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Relationship between Active Oxygen Species, Lipid Peroxidation, Necrosis, and Phytoalexin Production Induced by Elicitins in Nicotiana.

Plant physiology

Rusterucci C, Stallaert V, Milat ML, Pugin A, Ricci P, Blein JP.
PMID: 12226334
Plant Physiol. 1996 Jul;111(3):885-891. doi: 10.1104/pp.111.3.885.

Excised leaves of Nicotiana tabacum var Xanthi and Nicotiana rustica were treated with cryptogein and capsicein, basic and acidic elicitins, respectively. Both compounds induced leaf necrosis, the intensity of which depended on concentration and duration of treatment. N. tabacum...

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Rusterucci C, Stallaert V, Milat ML, et al. Relationship between Active Oxygen Species, Lipid Peroxidation, Necrosis, and Phytoalexin Production Induced by Elicitins in Nicotiana. Plant Physiol. 1996;111(3):885-891doi: 10.1104/pp.111.3.885.
Rusterucci, C., Stallaert, V., Milat, M. L., Pugin, A., Ricci, P., & Blein, J. P. (1996). Relationship between Active Oxygen Species, Lipid Peroxidation, Necrosis, and Phytoalexin Production Induced by Elicitins in Nicotiana. Plant physiology, 111(3), 885-891. https://doi.org/10.1104/pp.111.3.885
Rusterucci, C., et al. "Relationship between Active Oxygen Species, Lipid Peroxidation, Necrosis, and Phytoalexin Production Induced by Elicitins in Nicotiana." Plant physiology vol. 111,3 (1996): 885-891. doi: https://doi.org/10.1104/pp.111.3.885
Rusterucci C, Stallaert V, Milat ML, Pugin A, Ricci P, Blein JP. Relationship between Active Oxygen Species, Lipid Peroxidation, Necrosis, and Phytoalexin Production Induced by Elicitins in Nicotiana. Plant Physiol. 1996 Jul;111(3):885-891. doi: 10.1104/pp.111.3.885. PMID: 12226334; PMCID: PMC157907.
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Ligand functional selectivity and quantitative pharmacology at G protein-coupled receptors.

Expert opinion on drug discovery

Stallaert W, Christopoulos A, Bouvier M.
PMID: 22651124
Expert Opin Drug Discov. 2011 Aug;6(8):811-25. doi: 10.1517/17460441.2011.586691. Epub 2011 May 24.

INTRODUCTION: In recent years, it has become clear that individual GPCRs can elicit multiple G-protein-dependent and -independent cellular responses. This has led to the discovery that certain ligands can differentially modulate these responses, a concept known as functional selectivity.AREAS...

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Stallaert W, Christopoulos A, Bouvier M. Ligand functional selectivity and quantitative pharmacology at G protein-coupled receptors. Expert Opin Drug Discov. 2011;6(8):811-25doi: 10.1517/17460441.2011.586691.
Stallaert, W., Christopoulos, A., & Bouvier, M. (2011). Ligand functional selectivity and quantitative pharmacology at G protein-coupled receptors. Expert opinion on drug discovery, 6(8), 811-25. https://doi.org/10.1517/17460441.2011.586691
Stallaert, Wayne, et al. "Ligand functional selectivity and quantitative pharmacology at G protein-coupled receptors." Expert opinion on drug discovery vol. 6,8 (2011): 811-25. doi: https://doi.org/10.1517/17460441.2011.586691
Stallaert W, Christopoulos A, Bouvier M. Ligand functional selectivity and quantitative pharmacology at G protein-coupled receptors. Expert Opin Drug Discov. 2011 Aug;6(8):811-25. doi: 10.1517/17460441.2011.586691. Epub 2011 May 24. PMID: 22651124.
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Cyclin F drives proliferation through SCF-dependent degradation of the retinoblastoma-like tumor suppressor p130/RBL2.

eLife

Enrico TP, Stallaert W, Wick ET, Ngoi P, Wang X, Rubin SM, Brown NG, Purvis JE, Emanuele MJ.
PMID: 34851822
Elife. 2021 Dec 01;10. doi: 10.7554/eLife.70691.

