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Orotracheal treprostinil administration attenuates bleomycin-induced lung injury, vascular remodeling, and fibrosis in mice.

Pulmonary circulation

Nikitopoulou I, Manitsopoulos N, Kotanidou A, Tian X, Petrovic A, Magkou C, Ninou I, Aidinis V, Schermuly RT, Kosanovic D, Orfanos SE.
PMID: 31819797
Pulm Circ. 2019 Nov 15;9(4):2045894019881954. doi: 10.1177/2045894019881954. eCollection 2019.

Pulmonary fibrosis is a progressive disease characterized by disruption of lung architecture and deregulation of the pulmonary function. Prostacyclin, a metabolite of arachidonic acid, is a potential disease mediator since it exerts anti-inflammatory and anti-fibrotic actions. We investigated the...

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Nikitopoulou I, Manitsopoulos N, Kotanidou A, et al. Orotracheal treprostinil administration attenuates bleomycin-induced lung injury, vascular remodeling, and fibrosis in mice. Pulm Circ. 2019;9(4):2045894019881954doi: 10.1177/2045894019881954.
Nikitopoulou, I., Manitsopoulos, N., Kotanidou, A., Tian, X., Petrovic, A., Magkou, C., Ninou, I., Aidinis, V., Schermuly, R. T., Kosanovic, D., & Orfanos, S. E. (2019). Orotracheal treprostinil administration attenuates bleomycin-induced lung injury, vascular remodeling, and fibrosis in mice. Pulmonary circulation, 9(4), 2045894019881954. https://doi.org/10.1177/2045894019881954
Nikitopoulou, Ioanna, et al. "Orotracheal treprostinil administration attenuates bleomycin-induced lung injury, vascular remodeling, and fibrosis in mice." Pulmonary circulation vol. 9,4 (2019): 2045894019881954. doi: https://doi.org/10.1177/2045894019881954
Nikitopoulou I, Manitsopoulos N, Kotanidou A, Tian X, Petrovic A, Magkou C, Ninou I, Aidinis V, Schermuly RT, Kosanovic D, Orfanos SE. Orotracheal treprostinil administration attenuates bleomycin-induced lung injury, vascular remodeling, and fibrosis in mice. Pulm Circ. 2019 Nov 15;9(4):2045894019881954. doi: 10.1177/2045894019881954. eCollection 2019. PMID: 31819797; PMCID: PMC6883672.
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Empowering newborn screening programs in African countries through establishment of an international collaborative effort.

Journal of community genetics

Therrell BL, Lloyd-Puryear MA, Ohene-Frempong K, Ware RE, Padilla CD, Ambrose EE, Barkat A, Ghazal H, Kiyaga C, Mvalo T, Nnodu O, Ouldim K, Rahimy MC, Santos B, Tshilolo L, Yusuf C, Zarbalian G, Watson MS.
PMID: 32415570
J Community Genet. 2020 Jul;11(3):253-268. doi: 10.1007/s12687-020-00463-7. Epub 2020 May 15.

In an effort to explore new knowledge and to develop meaningful collaborations for improving child health, the First Pan African Workshop on Newborn Screening was convened in June 2019 in Rabat, Morocco. Participants included an informal network of newborn...

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Therrell BL, Lloyd-Puryear MA, Ohene-Frempong K, et al. Empowering newborn screening programs in African countries through establishment of an international collaborative effort. J Community Genet. 2020;11(3):253-268doi: 10.1007/s12687-020-00463-7.
Therrell, B. L., Lloyd-Puryear, M. A., Ohene-Frempong, K., Ware, R. E., Padilla, C. D., Ambrose, E. E., Barkat, A., Ghazal, H., Kiyaga, C., Mvalo, T., Nnodu, O., Ouldim, K., Rahimy, M. C., Santos, B., Tshilolo, L., Yusuf, C., Zarbalian, G., Watson, M. S. (2020). Empowering newborn screening programs in African countries through establishment of an international collaborative effort. Journal of community genetics, 11(3), 253-268. https://doi.org/10.1007/s12687-020-00463-7
Therrell, Bradford L, et al. "Empowering newborn screening programs in African countries through establishment of an international collaborative effort." Journal of community genetics vol. 11,3 (2020): 253-268. doi: https://doi.org/10.1007/s12687-020-00463-7
Therrell BL, Lloyd-Puryear MA, Ohene-Frempong K, Ware RE, Padilla CD, Ambrose EE, Barkat A, Ghazal H, Kiyaga C, Mvalo T, Nnodu O, Ouldim K, Rahimy MC, Santos B, Tshilolo L, Yusuf C, Zarbalian G, Watson MS. Empowering newborn screening programs in African countries through establishment of an international collaborative effort. J Community Genet. 2020 Jul;11(3):253-268. doi: 10.1007/s12687-020-00463-7. Epub 2020 May 15. PMID: 32415570; PMCID: PMC7295888.
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Correction to: Detailed clinical features and genotype-phenotype correlation in an OTOF-related hearing loss cohort in Japan.

