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The Influence of Explanations for Selection Test Use, Outcome Favorability, and Self-Efficacy on Test-Taker Perceptions.

Organizational behavior and human decision processes

Horvath M, Ryan AM, Stierwalt SL.
PMID: 11056073
Organ Behav Hum Decis Process. 2000 Nov;83(2):310-330. doi: 10.1006/obhd.2000.2911.

Previous research has shown that an explanation for an event can affect reactions to that event. This study examined this effect within a selection context by varying the type of explanation (causal, ideological, referential) and outcome favorability. A sample...

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Horvath M, Ryan AM, Stierwalt SL. The Influence of Explanations for Selection Test Use, Outcome Favorability, and Self-Efficacy on Test-Taker Perceptions. Organ Behav Hum Decis Process. 2000;83(2):310-330doi: 10.1006/obhd.2000.2911.
Horvath, M., Ryan, A. M., & Stierwalt, S. L. (2000). The Influence of Explanations for Selection Test Use, Outcome Favorability, and Self-Efficacy on Test-Taker Perceptions. Organizational behavior and human decision processes, 83(2), 310-330. https://doi.org/10.1006/obhd.2000.2911
Horvath, et al. "The Influence of Explanations for Selection Test Use, Outcome Favorability, and Self-Efficacy on Test-Taker Perceptions." Organizational behavior and human decision processes vol. 83,2 (2000): 310-330. doi: https://doi.org/10.1006/obhd.2000.2911
Horvath M, Ryan AM, Stierwalt SL. The Influence of Explanations for Selection Test Use, Outcome Favorability, and Self-Efficacy on Test-Taker Perceptions. Organ Behav Hum Decis Process. 2000 Nov;83(2):310-330. doi: 10.1006/obhd.2000.2911. PMID: 11056073.
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Peptidyl-tRNA and peptidyl transferase activity of skeletal muscle ribosomes. Effect of diabetes.

FEBS letters

Stirewalt WS, Wool IG.
PMID: 11945351
FEBS Lett. 1970 Sep 18;10(1):38-40. doi: 10.1016/0014-5793(70)80410-0.

No abstract available.

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Stirewalt WS, Wool IG. Peptidyl-tRNA and peptidyl transferase activity of skeletal muscle ribosomes. Effect of diabetes. FEBS Lett. 1970;10(1):38-40doi: 10.1016/0014-5793(70)80410-0.
Stirewalt, W. S., & Wool, I. G. (1970). Peptidyl-tRNA and peptidyl transferase activity of skeletal muscle ribosomes. Effect of diabetes. FEBS letters, 10(1), 38-40. https://doi.org/10.1016/0014-5793(70)80410-0
Stirewalt, William S., and Wool, Ira G.. "Peptidyl-tRNA and peptidyl transferase activity of skeletal muscle ribosomes. Effect of diabetes." FEBS letters vol. 10,1 (1970): 38-40. doi: https://doi.org/10.1016/0014-5793(70)80410-0
Stirewalt WS, Wool IG. Peptidyl-tRNA and peptidyl transferase activity of skeletal muscle ribosomes. Effect of diabetes. FEBS Lett. 1970 Sep 18;10(1):38-40. doi: 10.1016/0014-5793(70)80410-0. PMID: 11945351.
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The proteins of liver and muscle ribosomal subunits: Partial separation by carboxymethyl-cellulose column chromatography.

FEBS letters

Kanai K, Castles JJ, Wool IG, Stirewalt WS, Kanai A.
PMID: 11947241
FEBS Lett. 1969 Sep;5(1):68-72. doi: 10.1016/0014-5793(69)80295-4.

No abstract available.

