Advanced Search
Display options
Filter resources
Text Availability
Article type
Publication date
Species
Language
Sex
Age
Showing 1 to 2 of 2 entries
Sorted by: Best Match Show Resources per page
Effect of nitric oxide synthesis modification on renal function in gentamicin-induced nephrotoxicity.

Environmental toxicology and pharmacology

Valdivielso JM, Rivas-Cabañero L, Pérez-Barriocanal F, López-Novoa JM.
PMID: 21781769
Environ Toxicol Pharmacol. 1997 Jun 06;3(2):123-8. doi: 10.1016/s1382-6689(97)00148-8.

We evaluated the effect of acute or chronic nitric oxide (NO) synthesis activation or inhibition in rats with gentamicin-induced acute renal failure. Rats received gentamicin 100 mg/kg per day for 6 days, or isotonic saline. Some animals of each...

Cite
Valdivielso JM, Rivas-Cabañero L, Pérez-Barriocanal F, et al. Effect of nitric oxide synthesis modification on renal function in gentamicin-induced nephrotoxicity. Environ Toxicol Pharmacol. 1997;3(2):123-8doi: 10.1016/s1382-6689(97)00148-8.
Valdivielso, J. M., Rivas-Cabañero, L., Pérez-Barriocanal, F., & López-Novoa, J. M. (1997). Effect of nitric oxide synthesis modification on renal function in gentamicin-induced nephrotoxicity. Environmental toxicology and pharmacology, 3(2), 123-8. https://doi.org/10.1016/s1382-6689(97)00148-8
Valdivielso, J M, et al. "Effect of nitric oxide synthesis modification on renal function in gentamicin-induced nephrotoxicity." Environmental toxicology and pharmacology vol. 3,2 (1997): 123-8. doi: https://doi.org/10.1016/s1382-6689(97)00148-8
Valdivielso JM, Rivas-Cabañero L, Pérez-Barriocanal F, López-Novoa JM. Effect of nitric oxide synthesis modification on renal function in gentamicin-induced nephrotoxicity. Environ Toxicol Pharmacol. 1997 Jun 06;3(2):123-8. doi: 10.1016/s1382-6689(97)00148-8. PMID: 21781769.
Copy Download .nbib Format: NLM
Role of inflammation in túbulo-interstitial damage associated to obstructive nephropathy.

Journal of inflammation (London, England)

Grande MT, Pérez-Barriocanal F, López-Novoa JM.
PMID: 20412564
J Inflamm (Lond). 2010 Apr 22;7:19. doi: 10.1186/1476-9255-7-19.

Obstructive nephropathy is characterized by an inflammatory state in the kidney, that is promoted by cytokines and growth factors produced by damaged tubular cells, infiltrated macrophages and accumulated myofibroblasts. This inflammatory state contributes to tubular atrophy and interstitial fibrosis...

Cite
Grande MT, Pérez-Barriocanal F, López-Novoa JM. Role of inflammation in túbulo-interstitial damage associated to obstructive nephropathy. J Inflamm (Lond). 2010;7:19doi: 10.1186/1476-9255-7-19.
Grande, M. T., Pérez-Barriocanal, F., & López-Novoa, J. M. (2010). Role of inflammation in túbulo-interstitial damage associated to obstructive nephropathy. Journal of inflammation (London, England), 719. https://doi.org/10.1186/1476-9255-7-19
Grande, María T, et al. "Role of inflammation in túbulo-interstitial damage associated to obstructive nephropathy." Journal of inflammation (London, England) vol. 7 (2010): 19. doi: https://doi.org/10.1186/1476-9255-7-19
Grande MT, Pérez-Barriocanal F, López-Novoa JM. Role of inflammation in túbulo-interstitial damage associated to obstructive nephropathy. J Inflamm (Lond). 2010 Apr 22;7:19. doi: 10.1186/1476-9255-7-19. PMID: 20412564; PMCID: PMC2873503.
Copy Download .nbib Format: NLM
Showing 1 to 2 of 2 entries