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Self-organized shocks in the sedimentation of a granular gas.

Physical review. E, Statistical, nonlinear, and soft matter physics

Almazán L, Serero D, Salueña C, Pöschel T.
PMID: 26172712
Phys Rev E Stat Nonlin Soft Matter Phys. 2015 Jun;91(6):062214. doi: 10.1103/PhysRevE.91.062214. Epub 2015 Jun 26.

A granular gas in gravity heated from below develops a certain stationary density profile. When the heating is switched off, the granular gas collapses. We investigate the process of sedimentation using computational hydrodynamics, based on the Jenkins-Richman theory, and...

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Almazán L, Serero D, Salueña C, et al. Self-organized shocks in the sedimentation of a granular gas. Phys Rev E Stat Nonlin Soft Matter Phys. 2015;91(6):062214doi: 10.1103/PhysRevE.91.062214.
Almazán, L., Serero, D., Salueña, C., & Pöschel, T. (2015). Self-organized shocks in the sedimentation of a granular gas. Physical review. E, Statistical, nonlinear, and soft matter physics, 91(6), 062214. https://doi.org/10.1103/PhysRevE.91.062214
Almazán, Lidia, et al. "Self-organized shocks in the sedimentation of a granular gas." Physical review. E, Statistical, nonlinear, and soft matter physics vol. 91,6 (2015): 062214. doi: https://doi.org/10.1103/PhysRevE.91.062214
Almazán L, Serero D, Salueña C, Pöschel T. Self-organized shocks in the sedimentation of a granular gas. Phys Rev E Stat Nonlin Soft Matter Phys. 2015 Jun;91(6):062214. doi: 10.1103/PhysRevE.91.062214. Epub 2015 Jun 26. PMID: 26172712.
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The Prognostic Impact of Molecular Subtypes and Very Young Age on Breast Conserving Surgery in Early Stage Breast Cancer.

Cureus

Ordu C, McGuire K, Alco G, Nur Pilanci K, Koksal UI, Elbüken F, Erdogan Z, Agacayak F, Ilgun S, Sarsenov D, Öztürk A, İğdem Ş, Okkan S, Eralp Y, Dincer M, Ozmen V.
PMID: 27433412
Cureus. 2016 Jun 07;8(6):e633. doi: 10.7759/cureus.633.

BACKGROUND: Premenopausal breast cancer with a triple-negative phenotype (TNBC) has been associated with inferior locoregional recurrence free survival (LRFS) and overall survival (OS) after breast conserving surgery (BCS). The aim of this study is to analyze the association between...

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Ordu C, McGuire K, Alco G, et al. The Prognostic Impact of Molecular Subtypes and Very Young Age on Breast Conserving Surgery in Early Stage Breast Cancer. Cureus. 2016;8(6):e633doi: 10.7759/cureus.633.
Ordu, C., McGuire, K., Alco, G., Nur Pilanci, K., Koksal, U. I., Elbüken, F., Erdogan, Z., Agacayak, F., Ilgun, S., Sarsenov, D., Öztürk, A., İğdem, �. �., Okkan, S., Eralp, Y., Dincer, M., & Ozmen, V. (2016). The Prognostic Impact of Molecular Subtypes and Very Young Age on Breast Conserving Surgery in Early Stage Breast Cancer. Cureus, 8(6), e633. https://doi.org/10.7759/cureus.633
Ordu, Cetin, et al. "The Prognostic Impact of Molecular Subtypes and Very Young Age on Breast Conserving Surgery in Early Stage Breast Cancer." Cureus vol. 8,6 (2016): e633. doi: https://doi.org/10.7759/cureus.633
Ordu C, McGuire K, Alco G, Nur Pilanci K, Koksal UI, Elbüken F, Erdogan Z, Agacayak F, Ilgun S, Sarsenov D, Öztürk A, İğdem Ş, Okkan S, Eralp Y, Dincer M, Ozmen V. The Prognostic Impact of Molecular Subtypes and Very Young Age on Breast Conserving Surgery in Early Stage Breast Cancer. Cureus. 2016 Jun 07;8(6):e633. doi: 10.7759/cureus.633. PMID: 27433412; PMCID: PMC4938212.
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Novel .

