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Enhancement of methacholine-evoked tracheal contraction induced by bacterial lipopolysaccharides depends on epithelium and tumor necrosis factor.

Journal of allergy

Secher T, Rodrigues Coelho F, Noulin N, Lino Dos Santos Franco A, Quesniaux V, Lignon J, Mitchell J, Moser R, Gomes E, Mirotti L, Tavares-de-Lima W, Ryffel B, Vargaftig BB, Russo M.
PMID: 22481960
J Allergy (Cairo). 2012;2012:494085. doi: 10.1155/2012/494085. Epub 2012 Mar 11.

Inhaled bacterial lipopolysaccharides (LPSs) induce an acute tumour necrosis factor-alpha (TNF-α-) dependent inflammatory response in the murine airways mediated by Toll-like receptor 4 (TLR4) via the myeloid differentiation MyD88 adaptor protein pathway. However, the contractile response of the bronchial...

Cite
Secher T, Rodrigues Coelho F, Noulin N, et al. Enhancement of methacholine-evoked tracheal contraction induced by bacterial lipopolysaccharides depends on epithelium and tumor necrosis factor. J Allergy (Cairo). 2012;2012:494085doi: 10.1155/2012/494085.
Secher, T., Rodrigues Coelho, F., Noulin, N., Lino Dos Santos Franco, A., Quesniaux, V., Lignon, J., Mitchell, J., Moser, R., Gomes, E., Mirotti, L., Tavares-de-Lima, W., Ryffel, B., Vargaftig, B. B., & Russo, M. (2012). Enhancement of methacholine-evoked tracheal contraction induced by bacterial lipopolysaccharides depends on epithelium and tumor necrosis factor. Journal of allergy, 2012494085. https://doi.org/10.1155/2012/494085
Secher, T, et al. "Enhancement of methacholine-evoked tracheal contraction induced by bacterial lipopolysaccharides depends on epithelium and tumor necrosis factor." Journal of allergy vol. 2012 (2012): 494085. doi: https://doi.org/10.1155/2012/494085
Secher T, Rodrigues Coelho F, Noulin N, Lino Dos Santos Franco A, Quesniaux V, Lignon J, Mitchell J, Moser R, Gomes E, Mirotti L, Tavares-de-Lima W, Ryffel B, Vargaftig BB, Russo M. Enhancement of methacholine-evoked tracheal contraction induced by bacterial lipopolysaccharides depends on epithelium and tumor necrosis factor. J Allergy (Cairo). 2012;2012:494085. doi: 10.1155/2012/494085. Epub 2012 Mar 11. PMID: 22481960; PMCID: PMC3312294.
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Tumor necrosis factor neutralization combined with chemotherapy enhances Mycobacterium tuberculosis clearance and reduces lung pathology.

American journal of clinical and experimental immunology

Bourigault ML, Vacher R, Rose S, Olleros ML, Janssens JP, Quesniaux VF, Garcia I.
PMID: 23885330
Am J Clin Exp Immunol. 2013 Feb 27;2(1):124-34. Print 2013.

Tuberculosis (TB) is a major health problem requiring sustained immunity to inhibit Mycobacterium tuberculosis growth and appropriate antimicrobial therapy to prevent dissemination and drug resistance. Cell-mediated immune responses to M. tuberculosis involve the activation of cytokines such as Tumor...

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Bourigault ML, Vacher R, Rose S, et al. Tumor necrosis factor neutralization combined with chemotherapy enhances Mycobacterium tuberculosis clearance and reduces lung pathology. Am J Clin Exp Immunol. 2013;2(1):124-34
Bourigault, M. L., Vacher, R., Rose, S., Olleros, M. L., Janssens, J. P., Quesniaux, V. F., & Garcia, I. (2013). Tumor necrosis factor neutralization combined with chemotherapy enhances Mycobacterium tuberculosis clearance and reduces lung pathology. American journal of clinical and experimental immunology, 2(1), 124-34.
Bourigault, Marie-Laure, et al. "Tumor necrosis factor neutralization combined with chemotherapy enhances Mycobacterium tuberculosis clearance and reduces lung pathology." American journal of clinical and experimental immunology vol. 2,1 (2013): 124-34.
Bourigault ML, Vacher R, Rose S, Olleros ML, Janssens JP, Quesniaux VF, Garcia I. Tumor necrosis factor neutralization combined with chemotherapy enhances Mycobacterium tuberculosis clearance and reduces lung pathology. Am J Clin Exp Immunol. 2013 Feb 27;2(1):124-34. Print 2013. PMID: 23885330; PMCID: PMC3714199.
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Liver Immune Cells Release Type 1 Interferon Due to DNA Sensing and Amplify Liver Injury from Acetaminophen Overdose.

