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Perineural invasion is a strong and independent predictor of lymph node involvement in colorectal cancer.

Diseases of the colon and rectum

Betge J, Pollheimer MJ, Kornprat P, Rehak P, Vieth M, Langner C.
PMID: 21979193
Dis Colon Rectum. 2011 Nov;54(11):e273. doi: 10.1097/DCR.0b013e31822c67ea.

No abstract available.

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Betge J, Pollheimer MJ, Kornprat P, et al. Perineural invasion is a strong and independent predictor of lymph node involvement in colorectal cancer. Dis Colon Rectum. 2011;54(11):e273doi: 10.1097/DCR.0b013e31822c67ea.
Betge, J., Pollheimer, M. J., Kornprat, P., Rehak, P., Vieth, M., & Langner, C. (2011). Perineural invasion is a strong and independent predictor of lymph node involvement in colorectal cancer. Diseases of the colon and rectum, 54(11), e273. https://doi.org/10.1097/DCR.0b013e31822c67ea
Betge, Johannes, et al. "Perineural invasion is a strong and independent predictor of lymph node involvement in colorectal cancer." Diseases of the colon and rectum vol. 54,11 (2011): e273. doi: https://doi.org/10.1097/DCR.0b013e31822c67ea
Betge J, Pollheimer MJ, Kornprat P, Rehak P, Vieth M, Langner C. Perineural invasion is a strong and independent predictor of lymph node involvement in colorectal cancer. Dis Colon Rectum. 2011 Nov;54(11):e273. doi: 10.1097/DCR.0b013e31822c67ea. PMID: 21979193.
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Human diamine oxidase cellular binding and internalization in vitro and rapid clearance in vivo are not mediated by N-glycans but by heparan sulfate proteoglycan interactions.

Glycobiology

Gludovacz E, Schuetzenberger K, Resch M, Tillmann K, Petroczi K, Vondra S, Vakal S, Schosserer M, Virgolini N, Pollheimer J, Salminen TA, Jilma B, Borth N, Boehm T.
PMID: 32985651
Glycobiology. 2021 May 03;31(4):444-458. doi: 10.1093/glycob/cwaa090.

Human diamine oxidase (hDAO) rapidly inactivates histamine by deamination. No pharmacokinetic data are available to better understand its potential as a new therapeutic modality for diseases with excess local and systemic histamine, like anaphylaxis, urticaria or mastocytosis. After intravenous...

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Gludovacz E, Schuetzenberger K, Resch M, et al. Human diamine oxidase cellular binding and internalization in vitro and rapid clearance in vivo are not mediated by N-glycans but by heparan sulfate proteoglycan interactions. Glycobiology. 2021;31(4):444-458doi: 10.1093/glycob/cwaa090.
Gludovacz, E., Schuetzenberger, K., Resch, M., Tillmann, K., Petroczi, K., Vondra, S., Vakal, S., Schosserer, M., Virgolini, N., Pollheimer, J., Salminen, T. A., Jilma, B., Borth, N., & Boehm, T. (2021). Human diamine oxidase cellular binding and internalization in vitro and rapid clearance in vivo are not mediated by N-glycans but by heparan sulfate proteoglycan interactions. Glycobiology, 31(4), 444-458. https://doi.org/10.1093/glycob/cwaa090
Gludovacz, Elisabeth, et al. "Human diamine oxidase cellular binding and internalization in vitro and rapid clearance in vivo are not mediated by N-glycans but by heparan sulfate proteoglycan interactions." Glycobiology vol. 31,4 (2021): 444-458. doi: https://doi.org/10.1093/glycob/cwaa090
Gludovacz E, Schuetzenberger K, Resch M, Tillmann K, Petroczi K, Vondra S, Vakal S, Schosserer M, Virgolini N, Pollheimer J, Salminen TA, Jilma B, Borth N, Boehm T. Human diamine oxidase cellular binding and internalization in vitro and rapid clearance in vivo are not mediated by N-glycans but by heparan sulfate proteoglycan interactions. Glycobiology. 2021 May 03;31(4):444-458. doi: 10.1093/glycob/cwaa090. PMID: 32985651.
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Genetic loss of the muscarinic M.

