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Closing the Social Class Achievement Gap for First-Generation Students in Undergraduate Biology.

Journal of educational psychology

Harackiewicz JM, Canning EA, Tibbetts Y, Giffen CJ, Blair SS, Rouse DI, Hyde JS.
PMID: 25049437
J Educ Psychol. 2014 May 01;106(2):375-389. doi: 10.1037/a0034679.

Many students start college intending to pursue a career in the biosciences, but too many abandon this goal because they struggle in introductory biology. Interventions have been developed to close achievement gaps for underrepresented minority students and women, but...

Cite
Harackiewicz JM, Canning EA, Tibbetts Y, et al. Closing the Social Class Achievement Gap for First-Generation Students in Undergraduate Biology. J Educ Psychol. 2014;106(2):375-389doi: 10.1037/a0034679.
Harackiewicz, J. M., Canning, E. A., Tibbetts, Y., Giffen, C. J., Blair, S. S., Rouse, D. I., & Hyde, J. S. (2014). Closing the Social Class Achievement Gap for First-Generation Students in Undergraduate Biology. Journal of educational psychology, 106(2), 375-389. https://doi.org/10.1037/a0034679
Harackiewicz, Judith M, et al. "Closing the Social Class Achievement Gap for First-Generation Students in Undergraduate Biology." Journal of educational psychology vol. 106,2 (2014): 375-389. doi: https://doi.org/10.1037/a0034679
Harackiewicz JM, Canning EA, Tibbetts Y, Giffen CJ, Blair SS, Rouse DI, Hyde JS. Closing the Social Class Achievement Gap for First-Generation Students in Undergraduate Biology. J Educ Psychol. 2014 May 01;106(2):375-389. doi: 10.1037/a0034679. PMID: 25049437; PMCID: PMC4103196.
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Clonal selection of stable aneuploidies in progenitor cells drives high-prevalence tumorigenesis.

Genes & development

Trakala M, Aggarwal M, Sniffen C, Zasadil L, Carroll A, Ma D, Su XA, Wangsa D, Meyer A, Sieben CJ, Zhong J, Hsu PH, Paradis G, Ried T, Holland A, Van Deursen J, Amon A.
PMID: 34266888
Genes Dev. 2021 Aug 01;35(15):1079-1092. doi: 10.1101/gad.348341.121. Epub 2021 Jul 15.

Chromosome gains and losses are a frequent feature of human cancers. However, how these aberrations can outweigh the detrimental effects of aneuploidy remains unclear. An initial comparison of existing chromosomal instability (CIN) mouse models suggests that aneuploidy accumulates to...

Cite
Trakala M, Aggarwal M, Sniffen C, et al. Clonal selection of stable aneuploidies in progenitor cells drives high-prevalence tumorigenesis. Genes Dev. 2021;35(15):1079-1092doi: 10.1101/gad.348341.121.
Trakala, M., Aggarwal, M., Sniffen, C., Zasadil, L., Carroll, A., Ma, D., Su, X. A., Wangsa, D., Meyer, A., Sieben, C. J., Zhong, J., Hsu, P. H., Paradis, G., Ried, T., Holland, A., Van Deursen, J., & Amon, A. (2021). Clonal selection of stable aneuploidies in progenitor cells drives high-prevalence tumorigenesis. Genes & development, 35(15), 1079-1092. https://doi.org/10.1101/gad.348341.121
Trakala, Marianna, et al. "Clonal selection of stable aneuploidies in progenitor cells drives high-prevalence tumorigenesis." Genes & development vol. 35,15 (2021): 1079-1092. doi: https://doi.org/10.1101/gad.348341.121
Trakala M, Aggarwal M, Sniffen C, Zasadil L, Carroll A, Ma D, Su XA, Wangsa D, Meyer A, Sieben CJ, Zhong J, Hsu PH, Paradis G, Ried T, Holland A, Van Deursen J, Amon A. Clonal selection of stable aneuploidies in progenitor cells drives high-prevalence tumorigenesis. Genes Dev. 2021 Aug 01;35(15):1079-1092. doi: 10.1101/gad.348341.121. Epub 2021 Jul 15. PMID: 34266888; PMCID: PMC8336892.
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The value and progress of dental departments in mental hospitals.

The New York state dental journal

SNIFFEN DA.
PMID: 18895576
N Y State Dent J. 1947 Mar;13(3):144-7.

No abstract available.

