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Two-Year Outcomes from a Randomized Controlled Trial of Minimally Invasive Sacroiliac Joint Fusion vs. Non-Surgical Management for Sacroiliac Joint Dysfunction.

International journal of spine surgery

Polly DW, Swofford J, Whang PG, Frank CJ, Glaser JA, Limoni RP, Cher DJ, Wine KD, Sembrano JN.
PMID: 27652199
Int J Spine Surg. 2016 Aug 23;10:28. doi: 10.14444/3028. eCollection 2016.

BACKGROUND: Sacroiliac joint (SIJ) dysfunction is an important and underappreciated cause of chronic low back pain.OBJECTIVE: To prospectively and concurrently compare outcomes after surgical and non-surgical treatment for chronic SIJ dysfunction.METHODS: One hundred and forty-eight subjects with SIJ dysfunction...

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Polly DW, Swofford J, Whang PG, et al. Two-Year Outcomes from a Randomized Controlled Trial of Minimally Invasive Sacroiliac Joint Fusion vs. Non-Surgical Management for Sacroiliac Joint Dysfunction. Int J Spine Surg. 2016;10:28doi: 10.14444/3028.
Polly, D. W., Swofford, J., Whang, P. G., Frank, C. J., Glaser, J. A., Limoni, R. P., Cher, D. J., Wine, K. D., Sembrano, J. N. (2016). Two-Year Outcomes from a Randomized Controlled Trial of Minimally Invasive Sacroiliac Joint Fusion vs. Non-Surgical Management for Sacroiliac Joint Dysfunction. International journal of spine surgery, 1028. https://doi.org/10.14444/3028
Polly, David W, et al. "Two-Year Outcomes from a Randomized Controlled Trial of Minimally Invasive Sacroiliac Joint Fusion vs. Non-Surgical Management for Sacroiliac Joint Dysfunction." International journal of spine surgery vol. 10 (2016): 28. doi: https://doi.org/10.14444/3028
Polly DW, Swofford J, Whang PG, Frank CJ, Glaser JA, Limoni RP, Cher DJ, Wine KD, Sembrano JN. Two-Year Outcomes from a Randomized Controlled Trial of Minimally Invasive Sacroiliac Joint Fusion vs. Non-Surgical Management for Sacroiliac Joint Dysfunction. Int J Spine Surg. 2016 Aug 23;10:28. doi: 10.14444/3028. eCollection 2016. PMID: 27652199; PMCID: PMC5027818.
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[Conjugal violence during pregnancy: a literature review.].

Sante mentale au Quebec

Séguin L, Pimont M, Rinfret-Raynor M, Cantin S.
PMID: 18253581
Sante Ment Que. 2000;25(1):288-312.

Conjugal violence is a complicated issue which scope and consequences are increasingly documented. Among the many facets of the phenomenon, its occurrence during pregnancy is particularly worrisome. This article reviews the scientific literature pertaining to this issue. After establishing...

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Séguin L, Pimont M, Rinfret-Raynor M, et al. Violence conjugale pendant la grossesse : recension des écrits. [Conjugal violence during pregnancy: a literature review.]. Sante Ment Que. 2000;25(1):288-312
Séguin, L., Pimont, M., Rinfret-Raynor, M., & Cantin, S. (2000). Violence conjugale pendant la grossesse : recension des écrits. [Conjugal violence during pregnancy: a literature review.]. Sante mentale au Quebec, 25(1), 288-312.
Séguin, L, et al. "Violence conjugale pendant la grossesse : recension des écrits." [Conjugal violence during pregnancy: a literature review.]. Sante mentale au Quebec vol. 25,1 (2000): 288-312.
Séguin L, Pimont M, Rinfret-Raynor M, Cantin S. Violence conjugale pendant la grossesse : recension des écrits. [Conjugal violence during pregnancy: a literature review.]. Sante Ment Que. 2000;25(1):288-312. French. PMID: 18253581.
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Cardiomyogenic Differentiation Potential of Human Dilated Myocardium-Derived Mesenchymal Stem/Stromal Cells: The Impact of HDAC Inhibitor SAHA and Biomimetic Matrices.

