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Gray Matter Atrophy Is Primarily Related to Demyelination of Lesions in Multiple Sclerosis: A Diffusion Tensor Imaging MRI Study.

Frontiers in neuroanatomy

Tóth E, Szabó N, Csete G, Király A, Faragó P, Spisák T, Bencsik K, Vécsei L, Kincses ZT.
PMID: 28424595
Front Neuroanat. 2017 Mar 29;11:23. doi: 10.3389/fnana.2017.00023. eCollection 2017.

No abstract available.

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Tóth E, Szabó N, Csete G, et al. Gray Matter Atrophy Is Primarily Related to Demyelination of Lesions in Multiple Sclerosis: A Diffusion Tensor Imaging MRI Study. Front Neuroanat. 2017;11:23doi: 10.3389/fnana.2017.00023.
Tóth, E., Szabó, N., Csete, G., Király, A., Faragó, P., Spisák, T., Bencsik, K., Vécsei, L., & Kincses, Z. T. (2017). Gray Matter Atrophy Is Primarily Related to Demyelination of Lesions in Multiple Sclerosis: A Diffusion Tensor Imaging MRI Study. Frontiers in neuroanatomy, 1123. https://doi.org/10.3389/fnana.2017.00023
Tóth, Eszter, et al. "Gray Matter Atrophy Is Primarily Related to Demyelination of Lesions in Multiple Sclerosis: A Diffusion Tensor Imaging MRI Study." Frontiers in neuroanatomy vol. 11 (2017): 23. doi: https://doi.org/10.3389/fnana.2017.00023
Tóth E, Szabó N, Csete G, Király A, Faragó P, Spisák T, Bencsik K, Vécsei L, Kincses ZT. Gray Matter Atrophy Is Primarily Related to Demyelination of Lesions in Multiple Sclerosis: A Diffusion Tensor Imaging MRI Study. Front Neuroanat. 2017 Mar 29;11:23. doi: 10.3389/fnana.2017.00023. eCollection 2017. PMID: 28424595; PMCID: PMC5372801.
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CGRP antagonists and antibodies for the treatment of migraine.

Expert opinion on investigational drugs

Vécsei L, Szok D, Csáti A, Tajti J.
PMID: 25219387
Expert Opin Investig Drugs. 2015 Jan;24(1):31-41. doi: 10.1517/13543784.2015.960921. Epub 2014 Sep 15.

Introduction: Migraine is a highly devastating neurovascular disorder that affects up to 16% of the population worldwide. In spite of intensive research, its origin remains enigmatic with no therapeutic option appropriate for all migraine patients. One of the leading...

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Vécsei L, Szok D, Csáti A, et al. CGRP antagonists and antibodies for the treatment of migraine. Expert Opin Investig Drugs. 2014;24(1):31-41doi: 10.1517/13543784.2015.960921.
Vécsei, L., Szok, D., Csáti, A., & Tajti, J. (2015). CGRP antagonists and antibodies for the treatment of migraine. Expert opinion on investigational drugs, 24(1), 31-41. https://doi.org/10.1517/13543784.2015.960921
Vécsei, László, et al. "CGRP antagonists and antibodies for the treatment of migraine." Expert opinion on investigational drugs vol. 24,1 (2015): 31-41. doi: https://doi.org/10.1517/13543784.2015.960921
Vécsei L, Szok D, Csáti A, Tajti J. CGRP antagonists and antibodies for the treatment of migraine. Expert Opin Investig Drugs. 2015 Jan;24(1):31-41. doi: 10.1517/13543784.2015.960921. Epub 2014 Sep 15. PMID: 25219387.
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Autolysis and aging of Penicillium chrysogenum cultures under carbon starvation: Chitinase production and antifungal effect of allosamidin.

The Journal of general and applied microbiology

Sámi L, Pusztahelyi T, Emri T, Varecza Z, Fekete A, Grallert A, Karanyi Z, Kiss L, Pócsi I.
PMID: 12483620
J Gen Appl Microbiol. 2001 Aug;47(4):201-211. doi: 10.2323/jgam.47.201.

In carbon-depleted cultures of Penicillium chrysogenum, age-related chitinases were shown to play a crucial role in both autolysis and fragmentation as indicated by in vivo enzyme inhibition experiments using allosamidin. This pseudotrisaccharide even hindered significantly the outgrowth of new...

