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Formation of noble-gas hydrides and decay of solvated protons revisited: diffusion-controlled reactions and hydrogen atom losses in solid noble gases.

Physical chemistry chemical physics : PCCP

Tanskanen H, Khriachtchev L, Lignell A, Räsänen M, Johansson S, Khyzhniy I, Savchenko E.
PMID: 19791453
Phys Chem Chem Phys. 2008 Feb 07;10(5):692-701. doi: 10.1039/b713212c.

UV photolysis and annealing of C2H2/Xe, C2H2/Xe/Kr, and HBr/Xe matrices lead to complicated photochemical processes and reactions. The dominating products in these experiments are noble-gas hydrides with general formula HNgY (Ng = noble-gas atom, Y = electronegative fragment). We...

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Tanskanen H, Khriachtchev L, Lignell A, et al. Formation of noble-gas hydrides and decay of solvated protons revisited: diffusion-controlled reactions and hydrogen atom losses in solid noble gases. Phys Chem Chem Phys. 2008;10(5):692-701doi: 10.1039/b713212c.
Tanskanen, H., Khriachtchev, L., Lignell, A., Räsänen, M., Johansson, S., Khyzhniy, I., & Savchenko, E. (2008). Formation of noble-gas hydrides and decay of solvated protons revisited: diffusion-controlled reactions and hydrogen atom losses in solid noble gases. Physical chemistry chemical physics : PCCP, 10(5), 692-701. https://doi.org/10.1039/b713212c
Tanskanen, Hanna, et al. "Formation of noble-gas hydrides and decay of solvated protons revisited: diffusion-controlled reactions and hydrogen atom losses in solid noble gases." Physical chemistry chemical physics : PCCP vol. 10,5 (2008): 692-701. doi: https://doi.org/10.1039/b713212c
Tanskanen H, Khriachtchev L, Lignell A, Räsänen M, Johansson S, Khyzhniy I, Savchenko E. Formation of noble-gas hydrides and decay of solvated protons revisited: diffusion-controlled reactions and hydrogen atom losses in solid noble gases. Phys Chem Chem Phys. 2008 Feb 07;10(5):692-701. doi: 10.1039/b713212c. PMID: 19791453.
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Design of Liver Functional Reserve Estimation Technique Based on Optical Densitometry.

Diagnostics (Basel, Switzerland)

Savchenko E, Kolokolnikov I, Velichko E, Osovskikh V, Kiseleva L, Musakulova Z.
PMID: 32824396
Diagnostics (Basel). 2020 Aug 16;10(8). doi: 10.3390/diagnostics10080599.

This work is aimed at creating a modified invasive technique for assessing the liver's functional reserves. A study of the degree of hepatodepression is carried out by measuring the plasma elimination of indocyanine green using the method of optical...

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Savchenko E, Kolokolnikov I, Velichko E, et al. Design of Liver Functional Reserve Estimation Technique Based on Optical Densitometry. Diagnostics (Basel). 2020;10(8)doi: 10.3390/diagnostics10080599.
Savchenko, E., Kolokolnikov, I., Velichko, E., Osovskikh, V., Kiseleva, L., & Musakulova, Z. (2020). Design of Liver Functional Reserve Estimation Technique Based on Optical Densitometry. Diagnostics (Basel, Switzerland), 10(8), . https://doi.org/10.3390/diagnostics10080599
Savchenko, Ekaterina, et al. "Design of Liver Functional Reserve Estimation Technique Based on Optical Densitometry." Diagnostics (Basel, Switzerland) vol. 10,8 (2020). doi: https://doi.org/10.3390/diagnostics10080599
Savchenko E, Kolokolnikov I, Velichko E, Osovskikh V, Kiseleva L, Musakulova Z. Design of Liver Functional Reserve Estimation Technique Based on Optical Densitometry. Diagnostics (Basel). 2020 Aug 16;10(8). doi: 10.3390/diagnostics10080599. PMID: 32824396; PMCID: PMC7460397.
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Creation of permanent lattice defects via exciton self-trapping into molecular states in Xe matrix.

