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Benefits and costs of ecological restoration: Rapid assessment of changing ecosystem service values at a U.K. wetland.

Ecology and evolution

Peh KS, Balmford A, Field RH, Lamb A, Birch JC, Bradbury RB, Brown C, Butchart SH, Lester M, Morrison R, Sedgwick I, Soans C, Stattersfield AJ, Stroh PA, Swetnam RD, Thomas DH, Walpole M, Warrington S, Hughes FM.
PMID: 25505517
Ecol Evol. 2014 Oct;4(20):3875-86. doi: 10.1002/ece3.1248. Epub 2014 Sep 23.

Restoration of degraded land is recognized by the international community as an important way of enhancing both biodiversity and ecosystem services, but more information is needed about its costs and benefits. In Cambridgeshire, U.K., a long-term initiative to convert...

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Peh KS, Balmford A, Field RH, et al. Benefits and costs of ecological restoration: Rapid assessment of changing ecosystem service values at a U.K. wetland. Ecol Evol. 2014;4(20):3875-86doi: 10.1002/ece3.1248.
Peh, K. S., Balmford, A., Field, R. H., Lamb, A., Birch, J. C., Bradbury, R. B., Brown, C., Butchart, S. H., Lester, M., Morrison, R., Sedgwick, I., Soans, C., Stattersfield, A. J., Stroh, P. A., Swetnam, R. D., Thomas, D. H., Walpole, M., Warrington, S., & Hughes, F. M. (2014). Benefits and costs of ecological restoration: Rapid assessment of changing ecosystem service values at a U.K. wetland. Ecology and evolution, 4(20), 3875-86. https://doi.org/10.1002/ece3.1248
Peh, Kelvin S-H, et al. "Benefits and costs of ecological restoration: Rapid assessment of changing ecosystem service values at a U.K. wetland." Ecology and evolution vol. 4,20 (2014): 3875-86. doi: https://doi.org/10.1002/ece3.1248
Peh KS, Balmford A, Field RH, Lamb A, Birch JC, Bradbury RB, Brown C, Butchart SH, Lester M, Morrison R, Sedgwick I, Soans C, Stattersfield AJ, Stroh PA, Swetnam RD, Thomas DH, Walpole M, Warrington S, Hughes FM. Benefits and costs of ecological restoration: Rapid assessment of changing ecosystem service values at a U.K. wetland. Ecol Evol. 2014 Oct;4(20):3875-86. doi: 10.1002/ece3.1248. Epub 2014 Sep 23. PMID: 25505517; PMCID: PMC4242572.
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Recessive, gain-of-function toxicity in an APOL1 BAC transgenic mouse model mirrors human APOL1 kidney disease.

Disease models & mechanisms

McCarthy GM, Blasio A, Donovan OG, Schaller LB, Bock-Hughes A, Magraner JM, Suh JH, Tattersfield CF, Stillman IE, Shah SS, Zsengeller ZK, Subramanian B, Friedman DJ, Pollak MR.
PMID: 34350953
Dis Model Mech. 2021 Aug 01;14(8). doi: 10.1242/dmm.048952. Epub 2021 Aug 05.

People of recent sub-Saharan African ancestry develop kidney failure much more frequently than other groups. A large fraction of this disparity is due to two coding sequence variants in the APOL1 gene. Inheriting two copies of these APOL1 risk...

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McCarthy GM, Blasio A, Donovan OG, et al. Recessive, gain-of-function toxicity in an APOL1 BAC transgenic mouse model mirrors human APOL1 kidney disease. Dis Model Mech. 2021;14(8)doi: 10.1242/dmm.048952.
McCarthy, G. M., Blasio, A., Donovan, O. G., Schaller, L. B., Bock-Hughes, A., Magraner, J. M., Suh, J. H., Tattersfield, C. F., Stillman, I. E., Shah, S. S., Zsengeller, Z. K., Subramanian, B., Friedman, D. J., & Pollak, M. R. (2021). Recessive, gain-of-function toxicity in an APOL1 BAC transgenic mouse model mirrors human APOL1 kidney disease. Disease models & mechanisms, 14(8), . https://doi.org/10.1242/dmm.048952
McCarthy, Gizelle M, et al. "Recessive, gain-of-function toxicity in an APOL1 BAC transgenic mouse model mirrors human APOL1 kidney disease." Disease models & mechanisms vol. 14,8 (2021). doi: https://doi.org/10.1242/dmm.048952
McCarthy GM, Blasio A, Donovan OG, Schaller LB, Bock-Hughes A, Magraner JM, Suh JH, Tattersfield CF, Stillman IE, Shah SS, Zsengeller ZK, Subramanian B, Friedman DJ, Pollak MR. Recessive, gain-of-function toxicity in an APOL1 BAC transgenic mouse model mirrors human APOL1 kidney disease. Dis Model Mech. 2021 Aug 01;14(8). doi: 10.1242/dmm.048952. Epub 2021 Aug 05. PMID: 34350953; PMCID: PMC8353097.
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Lymphangioleiomyomatosis.