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Kanai K, Castles JJ, Wool IG, et al. The proteins of liver and muscle ribosomal subunits: Partial separation by carboxymethyl-cellulose column chromatography. FEBS Lett. 1969;5(1):68-72doi: 10.1016/0014-5793(69)80295-4.
Kanai, K., Castles, J. J., Wool, I. G., Stirewalt, W. S., & Kanai, A. (1969). The proteins of liver and muscle ribosomal subunits: Partial separation by carboxymethyl-cellulose column chromatography. FEBS letters, 5(1), 68-72. https://doi.org/10.1016/0014-5793(69)80295-4
Kanai, K, et al. "The proteins of liver and muscle ribosomal subunits: Partial separation by carboxymethyl-cellulose column chromatography." FEBS letters vol. 5,1 (1969): 68-72. doi: https://doi.org/10.1016/0014-5793(69)80295-4
Kanai K, Castles JJ, Wool IG, Stirewalt WS, Kanai A. The proteins of liver and muscle ribosomal subunits: Partial separation by carboxymethyl-cellulose column chromatography. FEBS Lett. 1969 Sep;5(1):68-72. doi: 10.1016/0014-5793(69)80295-4. PMID: 11947241.
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AML risk stratification models utilizing ELN-2017 guidelines and additional prognostic factors: a SWOG report.

Biomarker research

Pogosova-Agadjanyan EL, Moseley A, Othus M, Appelbaum FR, Chauncey TR, Chen IL, Erba HP, Godwin JE, Jenkins IC, Fang M, Huynh M, Kopecky KJ, List AF, Naru J, Radich JP, Stevens E, Willborg BE, Willman CL, Wood BL, Zhang Q, Meshinchi S, Stirewalt DL.
PMID: 32817791
Biomark Res. 2020 Aug 12;8:29. doi: 10.1186/s40364-020-00208-1. eCollection 2020.

BACKGROUND: The recently updated European LeukemiaNet risk stratification guidelines combine cytogenetic abnormalities and genetic mutations to provide the means to triage patients with acute myeloid leukemia for optimal therapies. Despite the identification of many prognostic factors, relatively few have...

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Pogosova-Agadjanyan EL, Moseley A, Othus M, et al. AML risk stratification models utilizing ELN-2017 guidelines and additional prognostic factors: a SWOG report. Biomark Res. 2020;8:29doi: 10.1186/s40364-020-00208-1.
Pogosova-Agadjanyan, E. L., Moseley, A., Othus, M., Appelbaum, F. R., Chauncey, T. R., Chen, I. L., Erba, H. P., Godwin, J. E., Jenkins, I. C., Fang, M., Huynh, M., Kopecky, K. J., List, A. F., Naru, J., Radich, J. P., Stevens, E., Willborg, B. E., Willman, C. L., Wood, B. L., Zhang, Q., Meshinchi, S., & Stirewalt, D. L. (2020). AML risk stratification models utilizing ELN-2017 guidelines and additional prognostic factors: a SWOG report. Biomarker research, 829. https://doi.org/10.1186/s40364-020-00208-1
Pogosova-Agadjanyan, Era L, et al. "AML risk stratification models utilizing ELN-2017 guidelines and additional prognostic factors: a SWOG report." Biomarker research vol. 8 (2020): 29. doi: https://doi.org/10.1186/s40364-020-00208-1
Pogosova-Agadjanyan EL, Moseley A, Othus M, Appelbaum FR, Chauncey TR, Chen IL, Erba HP, Godwin JE, Jenkins IC, Fang M, Huynh M, Kopecky KJ, List AF, Naru J, Radich JP, Stevens E, Willborg BE, Willman CL, Wood BL, Zhang Q, Meshinchi S, Stirewalt DL. AML risk stratification models utilizing ELN-2017 guidelines and additional prognostic factors: a SWOG report. Biomark Res. 2020 Aug 12;8:29. doi: 10.1186/s40364-020-00208-1. eCollection 2020. PMID: 32817791; PMCID: PMC7425159.
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Coordinated mis-splicing of TMEM14C and ABCB7 causes ring sideroblast formation in SF3B1-mutant myelodysplastic syndrome.

Blood

Clough CA, Pangallo J, Sarchi M, Ilagan JO, North K, Bergantinos R, Stolla MC, Naru J, Nugent P, Kim E, Stirewalt DL, Subramaniam AR, Abdel-Wahab O, Abkowitz JL, Bradley RK, Doulatov S.
PMID: 34861039
Blood. 2021 Dec 03; doi: 10.1182/blood.2021012652. Epub 2021 Dec 03.

