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Management of Crises and Risks in German Hospitals - Factors Influencing Medication Errors.

Value in health : the journal of the International Society for Pharmacoeconomics and Outcomes Research

Draheim M, Flessa S, Glasberg R, Hartmann M, Schermuly C, Stanchev V, Tamm G, Hessel FP.
PMID: 27201097
Value Health. 2014 Nov;17(7):A426. doi: 10.1016/j.jval.2014.08.1067. Epub 2014 Oct 26.

No abstract available.

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Draheim M, Flessa S, Glasberg R, et al. Management of Crises and Risks in German Hospitals - Factors Influencing Medication Errors. Value Health. 2014;17(7):A426doi: 10.1016/j.jval.2014.08.1067.
Draheim, M., Flessa, S., Glasberg, R., Hartmann, M., Schermuly, C., Stanchev, V., Tamm, G., & Hessel, F. P. (2014). Management of Crises and Risks in German Hospitals - Factors Influencing Medication Errors. Value in health : the journal of the International Society for Pharmacoeconomics and Outcomes Research, 17(7), A426. https://doi.org/10.1016/j.jval.2014.08.1067
Draheim, M, et al. "Management of Crises and Risks in German Hospitals - Factors Influencing Medication Errors." Value in health : the journal of the International Society for Pharmacoeconomics and Outcomes Research vol. 17,7 (2014): A426. doi: https://doi.org/10.1016/j.jval.2014.08.1067
Draheim M, Flessa S, Glasberg R, Hartmann M, Schermuly C, Stanchev V, Tamm G, Hessel FP. Management of Crises and Risks in German Hospitals - Factors Influencing Medication Errors. Value Health. 2014 Nov;17(7):A426. doi: 10.1016/j.jval.2014.08.1067. Epub 2014 Oct 26. PMID: 27201097.
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Rhodiola: an ordinary plant or a promising future therapy for pulmonary hypertension? a brief review.

Pulmonary circulation

Kosanovic D, Tian X, Pak O, Lai YJ, Hsieh YL, Seimetz M, Weissmann N, Schermuly RT, Dahal BK.
PMID: 24618536
Pulm Circ. 2013 Sep;3(3):499-506. doi: 10.1086/674303. Epub 2013 Nov 11.

Abstract Pulmonary hypertension (PH) is a chronic, complex, and progressive disease that eventuates in fatality. Research efforts over the past decades have resulted in therapeutic options that improve quality of life and prolong survival of patients, but they do...

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Kosanovic D, Tian X, Pak O, et al. Rhodiola: an ordinary plant or a promising future therapy for pulmonary hypertension? a brief review. Pulm Circ. 2013;3(3):499-506doi: 10.1086/674303.
Kosanovic, D., Tian, X., Pak, O., Lai, Y. J., Hsieh, Y. L., Seimetz, M., Weissmann, N., Schermuly, R. T., & Dahal, B. K. (2013). Rhodiola: an ordinary plant or a promising future therapy for pulmonary hypertension? a brief review. Pulmonary circulation, 3(3), 499-506. https://doi.org/10.1086/674303
Kosanovic, Djuro, et al. "Rhodiola: an ordinary plant or a promising future therapy for pulmonary hypertension? a brief review." Pulmonary circulation vol. 3,3 (2013): 499-506. doi: https://doi.org/10.1086/674303
Kosanovic D, Tian X, Pak O, Lai YJ, Hsieh YL, Seimetz M, Weissmann N, Schermuly RT, Dahal BK. Rhodiola: an ordinary plant or a promising future therapy for pulmonary hypertension? a brief review. Pulm Circ. 2013 Sep;3(3):499-506. doi: 10.1086/674303. Epub 2013 Nov 11. PMID: 24618536; PMCID: PMC4070792.
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Oxidative injury of the pulmonary circulation in the perinatal period: Short- and long-term consequences for the human cardiopulmonary system.

Pulmonary circulation

de Wijs-Meijler DP, Duncker DJ, Tibboel D, Schermuly RT, Weissmann N, Merkus D, Reiss IKM.
PMID: 28680565
Pulm Circ. 2017 Jan 01;7(1):55-66. doi: 10.1086/689748. eCollection 2017 Mar.