Journal of medical genetics

Soria-Valles C, Carrero D, Gabau E, Velasco G, Quesada V, Bárcena C, Moens M, Fieggen K, Möhrcken S, Owens M, Puente DA, Asensio Ó, Loeys B, Pérez A, Benoit V, Wuyts W, Lévy N, Hennekam RC, De Sandre-Giovannoli A, López-Otín C.
PMID: 27334370
J Med Genet. 2016 Nov;53(11):776-785. doi: 10.1136/jmedgenet-2015-103695. Epub 2016 Jun 22.

BACKGROUND: Progeroid syndromes are genetic disorders that recapitulate some phenotypes of physiological ageing. Classical progerias, such as Hutchinson-Gilford progeria syndrome (HGPS), are generally caused by mutations in METHODS AND RESULTS: Patients reported herein present an extremely homogeneous phenotype that...

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Soria-Valles C, Carrero D, Gabau E, et al. Novel . J Med Genet. 2016;53(11):776-785doi: 10.1136/jmedgenet-2015-103695.
Soria-Valles, C., Carrero, D., Gabau, E., Velasco, G., Quesada, V., Bárcena, C., Moens, M., Fieggen, K., Möhrcken, S., Owens, M., Puente, D. A., Asensio, �. �., Loeys, B., Pérez, A., Benoit, V., Wuyts, W., Lévy, N., Hennekam, R. C., De Sandre-Giovannoli, A., & López-Otín, C. (2016). Novel . Journal of medical genetics, 53(11), 776-785. https://doi.org/10.1136/jmedgenet-2015-103695
Soria-Valles, Clara, et al. "Novel ." Journal of medical genetics vol. 53,11 (2016): 776-785. doi: https://doi.org/10.1136/jmedgenet-2015-103695
Soria-Valles C, Carrero D, Gabau E, Velasco G, Quesada V, Bárcena C, Moens M, Fieggen K, Möhrcken S, Owens M, Puente DA, Asensio Ó, Loeys B, Pérez A, Benoit V, Wuyts W, Lévy N, Hennekam RC, De Sandre-Giovannoli A, López-Otín C. Novel . J Med Genet. 2016 Nov;53(11):776-785. doi: 10.1136/jmedgenet-2015-103695. Epub 2016 Jun 22. PMID: 27334370.
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Conditional Depletion of the Chlamydomonas Chloroplast ClpP Protease Activates Nuclear Genes Involved in Autophagy and Plastid Protein Quality Control.

The Plant cell

Ramundo S, Casero D, Mühlhaus T, Hemme D, Sommer F, Crèvecoeur M, Rahire M, Schroda M, Rusch J, Goodenough U, Pellegrini M, Perez-Perez ME, Crespo JL, Schaad O, Civic N, Rochaix JD.
PMID: 24879428
Plant Cell. 2014 May;26(5):2201-2222. doi: 10.1105/tpc.114.124842. Epub 2014 May 30.

Plastid protein homeostasis is critical during chloroplast biogenesis and responses to changes in environmental conditions. Proteases and molecular chaperones involved in plastid protein quality control are encoded by the nucleus except for the catalytic subunit of ClpP, an evolutionarily...

