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Study of the Effects of ATP Suppliers and Thiol Reductants on Toxicity of Pioglitazone in Isolated Rat Liver Mitochondria.

Iranian journal of pharmaceutical research : IJPR

Rezaiean Mehrabadi A, Jamshidzadeh A, Rashedinia M, Niknahad H.
PMID: 26330870
Iran J Pharm Res. 2015;14(3):825-32.

Pioglitazone (PG) is one of thiazolidinediones used for the treatment of type II diabetes mellitus. Some reports of its hepatotoxicity exist, but the mechanism of its hepatotoxicity is not well known. In the present study, the protective effect of...

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Rezaiean Mehrabadi A, Jamshidzadeh A, Rashedinia M, et al. Study of the Effects of ATP Suppliers and Thiol Reductants on Toxicity of Pioglitazone in Isolated Rat Liver Mitochondria. Iran J Pharm Res. 2015;14(3):825-32
Rezaiean Mehrabadi, A., Jamshidzadeh, A., Rashedinia, M., & Niknahad, H. (2015). Study of the Effects of ATP Suppliers and Thiol Reductants on Toxicity of Pioglitazone in Isolated Rat Liver Mitochondria. Iranian journal of pharmaceutical research : IJPR, 14(3), 825-32.
Rezaiean Mehrabadi, Abbas, et al. "Study of the Effects of ATP Suppliers and Thiol Reductants on Toxicity of Pioglitazone in Isolated Rat Liver Mitochondria." Iranian journal of pharmaceutical research : IJPR vol. 14,3 (2015): 825-32.
Rezaiean Mehrabadi A, Jamshidzadeh A, Rashedinia M, Niknahad H. Study of the Effects of ATP Suppliers and Thiol Reductants on Toxicity of Pioglitazone in Isolated Rat Liver Mitochondria. Iran J Pharm Res. 2015;14(3):825-32. PMID: 26330870; PMCID: PMC4518110.
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Effect of Thiol-reducing Agents and Antioxidants on Sulfasalazine-induced Hepatic Injury in Normotermic Recirculating Isolated Perfused Rat Liver.

Toxicological research

Heidari R, Esmailie N, Azarpira N, Najibi A, Niknahad H.
PMID: 27123164
Toxicol Res. 2016 Apr;32(2):133-40. doi: 10.5487/TR.2016.32.2.133. Epub 2016 Apr 30.

Sulfasalzine is a widely administered drug against inflammatory-based disorders in human. However several cases of liver injury are associated with its administration. There is no stabilized safe protective agent against sulfasalazine-induced liver injury. Current investigation was designed to evaluate...

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Heidari R, Esmailie N, Azarpira N, et al. Effect of Thiol-reducing Agents and Antioxidants on Sulfasalazine-induced Hepatic Injury in Normotermic Recirculating Isolated Perfused Rat Liver. Toxicol Res. 2016;32(2):133-40doi: 10.5487/TR.2016.32.2.133.
Heidari, R., Esmailie, N., Azarpira, N., Najibi, A., & Niknahad, H. (2016). Effect of Thiol-reducing Agents and Antioxidants on Sulfasalazine-induced Hepatic Injury in Normotermic Recirculating Isolated Perfused Rat Liver. Toxicological research, 32(2), 133-40. https://doi.org/10.5487/TR.2016.32.2.133
Heidari, Reza, et al. "Effect of Thiol-reducing Agents and Antioxidants on Sulfasalazine-induced Hepatic Injury in Normotermic Recirculating Isolated Perfused Rat Liver." Toxicological research vol. 32,2 (2016): 133-40. doi: https://doi.org/10.5487/TR.2016.32.2.133
Heidari R, Esmailie N, Azarpira N, Najibi A, Niknahad H. Effect of Thiol-reducing Agents and Antioxidants on Sulfasalazine-induced Hepatic Injury in Normotermic Recirculating Isolated Perfused Rat Liver. Toxicol Res. 2016 Apr;32(2):133-40. doi: 10.5487/TR.2016.32.2.133. Epub 2016 Apr 30. PMID: 27123164; PMCID: PMC4843982.
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Ammonia-induced mitochondrial dysfunction and energy metabolism disturbances in isolated brain and liver mitochondria, and the effect of taurine administration: relevance to hepatic encephalopathy treatment.