SF3B1 splicing factor mutations are near-universally found in myelodysplastic syndromes (MDS) with ring sideroblasts, a clonal hematopoietic disorder characterized by abnormal erythroid cells with iron-loaded mitochondria. Despite this remarkably strong genotype-to-phenotype correlation, the mechanism by which mutant SF3B1 dysregulates...

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Clough CA, Pangallo J, Sarchi M, et al. Coordinated mis-splicing of TMEM14C and ABCB7 causes ring sideroblast formation in SF3B1-mutant myelodysplastic syndrome. Blood. 2021;doi: 10.1182/blood.2021012652.
Clough, C. A., Pangallo, J., Sarchi, M., Ilagan, J. O., North, K., Bergantinos, R., Stolla, M. C., Naru, J., Nugent, P., Kim, E., Stirewalt, D. L., Subramaniam, A. R., Abdel-Wahab, O., Abkowitz, J. L., Bradley, R. K., & Doulatov, S. (2021). Coordinated mis-splicing of TMEM14C and ABCB7 causes ring sideroblast formation in SF3B1-mutant myelodysplastic syndrome. Blood, . https://doi.org/10.1182/blood.2021012652
Clough, Courtnee A, et al. "Coordinated mis-splicing of TMEM14C and ABCB7 causes ring sideroblast formation in SF3B1-mutant myelodysplastic syndrome." Blood vol. (2021). doi: https://doi.org/10.1182/blood.2021012652
Clough CA, Pangallo J, Sarchi M, Ilagan JO, North K, Bergantinos R, Stolla MC, Naru J, Nugent P, Kim E, Stirewalt DL, Subramaniam AR, Abdel-Wahab O, Abkowitz JL, Bradley RK, Doulatov S. Coordinated mis-splicing of TMEM14C and ABCB7 causes ring sideroblast formation in SF3B1-mutant myelodysplastic syndrome. Blood. 2021 Dec 03; doi: 10.1182/blood.2021012652. Epub 2021 Dec 03. PMID: 34861039.
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NCCN Guidelines® Insights: Older Adult Oncology, Version 1.2021.

Journal of the National Comprehensive Cancer Network : JNCCN

Dotan E, Walter LC, Browner IS, Clifton K, Cohen HJ, Extermann M, Gross C, Gupta S, Hollis G, Hubbard J, Jagsi R, Keating NL, Kessler E, Koll T, Korc-Grodzicki B, McKoy JM, Misra S, Moon D, O'Connor T, Owusu C, Rosko A, Russell M, Sedrak M, Siddiqui F, Stella A, Stirewalt DL, Subbiah IM, Tew WP, Williams GR, Hollinger L, George GV, Sundar H.
PMID: 34551388
J Natl Compr Canc Netw. 2021 Sep 20;19(9):1006-1019. doi: 10.6004/jnccn.2021.0043.

The NCCN Guidelines for Older Adult Oncology address specific issues related to the management of cancer in older adults, including screening and comprehensive geriatric assessment (CGA), assessing the risks and benefits of treatment, preventing or decreasing complications from therapy,...