Hepatology research : the official journal of the Japan Society of Hepatology

Durchschein F, Krones E, Pollheimer MJ, Zollner G, Wagner M, Raufman JP, Fickert P.
PMID: 28635176
Hepatol Res. 2018 Feb;48(3):E68-E77. doi: 10.1111/hepr.12928. Epub 2017 Aug 26.

AIM: Hepatic innervation represents a potentially underestimated regulator of liver function and regeneration. The muscarinic 3 receptor (MMETHODS: We compared bile flow and composition in MRESULTS: MCONCLUSION: Our experimental findings suggest that M

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Durchschein F, Krones E, Pollheimer MJ, et al. Genetic loss of the muscarinic M. Hepatol Res. 2017;48(3):E68-E77doi: 10.1111/hepr.12928.
Durchschein, F., Krones, E., Pollheimer, M. J., Zollner, G., Wagner, M., Raufman, J. P., & Fickert, P. (2018). Genetic loss of the muscarinic M. Hepatology research : the official journal of the Japan Society of Hepatology, 48(3), E68-E77. https://doi.org/10.1111/hepr.12928
Durchschein, Franziska, et al. "Genetic loss of the muscarinic M." Hepatology research : the official journal of the Japan Society of Hepatology vol. 48,3 (2018): E68-E77. doi: https://doi.org/10.1111/hepr.12928
Durchschein F, Krones E, Pollheimer MJ, Zollner G, Wagner M, Raufman JP, Fickert P. Genetic loss of the muscarinic M. Hepatol Res. 2018 Feb;48(3):E68-E77. doi: 10.1111/hepr.12928. Epub 2017 Aug 26. PMID: 28635176.
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PRG2 and AQPEP are misexpressed in fetal membranes in placenta previa and percreta†.

Biology of reproduction

Zhang ET, Hannibal RL, Badillo Rivera KM, Song JHT, McGowan K, Zhu X, Meinhardt G, Knöfler M, Pollheimer J, Urban AE, Folkins AK, Lyell DJ, Baker JC.
PMID: 33982062
Biol Reprod. 2021 Jul 02;105(1):244-257. doi: 10.1093/biolre/ioab068.

The obstetrical conditions placenta accreta spectrum (PAS) and placenta previa are a significant source of pregnancy-associated morbidity and mortality, yet the specific molecular and cellular underpinnings of these conditions are not known. In this study, we identified misregulated gene...

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Zhang ET, Hannibal RL, Badillo Rivera KM, et al. PRG2 and AQPEP are misexpressed in fetal membranes in placenta previa and percreta†. Biol Reprod. 2021;105(1):244-257doi: 10.1093/biolre/ioab068.
Zhang, E. T., Hannibal, R. L., Badillo Rivera, K. M., Song, J. H. T., McGowan, K., Zhu, X., Meinhardt, G., Knöfler, M., Pollheimer, J., Urban, A. E., Folkins, A. K., Lyell, D. J., & Baker, J. C. (2021). PRG2 and AQPEP are misexpressed in fetal membranes in placenta previa and percreta†. Biology of reproduction, 105(1), 244-257. https://doi.org/10.1093/biolre/ioab068
Zhang, Elisa T, et al. "PRG2 and AQPEP are misexpressed in fetal membranes in placenta previa and percreta†." Biology of reproduction vol. 105,1 (2021): 244-257. doi: https://doi.org/10.1093/biolre/ioab068
Zhang ET, Hannibal RL, Badillo Rivera KM, Song JHT, McGowan K, Zhu X, Meinhardt G, Knöfler M, Pollheimer J, Urban AE, Folkins AK, Lyell DJ, Baker JC. PRG2 and AQPEP are misexpressed in fetal membranes in placenta previa and percreta†. Biol Reprod. 2021 Jul 02;105(1):244-257. doi: 10.1093/biolre/ioab068. PMID: 33982062; PMCID: PMC8256106.
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Agonism of Prostaglandin E2 Receptor 4 Ameliorates Tubulointerstitial Injury in Nephrotoxic Serum Nephritis in Mice.

