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Distribution and Load of Amyloid-β Pathology in Parkinson Disease and Dementia with Lewy Bodies.

Journal of neuropathology and experimental neurology

Hepp DH, Vergoossen DL, Huisman E, Lemstra AW, Berendse HW, Rozemuller AJ, Foncke EM, van de Berg WD.
PMID: 27516115
J Neuropathol Exp Neurol. 2016 Oct;75(10):936-945. doi: 10.1093/jnen/nlw070. Epub 2016 Aug 11.

Parkinson disease (PD), Parkinson disease with dementia (PDD), and Dementia with Lewy bodies (DLB) differ clinically with regard to the presence and timing of dementia. In this postmortem study, we evaluated whether the burden and distribution pattern of amyloid-β...

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Hepp DH, Vergoossen DL, Huisman E, et al. Distribution and Load of Amyloid-β Pathology in Parkinson Disease and Dementia with Lewy Bodies. J Neuropathol Exp Neurol. 2016;75(10):936-945doi: 10.1093/jnen/nlw070.
Hepp, D. H., Vergoossen, D. L., Huisman, E., Lemstra, A. W., Berendse, H. W., Rozemuller, A. J., Foncke, E. M., & van de Berg, W. D. (2016). Distribution and Load of Amyloid-β Pathology in Parkinson Disease and Dementia with Lewy Bodies. Journal of neuropathology and experimental neurology, 75(10), 936-945. https://doi.org/10.1093/jnen/nlw070
Hepp, Dagmar H, et al. "Distribution and Load of Amyloid-β Pathology in Parkinson Disease and Dementia with Lewy Bodies." Journal of neuropathology and experimental neurology vol. 75,10 (2016): 936-945. doi: https://doi.org/10.1093/jnen/nlw070
Hepp DH, Vergoossen DL, Huisman E, Lemstra AW, Berendse HW, Rozemuller AJ, Foncke EM, van de Berg WD. Distribution and Load of Amyloid-β Pathology in Parkinson Disease and Dementia with Lewy Bodies. J Neuropathol Exp Neurol. 2016 Oct;75(10):936-945. doi: 10.1093/jnen/nlw070. Epub 2016 Aug 11. PMID: 27516115.
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Right temporal variant frontotemporal dementia is pathologically heterogeneous: a case-series and a systematic review.

Acta neuropathologica communications

Ulugut H, Dijkstra AA, Scarioni M, Barkhof F, Scheltens P, Rozemuller AJM, Pijnenburg YAL.
PMID: 34344452
Acta Neuropathol Commun. 2021 Aug 03;9(1):131. doi: 10.1186/s40478-021-01229-z.

Although the right temporal variant frontotemporal dementia (rtvFTD) is characterised by distinct clinical and radiological features, its underlying histopathology remains elusive. Being considered a right-sided variant of semantic variant primary progressive aphasia (svPPA), TDP-43 type C pathology has been...

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Ulugut H, Dijkstra AA, Scarioni M. Right temporal variant frontotemporal dementia is pathologically heterogeneous: a case-series and a systematic review. Acta Neuropathol Commun. 2021;9(1):131doi: 10.1186/s40478-021-01229-z.
Ulugut, H., Dijkstra, A. A., Scarioni, M., Barkhof, F., Scheltens, P., Rozemuller, A. J. M., & Pijnenburg, Y. A. L. (2021). Right temporal variant frontotemporal dementia is pathologically heterogeneous: a case-series and a systematic review. Acta neuropathologica communications, 9(1), 131. https://doi.org/10.1186/s40478-021-01229-z
Ulugut, Hulya, et al. "Right temporal variant frontotemporal dementia is pathologically heterogeneous: a case-series and a systematic review." Acta neuropathologica communications vol. 9,1 (2021): 131. doi: https://doi.org/10.1186/s40478-021-01229-z
Ulugut H, Dijkstra AA, Scarioni M, Barkhof F, Scheltens P, Rozemuller AJM, Pijnenburg YAL. Right temporal variant frontotemporal dementia is pathologically heterogeneous: a case-series and a systematic review. Acta Neuropathol Commun. 2021 Aug 03;9(1):131. doi: 10.1186/s40478-021-01229-z. PMID: 34344452; PMCID: PMC8330072.
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Erratum to: Neuropathological assessments of the pathology in frontotemporal lobar degeneration with TDP43-positive inclusions: an inter-laboratory study by the BrainNet Europe consortium.

