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Showing 1 to 11 of 11 entries
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Premature aging of leukocyte DNA methylation is associated with type 2 diabetes prevalence.

Clinical epigenetics

Toperoff G, Kark JD, Aran D, Nassar H, Ahmad WA, Sinnreich R, Azaiza D, Glaser B, Hellman A.
PMID: 25829970
Clin Epigenetics. 2015 Mar 28;7:35. doi: 10.1186/s13148-015-0069-1. eCollection 2015.

BACKGROUND: Type 2 diabetes mellitus (T2D) is highly prevalent in Middle-Eastern and North African Arab populations, but the molecular basis for this susceptibility is unknown. Altered DNA methylation levels were reported in insulin-secreting and responding tissues, but whether methylation...

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Toperoff G, Kark JD, Aran D, et al. Premature aging of leukocyte DNA methylation is associated with type 2 diabetes prevalence. Clin Epigenetics. 2015;7:35doi: 10.1186/s13148-015-0069-1.
Toperoff, G., Kark, J. D., Aran, D., Nassar, H., Ahmad, W. A., Sinnreich, R., Azaiza, D., Glaser, B., & Hellman, A. (2015). Premature aging of leukocyte DNA methylation is associated with type 2 diabetes prevalence. Clinical epigenetics, 735. https://doi.org/10.1186/s13148-015-0069-1
Toperoff, Gidon, et al. "Premature aging of leukocyte DNA methylation is associated with type 2 diabetes prevalence." Clinical epigenetics vol. 7 (2015): 35. doi: https://doi.org/10.1186/s13148-015-0069-1
Toperoff G, Kark JD, Aran D, Nassar H, Ahmad WA, Sinnreich R, Azaiza D, Glaser B, Hellman A. Premature aging of leukocyte DNA methylation is associated with type 2 diabetes prevalence. Clin Epigenetics. 2015 Mar 28;7:35. doi: 10.1186/s13148-015-0069-1. eCollection 2015. PMID: 25829970; PMCID: PMC4379765.
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Mitochondrial cytopathy with common MELAS mutation presenting as multiple system atrophy mimic.

Neurology. Genetics

Pröbstel AK, Schaller A, Lieb J, Hench J, Frank S, Fuhr P, Kappos L, Sinnreich M.
PMID: 27878137
Neurol Genet. 2016 Nov 17;2(6):e121. doi: 10.1212/NXG.0000000000000121. eCollection 2016 Dec.

Mitochondrial encephalomyopathy, lactic acidosis, and stroke-like episodes (MELAS) syndrome

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Pröbstel AK, Schaller A, Lieb J, et al. Mitochondrial cytopathy with common MELAS mutation presenting as multiple system atrophy mimic. Neurol Genet. 2016;2(6):e121doi: 10.1212/NXG.0000000000000121.
Pröbstel, A. K., Schaller, A., Lieb, J., Hench, J., Frank, S., Fuhr, P., Kappos, L., & Sinnreich, M. (2016). Mitochondrial cytopathy with common MELAS mutation presenting as multiple system atrophy mimic. Neurology. Genetics, 2(6), e121. https://doi.org/10.1212/NXG.0000000000000121
Pröbstel, Anne-Katrin, et al. "Mitochondrial cytopathy with common MELAS mutation presenting as multiple system atrophy mimic." Neurology. Genetics vol. 2,6 (2016): e121. doi: https://doi.org/10.1212/NXG.0000000000000121
Pröbstel AK, Schaller A, Lieb J, Hench J, Frank S, Fuhr P, Kappos L, Sinnreich M. Mitochondrial cytopathy with common MELAS mutation presenting as multiple system atrophy mimic. Neurol Genet. 2016 Nov 17;2(6):e121. doi: 10.1212/NXG.0000000000000121. eCollection 2016 Dec. PMID: 27878137; PMCID: PMC5114693.
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Lacrimal diverticula.

Orbit (Amsterdam, Netherlands)

Sinnreich Z.
PMID: 12048728
Orbit. 1998 Sep;17(3):195-200. doi: 10.1076/orbi.17.3.195.2750.

Lacrimal diverticula are outpouchings of the lacrimal drainage system. They are suspected clinically by the presence of an intermittent or permanent swelling near the lacrimal sac, which can be emptied, by compression, into the lacrimal system and by patency...