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Dotan E, Walter LC, Browner IS, et al. NCCN Guidelines® Insights: Older Adult Oncology, Version 1.2021. J Natl Compr Canc Netw. 2021;19(9):1006-1019doi: 10.6004/jnccn.2021.0043.
Dotan, E., Walter, L. C., Browner, I. S., Clifton, K., Cohen, H. J., Extermann, M., Gross, C., Gupta, S., Hollis, G., Hubbard, J., Jagsi, R., Keating, N. L., Kessler, E., Koll, T., Korc-Grodzicki, B., McKoy, J. M., Misra, S., Moon, D., O'Connor, T., Owusu, C., Rosko, A., Russell, M., Sedrak, M., Siddiqui, F., Stella, A., Stirewalt, D. L., Subbiah, I. M., Tew, W. P., Williams, G. R., Hollinger, L., George, G. V., & Sundar, H. (2021). NCCN Guidelines® Insights: Older Adult Oncology, Version 1.2021. Journal of the National Comprehensive Cancer Network : JNCCN, 19(9), 1006-1019. https://doi.org/10.6004/jnccn.2021.0043
Dotan, Efrat, et al. "NCCN Guidelines® Insights: Older Adult Oncology, Version 1.2021." Journal of the National Comprehensive Cancer Network : JNCCN vol. 19,9 (2021): 1006-1019. doi: https://doi.org/10.6004/jnccn.2021.0043
Dotan E, Walter LC, Browner IS, Clifton K, Cohen HJ, Extermann M, Gross C, Gupta S, Hollis G, Hubbard J, Jagsi R, Keating NL, Kessler E, Koll T, Korc-Grodzicki B, McKoy JM, Misra S, Moon D, O'Connor T, Owusu C, Rosko A, Russell M, Sedrak M, Siddiqui F, Stella A, Stirewalt DL, Subbiah IM, Tew WP, Williams GR, Hollinger L, George GV, Sundar H. NCCN Guidelines® Insights: Older Adult Oncology, Version 1.2021. J Natl Compr Canc Netw. 2021 Sep 20;19(9):1006-1019. doi: 10.6004/jnccn.2021.0043. PMID: 34551388.
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Malignancy: Tumor Suppressor Gene Aberrations in Acute Myelogenous Leukemia.

Hematology (Amsterdam, Netherlands)

Stirewalt DL, Radich JP.
PMID: 11399598
Hematology. 2000;5(1):15-25. doi: 10.1080/10245332.2000.11746484.

Acute myelogenous leukemia is a heterogeneous disease that appears to evade the normal regulatory controls of tumor suppressor genes. Studies in AML have documented mutations in both p53 and Retinoblastoma (Rb) genes, but these mutations are relatively uncommon, especially...

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Stirewalt DL, Radich JP. Malignancy: Tumor Suppressor Gene Aberrations in Acute Myelogenous Leukemia. Hematology. 2000;5(1):15-25doi: 10.1080/10245332.2000.11746484.
Stirewalt, D. L., & Radich, J. P. (2000). Malignancy: Tumor Suppressor Gene Aberrations in Acute Myelogenous Leukemia. Hematology (Amsterdam, Netherlands), 5(1), 15-25. https://doi.org/10.1080/10245332.2000.11746484
Stirewalt, DEREK L., and Radich, JERALD P.. "Malignancy: Tumor Suppressor Gene Aberrations in Acute Myelogenous Leukemia." Hematology (Amsterdam, Netherlands) vol. 5,1 (2000): 15-25. doi: https://doi.org/10.1080/10245332.2000.11746484
Stirewalt DL, Radich JP. Malignancy: Tumor Suppressor Gene Aberrations in Acute Myelogenous Leukemia. Hematology. 2000;5(1):15-25. doi: 10.1080/10245332.2000.11746484. PMID: 11399598.
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Coordinated mis-splicing of TMEM14C and ABCB7 causes ring sideroblast formation in SF3B1-mutant myelodysplastic syndrome.

Blood

Clough CA, Pangallo J, Sarchi M, Ilagan JO, North K, Bergantinos R, Stolla MC, Naru J, Nugent P, Kim E, Stirewalt DL, Subramaniam AR, Abdel-Wahab O, Abkowitz JL, Bradley RK, Doulatov S.
PMID: 34861039
Blood. 2021 Dec 03; doi: 10.1182/blood.2021012652. Epub 2021 Dec 03.

SF3B1 splicing factor mutations are near-universally found in myelodysplastic syndromes (MDS) with ring sideroblasts, a clonal hematopoietic disorder characterized by abnormal erythroid cells with iron-loaded mitochondria. Despite this remarkably strong genotype-to-phenotype correlation, the mechanism by which mutant SF3B1 dysregulates...