Journal of neural transmission (Vienna, Austria : 1996)

Alafuzoff I, Pikkarainen M, Neumann M, Arzberger T, Al-Sarraj S, Bodi I, Bogdanovic N, Bugiani O, Ferrer I, Gelpi E, Gentleman S, Giaccone G, Graeber MB, Hortobagyi T, Ince PG, Ironside JW, Kavantzas N, King A, Korkolopoulou P, Kovács GG, Meyronet D, Monoranu C, Nilsson T, Parchi P, Patsouris E, Revesz T, Roggendorf W, Rozemuller A, Seilhean D, Streichenberger N, Thal DR, Wharton SB, Kretzschmar H.
PMID: 25418279
J Neural Transm (Vienna). 2015 Jul;122(7):973-4. doi: 10.1007/s00702-014-1337-5.

No abstract available.

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Alafuzoff I, Pikkarainen M, Neumann M, et al. Erratum to: Neuropathological assessments of the pathology in frontotemporal lobar degeneration with TDP43-positive inclusions: an inter-laboratory study by the BrainNet Europe consortium. J Neural Transm (Vienna). 2015;122(7):973-4doi: 10.1007/s00702-014-1337-5.
Alafuzoff, I., Pikkarainen, M., Neumann, M., Arzberger, T., Al-Sarraj, S., Bodi, I., Bogdanovic, N., Bugiani, O., Ferrer, I., Gelpi, E., Gentleman, S., Giaccone, G., Graeber, M. B., Hortobagyi, T., Ince, P. G., Ironside, J. W., Kavantzas, N., King, A., Korkolopoulou, P., Kovács, G. G., Meyronet, D., Monoranu, C., Nilsson, T., Parchi, P., Patsouris, E., Revesz, T., Roggendorf, W., Rozemuller, A., Seilhean, D., Streichenberger, N., Thal, D. R., Wharton, S. B., & Kretzschmar, H. (2015). Erratum to: Neuropathological assessments of the pathology in frontotemporal lobar degeneration with TDP43-positive inclusions: an inter-laboratory study by the BrainNet Europe consortium. Journal of neural transmission (Vienna, Austria : 1996), 122(7), 973-4. https://doi.org/10.1007/s00702-014-1337-5
Alafuzoff, Irina, et al. "Erratum to: Neuropathological assessments of the pathology in frontotemporal lobar degeneration with TDP43-positive inclusions: an inter-laboratory study by the BrainNet Europe consortium." Journal of neural transmission (Vienna, Austria : 1996) vol. 122,7 (2015): 973-4. doi: https://doi.org/10.1007/s00702-014-1337-5
Alafuzoff I, Pikkarainen M, Neumann M, Arzberger T, Al-Sarraj S, Bodi I, Bogdanovic N, Bugiani O, Ferrer I, Gelpi E, Gentleman S, Giaccone G, Graeber MB, Hortobagyi T, Ince PG, Ironside JW, Kavantzas N, King A, Korkolopoulou P, Kovács GG, Meyronet D, Monoranu C, Nilsson T, Parchi P, Patsouris E, Revesz T, Roggendorf W, Rozemuller A, Seilhean D, Streichenberger N, Thal DR, Wharton SB, Kretzschmar H. Erratum to: Neuropathological assessments of the pathology in frontotemporal lobar degeneration with TDP43-positive inclusions: an inter-laboratory study by the BrainNet Europe consortium. J Neural Transm (Vienna). 2015 Jul;122(7):973-4. doi: 10.1007/s00702-014-1337-5. PMID: 25418279.
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MR1 encompasses at least six allele groups with coding region alterations.

