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An intrabody specific for the nucleophosmin carboxy-terminal mutant and fused to a nuclear localization sequence binds its antigen but fails to relocate it in the nucleus.

Biotechnology reports (Amsterdam, Netherlands)

Martinelli C, Colombo E, Piccini D, Sironi C, Pelicci PG, de Marco A.
PMID: 28626645
Biotechnol Rep (Amst). 2014 May 27;3:27-33. doi: 10.1016/j.btre.2014.05.008. eCollection 2014 Sep.

The cytoplasmic accumulation of NPM1 (NPMc+) is found in acute myeloid leukemia (AML) with NPM1 mutation. NPM1 must shuttle between nucleus and cytoplasm to assure physiological protein synthesis and, therefore, the elimination of NPMc+ is not a suitable therapeutic...

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Martinelli C, Colombo E, Piccini D, et al. An intrabody specific for the nucleophosmin carboxy-terminal mutant and fused to a nuclear localization sequence binds its antigen but fails to relocate it in the nucleus. Biotechnol Rep (Amst). 2014;3:27-33doi: 10.1016/j.btre.2014.05.008.
Martinelli, C., Colombo, E., Piccini, D., Sironi, C., Pelicci, P. G., & de Marco, A. (2014). An intrabody specific for the nucleophosmin carboxy-terminal mutant and fused to a nuclear localization sequence binds its antigen but fails to relocate it in the nucleus. Biotechnology reports (Amsterdam, Netherlands), 327-33. https://doi.org/10.1016/j.btre.2014.05.008
Martinelli, Chiara, et al. "An intrabody specific for the nucleophosmin carboxy-terminal mutant and fused to a nuclear localization sequence binds its antigen but fails to relocate it in the nucleus." Biotechnology reports (Amsterdam, Netherlands) vol. 3 (2014): 27-33. doi: https://doi.org/10.1016/j.btre.2014.05.008
Martinelli C, Colombo E, Piccini D, Sironi C, Pelicci PG, de Marco A. An intrabody specific for the nucleophosmin carboxy-terminal mutant and fused to a nuclear localization sequence binds its antigen but fails to relocate it in the nucleus. Biotechnol Rep (Amst). 2014 May 27;3:27-33. doi: 10.1016/j.btre.2014.05.008. eCollection 2014 Sep. PMID: 28626645; PMCID: PMC5466097.
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Acute promyelocytic leukemias share cooperative mutations with other myeloid-leukemia subgroups.

Blood cancer journal

Riva L, Ronchini C, Bodini M, Lo-Coco F, Lavorgna S, Ottone T, Martinelli G, Iacobucci I, Tarella C, Cignetti A, Volorio S, Bernard L, Russo A, Melloni GE, Luzi L, Alcalay M, Dellino GI, Pelicci PG.
PMID: 24658373
Blood Cancer J. 2014 Mar 21;4:e195. doi: 10.1038/bcj.2014.19.

No abstract available.

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Riva L, Ronchini C, Bodini M, et al. Acute promyelocytic leukemias share cooperative mutations with other myeloid-leukemia subgroups. Blood Cancer J. 2014;4:e195doi: 10.1038/bcj.2014.19.
Riva, L., Ronchini, C., Bodini, M., Lo-Coco, F., Lavorgna, S., Ottone, T., Martinelli, G., Iacobucci, I., Tarella, C., Cignetti, A., Volorio, S., Bernard, L., Russo, A., Melloni, G. E., Luzi, L., Alcalay, M., Dellino, G. I., & Pelicci, P. G. (2014). Acute promyelocytic leukemias share cooperative mutations with other myeloid-leukemia subgroups. Blood cancer journal, 4e195. https://doi.org/10.1038/bcj.2014.19
Riva, L, et al. "Acute promyelocytic leukemias share cooperative mutations with other myeloid-leukemia subgroups." Blood cancer journal vol. 4 (2014): e195. doi: https://doi.org/10.1038/bcj.2014.19
Riva L, Ronchini C, Bodini M, Lo-Coco F, Lavorgna S, Ottone T, Martinelli G, Iacobucci I, Tarella C, Cignetti A, Volorio S, Bernard L, Russo A, Melloni GE, Luzi L, Alcalay M, Dellino GI, Pelicci PG. Acute promyelocytic leukemias share cooperative mutations with other myeloid-leukemia subgroups. Blood Cancer J. 2014 Mar 21;4:e195. doi: 10.1038/bcj.2014.19. PMID: 24658373; PMCID: PMC3972704.
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Report from the Melanoma Independent Board First Melanoma MIB Conference, 21-22 October 2013.