Seminars in respiratory and critical care medicine

Johnson SR, Tattersfield AE.
PMID: 16088601
Semin Respir Crit Care Med. 2002 Apr;23(2):85-92. doi: 10.1055/s-2002-25298.

Lymphangioleiomyomatosis (LAM) is a rare disease categorized by an overgrowth of atypical smooth muscle cells (LAM cells) in the lungs and axial lymphatics, which is associated with the mesenchymal tumor angiomyolipoma. The disease occurs in women generally in their...

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Johnson SR, Tattersfield AE. Lymphangioleiomyomatosis. Semin Respir Crit Care Med. 2002;23(2):85-92doi: 10.1055/s-2002-25298.
Johnson, S. R., & Tattersfield, A. E. (2002). Lymphangioleiomyomatosis. Seminars in respiratory and critical care medicine, 23(2), 85-92. https://doi.org/10.1055/s-2002-25298
Johnson, Simon R, and Tattersfield, Anne E. "Lymphangioleiomyomatosis." Seminars in respiratory and critical care medicine vol. 23,2 (2002): 85-92. doi: https://doi.org/10.1055/s-2002-25298
Johnson SR, Tattersfield AE. Lymphangioleiomyomatosis. Semin Respir Crit Care Med. 2002 Apr;23(2):85-92. doi: 10.1055/s-2002-25298. PMID: 16088601.
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Recessive, gain-of-function toxicity in an APOL1 BAC transgenic mouse model mirrors human APOL1 kidney disease.

Disease models & mechanisms

McCarthy GM, Blasio A, Donovan OG, Schaller LB, Bock-Hughes A, Magraner JM, Suh JH, Tattersfield CF, Stillman IE, Shah SS, Zsengeller ZK, Subramanian B, Friedman DJ, Pollak MR.
PMID: 34350953
Dis Model Mech. 2021 Aug 01;14(8). doi: 10.1242/dmm.048952. Epub 2021 Aug 05.

People of recent sub-Saharan African ancestry develop kidney failure much more frequently than other groups. A large fraction of this disparity is due to two coding sequence variants in the APOL1 gene. Inheriting two copies of these APOL1 risk...

Cite
McCarthy GM, Blasio A, Donovan OG, et al. Recessive, gain-of-function toxicity in an APOL1 BAC transgenic mouse model mirrors human APOL1 kidney disease. Dis Model Mech. 2021;14(8)doi: 10.1242/dmm.048952.
McCarthy, G. M., Blasio, A., Donovan, O. G., Schaller, L. B., Bock-Hughes, A., Magraner, J. M., Suh, J. H., Tattersfield, C. F., Stillman, I. E., Shah, S. S., Zsengeller, Z. K., Subramanian, B., Friedman, D. J., & Pollak, M. R. (2021). Recessive, gain-of-function toxicity in an APOL1 BAC transgenic mouse model mirrors human APOL1 kidney disease. Disease models & mechanisms, 14(8), . https://doi.org/10.1242/dmm.048952
McCarthy, Gizelle M, et al. "Recessive, gain-of-function toxicity in an APOL1 BAC transgenic mouse model mirrors human APOL1 kidney disease." Disease models & mechanisms vol. 14,8 (2021). doi: https://doi.org/10.1242/dmm.048952
McCarthy GM, Blasio A, Donovan OG, Schaller LB, Bock-Hughes A, Magraner JM, Suh JH, Tattersfield CF, Stillman IE, Shah SS, Zsengeller ZK, Subramanian B, Friedman DJ, Pollak MR. Recessive, gain-of-function toxicity in an APOL1 BAC transgenic mouse model mirrors human APOL1 kidney disease. Dis Model Mech. 2021 Aug 01;14(8). doi: 10.1242/dmm.048952. Epub 2021 Aug 05. PMID: 34350953; PMCID: PMC8353097.
Copy Download .nbib Format: NLM
Showing 1 to 4 of 4 entries