International journal of molecular sciences

Miksiunas R, Aldonyte R, Vailionyte A, Jelinskas T, Eimont R, Stankeviciene G, Cepla V, Valiokas R, Rucinskas K, Janusauskas V, Labeit S, Bironaite D.
PMID: 34884505
Int J Mol Sci. 2021 Nov 24;22(23). doi: 10.3390/ijms222312702.

Dilated cardiomyopathy (DCM) is the most common type of nonischemic cardiomyopathy characterized by left ventricular or biventricular dilation and impaired contraction leading to heart failure and even patients' death. Therefore, it is important to search for new cardiac tissue...

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Miksiunas R, Aldonyte R, Vailionyte A, et al. Cardiomyogenic Differentiation Potential of Human Dilated Myocardium-Derived Mesenchymal Stem/Stromal Cells: The Impact of HDAC Inhibitor SAHA and Biomimetic Matrices. Int J Mol Sci. 2021;22(23)doi: 10.3390/ijms222312702.
Miksiunas, R., Aldonyte, R., Vailionyte, A., Jelinskas, T., Eimont, R., Stankeviciene, G., Cepla, V., Valiokas, R., Rucinskas, K., Janusauskas, V., Labeit, S., & Bironaite, D. (2021). Cardiomyogenic Differentiation Potential of Human Dilated Myocardium-Derived Mesenchymal Stem/Stromal Cells: The Impact of HDAC Inhibitor SAHA and Biomimetic Matrices. International journal of molecular sciences, 22(23), . https://doi.org/10.3390/ijms222312702
Miksiunas, Rokas, et al. "Cardiomyogenic Differentiation Potential of Human Dilated Myocardium-Derived Mesenchymal Stem/Stromal Cells: The Impact of HDAC Inhibitor SAHA and Biomimetic Matrices." International journal of molecular sciences vol. 22,23 (2021). doi: https://doi.org/10.3390/ijms222312702
Miksiunas R, Aldonyte R, Vailionyte A, Jelinskas T, Eimont R, Stankeviciene G, Cepla V, Valiokas R, Rucinskas K, Janusauskas V, Labeit S, Bironaite D. Cardiomyogenic Differentiation Potential of Human Dilated Myocardium-Derived Mesenchymal Stem/Stromal Cells: The Impact of HDAC Inhibitor SAHA and Biomimetic Matrices. Int J Mol Sci. 2021 Nov 24;22(23). doi: 10.3390/ijms222312702. PMID: 34884505.
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Increased likelihood of heat-induced large wildfires in the Mediterranean Basin.

Scientific reports

Ruffault J, Curt T, Moron V, Trigo RM, Mouillot F, Koutsias N, Pimont F, Martin-StPaul N, Barbero R, Dupuy JL, Russo A, Belhadj-Khedher C.
PMID: 32796945
Sci Rep. 2020 Aug 14;10(1):13790. doi: 10.1038/s41598-020-70069-z.

Wildfire activity is expected to increase across the Mediterranean Basin because of climate change. However, the effects of future climate change on the combinations of atmospheric conditions that promote wildfire activity remain largely unknown. Using a fire-weather based classification...

