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Neuronal dependence of extracellular dopamine, acetylcholine, glutamate, aspartate and gamma-aminobutyric acid (GABA) measured simultaneously from rat neostriatum using in vivo microdialysis: reciprocal interactions.

Amino acids

Herrera-Marschitz M, Meana JJ, O'Connor WT, Goiny M, Reid MS, Ungerstedt U.
PMID: 24194283
Amino Acids. 1992 Feb;2(1):157-79. doi: 10.1007/BF00806086.

The neuronal origin of extracellular levels of dopamine (DA), acetylcholine (ACh), glutamate (Glu), aspartate (Asp) and gamma-aminobutyric acid (GABA) simultaneously collected from the neostriatum of halothane anaesthetized rats with in vivo microdialysis was studied. The following criteria were applied...

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Herrera-Marschitz M, Meana JJ, O'Connor WT, et al. Neuronal dependence of extracellular dopamine, acetylcholine, glutamate, aspartate and gamma-aminobutyric acid (GABA) measured simultaneously from rat neostriatum using in vivo microdialysis: reciprocal interactions. Amino Acids. 1992;2(1):157-79doi: 10.1007/BF00806086.
Herrera-Marschitz, M., Meana, J. J., O'Connor, W. T., Goiny, M., Reid, M. S., & Ungerstedt, U. (1992). Neuronal dependence of extracellular dopamine, acetylcholine, glutamate, aspartate and gamma-aminobutyric acid (GABA) measured simultaneously from rat neostriatum using in vivo microdialysis: reciprocal interactions. Amino acids, 2(1), 157-79. https://doi.org/10.1007/BF00806086
Herrera-Marschitz, M, et al. "Neuronal dependence of extracellular dopamine, acetylcholine, glutamate, aspartate and gamma-aminobutyric acid (GABA) measured simultaneously from rat neostriatum using in vivo microdialysis: reciprocal interactions." Amino acids vol. 2,1 (1992): 157-79. doi: https://doi.org/10.1007/BF00806086
Herrera-Marschitz M, Meana JJ, O'Connor WT, Goiny M, Reid MS, Ungerstedt U. Neuronal dependence of extracellular dopamine, acetylcholine, glutamate, aspartate and gamma-aminobutyric acid (GABA) measured simultaneously from rat neostriatum using in vivo microdialysis: reciprocal interactions. Amino Acids. 1992 Feb;2(1):157-79. doi: 10.1007/BF00806086. PMID: 24194283.
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Allogeneic Hematopoietic Stem Cell Transplantation for Chronic Myelomonocytic Leukemia: Clinical and Molecular Genetic Prognostic Factors in a Nordic Population.

Transplantation and cellular therapy

Wedge E, Hansen JW, Dybedal I, Creignou M, Ejerblad E, Lorenz F, Werlenius O, Ungerstedt J, Holm MS, Nilsson L, Kittang AO, Antunovic P, Rohon P, Andersen MK, Papaemmanuil E, Bernard E, Jädersten M, Hellström-Lindberg E, Grønbæk K, Ljungman P, Friis LS.
PMID: 34500124
Transplant Cell Ther. 2021 Sep 06; doi: 10.1016/j.jtct.2021.08.028. Epub 2021 Sep 06.

Chronic myelomonocytic leukemia (CMML) is an aggressive disease in which survival after allogeneic hematopoietic stem cell transplantation (HCT) remains relatively poor. An assessment of prognostic factors is an important part of treatment decision making and has the potential to...