Human genetics

Iwasa YI, Nishio SY, Yoshimura H, Sugaya A, Kataoka Y, Maeda Y, Kanda Y, Nagai K, Naito Y, Yamazaki H, Ikezono T, Matsuda H, Nakai M, Tona R, Sakurai Y, Motegi R, Takeda H, Kobayashi M, Kihara C, Ishino T, Morita SY, Iwasaki S, Takahashi M, Furutate S, Oka SI, Kubota T, Arai Y, Kobayashi Y, Kikuchi D, Shintani T, Ogasawara N, Honkura Y, Izumi S, Hyogo M, Ninoyu Y, Suematsu M, Nakayama J, Tsuchihashi N, Okami M, Sakata H, Yoshihashi H, Kobayashi T, Kumakawa K, Yoshida T, Esaki T, Usami SI.
PMID: 34727261
Hum Genet. 2021 Nov 02; doi: 10.1007/s00439-021-02392-y. Epub 2021 Nov 02.

No abstract available.

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Iwasa YI, Nishio SY, Yoshimura H, et al. Correction to: Detailed clinical features and genotype-phenotype correlation in an OTOF-related hearing loss cohort in Japan. Hum Genet. 2021;doi: 10.1007/s00439-021-02392-y.
Iwasa, Y. I., Nishio, S. Y., Yoshimura, H., Sugaya, A., Kataoka, Y., Maeda, Y., Kanda, Y., Nagai, K., Naito, Y., Yamazaki, H., Ikezono, T., Matsuda, H., Nakai, M., Tona, R., Sakurai, Y., Motegi, R., Takeda, H., Kobayashi, M., Kihara, C., Ishino, T., Morita, S. Y., Iwasaki, S., Takahashi, M., Furutate, S., Oka, S. I., Kubota, T., Arai, Y., Kobayashi, Y., Kikuchi, D., Shintani, T., Ogasawara, N., Honkura, Y., Izumi, S., Hyogo, M., Ninoyu, Y., Suematsu, M., Nakayama, J., Tsuchihashi, N., Okami, M., Sakata, H., Yoshihashi, H., Kobayashi, T., Kumakawa, K., Yoshida, T., Esaki, T., & Usami, S. I. (2021). Correction to: Detailed clinical features and genotype-phenotype correlation in an OTOF-related hearing loss cohort in Japan. Human genetics, . https://doi.org/10.1007/s00439-021-02392-y
Iwasa, Yoh-Ichiro, et al. "Correction to: Detailed clinical features and genotype-phenotype correlation in an OTOF-related hearing loss cohort in Japan." Human genetics vol. (2021). doi: https://doi.org/10.1007/s00439-021-02392-y
Iwasa YI, Nishio SY, Yoshimura H, Sugaya A, Kataoka Y, Maeda Y, Kanda Y, Nagai K, Naito Y, Yamazaki H, Ikezono T, Matsuda H, Nakai M, Tona R, Sakurai Y, Motegi R, Takeda H, Kobayashi M, Kihara C, Ishino T, Morita SY, Iwasaki S, Takahashi M, Furutate S, Oka SI, Kubota T, Arai Y, Kobayashi Y, Kikuchi D, Shintani T, Ogasawara N, Honkura Y, Izumi S, Hyogo M, Ninoyu Y, Suematsu M, Nakayama J, Tsuchihashi N, Okami M, Sakata H, Yoshihashi H, Kobayashi T, Kumakawa K, Yoshida T, Esaki T, Usami SI. Correction to: Detailed clinical features and genotype-phenotype correlation in an OTOF-related hearing loss cohort in Japan. Hum Genet. 2021 Nov 02; doi: 10.1007/s00439-021-02392-y. Epub 2021 Nov 02. PMID: 34727261.
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Exploring Animal Models That Resemble Idiopathic Pulmonary Fibrosis.