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SNIFFEN DA. The value and progress of dental departments in mental hospitals. N Y State Dent J. 1947;13(3):144-7
SNIFFEN, D. A. (1947). The value and progress of dental departments in mental hospitals. The New York state dental journal, 13(3), 144-7.
SNIFFEN, D A. "The value and progress of dental departments in mental hospitals." The New York state dental journal vol. 13,3 (1947): 144-7.
SNIFFEN DA. The value and progress of dental departments in mental hospitals. N Y State Dent J. 1947 Mar;13(3):144-7. PMID: 18895576.
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A Novel Hyperactive Nud1 Mitotic Exit Network Scaffold Causes Spindle Position Checkpoint Bypass in Budding Yeast.

Cells

Vannini M, Mingione VR, Meyer A, Sniffen C, Whalen J, Seshan A.
PMID: 35011608
Cells. 2021 Dec 24;11(1). doi: 10.3390/cells11010046.

Mitotic exit is a critical cell cycle transition that requires the careful coordination of nuclear positioning and cyclin B destruction in budding yeast for the maintenance of genome integrity. The mitotic exit network (MEN) is a Ras-like signal transduction...

Cite
Vannini M, Mingione VR, Meyer A, et al. A Novel Hyperactive Nud1 Mitotic Exit Network Scaffold Causes Spindle Position Checkpoint Bypass in Budding Yeast. Cells. 2021;11(1)doi: 10.3390/cells11010046.
Vannini, M., Mingione, V. R., Meyer, A., Sniffen, C., Whalen, J., & Seshan, A. (2021). A Novel Hyperactive Nud1 Mitotic Exit Network Scaffold Causes Spindle Position Checkpoint Bypass in Budding Yeast. Cells, 11(1), . https://doi.org/10.3390/cells11010046
Vannini, Michael, et al. "A Novel Hyperactive Nud1 Mitotic Exit Network Scaffold Causes Spindle Position Checkpoint Bypass in Budding Yeast." Cells vol. 11,1 (2021). doi: https://doi.org/10.3390/cells11010046
Vannini M, Mingione VR, Meyer A, Sniffen C, Whalen J, Seshan A. A Novel Hyperactive Nud1 Mitotic Exit Network Scaffold Causes Spindle Position Checkpoint Bypass in Budding Yeast. Cells. 2021 Dec 24;11(1). doi: 10.3390/cells11010046. PMID: 35011608.
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Why Do ACG and AGA Guidelines Differ for the Use of Probiotics and the Prevention of CDI?.

The American journal of gastroenterology

McFarland LV, Kullar R, Johnson S, Sniffen JC, Woolard K, Goldstein EJC.
PMID: 35020606
Am J Gastroenterol. 2022 Jan 12; doi: 10.14309/ajg.0000000000001567. Epub 2022 Jan 12.

No abstract available.

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McFarland LV, Kullar R, Johnson S, et al. Why Do ACG and AGA Guidelines Differ for the Use of Probiotics and the Prevention of CDI?. Am J Gastroenterol. 2022;doi: 10.14309/ajg.0000000000001567.
McFarland, L. V., Kullar, R., Johnson, S., Sniffen, J. C., Woolard, K., & Goldstein, E. J. C. (2022). Why Do ACG and AGA Guidelines Differ for the Use of Probiotics and the Prevention of CDI?. The American journal of gastroenterology, . https://doi.org/10.14309/ajg.0000000000001567
McFarland, Lynne V, et al. "Why Do ACG and AGA Guidelines Differ for the Use of Probiotics and the Prevention of CDI?." The American journal of gastroenterology vol. (2022). doi: https://doi.org/10.14309/ajg.0000000000001567
McFarland LV, Kullar R, Johnson S, Sniffen JC, Woolard K, Goldstein EJC. Why Do ACG and AGA Guidelines Differ for the Use of Probiotics and the Prevention of CDI?. Am J Gastroenterol. 2022 Jan 12; doi: 10.14309/ajg.0000000000001567. Epub 2022 Jan 12. PMID: 35020606.
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Hemophagocytic lymphohistiocytosis and thrombotic microangiopathy after parvovirus B19 infection and renal transplantation: a case report.

BMC nephrology

Steffen CJ, Koch N, Eckardt KU, Amann K, Seelow E, Schreiber A.
PMID: 34641801
BMC Nephrol. 2021 Oct 12;22(1):337. doi: 10.1186/s12882-021-02538-0.

BACKGROUND: Hemophagocytic lymphohistiocytosis (HLH) is a rare and life-threatening disease characterized by hyperactivation of the immune system that causes hypercytokinemia and potentially multi organ failure. HLH can occur in patients with underlying rheumatic or autoinflammatory disorders. Additionally, HLH can...