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Ramundo S, Casero D, Mühlhaus T, et al. Conditional Depletion of the Chlamydomonas Chloroplast ClpP Protease Activates Nuclear Genes Involved in Autophagy and Plastid Protein Quality Control. Plant Cell. 2014;26(5):2201-2222doi: 10.1105/tpc.114.124842.
Ramundo, S., Casero, D., Mühlhaus, T., Hemme, D., Sommer, F., Crèvecoeur, M., Rahire, M., Schroda, M., Rusch, J., Goodenough, U., Pellegrini, M., Perez-Perez, M. E., Crespo, J. L., Schaad, O., Civic, N., & Rochaix, J. D. (2014). Conditional Depletion of the Chlamydomonas Chloroplast ClpP Protease Activates Nuclear Genes Involved in Autophagy and Plastid Protein Quality Control. The Plant cell, 26(5), 2201-2222. https://doi.org/10.1105/tpc.114.124842
Ramundo, Silvia, et al. "Conditional Depletion of the Chlamydomonas Chloroplast ClpP Protease Activates Nuclear Genes Involved in Autophagy and Plastid Protein Quality Control." The Plant cell vol. 26,5 (2014): 2201-2222. doi: https://doi.org/10.1105/tpc.114.124842
Ramundo S, Casero D, Mühlhaus T, Hemme D, Sommer F, Crèvecoeur M, Rahire M, Schroda M, Rusch J, Goodenough U, Pellegrini M, Perez-Perez ME, Crespo JL, Schaad O, Civic N, Rochaix JD. Conditional Depletion of the Chlamydomonas Chloroplast ClpP Protease Activates Nuclear Genes Involved in Autophagy and Plastid Protein Quality Control. Plant Cell. 2014 May;26(5):2201-2222. doi: 10.1105/tpc.114.124842. Epub 2014 May 30. PMID: 24879428; PMCID: PMC4079378.
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Do Genes Determine Our Health? Implications for Designing Lifestyle Interventions and Drug Trials.

Circulation. Cardiovascular genetics

Wessel J, Marrero D.
PMID: 26884607
Circ Cardiovasc Genet. 2016 Feb;9(1):2-3. doi: 10.1161/CIRCGENETICS.116.001367.

No abstract available.

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Wessel J, Marrero D. Do Genes Determine Our Health? Implications for Designing Lifestyle Interventions and Drug Trials. Circ Cardiovasc Genet. 2016;9(1):2-3doi: 10.1161/CIRCGENETICS.116.001367.
Wessel, J., & Marrero, D. (2016). Do Genes Determine Our Health? Implications for Designing Lifestyle Interventions and Drug Trials. Circulation. Cardiovascular genetics, 9(1), 2-3. https://doi.org/10.1161/CIRCGENETICS.116.001367
Wessel, Jennifer, and Marrero, David. "Do Genes Determine Our Health? Implications for Designing Lifestyle Interventions and Drug Trials." Circulation. Cardiovascular genetics vol. 9,1 (2016): 2-3. doi: https://doi.org/10.1161/CIRCGENETICS.116.001367
Wessel J, Marrero D. Do Genes Determine Our Health? Implications for Designing Lifestyle Interventions and Drug Trials. Circ Cardiovasc Genet. 2016 Feb;9(1):2-3. doi: 10.1161/CIRCGENETICS.116.001367. PMID: 26884607.
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Radiation Fibrosis Syndrome Imitating Breast Cancer Recurrence; A Case Report.

The journal of breast health

Sarsenov D, Aktepe F, Özmen V.
PMID: 28331767
J Breast Health. 2017 Jan 01;13(1):40-42. doi: 10.5152/tjbh.2016.3351. eCollection 2017 Jan.

Nowadays, surgery, radiotherapy and chemotherapy are the most frequently used modalities in the treatment of breast cancer. It is very well-known that some severe complications may result from after these treatments. Early and late complications of radiotherapy are well...