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Sinnreich Z. Lacrimal diverticula. Orbit. 1998;17(3):195-200doi: 10.1076/orbi.17.3.195.2750.
Sinnreich, Z. (1998). Lacrimal diverticula. Orbit (Amsterdam, Netherlands), 17(3), 195-200. https://doi.org/10.1076/orbi.17.3.195.2750
Sinnreich, Ze'ev. "Lacrimal diverticula." Orbit (Amsterdam, Netherlands) vol. 17,3 (1998): 195-200. doi: https://doi.org/10.1076/orbi.17.3.195.2750
Sinnreich Z. Lacrimal diverticula. Orbit. 1998 Sep;17(3):195-200. doi: 10.1076/orbi.17.3.195.2750. PMID: 12048728.
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The Significance of the Sulfatase Pathway for Local Estrogen Formation in Endometrial Cancer.

Frontiers in pharmacology

Sinreih M, Knific T, Anko M, Hevir N, Vouk K, Jerin A, Frković Grazio S, Rižner TL.
PMID: 28690541
Front Pharmacol. 2017 Jun 23;8:368. doi: 10.3389/fphar.2017.00368. eCollection 2017.

Endometrial cancer (EC) is the most common estrogen-dependent gynecological malignancy in the developed World. To investigate the local formation of estradiol (E2), we first measured the concentrations of the steroid precursor androstenedione (A-dione) and the most potent estrogen, E2,...

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Sinreih M, Knific T, Anko M, et al. The Significance of the Sulfatase Pathway for Local Estrogen Formation in Endometrial Cancer. Front Pharmacol. 2017;8:368doi: 10.3389/fphar.2017.00368.
Sinreih, M., Knific, T., Anko, M., Hevir, N., Vouk, K., Jerin, A., Frković Grazio, S., & Rižner, T. L. (2017). The Significance of the Sulfatase Pathway for Local Estrogen Formation in Endometrial Cancer. Frontiers in pharmacology, 8368. https://doi.org/10.3389/fphar.2017.00368
Sinreih, Maša, et al. "The Significance of the Sulfatase Pathway for Local Estrogen Formation in Endometrial Cancer." Frontiers in pharmacology vol. 8 (2017): 368. doi: https://doi.org/10.3389/fphar.2017.00368
Sinreih M, Knific T, Anko M, Hevir N, Vouk K, Jerin A, Frković Grazio S, Rižner TL. The Significance of the Sulfatase Pathway for Local Estrogen Formation in Endometrial Cancer. Front Pharmacol. 2017 Jun 23;8:368. doi: 10.3389/fphar.2017.00368. eCollection 2017. PMID: 28690541; PMCID: PMC5481366.
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In the Model Cell Lines of Moderately and Poorly Differentiated Endometrial Carcinoma, Estrogens Can Be Formed .

Frontiers in molecular biosciences

Pavlič R, Gjorgoska M, Hafner E, Sinreih M, Gajser K, Poschner S, Jäger W, Rižner TL.
PMID: 34805270
Front Mol Biosci. 2021 Nov 05;8:743403. doi: 10.3389/fmolb.2021.743403. eCollection 2021.

Endometrial cancer (EC) is the most common gynecological malignancy in resource-abundant countries. The majority of EC cases are estrogen dependent but the mechanisms of estrogen biosynthesis and oxidative metabolism and estrogen action are not completely understood. Here, we evaluated...

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Pavlič R, Gjorgoska M, Hafner E, et al. In the Model Cell Lines of Moderately and Poorly Differentiated Endometrial Carcinoma, Estrogens Can Be Formed . Front Mol Biosci. 2021;8:743403doi: 10.3389/fmolb.2021.743403.
Pavlič, R., Gjorgoska, M., Hafner, E., Sinreih, M., Gajser, K., Poschner, S., Jäger, W., & Rižner, T. L. (2021). In the Model Cell Lines of Moderately and Poorly Differentiated Endometrial Carcinoma, Estrogens Can Be Formed . Frontiers in molecular biosciences, 8743403. https://doi.org/10.3389/fmolb.2021.743403
Pavlič, Renata, et al. "In the Model Cell Lines of Moderately and Poorly Differentiated Endometrial Carcinoma, Estrogens Can Be Formed ." Frontiers in molecular biosciences vol. 8 (2021): 743403. doi: https://doi.org/10.3389/fmolb.2021.743403
Pavlič R, Gjorgoska M, Hafner E, Sinreih M, Gajser K, Poschner S, Jäger W, Rižner TL. In the Model Cell Lines of Moderately and Poorly Differentiated Endometrial Carcinoma, Estrogens Can Be Formed . Front Mol Biosci. 2021 Nov 05;8:743403. doi: 10.3389/fmolb.2021.743403. eCollection 2021. PMID: 34805270; PMCID: PMC8602794.
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Genetic characterization and improved genotyping of the dysferlin-deficient mouse strain Dysf (tm1Kcam).

