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Intracellular cholesterol pools regulate oncogenic signaling and epigenetic circuitries in Early T-cell Precursor Acute Lymphoblastic Leukemia.

Cancer discovery

Rashkovan M, Albero R, Gianni F, Perez-Duran P, Miller HI, Mackey AL, Paietta EM, Tallman MS, Rowe JM, Litzow MR, Wiernik PH, Luger S, Sulis ML, Soni RK, Ferrando AA.
PMID: 34711640
Cancer Discov. 2021 Oct 28; doi: 10.1158/2159-8290.CD-21-0551. Epub 2021 Oct 28.

Early T-cell acute lymphoblastic leukemia (ETP-ALL) is an aggressive hematologic malignancy associated with early relapse and poor prognosis that is genetically, immunophenotypically and transcriptionally distinct from more mature T-cell acute lymphoblastic (T-ALL) tumors. Here, we leveraged global metabolomic and...

Cite
Rashkovan M, Albero R, Gianni F, et al. Intracellular cholesterol pools regulate oncogenic signaling and epigenetic circuitries in Early T-cell Precursor Acute Lymphoblastic Leukemia. Cancer Discov. 2021;doi: 10.1158/2159-8290.CD-21-0551.
Rashkovan, M., Albero, R., Gianni, F., Perez-Duran, P., Miller, H. I., Mackey, A. L., Paietta, E. M., Tallman, M. S., Rowe, J. M., Litzow, M. R., Wiernik, P. H., Luger, S., Sulis, M. L., Soni, R. K., & Ferrando, A. A. (2021). Intracellular cholesterol pools regulate oncogenic signaling and epigenetic circuitries in Early T-cell Precursor Acute Lymphoblastic Leukemia. Cancer discovery, . https://doi.org/10.1158/2159-8290.CD-21-0551
Rashkovan, Marissa, et al. "Intracellular cholesterol pools regulate oncogenic signaling and epigenetic circuitries in Early T-cell Precursor Acute Lymphoblastic Leukemia." Cancer discovery vol. (2021). doi: https://doi.org/10.1158/2159-8290.CD-21-0551
Rashkovan M, Albero R, Gianni F, Perez-Duran P, Miller HI, Mackey AL, Paietta EM, Tallman MS, Rowe JM, Litzow MR, Wiernik PH, Luger S, Sulis ML, Soni RK, Ferrando AA. Intracellular cholesterol pools regulate oncogenic signaling and epigenetic circuitries in Early T-cell Precursor Acute Lymphoblastic Leukemia. Cancer Discov. 2021 Oct 28; doi: 10.1158/2159-8290.CD-21-0551. Epub 2021 Oct 28. PMID: 34711640.
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Anticancer potential of (-)-epicatechin in a triple-negative mammary gland model.

The Journal of pharmacy and pharmacology

Almaguer G, Ortiz-Vilchis P, Cordero P, Martinez-Vega R, Perez-Durán J, Meaney E, Villarreal F, Ceballos G, Nájera N.
PMID: 34473289
J Pharm Pharmacol. 2021 Dec 07;73(12):1675-1682. doi: 10.1093/jpp/rgab133.

OBJECTIVES: The main aim of this work was to analyse the potential tumour growth inhibition effects of (-)-epicatechin (EC). Triple-negative breast cancer (TNBC) is an invasive form of cancer characterized by the absence of progesterone receptor, estrogen receptor and...

Cite
Almaguer G, Ortiz-Vilchis P, Cordero P, et al. Anticancer potential of (-)-epicatechin in a triple-negative mammary gland model. J Pharm Pharmacol. 2021;73(12):1675-1682doi: 10.1093/jpp/rgab133.
Almaguer, G., Ortiz-Vilchis, P., Cordero, P., Martinez-Vega, R., Perez-Durán, J., Meaney, E., Villarreal, F., Ceballos, G., & Nájera, N. (2021). Anticancer potential of (-)-epicatechin in a triple-negative mammary gland model. The Journal of pharmacy and pharmacology, 73(12), 1675-1682. https://doi.org/10.1093/jpp/rgab133
Almaguer, Georgina, et al. "Anticancer potential of (-)-epicatechin in a triple-negative mammary gland model." The Journal of pharmacy and pharmacology vol. 73,12 (2021): 1675-1682. doi: https://doi.org/10.1093/jpp/rgab133
Almaguer G, Ortiz-Vilchis P, Cordero P, Martinez-Vega R, Perez-Durán J, Meaney E, Villarreal F, Ceballos G, Nájera N. Anticancer potential of (-)-epicatechin in a triple-negative mammary gland model. J Pharm Pharmacol. 2021 Dec 07;73(12):1675-1682. doi: 10.1093/jpp/rgab133. PMID: 34473289; PMCID: PMC8651247.
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PXR is a target of (-)-epicatechin in skeletal muscle.

