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OC-06 - Pro-thrombotic biomarkers in pancreatic diseases: are they specific of cancer?.

Thrombosis research

Faille D, Ajzenberg N, de Chaisemartin L, Granger V, Chollet-Martin S, de Raucourt E, Hammel P, Lévy P, Ruszniewski P, Rebours V.
PMID: 27161678
Thromb Res. 2016 Apr;140:S170-1. doi: 10.1016/S0049-3848(16)30123-2. Epub 2016 Apr 08.

INTRODUCTION: Venous thrombo-embolic events (VTE) occur frequently in patients with pancreatic cancer and contribute to elevated morbidity and mortality. Clinical risk factors for thrombosis such as cancer stage and tumor grade have been clearly identified. Recently, several biomarkers have...

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Faille D, Ajzenberg N, de Chaisemartin L, et al. OC-06 - Pro-thrombotic biomarkers in pancreatic diseases: are they specific of cancer?. Thromb Res. 2016;140:S170-1doi: 10.1016/S0049-3848(16)30123-2.
Faille, D., Ajzenberg, N., de Chaisemartin, L., Granger, V., Chollet-Martin, S., de Raucourt, E., Hammel, P., Lévy, P., Ruszniewski, P., & Rebours, V. (2016). OC-06 - Pro-thrombotic biomarkers in pancreatic diseases: are they specific of cancer?. Thrombosis research, 140S170-1. https://doi.org/10.1016/S0049-3848(16)30123-2
Faille, D, et al. "OC-06 - Pro-thrombotic biomarkers in pancreatic diseases: are they specific of cancer?." Thrombosis research vol. 140 (2016): S170-1. doi: https://doi.org/10.1016/S0049-3848(16)30123-2
Faille D, Ajzenberg N, de Chaisemartin L, Granger V, Chollet-Martin S, de Raucourt E, Hammel P, Lévy P, Ruszniewski P, Rebours V. OC-06 - Pro-thrombotic biomarkers in pancreatic diseases: are they specific of cancer?. Thromb Res. 2016 Apr;140:S170-1. doi: 10.1016/S0049-3848(16)30123-2. Epub 2016 Apr 08. PMID: 27161678.
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Innate immune deficiencies are associated with severity and poor prognosis in patients with COVID-19.

Scientific reports

Peyneau M, Granger V, Wicky PH, Khelifi-Touhami D, Timsit JF, Lescure FX, Yazdanpanah Y, Tran-Dinh A, Montravers P, Monteiro RC, Chollet-Martin S, Hurtado-Nedelec M, de Chaisemartin L.
PMID: 35022495
Sci Rep. 2022 Jan 12;12(1):638. doi: 10.1038/s41598-021-04705-7.

COVID-19 can cause acute respiratory distress syndrome, leading to death in many individuals. Evidence of a deleterious role of the innate immune system is accumulating, but the precise mechanisms involved remain unclear. In this study, we investigated the links...

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Peyneau M, Granger V, Wicky PH, et al. Innate immune deficiencies are associated with severity and poor prognosis in patients with COVID-19. Sci Rep. 2022;12(1):638doi: 10.1038/s41598-021-04705-7.
Peyneau, M., Granger, V., Wicky, P. H., Khelifi-Touhami, D., Timsit, J. F., Lescure, F. X., Yazdanpanah, Y., Tran-Dinh, A., Montravers, P., Monteiro, R. C., Chollet-Martin, S., Hurtado-Nedelec, M., & de Chaisemartin, L. (2022). Innate immune deficiencies are associated with severity and poor prognosis in patients with COVID-19. Scientific reports, 12(1), 638. https://doi.org/10.1038/s41598-021-04705-7
Peyneau, Marine, et al. "Innate immune deficiencies are associated with severity and poor prognosis in patients with COVID-19." Scientific reports vol. 12,1 (2022): 638. doi: https://doi.org/10.1038/s41598-021-04705-7
Peyneau M, Granger V, Wicky PH, Khelifi-Touhami D, Timsit JF, Lescure FX, Yazdanpanah Y, Tran-Dinh A, Montravers P, Monteiro RC, Chollet-Martin S, Hurtado-Nedelec M, de Chaisemartin L. Innate immune deficiencies are associated with severity and poor prognosis in patients with COVID-19. Sci Rep. 2022 Jan 12;12(1):638. doi: 10.1038/s41598-021-04705-7. PMID: 35022495.
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Neutrophil expression of glucocorticoid-induced leucine zipper (GILZ) anti-inflammatory protein is associated with acute respiratory distress syndrome severity.