Cells

Araujo AM, Antunes MM, Mattos MS, Diniz AB, Alvarenga DM, Nakagaki BN, Carvalho É, Lacerda VAS, Carvalho-Gontijo R, Goulart J, Mafra K, Freitas-Lopes MA, Oliveira HMC, Dutra CM, David BA, Mendes Silva A, Quesniaux V, Ryffel B, Oliveira SC, Barber GN, Mansur DS, Cunha TM, Rezende RM, Oliveira AG, Menezes GB.
PMID: 30060463
Cells. 2018 Jul 27;7(8). doi: 10.3390/cells7080088.

Hepatocytes may rupture after a drug overdose, and their intracellular contents act as damage-associated molecular patterns (DAMPs) that lead to additional leukocyte infiltration, amplifying the original injury. Necrosis-derived DNA can be recognized as a DAMP, activating liver non-parenchymal cells...

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Araujo AM, Antunes MM, Mattos MS, et al. Liver Immune Cells Release Type 1 Interferon Due to DNA Sensing and Amplify Liver Injury from Acetaminophen Overdose. Cells. 2018;7(8)doi: 10.3390/cells7080088.
Araujo, A. M., Antunes, M. M., Mattos, M. S., Diniz, A. B., Alvarenga, D. M., Nakagaki, B. N., Carvalho, �. �., Lacerda, V. A. S., Carvalho-Gontijo, R., Goulart, J., Mafra, K., Freitas-Lopes, M. A., Oliveira, H. M. C., Dutra, C. M., David, B. A., Mendes Silva, A., Quesniaux, V., Ryffel, B., Oliveira, S. C., Barber, G. N., Mansur, D. S., Cunha, T. M., Rezende, R. M., Oliveira, A. G., & Menezes, G. B. (2018). Liver Immune Cells Release Type 1 Interferon Due to DNA Sensing and Amplify Liver Injury from Acetaminophen Overdose. Cells, 7(8), . https://doi.org/10.3390/cells7080088
Araujo, Alan Moreira de, et al. "Liver Immune Cells Release Type 1 Interferon Due to DNA Sensing and Amplify Liver Injury from Acetaminophen Overdose." Cells vol. 7,8 (2018). doi: https://doi.org/10.3390/cells7080088
Araujo AM, Antunes MM, Mattos MS, Diniz AB, Alvarenga DM, Nakagaki BN, Carvalho É, Lacerda VAS, Carvalho-Gontijo R, Goulart J, Mafra K, Freitas-Lopes MA, Oliveira HMC, Dutra CM, David BA, Mendes Silva A, Quesniaux V, Ryffel B, Oliveira SC, Barber GN, Mansur DS, Cunha TM, Rezende RM, Oliveira AG, Menezes GB. Liver Immune Cells Release Type 1 Interferon Due to DNA Sensing and Amplify Liver Injury from Acetaminophen Overdose. Cells. 2018 Jul 27;7(8). doi: 10.3390/cells7080088. PMID: 30060463; PMCID: PMC6115735.
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Myeloid and T Cell-Derived TNF Protects against Central Nervous System Tuberculosis.

Frontiers in immunology

Hsu NJ, Francisco NM, Keeton R, Allie N, Quesniaux VF, Ryffel B, Jacobs M.
PMID: 28280495
Front Immunol. 2017 Feb 23;8:180. doi: 10.3389/fimmu.2017.00180. eCollection 2017.

Tuberculosis of the central nervous system (CNS-TB) is a devastating complication of tuberculosis, and tumor necrosis factor (TNF) is crucial for innate immunity and controlling the infection. TNF is produced by many cell types upon activation, in particularly the...