Physical chemistry chemical physics : PCCP

Savchenko E, Ogurtsov A, Khyzhniy I, Stryganyuk G, Zimmerer G.
PMID: 19791362
Phys Chem Chem Phys. 2005 Mar 07;7(5):785-90. doi: 10.1039/b415247f.

Electronic excitation is shown to affect the structural properties of the Xe matrix at low temperatures. The lattice defect creation was studied using the selective vacuum ultraviolet (VUV) spectroscopy methods. The samples were excited by synchrotron radiation in the...

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Savchenko E, Ogurtsov A, Khyzhniy I, et al. Creation of permanent lattice defects via exciton self-trapping into molecular states in Xe matrix. Phys Chem Chem Phys. 2005;7(5):785-90doi: 10.1039/b415247f.
Savchenko, E., Ogurtsov, A., Khyzhniy, I., Stryganyuk, G., & Zimmerer, G. (2005). Creation of permanent lattice defects via exciton self-trapping into molecular states in Xe matrix. Physical chemistry chemical physics : PCCP, 7(5), 785-90. https://doi.org/10.1039/b415247f
Savchenko, Elena, et al. "Creation of permanent lattice defects via exciton self-trapping into molecular states in Xe matrix." Physical chemistry chemical physics : PCCP vol. 7,5 (2005): 785-90. doi: https://doi.org/10.1039/b415247f
Savchenko E, Ogurtsov A, Khyzhniy I, Stryganyuk G, Zimmerer G. Creation of permanent lattice defects via exciton self-trapping into molecular states in Xe matrix. Phys Chem Chem Phys. 2005 Mar 07;7(5):785-90. doi: 10.1039/b415247f. PMID: 19791362.
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Reprint of: policy decisions on endocrine disruptors should be based on science across disciplines: a response to Dietrich, et al.

Frontiers in neuroendocrinology

Gore AC, Balthazart J, Bikle D, Carpenter DO, Crews D, Czernichow P, Diamanti-Kandarakis E, Dores RM, Grattan D, Hof PR, Hollenberg AN, Lange C, Lee AV, Levine JE, Millar RP, Nelson RJ, Porta M, Poth M, Power DM, Prins GS, Ridgway EC, Rissman EF, Romijn JA, Sawchenko PE, Sly PD, Söder O, Taylor HS, Tena-Sempere M, Vaudry H, Wallen K, Wang Z, Wartofsky L, Watson CS.
PMID: 24268499
Front Neuroendocrinol. 2014 Jan;35(1):2-5. doi: 10.1016/j.yfrne.2013.09.003. Epub 2013 Nov 21.

No abstract available.

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Gore AC, Balthazart J, Bikle D, et al. Reprint of: policy decisions on endocrine disruptors should be based on science across disciplines: a response to Dietrich, et al. Front Neuroendocrinol. 2013;35(1):2-5doi: 10.1016/j.yfrne.2013.09.003.
Gore, A. C., Balthazart, J., Bikle, D., Carpenter, D. O., Crews, D., Czernichow, P., Diamanti-Kandarakis, E., Dores, R. M., Grattan, D., Hof, P. R., Hollenberg, A. N., Lange, C., Lee, A. V., Levine, J. E., Millar, R. P., Nelson, R. J., Porta, M., Poth, M., Power, D. M., Prins, G. S., Ridgway, E. C., Rissman, E. F., Romijn, J. A., Sawchenko, P. E., Sly, P. D., Söder, O., Taylor, H. S., Tena-Sempere, M., Vaudry, H., Wallen, K., Wang, Z., Wartofsky, L., & Watson, C. S. (2014). Reprint of: policy decisions on endocrine disruptors should be based on science across disciplines: a response to Dietrich, et al. Frontiers in neuroendocrinology, 35(1), 2-5. https://doi.org/10.1016/j.yfrne.2013.09.003
Gore, A C, et al. "Reprint of: policy decisions on endocrine disruptors should be based on science across disciplines: a response to Dietrich, et al." Frontiers in neuroendocrinology vol. 35,1 (2014): 2-5. doi: https://doi.org/10.1016/j.yfrne.2013.09.003
Gore AC, Balthazart J, Bikle D, Carpenter DO, Crews D, Czernichow P, Diamanti-Kandarakis E, Dores RM, Grattan D, Hof PR, Hollenberg AN, Lange C, Lee AV, Levine JE, Millar RP, Nelson RJ, Porta M, Poth M, Power DM, Prins GS, Ridgway EC, Rissman EF, Romijn JA, Sawchenko PE, Sly PD, Söder O, Taylor HS, Tena-Sempere M, Vaudry H, Wallen K, Wang Z, Wartofsky L, Watson CS. Reprint of: policy decisions on endocrine disruptors should be based on science across disciplines: a response to Dietrich, et al. Front Neuroendocrinol. 2014 Jan;35(1):2-5. doi: 10.1016/j.yfrne.2013.09.003. Epub 2013 Nov 21. PMID: 24268499.
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The intracellular milieu of Parkinson's disease patient brain cells modulates alpha-synuclein protein aggregation.