Heliyon

Ortiz-Flores M, Portilla-Martínez A, Cabrera-Pérez F, Nájera N, Meaney E, Villarreal F, Pérez-Durán J, Ceballos G.
PMID: 33163657
Heliyon. 2020 Oct 30;6(10):e05357. doi: 10.1016/j.heliyon.2020.e05357. eCollection 2020 Oct.

(-)-Epicatechin (EC) is a flavanol that has shown numerous biological effects such as: decrease risk of cardiovascular dysfunction, metabolism regulation, skeletal muscle (SkM) performance improvement and SkM cells differentiation induction, among others. The described EC acceptor/receptor molecules do not...

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Ortiz-Flores M, Portilla-Martínez A, Cabrera-Pérez F, et al. PXR is a target of (-)-epicatechin in skeletal muscle. Heliyon. 2020;6(10):e05357doi: 10.1016/j.heliyon.2020.e05357.
Ortiz-Flores, M., Portilla-Martínez, A., Cabrera-Pérez, F., Nájera, N., Meaney, E., Villarreal, F., Pérez-Durán, J., & Ceballos, G. (2020). PXR is a target of (-)-epicatechin in skeletal muscle. Heliyon, 6(10), e05357. https://doi.org/10.1016/j.heliyon.2020.e05357
Ortiz-Flores, Miguel, et al. "PXR is a target of (-)-epicatechin in skeletal muscle." Heliyon vol. 6,10 (2020): e05357. doi: https://doi.org/10.1016/j.heliyon.2020.e05357
Ortiz-Flores M, Portilla-Martínez A, Cabrera-Pérez F, Nájera N, Meaney E, Villarreal F, Pérez-Durán J, Ceballos G. PXR is a target of (-)-epicatechin in skeletal muscle. Heliyon. 2020 Oct 30;6(10):e05357. doi: 10.1016/j.heliyon.2020.e05357. eCollection 2020 Oct. PMID: 33163657; PMCID: PMC7610271.
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Mutational and functional genetics mapping of chemotherapy resistance mechanisms in relapsed acute lymphoblastic leukemia.

Nature cancer

Oshima K, Zhao J, Pérez-Durán P, Brown JA, Patiño-Galindo JA, Chu T, Quinn A, Gunning T, Belver L, Ambesi-Impiombato A, Tosello V, Wang Z, Sulis ML, Kato M, Koh K, Paganin M, Basso G, Balbin M, Nicolas C, Gastier-Foster JM, Devidas M, Loh ML, Paietta E, Tallman MS, Rowe JM, Litzow M, Minden MD, Meijerink J, Rabadan R, Ferrando A.
PMID: 33796864
Nat Cancer. 2020 Nov;1(11):1113-1127. doi: 10.1038/s43018-020-00124-1. Epub 2020 Oct 19.

Multi-agent combination chemotherapy can be curative in acute lymphoblastic leukemia (ALL). Still, patients with primary refractory disease or with relapsed leukemia have a very poor prognosis. Here we integrate an in-depth dissection of the mutational landscape across diagnostic and...