Ecancermedicalscience

Testori A, Ascierto P, Chiarion Sileni V, De Lorenzo F, Pelicci P, Rossi C.
PMID: 25075214
Ecancermedicalscience. 2014 Jun 30;8:440. doi: 10.3332/ecancer.2014.440. eCollection 2014.

The Melanoma Independent Board (MIB) held its first conference from 21 to 22 October, 2013, in Rome, Italy. Like the MIB itself, the conference brought together specialists from all aspects of cancer care: doctors, patient associations, journalists, and representatives...

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Testori A, Ascierto P, Chiarion Sileni V, et al. Report from the Melanoma Independent Board First Melanoma MIB Conference, 21-22 October 2013. Ecancermedicalscience. 2014;8:440doi: 10.3332/ecancer.2014.440.
Testori, A., Ascierto, P., Chiarion Sileni, V., De Lorenzo, F., Pelicci, P., & Rossi, C. (2014). Report from the Melanoma Independent Board First Melanoma MIB Conference, 21-22 October 2013. Ecancermedicalscience, 8440. https://doi.org/10.3332/ecancer.2014.440
Testori, A, et al. "Report from the Melanoma Independent Board First Melanoma MIB Conference, 21-22 October 2013." Ecancermedicalscience vol. 8 (2014): 440. doi: https://doi.org/10.3332/ecancer.2014.440
Testori A, Ascierto P, Chiarion Sileni V, De Lorenzo F, Pelicci P, Rossi C. Report from the Melanoma Independent Board First Melanoma MIB Conference, 21-22 October 2013. Ecancermedicalscience. 2014 Jun 30;8:440. doi: 10.3332/ecancer.2014.440. eCollection 2014. PMID: 25075214; PMCID: PMC4096026.
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Is PML a Tumor Suppressor?.

Frontiers in oncology

Mazza M, Pelicci PG.
PMID: 23847764
Front Oncol. 2013 Jul 09;3:174. doi: 10.3389/fonc.2013.00174. eCollection 2013.

The role of the promyelocytic leukemia (PML) protein has been widely tested in many different contexts, as attested by the hundreds of papers present in the literature. In most of these studies, PML is regarded as a tumor suppressor,...

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Mazza M, Pelicci PG. Is PML a Tumor Suppressor?. Front Oncol. 2013;3:174doi: 10.3389/fonc.2013.00174.
Mazza, M., & Pelicci, P. G. (2013). Is PML a Tumor Suppressor?. Frontiers in oncology, 3174. https://doi.org/10.3389/fonc.2013.00174
Mazza, Massimiliano, and Pelicci, Pier Giuseppe. "Is PML a Tumor Suppressor?." Frontiers in oncology vol. 3 (2013): 174. doi: https://doi.org/10.3389/fonc.2013.00174
Mazza M, Pelicci PG. Is PML a Tumor Suppressor?. Front Oncol. 2013 Jul 09;3:174. doi: 10.3389/fonc.2013.00174. eCollection 2013. PMID: 23847764; PMCID: PMC3705425.
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Insights into the beneficial effect of caloric/ dietary restriction for a healthy and prolonged life.

Frontiers in physiology

Pallavi R, Giorgio M, Pelicci PG.
PMID: 22934068
Front Physiol. 2012 Aug 09;3:318. doi: 10.3389/fphys.2012.00318. eCollection 2012.

Over the last several years, new evidence has kept pouring in about the remarkable effect of caloric restriction (CR) on the conspicuous bedfellows- aging and cancer. Through the use of various animal models, it is now well established that...