Acta neuropathologica communications

Gustavsson N, Savchenko E, Klementieva O, Roybon L.
PMID: 34530929
Acta Neuropathol Commun. 2021 Sep 16;9(1):153. doi: 10.1186/s40478-021-01256-w.

Recent studies suggest that brain cell type specific intracellular environments may play important roles in the generation of structurally different protein aggregates that define neurodegenerative diseases. Using human induced pluripotent stem cells (hiPSC) and biochemical and vibrational spectroscopy techniques,...

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Gustavsson N, Savchenko E, Klementieva O, et al. The intracellular milieu of Parkinson's disease patient brain cells modulates alpha-synuclein protein aggregation. Acta Neuropathol Commun. 2021;9(1):153doi: 10.1186/s40478-021-01256-w.
Gustavsson, N., Savchenko, E., Klementieva, O., & Roybon, L. (2021). The intracellular milieu of Parkinson's disease patient brain cells modulates alpha-synuclein protein aggregation. Acta neuropathologica communications, 9(1), 153. https://doi.org/10.1186/s40478-021-01256-w
Gustavsson, Nadja, et al. "The intracellular milieu of Parkinson's disease patient brain cells modulates alpha-synuclein protein aggregation." Acta neuropathologica communications vol. 9,1 (2021): 153. doi: https://doi.org/10.1186/s40478-021-01256-w
Gustavsson N, Savchenko E, Klementieva O, Roybon L. The intracellular milieu of Parkinson's disease patient brain cells modulates alpha-synuclein protein aggregation. Acta Neuropathol Commun. 2021 Sep 16;9(1):153. doi: 10.1186/s40478-021-01256-w. PMID: 34530929; PMCID: PMC8444604.
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Human iPSC-derived hippocampal spheroids: An innovative tool for stratifying Alzheimer disease patient-specific cellular phenotypes and developing therapies.

Stem cell reports

Pomeshchik Y, Klementieva O, Gil J, Martinsson I, Hansen MG, de Vries T, Sancho-Balsells A, Russ K, Savchenko E, Collin A, Vaz AR, Bagnoli S, Nacmias B, Rampon C, Sorbi S, Brites D, Marko-Varga G, Kokaia Z, Rezeli M, Gouras GK, Roybon L.
PMID: 34758331
Stem Cell Reports. 2021 Nov 09;16(11):2838. doi: 10.1016/j.stemcr.2021.10.003.

No abstract available.

