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Quinoxaline-Based Cyclo(oligophenylenes).

The Journal of organic chemistry

Marin L, Kudrjasova J, Verstappen P, Penxten H, Robeyns K, Lutsen L, Vanderzande DJ, Maes W.
PMID: 25611254
J Org Chem. 2015 Feb 20;80(4):2425-30. doi: 10.1021/jo502771a. Epub 2015 Jan 30.

A series of fully conjugated quinoxaline-based oligophenylene macrocycles is synthesized by Ni(0)-mediated Yamamoto-type diaryl homocoupling of (fluorinated) 2,3-bis(4'-bromophenyl)quinoxaline precursors. Cyclotrimers and cyclotetramers are obtained as the dominant reaction products. The cyclooligomers are fully characterized, including single-crystal X-ray structures, and...

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Marin L, Kudrjasova J, Verstappen P, et al. Quinoxaline-Based Cyclo(oligophenylenes). J Org Chem. 2015;80(4):2425-30doi: 10.1021/jo502771a.
Marin, L., Kudrjasova, J., Verstappen, P., Penxten, H., Robeyns, K., Lutsen, L., Vanderzande, D. J., & Maes, W. (2015). Quinoxaline-Based Cyclo(oligophenylenes). The Journal of organic chemistry, 80(4), 2425-30. https://doi.org/10.1021/jo502771a
Marin, Lidia, et al. "Quinoxaline-Based Cyclo(oligophenylenes)." The Journal of organic chemistry vol. 80,4 (2015): 2425-30. doi: https://doi.org/10.1021/jo502771a
Marin L, Kudrjasova J, Verstappen P, Penxten H, Robeyns K, Lutsen L, Vanderzande DJ, Maes W. Quinoxaline-Based Cyclo(oligophenylenes). J Org Chem. 2015 Feb 20;80(4):2425-30. doi: 10.1021/jo502771a. Epub 2015 Jan 30. PMID: 25611254.
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Do we need bone mineral density to estimate osteoporotic fracture risk? A 10-year prospective multicentre validation study.

RMD open

Marques A, Lucas R, Simões E, Verstappen SMM, Jacobs JWG, da Silva JAP.
PMID: 29018567
RMD Open. 2017 Sep 26;3(2):e000509. doi: 10.1136/rmdopen-2017-000509. eCollection 2017.

OBJECTIVE: Evaluate the performance of FRAX®, with and without bone mineral densitometry (BMD), in predicting the occurrence of fragility fractures over 10 years.METHODS: Participants aged ≥40 years at baseline, with a complete set of data and a minimum of...

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Marques A, Lucas R, Simões E, et al. Do we need bone mineral density to estimate osteoporotic fracture risk? A 10-year prospective multicentre validation study. RMD Open. 2017;3(2):e000509doi: 10.1136/rmdopen-2017-000509.
Marques, A., Lucas, R., Simões, E., Verstappen, S. M. M., Jacobs, J. W. G., & da Silva, J. A. P. (2017). Do we need bone mineral density to estimate osteoporotic fracture risk? A 10-year prospective multicentre validation study. RMD open, 3(2), e000509. https://doi.org/10.1136/rmdopen-2017-000509
Marques, Andréa, et al. "Do we need bone mineral density to estimate osteoporotic fracture risk? A 10-year prospective multicentre validation study." RMD open vol. 3,2 (2017): e000509. doi: https://doi.org/10.1136/rmdopen-2017-000509
Marques A, Lucas R, Simões E, Verstappen SMM, Jacobs JWG, da Silva JAP. Do we need bone mineral density to estimate osteoporotic fracture risk? A 10-year prospective multicentre validation study. RMD Open. 2017 Sep 26;3(2):e000509. doi: 10.1136/rmdopen-2017-000509. eCollection 2017. PMID: 29018567; PMCID: PMC5623321.
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Reducing the immediate availability of red blood cells in cardiac surgery, a single-centre experience.

Netherlands heart journal : monthly journal of the Netherlands Society of Cardiology and the Netherlands Heart Foundation

Haanschoten MC, van Straten AH, Verstappen F, van de Kerkhof D, van Zundert AA, Soliman Hamad MA.
PMID: 25326103
Neth Heart J. 2015 Jan;23(1):28-32. doi: 10.1007/s12471-014-0618-9.