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Hsu NJ, Francisco NM, Keeton R, et al. Myeloid and T Cell-Derived TNF Protects against Central Nervous System Tuberculosis. Front Immunol. 2017;8:180doi: 10.3389/fimmu.2017.00180.
Hsu, N. J., Francisco, N. M., Keeton, R., Allie, N., Quesniaux, V. F., Ryffel, B., & Jacobs, M. (2017). Myeloid and T Cell-Derived TNF Protects against Central Nervous System Tuberculosis. Frontiers in immunology, 8180. https://doi.org/10.3389/fimmu.2017.00180
Hsu, Nai-Jen, et al. "Myeloid and T Cell-Derived TNF Protects against Central Nervous System Tuberculosis." Frontiers in immunology vol. 8 (2017): 180. doi: https://doi.org/10.3389/fimmu.2017.00180
Hsu NJ, Francisco NM, Keeton R, Allie N, Quesniaux VF, Ryffel B, Jacobs M. Myeloid and T Cell-Derived TNF Protects against Central Nervous System Tuberculosis. Front Immunol. 2017 Feb 23;8:180. doi: 10.3389/fimmu.2017.00180. eCollection 2017. PMID: 28280495; PMCID: PMC5322283.
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Blockade of IL-1R signaling diminishes Paneth cell depletion and Toxoplasma gondii induced ileitis in mice.

American journal of clinical and experimental immunology

Villeret B, Brault L, Couturier-Maillard A, Robinet P, Vasseur V, Secher T, Dimier-Poisson I, Jacobs M, Zheng SG, Quesniaux VF, Ryffel B.
PMID: 23885328
Am J Clin Exp Immunol. 2013 Feb 27;2(1):107-16. Print 2013.

Interleukin 1 is a critical inflammatory mediator and involved in host defense to several pathogens. Oral T. gondii infection causes lethal ileitis in C57BL/6 (BL6) mice and serves to investigate the mechanisms of acute intestinal inflammation. Here we show...

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Villeret B, Brault L, Couturier-Maillard A, et al. Blockade of IL-1R signaling diminishes Paneth cell depletion and Toxoplasma gondii induced ileitis in mice. Am J Clin Exp Immunol. 2013;2(1):107-16
Villeret, B., Brault, L., Couturier-Maillard, A., Robinet, P., Vasseur, V., Secher, T., Dimier-Poisson, I., Jacobs, M., Zheng, S. G., Quesniaux, V. F., & Ryffel, B. (2013). Blockade of IL-1R signaling diminishes Paneth cell depletion and Toxoplasma gondii induced ileitis in mice. American journal of clinical and experimental immunology, 2(1), 107-16.
Villeret, Bérengère, et al. "Blockade of IL-1R signaling diminishes Paneth cell depletion and Toxoplasma gondii induced ileitis in mice." American journal of clinical and experimental immunology vol. 2,1 (2013): 107-16.
Villeret B, Brault L, Couturier-Maillard A, Robinet P, Vasseur V, Secher T, Dimier-Poisson I, Jacobs M, Zheng SG, Quesniaux VF, Ryffel B. Blockade of IL-1R signaling diminishes Paneth cell depletion and Toxoplasma gondii induced ileitis in mice. Am J Clin Exp Immunol. 2013 Feb 27;2(1):107-16. Print 2013. PMID: 23885328; PMCID: PMC3714202.
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Enhancement of methacholine-evoked tracheal contraction induced by bacterial lipopolysaccharides depends on epithelium and tumor necrosis factor.

Journal of allergy

Secher T, Rodrigues Coelho F, Noulin N, Lino Dos Santos Franco A, Quesniaux V, Lignon J, Mitchell J, Moser R, Gomes E, Mirotti L, Tavares-de-Lima W, Ryffel B, Vargaftig BB, Russo M.
PMID: 22481960
J Allergy (Cairo). 2012;2012:494085. doi: 10.1155/2012/494085. Epub 2012 Mar 11.

Inhaled bacterial lipopolysaccharides (LPSs) induce an acute tumour necrosis factor-alpha (TNF-α-) dependent inflammatory response in the murine airways mediated by Toll-like receptor 4 (TLR4) via the myeloid differentiation MyD88 adaptor protein pathway. However, the contractile response of the bronchial...