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Pallavi R, Giorgio M, Pelicci PG. Insights into the beneficial effect of caloric/ dietary restriction for a healthy and prolonged life. Front Physiol. 2012;3:318doi: 10.3389/fphys.2012.00318.
Pallavi, R., Giorgio, M., & Pelicci, P. G. (2012). Insights into the beneficial effect of caloric/ dietary restriction for a healthy and prolonged life. Frontiers in physiology, 3318. https://doi.org/10.3389/fphys.2012.00318
Pallavi, Rani, et al. "Insights into the beneficial effect of caloric/ dietary restriction for a healthy and prolonged life." Frontiers in physiology vol. 3 (2012): 318. doi: https://doi.org/10.3389/fphys.2012.00318
Pallavi R, Giorgio M, Pelicci PG. Insights into the beneficial effect of caloric/ dietary restriction for a healthy and prolonged life. Front Physiol. 2012 Aug 09;3:318. doi: 10.3389/fphys.2012.00318. eCollection 2012. PMID: 22934068; PMCID: PMC3429088.
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Activated protein C reverses epigenetically sustained p66.

Communications biology

Shahzad K, Gadi I, Nazir S, Al-Dabet MM, Kohli S, Bock F, Breitenstein L, Ranjan S, Fuchs T, Halloul Z, Nawroth PP, Pelicci PG, Braun-Dullaeus RC, Camerer E, Esmon CT, Isermann B.
PMID: 30271984
Commun Biol. 2018 Aug 06;1:104. doi: 10.1038/s42003-018-0108-5. eCollection 2018.

Impaired activated protein C (aPC) generation is associated with atherosclerosis and diabetes mellitus. Diabetes-associated atherosclerosis is characterized by the hyperglycaemic memory, e.g., failure of disease improvement despite attenuation of hyperglycaemia. Therapies reversing the hyperglycaemic memory are lacking. Here we...

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Shahzad K, Gadi I, Nazir S, et al. Activated protein C reverses epigenetically sustained p66. Commun Biol. 2018;1:104doi: 10.1038/s42003-018-0108-5.
Shahzad, K., Gadi, I., Nazir, S., Al-Dabet, M. M., Kohli, S., Bock, F., Breitenstein, L., Ranjan, S., Fuchs, T., Halloul, Z., Nawroth, P. P., Pelicci, P. G., Braun-Dullaeus, R. C., Camerer, E., Esmon, C. T., & Isermann, B. (2018). Activated protein C reverses epigenetically sustained p66. Communications biology, 1104. https://doi.org/10.1038/s42003-018-0108-5
Shahzad, Khurrum, et al. "Activated protein C reverses epigenetically sustained p66." Communications biology vol. 1 (2018): 104. doi: https://doi.org/10.1038/s42003-018-0108-5
Shahzad K, Gadi I, Nazir S, Al-Dabet MM, Kohli S, Bock F, Breitenstein L, Ranjan S, Fuchs T, Halloul Z, Nawroth PP, Pelicci PG, Braun-Dullaeus RC, Camerer E, Esmon CT, Isermann B. Activated protein C reverses epigenetically sustained p66. Commun Biol. 2018 Aug 06;1:104. doi: 10.1038/s42003-018-0108-5. eCollection 2018. PMID: 30271984; PMCID: PMC6123684.
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Semantic and Geographical Analysis of COVID-19 Trials Reveals a Fragmented Clinical Research Landscape Likely to Impair Informativeness.

Frontiers in medicine

Tini G, Duso BA, Bellerba F, Corso F, Gandini S, Minucci S, Pelicci PG, Mazzarella L.
PMID: 32695791
Front Med (Lausanne). 2020 Jun 29;7:367. doi: 10.3389/fmed.2020.00367. eCollection 2020.

No abstract available.

