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[Not Available].

Annals of burns and fire disasters

Chaibdraa A, Medjelekh MS, Saouli A, Bentakouk MC.
PMID: 27279806
Ann Burns Fire Disasters. 2015 Sep 30;28(3):192-5.

Use of local/regional anesthesia in burn patients is limited by many factors. It is considered as marginal in the multimodal treatment of nociceptive pain. We conducted a retrospective study on regional anesthesia used for analgesia over a period of...

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Chaibdraa A, Medjelekh MS, Saouli A, et al. Quelle place pour l'anesthésie locorégionale chez les brûlés? [Not Available]. Ann Burns Fire Disasters. 2015;28(3):192-5
Chaibdraa, A., Medjelekh, M. S., Saouli, A., & Bentakouk, M. C. (2015). Quelle place pour l'anesthésie locorégionale chez les brûlés? [Not Available]. Annals of burns and fire disasters, 28(3), 192-5.
Chaibdraa, A, et al. "Quelle place pour l'anesthésie locorégionale chez les brûlés?" [Not Available]. Annals of burns and fire disasters vol. 28,3 (2015): 192-5.
Chaibdraa A, Medjelekh MS, Saouli A, Bentakouk MC. Quelle place pour l'anesthésie locorégionale chez les brûlés? [Not Available]. Ann Burns Fire Disasters. 2015 Sep 30;28(3):192-5. French. PMID: 27279806; PMCID: PMC4883604.
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The costimulatory molecule CD226 signals through VAV1 to amplify TCR signals and promote IL-17 production by CD4.

Science signaling

Gaud G, Roncagalli R, Chaoui K, Bernard I, Familiades J, Colacios C, Kassem S, Monsarrat B, Burlet-Schiltz O, de Peredo AG, Malissen B, Saoudi A.
PMID: 29991650
Sci Signal. 2018 Jul 10;11(538). doi: 10.1126/scisignal.aar3083.

The activation of T cells requires the guanine nucleotide exchange factor VAV1. Using mice in which a tag for affinity purification was attached to endogenous VAV1 molecules, we analyzed by quantitative mass spectrometry the signaling complex that assembles around...

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Gaud G, Roncagalli R, Chaoui K, et al. The costimulatory molecule CD226 signals through VAV1 to amplify TCR signals and promote IL-17 production by CD4. Sci Signal. 2018;11(538)doi: 10.1126/scisignal.aar3083.
Gaud, G., Roncagalli, R., Chaoui, K., Bernard, I., Familiades, J., Colacios, C., Kassem, S., Monsarrat, B., Burlet-Schiltz, O., de Peredo, A. G., Malissen, B., & Saoudi, A. (2018). The costimulatory molecule CD226 signals through VAV1 to amplify TCR signals and promote IL-17 production by CD4. Science signaling, 11(538), . https://doi.org/10.1126/scisignal.aar3083
Gaud, Guillaume, et al. "The costimulatory molecule CD226 signals through VAV1 to amplify TCR signals and promote IL-17 production by CD4." Science signaling vol. 11,538 (2018). doi: https://doi.org/10.1126/scisignal.aar3083
Gaud G, Roncagalli R, Chaoui K, Bernard I, Familiades J, Colacios C, Kassem S, Monsarrat B, Burlet-Schiltz O, de Peredo AG, Malissen B, Saoudi A. The costimulatory molecule CD226 signals through VAV1 to amplify TCR signals and promote IL-17 production by CD4. Sci Signal. 2018 Jul 10;11(538). doi: 10.1126/scisignal.aar3083. PMID: 29991650.
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[RNA splicing modulation: Therapeutic progress and perspectives].

Medecine sciences : M/S

Saoudi A, Goyenvalle A.
PMID: 34180822
Med Sci (Paris). 2021 Jun-Jul;37(6):625-631. doi: 10.1051/medsci/2021091. Epub 2021 Jun 28.

Advances in genetic and genomic research continue to increase our knowledge of hereditary diseases, and an increasing number of them are being attributed to aberrant splicing, thus representing ideal targets for RNA modulation therapies. New strategies to skip or...