Clinical and experimental hepatology

Niknahad H, Jamshidzadeh A, Heidari R, Zarei M, Ommati MM.
PMID: 29062904
Clin Exp Hepatol. 2017 Sep;3(3):141-151. doi: 10.5114/ceh.2017.68833. Epub 2017 Jul 05.

INTRODUCTION: Ammonia-induced oxidative stress, mitochondrial dysfunction, and energy crisis are known as some the major mechanisms of brain injury in hepatic encephalopathy (HE). Hyperammonemia also affects the liver and hepatocytes. Therefore, targeting mitochondria seems to be a therapeutic point...

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Niknahad H, Jamshidzadeh A, Heidari R, et al. Ammonia-induced mitochondrial dysfunction and energy metabolism disturbances in isolated brain and liver mitochondria, and the effect of taurine administration: relevance to hepatic encephalopathy treatment. Clin Exp Hepatol. 2017;3(3):141-151doi: 10.5114/ceh.2017.68833.
Niknahad, H., Jamshidzadeh, A., Heidari, R., Zarei, M., & Ommati, M. M. (2017). Ammonia-induced mitochondrial dysfunction and energy metabolism disturbances in isolated brain and liver mitochondria, and the effect of taurine administration: relevance to hepatic encephalopathy treatment. Clinical and experimental hepatology, 3(3), 141-151. https://doi.org/10.5114/ceh.2017.68833
Niknahad, Hossein, et al. "Ammonia-induced mitochondrial dysfunction and energy metabolism disturbances in isolated brain and liver mitochondria, and the effect of taurine administration: relevance to hepatic encephalopathy treatment." Clinical and experimental hepatology vol. 3,3 (2017): 141-151. doi: https://doi.org/10.5114/ceh.2017.68833
Niknahad H, Jamshidzadeh A, Heidari R, Zarei M, Ommati MM. Ammonia-induced mitochondrial dysfunction and energy metabolism disturbances in isolated brain and liver mitochondria, and the effect of taurine administration: relevance to hepatic encephalopathy treatment. Clin Exp Hepatol. 2017 Sep;3(3):141-151. doi: 10.5114/ceh.2017.68833. Epub 2017 Jul 05. PMID: 29062904; PMCID: PMC5649485.
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Sulfasalazine-induced renal and hepatic injury in rats and the protective role of taurine.

BioImpacts : BI

Heidari R, Rasti M, Shirazi Yeganeh B, Niknahad H, Saeedi A, Najibi A.
PMID: 27340618
Bioimpacts. 2016;6(1):3-8. doi: 10.15171/bi.2016.01. Epub 2016 Mar 28.

INTRODUCTION: Sulfasalazine is a drug commonly administrated against inflammatory-based disorders. On the other hand, kidney and liver injury are serious adverse events accompanied by sulfasalazine administration. No specific therapeutic option is available against this complication. The current investigation was...

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Heidari R, Rasti M, Shirazi Yeganeh B, et al. Sulfasalazine-induced renal and hepatic injury in rats and the protective role of taurine. Bioimpacts. 2016;6(1):3-8doi: 10.15171/bi.2016.01.
Heidari, R., Rasti, M., Shirazi Yeganeh, B., Niknahad, H., Saeedi, A., & Najibi, A. (2016). Sulfasalazine-induced renal and hepatic injury in rats and the protective role of taurine. BioImpacts : BI, 6(1), 3-8. https://doi.org/10.15171/bi.2016.01
Heidari, Reza, et al. "Sulfasalazine-induced renal and hepatic injury in rats and the protective role of taurine." BioImpacts : BI vol. 6,1 (2016): 3-8. doi: https://doi.org/10.15171/bi.2016.01
Heidari R, Rasti M, Shirazi Yeganeh B, Niknahad H, Saeedi A, Najibi A. Sulfasalazine-induced renal and hepatic injury in rats and the protective role of taurine. Bioimpacts. 2016;6(1):3-8. doi: 10.15171/bi.2016.01. Epub 2016 Mar 28. PMID: 27340618; PMCID: PMC4916549.
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Diazinon Interrupts Ovarian Steroidogenic Acute Regulatory (.