Skeletal muscle

Wiktorowicz T, Kinter J, Kobuke K, Campbell KP, Sinnreich M.
PMID: 26464793
Skelet Muscle. 2015 Oct 13;5:32. doi: 10.1186/s13395-015-0057-3. eCollection 2015.

BACKGROUND: Mouse models of dysferlinopathies are valuable tools with which to investigate the pathomechanisms underlying these diseases and to test novel therapeutic strategies. One such mouse model is the Dysf (tm1Kcam) strain, which was generated using a targeting vector...

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Wiktorowicz T, Kinter J, Kobuke K, et al. Genetic characterization and improved genotyping of the dysferlin-deficient mouse strain Dysf (tm1Kcam). Skelet Muscle. 2015;5:32doi: 10.1186/s13395-015-0057-3.
Wiktorowicz, T., Kinter, J., Kobuke, K., Campbell, K. P., & Sinnreich, M. (2015). Genetic characterization and improved genotyping of the dysferlin-deficient mouse strain Dysf (tm1Kcam). Skeletal muscle, 532. https://doi.org/10.1186/s13395-015-0057-3
Wiktorowicz, Tatiana, et al. "Genetic characterization and improved genotyping of the dysferlin-deficient mouse strain Dysf (tm1Kcam)." Skeletal muscle vol. 5 (2015): 32. doi: https://doi.org/10.1186/s13395-015-0057-3
Wiktorowicz T, Kinter J, Kobuke K, Campbell KP, Sinnreich M. Genetic characterization and improved genotyping of the dysferlin-deficient mouse strain Dysf (tm1Kcam). Skelet Muscle. 2015 Oct 13;5:32. doi: 10.1186/s13395-015-0057-3. eCollection 2015. PMID: 26464793; PMCID: PMC4603641.
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Proteasomal inhibition restores biological function of mis-sense mutated dysferlin in patient-derived muscle cells.

The Journal of biological chemistry

Azakir BA, Di Fulvio S, Kinter J, Sinnreich M.
PMID: 28754767
J Biol Chem. 2017 Jul 28;292(30):12542. doi: 10.1074/jbc.A111.329078.

No abstract available.

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Azakir BA, Di Fulvio S, Kinter J, et al. Proteasomal inhibition restores biological function of mis-sense mutated dysferlin in patient-derived muscle cells. J Biol Chem. 2017;292(30):12542doi: 10.1074/jbc.A111.329078.
Azakir, B. A., Di Fulvio, S., Kinter, J., & Sinnreich, M. (2017). Proteasomal inhibition restores biological function of mis-sense mutated dysferlin in patient-derived muscle cells. The Journal of biological chemistry, 292(30), 12542. https://doi.org/10.1074/jbc.A111.329078
Azakir, Bilal A, et al. "Proteasomal inhibition restores biological function of mis-sense mutated dysferlin in patient-derived muscle cells." The Journal of biological chemistry vol. 292,30 (2017): 12542. doi: https://doi.org/10.1074/jbc.A111.329078
Azakir BA, Di Fulvio S, Kinter J, Sinnreich M. Proteasomal inhibition restores biological function of mis-sense mutated dysferlin in patient-derived muscle cells. J Biol Chem. 2017 Jul 28;292(30):12542. doi: 10.1074/jbc.A111.329078. PMID: 28754767; PMCID: PMC5535028.
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Modular dispensability of dysferlin C2 domains reveals rational design for mini-dysferlin molecules.