Cell cycle gene expression programs fuel proliferation and are universally dysregulated in cancer. The retinoblastoma (RB)-family of proteins, RB1, RBL1/p107, and RBL2/p130, coordinately represses cell cycle gene expression, inhibiting proliferation, and suppressing tumorigenesis. Phosphorylation of RB-family proteins by cyclin-dependent...

Cite
Enrico TP, Stallaert W, Wick ET, et al. Cyclin F drives proliferation through SCF-dependent degradation of the retinoblastoma-like tumor suppressor p130/RBL2. Elife. 2021;10doi: 10.7554/eLife.70691.
Enrico, T. P., Stallaert, W., Wick, E. T., Ngoi, P., Wang, X., Rubin, S. M., Brown, N. G., Purvis, J. E., & Emanuele, M. J. (2021). Cyclin F drives proliferation through SCF-dependent degradation of the retinoblastoma-like tumor suppressor p130/RBL2. eLife, 10. https://doi.org/10.7554/eLife.70691
Enrico, Taylor P, et al. "Cyclin F drives proliferation through SCF-dependent degradation of the retinoblastoma-like tumor suppressor p130/RBL2." eLife vol. 10 (2021). doi: https://doi.org/10.7554/eLife.70691
Enrico TP, Stallaert W, Wick ET, Ngoi P, Wang X, Rubin SM, Brown NG, Purvis JE, Emanuele MJ. Cyclin F drives proliferation through SCF-dependent degradation of the retinoblastoma-like tumor suppressor p130/RBL2. Elife. 2021 Dec 01;10. doi: 10.7554/eLife.70691. PMID: 34851822; PMCID: PMC8670743.
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Growth factor-dependent ErbB vesicular dynamics couple receptor signaling to spatially and functionally distinct Erk pools.

Science signaling

Brüggemann Y, Karajannis LS, Stanoev A, Stallaert W, Bastiaens PIH.
PMID: 34006609
Sci Signal. 2021 May 18;14(683). doi: 10.1126/scisignal.abd9943.

Growth factor-dependent vesicular dynamics allow cells to regulate the spatial distribution of growth factor receptors and thereby their coupling to downstream signaling effectors that guide cellular responses. We found that the ErbB ligands epidermal growth factor (EGF) and heregulin...

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Brüggemann Y, Karajannis LS, Stanoev A, et al. Growth factor-dependent ErbB vesicular dynamics couple receptor signaling to spatially and functionally distinct Erk pools. Sci Signal. 2021;14(683)doi: 10.1126/scisignal.abd9943.
Brüggemann, Y., Karajannis, L. S., Stanoev, A., Stallaert, W., & Bastiaens, P. I. H. (2021). Growth factor-dependent ErbB vesicular dynamics couple receptor signaling to spatially and functionally distinct Erk pools. Science signaling, 14(683), . https://doi.org/10.1126/scisignal.abd9943
Brüggemann, Yannick, et al. "Growth factor-dependent ErbB vesicular dynamics couple receptor signaling to spatially and functionally distinct Erk pools." Science signaling vol. 14,683 (2021). doi: https://doi.org/10.1126/scisignal.abd9943
Brüggemann Y, Karajannis LS, Stanoev A, Stallaert W, Bastiaens PIH. Growth factor-dependent ErbB vesicular dynamics couple receptor signaling to spatially and functionally distinct Erk pools. Sci Signal. 2021 May 18;14(683). doi: 10.1126/scisignal.abd9943. PMID: 34006609.
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Cyclin F drives proliferation through SCF-dependent degradation of the retinoblastoma-like tumor suppressor p130/RBL2.

eLife

Enrico TP, Stallaert W, Wick ET, Ngoi P, Wang X, Rubin SM, Brown NG, Purvis JE, Emanuele MJ.
PMID: 34851822
Elife. 2021 Dec 01;10. doi: 10.7554/eLife.70691.

Cell cycle gene expression programs fuel proliferation and are universally dysregulated in cancer. The retinoblastoma (RB)-family of proteins, RB1, RBL1/p107, and RBL2/p130, coordinately represses cell cycle gene expression, inhibiting proliferation, and suppressing tumorigenesis. Phosphorylation of RB-family proteins by cyclin-dependent...