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Ruffault J, Curt T, Moron V, et al. Increased likelihood of heat-induced large wildfires in the Mediterranean Basin. Sci Rep. 2020;10(1):13790doi: 10.1038/s41598-020-70069-z.
Ruffault, J., Curt, T., Moron, V., Trigo, R. M., Mouillot, F., Koutsias, N., Pimont, F., Martin-StPaul, N., Barbero, R., Dupuy, J. L., Russo, A., & Belhadj-Khedher, C. (2020). Increased likelihood of heat-induced large wildfires in the Mediterranean Basin. Scientific reports, 10(1), 13790. https://doi.org/10.1038/s41598-020-70069-z
Ruffault, Julien, et al. "Increased likelihood of heat-induced large wildfires in the Mediterranean Basin." Scientific reports vol. 10,1 (2020): 13790. doi: https://doi.org/10.1038/s41598-020-70069-z
Ruffault J, Curt T, Moron V, Trigo RM, Mouillot F, Koutsias N, Pimont F, Martin-StPaul N, Barbero R, Dupuy JL, Russo A, Belhadj-Khedher C. Increased likelihood of heat-induced large wildfires in the Mediterranean Basin. Sci Rep. 2020 Aug 14;10(1):13790. doi: 10.1038/s41598-020-70069-z. PMID: 32796945; PMCID: PMC7427790.
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Enhancing and Extending Biological Performance and Resilience.

Dose-response : a publication of International Hormesis Society

Leak RK, Calabrese EJ, Kozumbo WJ, Gidday JM, Johnson TE, Mitchell JR, Ozaki CK, Wetzker R, Bast A, Belz RG, Bøtker HE, Koch S, Mattson MP, Simon RP, Jirtle RL, Andersen ME.
PMID: 30140178
Dose Response. 2018 Aug 15;16(3):1559325818784501. doi: 10.1177/1559325818784501. eCollection 2018.

Human performance, endurance, and resilience have biological limits that are genetically and epigenetically predetermined but perhaps not yet optimized. There are few systematic, rigorous studies on how to raise these limits and reach the true maxima. Achieving this goal...

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Leak RK, Calabrese EJ, Kozumbo WJ, et al. Enhancing and Extending Biological Performance and Resilience. Dose Response. 2018;16(3):1559325818784501doi: 10.1177/1559325818784501.
Leak, R. K., Calabrese, E. J., Kozumbo, W. J., Gidday, J. M., Johnson, T. E., Mitchell, J. R., Ozaki, C. K., Wetzker, R., Bast, A., Belz, R. G., Bøtker, H. E., Koch, S., Mattson, M. P., Simon, R. P., Jirtle, R. L., & Andersen, M. E. (2018). Enhancing and Extending Biological Performance and Resilience. Dose-response : a publication of International Hormesis Society, 16(3), 1559325818784501. https://doi.org/10.1177/1559325818784501
Leak, Rehana K, et al. "Enhancing and Extending Biological Performance and Resilience." Dose-response : a publication of International Hormesis Society vol. 16,3 (2018): 1559325818784501. doi: https://doi.org/10.1177/1559325818784501
Leak RK, Calabrese EJ, Kozumbo WJ, Gidday JM, Johnson TE, Mitchell JR, Ozaki CK, Wetzker R, Bast A, Belz RG, Bøtker HE, Koch S, Mattson MP, Simon RP, Jirtle RL, Andersen ME. Enhancing and Extending Biological Performance and Resilience. Dose Response. 2018 Aug 15;16(3):1559325818784501. doi: 10.1177/1559325818784501. eCollection 2018. PMID: 30140178; PMCID: PMC6096685.
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Adenosine dysfunction in astrogliosis: cause for seizure generation?.

Neuron glia biology

Li T, Quan Lan J, Fredholm BB, Simon RP, Boison D.
PMID: 18634566
Neuron Glia Biol. 2007 Nov;3(4):353-66. doi: 10.1017/S1740925X0800015X.

Epilepsy is characterized by both neuronal and astroglial dysfunction. The endogenous anticonvulsant adenosine, the level of which is largely controlled by astrocytes, might provide a crucial link between astrocyte and neuron dysfunction in epilepsy. Here we have studied astrogliosis,...