Development of the pulmonary circulation is a complex process with a spatial pattern that is tightly controlled. This process is vulnerable for disruption by various events in the prenatal and early postnatal periods. Disruption of normal pulmonary vascular development...

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de Wijs-Meijler DP, Duncker DJ, Tibboel D, et al. Oxidative injury of the pulmonary circulation in the perinatal period: Short- and long-term consequences for the human cardiopulmonary system. Pulm Circ. 2017;7(1):55-66doi: 10.1086/689748.
de Wijs-Meijler, D. P., Duncker, D. J., Tibboel, D., Schermuly, R. T., Weissmann, N., Merkus, D., & Reiss, I. K. M. (2017). Oxidative injury of the pulmonary circulation in the perinatal period: Short- and long-term consequences for the human cardiopulmonary system. Pulmonary circulation, 7(1), 55-66. https://doi.org/10.1086/689748
de Wijs-Meijler, Daphne P, et al. "Oxidative injury of the pulmonary circulation in the perinatal period: Short- and long-term consequences for the human cardiopulmonary system." Pulmonary circulation vol. 7,1 (2017): 55-66. doi: https://doi.org/10.1086/689748
de Wijs-Meijler DP, Duncker DJ, Tibboel D, Schermuly RT, Weissmann N, Merkus D, Reiss IKM. Oxidative injury of the pulmonary circulation in the perinatal period: Short- and long-term consequences for the human cardiopulmonary system. Pulm Circ. 2017 Jan 01;7(1):55-66. doi: 10.1086/689748. eCollection 2017 Mar. PMID: 28680565; PMCID: PMC5448552.
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Altered fibrin clot structure and dysregulated fibrinolysis contribute to thrombosis risk in severe COVID-19.

Blood advances

Wygrecka M, Birnhuber A, Seeliger B, Michalick L, Pak O, Schultz AS, Schramm F, Zacharias M, Gorkiewicz G, David S, Welte T, Schmidt JJ, Weissmann N, Schermuly RT, Barreto G, Schaefer L, Markart P, Brack MC, Hippenstiel S, Kurth F, Sander L, Witzenrath M, Kuebler W, Kwapiszewska G, Preissner KT.
PMID: 34861681
Blood Adv. 2021 Dec 03; doi: 10.1182/bloodadvances.2021004816. Epub 2021 Dec 03.

The high incidence of thrombotic events suggests a possible role of the contact system pathway in COVID-19 pathology. Here, we demonstrate altered levels of factor XII (FXII) and its activation products in critically ill COVID-19 patients in comparison to...

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Wygrecka M, Birnhuber A, Seeliger B, et al. Altered fibrin clot structure and dysregulated fibrinolysis contribute to thrombosis risk in severe COVID-19. Blood Adv. 2021;doi: 10.1182/bloodadvances.2021004816.
Wygrecka, M., Birnhuber, A., Seeliger, B., Michalick, L., Pak, O., Schultz, A. S., Schramm, F., Zacharias, M., Gorkiewicz, G., David, S., Welte, T., Schmidt, J. J., Weissmann, N., Schermuly, R. T., Barreto, G., Schaefer, L., Markart, P., Brack, M. C., Hippenstiel, S., Kurth, F., Sander, L., Witzenrath, M., Kuebler, W., Kwapiszewska, G., & Preissner, K. T. (2021). Altered fibrin clot structure and dysregulated fibrinolysis contribute to thrombosis risk in severe COVID-19. Blood advances, . https://doi.org/10.1182/bloodadvances.2021004816
Wygrecka, Malgorzata, et al. "Altered fibrin clot structure and dysregulated fibrinolysis contribute to thrombosis risk in severe COVID-19." Blood advances vol. (2021). doi: https://doi.org/10.1182/bloodadvances.2021004816
Wygrecka M, Birnhuber A, Seeliger B, Michalick L, Pak O, Schultz AS, Schramm F, Zacharias M, Gorkiewicz G, David S, Welte T, Schmidt JJ, Weissmann N, Schermuly RT, Barreto G, Schaefer L, Markart P, Brack MC, Hippenstiel S, Kurth F, Sander L, Witzenrath M, Kuebler W, Kwapiszewska G, Preissner KT. Altered fibrin clot structure and dysregulated fibrinolysis contribute to thrombosis risk in severe COVID-19. Blood Adv. 2021 Dec 03; doi: 10.1182/bloodadvances.2021004816. Epub 2021 Dec 03. PMID: 34861681; PMCID: PMC8648369.
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Correction to: Phosphodiesterase 6 subunits are expressed and altered in idiopathic pulmonary fibrosis.