Journal of clinical medicine

Aringer I, Artinger K, Schabhüttl C, Bärnthaler T, Mooslechner AA, Kirsch A, Pollheimer M, Eller P, Rosenkranz AR, Heinemann A, Eller K.
PMID: 33670614
J Clin Med. 2021 Feb 18;10(4). doi: 10.3390/jcm10040832.

Selectively targeting the E-type prostanoid receptor 4 (EP4) might be a new therapeutic option in the treatment of glomerulonephritis (GN), since the EP4 receptor is expressed on different immune cells, resident kidney cells, and endothelial cells, which are all...

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Aringer I, Artinger K, Schabhüttl C, et al. Agonism of Prostaglandin E2 Receptor 4 Ameliorates Tubulointerstitial Injury in Nephrotoxic Serum Nephritis in Mice. J Clin Med. 2021;10(4)doi: 10.3390/jcm10040832.
Aringer, I., Artinger, K., Schabhüttl, C., Bärnthaler, T., Mooslechner, A. A., Kirsch, A., Pollheimer, M., Eller, P., Rosenkranz, A. R., Heinemann, A., & Eller, K. (2021). Agonism of Prostaglandin E2 Receptor 4 Ameliorates Tubulointerstitial Injury in Nephrotoxic Serum Nephritis in Mice. Journal of clinical medicine, 10(4), . https://doi.org/10.3390/jcm10040832
Aringer, Ida, et al. "Agonism of Prostaglandin E2 Receptor 4 Ameliorates Tubulointerstitial Injury in Nephrotoxic Serum Nephritis in Mice." Journal of clinical medicine vol. 10,4 (2021). doi: https://doi.org/10.3390/jcm10040832
Aringer I, Artinger K, Schabhüttl C, Bärnthaler T, Mooslechner AA, Kirsch A, Pollheimer M, Eller P, Rosenkranz AR, Heinemann A, Eller K. Agonism of Prostaglandin E2 Receptor 4 Ameliorates Tubulointerstitial Injury in Nephrotoxic Serum Nephritis in Mice. J Clin Med. 2021 Feb 18;10(4). doi: 10.3390/jcm10040832. PMID: 33670614; PMCID: PMC7922874.
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Decidual stromal cells support tolerance at the human foetal-maternal interface by inducing regulatory M2 macrophages and regulatory T-cells.

Journal of reproductive immunology

Lindau R, Vondra S, Spreckels J, Solders M, Svensson-Arvelund J, Berg G, Pollheimer J, Kaipe H, Jenmalm MC, Ernerudh J.
PMID: 34049032
J Reprod Immunol. 2021 Aug;146:103330. doi: 10.1016/j.jri.2021.103330. Epub 2021 May 12.

During pregnancy, the semi-allogeneic nature of the foetus requires maternal immune adaption and acquisition of tolerance at the foetal-maternal interface. Macrophages with regulatory properties and regulatory T (Treg) cells are central in promoting foetal tolerance and are enriched in...

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Lindau R, Vondra S, Spreckels J, et al. Decidual stromal cells support tolerance at the human foetal-maternal interface by inducing regulatory M2 macrophages and regulatory T-cells. J Reprod Immunol. 2021;146:103330doi: 10.1016/j.jri.2021.103330.
Lindau, R., Vondra, S., Spreckels, J., Solders, M., Svensson-Arvelund, J., Berg, G., Pollheimer, J., Kaipe, H., Jenmalm, M. C., & Ernerudh, J. (2021). Decidual stromal cells support tolerance at the human foetal-maternal interface by inducing regulatory M2 macrophages and regulatory T-cells. Journal of reproductive immunology, 146103330. https://doi.org/10.1016/j.jri.2021.103330
Lindau, R, et al. "Decidual stromal cells support tolerance at the human foetal-maternal interface by inducing regulatory M2 macrophages and regulatory T-cells." Journal of reproductive immunology vol. 146 (2021): 103330. doi: https://doi.org/10.1016/j.jri.2021.103330
Lindau R, Vondra S, Spreckels J, Solders M, Svensson-Arvelund J, Berg G, Pollheimer J, Kaipe H, Jenmalm MC, Ernerudh J. Decidual stromal cells support tolerance at the human foetal-maternal interface by inducing regulatory M2 macrophages and regulatory T-cells. J Reprod Immunol. 2021 Aug;146:103330. doi: 10.1016/j.jri.2021.103330. Epub 2021 May 12. PMID: 34049032.
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Vitamin D modulates biliary fibrosis in ABCB4-deficient mice.