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Saoudi A, Goyenvalle A. Les approches thérapeutiques de modulation de l’épissage - Avancées et perspectives. [RNA splicing modulation: Therapeutic progress and perspectives]. Med Sci (Paris). 2021;37(6):625-631doi: 10.1051/medsci/2021091.
Saoudi, A., & Goyenvalle, A. (2021). Les approches thérapeutiques de modulation de l’épissage - Avancées et perspectives. [RNA splicing modulation: Therapeutic progress and perspectives]. Medecine sciences : M/S, 37(6), 625-631. https://doi.org/10.1051/medsci/2021091
Saoudi, Amel, and Goyenvalle, Aurélie. "Les approches thérapeutiques de modulation de l’épissage - Avancées et perspectives." [RNA splicing modulation: Therapeutic progress and perspectives]. Medecine sciences : M/S vol. 37,6 (2021): 625-631. doi: https://doi.org/10.1051/medsci/2021091
Saoudi A, Goyenvalle A. Les approches thérapeutiques de modulation de l’épissage - Avancées et perspectives. [RNA splicing modulation: Therapeutic progress and perspectives]. Med Sci (Paris). 2021 Jun-Jul;37(6):625-631. doi: 10.1051/medsci/2021091. Epub 2021 Jun 28. French. PMID: 34180822.
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A rectal foreign body: An unexpected cause of a rectovesical fistula with hematuria.

Urology case reports

Hosni A, Saouli A, Koutani A, Iben Attya Andalousi A, Jroundi L, Laamrani FZ.
PMID: 33604245
Urol Case Rep. 2021 Feb 04;36:101596. doi: 10.1016/j.eucr.2021.101596. eCollection 2021 May.

Cases of rectal foreign bodies (RFB) insertion are frequently encountred in the last years, leading to many complications, while many patients tends to obscure the context. This entity represent a particular form of rectal injuries that can be more...

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Hosni A, Saouli A, Koutani A, et al. A rectal foreign body: An unexpected cause of a rectovesical fistula with hematuria. Urol Case Rep. 2021;36:101596doi: 10.1016/j.eucr.2021.101596.
Hosni, A., Saouli, A., Koutani, A., Iben Attya Andalousi, A., Jroundi, L., & Laamrani, F. Z. (2021). A rectal foreign body: An unexpected cause of a rectovesical fistula with hematuria. Urology case reports, 36101596. https://doi.org/10.1016/j.eucr.2021.101596
Hosni, Abdelmoughit, et al. "A rectal foreign body: An unexpected cause of a rectovesical fistula with hematuria." Urology case reports vol. 36 (2021): 101596. doi: https://doi.org/10.1016/j.eucr.2021.101596
Hosni A, Saouli A, Koutani A, Iben Attya Andalousi A, Jroundi L, Laamrani FZ. A rectal foreign body: An unexpected cause of a rectovesical fistula with hematuria. Urol Case Rep. 2021 Feb 04;36:101596. doi: 10.1016/j.eucr.2021.101596. eCollection 2021 May. PMID: 33604245; PMCID: PMC7872978.
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Reversible CO.

Angewandte Chemie (International ed. in English)

Rodriguez R, Alvarado-Beltran I, Saouli J, Saffon-Merceron N, Baceiredo A, Branchadell V, Kato T.
PMID: 29336108
Angew Chem Int Ed Engl. 2018 Mar 01;57(10):2635-2638. doi: 10.1002/anie.201709787. Epub 2018 Feb 08.

The donor-stabilized sila-β-lactone 1 reacts with CO

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Rodriguez R, Alvarado-Beltran I, Saouli J, et al. Reversible CO. Angew Chem Int Ed Engl. 2018;57(10):2635-2638doi: 10.1002/anie.201709787.
Rodriguez, R., Alvarado-Beltran, I., Saouli, J., Saffon-Merceron, N., Baceiredo, A., Branchadell, V., & Kato, T. (2018). Reversible CO. Angewandte Chemie (International ed. in English), 57(10), 2635-2638. https://doi.org/10.1002/anie.201709787
Rodriguez, Ricardo, et al. "Reversible CO." Angewandte Chemie (International ed. in English) vol. 57,10 (2018): 2635-2638. doi: https://doi.org/10.1002/anie.201709787
Rodriguez R, Alvarado-Beltran I, Saouli J, Saffon-Merceron N, Baceiredo A, Branchadell V, Kato T. Reversible CO. Angew Chem Int Ed Engl. 2018 Mar 01;57(10):2635-2638. doi: 10.1002/anie.201709787. Epub 2018 Feb 08. PMID: 29336108.
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Immunotherapy of AD: Alum Demain?.

The journal of prevention of Alzheimer's disease

Liblau R, Saoudi A.
PMID: 29231228
J Prev Alzheimers Dis. 2015;2(2):80-81. doi: 10.14283/jpad.2015.64.