Respiratory research

Nikolova S, Guenther A, Savai R, Weissmann N, Ghofrani HA, Konigshoff M, Eickelberg O, Klepetko W, Voswinckel R, Seeger W, Grimminger F, Schermuly RT, Pullamsetti SS.
PMID: 34991577
Respir Res. 2022 Jan 06;23(1):2. doi: 10.1186/s12931-021-01907-5.

No abstract available.

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Nikolova S, Guenther A, Savai R, et al. Correction to: Phosphodiesterase 6 subunits are expressed and altered in idiopathic pulmonary fibrosis. Respir Res. 2022;23(1):2doi: 10.1186/s12931-021-01907-5.
Nikolova, S., Guenther, A., Savai, R., Weissmann, N., Ghofrani, H. A., Konigshoff, M., Eickelberg, O., Klepetko, W., Voswinckel, R., Seeger, W., Grimminger, F., Schermuly, R. T., & Pullamsetti, S. S. (2022). Correction to: Phosphodiesterase 6 subunits are expressed and altered in idiopathic pulmonary fibrosis. Respiratory research, 23(1), 2. https://doi.org/10.1186/s12931-021-01907-5
Nikolova, Sevdalina, et al. "Correction to: Phosphodiesterase 6 subunits are expressed and altered in idiopathic pulmonary fibrosis." Respiratory research vol. 23,1 (2022): 2. doi: https://doi.org/10.1186/s12931-021-01907-5
Nikolova S, Guenther A, Savai R, Weissmann N, Ghofrani HA, Konigshoff M, Eickelberg O, Klepetko W, Voswinckel R, Seeger W, Grimminger F, Schermuly RT, Pullamsetti SS. Correction to: Phosphodiesterase 6 subunits are expressed and altered in idiopathic pulmonary fibrosis. Respir Res. 2022 Jan 06;23(1):2. doi: 10.1186/s12931-021-01907-5. PMID: 34991577; PMCID: PMC8739988.
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Natural plant products in treatment of pulmonary arterial hypertension.

Pulmonary circulation

Xiang L, Li Y, Deng X, Kosanovic D, Schermuly RT, Li X.
PMID: 29869936
Pulm Circ. 2018 Jul-Sep;8(3):2045894018784033. doi: 10.1177/2045894018784033. Epub 2018 Jun 05.

Pulmonary arterial hypertension (PAH) is a severe disease characterized by progressive remodeling of distal pulmonary arteries and persistent elevation of pulmonary vascular resistance (PVR), which leads to right ventricular dysfunction, heart failure, and eventually death. Although treatment responsiveness for...

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Xiang L, Li Y, Deng X, et al. Natural plant products in treatment of pulmonary arterial hypertension. Pulm Circ. 2018;8(3):2045894018784033doi: 10.1177/2045894018784033.
Xiang, L., Li, Y., Deng, X., Kosanovic, D., Schermuly, R. T., & Li, X. (2018). Natural plant products in treatment of pulmonary arterial hypertension. Pulmonary circulation, 8(3), 2045894018784033. https://doi.org/10.1177/2045894018784033
Xiang, Lili, et al. "Natural plant products in treatment of pulmonary arterial hypertension." Pulmonary circulation vol. 8,3 (2018): 2045894018784033. doi: https://doi.org/10.1177/2045894018784033
Xiang L, Li Y, Deng X, Kosanovic D, Schermuly RT, Li X. Natural plant products in treatment of pulmonary arterial hypertension. Pulm Circ. 2018 Jul-Sep;8(3):2045894018784033. doi: 10.1177/2045894018784033. Epub 2018 Jun 05. PMID: 29869936; PMCID: PMC6055327.
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Orotracheal treprostinil administration attenuates bleomycin-induced lung injury, vascular remodeling, and fibrosis in mice.

Pulmonary circulation

Nikitopoulou I, Manitsopoulos N, Kotanidou A, Tian X, Petrovic A, Magkou C, Ninou I, Aidinis V, Schermuly RT, Kosanovic D, Orfanos SE.
PMID: 31819797
Pulm Circ. 2019 Nov 15;9(4):2045894019881954. doi: 10.1177/2045894019881954. eCollection 2019.