BACKGROUND: In our institution, we have redefined our criteria for direct availability of red blood cell (RBC) units in the operation room. In this study, we sought to evaluate the safety of applying this new logistical policy of blood...

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Haanschoten MC, van Straten AH, Verstappen F, et al. Reducing the immediate availability of red blood cells in cardiac surgery, a single-centre experience. Neth Heart J. 2015;23(1):28-32doi: 10.1007/s12471-014-0618-9.
Haanschoten, M. C., van Straten, A. H., Verstappen, F., van de Kerkhof, D., van Zundert, A. A., & Soliman Hamad, M. A. (2015). Reducing the immediate availability of red blood cells in cardiac surgery, a single-centre experience. Netherlands heart journal : monthly journal of the Netherlands Society of Cardiology and the Netherlands Heart Foundation, 23(1), 28-32. https://doi.org/10.1007/s12471-014-0618-9
Haanschoten, M C, et al. "Reducing the immediate availability of red blood cells in cardiac surgery, a single-centre experience." Netherlands heart journal : monthly journal of the Netherlands Society of Cardiology and the Netherlands Heart Foundation vol. 23,1 (2015): 28-32. doi: https://doi.org/10.1007/s12471-014-0618-9
Haanschoten MC, van Straten AH, Verstappen F, van de Kerkhof D, van Zundert AA, Soliman Hamad MA. Reducing the immediate availability of red blood cells in cardiac surgery, a single-centre experience. Neth Heart J. 2015 Jan;23(1):28-32. doi: 10.1007/s12471-014-0618-9. PMID: 25326103; PMCID: PMC4268205.
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A Gene-for-Gene Relationship Between Wheat and Mycosphaerella graminicola, the Septoria Tritici Blotch Pathogen.

Phytopathology

Brading PA, Verstappen EC, Kema GH, Brown JK.
PMID: 18942957
Phytopathology. 2002 Apr;92(4):439-45. doi: 10.1094/PHYTO.2002.92.4.439.

ABSTRACT Specific resistances to isolates of the ascomycete fungus Mycosphaerella graminicola, which causes Septoria tritici blotch of wheat, have been detected in many cultivars. Cvs. Flame and Hereward, which have specific resistance to the isolate IPO323, were crossed with...

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Brading PA, Verstappen EC, Kema GH, et al. A Gene-for-Gene Relationship Between Wheat and Mycosphaerella graminicola, the Septoria Tritici Blotch Pathogen. Phytopathology. 2002;92(4):439-45doi: 10.1094/PHYTO.2002.92.4.439.
Brading, P. A., Verstappen, E. C., Kema, G. H., & Brown, J. K. (2002). A Gene-for-Gene Relationship Between Wheat and Mycosphaerella graminicola, the Septoria Tritici Blotch Pathogen. Phytopathology, 92(4), 439-45. https://doi.org/10.1094/PHYTO.2002.92.4.439
Brading, Penny A, et al. "A Gene-for-Gene Relationship Between Wheat and Mycosphaerella graminicola, the Septoria Tritici Blotch Pathogen." Phytopathology vol. 92,4 (2002): 439-45. doi: https://doi.org/10.1094/PHYTO.2002.92.4.439
Brading PA, Verstappen EC, Kema GH, Brown JK. A Gene-for-Gene Relationship Between Wheat and Mycosphaerella graminicola, the Septoria Tritici Blotch Pathogen. Phytopathology. 2002 Apr;92(4):439-45. doi: 10.1094/PHYTO.2002.92.4.439. PMID: 18942957.
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Patient safety in Dutch primary care: a study protocol.

Implementation science : IS

Harmsen M, Gaal S, van Dulmen S, de Feijter E, Giesen P, Jacobs A, Martijn L, Mettes T, Verstappen W, Nijhuis-van der Sanden R, Wensing M.
PMID: 20584268
Implement Sci. 2010 Jun 28;5:50. doi: 10.1186/1748-5908-5-50.

BACKGROUND: Insight into the frequency and seriousness of potentially unsafe situations may be the first step towards improving patient safety. Most patient safety attention has been paid to patient safety in hospitals. However, in many countries, patients receive most...