Iranian journal of pharmaceutical research : IJPR

Siavashpour A, Ghasemi Y, Khalvati B, Jeivad F, Azarpira N, Niknahad H.
PMID: 29881411
Iran J Pharm Res. 2018;17(2):535-542.

Organophosphate pesticides are considered as endocrine disruptors that interfere with reproductive functions. The

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Siavashpour A, Ghasemi Y, Khalvati B, et al. Diazinon Interrupts Ovarian Steroidogenic Acute Regulatory (. Iran J Pharm Res. 2018;17(2):535-542
Siavashpour, A., Ghasemi, Y., Khalvati, B., Jeivad, F., Azarpira, N., & Niknahad, H. (2018). Diazinon Interrupts Ovarian Steroidogenic Acute Regulatory (. Iranian journal of pharmaceutical research : IJPR, 17(2), 535-542.
Siavashpour, Asma, et al. "Diazinon Interrupts Ovarian Steroidogenic Acute Regulatory (." Iranian journal of pharmaceutical research : IJPR vol. 17,2 (2018): 535-542.
Siavashpour A, Ghasemi Y, Khalvati B, Jeivad F, Azarpira N, Niknahad H. Diazinon Interrupts Ovarian Steroidogenic Acute Regulatory (. Iran J Pharm Res. 2018;17(2):535-542. PMID: 29881411; PMCID: PMC5985171.
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Recent Advances on Cell-Based Co-Culture Strategies for Prevascularization in Tissue Engineering.

Frontiers in bioengineering and biotechnology

Shafiee S, Shariatzadeh S, Zafari A, Majd A, Niknejad H.
PMID: 34900955
Front Bioeng Biotechnol. 2021 Nov 25;9:745314. doi: 10.3389/fbioe.2021.745314. eCollection 2021.

Currently, the fabrication of a functional vascular network to maintain the viability of engineered tissues is a major bottleneck in the way of developing a more advanced engineered construct. Inspired by vasculogenesis during the embryonic period, the

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Shafiee S, Shariatzadeh S, Zafari A, et al. Recent Advances on Cell-Based Co-Culture Strategies for Prevascularization in Tissue Engineering. Front Bioeng Biotechnol. 2021;9:745314doi: 10.3389/fbioe.2021.745314.
Shafiee, S., Shariatzadeh, S., Zafari, A., Majd, A., & Niknejad, H. (2021). Recent Advances on Cell-Based Co-Culture Strategies for Prevascularization in Tissue Engineering. Frontiers in bioengineering and biotechnology, 9745314. https://doi.org/10.3389/fbioe.2021.745314
Shafiee, Sepehr, et al. "Recent Advances on Cell-Based Co-Culture Strategies for Prevascularization in Tissue Engineering." Frontiers in bioengineering and biotechnology vol. 9 (2021): 745314. doi: https://doi.org/10.3389/fbioe.2021.745314
Shafiee S, Shariatzadeh S, Zafari A, Majd A, Niknejad H. Recent Advances on Cell-Based Co-Culture Strategies for Prevascularization in Tissue Engineering. Front Bioeng Biotechnol. 2021 Nov 25;9:745314. doi: 10.3389/fbioe.2021.745314. eCollection 2021. PMID: 34900955; PMCID: PMC8655789.
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Developing a pro-angiogenic placenta derived amniochorionic scaffold with two exposed basement membranes as substrates for cultivating endothelial cells.

Scientific reports

Shariatzadeh S, Shafiee S, Zafari A, Tayebi T, Yazdanpanah G, Majd A, Haj-Mirzaian A, Bahrami S, Niknejad H.
PMID: 34795361
Sci Rep. 2021 Nov 18;11(1):22508. doi: 10.1038/s41598-021-01922-y.