Pulmonary fibrosis is a progressive disease characterized by disruption of lung architecture and deregulation of the pulmonary function. Prostacyclin, a metabolite of arachidonic acid, is a potential disease mediator since it exerts anti-inflammatory and anti-fibrotic actions. We investigated the...

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Nikitopoulou I, Manitsopoulos N, Kotanidou A, et al. Orotracheal treprostinil administration attenuates bleomycin-induced lung injury, vascular remodeling, and fibrosis in mice. Pulm Circ. 2019;9(4):2045894019881954doi: 10.1177/2045894019881954.
Nikitopoulou, I., Manitsopoulos, N., Kotanidou, A., Tian, X., Petrovic, A., Magkou, C., Ninou, I., Aidinis, V., Schermuly, R. T., Kosanovic, D., & Orfanos, S. E. (2019). Orotracheal treprostinil administration attenuates bleomycin-induced lung injury, vascular remodeling, and fibrosis in mice. Pulmonary circulation, 9(4), 2045894019881954. https://doi.org/10.1177/2045894019881954
Nikitopoulou, Ioanna, et al. "Orotracheal treprostinil administration attenuates bleomycin-induced lung injury, vascular remodeling, and fibrosis in mice." Pulmonary circulation vol. 9,4 (2019): 2045894019881954. doi: https://doi.org/10.1177/2045894019881954
Nikitopoulou I, Manitsopoulos N, Kotanidou A, Tian X, Petrovic A, Magkou C, Ninou I, Aidinis V, Schermuly RT, Kosanovic D, Orfanos SE. Orotracheal treprostinil administration attenuates bleomycin-induced lung injury, vascular remodeling, and fibrosis in mice. Pulm Circ. 2019 Nov 15;9(4):2045894019881954. doi: 10.1177/2045894019881954. eCollection 2019. PMID: 31819797; PMCID: PMC6883672.
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Enhanced inflammatory cell profiles in schistosomiasis-induced pulmonary vascular remodeling.

Pulmonary circulation

Ali Z, Kosanovic D, Kolosionek E, Schermuly RT, Graham BB, Mathie A, Butrous G.
PMID: 28680583
Pulm Circ. 2017 Apr 04;7(1):244-252. doi: 10.1086/690687. eCollection 2017 Mar.

Schistosomiasis (bilharzia) is a neglected parasitic disease caused by trematode flatworms of the genus

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Ali Z, Kosanovic D, Kolosionek E, et al. Enhanced inflammatory cell profiles in schistosomiasis-induced pulmonary vascular remodeling. Pulm Circ. 2017;7(1):244-252doi: 10.1086/690687.
Ali, Z., Kosanovic, D., Kolosionek, E., Schermuly, R. T., Graham, B. B., Mathie, A., & Butrous, G. (2017). Enhanced inflammatory cell profiles in schistosomiasis-induced pulmonary vascular remodeling. Pulmonary circulation, 7(1), 244-252. https://doi.org/10.1086/690687
Ali, Zahara, et al. "Enhanced inflammatory cell profiles in schistosomiasis-induced pulmonary vascular remodeling." Pulmonary circulation vol. 7,1 (2017): 244-252. doi: https://doi.org/10.1086/690687
Ali Z, Kosanovic D, Kolosionek E, Schermuly RT, Graham BB, Mathie A, Butrous G. Enhanced inflammatory cell profiles in schistosomiasis-induced pulmonary vascular remodeling. Pulm Circ. 2017 Apr 04;7(1):244-252. doi: 10.1086/690687. eCollection 2017 Mar. PMID: 28680583; PMCID: PMC5448541.
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Circulating Apoptotic Signals During Acute and Chronic Exposure to High Altitude in Kyrgyz Population.

Frontiers in physiology

Kosanovic D, Platzek SM, Petrovic A, Sydykov A, Maripov A, Mamazhakypov A, Sartmyrzaeva M, Muratali Uulu K, Cholponbaeva M, Toktosunova A, Omurzakova N, Duishobaev M, Vroom C, Pak O, Weissmann N, Ghofrani HA, Sarybaev A, Schermuly RT.
PMID: 30804801
Front Physiol. 2019 Feb 05;10:54. doi: 10.3389/fphys.2019.00054. eCollection 2019.