The Journal of biological chemistry

Azakir BA, Di Fulvio S, Salomon S, Brockhoff M, Therrien C, Sinnreich M.
PMID: 28754768
J Biol Chem. 2017 Jul 28;292(30):12543. doi: 10.1074/jbc.A112.391722.

No abstract available.

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Azakir BA, Di Fulvio S, Salomon S, et al. Modular dispensability of dysferlin C2 domains reveals rational design for mini-dysferlin molecules. J Biol Chem. 2017;292(30):12543doi: 10.1074/jbc.A112.391722.
Azakir, B. A., Di Fulvio, S., Salomon, S., Brockhoff, M., Therrien, C., & Sinnreich, M. (2017). Modular dispensability of dysferlin C2 domains reveals rational design for mini-dysferlin molecules. The Journal of biological chemistry, 292(30), 12543. https://doi.org/10.1074/jbc.A112.391722
Azakir, Bilal A, et al. "Modular dispensability of dysferlin C2 domains reveals rational design for mini-dysferlin molecules." The Journal of biological chemistry vol. 292,30 (2017): 12543. doi: https://doi.org/10.1074/jbc.A112.391722
Azakir BA, Di Fulvio S, Salomon S, Brockhoff M, Therrien C, Sinnreich M. Modular dispensability of dysferlin C2 domains reveals rational design for mini-dysferlin molecules. J Biol Chem. 2017 Jul 28;292(30):12543. doi: 10.1074/jbc.A112.391722. PMID: 28754768; PMCID: PMC5535029.
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Exploration of the chemical space of novel naphthalene-sulfonamide and anthranilic Acid-based inhibitors of penicillin-binding proteins.

Acta chimica Slovenica

Sosič I, Turk S, Sinreih M, Trošt N, Verlaine O, Amoroso A, Zervosen A, Luxen A, Joris B, Gobec S.
PMID: 24061241
Acta Chim Slov. 2012 Jun;59(2):280-388.

Penicillin-binding proteins are a well established, validated and still a very promising target for the design and development of new antibacterial agents. Based on our previous discovery of several noncovalent small-molecule inhibitor hits for resistant PBPs we decided to...

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Sosič I, Turk S, Sinreih M, et al. Exploration of the chemical space of novel naphthalene-sulfonamide and anthranilic Acid-based inhibitors of penicillin-binding proteins. Acta Chim Slov. 2012;59(2):280-388
Sosič, I., Turk, S., Sinreih, M., Trošt, N., Verlaine, O., Amoroso, A., Zervosen, A., Luxen, A., Joris, B., & Gobec, S. (2012). Exploration of the chemical space of novel naphthalene-sulfonamide and anthranilic Acid-based inhibitors of penicillin-binding proteins. Acta chimica Slovenica, 59(2), 280-388.
Sosič, Izidor, et al. "Exploration of the chemical space of novel naphthalene-sulfonamide and anthranilic Acid-based inhibitors of penicillin-binding proteins." Acta chimica Slovenica vol. 59,2 (2012): 280-388.
Sosič I, Turk S, Sinreih M, Trošt N, Verlaine O, Amoroso A, Zervosen A, Luxen A, Joris B, Gobec S. Exploration of the chemical space of novel naphthalene-sulfonamide and anthranilic Acid-based inhibitors of penicillin-binding proteins. Acta Chim Slov. 2012 Jun;59(2):280-388. PMID: 24061241.
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Heterocyclic androstane and estrane d-ring modified steroids: Microwave-assisted synthesis, steroid-converting enzyme inhibition, apoptosis induction, and effects on genes encoding estrogen inactivating enzymes.

The Journal of steroid biochemistry and molecular biology

Kulmány ÁE, Herman BE, Zupkó I, Sinreih M, Rižner TL, Savić M, Oklješa A, Nikolić A, Nagy V, Ocsovszki I, Szécsi M, Jovanović-Šanta S.
PMID: 34509617
J Steroid Biochem Mol Biol. 2021 Nov;214:105997. doi: 10.1016/j.jsbmb.2021.105997. Epub 2021 Sep 10.

d-ring-fused and d-homo lactone compounds in estratriene and androstane series were synthesized using microwave-assisted reaction conditions. Microwave-irradiated synthesis methods were convenient and effective, and provided high yields with short reaction times. Their inhibition of C