Decellularized and de-epithelialized placenta membranes have widely been used as scaffolds and grafts in tissue engineering and regenerative medicine. Exceptional pro-angiogenic and biomechanical properties and low immunogenicity have made the amniochorionic membrane a unique substrate which provides an enriched...

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Shariatzadeh S, Shafiee S, Zafari A, et al. Developing a pro-angiogenic placenta derived amniochorionic scaffold with two exposed basement membranes as substrates for cultivating endothelial cells. Sci Rep. 2021;11(1):22508doi: 10.1038/s41598-021-01922-y.
Shariatzadeh, S., Shafiee, S., Zafari, A., Tayebi, T., Yazdanpanah, G., Majd, A., Haj-Mirzaian, A., Bahrami, S., & Niknejad, H. (2021). Developing a pro-angiogenic placenta derived amniochorionic scaffold with two exposed basement membranes as substrates for cultivating endothelial cells. Scientific reports, 11(1), 22508. https://doi.org/10.1038/s41598-021-01922-y
Shariatzadeh, Siavash, et al. "Developing a pro-angiogenic placenta derived amniochorionic scaffold with two exposed basement membranes as substrates for cultivating endothelial cells." Scientific reports vol. 11,1 (2021): 22508. doi: https://doi.org/10.1038/s41598-021-01922-y
Shariatzadeh S, Shafiee S, Zafari A, Tayebi T, Yazdanpanah G, Majd A, Haj-Mirzaian A, Bahrami S, Niknejad H. Developing a pro-angiogenic placenta derived amniochorionic scaffold with two exposed basement membranes as substrates for cultivating endothelial cells. Sci Rep. 2021 Nov 18;11(1):22508. doi: 10.1038/s41598-021-01922-y. PMID: 34795361; PMCID: PMC8602627.
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Targeted Mitochondrial Therapy With Over-Expressed MAVS Protein From Mesenchymal Stem Cells: A New Therapeutic Approach for COVID-19.

Frontiers in cell and developmental biology

Babajani A, Hosseini-Monfared P, Abbaspour S, Jamshidi E, Niknejad H.
PMID: 34179022
Front Cell Dev Biol. 2021 Jun 11;9:695362. doi: 10.3389/fcell.2021.695362. eCollection 2021.

The SARS-CoV-2, the virus that causes COVID-19, has infected millions of people worldwide. The symptoms of this disease are primarily due to pulmonary involvement, uncontrolled tissue inflammation, and inadequate immune response against the invader virus. Impaired interferon (IFN) production...

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Babajani A, Hosseini-Monfared P, Abbaspour S, et al. Targeted Mitochondrial Therapy With Over-Expressed MAVS Protein From Mesenchymal Stem Cells: A New Therapeutic Approach for COVID-19. Front Cell Dev Biol. 2021;9:695362doi: 10.3389/fcell.2021.695362.
Babajani, A., Hosseini-Monfared, P., Abbaspour, S., Jamshidi, E., & Niknejad, H. (2021). Targeted Mitochondrial Therapy With Over-Expressed MAVS Protein From Mesenchymal Stem Cells: A New Therapeutic Approach for COVID-19. Frontiers in cell and developmental biology, 9695362. https://doi.org/10.3389/fcell.2021.695362
Babajani, Amirhesam, et al. "Targeted Mitochondrial Therapy With Over-Expressed MAVS Protein From Mesenchymal Stem Cells: A New Therapeutic Approach for COVID-19." Frontiers in cell and developmental biology vol. 9 (2021): 695362. doi: https://doi.org/10.3389/fcell.2021.695362
Babajani A, Hosseini-Monfared P, Abbaspour S, Jamshidi E, Niknejad H. Targeted Mitochondrial Therapy With Over-Expressed MAVS Protein From Mesenchymal Stem Cells: A New Therapeutic Approach for COVID-19. Front Cell Dev Biol. 2021 Jun 11;9:695362. doi: 10.3389/fcell.2021.695362. eCollection 2021. PMID: 34179022; PMCID: PMC8226075.
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Developing Cytokine Storm-Sensitive Therapeutic Strategy in COVID-19 Using 8P9R Chimeric Peptide and Soluble ACE2.

