Advanced Search
Display options
Filter resources
Text Availability
Article type
Publication date
Species
Language
Sex
Age
Showing 1 to 6 of 6 entries
Sorted by: Best Match Show Resources per page
Thymosin β4 Is an Endogenous Iron Chelator and Molecular Switcher of Ferroptosis.

International journal of molecular sciences

Lachowicz JI, Pichiri G, Piludu M, Fais S, Orrù G, Congiu T, Piras M, Faa G, Fanni D, Dalla Torre G, Lopez X, Chandra K, Szczepski K, Jaremko L, Ghosh M, Emwas AH, Castagnola M, Jaremko M, Hannappel E, Coni P.
PMID: 35008976
Int J Mol Sci. 2022 Jan 04;23(1). doi: 10.3390/ijms23010551.

Thymosin β4 (Tβ4) was extracted forty years agofrom calf thymus. Since then, it has been identified as a G-actin binding protein involved in blood clotting, tissue regeneration, angiogenesis, and anti-inflammatory processes. Tβ4 has also been implicated in tumor metastasis...

Cite
Lachowicz JI, Pichiri G, Piludu M, et al. Thymosin β4 Is an Endogenous Iron Chelator and Molecular Switcher of Ferroptosis. Int J Mol Sci. 2022;23(1)doi: 10.3390/ijms23010551.
Lachowicz, J. I., Pichiri, G., Piludu, M., Fais, S., Orrù, G., Congiu, T., Piras, M., Faa, G., Fanni, D., Dalla Torre, G., Lopez, X., Chandra, K., Szczepski, K., Jaremko, L., Ghosh, M., Emwas, A. H., Castagnola, M., Jaremko, M., Hannappel, E., & Coni, P. (2022). Thymosin β4 Is an Endogenous Iron Chelator and Molecular Switcher of Ferroptosis. International journal of molecular sciences, 23(1), . https://doi.org/10.3390/ijms23010551
Lachowicz, Joanna I, et al. "Thymosin β4 Is an Endogenous Iron Chelator and Molecular Switcher of Ferroptosis." International journal of molecular sciences vol. 23,1 (2022). doi: https://doi.org/10.3390/ijms23010551
Lachowicz JI, Pichiri G, Piludu M, Fais S, Orrù G, Congiu T, Piras M, Faa G, Fanni D, Dalla Torre G, Lopez X, Chandra K, Szczepski K, Jaremko L, Ghosh M, Emwas AH, Castagnola M, Jaremko M, Hannappel E, Coni P. Thymosin β4 Is an Endogenous Iron Chelator and Molecular Switcher of Ferroptosis. Int J Mol Sci. 2022 Jan 04;23(1). doi: 10.3390/ijms23010551. PMID: 35008976.
Copy Download .nbib Format: NLM
[Diagnostic difficulties in the course of duodenal ulcer in a 10-year-old boy].

Wiadomosci lekarskie (Warsaw, Poland : 1960)

Szczepski O, Korman E, Lewandowska K, Korbas J.
PMID: 6061261
Wiad Lek. 1967 Jul 01;20(13):1295-7.

No abstract available.

Cite
Szczepski O, Korman E, Lewandowska K, et al. Trudności diagnostyczne w przebiegu choroby wrzodowej dwunastnicy u 10-letniego chłopca. [Diagnostic difficulties in the course of duodenal ulcer in a 10-year-old boy]. Wiad Lek. 1967;20(13):1295-7
Szczepski, O., Korman, E., Lewandowska, K., & Korbas, J. (1967). Trudności diagnostyczne w przebiegu choroby wrzodowej dwunastnicy u 10-letniego chłopca. [Diagnostic difficulties in the course of duodenal ulcer in a 10-year-old boy]. Wiadomosci lekarskie (Warsaw, Poland : 1960), 20(13), 1295-7.
Szczepski, O, et al. "Trudności diagnostyczne w przebiegu choroby wrzodowej dwunastnicy u 10-letniego chłopca." [Diagnostic difficulties in the course of duodenal ulcer in a 10-year-old boy]. Wiadomosci lekarskie (Warsaw, Poland : 1960) vol. 20,13 (1967): 1295-7.
Szczepski O, Korman E, Lewandowska K, Korbas J. Trudności diagnostyczne w przebiegu choroby wrzodowej dwunastnicy u 10-letniego chłopca. [Diagnostic difficulties in the course of duodenal ulcer in a 10-year-old boy]. Wiad Lek. 1967 Jul 01;20(13):1295-7. Polish. PMID: 6061261.
Copy Download .nbib Format: NLM
NMR as a "Gold Standard" Method in Drug Design and Discovery.