Frontiers in medicine

Tashiro J, Rubio GA, Limper AH, Williams K, Elliot SJ, Ninou I, Aidinis V, Tzouvelekis A, Glassberg MK.
PMID: 28804709
Front Med (Lausanne). 2017 Jul 28;4:118. doi: 10.3389/fmed.2017.00118. eCollection 2017.

Large multicenter clinical trials have led to two recently approved drugs for patients with idiopathic pulmonary fibrosis (IPF); yet, both of these therapies only slow disease progression and do not provide a definitive cure. Traditionally, preclinical trials have utilized...

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Tashiro J, Rubio GA, Limper AH, et al. Exploring Animal Models That Resemble Idiopathic Pulmonary Fibrosis. Front Med (Lausanne). 2017;4:118doi: 10.3389/fmed.2017.00118.
Tashiro, J., Rubio, G. A., Limper, A. H., Williams, K., Elliot, S. J., Ninou, I., Aidinis, V., Tzouvelekis, A., & Glassberg, M. K. (2017). Exploring Animal Models That Resemble Idiopathic Pulmonary Fibrosis. Frontiers in medicine, 4118. https://doi.org/10.3389/fmed.2017.00118
Tashiro, Jun, et al. "Exploring Animal Models That Resemble Idiopathic Pulmonary Fibrosis." Frontiers in medicine vol. 4 (2017): 118. doi: https://doi.org/10.3389/fmed.2017.00118
Tashiro J, Rubio GA, Limper AH, Williams K, Elliot SJ, Ninou I, Aidinis V, Tzouvelekis A, Glassberg MK. Exploring Animal Models That Resemble Idiopathic Pulmonary Fibrosis. Front Med (Lausanne). 2017 Jul 28;4:118. doi: 10.3389/fmed.2017.00118. eCollection 2017. PMID: 28804709; PMCID: PMC5532376.
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Salidroside Promotes Random Skin Flap Survival in Rats by Enhancing Angiogenesis and Inhibiting Apoptosis.

Journal of reconstructive microsurgery

Deheng C, Kailiang Z, Weidong W, Haiming J, Daoliang X, Ningyu C, Huazi X.
PMID: 27276197
J Reconstr Microsurg. 2016 Oct;32(8):580-586. doi: 10.1055/s-0036-1584205. Epub 2016 Jun 08.

No abstract available.

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Deheng C, Kailiang Z, Weidong W, et al. Salidroside Promotes Random Skin Flap Survival in Rats by Enhancing Angiogenesis and Inhibiting Apoptosis. J Reconstr Microsurg. 2016;32(8):580-586doi: 10.1055/s-0036-1584205.
Deheng, C., Kailiang, Z., Weidong, W., Haiming, J., Daoliang, X., Ningyu, C., & Huazi, X. (2016). Salidroside Promotes Random Skin Flap Survival in Rats by Enhancing Angiogenesis and Inhibiting Apoptosis. Journal of reconstructive microsurgery, 32(8), 580-586. https://doi.org/10.1055/s-0036-1584205
Deheng, Chen, et al. "Salidroside Promotes Random Skin Flap Survival in Rats by Enhancing Angiogenesis and Inhibiting Apoptosis." Journal of reconstructive microsurgery vol. 32,8 (2016): 580-586. doi: https://doi.org/10.1055/s-0036-1584205
Deheng C, Kailiang Z, Weidong W, Haiming J, Daoliang X, Ningyu C, Huazi X. Salidroside Promotes Random Skin Flap Survival in Rats by Enhancing Angiogenesis and Inhibiting Apoptosis. J Reconstr Microsurg. 2016 Oct;32(8):580-586. doi: 10.1055/s-0036-1584205. Epub 2016 Jun 08. PMID: 27276197.
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Autotaxin in Pathophysiology and Pulmonary Fibrosis.

Frontiers in medicine

Ninou I, Magkrioti C, Aidinis V.
PMID: 29951481
Front Med (Lausanne). 2018 Jun 13;5:180. doi: 10.3389/fmed.2018.00180. eCollection 2018.

Lysophospholipid signaling is emerging as a druggable regulator of pathophysiological responses, and especially fibrosis, exemplified by the relative ongoing clinical trials in idiopathic pulmonary fibrosis (IPF) patients. In this review, we focus on ectonucleotide pyrophosphatase-phosphodiesterase 2 (ENPP2), or as...