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Wedge E, Hansen JW, Dybedal I, et al. Allogeneic Hematopoietic Stem Cell Transplantation for Chronic Myelomonocytic Leukemia: Clinical and Molecular Genetic Prognostic Factors in a Nordic Population. Transplant Cell Ther. 2021;doi: 10.1016/j.jtct.2021.08.028.
Wedge, E., Hansen, J. W., Dybedal, I., Creignou, M., Ejerblad, E., Lorenz, F., Werlenius, O., Ungerstedt, J., Holm, M. S., Nilsson, L., Kittang, A. O., Antunovic, P., Rohon, P., Andersen, M. K., Papaemmanuil, E., Bernard, E., Jädersten, M., Hellström-Lindberg, E., Grønbæk, K., Ljungman, P., & Friis, L. S. (2021). Allogeneic Hematopoietic Stem Cell Transplantation for Chronic Myelomonocytic Leukemia: Clinical and Molecular Genetic Prognostic Factors in a Nordic Population. Transplantation and cellular therapy, . https://doi.org/10.1016/j.jtct.2021.08.028
Wedge, Eileen, et al. "Allogeneic Hematopoietic Stem Cell Transplantation for Chronic Myelomonocytic Leukemia: Clinical and Molecular Genetic Prognostic Factors in a Nordic Population." Transplantation and cellular therapy vol. (2021). doi: https://doi.org/10.1016/j.jtct.2021.08.028
Wedge E, Hansen JW, Dybedal I, Creignou M, Ejerblad E, Lorenz F, Werlenius O, Ungerstedt J, Holm MS, Nilsson L, Kittang AO, Antunovic P, Rohon P, Andersen MK, Papaemmanuil E, Bernard E, Jädersten M, Hellström-Lindberg E, Grønbæk K, Ljungman P, Friis LS. Allogeneic Hematopoietic Stem Cell Transplantation for Chronic Myelomonocytic Leukemia: Clinical and Molecular Genetic Prognostic Factors in a Nordic Population. Transplant Cell Ther. 2021 Sep 06; doi: 10.1016/j.jtct.2021.08.028. Epub 2021 Sep 06. PMID: 34500124.
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Simultaneous determination of cholecystokinin, dopamine, glutamate and aspartate in cortex and striatum of the rat using in vivo microdialysis.

Amino acids

Meana JJ, Herrera-Marschitz M, Brodin E, Hökfelt T, Ungerstedt U.
PMID: 24194176
Amino Acids. 1991 Oct;1(3):365-73. doi: 10.1007/BF00814005.

Extracellular levels of cholecystokinin (CCK), dopamine (DA), glutamate (Glu) and aspartate (Asp) were simultaneously monitored in the frontoparietal cortex and the striatum of halothane-anaesthetized rats using in vivo microdialysis. Under basal conditions, cortical and striatal CCK levels were 3.11...

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Meana JJ, Herrera-Marschitz M, Brodin E, et al. Simultaneous determination of cholecystokinin, dopamine, glutamate and aspartate in cortex and striatum of the rat using in vivo microdialysis. Amino Acids. 1991;1(3):365-73doi: 10.1007/BF00814005.
Meana, J. J., Herrera-Marschitz, M., Brodin, E., Hökfelt, T., & Ungerstedt, U. (1991). Simultaneous determination of cholecystokinin, dopamine, glutamate and aspartate in cortex and striatum of the rat using in vivo microdialysis. Amino acids, 1(3), 365-73. https://doi.org/10.1007/BF00814005
Meana, J J, et al. "Simultaneous determination of cholecystokinin, dopamine, glutamate and aspartate in cortex and striatum of the rat using in vivo microdialysis." Amino acids vol. 1,3 (1991): 365-73. doi: https://doi.org/10.1007/BF00814005
Meana JJ, Herrera-Marschitz M, Brodin E, Hökfelt T, Ungerstedt U. Simultaneous determination of cholecystokinin, dopamine, glutamate and aspartate in cortex and striatum of the rat using in vivo microdialysis. Amino Acids. 1991 Oct;1(3):365-73. doi: 10.1007/BF00814005. PMID: 24194176.
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Critical Role of Lama4 for Hematopoiesis Regeneration and Acute Myeloid Leukemia Progression.