HLA

Rozemuller E, Eckle SBG, McLaughlin I, Penning M, Mulder W, de Bruin H, van Wageningen S.
PMID: 34351076
HLA. 2021 Dec;98(6):509-516. doi: 10.1111/tan.14390. Epub 2021 Aug 23.

Unlike classical HLA class I genes, MR1 is assumed to have limited polymorphic positions. We developed a MR1 specific PCR assay and sequenced 56 DNA samples from cells with a diverse set of HLA genotypes. In this relatively small...

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Rozemuller E, Eckle SBG, McLaughlin I, et al. MR1 encompasses at least six allele groups with coding region alterations. HLA. 2021;98(6):509-516doi: 10.1111/tan.14390.
Rozemuller, E., Eckle, S. B. G., McLaughlin, I., Penning, M., Mulder, W., de Bruin, H., & van Wageningen, S. (2021). MR1 encompasses at least six allele groups with coding region alterations. HLA, 98(6), 509-516. https://doi.org/10.1111/tan.14390
Rozemuller, Erik, et al. "MR1 encompasses at least six allele groups with coding region alterations." HLA vol. 98,6 (2021): 509-516. doi: https://doi.org/10.1111/tan.14390
Rozemuller E, Eckle SBG, McLaughlin I, Penning M, Mulder W, de Bruin H, van Wageningen S. MR1 encompasses at least six allele groups with coding region alterations. HLA. 2021 Dec;98(6):509-516. doi: 10.1111/tan.14390. Epub 2021 Aug 23. PMID: 34351076.
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Whether, when and how chronic inflammation increases the risk of developing late-onset Alzheimer's disease.

Alzheimer's research & therapy

Eikelenboom P, Hoozemans JJ, Veerhuis R, van Exel E, Rozemuller AJ, van Gool WA.
PMID: 22647384
Alzheimers Res Ther. 2012 Jun 04;4(3):15. doi: 10.1186/alzrt118.

Neuropathological studies have revealed the presence of a broad variety of inflammation-related proteins (complement factors, acute-phase proteins, pro-inflammatory cytokines) in Alzheimer's disease (AD) brains. These constituents of innate immunity are involved in several crucial pathogenic events of the underlying...

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Eikelenboom P, Hoozemans JJ, Veerhuis R, et al. Whether, when and how chronic inflammation increases the risk of developing late-onset Alzheimer's disease. Alzheimers Res Ther. 2012;4(3):15doi: 10.1186/alzrt118.
Eikelenboom, P., Hoozemans, J. J., Veerhuis, R., van Exel, E., Rozemuller, A. J., & van Gool, W. A. (2012). Whether, when and how chronic inflammation increases the risk of developing late-onset Alzheimer's disease. Alzheimer's research & therapy, 4(3), 15. https://doi.org/10.1186/alzrt118
Eikelenboom, Piet, et al. "Whether, when and how chronic inflammation increases the risk of developing late-onset Alzheimer's disease." Alzheimer's research & therapy vol. 4,3 (2012): 15. doi: https://doi.org/10.1186/alzrt118
Eikelenboom P, Hoozemans JJ, Veerhuis R, van Exel E, Rozemuller AJ, van Gool WA. Whether, when and how chronic inflammation increases the risk of developing late-onset Alzheimer's disease. Alzheimers Res Ther. 2012 Jun 04;4(3):15. doi: 10.1186/alzrt118. PMID: 22647384; PMCID: PMC3506930.
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Distinctive pattern of temporal atrophy in patients with frontotemporal dementia and the I383V variant in .

Journal of neurology, neurosurgery, and psychiatry

Mol MO, Nijmeijer SWR, van Rooij JGJ, van Spaendonk RML, Pijnenburg YAL, van der Lee SJ, van Minkelen R, Donker Kaat L, Rozemuller AJM, Janse van Mantgem MR, van Rheenen W, van Es MA, Veldink JH, Hennekam FAM, Vernooij M, van Swieten JC, Cohn-Hokke PE, Seelaar H, Dopper EGP.
PMID: 33452055
J Neurol Neurosurg Psychiatry. 2021 Jul;92(7):787-789. doi: 10.1136/jnnp-2020-325150. Epub 2021 Jan 15.