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Ninou I, Magkrioti C, Aidinis V. Autotaxin in Pathophysiology and Pulmonary Fibrosis. Front Med (Lausanne). 2018;5:180doi: 10.3389/fmed.2018.00180.
Ninou, I., Magkrioti, C., & Aidinis, V. (2018). Autotaxin in Pathophysiology and Pulmonary Fibrosis. Frontiers in medicine, 5180. https://doi.org/10.3389/fmed.2018.00180
Ninou, Ioanna, et al. "Autotaxin in Pathophysiology and Pulmonary Fibrosis." Frontiers in medicine vol. 5 (2018): 180. doi: https://doi.org/10.3389/fmed.2018.00180
Ninou I, Magkrioti C, Aidinis V. Autotaxin in Pathophysiology and Pulmonary Fibrosis. Front Med (Lausanne). 2018 Jun 13;5:180. doi: 10.3389/fmed.2018.00180. eCollection 2018. PMID: 29951481; PMCID: PMC6008954.
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Detailed clinical features and genotype-phenotype correlation in an OTOF-related hearing loss cohort in Japan.

Human genetics

Iwasa YI, Nishio SY, Yoshimura H, Sugaya A, Kataoka Y, Maeda Y, Kanda Y, Nagai K, Naito Y, Yamazaki H, Ikezono T, Matsuda H, Nakai M, Tona R, Sakurai Y, Motegi R, Takeda H, Kobayashi M, Kihara C, Ishino T, Morita SY, Iwasaki S, Takahashi M, Furutate S, Oka SI, Kubota T, Arai Y, Kobayashi Y, Kikuchi D, Shintani T, Ogasawara N, Honkura Y, Izumi S, Hyogo M, Ninoyu Y, Suematsu M, Nakayama J, Tsuchihashi N, Okami M, Sakata H, Yoshihashi H, Kobayashi T, Kumakawa K, Yoshida T, Esaki T, Usami SI.
PMID: 34536124
Hum Genet. 2021 Sep 18; doi: 10.1007/s00439-021-02351-7. Epub 2021 Sep 18.

Mutations in the OTOF gene are a common cause of hereditary hearing loss and the main cause of auditory neuropathy spectrum disorder (ANSD). Although it is reported that most of the patients with OTOF mutations have stable, congenital or...

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Iwasa YI, Nishio SY, Yoshimura H, et al. Detailed clinical features and genotype-phenotype correlation in an OTOF-related hearing loss cohort in Japan. Hum Genet. 2021;doi: 10.1007/s00439-021-02351-7.
Iwasa, Y. I., Nishio, S. Y., Yoshimura, H., Sugaya, A., Kataoka, Y., Maeda, Y., Kanda, Y., Nagai, K., Naito, Y., Yamazaki, H., Ikezono, T., Matsuda, H., Nakai, M., Tona, R., Sakurai, Y., Motegi, R., Takeda, H., Kobayashi, M., Kihara, C., Ishino, T., Morita, S. Y., Iwasaki, S., Takahashi, M., Furutate, S., Oka, S. I., Kubota, T., Arai, Y., Kobayashi, Y., Kikuchi, D., Shintani, T., Ogasawara, N., Honkura, Y., Izumi, S., Hyogo, M., Ninoyu, Y., Suematsu, M., Nakayama, J., Tsuchihashi, N., Okami, M., Sakata, H., Yoshihashi, H., Kobayashi, T., Kumakawa, K., Yoshida, T., Esaki, T., & Usami, S. I. (2021). Detailed clinical features and genotype-phenotype correlation in an OTOF-related hearing loss cohort in Japan. Human genetics, . https://doi.org/10.1007/s00439-021-02351-7
Iwasa, Yoh-Ichiro, et al. "Detailed clinical features and genotype-phenotype correlation in an OTOF-related hearing loss cohort in Japan." Human genetics vol. (2021). doi: https://doi.org/10.1007/s00439-021-02351-7
Iwasa YI, Nishio SY, Yoshimura H, Sugaya A, Kataoka Y, Maeda Y, Kanda Y, Nagai K, Naito Y, Yamazaki H, Ikezono T, Matsuda H, Nakai M, Tona R, Sakurai Y, Motegi R, Takeda H, Kobayashi M, Kihara C, Ishino T, Morita SY, Iwasaki S, Takahashi M, Furutate S, Oka SI, Kubota T, Arai Y, Kobayashi Y, Kikuchi D, Shintani T, Ogasawara N, Honkura Y, Izumi S, Hyogo M, Ninoyu Y, Suematsu M, Nakayama J, Tsuchihashi N, Okami M, Sakata H, Yoshihashi H, Kobayashi T, Kumakawa K, Yoshida T, Esaki T, Usami SI. Detailed clinical features and genotype-phenotype correlation in an OTOF-related hearing loss cohort in Japan. Hum Genet. 2021 Sep 18; doi: 10.1007/s00439-021-02351-7. Epub 2021 Sep 18. PMID: 34536124.
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Implementation science research for the scale-up of evidence-based interventions for sickle cell disease in africa: a commentary.