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Oshima K, Zhao J, Pérez-Durán P, et al. Mutational and functional genetics mapping of chemotherapy resistance mechanisms in relapsed acute lymphoblastic leukemia. Nat Cancer. 2020;1(11):1113-1127doi: 10.1038/s43018-020-00124-1.
Oshima, K., Zhao, J., Pérez-Durán, P., Brown, J. A., Patiño-Galindo, J. A., Chu, T., Quinn, A., Gunning, T., Belver, L., Ambesi-Impiombato, A., Tosello, V., Wang, Z., Sulis, M. L., Kato, M., Koh, K., Paganin, M., Basso, G., Balbin, M., Nicolas, C., Gastier-Foster, J. M., Devidas, M., Loh, M. L., Paietta, E., Tallman, M. S., Rowe, J. M., Litzow, M., Minden, M. D., Meijerink, J., Rabadan, R., & Ferrando, A. (2020). Mutational and functional genetics mapping of chemotherapy resistance mechanisms in relapsed acute lymphoblastic leukemia. Nature cancer, 1(11), 1113-1127. https://doi.org/10.1038/s43018-020-00124-1
Oshima, Koichi, et al. "Mutational and functional genetics mapping of chemotherapy resistance mechanisms in relapsed acute lymphoblastic leukemia." Nature cancer vol. 1,11 (2020): 1113-1127. doi: https://doi.org/10.1038/s43018-020-00124-1
Oshima K, Zhao J, Pérez-Durán P, Brown JA, Patiño-Galindo JA, Chu T, Quinn A, Gunning T, Belver L, Ambesi-Impiombato A, Tosello V, Wang Z, Sulis ML, Kato M, Koh K, Paganin M, Basso G, Balbin M, Nicolas C, Gastier-Foster JM, Devidas M, Loh ML, Paietta E, Tallman MS, Rowe JM, Litzow M, Minden MD, Meijerink J, Rabadan R, Ferrando A. Mutational and functional genetics mapping of chemotherapy resistance mechanisms in relapsed acute lymphoblastic leukemia. Nat Cancer. 2020 Nov;1(11):1113-1127. doi: 10.1038/s43018-020-00124-1. Epub 2020 Oct 19. PMID: 33796864; PMCID: PMC8011577.
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Aneusomy detection with Karyolite-Bac on Beads® is a cost-efficient and high throughput strategy in the molecular analyses of the early pregnancy conception losses.

Molecular cytogenetics

Pérez-Durán J, Nájera Z, Trujillo-Cabrera Y, Martín-Saro M, García-Latorre E, Escarcega-Preciado J, Nájera N, Martínez-Galaviz T, Queipo G.
PMID: 26269714
Mol Cytogenet. 2015 Aug 12;8:63. doi: 10.1186/s13039-015-0168-x. eCollection 2015.

BACKGROUND: Approximately 10-15 % of all clinically recognized pregnancies end in miscarriage, the majority of them occur during the first trimester, underlying the cause of the loss. Genetic analysis of fetal tissues has the potential to provide valuable information...

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Pérez-Durán J, Nájera Z, Trujillo-Cabrera Y, et al. Aneusomy detection with Karyolite-Bac on Beads® is a cost-efficient and high throughput strategy in the molecular analyses of the early pregnancy conception losses. Mol Cytogenet. 2015;8:63doi: 10.1186/s13039-015-0168-x.
Pérez-Durán, J., Nájera, Z., Trujillo-Cabrera, Y., Martín-Saro, M., García-Latorre, E., Escarcega-Preciado, J., Nájera, N., Martínez-Galaviz, T., & Queipo, G. (2015). Aneusomy detection with Karyolite-Bac on Beads® is a cost-efficient and high throughput strategy in the molecular analyses of the early pregnancy conception losses. Molecular cytogenetics, 863. https://doi.org/10.1186/s13039-015-0168-x
Pérez-Durán, Javier, et al. "Aneusomy detection with Karyolite-Bac on Beads® is a cost-efficient and high throughput strategy in the molecular analyses of the early pregnancy conception losses." Molecular cytogenetics vol. 8 (2015): 63. doi: https://doi.org/10.1186/s13039-015-0168-x
Pérez-Durán J, Nájera Z, Trujillo-Cabrera Y, Martín-Saro M, García-Latorre E, Escarcega-Preciado J, Nájera N, Martínez-Galaviz T, Queipo G. Aneusomy detection with Karyolite-Bac on Beads® is a cost-efficient and high throughput strategy in the molecular analyses of the early pregnancy conception losses. Mol Cytogenet. 2015 Aug 12;8:63. doi: 10.1186/s13039-015-0168-x. eCollection 2015. PMID: 26269714; PMCID: PMC4534128.
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Intracellular cholesterol pools regulate oncogenic signaling and epigenetic circuitries in Early T-cell Precursor Acute Lymphoblastic Leukemia.

Cancer discovery

Rashkovan M, Albero R, Gianni F, Perez-Duran P, Miller HI, Mackey AL, Paietta EM, Tallman MS, Rowe JM, Litzow MR, Wiernik PH, Luger S, Sulis ML, Soni RK, Ferrando AA.
PMID: 34711640
Cancer Discov. 2021 Oct 28; doi: 10.1158/2159-8290.CD-21-0551. Epub 2021 Oct 28.