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Sarsenov D, Aktepe F, Özmen V. Radiation Fibrosis Syndrome Imitating Breast Cancer Recurrence; A Case Report. J Breast Health. 2017;13(1):40-42doi: 10.5152/tjbh.2016.3351.
Sarsenov, D., Aktepe, F., & Özmen, V. (2017). Radiation Fibrosis Syndrome Imitating Breast Cancer Recurrence; A Case Report. The journal of breast health, 13(1), 40-42. https://doi.org/10.5152/tjbh.2016.3351
Sarsenov, Dauren, et al. "Radiation Fibrosis Syndrome Imitating Breast Cancer Recurrence; A Case Report." The journal of breast health vol. 13,1 (2017): 40-42. doi: https://doi.org/10.5152/tjbh.2016.3351
Sarsenov D, Aktepe F, Özmen V. Radiation Fibrosis Syndrome Imitating Breast Cancer Recurrence; A Case Report. J Breast Health. 2017 Jan 01;13(1):40-42. doi: 10.5152/tjbh.2016.3351. eCollection 2017 Jan. PMID: 28331767; PMCID: PMC5351463.
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Molecularly targeted therapy for advanced hepatocellular carcinoma - a drug development crisis?.

World journal of gastrointestinal oncology

Thillai K, Ross P, Sarker D.
PMID: 26909132
World J Gastrointest Oncol. 2016 Feb 15;8(2):173-85. doi: 10.4251/wjgo.v8.i2.173.

Hepatocellular carcinoma is the fastest growing cause of cancer related death globally. Sorafenib, a multi-targeted kinase inhibitor, is the only drug proven to improve outcomes in patients with advanced disease offering modest survival benefit. Although comprehensive genomic mapping has...

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Thillai K, Ross P, Sarker D. Molecularly targeted therapy for advanced hepatocellular carcinoma - a drug development crisis?. World J Gastrointest Oncol. 2016;8(2):173-85doi: 10.4251/wjgo.v8.i2.173.
Thillai, K., Ross, P., & Sarker, D. (2016). Molecularly targeted therapy for advanced hepatocellular carcinoma - a drug development crisis?. World journal of gastrointestinal oncology, 8(2), 173-85. https://doi.org/10.4251/wjgo.v8.i2.173
Thillai, Kiruthikah, et al. "Molecularly targeted therapy for advanced hepatocellular carcinoma - a drug development crisis?." World journal of gastrointestinal oncology vol. 8,2 (2016): 173-85. doi: https://doi.org/10.4251/wjgo.v8.i2.173
Thillai K, Ross P, Sarker D. Molecularly targeted therapy for advanced hepatocellular carcinoma - a drug development crisis?. World J Gastrointest Oncol. 2016 Feb 15;8(2):173-85. doi: 10.4251/wjgo.v8.i2.173. PMID: 26909132; PMCID: PMC4753168.
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Draft Genome of Klebsiella pneumoniae Sequence Type 512, a Multidrug-Resistant Strain Isolated during a Recent KPC Outbreak in Italy.

Genome announcements

Comandatore F, Gaibani P, Ambretti S, Landini MP, Daffonchio D, Marone P, Sambri V, Bandi C, Sassera D.
PMID: 23405316
Genome Announc. 2013 Jan;1(1). doi: 10.1128/genomeA.00035-12. Epub 2013 Jan 15.

Here, we present the draft genome sequence of Klebsiella pneumoniae subsp. pneumoniae sequence type 512 (ST512) isolated during a KPC-producer outbreak. This strain is resistant to β-lactams, cephalosporins, fluoroquinolones, aminoglycosides, macrolides, tetracyclines, and carbapenems but susceptible to colistin. The...