No abstract available.

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Kosanovic D, Platzek SM, Petrovic A, et al. Circulating Apoptotic Signals During Acute and Chronic Exposure to High Altitude in Kyrgyz Population. Front Physiol. 2019;10:54doi: 10.3389/fphys.2019.00054.
Kosanovic, D., Platzek, S. M., Petrovic, A., Sydykov, A., Maripov, A., Mamazhakypov, A., Sartmyrzaeva, M., Muratali Uulu, K., Cholponbaeva, M., Toktosunova, A., Omurzakova, N., Duishobaev, M., Vroom, C., Pak, O., Weissmann, N., Ghofrani, H. A., Sarybaev, A., & Schermuly, R. T. (2019). Circulating Apoptotic Signals During Acute and Chronic Exposure to High Altitude in Kyrgyz Population. Frontiers in physiology, 1054. https://doi.org/10.3389/fphys.2019.00054
Kosanovic, Djuro, et al. "Circulating Apoptotic Signals During Acute and Chronic Exposure to High Altitude in Kyrgyz Population." Frontiers in physiology vol. 10 (2019): 54. doi: https://doi.org/10.3389/fphys.2019.00054
Kosanovic D, Platzek SM, Petrovic A, Sydykov A, Maripov A, Mamazhakypov A, Sartmyrzaeva M, Muratali Uulu K, Cholponbaeva M, Toktosunova A, Omurzakova N, Duishobaev M, Vroom C, Pak O, Weissmann N, Ghofrani HA, Sarybaev A, Schermuly RT. Circulating Apoptotic Signals During Acute and Chronic Exposure to High Altitude in Kyrgyz Population. Front Physiol. 2019 Feb 05;10:54. doi: 10.3389/fphys.2019.00054. eCollection 2019. PMID: 30804801; PMCID: PMC6370645.
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Short-term improvement in pulmonary hemodynamics is strongly predictive of long-term survival in patients with pulmonary arterial hypertension.

Pulmonary circulation

Tiede H, Sommer N, Milger K, Voswinckel R, Bandorski D, Schermuly RT, Weissmann N, Grimminger F, Seeger W, Ghofrani HA.
PMID: 24618538
Pulm Circ. 2013 Sep;3(3):523-32. doi: 10.1086/674338. Epub 2013 Nov 19.

Abstract Hemodynamic measurements provide important parameters for determining prognosis and therapy in patients with pulmonary arterial hypertension (PAH). Current guidelines do not incorporate the possible predictive value of individual changes in hemodynamic variables during the disease time course, and...

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Tiede H, Sommer N, Milger K, et al. Short-term improvement in pulmonary hemodynamics is strongly predictive of long-term survival in patients with pulmonary arterial hypertension. Pulm Circ. 2013;3(3):523-32doi: 10.1086/674338.
Tiede, H., Sommer, N., Milger, K., Voswinckel, R., Bandorski, D., Schermuly, R. T., Weissmann, N., Grimminger, F., Seeger, W., & Ghofrani, H. A. (2013). Short-term improvement in pulmonary hemodynamics is strongly predictive of long-term survival in patients with pulmonary arterial hypertension. Pulmonary circulation, 3(3), 523-32. https://doi.org/10.1086/674338
Tiede, Henning, et al. "Short-term improvement in pulmonary hemodynamics is strongly predictive of long-term survival in patients with pulmonary arterial hypertension." Pulmonary circulation vol. 3,3 (2013): 523-32. doi: https://doi.org/10.1086/674338
Tiede H, Sommer N, Milger K, Voswinckel R, Bandorski D, Schermuly RT, Weissmann N, Grimminger F, Seeger W, Ghofrani HA. Short-term improvement in pulmonary hemodynamics is strongly predictive of long-term survival in patients with pulmonary arterial hypertension. Pulm Circ. 2013 Sep;3(3):523-32. doi: 10.1086/674338. Epub 2013 Nov 19. PMID: 24618538; PMCID: PMC4070816.
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Elevated protein arginine methyltransferase 1 expression regulates fibroblast motility in pulmonary fibrosis.