Blood

Cai H, Kondo M, Sandhow L, Xiao P, Johansson AS, Sasaki T, Zawacka-Pankau J, Tryggvason K, Ungerstedt JS, Walfridsson J, Ekblom M, Qian H.
PMID: 34958665
Blood. 2021 Dec 27; doi: 10.1182/blood.2021011510. Epub 2021 Dec 27.

Impairement of normal hmatopoiesis and leukemia progression are two well-linked processes during leukemia development and controlled by the bone marrow (BM) niche. Extracellular matrix proteins including laminin are important BM niche components. However, their role in hematopoiesis regeneration and...

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Cai H, Kondo M, Sandhow L, et al. Critical Role of Lama4 for Hematopoiesis Regeneration and Acute Myeloid Leukemia Progression. Blood. 2021;doi: 10.1182/blood.2021011510.
Cai, H., Kondo, M., Sandhow, L., Xiao, P., Johansson, A. S., Sasaki, T., Zawacka-Pankau, J., Tryggvason, K., Ungerstedt, J. S., Walfridsson, J., Ekblom, M., & Qian, H. (2021). Critical Role of Lama4 for Hematopoiesis Regeneration and Acute Myeloid Leukemia Progression. Blood, . https://doi.org/10.1182/blood.2021011510
Cai, Huan, et al. "Critical Role of Lama4 for Hematopoiesis Regeneration and Acute Myeloid Leukemia Progression." Blood vol. (2021). doi: https://doi.org/10.1182/blood.2021011510
Cai H, Kondo M, Sandhow L, Xiao P, Johansson AS, Sasaki T, Zawacka-Pankau J, Tryggvason K, Ungerstedt JS, Walfridsson J, Ekblom M, Qian H. Critical Role of Lama4 for Hematopoiesis Regeneration and Acute Myeloid Leukemia Progression. Blood. 2021 Dec 27; doi: 10.1182/blood.2021011510. Epub 2021 Dec 27. PMID: 34958665.
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Glutaredoxin mediated redox effects of coenzyme Q10 treatment in type 1 and type 2 diabetes patients.

BBA clinical

Montano SJ, Grünler J, Nair D, Tekle M, Fernandes AP, Hua X, Holmgren A, Brismar K, Ungerstedt JS.
PMID: 26966682
BBA Clin. 2015 Jun 10;4:14-20. doi: 10.1016/j.bbacli.2015.06.001. eCollection 2015 Dec.

The possible beneficial effects of coenzyme Q10 (CoQ10) supplementation on disease progression and oxidant status in diabetes remains debated. In the present study, patients with type 1 and type 2 diabetes were treated with oral CoQ10, 100 mg twice...

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Montano SJ, Grünler J, Nair D, et al. Glutaredoxin mediated redox effects of coenzyme Q10 treatment in type 1 and type 2 diabetes patients. BBA Clin. 2015;4:14-20doi: 10.1016/j.bbacli.2015.06.001.
Montano, S. J., Grünler, J., Nair, D., Tekle, M., Fernandes, A. P., Hua, X., Holmgren, A., Brismar, K., & Ungerstedt, J. S. (2015). Glutaredoxin mediated redox effects of coenzyme Q10 treatment in type 1 and type 2 diabetes patients. BBA clinical, 414-20. https://doi.org/10.1016/j.bbacli.2015.06.001
Montano, Sergio J, et al. "Glutaredoxin mediated redox effects of coenzyme Q10 treatment in type 1 and type 2 diabetes patients." BBA clinical vol. 4 (2015): 14-20. doi: https://doi.org/10.1016/j.bbacli.2015.06.001
Montano SJ, Grünler J, Nair D, Tekle M, Fernandes AP, Hua X, Holmgren A, Brismar K, Ungerstedt JS. Glutaredoxin mediated redox effects of coenzyme Q10 treatment in type 1 and type 2 diabetes patients. BBA Clin. 2015 Jun 10;4:14-20. doi: 10.1016/j.bbacli.2015.06.001. eCollection 2015 Dec. PMID: 26966682; PMCID: PMC4737908.
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Effects of chronic treatment with uridine on striatal dopamine release and dopamine related behaviours in the absence or the presence of chronic treatment with haloperidol.