No abstract available.

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Mol MO, Nijmeijer SWR, van Rooij JGJ, et al. Distinctive pattern of temporal atrophy in patients with frontotemporal dementia and the I383V variant in . J Neurol Neurosurg Psychiatry. 2021;92(7):787-789doi: 10.1136/jnnp-2020-325150.
Mol, M. O., Nijmeijer, S. W. R., van Rooij, J. G. J., van Spaendonk, R. M. L., Pijnenburg, Y. A. L., van der Lee, S. J., van Minkelen, R., Donker Kaat, L., Rozemuller, A. J. M., Janse van Mantgem, M. R., van Rheenen, W., van Es, M. A., Veldink, J. H., Hennekam, F. A. M., Vernooij, M., van Swieten, J. C., Cohn-Hokke, P. E., Seelaar, H., & Dopper, E. G. P. (2021). Distinctive pattern of temporal atrophy in patients with frontotemporal dementia and the I383V variant in . Journal of neurology, neurosurgery, and psychiatry, 92(7), 787-789. https://doi.org/10.1136/jnnp-2020-325150
Mol, Merel O, et al. "Distinctive pattern of temporal atrophy in patients with frontotemporal dementia and the I383V variant in ." Journal of neurology, neurosurgery, and psychiatry vol. 92,7 (2021): 787-789. doi: https://doi.org/10.1136/jnnp-2020-325150
Mol MO, Nijmeijer SWR, van Rooij JGJ, van Spaendonk RML, Pijnenburg YAL, van der Lee SJ, van Minkelen R, Donker Kaat L, Rozemuller AJM, Janse van Mantgem MR, van Rheenen W, van Es MA, Veldink JH, Hennekam FAM, Vernooij M, van Swieten JC, Cohn-Hokke PE, Seelaar H, Dopper EGP. Distinctive pattern of temporal atrophy in patients with frontotemporal dementia and the I383V variant in . J Neurol Neurosurg Psychiatry. 2021 Jul;92(7):787-789. doi: 10.1136/jnnp-2020-325150. Epub 2021 Jan 15. PMID: 33452055; PMCID: PMC8223666.
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Distinctive pattern of temporal atrophy in patients with frontotemporal dementia and the I383V variant in .

Journal of neurology, neurosurgery, and psychiatry

Mol MO, Nijmeijer SWR, van Rooij JGJ, van Spaendonk RML, Pijnenburg YAL, van der Lee SJ, van Minkelen R, Donker Kaat L, Rozemuller AJM, Janse van Mantgem MR, van Rheenen W, van Es MA, Veldink JH, Hennekam FAM, Vernooij M, van Swieten JC, Cohn-Hokke PE, Seelaar H, Dopper EGP.
PMID: 33452055
J Neurol Neurosurg Psychiatry. 2021 Jul;92(7):787-789. doi: 10.1136/jnnp-2020-325150. Epub 2021 Jan 15.

No abstract available.