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Harmsen M, Gaal S, van Dulmen S, et al. Patient safety in Dutch primary care: a study protocol. Implement Sci. 2010;5:50doi: 10.1186/1748-5908-5-50.
Harmsen, M., Gaal, S., van Dulmen, S., de Feijter, E., Giesen, P., Jacobs, A., Martijn, L., Mettes, T., Verstappen, W., Nijhuis-van der Sanden, R., & Wensing, M. (2010). Patient safety in Dutch primary care: a study protocol. Implementation science : IS, 550. https://doi.org/10.1186/1748-5908-5-50
Harmsen, Mirjam, et al. "Patient safety in Dutch primary care: a study protocol." Implementation science : IS vol. 5 (2010): 50. doi: https://doi.org/10.1186/1748-5908-5-50
Harmsen M, Gaal S, van Dulmen S, de Feijter E, Giesen P, Jacobs A, Martijn L, Mettes T, Verstappen W, Nijhuis-van der Sanden R, Wensing M. Patient safety in Dutch primary care: a study protocol. Implement Sci. 2010 Jun 28;5:50. doi: 10.1186/1748-5908-5-50. PMID: 20584268; PMCID: PMC2914083.
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[No title available]

Frontiers in veterinary science

Verstappen KM, Willems E, Fluit AC, Duim B, Martens M, Wagenaar JA.
PMID: 28691012
Front Vet Sci. 2017 Jun 23;4:97. doi: 10.3389/fvets.2017.00097. eCollection 2017.

IN CONCLUSION: (i) pigs from different genetic lineages have different susceptibilities for colonization with

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Verstappen KM, Willems E, Fluit AC, et al. . Front Vet Sci. 2017;4:97doi: 10.3389/fvets.2017.00097.
Verstappen, K. M., Willems, E., Fluit, A. C., Duim, B., Martens, M., & Wagenaar, J. A. (2017). . Frontiers in veterinary science, 497. https://doi.org/10.3389/fvets.2017.00097
Verstappen, Koen M, et al. "." Frontiers in veterinary science vol. 4 (2017): 97. doi: https://doi.org/10.3389/fvets.2017.00097
Verstappen KM, Willems E, Fluit AC, Duim B, Martens M, Wagenaar JA. . Front Vet Sci. 2017 Jun 23;4:97. doi: 10.3389/fvets.2017.00097. eCollection 2017. PMID: 28691012; PMCID: PMC5481302.
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Correction: Development of a real-time PCR for detection of Staphylococcus pseudintermedius using a novel automated comparison of whole-genome sequences.

PloS one

Verstappen KM, Huijbregts L, Spaninks M, Wagenaar JA, Fluit AC, Duim B.
PMID: 29211783
PLoS One. 2017 Dec 06;12(12):e0189520. doi: 10.1371/journal.pone.0189520. eCollection 2017.

[This corrects the article DOI: 10.1371/journal.pone.0183925.].

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Verstappen KM, Huijbregts L, Spaninks M, et al. Correction: Development of a real-time PCR for detection of Staphylococcus pseudintermedius using a novel automated comparison of whole-genome sequences. PLoS One. 2017;12(12):e0189520doi: 10.1371/journal.pone.0189520.
Verstappen, K. M., Huijbregts, L., Spaninks, M., Wagenaar, J. A., Fluit, A. C., & Duim, B. (2017). Correction: Development of a real-time PCR for detection of Staphylococcus pseudintermedius using a novel automated comparison of whole-genome sequences. PloS one, 12(12), e0189520. https://doi.org/10.1371/journal.pone.0189520
Verstappen, Koen M, et al. "Correction: Development of a real-time PCR for detection of Staphylococcus pseudintermedius using a novel automated comparison of whole-genome sequences." PloS one vol. 12,12 (2017): e0189520. doi: https://doi.org/10.1371/journal.pone.0189520
Verstappen KM, Huijbregts L, Spaninks M, Wagenaar JA, Fluit AC, Duim B. Correction: Development of a real-time PCR for detection of Staphylococcus pseudintermedius using a novel automated comparison of whole-genome sequences. PLoS One. 2017 Dec 06;12(12):e0189520. doi: 10.1371/journal.pone.0189520. eCollection 2017. PMID: 29211783; PMCID: PMC5718433.
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T cells in primary Sjögren's syndrome: targets for early intervention.

Rheumatology (Oxford, England)

Verstappen GM, Kroese FGM, Bootsma H.
PMID: 30770920
Rheumatology (Oxford). 2019 Feb 15; doi: 10.1093/rheumatology/kez004. Epub 2019 Feb 15.

