimani M, et al. Effect of Pentoxifylline on Ischemia- induced Brain Damage and Spatial Memory Impairment in Rat. Iran J Basic Med Sci. 2012;15(5):1083-90
Movassaghi, S., Nadia Sharifi, Z., Soleimani, M., Joghataii, M. T., Hashemi, M., Shafaroodi, H., & Mehdizadeh, M. (2012). Effect of Pentoxifylline on Ischemia- induced Brain Damage and Spatial Memory Impairment in Rat. Iranian journal of basic medical sciences, 15(5), 1083-90.
Movassaghi, Shabnam, et al. "Effect of Pentoxifylline on Ischemia- induced Brain Damage and Spatial Memory Impairment in Rat." Iranian journal of basic medical sciences vol. 15,5 (2012): 1083-90.
Movassaghi S, Nadia Sharifi Z, Soleimani M, Joghataii MT, Hashemi M, Shafaroodi H, Mehdizadeh M. Effect of Pentoxifylline on Ischemia- induced Brain Damage and Spatial Memory Impairment in Rat. Iran J Basic Med Sci. 2012 Sep;15(5):1083-90. PMID: 23493977; PMCID: PMC3586929.
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Stability of Periodically Driven Topological Phases against Disorder.

Physical review letters

Shtanko O, Movassagh R.
PMID: 30296124
Phys Rev Lett. 2018 Sep 21;121(12):126803. doi: 10.1103/PhysRevLett.121.126803.

In recent experiments, time-dependent periodic fields are used to create exotic topological phases of matter with potential applications ranging from quantum transport to quantum computing. These nonequilibrium states, at high driving frequencies, exhibit the quintessential robustness against local disorder...

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Shtanko O, Movassagh R. Stability of Periodically Driven Topological Phases against Disorder. Phys Rev Lett. 2018;121(12):126803doi: 10.1103/PhysRevLett.121.126803.
Shtanko, O., & Movassagh, R. (2018). Stability of Periodically Driven Topological Phases against Disorder. Physical review letters, 121(12), 126803. https://doi.org/10.1103/PhysRevLett.121.126803
Shtanko, Oles, and Movassagh, Ramis. "Stability of Periodically Driven Topological Phases against Disorder." Physical review letters vol. 121,12 (2018): 126803. doi: https://doi.org/10.1103/PhysRevLett.121.126803
Shtanko O, Movassagh R. Stability of Periodically Driven Topological Phases against Disorder. Phys Rev Lett. 2018 Sep 21;121(12):126803. doi: 10.1103/PhysRevLett.121.126803. PMID: 30296124.
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Effect of Different Doses and Times of FK506 on Different Areas of the Hippocampus in the Rat Model of Transient Global Cerebral Ischemia-Reperfusion.

Neurology research international

Sharifi ZN, Zaferani Arani H, Olya M, Adin Atashi H, Movassaghi S.
PMID: 31467711
Neurol Res Int. 2019 Jul 29;2019:8047672. doi: 10.1155/2019/8047672. eCollection 2019.

BACKGROUND: Stroke is a major worldwide problem that is leading to a high mortality rate in humans. Ischemia, as the most common type of stroke, is characterized by tissue damage that can occur due to insufficient blood flow to...

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Sharifi ZN, Zaferani Arani H, Olya M, et al. Effect of Different Doses and Times of FK506 on Different Areas of the Hippocampus in the Rat Model of Transient Global Cerebral Ischemia-Reperfusion. Neurol Res Int. 2019;2019:8047672doi: 10.1155/2019/8047672.
Sharifi, Z. N., Zaferani Arani, H., Olya, M., Adin Atashi, H., & Movassaghi, S. (2019). Effect of Different Doses and Times of FK506 on Different Areas of the Hippocampus in the Rat Model of Transient Global Cerebral Ischemia-Reperfusion. Neurology research international, 20198047672. https://doi.org/10.1155/2019/8047672
Sharifi, Zahra Nadia, et al. "Effect of Different Doses and Times of FK506 on Different Areas of the Hippocampus in the Rat Model of Transient Global Cerebral Ischemia-Reperfusion." Neurology research international vol. 2019 (2019): 8047672. doi: https://doi.org/10.1155/2019/8047672
Sharifi ZN, Zaferani Arani H, Olya M, Adin Atashi H, Movassaghi S. Effect of Different Doses and Times of FK506 on Different Areas of the Hippocampus in the Rat Model of Transient Global Cerebral Ischemia-Reperfusion. Neurol Res Int. 2019 Jul 29;2019:8047672. doi: 10.1155/2019/8047672. eCollection 2019. PMID: 31467711; PMCID: PMC6699343.
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[Hyperuricemia in systemic lupus erythematosus: is it associated with the neuropsychiatric manifestations of the disease?].

Revista brasileira de reumatologia

Sheikh M, Movassaghi S, Khaledi M, Moghaddassi M.
PMID: 26232280
Rev Bras Reumatol. 2015 Jul 17; doi: 10.1016/j.rbr.2015.05.002. Epub 2015 Jul 17.

OBJECTIVES: To assess the association between hyperuricemia and different neuropsychiatric manifestations and stroke risk factors in systematic lupus erythematosus (SLE) patients.METHODS: This study was conducted on 204 SLE patients who were admitted to a tertiary referral center. A standardized...

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Sheikh M, Movassaghi S, Khaledi M, et al. Hiperuricemia no lúpus eritematoso sistêmico: está associada a manifestações neuropsiquiátricas da doença? [Hyperuricemia in systemic lupus erythematosus: is it associated with the neuropsychiatric manifestations of the disease?]. Rev Bras Reumatol. 2015;doi: 10.1016/j.rbr.2015.05.002.
Sheikh, M., Movassaghi, S., Khaledi, M., & Moghaddassi, M. (2015). Hiperuricemia no lúpus eritematoso sistêmico: está associada a manifestações neuropsiquiátricas da doença? [Hyperuricemia in systemic lupus erythematosus: is it associated with the neuropsychiatric manifestations of the disease?]. Revista brasileira de reumatologia, . https://doi.org/10.1016/j.rbr.2015.05.002
Sheikh, Mahdi, et al. "Hiperuricemia no lúpus eritematoso sistêmico: está associada a manifestações neuropsiquiátricas da doença?" [Hyperuricemia in systemic lupus erythematosus: is it associated with the neuropsychiatric manifestations of the disease?]. Revista brasileira de reumatologia vol. (2015). doi: https://doi.org/10.1016/j.rbr.2015.05.002
Sheikh M, Movassaghi S, Khaledi M, Moghaddassi M. Hiperuricemia no lúpus eritematoso sistêmico: está associada a manifestações neuropsiquiátricas da doença? [Hyperuricemia in systemic lupus erythematosus: is it associated with the neuropsychiatric manifestations of the disease?]. Rev Bras Reumatol. 2015 Jul 17; doi: 10.1016/j.rbr.2015.05.002. Epub 2015 Jul 17. Portuguese. PMID: 26232280.
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Showing 1 to 12 of 103 entries