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Comandatore F, Gaibani P, Ambretti S, et al. Draft Genome of Klebsiella pneumoniae Sequence Type 512, a Multidrug-Resistant Strain Isolated during a Recent KPC Outbreak in Italy. Genome Announc. 2013;1(1)doi: 10.1128/genomeA.00035-12.
Comandatore, F., Gaibani, P., Ambretti, S., Landini, M. P., Daffonchio, D., Marone, P., Sambri, V., Bandi, C., & Sassera, D. (2013). Draft Genome of Klebsiella pneumoniae Sequence Type 512, a Multidrug-Resistant Strain Isolated during a Recent KPC Outbreak in Italy. Genome announcements, 1(1), . https://doi.org/10.1128/genomeA.00035-12
Comandatore, Francesco, et al. "Draft Genome of Klebsiella pneumoniae Sequence Type 512, a Multidrug-Resistant Strain Isolated during a Recent KPC Outbreak in Italy." Genome announcements vol. 1,1 (2013). doi: https://doi.org/10.1128/genomeA.00035-12
Comandatore F, Gaibani P, Ambretti S, Landini MP, Daffonchio D, Marone P, Sambri V, Bandi C, Sassera D. Draft Genome of Klebsiella pneumoniae Sequence Type 512, a Multidrug-Resistant Strain Isolated during a Recent KPC Outbreak in Italy. Genome Announc. 2013 Jan;1(1). doi: 10.1128/genomeA.00035-12. Epub 2013 Jan 15. PMID: 23405316; PMCID: PMC3569305.
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Delayed adverse events in phase I trials of molecularly targeted and cytotoxic agents.

Oncotarget

Jordan EJ, Spicer J, Sarker D.
PMID: 30338038
Oncotarget. 2018 Sep 21;9(74):33961-33971. doi: 10.18632/oncotarget.26104. eCollection 2018 Sep 21.

BACKGROUND: Grade 3 and 4 adverse events (AEs) during cycle 1 are traditionally used for dose escalation decisions in Phase I oncology trials. With molecularly targeted agents (MTAs), assessment of lower grade AEs and those in later cycles is...

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Jordan EJ, Spicer J, Sarker D. Delayed adverse events in phase I trials of molecularly targeted and cytotoxic agents. Oncotarget. 2018;9(74):33961-33971doi: 10.18632/oncotarget.26104.
Jordan, E. J., Spicer, J., & Sarker, D. (2018). Delayed adverse events in phase I trials of molecularly targeted and cytotoxic agents. Oncotarget, 9(74), 33961-33971. https://doi.org/10.18632/oncotarget.26104
Jordan, Emma J, et al. "Delayed adverse events in phase I trials of molecularly targeted and cytotoxic agents." Oncotarget vol. 9,74 (2018): 33961-33971. doi: https://doi.org/10.18632/oncotarget.26104
Jordan EJ, Spicer J, Sarker D. Delayed adverse events in phase I trials of molecularly targeted and cytotoxic agents. Oncotarget. 2018 Sep 21;9(74):33961-33971. doi: 10.18632/oncotarget.26104. eCollection 2018 Sep 21. PMID: 30338038; PMCID: PMC6188052.
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Pharmacodynamic Biomarker Development for PI3K Pathway Therapeutics.

Translational oncogenomics

Josephs DH, Sarker D.
PMID: 26917948
Transl Oncogenomics. 2016 Feb 21;7:33-49. doi: 10.4137/TOG.S30529. eCollection 2015.

The phosphatidylinositol 3-kinase (PI3K) signaling pathway is integral to many essential cell processes, including cell growth, differentiation, proliferation, motility, and metabolism. Somatic mutations and genetic amplifications that result in activation of the pathway are frequently detected in cancer. This...

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Josephs DH, Sarker D. Pharmacodynamic Biomarker Development for PI3K Pathway Therapeutics. Transl Oncogenomics. 2016;7:33-49doi: 10.4137/TOG.S30529.
Josephs, D. H., & Sarker, D. (2015). Pharmacodynamic Biomarker Development for PI3K Pathway Therapeutics. Translational oncogenomics, 733-49. https://doi.org/10.4137/TOG.S30529
Josephs, Debra H, and Sarker, Debashis. "Pharmacodynamic Biomarker Development for PI3K Pathway Therapeutics." Translational oncogenomics vol. 7 (2015): 33-49. doi: https://doi.org/10.4137/TOG.S30529
Josephs DH, Sarker D. Pharmacodynamic Biomarker Development for PI3K Pathway Therapeutics. Transl Oncogenomics. 2016 Feb 21;7:33-49. doi: 10.4137/TOG.S30529. eCollection 2015. PMID: 26917948; PMCID: PMC4762492.
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Correction: Disparities in outcomes of patients admitted with diabetic foot infections.