Neurochemistry international

Agnati LF, Fuxe K, Ruggeri M, Merlo Pich E, Benfenati F, Volterra V, Ungerstedt U, Zini I.
PMID: 20504471
Neurochem Int. 1989;15(1):107-13. doi: 10.1016/0197-0186(89)90082-x.

Uridine (15mg/kg/day, i.p.), haloperidol (1mg/kg/day, i.p.), uridine (15mg/kg/day, i.p.) plus haloperidol (1mg/kg/day, i.p.) or saline have been chronically administered to Sprague-Dawley male rats. Following 1 week of wash-out, the effects of these treatments on basal striatal dopamine (DA) release...

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Agnati LF, Fuxe K, Ruggeri M, et al. Effects of chronic treatment with uridine on striatal dopamine release and dopamine related behaviours in the absence or the presence of chronic treatment with haloperidol. Neurochem Int. 1989;15(1):107-13doi: 10.1016/0197-0186(89)90082-x.
Agnati, L. F., Fuxe, K., Ruggeri, M., Merlo Pich, E., Benfenati, F., Volterra, V., Ungerstedt, U., & Zini, I. (1989). Effects of chronic treatment with uridine on striatal dopamine release and dopamine related behaviours in the absence or the presence of chronic treatment with haloperidol. Neurochemistry international, 15(1), 107-13. https://doi.org/10.1016/0197-0186(89)90082-x
Agnati, L F, et al. "Effects of chronic treatment with uridine on striatal dopamine release and dopamine related behaviours in the absence or the presence of chronic treatment with haloperidol." Neurochemistry international vol. 15,1 (1989): 107-13. doi: https://doi.org/10.1016/0197-0186(89)90082-x
Agnati LF, Fuxe K, Ruggeri M, Merlo Pich E, Benfenati F, Volterra V, Ungerstedt U, Zini I. Effects of chronic treatment with uridine on striatal dopamine release and dopamine related behaviours in the absence or the presence of chronic treatment with haloperidol. Neurochem Int. 1989;15(1):107-13. doi: 10.1016/0197-0186(89)90082-x. PMID: 20504471.
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Aspects of neural plasticity in the central nervous system-III. Methodological studies on the microdialysis technique.

Neurochemistry international

Ruggeri M, Zoli M, Grimaldi R, Ungerstedt U, Eliasson A, Agnati LF, Fuxe K.
PMID: 20504583
Neurochem Int. 1990;16(4):427-35. doi: 10.1016/0197-0186(90)90004-d.

Some methodological aspects of the intracerebral microdialysis technique have been investigated: the existence of a pressure gradient at the level of the dialyzing membrane, the substance diffusion from the microdialysis probe and the extent of tissue damage induced by...

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Ruggeri M, Zoli M, Grimaldi R, et al. Aspects of neural plasticity in the central nervous system-III. Methodological studies on the microdialysis technique. Neurochem Int. 1990;16(4):427-35doi: 10.1016/0197-0186(90)90004-d.
Ruggeri, M., Zoli, M., Grimaldi, R., Ungerstedt, U., Eliasson, A., Agnati, L. F., & Fuxe, K. (1990). Aspects of neural plasticity in the central nervous system-III. Methodological studies on the microdialysis technique. Neurochemistry international, 16(4), 427-35. https://doi.org/10.1016/0197-0186(90)90004-d
Ruggeri, M, et al. "Aspects of neural plasticity in the central nervous system-III. Methodological studies on the microdialysis technique." Neurochemistry international vol. 16,4 (1990): 427-35. doi: https://doi.org/10.1016/0197-0186(90)90004-d
Ruggeri M, Zoli M, Grimaldi R, Ungerstedt U, Eliasson A, Agnati LF, Fuxe K. Aspects of neural plasticity in the central nervous system-III. Methodological studies on the microdialysis technique. Neurochem Int. 1990;16(4):427-35. doi: 10.1016/0197-0186(90)90004-d. PMID: 20504583.
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Prevention of mortality induced by perinatal asphyxia: Hypothermia or glutamate antagonism?.