Cite
Enrico TP, Stallaert W, Wick ET, et al. Cyclin F drives proliferation through SCF-dependent degradation of the retinoblastoma-like tumor suppressor p130/RBL2. Elife. 2021;10doi: 10.7554/eLife.70691.
Enrico, T. P., Stallaert, W., Wick, E. T., Ngoi, P., Wang, X., Rubin, S. M., Brown, N. G., Purvis, J. E., & Emanuele, M. J. (2021). Cyclin F drives proliferation through SCF-dependent degradation of the retinoblastoma-like tumor suppressor p130/RBL2. eLife, 10. https://doi.org/10.7554/eLife.70691
Enrico, Taylor P, et al. "Cyclin F drives proliferation through SCF-dependent degradation of the retinoblastoma-like tumor suppressor p130/RBL2." eLife vol. 10 (2021). doi: https://doi.org/10.7554/eLife.70691
Enrico TP, Stallaert W, Wick ET, Ngoi P, Wang X, Rubin SM, Brown NG, Purvis JE, Emanuele MJ. Cyclin F drives proliferation through SCF-dependent degradation of the retinoblastoma-like tumor suppressor p130/RBL2. Elife. 2021 Dec 01;10. doi: 10.7554/eLife.70691. PMID: 34851822.
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Growth factor-dependent ErbB vesicular dynamics couple receptor signaling to spatially and functionally distinct Erk pools.

Science signaling

Brüggemann Y, Karajannis LS, Stanoev A, Stallaert W, Bastiaens PIH.
PMID: 34006609
Sci Signal. 2021 May 18;14(683). doi: 10.1126/scisignal.abd9943.

Growth factor-dependent vesicular dynamics allow cells to regulate the spatial distribution of growth factor receptors and thereby their coupling to downstream signaling effectors that guide cellular responses. We found that the ErbB ligands epidermal growth factor (EGF) and heregulin...

Cite
Brüggemann Y, Karajannis LS, Stanoev A, et al. Growth factor-dependent ErbB vesicular dynamics couple receptor signaling to spatially and functionally distinct Erk pools. Sci Signal. 2021;14(683)doi: 10.1126/scisignal.abd9943.
Brüggemann, Y., Karajannis, L. S., Stanoev, A., Stallaert, W., & Bastiaens, P. I. H. (2021). Growth factor-dependent ErbB vesicular dynamics couple receptor signaling to spatially and functionally distinct Erk pools. Science signaling, 14(683), . https://doi.org/10.1126/scisignal.abd9943
Brüggemann, Yannick, et al. "Growth factor-dependent ErbB vesicular dynamics couple receptor signaling to spatially and functionally distinct Erk pools." Science signaling vol. 14,683 (2021). doi: https://doi.org/10.1126/scisignal.abd9943
Brüggemann Y, Karajannis LS, Stanoev A, Stallaert W, Bastiaens PIH. Growth factor-dependent ErbB vesicular dynamics couple receptor signaling to spatially and functionally distinct Erk pools. Sci Signal. 2021 May 18;14(683). doi: 10.1126/scisignal.abd9943. PMID: 34006609.
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The structure of the human cell cycle.

Cell systems

Stallaert W, Kedziora KM, Taylor CD, Zikry TM, Ranek JS, Sobon HK, Taylor SR, Young CL, Cook JG, Purvis JE.
PMID: 34800361
Cell Syst. 2021 Nov 17; doi: 10.1016/j.cels.2021.10.007. Epub 2021 Nov 17.

Understanding the organization of the cell cycle has been a longstanding goal in cell biology. We combined time-lapse microscopy, highly multiplexed single-cell imaging of 48 core cell cycle proteins, and manifold learning to render a visualization of the human...

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Stallaert W, Kedziora KM, Taylor CD, et al. The structure of the human cell cycle. Cell Syst. 2021;doi: 10.1016/j.cels.2021.10.007.
Stallaert, W., Kedziora, K. M., Taylor, C. D., Zikry, T. M., Ranek, J. S., Sobon, H. K., Taylor, S. R., Young, C. L., Cook, J. G., & Purvis, J. E. (2021). The structure of the human cell cycle. Cell systems, . https://doi.org/10.1016/j.cels.2021.10.007
Stallaert, Wayne, et al. "The structure of the human cell cycle." Cell systems vol. (2021). doi: https://doi.org/10.1016/j.cels.2021.10.007
Stallaert W, Kedziora KM, Taylor CD, Zikry TM, Ranek JS, Sobon HK, Taylor SR, Young CL, Cook JG, Purvis JE. The structure of the human cell cycle. Cell Syst. 2021 Nov 17; doi: 10.1016/j.cels.2021.10.007. Epub 2021 Nov 17. PMID: 34800361.
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Cyclin F drives proliferation through SCF-dependent degradation of the retinoblastoma-like tumor suppressor p130/RBL2.