Frontiers in cell and developmental biology

Nazerian Y, Vakili K, Ebrahimi A, Niknejad H.
PMID: 34540833
Front Cell Dev Biol. 2021 Sep 03;9:717587. doi: 10.3389/fcell.2021.717587. eCollection 2021.

Currently, the COVID-19 pandemic is an international challenge, largely due to lack of effective therapies. Pharmacotherapy has not yet been able to find a definitive treatment for COVID-19. Since SARS-CoV-2 affects several organs, treatment strategies that target the virus...

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Nazerian Y, Vakili K, Ebrahimi A, et al. Developing Cytokine Storm-Sensitive Therapeutic Strategy in COVID-19 Using 8P9R Chimeric Peptide and Soluble ACE2. Front Cell Dev Biol. 2021;9:717587doi: 10.3389/fcell.2021.717587.
Nazerian, Y., Vakili, K., Ebrahimi, A., & Niknejad, H. (2021). Developing Cytokine Storm-Sensitive Therapeutic Strategy in COVID-19 Using 8P9R Chimeric Peptide and Soluble ACE2. Frontiers in cell and developmental biology, 9717587. https://doi.org/10.3389/fcell.2021.717587
Nazerian, Yasaman, et al. "Developing Cytokine Storm-Sensitive Therapeutic Strategy in COVID-19 Using 8P9R Chimeric Peptide and Soluble ACE2." Frontiers in cell and developmental biology vol. 9 (2021): 717587. doi: https://doi.org/10.3389/fcell.2021.717587
Nazerian Y, Vakili K, Ebrahimi A, Niknejad H. Developing Cytokine Storm-Sensitive Therapeutic Strategy in COVID-19 Using 8P9R Chimeric Peptide and Soluble ACE2. Front Cell Dev Biol. 2021 Sep 03;9:717587. doi: 10.3389/fcell.2021.717587. eCollection 2021. PMID: 34540833; PMCID: PMC8446510.
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A Review on the Kinetics of Iron Ore Reduction by Hydrogen.

Materials (Basel, Switzerland)

Heidari A, Niknahad N, Iljana M, Fabritius T.
PMID: 34947143
Materials (Basel). 2021 Dec 09;14(24). doi: 10.3390/ma14247540.

A clean energy revolution is occurring across the world. As iron and steelmaking have a tremendous impact on the amount of CO

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Heidari A, Niknahad N, Iljana M, et al. A Review on the Kinetics of Iron Ore Reduction by Hydrogen. Materials (Basel). 2021;14(24)doi: 10.3390/ma14247540.
Heidari, A., Niknahad, N., Iljana, M., & Fabritius, T. (2021). A Review on the Kinetics of Iron Ore Reduction by Hydrogen. Materials (Basel, Switzerland), 14(24), . https://doi.org/10.3390/ma14247540
Heidari, Aidin, et al. "A Review on the Kinetics of Iron Ore Reduction by Hydrogen." Materials (Basel, Switzerland) vol. 14,24 (2021). doi: https://doi.org/10.3390/ma14247540
Heidari A, Niknahad N, Iljana M, Fabritius T. A Review on the Kinetics of Iron Ore Reduction by Hydrogen. Materials (Basel). 2021 Dec 09;14(24). doi: 10.3390/ma14247540. PMID: 34947143.
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Oral administration of thiol-reducing agents mitigates gut barrier disintegrity and bacterial lipopolysaccharide translocation in a rat model of biliary obstruction.

Current research in pharmacology and drug discovery

Ommati MM, Farshad O, Niknahad H, Mousavi K, Moein M, Azarpira N, Mohammadi H, Jamshidzadeh A, Heidari R.
PMID: 34909638
Curr Res Pharmacol Drug Discov. 2020 Jun 16;1:10-18. doi: 10.1016/j.crphar.2020.06.001. eCollection 2020 Apr.