No abstract available.

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Liblau R, Saoudi A. Immunotherapy of AD: Alum Demain?. J Prev Alzheimers Dis. 2015;2(2):80-81doi: 10.14283/jpad.2015.64.
Liblau, R., & Saoudi, A. (2015). Immunotherapy of AD: Alum Demain?. The journal of prevention of Alzheimer's disease, 2(2), 80-81. https://doi.org/10.14283/jpad.2015.64
Liblau, R, and Saoudi, A. "Immunotherapy of AD: Alum Demain?." The journal of prevention of Alzheimer's disease vol. 2,2 (2015): 80-81. doi: https://doi.org/10.14283/jpad.2015.64
Liblau R, Saoudi A. Immunotherapy of AD: Alum Demain?. J Prev Alzheimers Dis. 2015;2(2):80-81. doi: 10.14283/jpad.2015.64. PMID: 29231228.
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A Stable Monomeric SiO.

Angewandte Chemie (International ed. in English)

Rodriguez R, Gau D, Saouli J, Baceiredo A, Saffon-Merceron N, Branchadell V, Kato T.
PMID: 28247559
Angew Chem Int Ed Engl. 2017 Mar 27;56(14):3935-3939. doi: 10.1002/anie.201611851. Epub 2017 Mar 01.

Isolation of a monomeric SiO

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Rodriguez R, Gau D, Saouli J, et al. A Stable Monomeric SiO. Angew Chem Int Ed Engl. 2017;56(14):3935-3939doi: 10.1002/anie.201611851.
Rodriguez, R., Gau, D., Saouli, J., Baceiredo, A., Saffon-Merceron, N., Branchadell, V., & Kato, T. (2017). A Stable Monomeric SiO. Angewandte Chemie (International ed. in English), 56(14), 3935-3939. https://doi.org/10.1002/anie.201611851
Rodriguez, Ricardo, et al. "A Stable Monomeric SiO." Angewandte Chemie (International ed. in English) vol. 56,14 (2017): 3935-3939. doi: https://doi.org/10.1002/anie.201611851
Rodriguez R, Gau D, Saouli J, Baceiredo A, Saffon-Merceron N, Branchadell V, Kato T. A Stable Monomeric SiO. Angew Chem Int Ed Engl. 2017 Mar 27;56(14):3935-3939. doi: 10.1002/anie.201611851. Epub 2017 Mar 01. PMID: 28247559.
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[Not Available].

Annals of burns and fire disasters

Chaibdraa A, Medjellekh MS, Saouli A, Bentakouk MC.
PMID: 21991140
Ann Burns Fire Disasters. 2008 Dec 31;21(4):210-8.

Le Pseudomonasest un agent pathogène à l'origine d'infections nosocomiales graves dans les centres des brûlés. Son opportunisme et sa virulence en font une préoccupation majeure. Ce travail se propose d'évaluer la place de cette bactérie dans l'écologie bactérienne locale...

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Chaibdraa A, Medjellekh MS, Saouli A, et al. Le Pseudomonas: Experience du Centre des Brules D'Annaba et Revue de la Litterature. [Not Available]. Ann Burns Fire Disasters. 2008;21(4):210-8
Chaibdraa, A., Medjellekh, M. S., Saouli, A., & Bentakouk, M. C. (2008). Le Pseudomonas: Experience du Centre des Brules D'Annaba et Revue de la Litterature. [Not Available]. Annals of burns and fire disasters, 21(4), 210-8.
Chaibdraa, A, et al. "Le Pseudomonas: Experience du Centre des Brules D'Annaba et Revue de la Litterature." [Not Available]. Annals of burns and fire disasters vol. 21,4 (2008): 210-8.
Chaibdraa A, Medjellekh MS, Saouli A, Bentakouk MC. Le Pseudomonas: Experience du Centre des Brules D'Annaba et Revue de la Litterature. [Not Available]. Ann Burns Fire Disasters. 2008 Dec 31;21(4):210-8. French. PMID: 21991140; PMCID: PMC3188199.
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Quantification of the heat wave effect on mortality in nine French cities during summer 2006.

PLoS currents

Pascal M, Le Tertre A, Saoudi A.
PMID: 22453900
PLoS Curr. 2012 Feb 23;4:RRN1307. doi: 10.1371/currents.RRN1307.

BACKGROUND: July 2006 was the first major heat wave in France after the creation of a heat prevention plan. Understanding its impacts on health will help improving the efficiency of this plan. We assessed the mortality impact of the...