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Steffen CJ, Koch N, Eckardt KU, et al. Hemophagocytic lymphohistiocytosis and thrombotic microangiopathy after parvovirus B19 infection and renal transplantation: a case report. BMC Nephrol. 2021;22(1):337doi: 10.1186/s12882-021-02538-0.
Steffen, C. J., Koch, N., Eckardt, K. U., Amann, K., Seelow, E., & Schreiber, A. (2021). Hemophagocytic lymphohistiocytosis and thrombotic microangiopathy after parvovirus B19 infection and renal transplantation: a case report. BMC nephrology, 22(1), 337. https://doi.org/10.1186/s12882-021-02538-0
Steffen, C J, et al. "Hemophagocytic lymphohistiocytosis and thrombotic microangiopathy after parvovirus B19 infection and renal transplantation: a case report." BMC nephrology vol. 22,1 (2021): 337. doi: https://doi.org/10.1186/s12882-021-02538-0
Steffen CJ, Koch N, Eckardt KU, Amann K, Seelow E, Schreiber A. Hemophagocytic lymphohistiocytosis and thrombotic microangiopathy after parvovirus B19 infection and renal transplantation: a case report. BMC Nephrol. 2021 Oct 12;22(1):337. doi: 10.1186/s12882-021-02538-0. PMID: 34641801; PMCID: PMC8506469.
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Renin Angiotensin Aldosterone System Antagonism in 2019 Novel Coronavirus Acute Lung Injury.

Open forum infectious diseases

Ventura D, Carr AL, Davis RD, Silvestry S, Bogar L, Raval N, Gries C, Hayes JE, Oliveira E, Sniffen J, Allison SL, Herrera V, Jennings DL, Page RL, McDyer JF, Ensor CR.
PMID: 34642634
Open Forum Infect Dis. 2021 Apr 04;8(10):ofab170. doi: 10.1093/ofid/ofab170. eCollection 2021 Oct.

It has been established that severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) uses angiotensin-converting enzyme 2 (ACE2), a membrane-bound regulatory peptide, for host cell entry. Renin-angiotensin-aldosterone system (RAAS) inhibitors have been reported to increase ACE2 in type 2 pneumocyte...

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Ventura D, Carr AL, Davis RD, et al. Renin Angiotensin Aldosterone System Antagonism in 2019 Novel Coronavirus Acute Lung Injury. Open Forum Infect Dis. 2021;8(10):ofab170doi: 10.1093/ofid/ofab170.
Ventura, D., Carr, A. L., Davis, R. D., Silvestry, S., Bogar, L., Raval, N., Gries, C., Hayes, J. E., Oliveira, E., Sniffen, J., Allison, S. L., Herrera, V., Jennings, D. L., Page, R. L., McDyer, J. F., & Ensor, C. R. (2021). Renin Angiotensin Aldosterone System Antagonism in 2019 Novel Coronavirus Acute Lung Injury. Open forum infectious diseases, 8(10), ofab170. https://doi.org/10.1093/ofid/ofab170
Ventura, Davide, et al. "Renin Angiotensin Aldosterone System Antagonism in 2019 Novel Coronavirus Acute Lung Injury." Open forum infectious diseases vol. 8,10 (2021): ofab170. doi: https://doi.org/10.1093/ofid/ofab170
Ventura D, Carr AL, Davis RD, Silvestry S, Bogar L, Raval N, Gries C, Hayes JE, Oliveira E, Sniffen J, Allison SL, Herrera V, Jennings DL, Page RL, McDyer JF, Ensor CR. Renin Angiotensin Aldosterone System Antagonism in 2019 Novel Coronavirus Acute Lung Injury. Open Forum Infect Dis. 2021 Apr 04;8(10):ofab170. doi: 10.1093/ofid/ofab170. eCollection 2021 Oct. PMID: 34642634; PMCID: PMC8083494.
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Clonal selection of stable aneuploidies in progenitor cells drives high-prevalence tumorigenesis.

Genes & development

Trakala M, Aggarwal M, Sniffen C, Zasadil L, Carroll A, Ma D, Su XA, Wangsa D, Meyer A, Sieben CJ, Zhong J, Hsu PH, Paradis G, Ried T, Holland A, Van Deursen J, Amon A.
PMID: 34266888
Genes Dev. 2021 Aug 01;35(15):1079-1092. doi: 10.1101/gad.348341.121. Epub 2021 Jul 15.

Chromosome gains and losses are a frequent feature of human cancers. However, how these aberrations can outweigh the detrimental effects of aneuploidy remains unclear. An initial comparison of existing chromosomal instability (CIN) mouse models suggests that aneuploidy accumulates to...