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Li T, Quan Lan J, Fredholm BB, et al. Adenosine dysfunction in astrogliosis: cause for seizure generation?. Neuron Glia Biol. 2007;3(4):353-66doi: 10.1017/S1740925X0800015X.
Li, T., Quan Lan, J., Fredholm, B. B., Simon, R. P., & Boison, D. (2007). Adenosine dysfunction in astrogliosis: cause for seizure generation?. Neuron glia biology, 3(4), 353-66. https://doi.org/10.1017/S1740925X0800015X
Li, Tianfu, et al. "Adenosine dysfunction in astrogliosis: cause for seizure generation?." Neuron glia biology vol. 3,4 (2007): 353-66. doi: https://doi.org/10.1017/S1740925X0800015X
Li T, Quan Lan J, Fredholm BB, Simon RP, Boison D. Adenosine dysfunction in astrogliosis: cause for seizure generation?. Neuron Glia Biol. 2007 Nov;3(4):353-66. doi: 10.1017/S1740925X0800015X. PMID: 18634566; PMCID: PMC2561997.
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Expression of the proto-oncogene bcl-2 is increased in the rat brain following kainate-induced seizures.

Restorative neurology and neuroscience

Graham SH, Chen J, Stetler RA, Zhu RL, Jin KL, Simon RP.
PMID: 21551913
Restor Neurol Neurosci. 1996 Jan 01;9(4):243-50. doi: 10.3233/RNN-1996-9407.

The proto-oncogene bcl-2 is an important suppressor of apoptotic cell death in development and of both apoptotic and necrotic cell death in mature neurons. We studied expression of bcl-2 and the related gene, bax, which may promote cell death,...

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Graham SH, Chen J, Stetler RA, et al. Expression of the proto-oncogene bcl-2 is increased in the rat brain following kainate-induced seizures. Restor Neurol Neurosci. 1996;9(4):243-50doi: 10.3233/RNN-1996-9407.
Graham, S. H., Chen, J., Stetler, R. A., Zhu, R. L., Jin, K. L., & Simon, R. P. (1996). Expression of the proto-oncogene bcl-2 is increased in the rat brain following kainate-induced seizures. Restorative neurology and neuroscience, 9(4), 243-50. https://doi.org/10.3233/RNN-1996-9407
Graham, S H, et al. "Expression of the proto-oncogene bcl-2 is increased in the rat brain following kainate-induced seizures." Restorative neurology and neuroscience vol. 9,4 (1996): 243-50. doi: https://doi.org/10.3233/RNN-1996-9407
Graham SH, Chen J, Stetler RA, Zhu RL, Jin KL, Simon RP. Expression of the proto-oncogene bcl-2 is increased in the rat brain following kainate-induced seizures. Restor Neurol Neurosci. 1996 Jan 01;9(4):243-50. doi: 10.3233/RNN-1996-9407. PMID: 21551913.
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Expression of neurogenesis genes in human temporal lobe epilepsy with hippocampal sclerosis.

International journal of physiology, pathophysiology and pharmacology

Engel T, Schindler CK, Sanz-Rodriguez A, Conroy RM, Meller R, Simon RP, Henshall DC.
PMID: 21479101
Int J Physiol Pathophysiol Pharmacol. 2011;3(1):38-47. Epub 2010 Dec 15.

Both evoked and spontaneous seizures have been reported to increase neurogenesis in animal models. Less is known about whether neurogenesis and markers thereof are aberrantly expressed in human temporal lobe epilepsy (TLE) with hippocampal sclerosis. In the present study...

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Engel T, Schindler CK, Sanz-Rodriguez A, et al. Expression of neurogenesis genes in human temporal lobe epilepsy with hippocampal sclerosis. Int J Physiol Pathophysiol Pharmacol. 2010;3(1):38-47
Engel, T., Schindler, C. K., Sanz-Rodriguez, A., Conroy, R. M., Meller, R., Simon, R. P., & Henshall, D. C. (2011). Expression of neurogenesis genes in human temporal lobe epilepsy with hippocampal sclerosis. International journal of physiology, pathophysiology and pharmacology, 3(1), 38-47.
Engel, Tobias, et al. "Expression of neurogenesis genes in human temporal lobe epilepsy with hippocampal sclerosis." International journal of physiology, pathophysiology and pharmacology vol. 3,1 (2011): 38-47.
Engel T, Schindler CK, Sanz-Rodriguez A, Conroy RM, Meller R, Simon RP, Henshall DC. Expression of neurogenesis genes in human temporal lobe epilepsy with hippocampal sclerosis. Int J Physiol Pathophysiol Pharmacol. 2011;3(1):38-47. Epub 2010 Dec 15. PMID: 21479101; PMCID: PMC3068852.
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Transcriptional Response of Polycomb Group Genes to Status Epilepticus in Mice is Modified by Prior Exposure to Epileptic Preconditioning.