Cite
Clough CA, Pangallo J, Sarchi M, et al. Coordinated mis-splicing of TMEM14C and ABCB7 causes ring sideroblast formation in SF3B1-mutant myelodysplastic syndrome. Blood. 2021;doi: 10.1182/blood.2021012652.
Clough, C. A., Pangallo, J., Sarchi, M., Ilagan, J. O., North, K., Bergantinos, R., Stolla, M. C., Naru, J., Nugent, P., Kim, E., Stirewalt, D. L., Subramaniam, A. R., Abdel-Wahab, O., Abkowitz, J. L., Bradley, R. K., & Doulatov, S. (2021). Coordinated mis-splicing of TMEM14C and ABCB7 causes ring sideroblast formation in SF3B1-mutant myelodysplastic syndrome. Blood, . https://doi.org/10.1182/blood.2021012652
Clough, Courtnee A, et al. "Coordinated mis-splicing of TMEM14C and ABCB7 causes ring sideroblast formation in SF3B1-mutant myelodysplastic syndrome." Blood vol. (2021). doi: https://doi.org/10.1182/blood.2021012652
Clough CA, Pangallo J, Sarchi M, Ilagan JO, North K, Bergantinos R, Stolla MC, Naru J, Nugent P, Kim E, Stirewalt DL, Subramaniam AR, Abdel-Wahab O, Abkowitz JL, Bradley RK, Doulatov S. Coordinated mis-splicing of TMEM14C and ABCB7 causes ring sideroblast formation in SF3B1-mutant myelodysplastic syndrome. Blood. 2021 Dec 03; doi: 10.1182/blood.2021012652. Epub 2021 Dec 03. PMID: 34861039.
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Coordinated mis-splicing of TMEM14C and ABCB7 causes ring sideroblast formation in SF3B1-mutant myelodysplastic syndrome.

Blood

Clough CA, Pangallo J, Sarchi M, Ilagan JO, North K, Bergantinos R, Stolla MC, Naru J, Nugent P, Kim E, Stirewalt DL, Subramaniam AR, Abdel-Wahab O, Abkowitz JL, Bradley RK, Doulatov S.
PMID: 34861039
Blood. 2021 Dec 03; doi: 10.1182/blood.2021012652. Epub 2021 Dec 03.

SF3B1 splicing factor mutations are near-universally found in myelodysplastic syndromes (MDS) with ring sideroblasts, a clonal hematopoietic disorder characterized by abnormal erythroid cells with iron-loaded mitochondria. Despite this remarkably strong genotype-to-phenotype correlation, the mechanism by which mutant SF3B1 dysregulates...

Cite
Clough CA, Pangallo J, Sarchi M, et al. Coordinated mis-splicing of TMEM14C and ABCB7 causes ring sideroblast formation in SF3B1-mutant myelodysplastic syndrome. Blood. 2021;doi: 10.1182/blood.2021012652.
Clough, C. A., Pangallo, J., Sarchi, M., Ilagan, J. O., North, K., Bergantinos, R., Stolla, M. C., Naru, J., Nugent, P., Kim, E., Stirewalt, D. L., Subramaniam, A. R., Abdel-Wahab, O., Abkowitz, J. L., Bradley, R. K., & Doulatov, S. (2021). Coordinated mis-splicing of TMEM14C and ABCB7 causes ring sideroblast formation in SF3B1-mutant myelodysplastic syndrome. Blood, . https://doi.org/10.1182/blood.2021012652
Clough, Courtnee A, et al. "Coordinated mis-splicing of TMEM14C and ABCB7 causes ring sideroblast formation in SF3B1-mutant myelodysplastic syndrome." Blood vol. (2021). doi: https://doi.org/10.1182/blood.2021012652
Clough CA, Pangallo J, Sarchi M, Ilagan JO, North K, Bergantinos R, Stolla MC, Naru J, Nugent P, Kim E, Stirewalt DL, Subramaniam AR, Abdel-Wahab O, Abkowitz JL, Bradley RK, Doulatov S. Coordinated mis-splicing of TMEM14C and ABCB7 causes ring sideroblast formation in SF3B1-mutant myelodysplastic syndrome. Blood. 2021 Dec 03; doi: 10.1182/blood.2021012652. Epub 2021 Dec 03. PMID: 34861039.
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Family Conference Simulation Designed for Physician Assistant Students and Chaplain Residents.