Amino acids

Herrera-Marschitz M, Loidl CF, Andersson K, Ungerstedt U.
PMID: 24190712
Amino Acids. 1993 Oct;5(3):413-9. doi: 10.1007/BF00806959.

Perinatal asphyxia was induced by keeping pups-containing uterus horns, removed by hysterectomy, in a 37°C or a 30°C water bath. Asphyxia for a period of 21-22 min at 37°C led to a 97% mortality within the first 20 min...

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Herrera-Marschitz M, Loidl CF, Andersson K, et al. Prevention of mortality induced by perinatal asphyxia: Hypothermia or glutamate antagonism?. Amino Acids. 1993;5(3):413-9doi: 10.1007/BF00806959.
Herrera-Marschitz, M., Loidl, C. F., Andersson, K., & Ungerstedt, U. (1993). Prevention of mortality induced by perinatal asphyxia: Hypothermia or glutamate antagonism?. Amino acids, 5(3), 413-9. https://doi.org/10.1007/BF00806959
Herrera-Marschitz, M, et al. "Prevention of mortality induced by perinatal asphyxia: Hypothermia or glutamate antagonism?." Amino acids vol. 5,3 (1993): 413-9. doi: https://doi.org/10.1007/BF00806959
Herrera-Marschitz M, Loidl CF, Andersson K, Ungerstedt U. Prevention of mortality induced by perinatal asphyxia: Hypothermia or glutamate antagonism?. Amino Acids. 1993 Oct;5(3):413-9. doi: 10.1007/BF00806959. PMID: 24190712.
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Allogeneic Hematopoietic Stem Cell Transplantation for Chronic Myelomonocytic Leukemia: Clinical and Molecular Genetic Prognostic Factors in a Nordic Population.

Transplantation and cellular therapy

Wedge E, Hansen JW, Dybedal I, Creignou M, Ejerblad E, Lorenz F, Werlenius O, Ungerstedt J, Holm MS, Nilsson L, Kittang AO, Antunovic P, Rohon P, Andersen MK, Papaemmanuil E, Bernard E, Jädersten M, Hellström-Lindberg E, Grønbæk K, Ljungman P, Friis LS.
PMID: 34500124
Transplant Cell Ther. 2021 Dec;27(12):991.e1-991.e9. doi: 10.1016/j.jtct.2021.08.028. Epub 2021 Sep 06.

Chronic myelomonocytic leukemia (CMML) is an aggressive disease in which survival after allogeneic hematopoietic stem cell transplantation (HCT) remains relatively poor. An assessment of prognostic factors is an important part of treatment decision making and has the potential to...