Molecules (Basel, Switzerland)

Emwas AH, Szczepski K, Poulson BG, Chandra K, McKay RT, Dhahri M, Alahmari F, Jaremko L, Lachowicz JI, Jaremko M.
PMID: 33050240
Molecules. 2020 Oct 09;25(20). doi: 10.3390/molecules25204597.

Studying disease models at the molecular level is vital for drug development in order to improve treatment and prevent a wide range of human pathologies. Microbial infections are still a major challenge because pathogens rapidly and continually evolve developing...

Cite
Emwas AH, Szczepski K, Poulson BG, et al. NMR as a "Gold Standard" Method in Drug Design and Discovery. Molecules. 2020;25(20)doi: 10.3390/molecules25204597.
Emwas, A. H., Szczepski, K., Poulson, B. G., Chandra, K., McKay, R. T., Dhahri, M., Alahmari, F., Jaremko, L., Lachowicz, J. I., & Jaremko, M. (2020). NMR as a "Gold Standard" Method in Drug Design and Discovery. Molecules (Basel, Switzerland), 25(20), . https://doi.org/10.3390/molecules25204597
Emwas, Abdul-Hamid, et al. "NMR as a "Gold Standard" Method in Drug Design and Discovery." Molecules (Basel, Switzerland) vol. 25,20 (2020). doi: https://doi.org/10.3390/molecules25204597
Emwas AH, Szczepski K, Poulson BG, Chandra K, McKay RT, Dhahri M, Alahmari F, Jaremko L, Lachowicz JI, Jaremko M. NMR as a "Gold Standard" Method in Drug Design and Discovery. Molecules. 2020 Oct 09;25(20). doi: 10.3390/molecules25204597. PMID: 33050240; PMCID: PMC7594251.
Copy Download .nbib Format: NLM
Thymosin β4 Is an Endogenous Iron Chelator and Molecular Switcher of Ferroptosis.

International journal of molecular sciences

Lachowicz JI, Pichiri G, Piludu M, Fais S, Orrù G, Congiu T, Piras M, Faa G, Fanni D, Dalla Torre G, Lopez X, Chandra K, Szczepski K, Jaremko L, Ghosh M, Emwas AH, Castagnola M, Jaremko M, Hannappel E, Coni P.
PMID: 35008976
Int J Mol Sci. 2022 Jan 04;23(1). doi: 10.3390/ijms23010551.

Thymosin β4 (Tβ4) was extracted forty years agofrom calf thymus. Since then, it has been identified as a G-actin binding protein involved in blood clotting, tissue regeneration, angiogenesis, and anti-inflammatory processes. Tβ4 has also been implicated in tumor metastasis...

Cite
Lachowicz JI, Pichiri G, Piludu M, et al. Thymosin β4 Is an Endogenous Iron Chelator and Molecular Switcher of Ferroptosis. Int J Mol Sci. 2022;23(1)doi: 10.3390/ijms23010551.
Lachowicz, J. I., Pichiri, G., Piludu, M., Fais, S., Orrù, G., Congiu, T., Piras, M., Faa, G., Fanni, D., Dalla Torre, G., Lopez, X., Chandra, K., Szczepski, K., Jaremko, L., Ghosh, M., Emwas, A. H., Castagnola, M., Jaremko, M., Hannappel, E., & Coni, P. (2022). Thymosin β4 Is an Endogenous Iron Chelator and Molecular Switcher of Ferroptosis. International journal of molecular sciences, 23(1), . https://doi.org/10.3390/ijms23010551
Lachowicz, Joanna I, et al. "Thymosin β4 Is an Endogenous Iron Chelator and Molecular Switcher of Ferroptosis." International journal of molecular sciences vol. 23,1 (2022). doi: https://doi.org/10.3390/ijms23010551
Lachowicz JI, Pichiri G, Piludu M, Fais S, Orrù G, Congiu T, Piras M, Faa G, Fanni D, Dalla Torre G, Lopez X, Chandra K, Szczepski K, Jaremko L, Ghosh M, Emwas AH, Castagnola M, Jaremko M, Hannappel E, Coni P. Thymosin β4 Is an Endogenous Iron Chelator and Molecular Switcher of Ferroptosis. Int J Mol Sci. 2022 Jan 04;23(1). doi: 10.3390/ijms23010551. PMID: 35008976; PMCID: PMC8745404.
Copy Download .nbib Format: NLM
[Effect of stimulating substances with special reference to dibazol after subsiding of acute symptoms].