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Pomeshchik Y, Klementieva O, Gil J, et al. Human iPSC-derived hippocampal spheroids: An innovative tool for stratifying Alzheimer disease patient-specific cellular phenotypes and developing therapies. Stem Cell Reports. 2021;16(11):2838doi: 10.1016/j.stemcr.2021.10.003.
Pomeshchik, Y., Klementieva, O., Gil, J., Martinsson, I., Hansen, M. G., de Vries, T., Sancho-Balsells, A., Russ, K., Savchenko, E., Collin, A., Vaz, A. R., Bagnoli, S., Nacmias, B., Rampon, C., Sorbi, S., Brites, D., Marko-Varga, G., Kokaia, Z., Rezeli, M., Gouras, G. K., & Roybon, L. (2021). Human iPSC-derived hippocampal spheroids: An innovative tool for stratifying Alzheimer disease patient-specific cellular phenotypes and developing therapies. Stem cell reports, 16(11), 2838. https://doi.org/10.1016/j.stemcr.2021.10.003
Pomeshchik, Yuriy, et al. "Human iPSC-derived hippocampal spheroids: An innovative tool for stratifying Alzheimer disease patient-specific cellular phenotypes and developing therapies." Stem cell reports vol. 16,11 (2021): 2838. doi: https://doi.org/10.1016/j.stemcr.2021.10.003
Pomeshchik Y, Klementieva O, Gil J, Martinsson I, Hansen MG, de Vries T, Sancho-Balsells A, Russ K, Savchenko E, Collin A, Vaz AR, Bagnoli S, Nacmias B, Rampon C, Sorbi S, Brites D, Marko-Varga G, Kokaia Z, Rezeli M, Gouras GK, Roybon L. Human iPSC-derived hippocampal spheroids: An innovative tool for stratifying Alzheimer disease patient-specific cellular phenotypes and developing therapies. Stem Cell Reports. 2021 Nov 09;16(11):2838. doi: 10.1016/j.stemcr.2021.10.003. PMID: 34758331; PMCID: PMC8581187.
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Long-term interleukin-33 treatment delays disease onset and alleviates astrocytic activation in a transgenic mouse model of amyotrophic lateral sclerosis.

IBRO reports

Korhonen P, Pollari E, Kanninen KM, Savchenko E, Lehtonen Š, Wojciechowski S, Pomeshchik Y, Van Den Bosch L, Goldsteins G, Koistinaho J, Malm T.
PMID: 30705990
IBRO Rep. 2019 Jan 11;6:74-86. doi: 10.1016/j.ibror.2019.01.005. eCollection 2019 Jun.

Inflammation is a prominent feature of the neuropathology of amyotrophic lateral sclerosis (ALS). Emerging evidence suggests that inflammatory cascades contributing to the disease progression are not restricted to the central nervous system (CNS) but also occur peripherally. Indeed, alterations...

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Korhonen P, Pollari E, Kanninen KM, et al. Long-term interleukin-33 treatment delays disease onset and alleviates astrocytic activation in a transgenic mouse model of amyotrophic lateral sclerosis. IBRO Rep. 2019;6:74-86doi: 10.1016/j.ibror.2019.01.005.
Korhonen, P., Pollari, E., Kanninen, K. M., Savchenko, E., Lehtonen, �. �., Wojciechowski, S., Pomeshchik, Y., Van Den Bosch, L., Goldsteins, G., Koistinaho, J., & Malm, T. (2019). Long-term interleukin-33 treatment delays disease onset and alleviates astrocytic activation in a transgenic mouse model of amyotrophic lateral sclerosis. IBRO reports, 674-86. https://doi.org/10.1016/j.ibror.2019.01.005
Korhonen, Paula, et al. "Long-term interleukin-33 treatment delays disease onset and alleviates astrocytic activation in a transgenic mouse model of amyotrophic lateral sclerosis." IBRO reports vol. 6 (2019): 74-86. doi: https://doi.org/10.1016/j.ibror.2019.01.005
Korhonen P, Pollari E, Kanninen KM, Savchenko E, Lehtonen Š, Wojciechowski S, Pomeshchik Y, Van Den Bosch L, Goldsteins G, Koistinaho J, Malm T. Long-term interleukin-33 treatment delays disease onset and alleviates astrocytic activation in a transgenic mouse model of amyotrophic lateral sclerosis. IBRO Rep. 2019 Jan 11;6:74-86. doi: 10.1016/j.ibror.2019.01.005. eCollection 2019 Jun. PMID: 30705990; PMCID: PMC6348738.
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Infrared absorption spectrum of matrix-isolated noble-gas hydride molecules: fingerprints of specific interactions and hindered rotation.