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Sámi L, Pusztahelyi T, Emri T, et al. Autolysis and aging of Penicillium chrysogenum cultures under carbon starvation: Chitinase production and antifungal effect of allosamidin. J Gen Appl Microbiol. 2001;47(4):201-211doi: 10.2323/jgam.47.201.
Sámi, L., Pusztahelyi, T., Emri, T., Varecza, Z., Fekete, A., Grallert, A., Karanyi, Z., Kiss, L., & Pócsi, I. (2001). Autolysis and aging of Penicillium chrysogenum cultures under carbon starvation: Chitinase production and antifungal effect of allosamidin. The Journal of general and applied microbiology, 47(4), 201-211. https://doi.org/10.2323/jgam.47.201
Sámi, László, et al. "Autolysis and aging of Penicillium chrysogenum cultures under carbon starvation: Chitinase production and antifungal effect of allosamidin." The Journal of general and applied microbiology vol. 47,4 (2001): 201-211. doi: https://doi.org/10.2323/jgam.47.201
Sámi L, Pusztahelyi T, Emri T, Varecza Z, Fekete A, Grallert A, Karanyi Z, Kiss L, Pócsi I. Autolysis and aging of Penicillium chrysogenum cultures under carbon starvation: Chitinase production and antifungal effect of allosamidin. J Gen Appl Microbiol. 2001 Aug;47(4):201-211. doi: 10.2323/jgam.47.201. PMID: 12483620.
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Central precocious puberty and abnormal chromosomal patterns.

Endocrine pathology

Grosso S, Anichini C, Berardi R, Balestri P, Pucci L, Morgese G.
PMID: 15304841
Endocr Pathol. 2000;11(1):69-75. doi: 10.1385/ep:11:1:69.

Central precocious puberty (PP) can be caused by chromosomal aberrations. We report three patients presenting with central PP in whom karyotype analysis demonstrated abnormal chromosomal patterns. The first patient was affected by the triple-X syndrome, commonly characterized by premature...

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Grosso S, Anichini C, Berardi R, et al. Central precocious puberty and abnormal chromosomal patterns. Endocr Pathol. 2000;11(1):69-75doi: 10.1385/ep:11:1:69.
Grosso, S., Anichini, C., Berardi, R., Balestri, P., Pucci, L., & Morgese, G. (2000). Central precocious puberty and abnormal chromosomal patterns. Endocrine pathology, 11(1), 69-75. https://doi.org/10.1385/ep:11:1:69
Grosso, S, et al. "Central precocious puberty and abnormal chromosomal patterns." Endocrine pathology vol. 11,1 (2000): 69-75. doi: https://doi.org/10.1385/ep:11:1:69
Grosso S, Anichini C, Berardi R, Balestri P, Pucci L, Morgese G. Central precocious puberty and abnormal chromosomal patterns. Endocr Pathol. 2000;11(1):69-75. doi: 10.1385/ep:11:1:69. PMID: 15304841.
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The involvement of catecholaminergic mechanisms in the behavioural action of vasopressin.

Neuroscience letters

Kovács GL, Vécsei L, Szabó G, Telegdy G.
PMID: 19605017
Neurosci Lett. 1977 Sep;5(6):337-44. doi: 10.1016/0304-3940(77)90110-0.

The effect of lysine-8-vasopressin (300 mU/kg b.w.) has been tested in a single-trial step-down passive avoidance test. Vasopressin treatment resulted in an improvement of step-down latency in normal rats, but this action disappeared when the animals were pretreated with...

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Kovács GL, Vécsei L, Szabó G, et al. The involvement of catecholaminergic mechanisms in the behavioural action of vasopressin. Neurosci Lett. 1977;5(6):337-44doi: 10.1016/0304-3940(77)90110-0.
Kovács, G. L., Vécsei, L., Szabó, G., & Telegdy, G. (1977). The involvement of catecholaminergic mechanisms in the behavioural action of vasopressin. Neuroscience letters, 5(6), 337-44. https://doi.org/10.1016/0304-3940(77)90110-0
Kovács, G L, et al. "The involvement of catecholaminergic mechanisms in the behavioural action of vasopressin." Neuroscience letters vol. 5,6 (1977): 337-44. doi: https://doi.org/10.1016/0304-3940(77)90110-0
Kovács GL, Vécsei L, Szabó G, Telegdy G. The involvement of catecholaminergic mechanisms in the behavioural action of vasopressin. Neurosci Lett. 1977 Sep;5(6):337-44. doi: 10.1016/0304-3940(77)90110-0. PMID: 19605017.
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Genetic and molecular aspects of spinocerebellar ataxias.

Neuropsychiatric disease and treatment

Honti V, Vécsei L.
PMID: 18568057
Neuropsychiatr Dis Treat. 2005 Jun;1(2):125-33. doi: 10.2147/nedt.1.2.125.61044.