Globalization and health

Gyamfi J, Ojo T, Iwelunmor J, Ogedegbe G, Ryan N, Diawara A, Nnodu O, Wonkam A, Royal C, Peprah E.
PMID: 33596947
Global Health. 2021 Feb 17;17(1):20. doi: 10.1186/s12992-021-00671-x.

BACKGROUND: The burden of sickle cell disease (SCD) is greatest among African nations. Effective scalability of evidence-based interventions (e.g., newborn screening, health education, prophylaxis for infection, optimal nutrition and hydration, hydroxyurea therapy, blood transfusions, and transcranial Doppler (TCD) screening)...

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Gyamfi J, Ojo T, Iwelunmor J, et al. Implementation science research for the scale-up of evidence-based interventions for sickle cell disease in africa: a commentary. Global Health. 2021;17(1):20doi: 10.1186/s12992-021-00671-x.
Gyamfi, J., Ojo, T., Iwelunmor, J., Ogedegbe, G., Ryan, N., Diawara, A., Nnodu, O., Wonkam, A., Royal, C., & Peprah, E. (2021). Implementation science research for the scale-up of evidence-based interventions for sickle cell disease in africa: a commentary. Globalization and health, 17(1), 20. https://doi.org/10.1186/s12992-021-00671-x
Gyamfi, Joyce, et al. "Implementation science research for the scale-up of evidence-based interventions for sickle cell disease in africa: a commentary." Globalization and health vol. 17,1 (2021): 20. doi: https://doi.org/10.1186/s12992-021-00671-x
Gyamfi J, Ojo T, Iwelunmor J, Ogedegbe G, Ryan N, Diawara A, Nnodu O, Wonkam A, Royal C, Peprah E. Implementation science research for the scale-up of evidence-based interventions for sickle cell disease in africa: a commentary. Global Health. 2021 Feb 17;17(1):20. doi: 10.1186/s12992-021-00671-x. PMID: 33596947; PMCID: PMC7888072.
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Long Non-Coding RNA .

Frontiers in cell and developmental biology

Ninou E, Michail A, Politis PK.
PMID: 34900989
Front Cell Dev Biol. 2021 Nov 24;9:726857. doi: 10.3389/fcell.2021.726857. eCollection 2021.

Although long non-coding RNAs (lncRNAs) is one of the most abundant classes of RNAs encoded within the mammalian genome and are highly expressed in the adult brain, they remain poorly characterized and their roles in the brain development are...

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Ninou E, Michail A, Politis PK. Long Non-Coding RNA . Front Cell Dev Biol. 2021;9:726857doi: 10.3389/fcell.2021.726857.
Ninou, E., Michail, A., & Politis, P. K. (2021). Long Non-Coding RNA . Frontiers in cell and developmental biology, 9726857. https://doi.org/10.3389/fcell.2021.726857
Ninou, Elpinickie, et al. "Long Non-Coding RNA ." Frontiers in cell and developmental biology vol. 9 (2021): 726857. doi: https://doi.org/10.3389/fcell.2021.726857
Ninou E, Michail A, Politis PK. Long Non-Coding RNA . Front Cell Dev Biol. 2021 Nov 24;9:726857. doi: 10.3389/fcell.2021.726857. eCollection 2021. PMID: 34900989; PMCID: PMC8653915.
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Proceedings of a Sickle Cell Disease Ontology workshop - Towards the first comprehensive ontology for Sickle Cell Disease.