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Tini G, Duso BA, Bellerba F, et al. Semantic and Geographical Analysis of COVID-19 Trials Reveals a Fragmented Clinical Research Landscape Likely to Impair Informativeness. Front Med (Lausanne). 2020;7:367doi: 10.3389/fmed.2020.00367.
Tini, G., Duso, B. A., Bellerba, F., Corso, F., Gandini, S., Minucci, S., Pelicci, P. G., & Mazzarella, L. (2020). Semantic and Geographical Analysis of COVID-19 Trials Reveals a Fragmented Clinical Research Landscape Likely to Impair Informativeness. Frontiers in medicine, 7367. https://doi.org/10.3389/fmed.2020.00367
Tini, Giulia, et al. "Semantic and Geographical Analysis of COVID-19 Trials Reveals a Fragmented Clinical Research Landscape Likely to Impair Informativeness." Frontiers in medicine vol. 7 (2020): 367. doi: https://doi.org/10.3389/fmed.2020.00367
Tini G, Duso BA, Bellerba F, Corso F, Gandini S, Minucci S, Pelicci PG, Mazzarella L. Semantic and Geographical Analysis of COVID-19 Trials Reveals a Fragmented Clinical Research Landscape Likely to Impair Informativeness. Front Med (Lausanne). 2020 Jun 29;7:367. doi: 10.3389/fmed.2020.00367. eCollection 2020. PMID: 32695791; PMCID: PMC7336807.
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Persistence of Anti-SARS-CoV-2 Antibodies in Non-Hospitalized COVID-19 Convalescent Health Care Workers.

Journal of clinical medicine

Bruni M, Cecatiello V, Diaz-Basabe A, Lattanzi G, Mileti E, Monzani S, Pirovano L, Rizzelli F, Visintin C, Bonizzi G, Giani M, Lavitrano M, Faravelli S, Forneris F, Caprioli F, Pelicci PG, Natoli G, Pasqualato S, Mapelli M, Facciotti F.
PMID: 33019628
J Clin Med. 2020 Oct 01;9(10). doi: 10.3390/jcm9103188.

Although antibody response to SARS-CoV-2 can be detected early during the infection, several outstanding questions remain to be addressed regarding the magnitude and persistence of antibody titer against different viral proteins and their correlation with the strength of the...

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Bruni M, Cecatiello V, Diaz-Basabe A, et al. Persistence of Anti-SARS-CoV-2 Antibodies in Non-Hospitalized COVID-19 Convalescent Health Care Workers. J Clin Med. 2020;9(10)doi: 10.3390/jcm9103188.
Bruni, M., Cecatiello, V., Diaz-Basabe, A., Lattanzi, G., Mileti, E., Monzani, S., Pirovano, L., Rizzelli, F., Visintin, C., Bonizzi, G., Giani, M., Lavitrano, M., Faravelli, S., Forneris, F., Caprioli, F., Pelicci, P. G., Natoli, G., Pasqualato, S., Mapelli, M., & Facciotti, F. (2020). Persistence of Anti-SARS-CoV-2 Antibodies in Non-Hospitalized COVID-19 Convalescent Health Care Workers. Journal of clinical medicine, 9(10), . https://doi.org/10.3390/jcm9103188
Bruni, Margherita, et al. "Persistence of Anti-SARS-CoV-2 Antibodies in Non-Hospitalized COVID-19 Convalescent Health Care Workers." Journal of clinical medicine vol. 9,10 (2020). doi: https://doi.org/10.3390/jcm9103188
Bruni M, Cecatiello V, Diaz-Basabe A, Lattanzi G, Mileti E, Monzani S, Pirovano L, Rizzelli F, Visintin C, Bonizzi G, Giani M, Lavitrano M, Faravelli S, Forneris F, Caprioli F, Pelicci PG, Natoli G, Pasqualato S, Mapelli M, Facciotti F. Persistence of Anti-SARS-CoV-2 Antibodies in Non-Hospitalized COVID-19 Convalescent Health Care Workers. J Clin Med. 2020 Oct 01;9(10). doi: 10.3390/jcm9103188. PMID: 33019628; PMCID: PMC7600936.
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Biomedical omics: first insights of a new MSc degree of the University of Milan.

Tumori

Alcalay M, Jereczek-Fossa BA, Pepa M, Volpe S, Zaffaroni M, Fiore F, Marvaso G, Pravettoni G, Curigliano G, Santaguida S, Pelicci PG.
PMID: 34585604
Tumori. 2021 Sep 29;3008916211047268. doi: 10.1177/03008916211047268. Epub 2021 Sep 29.