The group of spinocerebellar ataxias (SCAs) includes more than 20 subgroups based only on genetic research. The "ataxia genes" are autosomal; the "disease-alleles" are dominant, and many of them, but not all, encode a protein with an abnormally long...

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Honti V, Vécsei L. Genetic and molecular aspects of spinocerebellar ataxias. Neuropsychiatr Dis Treat. 2005;1(2):125-33doi: 10.2147/nedt.1.2.125.61044.
Honti, V., & Vécsei, L. (2005). Genetic and molecular aspects of spinocerebellar ataxias. Neuropsychiatric disease and treatment, 1(2), 125-33. https://doi.org/10.2147/nedt.1.2.125.61044
Honti, Viktor, and Vécsei, László. "Genetic and molecular aspects of spinocerebellar ataxias." Neuropsychiatric disease and treatment vol. 1,2 (2005): 125-33. doi: https://doi.org/10.2147/nedt.1.2.125.61044
Honti V, Vécsei L. Genetic and molecular aspects of spinocerebellar ataxias. Neuropsychiatr Dis Treat. 2005 Jun;1(2):125-33. doi: 10.2147/nedt.1.2.125.61044. PMID: 18568057; PMCID: PMC2413192.
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Management of multiple sclerosis patients in central European countries: current needs and potential solutions.

Therapeutic advances in neurological disorders

Berger T, Adamczyk-Sowa M, Csépány T, Fazekas F, Hojs Fabjan T, Horáková D, Illes Z, Klimová E, Leutmezer F, Rejdak K, Rozsa C, Šega Jazbec S, Szilasiová J, Turčáni P, Vachová M, Vécsei L, Havrdová E.
PMID: 29511382
Ther Adv Neurol Disord. 2018 Feb 22;11:1756286418759189. doi: 10.1177/1756286418759189. eCollection 2018.

Multiple sclerosis (MS) experts in Europe are facing rapidly rising demands of excellence due to the increasing complexity of MS therapy and management. A central European expert board of MS experts met to identify needs and obstacles with respect...

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Berger T, Adamczyk-Sowa M, Csépány T, et al. Management of multiple sclerosis patients in central European countries: current needs and potential solutions. Ther Adv Neurol Disord. 2018;11:1756286418759189doi: 10.1177/1756286418759189.
Berger, T., Adamczyk-Sowa, M., Csépány, T., Fazekas, F., Hojs Fabjan, T., Horáková, D., Illes, Z., Klimová, E., Leutmezer, F., Rejdak, K., Rozsa, C., Šega Jazbec, S., Szilasiová, J., Turčáni, P., Vachová, M., Vécsei, L., & Havrdová, E. (2018). Management of multiple sclerosis patients in central European countries: current needs and potential solutions. Therapeutic advances in neurological disorders, 111756286418759189. https://doi.org/10.1177/1756286418759189
Berger, Thomas, et al. "Management of multiple sclerosis patients in central European countries: current needs and potential solutions." Therapeutic advances in neurological disorders vol. 11 (2018): 1756286418759189. doi: https://doi.org/10.1177/1756286418759189
Berger T, Adamczyk-Sowa M, Csépány T, Fazekas F, Hojs Fabjan T, Horáková D, Illes Z, Klimová E, Leutmezer F, Rejdak K, Rozsa C, Šega Jazbec S, Szilasiová J, Turčáni P, Vachová M, Vécsei L, Havrdová E. Management of multiple sclerosis patients in central European countries: current needs and potential solutions. Ther Adv Neurol Disord. 2018 Feb 22;11:1756286418759189. doi: 10.1177/1756286418759189. eCollection 2018. PMID: 29511382; PMCID: PMC5826096.
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Effectiveness of an Oral Health Education Program for Obstetrician/Gynecologist Residents at Tufts Medical Center.

Journal of International Society of Preventive & Community Dentistry

Shah D, Parsi L, Bagher S, Finkelman M, Loo C.
PMID: 29184837
J Int Soc Prev Community Dent. 2017 Oct;7:S107-S112. doi: 10.4103/jispcd.JISPCD_195_17. Epub 2017 Oct 30.

AIM AND OBJECTIVES: To assess Tufts Medical Center obstetrician/gynecologist (OB/GYN) residents' knowledge, beliefs, and previous training in oral health and to assess the effect of an oral health educational seminar on their knowledge and beliefs.MATERIALS AND METHODS: A preseminar...