Annals of intensive care

Espinasse MA, Hajage D, Montravers P, Piednoir P, Dufour G, Tubach F, Granger V, de Chaisemartin L, Noël B, Pallardy M, Chollet-Martin S, Biola-Vidamment A.
PMID: 27807817
Ann Intensive Care. 2016 Dec;6(1):105. doi: 10.1186/s13613-016-0210-0. Epub 2016 Nov 02.

BACKGROUND: Glucocorticoid-induced leucine zipper (GILZ) is a potent anti-inflammatory protein involved in neutrophil apoptosis and the resolution of inflammation. Given the numerous pathophysiologic roles of neutrophils in the acute respiratory distress syndrome (ARDS), we postulated that neutrophil GILZ expression...

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Espinasse MA, Hajage D, Montravers P, et al. Neutrophil expression of glucocorticoid-induced leucine zipper (GILZ) anti-inflammatory protein is associated with acute respiratory distress syndrome severity. Ann Intensive Care. 2016;6(1):105doi: 10.1186/s13613-016-0210-0.
Espinasse, M. A., Hajage, D., Montravers, P., Piednoir, P., Dufour, G., Tubach, F., Granger, V., de Chaisemartin, L., Noël, B., Pallardy, M., Chollet-Martin, S., & Biola-Vidamment, A. (2016). Neutrophil expression of glucocorticoid-induced leucine zipper (GILZ) anti-inflammatory protein is associated with acute respiratory distress syndrome severity. Annals of intensive care, 6(1), 105. https://doi.org/10.1186/s13613-016-0210-0
Espinasse, Marie-Alix, et al. "Neutrophil expression of glucocorticoid-induced leucine zipper (GILZ) anti-inflammatory protein is associated with acute respiratory distress syndrome severity." Annals of intensive care vol. 6,1 (2016): 105. doi: https://doi.org/10.1186/s13613-016-0210-0
Espinasse MA, Hajage D, Montravers P, Piednoir P, Dufour G, Tubach F, Granger V, de Chaisemartin L, Noël B, Pallardy M, Chollet-Martin S, Biola-Vidamment A. Neutrophil expression of glucocorticoid-induced leucine zipper (GILZ) anti-inflammatory protein is associated with acute respiratory distress syndrome severity. Ann Intensive Care. 2016 Dec;6(1):105. doi: 10.1186/s13613-016-0210-0. Epub 2016 Nov 02. PMID: 27807817; PMCID: PMC5093102.
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Innate immune deficiencies are associated with severity and poor prognosis in patients with COVID-19.

Scientific reports

Peyneau M, Granger V, Wicky PH, Khelifi-Touhami D, Timsit JF, Lescure FX, Yazdanpanah Y, Tran-Dinh A, Montravers P, Monteiro RC, Chollet-Martin S, Hurtado-Nedelec M, de Chaisemartin L.
PMID: 35022495
Sci Rep. 2022 Jan 12;12(1):638. doi: 10.1038/s41598-021-04705-7.

COVID-19 can cause acute respiratory distress syndrome, leading to death in many individuals. Evidence of a deleterious role of the innate immune system is accumulating, but the precise mechanisms involved remain unclear. In this study, we investigated the links...

Cite
Peyneau M, Granger V, Wicky PH, et al. Innate immune deficiencies are associated with severity and poor prognosis in patients with COVID-19. Sci Rep. 2022;12(1):638doi: 10.1038/s41598-021-04705-7.
Peyneau, M., Granger, V., Wicky, P. H., Khelifi-Touhami, D., Timsit, J. F., Lescure, F. X., Yazdanpanah, Y., Tran-Dinh, A., Montravers, P., Monteiro, R. C., Chollet-Martin, S., Hurtado-Nedelec, M., & de Chaisemartin, L. (2022). Innate immune deficiencies are associated with severity and poor prognosis in patients with COVID-19. Scientific reports, 12(1), 638. https://doi.org/10.1038/s41598-021-04705-7
Peyneau, Marine, et al. "Innate immune deficiencies are associated with severity and poor prognosis in patients with COVID-19." Scientific reports vol. 12,1 (2022): 638. doi: https://doi.org/10.1038/s41598-021-04705-7
Peyneau M, Granger V, Wicky PH, Khelifi-Touhami D, Timsit JF, Lescure FX, Yazdanpanah Y, Tran-Dinh A, Montravers P, Monteiro RC, Chollet-Martin S, Hurtado-Nedelec M, de Chaisemartin L. Innate immune deficiencies are associated with severity and poor prognosis in patients with COVID-19. Sci Rep. 2022 Jan 12;12(1):638. doi: 10.1038/s41598-021-04705-7. PMID: 35022495.
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An improved strategy to recover large fragments of functional human neutrophil extracellular traps.