Cite
Secher T, Rodrigues Coelho F, Noulin N, et al. Enhancement of methacholine-evoked tracheal contraction induced by bacterial lipopolysaccharides depends on epithelium and tumor necrosis factor. J Allergy (Cairo). 2012;2012:494085doi: 10.1155/2012/494085.
Secher, T., Rodrigues Coelho, F., Noulin, N., Lino Dos Santos Franco, A., Quesniaux, V., Lignon, J., Mitchell, J., Moser, R., Gomes, E., Mirotti, L., Tavares-de-Lima, W., Ryffel, B., Vargaftig, B. B., & Russo, M. (2012). Enhancement of methacholine-evoked tracheal contraction induced by bacterial lipopolysaccharides depends on epithelium and tumor necrosis factor. Journal of allergy, 2012494085. https://doi.org/10.1155/2012/494085
Secher, T, et al. "Enhancement of methacholine-evoked tracheal contraction induced by bacterial lipopolysaccharides depends on epithelium and tumor necrosis factor." Journal of allergy vol. 2012 (2012): 494085. doi: https://doi.org/10.1155/2012/494085
Secher T, Rodrigues Coelho F, Noulin N, Lino Dos Santos Franco A, Quesniaux V, Lignon J, Mitchell J, Moser R, Gomes E, Mirotti L, Tavares-de-Lima W, Ryffel B, Vargaftig BB, Russo M. Enhancement of methacholine-evoked tracheal contraction induced by bacterial lipopolysaccharides depends on epithelium and tumor necrosis factor. J Allergy (Cairo). 2012;2012:494085. doi: 10.1155/2012/494085. Epub 2012 Mar 11. PMID: 22481960; PMCID: PMC3312294.
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Inflammasome, IL-1 and inflammation in ozone-induced lung injury.

American journal of clinical and experimental immunology

Michaudel C, Couturier-Maillard A, Chenuet P, Maillet I, Mura C, Couillin I, Gombault A, Quesniaux VF, Huaux F, Ryffel B.
PMID: 27168953
Am J Clin Exp Immunol. 2016 Mar 23;5(1):33-40. eCollection 2016.

Exposure to ambient ozone causes airway hyperreactivity and lung inflammation, which represent an important health concern in humans. Recent clinical and experimental studies contributed to the understanding of the mechanisms of epithelial injury, inflammation and airway hyperreactivity, which is...

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Michaudel C, Couturier-Maillard A, Chenuet P, et al. Inflammasome, IL-1 and inflammation in ozone-induced lung injury. Am J Clin Exp Immunol. 2016;5(1):33-40
Michaudel, C., Couturier-Maillard, A., Chenuet, P., Maillet, I., Mura, C., Couillin, I., Gombault, A., Quesniaux, V. F., Huaux, F., & Ryffel, B. (2016). Inflammasome, IL-1 and inflammation in ozone-induced lung injury. American journal of clinical and experimental immunology, 5(1), 33-40.
Michaudel, Chloé, et al. "Inflammasome, IL-1 and inflammation in ozone-induced lung injury." American journal of clinical and experimental immunology vol. 5,1 (2016): 33-40.
Michaudel C, Couturier-Maillard A, Chenuet P, Maillet I, Mura C, Couillin I, Gombault A, Quesniaux VF, Huaux F, Ryffel B. Inflammasome, IL-1 and inflammation in ozone-induced lung injury. Am J Clin Exp Immunol. 2016 Mar 23;5(1):33-40. eCollection 2016. PMID: 27168953; PMCID: PMC4858604.
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Correction: Interleukin-22-deficiency and microbiota contribute to the exacerbation of Toxoplasma gondii-induced intestinal inflammation.

Mucosal immunology

Couturier-Maillard A, Froux N, Piotet-Morin J, Michaudel C, Brault L, Le Bérichel J, Sénéchal A, Robinet P, Chenuet P, Jejou S, Dumoutier L, Renauld JC, Iovanna J, Huber S, Chamaillard M, Quesniaux VFJ, Sokol H, Ryffel B.
PMID: 30504789
Mucosal Immunol. 2019 Jan;12(1):290. doi: 10.1038/s41385-018-0112-6.

The original version of this Article omitted the author Dr Mathias Chamaillard from the l'Institut de Pasteur, Lille, France. This has been corrected in both the PDF and HTML versions of the Article.

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Couturier-Maillard A, Froux N, Piotet-Morin J, et al. Correction: Interleukin-22-deficiency and microbiota contribute to the exacerbation of Toxoplasma gondii-induced intestinal inflammation. Mucosal Immunol. 2019;12(1):290doi: 10.1038/s41385-018-0112-6.
Couturier-Maillard, A., Froux, N., Piotet-Morin, J., Michaudel, C., Brault, L., Le Bérichel, J., Sénéchal, A., Robinet, P., Chenuet, P., Jejou, S., Dumoutier, L., Renauld, J. C., Iovanna, J., Huber, S., Chamaillard, M., Quesniaux, V. F. J., Sokol, H., & Ryffel, B. (2019). Correction: Interleukin-22-deficiency and microbiota contribute to the exacerbation of Toxoplasma gondii-induced intestinal inflammation. Mucosal immunology, 12(1), 290. https://doi.org/10.1038/s41385-018-0112-6
Couturier-Maillard, A, et al. "Correction: Interleukin-22-deficiency and microbiota contribute to the exacerbation of Toxoplasma gondii-induced intestinal inflammation." Mucosal immunology vol. 12,1 (2019): 290. doi: https://doi.org/10.1038/s41385-018-0112-6
Couturier-Maillard A, Froux N, Piotet-Morin J, Michaudel C, Brault L, Le Bérichel J, Sénéchal A, Robinet P, Chenuet P, Jejou S, Dumoutier L, Renauld JC, Iovanna J, Huber S, Chamaillard M, Quesniaux VFJ, Sokol H, Ryffel B. Correction: Interleukin-22-deficiency and microbiota contribute to the exacerbation of Toxoplasma gondii-induced intestinal inflammation. Mucosal Immunol. 2019 Jan;12(1):290. doi: 10.1038/s41385-018-0112-6. PMID: 30504789.
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Perinatal Exposure to Glufosinate Ammonium Herbicide Impairs Neurogenesis and Neuroblast Migration through Cytoskeleton Destabilization.