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Shah D, Parsi L, Bagher S, et al. Effectiveness of an Oral Health Education Program for Obstetrician/Gynecologist Residents at Tufts Medical Center. J Int Soc Prev Community Dent. 2017;7:S107-S112doi: 10.4103/jispcd.JISPCD_195_17.
Shah, D., Parsi, L., Bagher, S., Finkelman, M., & Loo, C. (2017). Effectiveness of an Oral Health Education Program for Obstetrician/Gynecologist Residents at Tufts Medical Center. Journal of International Society of Preventive & Community Dentistry, 7S107-S112. https://doi.org/10.4103/jispcd.JISPCD_195_17
Shah, Devina, et al. "Effectiveness of an Oral Health Education Program for Obstetrician/Gynecologist Residents at Tufts Medical Center." Journal of International Society of Preventive & Community Dentistry vol. 7 (2017): S107-S112. doi: https://doi.org/10.4103/jispcd.JISPCD_195_17
Shah D, Parsi L, Bagher S, Finkelman M, Loo C. Effectiveness of an Oral Health Education Program for Obstetrician/Gynecologist Residents at Tufts Medical Center. J Int Soc Prev Community Dent. 2017 Oct;7:S107-S112. doi: 10.4103/jispcd.JISPCD_195_17. Epub 2017 Oct 30. PMID: 29184837; PMCID: PMC5682702.
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MeaB-dependent nutrition sensing regulates autolysis in carbon starved Aspergillus nidulans cultures.

Indian journal of microbiology

Szilágyi M, Pócsi I, Forgács K, Emri T.
PMID: 23100816
Indian J Microbiol. 2010 Mar;50(1):104-8. doi: 10.1007/s12088-010-0023-z. Epub 2010 Mar 16.

Carbon starvation induced autolysis is an active process of self-digestion and is under complex regulation in Aspergillus nidulans. In this study we investigated how autolysis depends on the composition of the culture medium, especially on the presence of yeast...

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Szilágyi M, Pócsi I, Forgács K, et al. MeaB-dependent nutrition sensing regulates autolysis in carbon starved Aspergillus nidulans cultures. Indian J Microbiol. 2010;50(1):104-8doi: 10.1007/s12088-010-0023-z.
Szilágyi, M., Pócsi, I., Forgács, K., & Emri, T. (2010). MeaB-dependent nutrition sensing regulates autolysis in carbon starved Aspergillus nidulans cultures. Indian journal of microbiology, 50(1), 104-8. https://doi.org/10.1007/s12088-010-0023-z
Szilágyi, Melinda, et al. "MeaB-dependent nutrition sensing regulates autolysis in carbon starved Aspergillus nidulans cultures." Indian journal of microbiology vol. 50,1 (2010): 104-8. doi: https://doi.org/10.1007/s12088-010-0023-z
Szilágyi M, Pócsi I, Forgács K, Emri T. MeaB-dependent nutrition sensing regulates autolysis in carbon starved Aspergillus nidulans cultures. Indian J Microbiol. 2010 Mar;50(1):104-8. doi: 10.1007/s12088-010-0023-z. Epub 2010 Mar 16. PMID: 23100816; PMCID: PMC3450277.
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PPIs Are Not Responsible for Elevating Cardiovascular Risk in Patients on Clopidogrel-A Systematic Review and Meta-Analysis.

Frontiers in physiology

Demcsák A, Lantos T, Bálint ER, Hartmann P, Vincze Á, Bajor J, Czopf L, Alizadeh H, Gyöngyi Z, Márta K, Mikó A, Szakács Z, Pécsi D, Hegyi P, Szabó IL.
PMID: 30510515
Front Physiol. 2018 Nov 19;9:1550. doi: 10.3389/fphys.2018.01550. eCollection 2018.

No abstract available.