Applied & translational genomics

Mulder N, Nembaware V, Adekile A, Anie KA, Inusa B, Brown B, Campbell A, Chinenere F, Chunda-Liyoka C, Derebail VK, Geard A, Ghedira K, Hamilton CM, Hanchard NA, Haendel M, Huggins W, Ibrahim M, Jupp S, Kamga KK, Knight-Madden J, Lopez-Sall P, Mbiyavanga M, Munube D, Nirenberg D, Nnodu O, Ofori-Acquah SF, Ohene-Frempong K, Opap KB, Panji S, Park M, Pule G, Royal C, Sangeda R, Tayo B, Treadwell M, Tshilolo L, Wonkam A.
PMID: 27354937
Appl Transl Genom. 2016 Mar 15;9:23-9. doi: 10.1016/j.atg.2016.03.005. eCollection 2016 Jun.

Sickle cell disease (SCD) is a debilitating single gene disorder caused by a single point mutation that results in physical deformation (i.e. sickling) of erythrocytes at reduced oxygen tensions. Up to 75% of SCD in newborns world-wide occurs in...

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Mulder N, Nembaware V, Adekile A, et al. Proceedings of a Sickle Cell Disease Ontology workshop - Towards the first comprehensive ontology for Sickle Cell Disease. Appl Transl Genom. 2016;9:23-9doi: 10.1016/j.atg.2016.03.005.
Mulder, N., Nembaware, V., Adekile, A., Anie, K. A., Inusa, B., Brown, B., Campbell, A., Chinenere, F., Chunda-Liyoka, C., Derebail, V. K., Geard, A., Ghedira, K., Hamilton, C. M., Hanchard, N. A., Haendel, M., Huggins, W., Ibrahim, M., Jupp, S., Kamga, K. K., Knight-Madden, J., Lopez-Sall, P., Mbiyavanga, M., Munube, D., Nirenberg, D., Nnodu, O., Ofori-Acquah, S. F., Ohene-Frempong, K., Opap, K. B., Panji, S., Park, M., Pule, G., Royal, C., Sangeda, R., Tayo, B., Treadwell, M., Tshilolo, L., & Wonkam, A. (2016). Proceedings of a Sickle Cell Disease Ontology workshop - Towards the first comprehensive ontology for Sickle Cell Disease. Applied & translational genomics, 923-9. https://doi.org/10.1016/j.atg.2016.03.005
Mulder, Nicola, et al. "Proceedings of a Sickle Cell Disease Ontology workshop - Towards the first comprehensive ontology for Sickle Cell Disease." Applied & translational genomics vol. 9 (2016): 23-9. doi: https://doi.org/10.1016/j.atg.2016.03.005
Mulder N, Nembaware V, Adekile A, Anie KA, Inusa B, Brown B, Campbell A, Chinenere F, Chunda-Liyoka C, Derebail VK, Geard A, Ghedira K, Hamilton CM, Hanchard NA, Haendel M, Huggins W, Ibrahim M, Jupp S, Kamga KK, Knight-Madden J, Lopez-Sall P, Mbiyavanga M, Munube D, Nirenberg D, Nnodu O, Ofori-Acquah SF, Ohene-Frempong K, Opap KB, Panji S, Park M, Pule G, Royal C, Sangeda R, Tayo B, Treadwell M, Tshilolo L, Wonkam A. Proceedings of a Sickle Cell Disease Ontology workshop - Towards the first comprehensive ontology for Sickle Cell Disease. Appl Transl Genom. 2016 Mar 15;9:23-9. doi: 10.1016/j.atg.2016.03.005. eCollection 2016 Jun. PMID: 27354937; PMCID: PMC4911424.
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Corrigendum to "Sickle cell disease clinical phenotypes in Nigeria: A preliminary analysis of the Sickle Pan Africa Research Consortium Nigeria database" [Blood Cells, Molecules and Diseases (2020) Start page-End page/202438].

Blood cells, molecules & diseases

Isa H, Adegoke S, Madu A, Hassan AA, Ohiaeri C, Chianumba R, Brown B, Okocha E, Ugwu N, Diaku-Akinwumi I, Adeyemo T, Kuliya-Gwarzo A, Dogara L, Lawal H, Tanko Y, Ladu A, Kangiwa U, Ekwem L, Oniyangi S, Wakama T, Umoru D, Olanrewaju O, Akinola N, Nnebe-Agumadu U, Asala S, Adekile A, Olaniyi J, Sangeda R, Nnodu O.
PMID: 32739321
Blood Cells Mol Dis. 2021 May;88:102480. doi: 10.1016/j.bcmd.2020.102480. Epub 2020 Jul 29.