Frontiers in immunology

Barrientos L, Marin-Esteban V, de Chaisemartin L, Le-Moal VL, Sandré C, Bianchini E, Nicolas V, Pallardy M, Chollet-Martin S.
PMID: 23805143
Front Immunol. 2013 Jun 24;4:166. doi: 10.3389/fimmu.2013.00166. eCollection 2013.

Netosis is a recently described neutrophil function that leads to the release of neutrophil extracellular traps (NETs) in response to various stimuli. NETs are filaments of decondensed chromatin associated with granular proteins. In addition to their role against microorganisms,...

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Barrientos L, Marin-Esteban V, de Chaisemartin L, et al. An improved strategy to recover large fragments of functional human neutrophil extracellular traps. Front Immunol. 2013;4:166doi: 10.3389/fimmu.2013.00166.
Barrientos, L., Marin-Esteban, V., de Chaisemartin, L., Le-Moal, V. L., Sandré, C., Bianchini, E., Nicolas, V., Pallardy, M., & Chollet-Martin, S. (2013). An improved strategy to recover large fragments of functional human neutrophil extracellular traps. Frontiers in immunology, 4166. https://doi.org/10.3389/fimmu.2013.00166
Barrientos, Lorena, et al. "An improved strategy to recover large fragments of functional human neutrophil extracellular traps." Frontiers in immunology vol. 4 (2013): 166. doi: https://doi.org/10.3389/fimmu.2013.00166
Barrientos L, Marin-Esteban V, de Chaisemartin L, Le-Moal VL, Sandré C, Bianchini E, Nicolas V, Pallardy M, Chollet-Martin S. An improved strategy to recover large fragments of functional human neutrophil extracellular traps. Front Immunol. 2013 Jun 24;4:166. doi: 10.3389/fimmu.2013.00166. eCollection 2013. PMID: 23805143; PMCID: PMC3690357.
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The immune response in SAPHO syndrome: deficiency, hyper- responsiveness, or both?.

Current rheumatology reviews

Hayem G, Hurtado-Nedelec M, Chollet-Martin S.
PMID: 25198361
Curr Rheumatol Rev. 2013;9(1):11-4. doi: 10.2174/1573397111309010004.

The pathophysiology of SAPHO syndrome still remains to be determined. However, like in other forms of spondylarthritides, this rare condition seems to result from the combination of genetic, environmental and immunological factors. Surely, SAPHO syndrome cannot be simply regarded...

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Hayem G, Hurtado-Nedelec M, Chollet-Martin S. The immune response in SAPHO syndrome: deficiency, hyper- responsiveness, or both?. Curr Rheumatol Rev. 2013;9(1):11-4doi: 10.2174/1573397111309010004.
Hayem, G., Hurtado-Nedelec, M., & Chollet-Martin, S. (2013). The immune response in SAPHO syndrome: deficiency, hyper- responsiveness, or both?. Current rheumatology reviews, 9(1), 11-4. https://doi.org/10.2174/1573397111309010004
Hayem, Gilles, et al. "The immune response in SAPHO syndrome: deficiency, hyper- responsiveness, or both?." Current rheumatology reviews vol. 9,1 (2013): 11-4. doi: https://doi.org/10.2174/1573397111309010004
Hayem G, Hurtado-Nedelec M, Chollet-Martin S. The immune response in SAPHO syndrome: deficiency, hyper- responsiveness, or both?. Curr Rheumatol Rev. 2013;9(1):11-4. doi: 10.2174/1573397111309010004. PMID: 25198361.
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In reply.

Anesthesiology

Laroche D, Chollet-Martin S, Léturgie P, Malzac L, Vergnaud MC, Neukirch C, Venemalm L, Guéant JL, Roland PN.
PMID: 21862894
Anesthesiology. 2011 Sep;115(3):659-60. doi: 10.1097/ALN.0b013e3182268153.

No abstract available.