Cite
Mol MO, Nijmeijer SWR, van Rooij JGJ, et al. Distinctive pattern of temporal atrophy in patients with frontotemporal dementia and the I383V variant in . J Neurol Neurosurg Psychiatry. 2021;92(7):787-789doi: 10.1136/jnnp-2020-325150.
Mol, M. O., Nijmeijer, S. W. R., van Rooij, J. G. J., van Spaendonk, R. M. L., Pijnenburg, Y. A. L., van der Lee, S. J., van Minkelen, R., Donker Kaat, L., Rozemuller, A. J. M., Janse van Mantgem, M. R., van Rheenen, W., van Es, M. A., Veldink, J. H., Hennekam, F. A. M., Vernooij, M., van Swieten, J. C., Cohn-Hokke, P. E., Seelaar, H., & Dopper, E. G. P. (2021). Distinctive pattern of temporal atrophy in patients with frontotemporal dementia and the I383V variant in . Journal of neurology, neurosurgery, and psychiatry, 92(7), 787-789. https://doi.org/10.1136/jnnp-2020-325150
Mol, Merel O, et al. "Distinctive pattern of temporal atrophy in patients with frontotemporal dementia and the I383V variant in ." Journal of neurology, neurosurgery, and psychiatry vol. 92,7 (2021): 787-789. doi: https://doi.org/10.1136/jnnp-2020-325150
Mol MO, Nijmeijer SWR, van Rooij JGJ, van Spaendonk RML, Pijnenburg YAL, van der Lee SJ, van Minkelen R, Donker Kaat L, Rozemuller AJM, Janse van Mantgem MR, van Rheenen W, van Es MA, Veldink JH, Hennekam FAM, Vernooij M, van Swieten JC, Cohn-Hokke PE, Seelaar H, Dopper EGP. Distinctive pattern of temporal atrophy in patients with frontotemporal dementia and the I383V variant in . J Neurol Neurosurg Psychiatry. 2021 Jul;92(7):787-789. doi: 10.1136/jnnp-2020-325150. Epub 2021 Jan 15. PMID: 33452055; PMCID: PMC8223666.
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Amyloid-β, p-tau and reactive microglia are pathological correlates of MRI cortical atrophy in Alzheimer's disease.

Brain communications

Frigerio I, Boon BDC, Lin CP, Galis-de Graaf Y, Bol J, Preziosa P, Twisk J, Barkhof F, Hoozemans JJM, Bouwman FH, Rozemuller AJM, van de Berg WDJ, Jonkman LE.
PMID: 34927073
Brain Commun. 2021 Nov 24;3(4):fcab281. doi: 10.1093/braincomms/fcab281. eCollection 2021.

Alzheimer's disease is characterized by cortical atrophy on MRI and abnormal depositions of amyloid-beta, phosphorylated-tau and inflammation pathologically. However, the relative contribution of these pathological hallmarks to cortical atrophy, a widely used MRI biomarker in Alzheimer's disease, is yet...

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Frigerio I, Boon BDC, Lin CP, et al. Amyloid-β, p-tau and reactive microglia are pathological correlates of MRI cortical atrophy in Alzheimer's disease. Brain Commun. 2021;3(4):fcab281doi: 10.1093/braincomms/fcab281.
Frigerio, I., Boon, B. D. C., Lin, C. P., Galis-de Graaf, Y., Bol, J., Preziosa, P., Twisk, J., Barkhof, F., Hoozemans, J. J. M., Bouwman, F. H., Rozemuller, A. J. M., van de Berg, W. D. J., & Jonkman, L. E. (2021). Amyloid-β, p-tau and reactive microglia are pathological correlates of MRI cortical atrophy in Alzheimer's disease. Brain communications, 3(4), fcab281. https://doi.org/10.1093/braincomms/fcab281
Frigerio, Irene, et al. "Amyloid-β, p-tau and reactive microglia are pathological correlates of MRI cortical atrophy in Alzheimer's disease." Brain communications vol. 3,4 (2021): fcab281. doi: https://doi.org/10.1093/braincomms/fcab281
Frigerio I, Boon BDC, Lin CP, Galis-de Graaf Y, Bol J, Preziosa P, Twisk J, Barkhof F, Hoozemans JJM, Bouwman FH, Rozemuller AJM, van de Berg WDJ, Jonkman LE. Amyloid-β, p-tau and reactive microglia are pathological correlates of MRI cortical atrophy in Alzheimer's disease. Brain Commun. 2021 Nov 24;3(4):fcab281. doi: 10.1093/braincomms/fcab281. eCollection 2021. PMID: 34927073; PMCID: PMC8677327.
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Neuropathology of .

