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Activation of dihydropyridine-sensitive calcium channels induces somatostatin release from hypothalamic neurons. Pharmacological characterization.

Neurochemistry international

Tapia-Arancibia L, Humbert T.
PMID: 20504713
Neurochem Int. 1991;18(3):367-71. doi: 10.1016/0197-0186(91)90168-d.

The current study was designed to determine whether dihydropyridine voltage-sensitive calcium channels were involved in the release of somatostatin from hypothalamic neurons, BAY K 8644 (?) a dihydropyridine with Ca(2+) channel agonistic properties elicited a dose-response release of somatostatin....

Glutamate peripherally administered exerts somatostatin-releasing action in the conscious rat.

Journal of neuroendocrinology

Benyassi A, Tapia-Arancibia L, Arancibia S.
PMID: 19215488
J Neuroendocrinol. 1991 Aug 01;3(4):429-32. doi: 10.1111/j.1365-2826.1991.tb00299.x.

Abstract This study was designed to determine whether glutamate is able to stimulate somatostatin release from in vivo conscious animals when somatostatin release is monitored in unanaesthetized rats stereotaxically implanted with a push-pull cannula in the median eminence. One...

Phospholipase A and Somatostatin Release are Activated in Response to N-Methyl-D-Aspartate Receptor Stimulation in Hypothalamic Neurons in Primary Culture.

Journal of neuroendocrinology

Rage F, Pin JP, Tapia-Arancibia L.
PMID: 19215501
J Neuroendocrinol. 1991 Oct 01;3(5):515-22. doi: 10.1111/j.1365-2826.1991.tb00312.x.

Abstract We have recently shown that glutamate primarily induces somatostatin release in hypothalamic neurons through N-methyl-D-aspartate (NMDA)-type receptor sites. Here we report that glutamate and NMDA also stimulate the release of [(3)H]arachidonic acid in a dose-dependent manner. The NMDA-induced...

Showing 1 to 3 of 3 entries