Biochimica et biophysica acta

Zakrzewicz D, Zakrzewicz A, Didiasova M, Korencak M, Kosanovic D, Schermuly RT, Markart P, Wygrecka M.
PMID: 26391253
Biochim Biophys Acta. 2015 Dec;1852(12):2678-88. doi: 10.1016/j.bbadis.2015.09.008. Epub 2015 Sep 21.

OBJECTIVE: Idiopathic pulmonary fibrosis (IPF) is a devastating disease characterized by epithelial cell injury, fibroblast activation and excessive extracellular matrix deposition. Although protein arginine methyltransferase 1 (PRMT1) was found to regulate cell proliferation, differentiation and migration, its role in...

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Zakrzewicz D, Zakrzewicz A, Didiasova M, et al. Elevated protein arginine methyltransferase 1 expression regulates fibroblast motility in pulmonary fibrosis. Biochim Biophys Acta. 2015;1852(12):2678-88doi: 10.1016/j.bbadis.2015.09.008.
Zakrzewicz, D., Zakrzewicz, A., Didiasova, M., Korencak, M., Kosanovic, D., Schermuly, R. T., Markart, P., & Wygrecka, M. (2015). Elevated protein arginine methyltransferase 1 expression regulates fibroblast motility in pulmonary fibrosis. Biochimica et biophysica acta, 1852(12), 2678-88. https://doi.org/10.1016/j.bbadis.2015.09.008
Zakrzewicz, Dariusz, et al. "Elevated protein arginine methyltransferase 1 expression regulates fibroblast motility in pulmonary fibrosis." Biochimica et biophysica acta vol. 1852,12 (2015): 2678-88. doi: https://doi.org/10.1016/j.bbadis.2015.09.008
Zakrzewicz D, Zakrzewicz A, Didiasova M, Korencak M, Kosanovic D, Schermuly RT, Markart P, Wygrecka M. Elevated protein arginine methyltransferase 1 expression regulates fibroblast motility in pulmonary fibrosis. Biochim Biophys Acta. 2015 Dec;1852(12):2678-88. doi: 10.1016/j.bbadis.2015.09.008. Epub 2015 Sep 21. PMID: 26391253.
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Lysine methyltransferase Smyd2 suppresses p53-dependent cardiomyocyte apoptosis.

Biochimica et biophysica acta

Sajjad A, Novoyatleva T, Vergarajauregui S, Troidl C, Schermuly RT, Tucker HO, Engel FB.
PMID: 25014164
Biochim Biophys Acta. 2014 Nov;1843(11):2556-62. doi: 10.1016/j.bbamcr.2014.06.019. Epub 2014 Jul 08.

Apoptosis, or programmed cell death, is an essential physiological process for proper embryogenesis as well as for homeostasis during aging. In addition, apoptosis is one of the major mechanisms causing cell loss in pathophysiological conditions such as heart failure....

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Sajjad A, Novoyatleva T, Vergarajauregui S, et al. Lysine methyltransferase Smyd2 suppresses p53-dependent cardiomyocyte apoptosis. Biochim Biophys Acta. 2014;1843(11):2556-62doi: 10.1016/j.bbamcr.2014.06.019.
Sajjad, A., Novoyatleva, T., Vergarajauregui, S., Troidl, C., Schermuly, R. T., Tucker, H. O., & Engel, F. B. (2014). Lysine methyltransferase Smyd2 suppresses p53-dependent cardiomyocyte apoptosis. Biochimica et biophysica acta, 1843(11), 2556-62. https://doi.org/10.1016/j.bbamcr.2014.06.019
Sajjad, Amna, et al. "Lysine methyltransferase Smyd2 suppresses p53-dependent cardiomyocyte apoptosis." Biochimica et biophysica acta vol. 1843,11 (2014): 2556-62. doi: https://doi.org/10.1016/j.bbamcr.2014.06.019
Sajjad A, Novoyatleva T, Vergarajauregui S, Troidl C, Schermuly RT, Tucker HO, Engel FB. Lysine methyltransferase Smyd2 suppresses p53-dependent cardiomyocyte apoptosis. Biochim Biophys Acta. 2014 Nov;1843(11):2556-62. doi: 10.1016/j.bbamcr.2014.06.019. Epub 2014 Jul 08. PMID: 25014164; PMCID: PMC4157957.
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