Frontiers in cellular neuroscience

Herzine A, Laugeray A, Feat J, Menuet A, Quesniaux V, Richard O, Pichon J, Montécot-Dubourg C, Perche O, Mortaud S.
PMID: 27555806
Front Cell Neurosci. 2016 Aug 09;10:191. doi: 10.3389/fncel.2016.00191. eCollection 2016.

Neurogenesis, a process of generating functional neurons from neural precursors, occurs throughout life in restricted brain regions such as the subventricular zone (SVZ). During this process, newly generated neurons migrate along the rostral migratory stream to the olfactory bulb...

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Herzine A, Laugeray A, Feat J, et al. Perinatal Exposure to Glufosinate Ammonium Herbicide Impairs Neurogenesis and Neuroblast Migration through Cytoskeleton Destabilization. Front Cell Neurosci. 2016;10:191doi: 10.3389/fncel.2016.00191.
Herzine, A., Laugeray, A., Feat, J., Menuet, A., Quesniaux, V., Richard, O., Pichon, J., Montécot-Dubourg, C., Perche, O., & Mortaud, S. (2016). Perinatal Exposure to Glufosinate Ammonium Herbicide Impairs Neurogenesis and Neuroblast Migration through Cytoskeleton Destabilization. Frontiers in cellular neuroscience, 10191. https://doi.org/10.3389/fncel.2016.00191
Herzine, Ameziane, et al. "Perinatal Exposure to Glufosinate Ammonium Herbicide Impairs Neurogenesis and Neuroblast Migration through Cytoskeleton Destabilization." Frontiers in cellular neuroscience vol. 10 (2016): 191. doi: https://doi.org/10.3389/fncel.2016.00191
Herzine A, Laugeray A, Feat J, Menuet A, Quesniaux V, Richard O, Pichon J, Montécot-Dubourg C, Perche O, Mortaud S. Perinatal Exposure to Glufosinate Ammonium Herbicide Impairs Neurogenesis and Neuroblast Migration through Cytoskeleton Destabilization. Front Cell Neurosci. 2016 Aug 09;10:191. doi: 10.3389/fncel.2016.00191. eCollection 2016. PMID: 27555806; PMCID: PMC4977287.
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Tumor Necrosis Factor-Alpha Targeting Can Protect against Arthritis with Low Sensitization to Infection.

Frontiers in immunology

Belmellat N, Semerano L, Segueni N, Damotte D, Decker P, Ryffel B, Quesniaux V, Boissier MC, Assier E.
PMID: 29184553
Front Immunol. 2017 Nov 14;8:1533. doi: 10.3389/fimmu.2017.01533. eCollection 2017.

Tumor necrosis factor-alpha (TNF-α) blockade is an effective treatment for rheumatoid arthritis (RA) and other inflammatory diseases, but in patients, it is associated with reduced resistance to the infectious agents