Cite
Wedge E, Hansen JW, Dybedal I, et al. Allogeneic Hematopoietic Stem Cell Transplantation for Chronic Myelomonocytic Leukemia: Clinical and Molecular Genetic Prognostic Factors in a Nordic Population. Transplant Cell Ther. 2021;27(12):991.e1-991.e9doi: 10.1016/j.jtct.2021.08.028.
Wedge, E., Hansen, J. W., Dybedal, I., Creignou, M., Ejerblad, E., Lorenz, F., Werlenius, O., Ungerstedt, J., Holm, M. S., Nilsson, L., Kittang, A. O., Antunovic, P., Rohon, P., Andersen, M. K., Papaemmanuil, E., Bernard, E., Jädersten, M., Hellström-Lindberg, E., Grønbæk, K., Ljungman, P., & Friis, L. S. (2021). Allogeneic Hematopoietic Stem Cell Transplantation for Chronic Myelomonocytic Leukemia: Clinical and Molecular Genetic Prognostic Factors in a Nordic Population. Transplantation and cellular therapy, 27(12), 991.e1-991.e9. https://doi.org/10.1016/j.jtct.2021.08.028
Wedge, Eileen, et al. "Allogeneic Hematopoietic Stem Cell Transplantation for Chronic Myelomonocytic Leukemia: Clinical and Molecular Genetic Prognostic Factors in a Nordic Population." Transplantation and cellular therapy vol. 27,12 (2021): 991.e1-991.e9. doi: https://doi.org/10.1016/j.jtct.2021.08.028
Wedge E, Hansen JW, Dybedal I, Creignou M, Ejerblad E, Lorenz F, Werlenius O, Ungerstedt J, Holm MS, Nilsson L, Kittang AO, Antunovic P, Rohon P, Andersen MK, Papaemmanuil E, Bernard E, Jädersten M, Hellström-Lindberg E, Grønbæk K, Ljungman P, Friis LS. Allogeneic Hematopoietic Stem Cell Transplantation for Chronic Myelomonocytic Leukemia: Clinical and Molecular Genetic Prognostic Factors in a Nordic Population. Transplant Cell Ther. 2021 Dec;27(12):991.e1-991.e9. doi: 10.1016/j.jtct.2021.08.028. Epub 2021 Sep 06. PMID: 34500124.
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Kynurenic Acid in the Quinolinate-lesioned Rat Hippocampus: Studies In Vitro and In Vivo.

The European journal of neuroscience

Wu HQ, Baran H, Ungerstedt U, Schwarcz R.
PMID: 12106390
Eur J Neurosci. 1992;4(12):1264-1270. doi: 10.1111/j.1460-9568.1992.tb00152.x.

The present study was designed to examine the cellular localization and biosynthetic machinery of the broad-spectrum excitatory amino acid receptor antagonist kynurenic acid in the lesioned rat hippocampus. Seven days after an intrahippocampal injection of 120 nmol quinolinic acid,...

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Wu HQ, Baran H, Ungerstedt U, et al. Kynurenic Acid in the Quinolinate-lesioned Rat Hippocampus: Studies In Vitro and In Vivo. Eur J Neurosci. 1992;4(12):1264-1270doi: 10.1111/j.1460-9568.1992.tb00152.x.
Wu, H. Q., Baran, H., Ungerstedt, U., & Schwarcz, R. (1992). Kynurenic Acid in the Quinolinate-lesioned Rat Hippocampus: Studies In Vitro and In Vivo. The European journal of neuroscience, 4(12), 1264-1270. https://doi.org/10.1111/j.1460-9568.1992.tb00152.x
Wu, Hui-Qiu, et al. "Kynurenic Acid in the Quinolinate-lesioned Rat Hippocampus: Studies In Vitro and In Vivo." The European journal of neuroscience vol. 4,12 (1992): 1264-1270. doi: https://doi.org/10.1111/j.1460-9568.1992.tb00152.x
Wu HQ, Baran H, Ungerstedt U, Schwarcz R. Kynurenic Acid in the Quinolinate-lesioned Rat Hippocampus: Studies In Vitro and In Vivo. Eur J Neurosci. 1992;4(12):1264-1270. doi: 10.1111/j.1460-9568.1992.tb00152.x. PMID: 12106390.
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Effects of cholecystokinin peptides and neurotensin on dopamine release and metabolism in the rostral and caudal part of the nucleus accumbens using intracerebral dialysis in the anaesthetized rat.

Neurochemistry international

Ruggeri M, Ungerstedt U, Agnati LF, Mutt V, Härfstrand A, Fuxe K.
PMID: 20501123
Neurochem Int. 1987;10(4):509-20. doi: 10.1016/0197-0186(87)90077-5.

By means of intracerebral microdialysis effects of cholecystokinin peptides and neurotensin administered via the microdialysis probe have been studied on dopamine release and metabolism in the nucleus accumbens and neostriatum of the halothane anaesthetized male rat. Levels of extra...