eLife

Enrico TP, Stallaert W, Wick ET, Ngoi P, Wang X, Rubin SM, Brown NG, Purvis J, Emanuele MJ.
PMID: 34851822
Elife. 2021 Dec 01;10. doi: 10.7554/eLife.70691. Epub 2021 Dec 01.

Cell cycle gene expression programs fuel proliferation and are universally dysregulated in cancer. The retinoblastoma (RB)-family of proteins, RB1, RBL1/p107 and RBL2/p130, coordinately repress cell cycle gene expression, inhibiting proliferation and suppressing tumorigenesis. Phosphorylation of RB-family proteins by cyclin...

Cite
Enrico TP, Stallaert W, Wick ET, et al. Cyclin F drives proliferation through SCF-dependent degradation of the retinoblastoma-like tumor suppressor p130/RBL2. Elife. 2021;10doi: 10.7554/eLife.70691.
Enrico, T. P., Stallaert, W., Wick, E. T., Ngoi, P., Wang, X., Rubin, S. M., Brown, N. G., Purvis, J., & Emanuele, M. J. (2021). Cyclin F drives proliferation through SCF-dependent degradation of the retinoblastoma-like tumor suppressor p130/RBL2. eLife, 10. https://doi.org/10.7554/eLife.70691
Enrico, Taylor P, et al. "Cyclin F drives proliferation through SCF-dependent degradation of the retinoblastoma-like tumor suppressor p130/RBL2." eLife vol. 10 (2021). doi: https://doi.org/10.7554/eLife.70691
Enrico TP, Stallaert W, Wick ET, Ngoi P, Wang X, Rubin SM, Brown NG, Purvis J, Emanuele MJ. Cyclin F drives proliferation through SCF-dependent degradation of the retinoblastoma-like tumor suppressor p130/RBL2. Elife. 2021 Dec 01;10. doi: 10.7554/eLife.70691. Epub 2021 Dec 01. PMID: 34851822.
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Cyclin F drives proliferation through SCF-dependent degradation of the retinoblastoma-like tumor suppressor p130/RBL2.

eLife

Enrico TP, Stallaert W, Wick ET, Ngoi P, Wang X, Rubin SM, Brown NG, Purvis JE, Emanuele MJ.
PMID: 34851822
Elife. 2021 Dec 01;10. doi: 10.7554/eLife.70691.

Cell cycle gene expression programs fuel proliferation and are universally dysregulated in cancer. The retinoblastoma (RB)-family of proteins, RB1, RBL1/p107, and RBL2/p130, coordinately represses cell cycle gene expression, inhibiting proliferation, and suppressing tumorigenesis. Phosphorylation of RB-family proteins by cyclin-dependent...

Cite
Enrico TP, Stallaert W, Wick ET, et al. Cyclin F drives proliferation through SCF-dependent degradation of the retinoblastoma-like tumor suppressor p130/RBL2. Elife. 2021;10doi: 10.7554/eLife.70691.
Enrico, T. P., Stallaert, W., Wick, E. T., Ngoi, P., Wang, X., Rubin, S. M., Brown, N. G., Purvis, J. E., & Emanuele, M. J. (2021). Cyclin F drives proliferation through SCF-dependent degradation of the retinoblastoma-like tumor suppressor p130/RBL2. eLife, 10. https://doi.org/10.7554/eLife.70691
Enrico, Taylor P, et al. "Cyclin F drives proliferation through SCF-dependent degradation of the retinoblastoma-like tumor suppressor p130/RBL2." eLife vol. 10 (2021). doi: https://doi.org/10.7554/eLife.70691
Enrico TP, Stallaert W, Wick ET, Ngoi P, Wang X, Rubin SM, Brown NG, Purvis JE, Emanuele MJ. Cyclin F drives proliferation through SCF-dependent degradation of the retinoblastoma-like tumor suppressor p130/RBL2. Elife. 2021 Dec 01;10. doi: 10.7554/eLife.70691. PMID: 34851822; PMCID: PMC8670743.
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Cyclin F drives proliferation through SCF-dependent degradation of the retinoblastoma-like tumor suppressor p130/RBL2.

eLife

Enrico TP, Stallaert W, Wick ET, Ngoi P, Wang X, Rubin SM, Brown NG, Purvis JE, Emanuele MJ.
PMID: 34851822
Elife. 2021 Dec 01;10. doi: 10.7554/eLife.70691.

