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Taking a back seat: synaptic vesicle clustering in presynaptic terminals.

Frontiers in synaptic neuroscience

Pechstein A, Shupliakov O.
PMID: 21423529
Front Synaptic Neurosci. 2010 Sep 15;2:143. doi: 10.3389/fnsyn.2010.00143. eCollection 2010.

Central inter-neuronal synapses employ various molecular mechanisms to sustain neurotransmitter release during phases of high-frequency synaptic activity. One of the features ensuring this property is the presence of a pool of synaptic vesicles (SVs) in the presynaptic terminal. At...

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Pechstein A, Shupliakov O. Taking a back seat: synaptic vesicle clustering in presynaptic terminals. Front Synaptic Neurosci. 2010;2:143doi: 10.3389/fnsyn.2010.00143.
Pechstein, A., & Shupliakov, O. (2010). Taking a back seat: synaptic vesicle clustering in presynaptic terminals. Frontiers in synaptic neuroscience, 2143. https://doi.org/10.3389/fnsyn.2010.00143
Pechstein, Arndt, and Shupliakov, Oleg. "Taking a back seat: synaptic vesicle clustering in presynaptic terminals." Frontiers in synaptic neuroscience vol. 2 (2010): 143. doi: https://doi.org/10.3389/fnsyn.2010.00143
Pechstein A, Shupliakov O. Taking a back seat: synaptic vesicle clustering in presynaptic terminals. Front Synaptic Neurosci. 2010 Sep 15;2:143. doi: 10.3389/fnsyn.2010.00143. eCollection 2010. PMID: 21423529; PMCID: PMC3059686.
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On the Distribution of GAP-43 and its Relation to Serotonin in Adult Monkey and Cat Spinal Cord and Lower Brainstem.

The European journal of neuroscience

Arvidsson U, Risling M, Cullheim S, Dagerlind A, Lindå H, Shupliakov O, Ulfhake B, Hökfelt T.
PMID: 12106322
Eur J Neurosci. 1992;4(8):777-784. doi: 10.1111/j.1460-9568.1992.tb00187.x.

By use of a monoclonal antibody, the distribution of growth-associated protein (GAP)-43-like immunoreactivity (LI) has been studied in the spinal cord of adult grey monkeys (Macaca fascicularis) and adult cats by use of immunofluorescence and peroxidase - antiperoxidase techniques....

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Arvidsson U, Risling M, Cullheim S, et al. On the Distribution of GAP-43 and its Relation to Serotonin in Adult Monkey and Cat Spinal Cord and Lower Brainstem. Eur J Neurosci. 1992;4(8):777-784doi: 10.1111/j.1460-9568.1992.tb00187.x.
Arvidsson, U., Risling, M., Cullheim, S., Dagerlind, A., Lindå, H., Shupliakov, O., Ulfhake, B., & Hökfelt, T. (1992). On the Distribution of GAP-43 and its Relation to Serotonin in Adult Monkey and Cat Spinal Cord and Lower Brainstem. The European journal of neuroscience, 4(8), 777-784. https://doi.org/10.1111/j.1460-9568.1992.tb00187.x
Arvidsson, Ulf, et al. "On the Distribution of GAP-43 and its Relation to Serotonin in Adult Monkey and Cat Spinal Cord and Lower Brainstem." The European journal of neuroscience vol. 4,8 (1992): 777-784. doi: https://doi.org/10.1111/j.1460-9568.1992.tb00187.x
Arvidsson U, Risling M, Cullheim S, Dagerlind A, Lindå H, Shupliakov O, Ulfhake B, Hökfelt T. On the Distribution of GAP-43 and its Relation to Serotonin in Adult Monkey and Cat Spinal Cord and Lower Brainstem. Eur J Neurosci. 1992;4(8):777-784. doi: 10.1111/j.1460-9568.1992.tb00187.x. PMID: 12106322.
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Retromer in Synaptic Function and Pathology.

Frontiers in synaptic neuroscience

Brodin L, Shupliakov O.
PMID: 30405388
Front Synaptic Neurosci. 2018 Oct 24;10:37. doi: 10.3389/fnsyn.2018.00037. eCollection 2018.

The retromer complex mediates export of select transmembrane proteins from endosomes to the trans-Golgi network (TGN) or to the plasma membrane. Dysfunction of retromer has been linked with slowly progressing neurodegenerative disorders, including Alzheimer's and Parkinson's disease (AD and...

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Brodin L, Shupliakov O. Retromer in Synaptic Function and Pathology. Front Synaptic Neurosci. 2018;10:37doi: 10.3389/fnsyn.2018.00037.
Brodin, L., & Shupliakov, O. (2018). Retromer in Synaptic Function and Pathology. Frontiers in synaptic neuroscience, 1037. https://doi.org/10.3389/fnsyn.2018.00037
Brodin, Lennart, and Shupliakov, Oleg. "Retromer in Synaptic Function and Pathology." Frontiers in synaptic neuroscience vol. 10 (2018): 37. doi: https://doi.org/10.3389/fnsyn.2018.00037
Brodin L, Shupliakov O. Retromer in Synaptic Function and Pathology. Front Synaptic Neurosci. 2018 Oct 24;10:37. doi: 10.3389/fnsyn.2018.00037. eCollection 2018. PMID: 30405388; PMCID: PMC6207580.
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Mitochondrial dysfunction in adult midbrain dopamine neurons triggers an early immune response.

PLoS genetics

Filograna R, Lee S, Tiklová K, Mennuni M, Jonsson V, Ringnér M, Gillberg L, Sopova E, Shupliakov O, Koolmeister C, Olson L, Perlmann T, Larsson NG.
PMID: 34570766
PLoS Genet. 2021 Sep 27;17(9):e1009822. doi: 10.1371/journal.pgen.1009822. eCollection 2021 Sep.

Dopamine (DA) neurons of the midbrain are at risk to become affected by mitochondrial damage over time and mitochondrial defects have been frequently reported in Parkinson's disease (PD) patients. However, the causal contribution of adult-onset mitochondrial dysfunction to PD...

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Filograna R, Lee S, Tiklová K, et al. Mitochondrial dysfunction in adult midbrain dopamine neurons triggers an early immune response. PLoS Genet. 2021;17(9):e1009822doi: 10.1371/journal.pgen.1009822.
Filograna, R., Lee, S., Tiklová, K., Mennuni, M., Jonsson, V., Ringnér, M., Gillberg, L., Sopova, E., Shupliakov, O., Koolmeister, C., Olson, L., Perlmann, T., & Larsson, N. G. (2021). Mitochondrial dysfunction in adult midbrain dopamine neurons triggers an early immune response. PLoS genetics, 17(9), e1009822. https://doi.org/10.1371/journal.pgen.1009822
Filograna, Roberta, et al. "Mitochondrial dysfunction in adult midbrain dopamine neurons triggers an early immune response." PLoS genetics vol. 17,9 (2021): e1009822. doi: https://doi.org/10.1371/journal.pgen.1009822
Filograna R, Lee S, Tiklová K, Mennuni M, Jonsson V, Ringnér M, Gillberg L, Sopova E, Shupliakov O, Koolmeister C, Olson L, Perlmann T, Larsson NG. Mitochondrial dysfunction in adult midbrain dopamine neurons triggers an early immune response. PLoS Genet. 2021 Sep 27;17(9):e1009822. doi: 10.1371/journal.pgen.1009822. eCollection 2021 Sep. PMID: 34570766; PMCID: PMC8496783.
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Showing 1 to 4 of 4 entries