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Ruggeri M, Ungerstedt U, Agnati LF, et al. Effects of cholecystokinin peptides and neurotensin on dopamine release and metabolism in the rostral and caudal part of the nucleus accumbens using intracerebral dialysis in the anaesthetized rat. Neurochem Int. 1987;10(4):509-20doi: 10.1016/0197-0186(87)90077-5.
Ruggeri, M., Ungerstedt, U., Agnati, L. F., Mutt, V., Härfstrand, A., & Fuxe, K. (1987). Effects of cholecystokinin peptides and neurotensin on dopamine release and metabolism in the rostral and caudal part of the nucleus accumbens using intracerebral dialysis in the anaesthetized rat. Neurochemistry international, 10(4), 509-20. https://doi.org/10.1016/0197-0186(87)90077-5
Ruggeri, M, et al. "Effects of cholecystokinin peptides and neurotensin on dopamine release and metabolism in the rostral and caudal part of the nucleus accumbens using intracerebral dialysis in the anaesthetized rat." Neurochemistry international vol. 10,4 (1987): 509-20. doi: https://doi.org/10.1016/0197-0186(87)90077-5
Ruggeri M, Ungerstedt U, Agnati LF, Mutt V, Härfstrand A, Fuxe K. Effects of cholecystokinin peptides and neurotensin on dopamine release and metabolism in the rostral and caudal part of the nucleus accumbens using intracerebral dialysis in the anaesthetized rat. Neurochem Int. 1987;10(4):509-20. doi: 10.1016/0197-0186(87)90077-5. PMID: 20501123.
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Methylseleninic Acid Sensitizes Ovarian Cancer Cells to T-Cell Mediated Killing by Decreasing PDL1 and VEGF Levels.

Frontiers in oncology

Nair D, Rådestad E, Khalkar P, Diaz-Argelich N, Schröder A, Klynning C, Ungerstedt J, Uhlin M, Fernandes AP.
PMID: 30324091
Front Oncol. 2018 Sep 28;8:407. doi: 10.3389/fonc.2018.00407. eCollection 2018.

Redox active selenium (Se) compounds at sub toxic doses act as pro-oxidants with cytotoxic effects on tumor cells and are promising future chemotherapeutic agents. However, little is known about how Se compounds affect immune cells in the tumor microenvironment....

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Nair D, Rådestad E, Khalkar P, et al. Methylseleninic Acid Sensitizes Ovarian Cancer Cells to T-Cell Mediated Killing by Decreasing PDL1 and VEGF Levels. Front Oncol. 2018;8:407doi: 10.3389/fonc.2018.00407.
Nair, D., Rådestad, E., Khalkar, P., Diaz-Argelich, N., Schröder, A., Klynning, C., Ungerstedt, J., Uhlin, M., & Fernandes, A. P. (2018). Methylseleninic Acid Sensitizes Ovarian Cancer Cells to T-Cell Mediated Killing by Decreasing PDL1 and VEGF Levels. Frontiers in oncology, 8407. https://doi.org/10.3389/fonc.2018.00407
Nair, Deepika, et al. "Methylseleninic Acid Sensitizes Ovarian Cancer Cells to T-Cell Mediated Killing by Decreasing PDL1 and VEGF Levels." Frontiers in oncology vol. 8 (2018): 407. doi: https://doi.org/10.3389/fonc.2018.00407
Nair D, Rådestad E, Khalkar P, Diaz-Argelich N, Schröder A, Klynning C, Ungerstedt J, Uhlin M, Fernandes AP. Methylseleninic Acid Sensitizes Ovarian Cancer Cells to T-Cell Mediated Killing by Decreasing PDL1 and VEGF Levels. Front Oncol. 2018 Sep 28;8:407. doi: 10.3389/fonc.2018.00407. eCollection 2018. PMID: 30324091; PMCID: PMC6172341.
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Showing 1 to 12 of 12 entries