Cite
Trakala M, Aggarwal M, Sniffen C, et al. Clonal selection of stable aneuploidies in progenitor cells drives high-prevalence tumorigenesis. Genes Dev. 2021;35(15):1079-1092doi: 10.1101/gad.348341.121.
Trakala, M., Aggarwal, M., Sniffen, C., Zasadil, L., Carroll, A., Ma, D., Su, X. A., Wangsa, D., Meyer, A., Sieben, C. J., Zhong, J., Hsu, P. H., Paradis, G., Ried, T., Holland, A., Van Deursen, J., & Amon, A. (2021). Clonal selection of stable aneuploidies in progenitor cells drives high-prevalence tumorigenesis. Genes & development, 35(15), 1079-1092. https://doi.org/10.1101/gad.348341.121
Trakala, Marianna, et al. "Clonal selection of stable aneuploidies in progenitor cells drives high-prevalence tumorigenesis." Genes & development vol. 35,15 (2021): 1079-1092. doi: https://doi.org/10.1101/gad.348341.121
Trakala M, Aggarwal M, Sniffen C, Zasadil L, Carroll A, Ma D, Su XA, Wangsa D, Meyer A, Sieben CJ, Zhong J, Hsu PH, Paradis G, Ried T, Holland A, Van Deursen J, Amon A. Clonal selection of stable aneuploidies in progenitor cells drives high-prevalence tumorigenesis. Genes Dev. 2021 Aug 01;35(15):1079-1092. doi: 10.1101/gad.348341.121. Epub 2021 Jul 15. PMID: 34266888; PMCID: PMC8336892.
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A Novel Hyperactive Nud1 Mitotic Exit Network Scaffold Causes Spindle Position Checkpoint Bypass in Budding Yeast.

Cells

Vannini M, Mingione VR, Meyer A, Sniffen C, Whalen J, Seshan A.
PMID: 35011608
Cells. 2021 Dec 24;11(1). doi: 10.3390/cells11010046.

Mitotic exit is a critical cell cycle transition that requires the careful coordination of nuclear positioning and cyclin B destruction in budding yeast for the maintenance of genome integrity. The mitotic exit network (MEN) is a Ras-like signal transduction...

Cite
Vannini M, Mingione VR, Meyer A, et al. A Novel Hyperactive Nud1 Mitotic Exit Network Scaffold Causes Spindle Position Checkpoint Bypass in Budding Yeast. Cells. 2021;11(1)doi: 10.3390/cells11010046.
Vannini, M., Mingione, V. R., Meyer, A., Sniffen, C., Whalen, J., & Seshan, A. (2021). A Novel Hyperactive Nud1 Mitotic Exit Network Scaffold Causes Spindle Position Checkpoint Bypass in Budding Yeast. Cells, 11(1), . https://doi.org/10.3390/cells11010046
Vannini, Michael, et al. "A Novel Hyperactive Nud1 Mitotic Exit Network Scaffold Causes Spindle Position Checkpoint Bypass in Budding Yeast." Cells vol. 11,1 (2021). doi: https://doi.org/10.3390/cells11010046
Vannini M, Mingione VR, Meyer A, Sniffen C, Whalen J, Seshan A. A Novel Hyperactive Nud1 Mitotic Exit Network Scaffold Causes Spindle Position Checkpoint Bypass in Budding Yeast. Cells. 2021 Dec 24;11(1). doi: 10.3390/cells11010046. PMID: 35011608; PMCID: PMC8750578.
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Closing the Social Class Achievement Gap for First-Generation Students in Undergraduate Biology.

Harackiewicz JM, Hyde JS, Canning EA, Tibbetts Y, Giffen CJ, Blair SS, Rouse DI.
UIID-AD: 454
2014;106:375-389. doi: 10.1037/a0034679.

Many students start college intending to pursue a career in the biosciences, but too many abandon this goal because they struggle in introductory biology. Interventions have been developed to close achievement gaps for underrepresented minority students and women, but...

Cite
Harackiewicz JM, Hyde JS, Canning EA, et al. Closing the Social Class Achievement Gap for First-Generation Students in Undergraduate Biology. 2014;106:375-389doi: 10.1037/a0034679.
Harackiewicz, J. M., Hyde, J. S., Canning, E. A., Tibbetts, Y., Giffen, C. J., Blair, S. S., & Rouse, D. I. (2014). Closing the Social Class Achievement Gap for First-Generation Students in Undergraduate Biology. 106375-389. https://doi.org/10.1037/a0034679
Harackiewicz, J M, et al. "Closing the Social Class Achievement Gap for First-Generation Students in Undergraduate Biology." vol. 106 (2014): 375-389. doi: https://doi.org/10.1037/a0034679
Harackiewicz JM, Hyde JS, Canning EA, Tibbetts Y, Giffen CJ, Blair SS, Rouse DI. Closing the Social Class Achievement Gap for First-Generation Students in Undergraduate Biology. 2014;106:375-389. doi: 10.1037/a0034679. UIID-AD: 454.
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Showing 1 to 10 of 10 entries