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Belmellat N, Semerano L, Segueni N, et al. Tumor Necrosis Factor-Alpha Targeting Can Protect against Arthritis with Low Sensitization to Infection. Front Immunol. 2017;8:1533doi: 10.3389/fimmu.2017.01533.
Belmellat, N., Semerano, L., Segueni, N., Damotte, D., Decker, P., Ryffel, B., Quesniaux, V., Boissier, M. C., & Assier, E. (2017). Tumor Necrosis Factor-Alpha Targeting Can Protect against Arthritis with Low Sensitization to Infection. Frontiers in immunology, 81533. https://doi.org/10.3389/fimmu.2017.01533
Belmellat, Nadia, et al. "Tumor Necrosis Factor-Alpha Targeting Can Protect against Arthritis with Low Sensitization to Infection." Frontiers in immunology vol. 8 (2017): 1533. doi: https://doi.org/10.3389/fimmu.2017.01533
Belmellat N, Semerano L, Segueni N, Damotte D, Decker P, Ryffel B, Quesniaux V, Boissier MC, Assier E. Tumor Necrosis Factor-Alpha Targeting Can Protect against Arthritis with Low Sensitization to Infection. Front Immunol. 2017 Nov 14;8:1533. doi: 10.3389/fimmu.2017.01533. eCollection 2017. PMID: 29184553; PMCID: PMC5694445.
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Transmembrane Tumor Necrosis Factor Controls Myeloid-Derived Suppressor Cell Activity .

Frontiers in immunology

Chavez-Galan L, Vesin D, Uysal H, Blaser G, Benkhoucha M, Ryffel B, Quesniaux VFJ, Garcia I.
PMID: 28890718
Front Immunol. 2017 Aug 25;8:999. doi: 10.3389/fimmu.2017.00999. eCollection 2017.

Pleural tuberculosis (TB) is a form of extra-pulmonary TB observed in patients infected with

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Chavez-Galan L, Vesin D, Uysal H, et al. Transmembrane Tumor Necrosis Factor Controls Myeloid-Derived Suppressor Cell Activity . Front Immunol. 2017;8:999doi: 10.3389/fimmu.2017.00999.
Chavez-Galan, L., Vesin, D., Uysal, H., Blaser, G., Benkhoucha, M., Ryffel, B., Quesniaux, V. F. J., & Garcia, I. (2017). Transmembrane Tumor Necrosis Factor Controls Myeloid-Derived Suppressor Cell Activity . Frontiers in immunology, 8999. https://doi.org/10.3389/fimmu.2017.00999
Chavez-Galan, Leslie, et al. "Transmembrane Tumor Necrosis Factor Controls Myeloid-Derived Suppressor Cell Activity ." Frontiers in immunology vol. 8 (2017): 999. doi: https://doi.org/10.3389/fimmu.2017.00999
Chavez-Galan L, Vesin D, Uysal H, Blaser G, Benkhoucha M, Ryffel B, Quesniaux VFJ, Garcia I. Transmembrane Tumor Necrosis Factor Controls Myeloid-Derived Suppressor Cell Activity . Front Immunol. 2017 Aug 25;8:999. doi: 10.3389/fimmu.2017.00999. eCollection 2017. PMID: 28890718; PMCID: PMC5574880.
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Chronic .

ImmunoHorizons

Mackowiak C, Marchiol T, Paljetak HC, Fauconnier L, Palomo J, Secher T, Panek C, Sedda D, Savigny F, Erard F, Bragonzi A, Huaux F, Stoeger T, Schiller HB, Sirard JC, Le Bert M, Couillin I, Quesniaux VFJ, Togbe D, Ryffel B.
PMID: 33958388
Immunohorizons. 2021 May 06;5(5):273-283. doi: 10.4049/immunohorizons.2000095.

Cystic fibrosis is associated with chronic

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Mackowiak C, Marchiol T, Paljetak HC, et al. Chronic . Immunohorizons. 2021;5(5):273-283doi: 10.4049/immunohorizons.2000095.
Mackowiak, C., Marchiol, T., Paljetak, H. C., Fauconnier, L., Palomo, J., Secher, T., Panek, C., Sedda, D., Savigny, F., Erard, F., Bragonzi, A., Huaux, F., Stoeger, T., Schiller, H. B., Sirard, J. C., Le Bert, M., Couillin, I., Quesniaux, V. F. J., Togbe, D., & Ryffel, B. (2021). Chronic . ImmunoHorizons, 5(5), 273-283. https://doi.org/10.4049/immunohorizons.2000095
Mackowiak, Claire, et al. "Chronic ." ImmunoHorizons vol. 5,5 (2021): 273-283. doi: https://doi.org/10.4049/immunohorizons.2000095
Mackowiak C, Marchiol T, Paljetak HC, Fauconnier L, Palomo J, Secher T, Panek C, Sedda D, Savigny F, Erard F, Bragonzi A, Huaux F, Stoeger T, Schiller HB, Sirard JC, Le Bert M, Couillin I, Quesniaux VFJ, Togbe D, Ryffel B. Chronic . Immunohorizons. 2021 May 06;5(5):273-283. doi: 10.4049/immunohorizons.2000095. PMID: 33958388.
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