The Journal of chemical physics

Khriachtchev L, Lignell A, Juselius J, Räsänen M, Savchenko E.
PMID: 15638677
J Chem Phys. 2005 Jan 01;122(1):14510. doi: 10.1063/1.1827592.

Noble-gas hydride molecules with the general formula HNgY (Ng denotes noble-gas atom and Y denotes electronegative fragment) are usually prepared in solid noble gases. In many cases, the matrix-isolated HNgY molecules show a characteristic structure of the H-Ng stretching...

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Khriachtchev L, Lignell A, Juselius J, et al. Infrared absorption spectrum of matrix-isolated noble-gas hydride molecules: fingerprints of specific interactions and hindered rotation. J Chem Phys. 2005;122(1):14510doi: 10.1063/1.1827592.
Khriachtchev, L., Lignell, A., Juselius, J., Räsänen, M., & Savchenko, E. (2005). Infrared absorption spectrum of matrix-isolated noble-gas hydride molecules: fingerprints of specific interactions and hindered rotation. The Journal of chemical physics, 122(1), 14510. https://doi.org/10.1063/1.1827592
Khriachtchev, Leonid, et al. "Infrared absorption spectrum of matrix-isolated noble-gas hydride molecules: fingerprints of specific interactions and hindered rotation." The Journal of chemical physics vol. 122,1 (2005): 14510. doi: https://doi.org/10.1063/1.1827592
Khriachtchev L, Lignell A, Juselius J, Räsänen M, Savchenko E. Infrared absorption spectrum of matrix-isolated noble-gas hydride molecules: fingerprints of specific interactions and hindered rotation. J Chem Phys. 2005 Jan 01;122(1):14510. doi: 10.1063/1.1827592. PMID: 15638677.
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Aβ and Inflammatory Stimulus Activate Diverse Signaling Pathways in Monocytic Cells: Implications in Retaining Phagocytosis in Aβ-Laden Environment.

Frontiers in cellular neuroscience

Savchenko E, Malm T, Konttinen H, Hämäläinen RH, Guerrero-Toro C, Wojciechowski S, Giniatullin R, Koistinaho J, Magga J.
PMID: 27994540
Front Cell Neurosci. 2016 Dec 05;10:279. doi: 10.3389/fncel.2016.00279. eCollection 2016.

No abstract available.

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Savchenko E, Malm T, Konttinen H, et al. Aβ and Inflammatory Stimulus Activate Diverse Signaling Pathways in Monocytic Cells: Implications in Retaining Phagocytosis in Aβ-Laden Environment. Front Cell Neurosci. 2016;10:279doi: 10.3389/fncel.2016.00279.
Savchenko, E., Malm, T., Konttinen, H., Hämäläinen, R. H., Guerrero-Toro, C., Wojciechowski, S., Giniatullin, R., Koistinaho, J., & Magga, J. (2016). Aβ and Inflammatory Stimulus Activate Diverse Signaling Pathways in Monocytic Cells: Implications in Retaining Phagocytosis in Aβ-Laden Environment. Frontiers in cellular neuroscience, 10279. https://doi.org/10.3389/fncel.2016.00279
Savchenko, Ekaterina, et al. "Aβ and Inflammatory Stimulus Activate Diverse Signaling Pathways in Monocytic Cells: Implications in Retaining Phagocytosis in Aβ-Laden Environment." Frontiers in cellular neuroscience vol. 10 (2016): 279. doi: https://doi.org/10.3389/fncel.2016.00279
Savchenko E, Malm T, Konttinen H, Hämäläinen RH, Guerrero-Toro C, Wojciechowski S, Giniatullin R, Koistinaho J, Magga J. Aβ and Inflammatory Stimulus Activate Diverse Signaling Pathways in Monocytic Cells: Implications in Retaining Phagocytosis in Aβ-Laden Environment. Front Cell Neurosci. 2016 Dec 05;10:279. doi: 10.3389/fncel.2016.00279. eCollection 2016. PMID: 27994540; PMCID: PMC5136556.
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Novel hypothalamic and preoptic sites of prepro-melanin-concentrating hormone messenger ribonucleic Acid and Peptide expression in lactating rats.