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Pascal M, Le Tertre A, Saoudi A. Quantification of the heat wave effect on mortality in nine French cities during summer 2006. PLoS Curr. 2012;4:RRN1307doi: 10.1371/currents.RRN1307.
Pascal, M., Le Tertre, A., & Saoudi, A. (2012). Quantification of the heat wave effect on mortality in nine French cities during summer 2006. PLoS currents, 4RRN1307. https://doi.org/10.1371/currents.RRN1307
Pascal, Mathilde, et al. "Quantification of the heat wave effect on mortality in nine French cities during summer 2006." PLoS currents vol. 4 (2012): RRN1307. doi: https://doi.org/10.1371/currents.RRN1307
Pascal M, Le Tertre A, Saoudi A. Quantification of the heat wave effect on mortality in nine French cities during summer 2006. PLoS Curr. 2012 Feb 23;4:RRN1307. doi: 10.1371/currents.RRN1307. PMID: 22453900; PMCID: PMC3289125.1.
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Emotional behavior and brain anatomy of the mdx52 mouse model of Duchenne muscular dystrophy.

Disease models & mechanisms

Saoudi A, Zarrouki F, Sebrié C, Izabelle C, Goyenvalle A, Vaillend C.
PMID: 34546327
Dis Model Mech. 2021 Sep 01;14(9). doi: 10.1242/dmm.049028. Epub 2021 Sep 21.

The exon-52-deleted mdx52 mouse is a critical model of Duchenne muscular dystrophy (DMD), as it features a deletion in a hotspot region of the DMD gene, frequently mutated in patients. Deletion of exon 52 impedes expression of several brain...

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Saoudi A, Zarrouki F, Sebrié C, et al. Emotional behavior and brain anatomy of the mdx52 mouse model of Duchenne muscular dystrophy. Dis Model Mech. 2021;14(9)doi: 10.1242/dmm.049028.
Saoudi, A., Zarrouki, F., Sebrié, C., Izabelle, C., Goyenvalle, A., & Vaillend, C. (2021). Emotional behavior and brain anatomy of the mdx52 mouse model of Duchenne muscular dystrophy. Disease models & mechanisms, 14(9), . https://doi.org/10.1242/dmm.049028
Saoudi, Amel, et al. "Emotional behavior and brain anatomy of the mdx52 mouse model of Duchenne muscular dystrophy." Disease models & mechanisms vol. 14,9 (2021). doi: https://doi.org/10.1242/dmm.049028
Saoudi A, Zarrouki F, Sebrié C, Izabelle C, Goyenvalle A, Vaillend C. Emotional behavior and brain anatomy of the mdx52 mouse model of Duchenne muscular dystrophy. Dis Model Mech. 2021 Sep 01;14(9). doi: 10.1242/dmm.049028. Epub 2021 Sep 21. PMID: 34546327; PMCID: PMC8476816.
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Early and late ventricular arrhythmias complicating ST-segment elevation myocardial infarction.

Archives of cardiovascular diseases

Auffret V, Bourenane H, Sharobeem S, Leurent G, Didier R, Gilard M, Nicol PP, Payot L, Filippi E, Hacot JP, Rouault G, Saouli D, Druelles P, Coudert I, Boulanger B, Cherfaoui T, Treuil J, Bedossa M, Boulmier D, Le Guellec M, Martins RP, Le Breton H.
PMID: 34953752
Arch Cardiovasc Dis. 2022 Jan;115(1):4-16. doi: 10.1016/j.acvd.2021.10.012. Epub 2021 Dec 23.

BACKGROUND: Ventricular arrhythmias can be life-threatening complications of ST-segment elevation myocardial infarction (STEMI).AIMS: To describe the incidence, predictors and in-hospital impact of early ventricular arrhythmia (EVA, occurring