Frontiers in neurology

Reynolds JP, Miller-Delaney SF, Jimenez-Mateos EM, Sano T, McKiernan RC, Simon RP, Henshall DC.
PMID: 25806020
Front Neurol. 2015 Mar 10;6:46. doi: 10.3389/fneur.2015.00046. eCollection 2015.

Exposure of the brain to brief, non-harmful seizures can activate protective mechanisms that temporarily generate a damage-refractory state. This process, termed epileptic tolerance, is associated with large-scale down-regulation of gene expression. Polycomb group (PcG) proteins are master controllers of...

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Reynolds JP, Miller-Delaney SF, Jimenez-Mateos EM, et al. Transcriptional Response of Polycomb Group Genes to Status Epilepticus in Mice is Modified by Prior Exposure to Epileptic Preconditioning. Front Neurol. 2015;6:46doi: 10.3389/fneur.2015.00046.
Reynolds, J. P., Miller-Delaney, S. F., Jimenez-Mateos, E. M., Sano, T., McKiernan, R. C., Simon, R. P., & Henshall, D. C. (2015). Transcriptional Response of Polycomb Group Genes to Status Epilepticus in Mice is Modified by Prior Exposure to Epileptic Preconditioning. Frontiers in neurology, 646. https://doi.org/10.3389/fneur.2015.00046
Reynolds, James P, et al. "Transcriptional Response of Polycomb Group Genes to Status Epilepticus in Mice is Modified by Prior Exposure to Epileptic Preconditioning." Frontiers in neurology vol. 6 (2015): 46. doi: https://doi.org/10.3389/fneur.2015.00046
Reynolds JP, Miller-Delaney SF, Jimenez-Mateos EM, Sano T, McKiernan RC, Simon RP, Henshall DC. Transcriptional Response of Polycomb Group Genes to Status Epilepticus in Mice is Modified by Prior Exposure to Epileptic Preconditioning. Front Neurol. 2015 Mar 10;6:46. doi: 10.3389/fneur.2015.00046. eCollection 2015. PMID: 25806020; PMCID: PMC4354380.
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Blood transcriptome changes after stroke in an African American population.

Annals of clinical and translational neurology

Meller R, Pearson AN, Hardy JJ, Hall CL, McGuire D, Frankel MR, Simon RP.
PMID: 26900583
Ann Clin Transl Neurol. 2016 Jan 15;3(2):70-81. doi: 10.1002/acn3.272. eCollection 2016 Feb.

OBJECTIVE: Molecular diagnostic medicine holds much promise to change point of care treatment. An area where additional diagnostic tools are needed is in acute stroke care, to assist in diagnosis and prognosis. Previous studies using microarray-based gene expression analysis...

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Meller R, Pearson AN, Hardy JJ, et al. Blood transcriptome changes after stroke in an African American population. Ann Clin Transl Neurol. 2016;3(2):70-81doi: 10.1002/acn3.272.
Meller, R., Pearson, A. N., Hardy, J. J., Hall, C. L., McGuire, D., Frankel, M. R., & Simon, R. P. (2016). Blood transcriptome changes after stroke in an African American population. Annals of clinical and translational neurology, 3(2), 70-81. https://doi.org/10.1002/acn3.272
Meller, Robert, et al. "Blood transcriptome changes after stroke in an African American population." Annals of clinical and translational neurology vol. 3,2 (2016): 70-81. doi: https://doi.org/10.1002/acn3.272
Meller R, Pearson AN, Hardy JJ, Hall CL, McGuire D, Frankel MR, Simon RP. Blood transcriptome changes after stroke in an African American population. Ann Clin Transl Neurol. 2016 Jan 15;3(2):70-81. doi: 10.1002/acn3.272. eCollection 2016 Feb. PMID: 26900583; PMCID: PMC4748310.
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MALDI-TOF-Based Affinity Selection Mass Spectrometry for Automated Screening of Protein-Ligand Interactions at High Throughput.