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Kulmány ÁE, Herman BE, Zupkó I, et al. Heterocyclic androstane and estrane d-ring modified steroids: Microwave-assisted synthesis, steroid-converting enzyme inhibition, apoptosis induction, and effects on genes encoding estrogen inactivating enzymes. J Steroid Biochem Mol Biol. 2021;214:105997doi: 10.1016/j.jsbmb.2021.105997.
Kulmány, �. �. E., Herman, B. E., Zupkó, I., Sinreih, M., Rižner, T. L., Savić, M., Oklješa, A., Nikolić, A., Nagy, V., Ocsovszki, I., Szécsi, M., & Jovanović-Šanta, S. (2021). Heterocyclic androstane and estrane d-ring modified steroids: Microwave-assisted synthesis, steroid-converting enzyme inhibition, apoptosis induction, and effects on genes encoding estrogen inactivating enzymes. The Journal of steroid biochemistry and molecular biology, 214105997. https://doi.org/10.1016/j.jsbmb.2021.105997
Kulmány, Ágnes Erika, et al. "Heterocyclic androstane and estrane d-ring modified steroids: Microwave-assisted synthesis, steroid-converting enzyme inhibition, apoptosis induction, and effects on genes encoding estrogen inactivating enzymes." The Journal of steroid biochemistry and molecular biology vol. 214 (2021): 105997. doi: https://doi.org/10.1016/j.jsbmb.2021.105997
Kulmány ÁE, Herman BE, Zupkó I, Sinreih M, Rižner TL, Savić M, Oklješa A, Nikolić A, Nagy V, Ocsovszki I, Szécsi M, Jovanović-Šanta S. Heterocyclic androstane and estrane d-ring modified steroids: Microwave-assisted synthesis, steroid-converting enzyme inhibition, apoptosis induction, and effects on genes encoding estrogen inactivating enzymes. J Steroid Biochem Mol Biol. 2021 Nov;214:105997. doi: 10.1016/j.jsbmb.2021.105997. Epub 2021 Sep 10. PMID: 34509617.
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Data on expression of genes involved in estrogen and progesterone action, inflammation and differentiation according to demographic, histopathological and clinical characteristics of endometrial cancer patients.

Data in brief

Sinreih M, Štupar S, Čemažar L, Verdenik I, Frković Grazio S, Smrkolj Š, Lanišnik Rižner T.
PMID: 28540356
Data Brief. 2017 May 04;12:632-643. doi: 10.1016/j.dib.2017.04.050. eCollection 2017 Jun.

Endometrial cancer is the sixth most common cancer in women worldwide. It is associated with aberrant actions of steroid hormones, estrogens and progesterone, but also with enhanced inflammation and reduced cellular differentiation. Here, we show data on demographic and...

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Sinreih M, Štupar S, Čemažar L, et al. Data on expression of genes involved in estrogen and progesterone action, inflammation and differentiation according to demographic, histopathological and clinical characteristics of endometrial cancer patients. Data Brief. 2017;12:632-643doi: 10.1016/j.dib.2017.04.050.
Sinreih, M., Štupar, S., Čemažar, L., Verdenik, I., Frković Grazio, S., Smrkolj, �. �., & Lanišnik Rižner, T. (2017). Data on expression of genes involved in estrogen and progesterone action, inflammation and differentiation according to demographic, histopathological and clinical characteristics of endometrial cancer patients. Data in brief, 12632-643. https://doi.org/10.1016/j.dib.2017.04.050
Sinreih, Maša, et al. "Data on expression of genes involved in estrogen and progesterone action, inflammation and differentiation according to demographic, histopathological and clinical characteristics of endometrial cancer patients." Data in brief vol. 12 (2017): 632-643. doi: https://doi.org/10.1016/j.dib.2017.04.050
Sinreih M, Štupar S, Čemažar L, Verdenik I, Frković Grazio S, Smrkolj Š, Lanišnik Rižner T. Data on expression of genes involved in estrogen and progesterone action, inflammation and differentiation according to demographic, histopathological and clinical characteristics of endometrial cancer patients. Data Brief. 2017 May 04;12:632-643. doi: 10.1016/j.dib.2017.04.050. eCollection 2017 Jun. PMID: 28540356; PMCID: PMC5430149.
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