A histologic hallmark of primary SS (pSS) is lymphocytic infiltration of the salivary and lacrimal glands, in particular by CD4+ T and B cells. In the early stages of the disease, infiltrates are dominated by CD4+ T cells, while...

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Verstappen GM, Kroese FGM, Bootsma H. T cells in primary Sjögren's syndrome: targets for early intervention. Rheumatology (Oxford). 2019;doi: 10.1093/rheumatology/kez004.
Verstappen, G. M., Kroese, F. G. M., & Bootsma, H. (2019). T cells in primary Sjögren's syndrome: targets for early intervention. Rheumatology (Oxford, England), . https://doi.org/10.1093/rheumatology/kez004
Verstappen, Gwenny M, et al. "T cells in primary Sjögren's syndrome: targets for early intervention." Rheumatology (Oxford, England) vol. (2019). doi: https://doi.org/10.1093/rheumatology/kez004
Verstappen GM, Kroese FGM, Bootsma H. T cells in primary Sjögren's syndrome: targets for early intervention. Rheumatology (Oxford). 2019 Feb 15; doi: 10.1093/rheumatology/kez004. Epub 2019 Feb 15. PMID: 30770920; PMCID: PMC8516500.
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Formation of haematopoietic microenvironment and haematopoietic stem cells from single human bone marrow stem cells.

Nature

Huang S, Terstappen LW.
PMID: 8152468
Nature. 1994 Apr 14;368(6472):664. doi: 10.1038/368664a0.

No abstract available.

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Huang S, Terstappen LW. Formation of haematopoietic microenvironment and haematopoietic stem cells from single human bone marrow stem cells. Nature. 1994;368(6472):664doi: 10.1038/368664a0.
Huang, S., & Terstappen, L. W. (1994). Formation of haematopoietic microenvironment and haematopoietic stem cells from single human bone marrow stem cells. Nature, 368(6472), 664. https://doi.org/10.1038/368664a0
Huang, S, and Terstappen, L W. "Formation of haematopoietic microenvironment and haematopoietic stem cells from single human bone marrow stem cells." Nature vol. 368,6472 (1994): 664. doi: https://doi.org/10.1038/368664a0
Huang S, Terstappen LW. Formation of haematopoietic microenvironment and haematopoietic stem cells from single human bone marrow stem cells. Nature. 1994 Apr 14;368(6472):664. doi: 10.1038/368664a0. PMID: 8152468.
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Do people with rheumatoid arthritis maintain their physical activity level at treatment onset over the first year of methotrexate therapy?.

Rheumatology (Oxford, England)

Gwinnutt JM, Alsafar H, Hyrich KL, Lunt M, Barton A, Verstappen SMM.
PMID: 33605404
Rheumatology (Oxford). 2021 Oct 02;60(10):4633-4642. doi: 10.1093/rheumatology/keab060.

OBJECTIVES: To describe how many people with RA reduce their baseline physical activity level over the first year of MTX treatment, and which factors predict this.METHODS: Data came from the Rheumatoid Arthritis Medication Study (RAMS), a prospective cohort of...

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Gwinnutt JM, Alsafar H, Hyrich KL, et al. Do people with rheumatoid arthritis maintain their physical activity level at treatment onset over the first year of methotrexate therapy?. Rheumatology (Oxford). 2021;60(10):4633-4642doi: 10.1093/rheumatology/keab060.
Gwinnutt, J. M., Alsafar, H., Hyrich, K. L., Lunt, M., Barton, A., Verstappen, S. M. M. (2021). Do people with rheumatoid arthritis maintain their physical activity level at treatment onset over the first year of methotrexate therapy?. Rheumatology (Oxford, England), 60(10), 4633-4642. https://doi.org/10.1093/rheumatology/keab060
Gwinnutt, James M, et al. "Do people with rheumatoid arthritis maintain their physical activity level at treatment onset over the first year of methotrexate therapy?." Rheumatology (Oxford, England) vol. 60,10 (2021): 4633-4642. doi: https://doi.org/10.1093/rheumatology/keab060
Gwinnutt JM, Alsafar H, Hyrich KL, Lunt M, Barton A, Verstappen SMM. Do people with rheumatoid arthritis maintain their physical activity level at treatment onset over the first year of methotrexate therapy?. Rheumatology (Oxford). 2021 Oct 02;60(10):4633-4642. doi: 10.1093/rheumatology/keab060. PMID: 33605404; PMCID: PMC8487269.
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Increased frailty in people with osteoarthritis and rheumatoid arthritis and the influence of co-morbidity: an analysis of the UK Biobank cohort.