Brain communications

Dijkstra AA, Haify SN, Verwey NA, Prins ND, van der Toorn EC, Rozemuller AJM, Bugiani M, den Dunnen WFA, Todd PK, Charlet-Berguerand N, Willemsen R, Hukema RK, Hoozemans JJM.
PMID: 33709078
Brain Commun. 2021 Jan 27;3(1):fcab007. doi: 10.1093/braincomms/fcab007. eCollection 2021.

CGG repeat expansions within the premutation range (55-200) of the

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Dijkstra AA, Haify SN, Verwey NA, et al. Neuropathology of . Brain Commun. 2021;3(1):fcab007doi: 10.1093/braincomms/fcab007.
Dijkstra, A. A., Haify, S. N., Verwey, N. A., Prins, N. D., van der Toorn, E. C., Rozemuller, A. J. M., Bugiani, M., den Dunnen, W. F. A., Todd, P. K., Charlet-Berguerand, N., Willemsen, R., Hukema, R. K., & Hoozemans, J. J. M. (2021). Neuropathology of . Brain communications, 3(1), fcab007. https://doi.org/10.1093/braincomms/fcab007
Dijkstra, Anke A, et al. "Neuropathology of ." Brain communications vol. 3,1 (2021): fcab007. doi: https://doi.org/10.1093/braincomms/fcab007
Dijkstra AA, Haify SN, Verwey NA, Prins ND, van der Toorn EC, Rozemuller AJM, Bugiani M, den Dunnen WFA, Todd PK, Charlet-Berguerand N, Willemsen R, Hukema RK, Hoozemans JJM. Neuropathology of . Brain Commun. 2021 Jan 27;3(1):fcab007. doi: 10.1093/braincomms/fcab007. eCollection 2021. PMID: 33709078; PMCID: PMC7936660.
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Associating Alzheimer's disease pathology with its cerebrospinal fluid biomarkers.

Brain : a journal of neurology

Bridel C, Somers C, Sieben A, Rozemuller A, Niemantsverdriet E, Struyfs H, Vermeiren Y, Van Broeckhoven C, De Deyn PP, Bjerke M, Nagels G, Teunissen CE, Engelborghs S.
PMID: 35026840
Brain. 2022 Jan 13; doi: 10.1093/brain/awac013. Epub 2022 Jan 13.

Alzheimer's disease cerebrospinal fluid (CSF) biomarkers 42 amino acid long amyloid-β peptide (Aβ1-42), total tau protein (T-tau), and tau protein phosphorylated at threonine 181 (P-tau181) are considered surrogate biomarkers of Alzheimer's disease pathology, and significantly improve diagnostic accuracy. Their...

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Bridel C, Somers C, Sieben A, et al. Associating Alzheimer's disease pathology with its cerebrospinal fluid biomarkers. Brain. 2022;doi: 10.1093/brain/awac013.
Bridel, C., Somers, C., Sieben, A., Rozemuller, A., Niemantsverdriet, E., Struyfs, H., Vermeiren, Y., Van Broeckhoven, C., De Deyn, P. P., Bjerke, M., Nagels, G., Teunissen, C. E., & Engelborghs, S. (2022). Associating Alzheimer's disease pathology with its cerebrospinal fluid biomarkers. Brain : a journal of neurology, . https://doi.org/10.1093/brain/awac013
Bridel, Claire, et al. "Associating Alzheimer's disease pathology with its cerebrospinal fluid biomarkers." Brain : a journal of neurology vol. (2022). doi: https://doi.org/10.1093/brain/awac013
Bridel C, Somers C, Sieben A, Rozemuller A, Niemantsverdriet E, Struyfs H, Vermeiren Y, Van Broeckhoven C, De Deyn PP, Bjerke M, Nagels G, Teunissen CE, Engelborghs S. Associating Alzheimer's disease pathology with its cerebrospinal fluid biomarkers. Brain. 2022 Jan 13; doi: 10.1093/brain/awac013. Epub 2022 Jan 13. PMID: 35026840.
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Schizophrenia as a mimic of behavioral variant frontotemporal dementia.