Journal of palliative medicine

Grant E, Sisson CB, Hiatt TL, Stirewalt FK, Crandall SJ.
PMID: 34042524
J Palliat Med. 2021 Dec;24(12):1816-1822. doi: 10.1089/jpm.2019.0563. Epub 2021 May 25.

No abstract available.

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Grant E, Sisson CB, Hiatt TL, et al. Family Conference Simulation Designed for Physician Assistant Students and Chaplain Residents. J Palliat Med. 2021;24(12):1816-1822doi: 10.1089/jpm.2019.0563.
Grant, E., Sisson, C. B., Hiatt, T. L., Stirewalt, F. K., & Crandall, S. J. (2021). Family Conference Simulation Designed for Physician Assistant Students and Chaplain Residents. Journal of palliative medicine, 24(12), 1816-1822. https://doi.org/10.1089/jpm.2019.0563
Grant, Erich, et al. "Family Conference Simulation Designed for Physician Assistant Students and Chaplain Residents." Journal of palliative medicine vol. 24,12 (2021): 1816-1822. doi: https://doi.org/10.1089/jpm.2019.0563
Grant E, Sisson CB, Hiatt TL, Stirewalt FK, Crandall SJ. Family Conference Simulation Designed for Physician Assistant Students and Chaplain Residents. J Palliat Med. 2021 Dec;24(12):1816-1822. doi: 10.1089/jpm.2019.0563. Epub 2021 May 25. PMID: 34042524.
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Infrared spectral emittance measurements of optical materials.

Applied optics

Stierwalt DL.
PMID: 20057659
Appl Opt. 1966 Dec 01;5(12):1911-5. doi: 10.1364/AO.5.001911.

Optical properties of solids are usually determined by measurements of either the transmittance or the reflectance. Each of these methods has advantages and disadvantages, depending upon the spectral region and the nature of the material being studied. A third...

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Stierwalt DL. Infrared spectral emittance measurements of optical materials. Appl Opt. 1966;5(12):1911-5doi: 10.1364/AO.5.001911.
Stierwalt, D. L. (1966). Infrared spectral emittance measurements of optical materials. Applied optics, 5(12), 1911-5. https://doi.org/10.1364/AO.5.001911
Stierwalt, D L. "Infrared spectral emittance measurements of optical materials." Applied optics vol. 5,12 (1966): 1911-5. doi: https://doi.org/10.1364/AO.5.001911
Stierwalt DL. Infrared spectral emittance measurements of optical materials. Appl Opt. 1966 Dec 01;5(12):1911-5. doi: 10.1364/AO.5.001911. PMID: 20057659.
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Surface and bulk absorption characteristics of chemically vapor-deposited zinc selenide in the infrared.

Applied optics

Klein CA, Miller RP, Stierwalt DL.
PMID: 20935788
Appl Opt. 1994 Jul 01;33(19):4304-13. doi: 10.1364/AO.33.004304.

Chemically vapor-deposited zinc selenide exhibits outstanding properties in the infrared and has been established as a prime material for transmissive optics applications. Here we present and discuss data relating to the surface and the bulk absorption forward-looking infrared- (FLIR-)...

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Klein CA, Miller RP, Stierwalt DL. Surface and bulk absorption characteristics of chemically vapor-deposited zinc selenide in the infrared. Appl Opt. 1994;33(19):4304-13doi: 10.1364/AO.33.004304.
Klein, C. A., Miller, R. P., & Stierwalt, D. L. (1994). Surface and bulk absorption characteristics of chemically vapor-deposited zinc selenide in the infrared. Applied optics, 33(19), 4304-13. https://doi.org/10.1364/AO.33.004304
Klein, C A, et al. "Surface and bulk absorption characteristics of chemically vapor-deposited zinc selenide in the infrared." Applied optics vol. 33,19 (1994): 4304-13. doi: https://doi.org/10.1364/AO.33.004304
Klein CA, Miller RP, Stierwalt DL. Surface and bulk absorption characteristics of chemically vapor-deposited zinc selenide in the infrared. Appl Opt. 1994 Jul 01;33(19):4304-13. doi: 10.1364/AO.33.004304. PMID: 20935788.
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