Early T-cell acute lymphoblastic leukemia (ETP-ALL) is an aggressive hematologic malignancy associated with early relapse and poor prognosis that is genetically, immunophenotypically and transcriptionally distinct from more mature T-cell acute lymphoblastic (T-ALL) tumors. Here, we leveraged global metabolomic and...

Cite
Rashkovan M, Albero R, Gianni F, et al. Intracellular cholesterol pools regulate oncogenic signaling and epigenetic circuitries in Early T-cell Precursor Acute Lymphoblastic Leukemia. Cancer Discov. 2021;doi: 10.1158/2159-8290.CD-21-0551.
Rashkovan, M., Albero, R., Gianni, F., Perez-Duran, P., Miller, H. I., Mackey, A. L., Paietta, E. M., Tallman, M. S., Rowe, J. M., Litzow, M. R., Wiernik, P. H., Luger, S., Sulis, M. L., Soni, R. K., & Ferrando, A. A. (2021). Intracellular cholesterol pools regulate oncogenic signaling and epigenetic circuitries in Early T-cell Precursor Acute Lymphoblastic Leukemia. Cancer discovery, . https://doi.org/10.1158/2159-8290.CD-21-0551
Rashkovan, Marissa, et al. "Intracellular cholesterol pools regulate oncogenic signaling and epigenetic circuitries in Early T-cell Precursor Acute Lymphoblastic Leukemia." Cancer discovery vol. (2021). doi: https://doi.org/10.1158/2159-8290.CD-21-0551
Rashkovan M, Albero R, Gianni F, Perez-Duran P, Miller HI, Mackey AL, Paietta EM, Tallman MS, Rowe JM, Litzow MR, Wiernik PH, Luger S, Sulis ML, Soni RK, Ferrando AA. Intracellular cholesterol pools regulate oncogenic signaling and epigenetic circuitries in Early T-cell Precursor Acute Lymphoblastic Leukemia. Cancer Discov. 2021 Oct 28; doi: 10.1158/2159-8290.CD-21-0551. Epub 2021 Oct 28. PMID: 34711640.
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Anticancer potential of (-)-epicatechin in a triple-negative mammary gland model.

The Journal of pharmacy and pharmacology

Almaguer G, Ortiz-Vilchis P, Cordero P, Martinez-Vega R, Perez-Durán J, Meaney E, Villarreal F, Ceballos G, Nájera N.
PMID: 34473289
J Pharm Pharmacol. 2021 Dec 07;73(12):1675-1682. doi: 10.1093/jpp/rgab133.

OBJECTIVES: The main aim of this work was to analyse the potential tumour growth inhibition effects of (-)-epicatechin (EC). Triple-negative breast cancer (TNBC) is an invasive form of cancer characterized by the absence of progesterone receptor, estrogen receptor and...

Cite
Almaguer G, Ortiz-Vilchis P, Cordero P, et al. Anticancer potential of (-)-epicatechin in a triple-negative mammary gland model. J Pharm Pharmacol. 2021;73(12):1675-1682doi: 10.1093/jpp/rgab133.
Almaguer, G., Ortiz-Vilchis, P., Cordero, P., Martinez-Vega, R., Perez-Durán, J., Meaney, E., Villarreal, F., Ceballos, G., & Nájera, N. (2021). Anticancer potential of (-)-epicatechin in a triple-negative mammary gland model. The Journal of pharmacy and pharmacology, 73(12), 1675-1682. https://doi.org/10.1093/jpp/rgab133
Almaguer, Georgina, et al. "Anticancer potential of (-)-epicatechin in a triple-negative mammary gland model." The Journal of pharmacy and pharmacology vol. 73,12 (2021): 1675-1682. doi: https://doi.org/10.1093/jpp/rgab133
Almaguer G, Ortiz-Vilchis P, Cordero P, Martinez-Vega R, Perez-Durán J, Meaney E, Villarreal F, Ceballos G, Nájera N. Anticancer potential of (-)-epicatechin in a triple-negative mammary gland model. J Pharm Pharmacol. 2021 Dec 07;73(12):1675-1682. doi: 10.1093/jpp/rgab133. PMID: 34473289; PMCID: PMC8651247.
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Showing 1 to 7 of 7 entries