The advent of technologies allowing the global analysis of biological phenomena, referred to as "omics" (genomics, epigenomics, proteomics, metabolomics, microbiomics, radiomics, and radiogenomics), has revolutionized the study of human diseases and traced the path for quantitative personalized medicine. The...

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Alcalay M, Jereczek-Fossa BA, Pepa M, et al. Biomedical omics: first insights of a new MSc degree of the University of Milan. Tumori. 2021;3008916211047268doi: 10.1177/03008916211047268.
Alcalay, M., Jereczek-Fossa, B. A., Pepa, M., Volpe, S., Zaffaroni, M., Fiore, F., Marvaso, G., Pravettoni, G., Curigliano, G., Santaguida, S., & Pelicci, P. G. (2021). Biomedical omics: first insights of a new MSc degree of the University of Milan. Tumori, 3008916211047268. https://doi.org/10.1177/03008916211047268
Alcalay, Myriam, et al. "Biomedical omics: first insights of a new MSc degree of the University of Milan." Tumori vol. (2021): 3008916211047268. doi: https://doi.org/10.1177/03008916211047268
Alcalay M, Jereczek-Fossa BA, Pepa M, Volpe S, Zaffaroni M, Fiore F, Marvaso G, Pravettoni G, Curigliano G, Santaguida S, Pelicci PG. Biomedical omics: first insights of a new MSc degree of the University of Milan. Tumori. 2021 Sep 29;3008916211047268. doi: 10.1177/03008916211047268. Epub 2021 Sep 29. PMID: 34585604.
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Adaptive mechanoproperties mediated by the formin FMN1 characterize glioblastoma fitness for invasion.

Developmental cell

Monzo P, Crestani M, Chong YK, Ghisleni A, Hennig K, Li Q, Kakogiannos N, Giannotta M, Richichi C, Dini T, Dejana E, Maiuri P, Balland M, Sheetz MP, Pelicci G, Ang BT, Tang C, Gauthier NC.
PMID: 34559979
Dev Cell. 2021 Oct 25;56(20):2841-2855.e8. doi: 10.1016/j.devcel.2021.09.007. Epub 2021 Sep 23.

Glioblastoma are heterogeneous tumors composed of highly invasive and highly proliferative clones. Heterogeneity in invasiveness could emerge from discrete biophysical properties linked to specific molecular expression. We identified clones of patient-derived glioma propagating cells that were either highly proliferative...

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Monzo P, Crestani M, Chong YK, et al. Adaptive mechanoproperties mediated by the formin FMN1 characterize glioblastoma fitness for invasion. Dev Cell. 2021;56(20):2841-2855.e8doi: 10.1016/j.devcel.2021.09.007.
Monzo, P., Crestani, M., Chong, Y. K., Ghisleni, A., Hennig, K., Li, Q., Kakogiannos, N., Giannotta, M., Richichi, C., Dini, T., Dejana, E., Maiuri, P., Balland, M., Sheetz, M. P., Pelicci, G., Ang, B. T., Tang, C., & Gauthier, N. C. (2021). Adaptive mechanoproperties mediated by the formin FMN1 characterize glioblastoma fitness for invasion. Developmental cell, 56(20), 2841-2855.e8. https://doi.org/10.1016/j.devcel.2021.09.007
Monzo, Pascale, et al. "Adaptive mechanoproperties mediated by the formin FMN1 characterize glioblastoma fitness for invasion." Developmental cell vol. 56,20 (2021): 2841-2855.e8. doi: https://doi.org/10.1016/j.devcel.2021.09.007
Monzo P, Crestani M, Chong YK, Ghisleni A, Hennig K, Li Q, Kakogiannos N, Giannotta M, Richichi C, Dini T, Dejana E, Maiuri P, Balland M, Sheetz MP, Pelicci G, Ang BT, Tang C, Gauthier NC. Adaptive mechanoproperties mediated by the formin FMN1 characterize glioblastoma fitness for invasion. Dev Cell. 2021 Oct 25;56(20):2841-2855.e8. doi: 10.1016/j.devcel.2021.09.007. Epub 2021 Sep 23. PMID: 34559979.
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Understanding COVID-19 in children may provide clues to protect at-risk populations.