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Auffret V, Bourenane H, Sharobeem S, et al. Early and late ventricular arrhythmias complicating ST-segment elevation myocardial infarction. Arch Cardiovasc Dis. 2021;115(1):4-16doi: 10.1016/j.acvd.2021.10.012.
Auffret, V., Bourenane, H., Sharobeem, S., Leurent, G., Didier, R., Gilard, M., Nicol, P. P., Payot, L., Filippi, E., Hacot, J. P., Rouault, G., Saouli, D., Druelles, P., Coudert, I., Boulanger, B., Cherfaoui, T., Treuil, J., Bedossa, M., Boulmier, D., Le Guellec, M., Martins, R. P., & Le Breton, H. (2022). Early and late ventricular arrhythmias complicating ST-segment elevation myocardial infarction. Archives of cardiovascular diseases, 115(1), 4-16. https://doi.org/10.1016/j.acvd.2021.10.012
Auffret, Vincent, et al. "Early and late ventricular arrhythmias complicating ST-segment elevation myocardial infarction." Archives of cardiovascular diseases vol. 115,1 (2022): 4-16. doi: https://doi.org/10.1016/j.acvd.2021.10.012
Auffret V, Bourenane H, Sharobeem S, Leurent G, Didier R, Gilard M, Nicol PP, Payot L, Filippi E, Hacot JP, Rouault G, Saouli D, Druelles P, Coudert I, Boulanger B, Cherfaoui T, Treuil J, Bedossa M, Boulmier D, Le Guellec M, Martins RP, Le Breton H. Early and late ventricular arrhythmias complicating ST-segment elevation myocardial infarction. Arch Cardiovasc Dis. 2022 Jan;115(1):4-16. doi: 10.1016/j.acvd.2021.10.012. Epub 2021 Dec 23. PMID: 34953752.
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VAV1 and BAFF, via NFκB pathway, are genetic risk factors for myasthenia gravis.

Annals of clinical and translational neurology

Avidan N, Le Panse R, Harbo HF, Bernasconi P, Poulas K, Ginzburg E, Cavalcante P, Colleoni L, Baggi F, Antozzi C, Truffault F, Horn-Saban S, Pöschel S, Zagoriti Z, Maniaol A, Lie BA, Bernard I, Saoudi A, Illes Z, Casasnovas Pons C, Melms A, Tzartos S, Willcox N, Kostera-Pruszczyk A, Tallaksen C, Mantegazza R, Berrih-Aknin S, Miller A.
PMID: 25356403
Ann Clin Transl Neurol. 2014 May;1(5):329-39. doi: 10.1002/acn3.51. Epub 2014 Apr 11.

OBJECTIVE: To identify novel genetic loci that predispose to early-onset myasthenia gravis (EOMG) applying a two-stage association study, exploration, and replication strategy.METHODS: Thirty-four loci and one confirmation loci, human leukocyte antigen (HLA)-DRA, were selected as candidate genes by team...

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Avidan N, Le Panse R, Harbo HF, et al. VAV1 and BAFF, via NFκB pathway, are genetic risk factors for myasthenia gravis. Ann Clin Transl Neurol. 2014;1(5):329-39doi: 10.1002/acn3.51.
Avidan, N., Le Panse, R., Harbo, H. F., Bernasconi, P., Poulas, K., Ginzburg, E., Cavalcante, P., Colleoni, L., Baggi, F., Antozzi, C., Truffault, F., Horn-Saban, S., Pöschel, S., Zagoriti, Z., Maniaol, A., Lie, B. A., Bernard, I., Saoudi, A., Illes, Z., Casasnovas Pons, C., Melms, A., Tzartos, S., Willcox, N., Kostera-Pruszczyk, A., Tallaksen, C., Mantegazza, R., Berrih-Aknin, S., & Miller, A. (2014). VAV1 and BAFF, via NFκB pathway, are genetic risk factors for myasthenia gravis. Annals of clinical and translational neurology, 1(5), 329-39. https://doi.org/10.1002/acn3.51
Avidan, Nili, et al. "VAV1 and BAFF, via NFκB pathway, are genetic risk factors for myasthenia gravis." Annals of clinical and translational neurology vol. 1,5 (2014): 329-39. doi: https://doi.org/10.1002/acn3.51
Avidan N, Le Panse R, Harbo HF, Bernasconi P, Poulas K, Ginzburg E, Cavalcante P, Colleoni L, Baggi F, Antozzi C, Truffault F, Horn-Saban S, Pöschel S, Zagoriti Z, Maniaol A, Lie BA, Bernard I, Saoudi A, Illes Z, Casasnovas Pons C, Melms A, Tzartos S, Willcox N, Kostera-Pruszczyk A, Tallaksen C, Mantegazza R, Berrih-Aknin S, Miller A. VAV1 and BAFF, via NFκB pathway, are genetic risk factors for myasthenia gravis. Ann Clin Transl Neurol. 2014 May;1(5):329-39. doi: 10.1002/acn3.51. Epub 2014 Apr 11. PMID: 25356403; PMCID: PMC4184684.
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Showing 1 to 12 of 31 entries