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Laroche D, Chollet-Martin S, Léturgie P, et al. In reply. Anesthesiology. 2011;115(3):659-60doi: 10.1097/ALN.0b013e3182268153.
Laroche, D., Chollet-Martin, S., Léturgie, P., Malzac, L., Vergnaud, M. C., Neukirch, C., Venemalm, L., Guéant, J. L., & Roland, P. N. (2011). In reply. Anesthesiology, 115(3), 659-60. https://doi.org/10.1097/ALN.0b013e3182268153
Laroche, Dominique, et al. "In reply." Anesthesiology vol. 115,3 (2011): 659-60. doi: https://doi.org/10.1097/ALN.0b013e3182268153
Laroche D, Chollet-Martin S, Léturgie P, Malzac L, Vergnaud MC, Neukirch C, Venemalm L, Guéant JL, Roland PN. In reply. Anesthesiology. 2011 Sep;115(3):659-60. doi: 10.1097/ALN.0b013e3182268153. PMID: 21862894.
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Innate immune deficiencies are associated with severity and poor prognosis in patients with COVID-19.

Scientific reports

Peyneau M, Granger V, Wicky PH, Khelifi-Touhami D, Timsit JF, Lescure FX, Yazdanpanah Y, Tran-Dinh A, Montravers P, Monteiro RC, Chollet-Martin S, Hurtado-Nedelec M, de Chaisemartin L.
PMID: 35022495
Sci Rep. 2022 Jan 12;12(1):638. doi: 10.1038/s41598-021-04705-7.

COVID-19 can cause acute respiratory distress syndrome, leading to death in many individuals. Evidence of a deleterious role of the innate immune system is accumulating, but the precise mechanisms involved remain unclear. In this study, we investigated the links...

Cite
Peyneau M, Granger V, Wicky PH, et al. Innate immune deficiencies are associated with severity and poor prognosis in patients with COVID-19. Sci Rep. 2022;12(1):638doi: 10.1038/s41598-021-04705-7.
Peyneau, M., Granger, V., Wicky, P. H., Khelifi-Touhami, D., Timsit, J. F., Lescure, F. X., Yazdanpanah, Y., Tran-Dinh, A., Montravers, P., Monteiro, R. C., Chollet-Martin, S., Hurtado-Nedelec, M., & de Chaisemartin, L. (2022). Innate immune deficiencies are associated with severity and poor prognosis in patients with COVID-19. Scientific reports, 12(1), 638. https://doi.org/10.1038/s41598-021-04705-7
Peyneau, Marine, et al. "Innate immune deficiencies are associated with severity and poor prognosis in patients with COVID-19." Scientific reports vol. 12,1 (2022): 638. doi: https://doi.org/10.1038/s41598-021-04705-7
Peyneau M, Granger V, Wicky PH, Khelifi-Touhami D, Timsit JF, Lescure FX, Yazdanpanah Y, Tran-Dinh A, Montravers P, Monteiro RC, Chollet-Martin S, Hurtado-Nedelec M, de Chaisemartin L. Innate immune deficiencies are associated with severity and poor prognosis in patients with COVID-19. Sci Rep. 2022 Jan 12;12(1):638. doi: 10.1038/s41598-021-04705-7. PMID: 35022495; PMCID: PMC8755788.
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Erratum to: "IL-33/ST2 pathway regulates neutrophil migration and predicts outcome in patients with severe alcoholic hepatitis".

Journal of hepatology

Artru F, Bou Saleh M, Maggiotto F, Lassailly G, Ningarhari M, Demaret J, Ntandja-Wandji LC, Pais de Barros JP, Labreuche J, Drumez E, Helou DG, Dharancy S, Gantier E, Périanin A, Chollet-Martin S, Bataller R, Mathurin P, Dubuquoy L, Louvet A.
PMID: 33715895
J Hepatol. 2021 May;74(5):1272-1273. doi: 10.1016/j.jhep.2021.02.016. Epub 2021 Mar 11.

No abstract available.