PloS one

Tan TW, Shih CD, Concha-Moore KC, Diri MM, Hu B, Marrero D, Zhou W, Armstrong DG.
PMID: 30973946
PLoS One. 2019 Apr 11;14(4):e0215532. doi: 10.1371/journal.pone.0215532. eCollection 2019.

[This corrects the article DOI: 10.1371/journal.pone.0211481.].

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Tan TW, Shih CD, Concha-Moore KC, et al. Correction: Disparities in outcomes of patients admitted with diabetic foot infections. PLoS One. 2019;14(4):e0215532doi: 10.1371/journal.pone.0215532.
Tan, T. W., Shih, C. D., Concha-Moore, K. C., Diri, M. M., Hu, B., Marrero, D., Zhou, W., & Armstrong, D. G. (2019). Correction: Disparities in outcomes of patients admitted with diabetic foot infections. PloS one, 14(4), e0215532. https://doi.org/10.1371/journal.pone.0215532
Tan, Tze-Woei, et al. "Correction: Disparities in outcomes of patients admitted with diabetic foot infections." PloS one vol. 14,4 (2019): e0215532. doi: https://doi.org/10.1371/journal.pone.0215532
Tan TW, Shih CD, Concha-Moore KC, Diri MM, Hu B, Marrero D, Zhou W, Armstrong DG. Correction: Disparities in outcomes of patients admitted with diabetic foot infections. PLoS One. 2019 Apr 11;14(4):e0215532. doi: 10.1371/journal.pone.0215532. eCollection 2019. PMID: 30973946; PMCID: PMC6459523.
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Can Insertion Sequences Proliferation Influence Genomic Plasticity? Comparative Analysis of .

Frontiers in microbiology

Gaiarsa S, Bitar I, Comandatore F, Corbella M, Piazza A, Scaltriti E, Villa L, Postiglione U, Marone P, Nucleo E, Pongolini S, Migliavacca R, Sassera D.
PMID: 31572316
Front Microbiol. 2019 Sep 12;10:2080. doi: 10.3389/fmicb.2019.02080. eCollection 2019.

No abstract available.

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Gaiarsa S, Bitar I, Comandatore F, et al. Can Insertion Sequences Proliferation Influence Genomic Plasticity? Comparative Analysis of . Front Microbiol. 2019;10:2080doi: 10.3389/fmicb.2019.02080.
Gaiarsa, S., Bitar, I., Comandatore, F., Corbella, M., Piazza, A., Scaltriti, E., Villa, L., Postiglione, U., Marone, P., Nucleo, E., Pongolini, S., Migliavacca, R., & Sassera, D. (2019). Can Insertion Sequences Proliferation Influence Genomic Plasticity? Comparative Analysis of . Frontiers in microbiology, 102080. https://doi.org/10.3389/fmicb.2019.02080
Gaiarsa, Stefano, et al. "Can Insertion Sequences Proliferation Influence Genomic Plasticity? Comparative Analysis of ." Frontiers in microbiology vol. 10 (2019): 2080. doi: https://doi.org/10.3389/fmicb.2019.02080
Gaiarsa S, Bitar I, Comandatore F, Corbella M, Piazza A, Scaltriti E, Villa L, Postiglione U, Marone P, Nucleo E, Pongolini S, Migliavacca R, Sassera D. Can Insertion Sequences Proliferation Influence Genomic Plasticity? Comparative Analysis of . Front Microbiol. 2019 Sep 12;10:2080. doi: 10.3389/fmicb.2019.02080. eCollection 2019. PMID: 31572316; PMCID: PMC6751323.
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Showing 1 to 12 of 110 entries