SLAS discovery : advancing life sciences R & D

Simon RP, Winter M, Kleiner C, Wehrle L, Karnath M, Ries R, Zeeb M, Schnapp G, Fiegen D, Häbe TT, Runge F, Bretschneider T, Luippold AH, Bischoff D, Reindl W, Büttner FH.
PMID: 33073664
SLAS Discov. 2021 Jan;26(1):44-57. doi: 10.1177/2472555220959266. Epub 2020 Oct 17.

Demonstration of in vitro target engagement for small-molecule ligands by measuring binding to a molecular target is an established approach in early drug discovery and a pivotal step in high-throughput screening (HTS)-based compound triaging. We describe the setup, evaluation,...

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Simon RP, Winter M, Kleiner C, et al. MALDI-TOF-Based Affinity Selection Mass Spectrometry for Automated Screening of Protein-Ligand Interactions at High Throughput. SLAS Discov. 2020;26(1):44-57doi: 10.1177/2472555220959266.
Simon, R. P., Winter, M., Kleiner, C., Wehrle, L., Karnath, M., Ries, R., Zeeb, M., Schnapp, G., Fiegen, D., Häbe, T. T., Runge, F., Bretschneider, T., Luippold, A. H., Bischoff, D., Reindl, W., & Büttner, F. H. (2021). MALDI-TOF-Based Affinity Selection Mass Spectrometry for Automated Screening of Protein-Ligand Interactions at High Throughput. SLAS discovery : advancing life sciences R & D, 26(1), 44-57. https://doi.org/10.1177/2472555220959266
Simon, Roman P, et al. "MALDI-TOF-Based Affinity Selection Mass Spectrometry for Automated Screening of Protein-Ligand Interactions at High Throughput." SLAS discovery : advancing life sciences R & D vol. 26,1 (2021): 44-57. doi: https://doi.org/10.1177/2472555220959266
Simon RP, Winter M, Kleiner C, Wehrle L, Karnath M, Ries R, Zeeb M, Schnapp G, Fiegen D, Häbe TT, Runge F, Bretschneider T, Luippold AH, Bischoff D, Reindl W, Büttner FH. MALDI-TOF-Based Affinity Selection Mass Spectrometry for Automated Screening of Protein-Ligand Interactions at High Throughput. SLAS Discov. 2021 Jan;26(1):44-57. doi: 10.1177/2472555220959266. Epub 2020 Oct 17. PMID: 33073664.
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Diagnosing and treating insomnia in older patients.

Journal of the American Medical Directors Association

Erman MK, Simon RP, Smith DA.
PMID: 12818010
J Am Med Dir Assoc. 2000 Nov-Dec;1(6):S15-9.

No abstract available.

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Erman MK, Simon RP, Smith DA. Diagnosing and treating insomnia in older patients. J Am Med Dir Assoc. 2000;1(6):S15-9
Erman, M. K., Simon, R. P., & Smith, D. A. (2000). Diagnosing and treating insomnia in older patients. Journal of the American Medical Directors Association, 1(6), S15-9.
Erman, M K, et al. "Diagnosing and treating insomnia in older patients." Journal of the American Medical Directors Association vol. 1,6 (2000): S15-9.
Erman MK, Simon RP, Smith DA. Diagnosing and treating insomnia in older patients. J Am Med Dir Assoc. 2000 Nov-Dec;1(6):S15-9. PMID: 12818010.
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Showing 1 to 12 of 21 entries