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Artru F, Bou Saleh M, Maggiotto F, et al. Erratum to: "IL-33/ST2 pathway regulates neutrophil migration and predicts outcome in patients with severe alcoholic hepatitis". J Hepatol. 2021;74(5):1272-1273doi: 10.1016/j.jhep.2021.02.016.
Artru, F., Bou Saleh, M., Maggiotto, F., Lassailly, G., Ningarhari, M., Demaret, J., Ntandja-Wandji, L. C., Pais de Barros, J. P., Labreuche, J., Drumez, E., Helou, D. G., Dharancy, S., Gantier, E., Périanin, A., Chollet-Martin, S., Bataller, R., Mathurin, P., Dubuquoy, L., & Louvet, A. (2021). Erratum to: "IL-33/ST2 pathway regulates neutrophil migration and predicts outcome in patients with severe alcoholic hepatitis". Journal of hepatology, 74(5), 1272-1273. https://doi.org/10.1016/j.jhep.2021.02.016
Artru, Florent, et al. "Erratum to: "IL-33/ST2 pathway regulates neutrophil migration and predicts outcome in patients with severe alcoholic hepatitis"." Journal of hepatology vol. 74,5 (2021): 1272-1273. doi: https://doi.org/10.1016/j.jhep.2021.02.016
Artru F, Bou Saleh M, Maggiotto F, Lassailly G, Ningarhari M, Demaret J, Ntandja-Wandji LC, Pais de Barros JP, Labreuche J, Drumez E, Helou DG, Dharancy S, Gantier E, Périanin A, Chollet-Martin S, Bataller R, Mathurin P, Dubuquoy L, Louvet A. Erratum to: "IL-33/ST2 pathway regulates neutrophil migration and predicts outcome in patients with severe alcoholic hepatitis". J Hepatol. 2021 May;74(5):1272-1273. doi: 10.1016/j.jhep.2021.02.016. Epub 2021 Mar 11. PMID: 33715895.
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The Proteolytic Inactivation of Protein Z-Dependent Protease Inhibitor by Neutrophil Elastase Might Promote the Procoagulant Activity of Neutrophil Extracellular Traps in Sepsis.

Thrombosis and haemostasis

Bianchini EP, Razanakolona M, Helms J, Zouiti F, Couteau-Chardon A, Marin-Esteban V, Chaisemartin L, De-Carvalho A, Bironien R, Chollet-Martin S, Denis CV, Diehl JL, Vasse M, Meziani F, Borgel D.
PMID: 34134169
Thromb Haemost. 2021 Jun 16; doi: 10.1055/a-1530-3980. Epub 2021 Jun 16.

Septic shock is the archetypal clinical setting in which extensive crosstalk between inflammation and coagulation dysregulates the latter. The main anticoagulant systems are systematically impaired, depleted, and/or downregulated. Protein Z-dependent protease inhibitor (ZPI) is an anticoagulant serpin that not...

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Bianchini EP, Razanakolona M, Helms J, et al. The Proteolytic Inactivation of Protein Z-Dependent Protease Inhibitor by Neutrophil Elastase Might Promote the Procoagulant Activity of Neutrophil Extracellular Traps in Sepsis. Thromb Haemost. 2021;doi: 10.1055/a-1530-3980.
Bianchini, E. P., Razanakolona, M., Helms, J., Zouiti, F., Couteau-Chardon, A., Marin-Esteban, V., Chaisemartin, L., De-Carvalho, A., Bironien, R., Chollet-Martin, S., Denis, C. V., Diehl, J. L., Vasse, M., Meziani, F., & Borgel, D. (2021). The Proteolytic Inactivation of Protein Z-Dependent Protease Inhibitor by Neutrophil Elastase Might Promote the Procoagulant Activity of Neutrophil Extracellular Traps in Sepsis. Thrombosis and haemostasis, . https://doi.org/10.1055/a-1530-3980
Bianchini, Elsa P, et al. "The Proteolytic Inactivation of Protein Z-Dependent Protease Inhibitor by Neutrophil Elastase Might Promote the Procoagulant Activity of Neutrophil Extracellular Traps in Sepsis." Thrombosis and haemostasis vol. (2021). doi: https://doi.org/10.1055/a-1530-3980
Bianchini EP, Razanakolona M, Helms J, Zouiti F, Couteau-Chardon A, Marin-Esteban V, Chaisemartin L, De-Carvalho A, Bironien R, Chollet-Martin S, Denis CV, Diehl JL, Vasse M, Meziani F, Borgel D. The Proteolytic Inactivation of Protein Z-Dependent Protease Inhibitor by Neutrophil Elastase Might Promote the Procoagulant Activity of Neutrophil Extracellular Traps in Sepsis. Thromb Haemost. 2021 Jun 16; doi: 10.1055/a-1530-3980. Epub 2021 Jun 16. PMID: 34134169.
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Showing 1 to 10 of 10 entries