Neurocase

Kerssens CJ, Krudop WA, Prins ND, van Berckel BN, Rozemuller A, Seeley WW, Scheltens P, Stek ML, Pijnenburg YA.
PMID: 27223596
Neurocase. 2016 Jun;22(3):285-8. doi: 10.1080/13554794.2016.1187178. Epub 2016 May 25.

Recently, the diagnostic criteria for the behavioral variant of frontotemporal dementia were revised. Although these criteria offer a relatively high sensitivity, their specificity is yet unknown. We describe a 54-year-old woman fulfilling criteria for both late-onset schizophrenia and probable...

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Kerssens CJ, Krudop WA, Prins ND, et al. Schizophrenia as a mimic of behavioral variant frontotemporal dementia. Neurocase. 2016;22(3):285-8doi: 10.1080/13554794.2016.1187178.
Kerssens, C. J., Krudop, W. A., Prins, N. D., van Berckel, B. N., Rozemuller, A., Seeley, W. W., Scheltens, P., Stek, M. L., & Pijnenburg, Y. A. (2016). Schizophrenia as a mimic of behavioral variant frontotemporal dementia. Neurocase, 22(3), 285-8. https://doi.org/10.1080/13554794.2016.1187178
Kerssens, Cora J, et al. "Schizophrenia as a mimic of behavioral variant frontotemporal dementia." Neurocase vol. 22,3 (2016): 285-8. doi: https://doi.org/10.1080/13554794.2016.1187178
Kerssens CJ, Krudop WA, Prins ND, van Berckel BN, Rozemuller A, Seeley WW, Scheltens P, Stek ML, Pijnenburg YA. Schizophrenia as a mimic of behavioral variant frontotemporal dementia. Neurocase. 2016 Jun;22(3):285-8. doi: 10.1080/13554794.2016.1187178. Epub 2016 May 25. PMID: 27223596; PMCID: PMC5790990.
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Prion protein codon 129 polymorphism in mild cognitive impairment and dementia: the Rotterdam Study.

Brain communications

Karamujić-Čomić H, Ahmad S, Lysen TS, Heshmatollah A, Roshchupkin GV, Vernooij MW, Rozemuller AJM, Ikram MA, Amin N, van Duijn CM.
PMID: 32954288
Brain Commun. 2020 Mar 20;2(1):fcaa030. doi: 10.1093/braincomms/fcaa030. eCollection 2020.

Creutzfeldt-Jakob disease is a rare, fatal, neurodegenerative disease caused by the accumulation of abnormally folded prion proteins. The common polymorphism at codon 129 (methionine/valine) in the prion protein (

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Karamujić-Čomić H, Ahmad S, Lysen TS, et al. Prion protein codon 129 polymorphism in mild cognitive impairment and dementia: the Rotterdam Study. Brain Commun. 2020;2(1):fcaa030doi: 10.1093/braincomms/fcaa030.
Karamujić-Čomić, H., Ahmad, S., Lysen, T. S., Heshmatollah, A., Roshchupkin, G. V., Vernooij, M. W., Rozemuller, A. J. M., Ikram, M. A., Amin, N., & van Duijn, C. M. (2020). Prion protein codon 129 polymorphism in mild cognitive impairment and dementia: the Rotterdam Study. Brain communications, 2(1), fcaa030. https://doi.org/10.1093/braincomms/fcaa030
Karamujić-Čomić, Hata, et al. "Prion protein codon 129 polymorphism in mild cognitive impairment and dementia: the Rotterdam Study." Brain communications vol. 2,1 (2020): fcaa030. doi: https://doi.org/10.1093/braincomms/fcaa030
Karamujić-Čomić H, Ahmad S, Lysen TS, Heshmatollah A, Roshchupkin GV, Vernooij MW, Rozemuller AJM, Ikram MA, Amin N, van Duijn CM. Prion protein codon 129 polymorphism in mild cognitive impairment and dementia: the Rotterdam Study. Brain Commun. 2020 Mar 20;2(1):fcaa030. doi: 10.1093/braincomms/fcaa030. eCollection 2020. PMID: 32954288; PMCID: PMC7425338.
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