It has been well documented that cirrhosis is associated with the intestinal injury. Intestinal injury in cirrhosis could lead to bacterial lipopolysaccharide (LPS) translocation to the systemic circulation. It has been found that high plasma LPS is connected with...

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Ommati MM, Farshad O, Niknahad H, et al. Oral administration of thiol-reducing agents mitigates gut barrier disintegrity and bacterial lipopolysaccharide translocation in a rat model of biliary obstruction. Curr Res Pharmacol Drug Discov. 2020;1:10-18doi: 10.1016/j.crphar.2020.06.001.
Ommati, M. M., Farshad, O., Niknahad, H., Mousavi, K., Moein, M., Azarpira, N., Mohammadi, H., Jamshidzadeh, A., & Heidari, R. (2020). Oral administration of thiol-reducing agents mitigates gut barrier disintegrity and bacterial lipopolysaccharide translocation in a rat model of biliary obstruction. Current research in pharmacology and drug discovery, 110-18. https://doi.org/10.1016/j.crphar.2020.06.001
Ommati, Mohammad Mehdi, et al. "Oral administration of thiol-reducing agents mitigates gut barrier disintegrity and bacterial lipopolysaccharide translocation in a rat model of biliary obstruction." Current research in pharmacology and drug discovery vol. 1 (2020): 10-18. doi: https://doi.org/10.1016/j.crphar.2020.06.001
Ommati MM, Farshad O, Niknahad H, Mousavi K, Moein M, Azarpira N, Mohammadi H, Jamshidzadeh A, Heidari R. Oral administration of thiol-reducing agents mitigates gut barrier disintegrity and bacterial lipopolysaccharide translocation in a rat model of biliary obstruction. Curr Res Pharmacol Drug Discov. 2020 Jun 16;1:10-18. doi: 10.1016/j.crphar.2020.06.001. eCollection 2020 Apr. PMID: 34909638; PMCID: PMC8663936.
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Poly (ADP-Ribose) polymerase-1 (PARP-1) overactivity plays a pathogenic role in bile acids-induced nephrotoxicity in cholestatic rats.

Toxicology letters

Siavashpour A, Khalvati B, Azarpira N, Mohammadi H, Niknahad H, Heidari R.
PMID: 32422328
Toxicol Lett. 2020 May 16;330:144-158. doi: 10.1016/j.toxlet.2020.05.012. Epub 2020 May 16.

Cholestatic liver disease is a clinical complication with a wide range of etiologies. The liver is the primary organ influenced by cholestasis. Other organs, rather than the liver (e.g., kidneys), could also be affected by cholestatic liver disease. Cholestasis-induced...

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Siavashpour A, Khalvati B, Azarpira N, et al. Poly (ADP-Ribose) polymerase-1 (PARP-1) overactivity plays a pathogenic role in bile acids-induced nephrotoxicity in cholestatic rats. Toxicol Lett. 2020;330:144-158doi: 10.1016/j.toxlet.2020.05.012.
Siavashpour, A., Khalvati, B., Azarpira, N., Mohammadi, H., Niknahad, H., & Heidari, R. (2020). Poly (ADP-Ribose) polymerase-1 (PARP-1) overactivity plays a pathogenic role in bile acids-induced nephrotoxicity in cholestatic rats. Toxicology letters, 330144-158. https://doi.org/10.1016/j.toxlet.2020.05.012
Siavashpour, Asma, et al. "Poly (ADP-Ribose) polymerase-1 (PARP-1) overactivity plays a pathogenic role in bile acids-induced nephrotoxicity in cholestatic rats." Toxicology letters vol. 330 (2020): 144-158. doi: https://doi.org/10.1016/j.toxlet.2020.05.012
Siavashpour A, Khalvati B, Azarpira N, Mohammadi H, Niknahad H, Heidari R. Poly (ADP-Ribose) polymerase-1 (PARP-1) overactivity plays a pathogenic role in bile acids-induced nephrotoxicity in cholestatic rats. Toxicol Lett. 2020 May 16;330:144-158. doi: 10.1016/j.toxlet.2020.05.012. Epub 2020 May 16. PMID: 32422328.
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