Hepatology international

Hochrath K, Stokes CS, Geisel J, Pollheimer MJ, Fickert P, Dooley S, Lammert F.
PMID: 25191532
Hepatol Int. 2014 Jun 21;8(3):443-52. doi: 10.1007/s12072-014-9548-2. eCollection 2014 Jul.

PURPOSE: Impaired vitamin D receptor signaling represents an aggravating factor during liver injury, and recent studies suggest that vitamin D might exert a protective role in chronic hepatobiliary diseases. We hypothesized that vitamin D supplementation would ameliorate liver fibrosis...

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Hochrath K, Stokes CS, Geisel J, et al. Vitamin D modulates biliary fibrosis in ABCB4-deficient mice. Hepatol Int. 2014;8(3):443-52doi: 10.1007/s12072-014-9548-2.
Hochrath, K., Stokes, C. S., Geisel, J., Pollheimer, M. J., Fickert, P., Dooley, S., & Lammert, F. (2014). Vitamin D modulates biliary fibrosis in ABCB4-deficient mice. Hepatology international, 8(3), 443-52. https://doi.org/10.1007/s12072-014-9548-2
Hochrath, Katrin, et al. "Vitamin D modulates biliary fibrosis in ABCB4-deficient mice." Hepatology international vol. 8,3 (2014): 443-52. doi: https://doi.org/10.1007/s12072-014-9548-2
Hochrath K, Stokes CS, Geisel J, Pollheimer MJ, Fickert P, Dooley S, Lammert F. Vitamin D modulates biliary fibrosis in ABCB4-deficient mice. Hepatol Int. 2014 Jun 21;8(3):443-52. doi: 10.1007/s12072-014-9548-2. eCollection 2014 Jul. PMID: 25191532; PMCID: PMC4148166.
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Human placental trophoblast invasion and differentiation: a particular focus on Wnt signaling.

Frontiers in genetics

Knöfler M, Pollheimer J.
PMID: 24133501
Front Genet. 2013 Sep 26;4:190. doi: 10.3389/fgene.2013.00190.

Wingless ligands, a family of secreted proteins, are critically involved in organ development and tissue homeostasis by ensuring balanced rates of stem cell proliferation, cell death and differentiation. Wnt signaling components also play crucial roles in murine placental development...

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Knöfler M, Pollheimer J. Human placental trophoblast invasion and differentiation: a particular focus on Wnt signaling. Front Genet. 2013;4:190doi: 10.3389/fgene.2013.00190.
Knöfler, M., & Pollheimer, J. (2013). Human placental trophoblast invasion and differentiation: a particular focus on Wnt signaling. Frontiers in genetics, 4190. https://doi.org/10.3389/fgene.2013.00190
Knöfler, Martin, and Pollheimer, Jürgen. "Human placental trophoblast invasion and differentiation: a particular focus on Wnt signaling." Frontiers in genetics vol. 4 (2013): 190. doi: https://doi.org/10.3389/fgene.2013.00190
Knöfler M, Pollheimer J. Human placental trophoblast invasion and differentiation: a particular focus on Wnt signaling. Front Genet. 2013 Sep 26;4:190. doi: 10.3389/fgene.2013.00190. PMID: 24133501; PMCID: PMC3783976.
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3-Dimensional JEG-3 choriocarcinoma cell organoids as a model for trophoblast expansion and differentiation.