Arthritis care & research

Cook MJ, Verstappen SMM, Lunt M, O'Neill TW.
PMID: 34235888
Arthritis Care Res (Hoboken). 2021 Jul 07; doi: 10.1002/acr.24747. Epub 2021 Jul 07.

OBJECTIVES: To determine the association between osteoarthritis (OA), rheumatoid arthritis (RA) and frailty and to determine whether co-morbidities interact with OA and RA to further increase the likelihood of frailty.METHODS: Participants of the UK Biobank aged 40-69 years at...

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Cook MJ, Verstappen SMM, Lunt M, et al. Increased frailty in people with osteoarthritis and rheumatoid arthritis and the influence of co-morbidity: an analysis of the UK Biobank cohort. Arthritis Care Res (Hoboken). 2021;doi: 10.1002/acr.24747.
Cook, M. J., Verstappen, S. M. M., Lunt, M., & O'Neill, T. W. (2021). Increased frailty in people with osteoarthritis and rheumatoid arthritis and the influence of co-morbidity: an analysis of the UK Biobank cohort. Arthritis care & research, . https://doi.org/10.1002/acr.24747
Cook, Michael J, et al. "Increased frailty in people with osteoarthritis and rheumatoid arthritis and the influence of co-morbidity: an analysis of the UK Biobank cohort." Arthritis care & research vol. (2021). doi: https://doi.org/10.1002/acr.24747
Cook MJ, Verstappen SMM, Lunt M, O'Neill TW. Increased frailty in people with osteoarthritis and rheumatoid arthritis and the influence of co-morbidity: an analysis of the UK Biobank cohort. Arthritis Care Res (Hoboken). 2021 Jul 07; doi: 10.1002/acr.24747. Epub 2021 Jul 07. PMID: 34235888.
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Unraveling heterogeneity within ACPA-negative rheumatoid arthritis: the subgroup of patients with a strong clinical and serological response to initiation of DMARD treatment favor disease resolution.

Arthritis research & therapy

Verstappen M, van Steenbergen HW, de Jong PHP, van der Helm-van Mil AHM.
PMID: 34980246
Arthritis Res Ther. 2022 Jan 03;24(1):4. doi: 10.1186/s13075-021-02671-z.

BACKGROUND: Rheumatoid arthritis (RA) is a heterogeneous disease, as evidenced by the differences in long-term outcomes. This applies especially to anti-citrullinated protein antibodies (ACPA)-negative RA, where a proportion achieves sustained DMARD-free remission (SDFR; sustained absence of synovitis after DMARD...

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Verstappen M, van Steenbergen HW, de Jong PHP, et al. Unraveling heterogeneity within ACPA-negative rheumatoid arthritis: the subgroup of patients with a strong clinical and serological response to initiation of DMARD treatment favor disease resolution. Arthritis Res Ther. 2022;24(1):4doi: 10.1186/s13075-021-02671-z.
Verstappen, M., van Steenbergen, H. W., de Jong, P. H. P., & van der Helm-van Mil, A. H. M. (2022). Unraveling heterogeneity within ACPA-negative rheumatoid arthritis: the subgroup of patients with a strong clinical and serological response to initiation of DMARD treatment favor disease resolution. Arthritis research & therapy, 24(1), 4. https://doi.org/10.1186/s13075-021-02671-z
Verstappen, M, et al. "Unraveling heterogeneity within ACPA-negative rheumatoid arthritis: the subgroup of patients with a strong clinical and serological response to initiation of DMARD treatment favor disease resolution." Arthritis research & therapy vol. 24,1 (2022): 4. doi: https://doi.org/10.1186/s13075-021-02671-z
Verstappen M, van Steenbergen HW, de Jong PHP, van der Helm-van Mil AHM. Unraveling heterogeneity within ACPA-negative rheumatoid arthritis: the subgroup of patients with a strong clinical and serological response to initiation of DMARD treatment favor disease resolution. Arthritis Res Ther. 2022 Jan 03;24(1):4. doi: 10.1186/s13075-021-02671-z. PMID: 34980246; PMCID: PMC8722281.
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Showing 1 to 12 of 120 entries