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Demcsák A, Lantos T, Bálint ER, et al. PPIs Are Not Responsible for Elevating Cardiovascular Risk in Patients on Clopidogrel-A Systematic Review and Meta-Analysis. Front Physiol. 2018;9:1550doi: 10.3389/fphys.2018.01550.
Demcsák, A., Lantos, T., Bálint, E. R., Hartmann, P., Vincze, �. �., Bajor, J., Czopf, L., Alizadeh, H., Gyöngyi, Z., Márta, K., Mikó, A., Szakács, Z., Pécsi, D., Hegyi, P., & Szabó, I. L. (2018). PPIs Are Not Responsible for Elevating Cardiovascular Risk in Patients on Clopidogrel-A Systematic Review and Meta-Analysis. Frontiers in physiology, 91550. https://doi.org/10.3389/fphys.2018.01550
Demcsák, Alexandra, et al. "PPIs Are Not Responsible for Elevating Cardiovascular Risk in Patients on Clopidogrel-A Systematic Review and Meta-Analysis." Frontiers in physiology vol. 9 (2018): 1550. doi: https://doi.org/10.3389/fphys.2018.01550
Demcsák A, Lantos T, Bálint ER, Hartmann P, Vincze Á, Bajor J, Czopf L, Alizadeh H, Gyöngyi Z, Márta K, Mikó A, Szakács Z, Pécsi D, Hegyi P, Szabó IL. PPIs Are Not Responsible for Elevating Cardiovascular Risk in Patients on Clopidogrel-A Systematic Review and Meta-Analysis. Front Physiol. 2018 Nov 19;9:1550. doi: 10.3389/fphys.2018.01550. eCollection 2018. PMID: 30510515; PMCID: PMC6252380.
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Transcriptome-Based Modeling Reveals that Oxidative Stress Induces Modulation of the AtfA-Dependent Signaling Networks in .

International journal of genomics

Orosz E, Antal K, Gazdag Z, Szabó Z, Han KH, Yu JH, Pócsi I, Emri T.
PMID: 28770220
Int J Genomics. 2017;2017:6923849. doi: 10.1155/2017/6923849. Epub 2017 Jul 09.

To better understand the molecular functions of the master stress-response regulator AtfA in

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Orosz E, Antal K, Gazdag Z, et al. Transcriptome-Based Modeling Reveals that Oxidative Stress Induces Modulation of the AtfA-Dependent Signaling Networks in . Int J Genomics. 2017;2017:6923849doi: 10.1155/2017/6923849.
Orosz, E., Antal, K., Gazdag, Z., Szabó, Z., Han, K. H., Yu, J. H., Pócsi, I., & Emri, T. (2017). Transcriptome-Based Modeling Reveals that Oxidative Stress Induces Modulation of the AtfA-Dependent Signaling Networks in . International journal of genomics, 20176923849. https://doi.org/10.1155/2017/6923849
Orosz, Erzsébet, et al. "Transcriptome-Based Modeling Reveals that Oxidative Stress Induces Modulation of the AtfA-Dependent Signaling Networks in ." International journal of genomics vol. 2017 (2017): 6923849. doi: https://doi.org/10.1155/2017/6923849
Orosz E, Antal K, Gazdag Z, Szabó Z, Han KH, Yu JH, Pócsi I, Emri T. Transcriptome-Based Modeling Reveals that Oxidative Stress Induces Modulation of the AtfA-Dependent Signaling Networks in . Int J Genomics. 2017;2017:6923849. doi: 10.1155/2017/6923849. Epub 2017 Jul 09. PMID: 28770220; PMCID: PMC5523550.
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Are Migraine With and Without Aura Really Different Entities?.

Frontiers in neurology

Kincses ZT, Veréb D, Faragó P, Tóth E, Kocsis K, Kincses B, Király A, Bozsik B, Párdutz Á, Szok D, Tajti J, Vécsei L, Tuka B, Szabó N.
PMID: 31632329
Front Neurol. 2019 Oct 02;10:982. doi: 10.3389/fneur.2019.00982. eCollection 2019.

No abstract available.

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Kincses ZT, Veréb D, Faragó P, et al. Are Migraine With and Without Aura Really Different Entities?. Front Neurol. 2019;10:982doi: 10.3389/fneur.2019.00982.
Kincses, Z. T., Veréb, D., Faragó, P., Tóth, E., Kocsis, K., Kincses, B., Király, A., Bozsik, B., Párdutz, �. �., Szok, D., Tajti, J., Vécsei, L., Tuka, B., & Szabó, N. (2019). Are Migraine With and Without Aura Really Different Entities?. Frontiers in neurology, 10982. https://doi.org/10.3389/fneur.2019.00982
Kincses, Zsigmond Tamás, et al. "Are Migraine With and Without Aura Really Different Entities?." Frontiers in neurology vol. 10 (2019): 982. doi: https://doi.org/10.3389/fneur.2019.00982
Kincses ZT, Veréb D, Faragó P, Tóth E, Kocsis K, Kincses B, Király A, Bozsik B, Párdutz Á, Szok D, Tajti J, Vécsei L, Tuka B, Szabó N. Are Migraine With and Without Aura Really Different Entities?. Front Neurol. 2019 Oct 02;10:982. doi: 10.3389/fneur.2019.00982. eCollection 2019. PMID: 31632329; PMCID: PMC6783501.
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Showing 1 to 12 of 194 entries