No abstract available.

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Isa H, Adegoke S, Madu A, et al. Corrigendum to "Sickle cell disease clinical phenotypes in Nigeria: A preliminary analysis of the Sickle Pan Africa Research Consortium Nigeria database" [Blood Cells, Molecules and Diseases (2020) Start page-End page/202438]. Blood Cells Mol Dis. 2020;88:102480doi: 10.1016/j.bcmd.2020.102480.
Isa, H., Adegoke, S., Madu, A., Hassan, A. A., Ohiaeri, C., Chianumba, R., Brown, B., Okocha, E., Ugwu, N., Diaku-Akinwumi, I., Adeyemo, T., Kuliya-Gwarzo, A., Dogara, L., Lawal, H., Tanko, Y., Ladu, A., Kangiwa, U., Ekwem, L., Oniyangi, S., Wakama, T., Umoru, D., Olanrewaju, O., Akinola, N., Nnebe-Agumadu, U., Asala, S., Adekile, A., Olaniyi, J., Sangeda, R., & Nnodu, O. (2021). Corrigendum to "Sickle cell disease clinical phenotypes in Nigeria: A preliminary analysis of the Sickle Pan Africa Research Consortium Nigeria database" [Blood Cells, Molecules and Diseases (2020) Start page-End page/202438]. Blood cells, molecules & diseases, 88102480. https://doi.org/10.1016/j.bcmd.2020.102480
Isa, Hezekiah, et al. "Corrigendum to "Sickle cell disease clinical phenotypes in Nigeria: A preliminary analysis of the Sickle Pan Africa Research Consortium Nigeria database" [Blood Cells, Molecules and Diseases (2020) Start page-End page/202438]." Blood cells, molecules & diseases vol. 88 (2021): 102480. doi: https://doi.org/10.1016/j.bcmd.2020.102480
Isa H, Adegoke S, Madu A, Hassan AA, Ohiaeri C, Chianumba R, Brown B, Okocha E, Ugwu N, Diaku-Akinwumi I, Adeyemo T, Kuliya-Gwarzo A, Dogara L, Lawal H, Tanko Y, Ladu A, Kangiwa U, Ekwem L, Oniyangi S, Wakama T, Umoru D, Olanrewaju O, Akinola N, Nnebe-Agumadu U, Asala S, Adekile A, Olaniyi J, Sangeda R, Nnodu O. Corrigendum to "Sickle cell disease clinical phenotypes in Nigeria: A preliminary analysis of the Sickle Pan Africa Research Consortium Nigeria database" [Blood Cells, Molecules and Diseases (2020) Start page-End page/202438]. Blood Cells Mol Dis. 2021 May;88:102480. doi: 10.1016/j.bcmd.2020.102480. Epub 2020 Jul 29. PMID: 32739321.
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Long Non-Coding RNA .

Frontiers in cell and developmental biology

Ninou E, Michail A, Politis PK.
PMID: 34900989
Front Cell Dev Biol. 2021 Nov 24;9:726857. doi: 10.3389/fcell.2021.726857. eCollection 2021.

Although long non-coding RNAs (lncRNAs) is one of the most abundant classes of RNAs encoded within the mammalian genome and are highly expressed in the adult brain, they remain poorly characterized and their roles in the brain development are...

Cite
Ninou E, Michail A, Politis PK. Long Non-Coding RNA . Front Cell Dev Biol. 2021;9:726857doi: 10.3389/fcell.2021.726857.
Ninou, E., Michail, A., & Politis, P. K. (2021). Long Non-Coding RNA . Frontiers in cell and developmental biology, 9726857. https://doi.org/10.3389/fcell.2021.726857
Ninou, Elpinickie, et al. "Long Non-Coding RNA ." Frontiers in cell and developmental biology vol. 9 (2021): 726857. doi: https://doi.org/10.3389/fcell.2021.726857
Ninou E, Michail A, Politis PK. Long Non-Coding RNA . Front Cell Dev Biol. 2021 Nov 24;9:726857. doi: 10.3389/fcell.2021.726857. eCollection 2021. PMID: 34900989; PMCID: PMC8653915.
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