Cell cycle gene expression programs fuel proliferation and are universally dysregulated in cancer. The retinoblastoma (RB)-family of proteins, RB1, RBL1/p107, and RBL2/p130, coordinately represses cell cycle gene expression, inhibiting proliferation, and suppressing tumorigenesis. Phosphorylation of RB-family proteins by cyclin-dependent...

Cite
Enrico TP, Stallaert W, Wick ET, et al. Cyclin F drives proliferation through SCF-dependent degradation of the retinoblastoma-like tumor suppressor p130/RBL2. Elife. 2021;10doi: 10.7554/eLife.70691.
Enrico, T. P., Stallaert, W., Wick, E. T., Ngoi, P., Wang, X., Rubin, S. M., Brown, N. G., Purvis, J. E., & Emanuele, M. J. (2021). Cyclin F drives proliferation through SCF-dependent degradation of the retinoblastoma-like tumor suppressor p130/RBL2. eLife, 10. https://doi.org/10.7554/eLife.70691
Enrico, Taylor P, et al. "Cyclin F drives proliferation through SCF-dependent degradation of the retinoblastoma-like tumor suppressor p130/RBL2." eLife vol. 10 (2021). doi: https://doi.org/10.7554/eLife.70691
Enrico TP, Stallaert W, Wick ET, Ngoi P, Wang X, Rubin SM, Brown NG, Purvis JE, Emanuele MJ. Cyclin F drives proliferation through SCF-dependent degradation of the retinoblastoma-like tumor suppressor p130/RBL2. Elife. 2021 Dec 01;10. doi: 10.7554/eLife.70691. PMID: 34851822; PMCID: PMC8670743.
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Cyclin F drives proliferation through SCF-dependent degradation of the retinoblastoma-like tumor suppressor p130/RBL2.

eLife

Enrico TP, Stallaert W, Wick ET, Ngoi P, Wang X, Rubin SM, Brown NG, Purvis JE, Emanuele MJ.
PMID: 34851822
Elife. 2021 Dec 01;10. doi: 10.7554/eLife.70691.

Cell cycle gene expression programs fuel proliferation and are universally dysregulated in cancer. The retinoblastoma (RB)-family of proteins, RB1, RBL1/p107, and RBL2/p130, coordinately represses cell cycle gene expression, inhibiting proliferation, and suppressing tumorigenesis. Phosphorylation of RB-family proteins by cyclin-dependent...

Cite
Enrico TP, Stallaert W, Wick ET, et al. Cyclin F drives proliferation through SCF-dependent degradation of the retinoblastoma-like tumor suppressor p130/RBL2. Elife. 2021;10doi: 10.7554/eLife.70691.
Enrico, T. P., Stallaert, W., Wick, E. T., Ngoi, P., Wang, X., Rubin, S. M., Brown, N. G., Purvis, J. E., & Emanuele, M. J. (2021). Cyclin F drives proliferation through SCF-dependent degradation of the retinoblastoma-like tumor suppressor p130/RBL2. eLife, 10. https://doi.org/10.7554/eLife.70691
Enrico, Taylor P, et al. "Cyclin F drives proliferation through SCF-dependent degradation of the retinoblastoma-like tumor suppressor p130/RBL2." eLife vol. 10 (2021). doi: https://doi.org/10.7554/eLife.70691
Enrico TP, Stallaert W, Wick ET, Ngoi P, Wang X, Rubin SM, Brown NG, Purvis JE, Emanuele MJ. Cyclin F drives proliferation through SCF-dependent degradation of the retinoblastoma-like tumor suppressor p130/RBL2. Elife. 2021 Dec 01;10. doi: 10.7554/eLife.70691. PMID: 34851822.
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Showing 1 to 11 of 11 entries