Placenta

Dietrich B, Kunihs V, Haider S, Pollheimer J, Knöfler M.
PMID: 33461068
Placenta. 2021 Jan 15;104:243-246. doi: 10.1016/j.placenta.2020.12.013. Epub 2020 Dec 28.

JEG-3 choriocarcinoma cells have been widely used as a model for placental trophoblast. Herein, 3-dimensional (3D) JEG-3 organoids (JEG-3-ORGs) were established using a protocol that we recently developed for primary cytotrophoblast organoids (CTB-ORGs). 3D JEG-3-ORGs, cultivated in basic culture...

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Dietrich B, Kunihs V, Haider S, et al. 3-Dimensional JEG-3 choriocarcinoma cell organoids as a model for trophoblast expansion and differentiation. Placenta. 2020;104:243-246doi: 10.1016/j.placenta.2020.12.013.
Dietrich, B., Kunihs, V., Haider, S., Pollheimer, J., & Knöfler, M. (2021). 3-Dimensional JEG-3 choriocarcinoma cell organoids as a model for trophoblast expansion and differentiation. Placenta, 104243-246. https://doi.org/10.1016/j.placenta.2020.12.013
Dietrich, B, et al. "3-Dimensional JEG-3 choriocarcinoma cell organoids as a model for trophoblast expansion and differentiation." Placenta vol. 104 (2021): 243-246. doi: https://doi.org/10.1016/j.placenta.2020.12.013
Dietrich B, Kunihs V, Haider S, Pollheimer J, Knöfler M. 3-Dimensional JEG-3 choriocarcinoma cell organoids as a model for trophoblast expansion and differentiation. Placenta. 2021 Jan 15;104:243-246. doi: 10.1016/j.placenta.2020.12.013. Epub 2020 Dec 28. PMID: 33461068.
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Graft Growth and Podocyte Dedifferentiation in Donor-Recipient Size Mismatch Kidney Transplants.

Transplantation direct

Müller-Deile J, Bräsen JH, Pollheimer M, Ratschek M, Haller H, Pape L, Schiffer M.
PMID: 29026873
Transplant Direct. 2017 Sep 05;3(10):e210. doi: 10.1097/TXD.0000000000000728. eCollection 2017 Oct.

BACKGROUND: Kidney transplantation is the treatment choice for patients with end-stage renal diseases. Because of good long-term outcome, pediatric kidney grafts are also accepted for transplantation in adult recipients despite a significant mismatch in body size and age between...

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Müller-Deile J, Bräsen JH, Pollheimer M, et al. Graft Growth and Podocyte Dedifferentiation in Donor-Recipient Size Mismatch Kidney Transplants. Transplant Direct. 2017;3(10):e210doi: 10.1097/TXD.0000000000000728.
Müller-Deile, J., Bräsen, J. H., Pollheimer, M., Ratschek, M., Haller, H., Pape, L., & Schiffer, M. (2017). Graft Growth and Podocyte Dedifferentiation in Donor-Recipient Size Mismatch Kidney Transplants. Transplantation direct, 3(10), e210. https://doi.org/10.1097/TXD.0000000000000728
Müller-Deile, Janina, et al. "Graft Growth and Podocyte Dedifferentiation in Donor-Recipient Size Mismatch Kidney Transplants." Transplantation direct vol. 3,10 (2017): e210. doi: https://doi.org/10.1097/TXD.0000000000000728
Müller-Deile J, Bräsen JH, Pollheimer M, Ratschek M, Haller H, Pape L, Schiffer M. Graft Growth and Podocyte Dedifferentiation in Donor-Recipient Size Mismatch Kidney Transplants. Transplant Direct. 2017 Sep 05;3(10):e210. doi: 10.1097/TXD.0000000000000728. eCollection 2017 Oct. PMID: 29026873; PMCID: PMC5627741.
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Downgrading of a G3 Neuroendocrine Tumor to a G2 Tumor: Can First-Line Cytotoxic Chemotherapy Change the Tumor Biology?.