Pediatria polska

GERWELOWA H, GSODAM B, HOŁOGA U, SKALMOWSKI T, SZCZEPSKI O.
PMID: 14948324
Pediatr Pol. 1952 Jul;27(7):835-44.

No abstract available.

Cite
GERWELOWA H, GSODAM B, HOŁOGA U, et al. Wpływ środków podniecajacych ze szczególnym uwzglednieniem dibazolu na poliomyelitis po ustapieniu objawów ostrych. [Effect of stimulating substances with special reference to dibazol after subsiding of acute symptoms]. Pediatr Pol. 1952;27(7):835-44
GERWELOWA, H., GSODAM, B., HOŁOGA, U., SKALMOWSKI, T., & SZCZEPSKI, O. (1952). Wpływ środków podniecajacych ze szczególnym uwzglednieniem dibazolu na poliomyelitis po ustapieniu objawów ostrych. [Effect of stimulating substances with special reference to dibazol after subsiding of acute symptoms]. Pediatria polska, 27(7), 835-44.
GERWELOWA, H, et al. "Wpływ środków podniecajacych ze szczególnym uwzglednieniem dibazolu na poliomyelitis po ustapieniu objawów ostrych." [Effect of stimulating substances with special reference to dibazol after subsiding of acute symptoms]. Pediatria polska vol. 27,7 (1952): 835-44.
GERWELOWA H, GSODAM B, HOŁOGA U, SKALMOWSKI T, SZCZEPSKI O. Wpływ środków podniecajacych ze szczególnym uwzglednieniem dibazolu na poliomyelitis po ustapieniu objawów ostrych. [Effect of stimulating substances with special reference to dibazol after subsiding of acute symptoms]. Pediatr Pol. 1952 Jul;27(7):835-44. Undetermined. PMID: 14948324.
Copy Download .nbib Format: NLM
You Are What You Eat: Application of Metabolomics Approaches to Advance Nutrition Research.

Foods (Basel, Switzerland)

Emwas AM, Al-Rifai N, Szczepski K, Alsuhaymi S, Rayyan S, Almahasheer H, Jaremko M, Brennan L, Lachowicz JI.
PMID: 34072780
Foods. 2021 May 31;10(6). doi: 10.3390/foods10061249.

A healthy condition is defined by complex human metabolic pathways that only function properly when fully satisfied by nutritional inputs. Poor nutritional intakes are associated with a number of metabolic diseases, such as diabetes, obesity, atherosclerosis, hypertension, and osteoporosis....

Cite
Emwas AM, Al-Rifai N, Szczepski K, et al. You Are What You Eat: Application of Metabolomics Approaches to Advance Nutrition Research. Foods. 2021;10(6)doi: 10.3390/foods10061249.
Emwas, A. M., Al-Rifai, N., Szczepski, K., Alsuhaymi, S., Rayyan, S., Almahasheer, H., Jaremko, M., Brennan, L., & Lachowicz, J. I. (2021). You Are What You Eat: Application of Metabolomics Approaches to Advance Nutrition Research. Foods (Basel, Switzerland), 10(6), . https://doi.org/10.3390/foods10061249
Emwas, Abdul-Hamid M, et al. "You Are What You Eat: Application of Metabolomics Approaches to Advance Nutrition Research." Foods (Basel, Switzerland) vol. 10,6 (2021). doi: https://doi.org/10.3390/foods10061249
Emwas AM, Al-Rifai N, Szczepski K, Alsuhaymi S, Rayyan S, Almahasheer H, Jaremko M, Brennan L, Lachowicz JI. You Are What You Eat: Application of Metabolomics Approaches to Advance Nutrition Research. Foods. 2021 May 31;10(6). doi: 10.3390/foods10061249. PMID: 34072780; PMCID: PMC8229064.
Copy Download .nbib Format: NLM
Showing 1 to 6 of 6 entries