BMJ paediatrics open

Do LAH, Anderson J, Sutton P, Pellicci DG, Mulholland K, Licciardi PV.
PMID: 32455170
BMJ Paediatr Open. 2020 May 14;4(1):e000702. doi: 10.1136/bmjpo-2020-000702. eCollection 2020.

No abstract available.

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Do LAH, Anderson J, Sutton P, et al. Understanding COVID-19 in children may provide clues to protect at-risk populations. BMJ Paediatr Open. 2020;4(1):e000702doi: 10.1136/bmjpo-2020-000702.
Do, L. A. H., Anderson, J., Sutton, P., Pellicci, D. G., Mulholland, K., & Licciardi, P. V. (2020). Understanding COVID-19 in children may provide clues to protect at-risk populations. BMJ paediatrics open, 4(1), e000702. https://doi.org/10.1136/bmjpo-2020-000702
Do, Lien Anh Ha, et al. "Understanding COVID-19 in children may provide clues to protect at-risk populations." BMJ paediatrics open vol. 4,1 (2020): e000702. doi: https://doi.org/10.1136/bmjpo-2020-000702
Do LAH, Anderson J, Sutton P, Pellicci DG, Mulholland K, Licciardi PV. Understanding COVID-19 in children may provide clues to protect at-risk populations. BMJ Paediatr Open. 2020 May 14;4(1):e000702. doi: 10.1136/bmjpo-2020-000702. eCollection 2020. PMID: 32455170; PMCID: PMC7234865.
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Correction: The Adaptor Protein Rai/ShcC Promotes Astrocyte-Dependent Inflammation during Experimental Autoimmune Encephalomyelitis.

Journal of immunology (Baltimore, Md. : 1950)

Ulivieri C, Savino MT, Luccarini I, Fanigliulo E, Aldinucci A, Bonechi E, Benagiano M, Ortensi B, Pelicci G, D'Elios MM, Ballerini C, Baldari CT.
PMID: 27815449
J Immunol. 2016 Nov 15;197(10):4178. doi: 10.4049/jimmunol.1601660.

No abstract available.

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Ulivieri C, Savino MT, Luccarini I, et al. Correction: The Adaptor Protein Rai/ShcC Promotes Astrocyte-Dependent Inflammation during Experimental Autoimmune Encephalomyelitis. J Immunol. 2016;197(10):4178doi: 10.4049/jimmunol.1601660.
Ulivieri, C., Savino, M. T., Luccarini, I., Fanigliulo, E., Aldinucci, A., Bonechi, E., Benagiano, M., Ortensi, B., Pelicci, G., D'Elios, M. M., Ballerini, C., & Baldari, C. T. (2016). Correction: The Adaptor Protein Rai/ShcC Promotes Astrocyte-Dependent Inflammation during Experimental Autoimmune Encephalomyelitis. Journal of immunology (Baltimore, Md. : 1950), 197(10), 4178. https://doi.org/10.4049/jimmunol.1601660
Ulivieri, Cristina, et al. "Correction: The Adaptor Protein Rai/ShcC Promotes Astrocyte-Dependent Inflammation during Experimental Autoimmune Encephalomyelitis." Journal of immunology (Baltimore, Md. : 1950) vol. 197,10 (2016): 4178. doi: https://doi.org/10.4049/jimmunol.1601660
Ulivieri C, Savino MT, Luccarini I, Fanigliulo E, Aldinucci A, Bonechi E, Benagiano M, Ortensi B, Pelicci G, D'Elios MM, Ballerini C, Baldari CT. Correction: The Adaptor Protein Rai/ShcC Promotes Astrocyte-Dependent Inflammation during Experimental Autoimmune Encephalomyelitis. J Immunol. 2016 Nov 15;197(10):4178. doi: 10.4049/jimmunol.1601660. PMID: 27815449.
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