Case reports in oncology

Blesl A, Krones E, Pollheimer MJ, Haybaeck J, Wiesspeiner U, Lipp RW, Kump P.
PMID: 29430237
Case Rep Oncol. 2017 Dec 20;10(3):1121-1126. doi: 10.1159/000484473. eCollection 2017.

The antiproliferative treatment options for neuroendocrine tumors (NET)/neuroendocrine carcinomas of the gastrointestinal tract critically depend on the proliferation rate, evaluated by immunohistochemical staining for Ki-67. According to their grading, tumors are treated with somatostatin analogs, mTOR inhibitors, or cytotoxic...

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Blesl A, Krones E, Pollheimer MJ, et al. Downgrading of a G3 Neuroendocrine Tumor to a G2 Tumor: Can First-Line Cytotoxic Chemotherapy Change the Tumor Biology?. Case Rep Oncol. 2017;10(3):1121-1126doi: 10.1159/000484473.
Blesl, A., Krones, E., Pollheimer, M. J., Haybaeck, J., Wiesspeiner, U., Lipp, R. W., & Kump, P. (2017). Downgrading of a G3 Neuroendocrine Tumor to a G2 Tumor: Can First-Line Cytotoxic Chemotherapy Change the Tumor Biology?. Case reports in oncology, 10(3), 1121-1126. https://doi.org/10.1159/000484473
Blesl, Andreas, et al. "Downgrading of a G3 Neuroendocrine Tumor to a G2 Tumor: Can First-Line Cytotoxic Chemotherapy Change the Tumor Biology?." Case reports in oncology vol. 10,3 (2017): 1121-1126. doi: https://doi.org/10.1159/000484473
Blesl A, Krones E, Pollheimer MJ, Haybaeck J, Wiesspeiner U, Lipp RW, Kump P. Downgrading of a G3 Neuroendocrine Tumor to a G2 Tumor: Can First-Line Cytotoxic Chemotherapy Change the Tumor Biology?. Case Rep Oncol. 2017 Dec 20;10(3):1121-1126. doi: 10.1159/000484473. eCollection 2017. PMID: 29430237; PMCID: PMC5803685.
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[No title available]

RoFo : Fortschritte auf dem Gebiete der Rontgenstrahlen und der Nuklearmedizin

Adelsmayr G, Pollheimer M, Pfeifer J.
PMID: 34933354
Rofo. 2021 Dec 21; doi: 10.1055/a-1712-4529. Epub 2021 Dec 21.

No abstract available.

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Adelsmayr G, Pollheimer M, Pfeifer J. Invagination und Bezoar: außergewöhnliche Komplikationen einer Gastrojejunostomie-Sonde bei einer Patientin mit Morbus Parkinson. . Rofo. 2021;doi: 10.1055/a-1712-4529.
Adelsmayr, G., Pollheimer, M., & Pfeifer, J. (2021). Invagination und Bezoar: außergewöhnliche Komplikationen einer Gastrojejunostomie-Sonde bei einer Patientin mit Morbus Parkinson. . RoFo : Fortschritte auf dem Gebiete der Rontgenstrahlen und der Nuklearmedizin, . https://doi.org/10.1055/a-1712-4529
Adelsmayr, Gabriel, et al. "Invagination und Bezoar: außergewöhnliche Komplikationen einer Gastrojejunostomie-Sonde bei einer Patientin mit Morbus Parkinson." . RoFo : Fortschritte auf dem Gebiete der Rontgenstrahlen und der Nuklearmedizin vol. (2021). doi: https://doi.org/10.1055/a-1712-4529
Adelsmayr G, Pollheimer M, Pfeifer J. Invagination und Bezoar: außergewöhnliche Komplikationen einer Gastrojejunostomie-Sonde bei einer Patientin mit Morbus Parkinson. . Rofo. 2021 Dec 21; doi: 10.1055/a-1712-4529. Epub 2021 Dec 21. German. PMID: 34933354.
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