Journal of neuroendocrinology

Knollema S, Brown ER, Vale W, Sawchenko PE.
PMID: 21554658
J Neuroendocrinol. 1992 Dec;4(6):709-17. doi: 10.1111/j.1365-2826.1992.tb00222.x.

The deduced structure of the rat melanin-concentrating hormone (MCH) precursor predicted the existence of at least two peptides that may be processed from it, one similar to teleost MCH and a second novel neuropeptide, NEI. Cellular localization studies confirmed...

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Knollema S, Brown ER, Vale W, et al. Novel hypothalamic and preoptic sites of prepro-melanin-concentrating hormone messenger ribonucleic Acid and Peptide expression in lactating rats. J Neuroendocrinol. 1992;4(6):709-17doi: 10.1111/j.1365-2826.1992.tb00222.x.
Knollema, S., Brown, E. R., Vale, W., & Sawchenko, P. E. (1992). Novel hypothalamic and preoptic sites of prepro-melanin-concentrating hormone messenger ribonucleic Acid and Peptide expression in lactating rats. Journal of neuroendocrinology, 4(6), 709-17. https://doi.org/10.1111/j.1365-2826.1992.tb00222.x
Knollema, S, et al. "Novel hypothalamic and preoptic sites of prepro-melanin-concentrating hormone messenger ribonucleic Acid and Peptide expression in lactating rats." Journal of neuroendocrinology vol. 4,6 (1992): 709-17. doi: https://doi.org/10.1111/j.1365-2826.1992.tb00222.x
Knollema S, Brown ER, Vale W, Sawchenko PE. Novel hypothalamic and preoptic sites of prepro-melanin-concentrating hormone messenger ribonucleic Acid and Peptide expression in lactating rats. J Neuroendocrinol. 1992 Dec;4(6):709-17. doi: 10.1111/j.1365-2826.1992.tb00222.x. PMID: 21554658.
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Synapsin-caveolin-1 gene therapy preserves neuronal and synaptic morphology and prevents neurodegeneration in a mouse model of AD.

Molecular therapy. Methods & clinical development

Wang S, Leem JS, Podvin S, Hook V, Kleschevnikov N, Savchenko P, Dhanani M, Zhou K, Kelly IC, Zhang T, Miyanohara A, Nguyen P, Kleschevnikov A, Wagner SL, Trojanowski JQ, Roth DM, Patel HH, Patel PM, Head BP.
PMID: 33981778
Mol Ther Methods Clin Dev. 2021 Mar 29;21:434-450. doi: 10.1016/j.omtm.2021.03.021. eCollection 2021 Jun 11.

Alzheimer's disease (AD) is the most common form of neurodegeneration and cognitive dysfunction in the elderly. Identifying molecular signals that mitigate and reverse neurodegeneration in AD may be exploited therapeutically. Transgenic AD mice (PSAPP) exhibit learning and memory deficits...

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Wang S, Leem JS, Podvin S, et al. Synapsin-caveolin-1 gene therapy preserves neuronal and synaptic morphology and prevents neurodegeneration in a mouse model of AD. Mol Ther Methods Clin Dev. 2021;21:434-450doi: 10.1016/j.omtm.2021.03.021.
Wang, S., Leem, J. S., Podvin, S., Hook, V., Kleschevnikov, N., Savchenko, P., Dhanani, M., Zhou, K., Kelly, I. C., Zhang, T., Miyanohara, A., Nguyen, P., Kleschevnikov, A., Wagner, S. L., Trojanowski, J. Q., Roth, D. M., Patel, H. H., Patel, P. M., & Head, B. P. (2021). Synapsin-caveolin-1 gene therapy preserves neuronal and synaptic morphology and prevents neurodegeneration in a mouse model of AD. Molecular therapy. Methods & clinical development, 21434-450. https://doi.org/10.1016/j.omtm.2021.03.021
Wang, Shanshan, et al. "Synapsin-caveolin-1 gene therapy preserves neuronal and synaptic morphology and prevents neurodegeneration in a mouse model of AD." Molecular therapy. Methods & clinical development vol. 21 (2021): 434-450. doi: https://doi.org/10.1016/j.omtm.2021.03.021
Wang S, Leem JS, Podvin S, Hook V, Kleschevnikov N, Savchenko P, Dhanani M, Zhou K, Kelly IC, Zhang T, Miyanohara A, Nguyen P, Kleschevnikov A, Wagner SL, Trojanowski JQ, Roth DM, Patel HH, Patel PM, Head BP. Synapsin-caveolin-1 gene therapy preserves neuronal and synaptic morphology and prevents neurodegeneration in a mouse model of AD. Mol Ther Methods Clin Dev. 2021 Mar 29;21:434-450. doi: 10.1016/j.omtm.2021.03.021. eCollection 2021 Jun 11. PMID: 33981778; PMCID: PMC8065227.
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TNF-α and α-synuclein fibrils differently regulate human astrocyte immune reactivity and impair mitochondrial respiration.

Cell reports

Russ K, Teku G, Bousset L, Redeker V, Piel S, Savchenko E, Pomeshchik Y, Savistchenko J, Stummann TC, Azevedo C, Collin A, Goldwurm S, Fog K, Elmer E, Vihinen M, Melki R, Roybon L.
PMID: 33761362
Cell Rep. 2021 Mar 23;34(12):108895. doi: 10.1016/j.celrep.2021.108895.

Here, we examine the cellular changes triggered by tumor necrosis factor alpha (TNF-α) and different alpha-synuclein (αSYN) species in astrocytes derived from induced pluripotent stem cells. Human astrocytes treated with TNF-α display a strong reactive pro-inflammatory phenotype with upregulation...

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Russ K, Teku G, Bousset L, et al. TNF-α and α-synuclein fibrils differently regulate human astrocyte immune reactivity and impair mitochondrial respiration. Cell Rep. 2021;34(12):108895doi: 10.1016/j.celrep.2021.108895.
Russ, K., Teku, G., Bousset, L., Redeker, V., Piel, S., Savchenko, E., Pomeshchik, Y., Savistchenko, J., Stummann, T. C., Azevedo, C., Collin, A., Goldwurm, S., Fog, K., Elmer, E., Vihinen, M., Melki, R., & Roybon, L. (2021). TNF-α and α-synuclein fibrils differently regulate human astrocyte immune reactivity and impair mitochondrial respiration. Cell reports, 34(12), 108895. https://doi.org/10.1016/j.celrep.2021.108895
Russ, Kaspar, et al. "TNF-α and α-synuclein fibrils differently regulate human astrocyte immune reactivity and impair mitochondrial respiration." Cell reports vol. 34,12 (2021): 108895. doi: https://doi.org/10.1016/j.celrep.2021.108895
Russ K, Teku G, Bousset L, Redeker V, Piel S, Savchenko E, Pomeshchik Y, Savistchenko J, Stummann TC, Azevedo C, Collin A, Goldwurm S, Fog K, Elmer E, Vihinen M, Melki R, Roybon L. TNF-α and α-synuclein fibrils differently regulate human astrocyte immune reactivity and impair mitochondrial respiration. Cell Rep. 2021 Mar 23;34(12):108895. doi: 10.1016/j.celrep.2021.108895. PMID: 33761362.
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