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Showing 1 to 12 of 66 entries
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Erratum: In vivo single-molecule imaging of syntaxin1A reveals polyphosphoinositide- and activity-dependent trapping in presynaptic nanoclusters.

Nature communications

Bademosi AT, Lauwers E, Padmanabhan P, Odierna L, Chai YJ, Papadopulos A, Goodhill GJ, Verstreken P, van Swinderen B, Meunier FA.
PMID: 28181515
Nat Commun. 2017 Feb 09;8:14492. doi: 10.1038/ncomms14492.

No abstract available.

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Bademosi AT, Lauwers E, Padmanabhan P, et al. Erratum: In vivo single-molecule imaging of syntaxin1A reveals polyphosphoinositide- and activity-dependent trapping in presynaptic nanoclusters. Nat Commun. 2017;8:14492doi: 10.1038/ncomms14492.
Bademosi, A. T., Lauwers, E., Padmanabhan, P., Odierna, L., Chai, Y. J., Papadopulos, A., Goodhill, G. J., Verstreken, P., van Swinderen, B., & Meunier, F. A. (2017). Erratum: In vivo single-molecule imaging of syntaxin1A reveals polyphosphoinositide- and activity-dependent trapping in presynaptic nanoclusters. Nature communications, 814492. https://doi.org/10.1038/ncomms14492
Bademosi, Adekunle T, et al. "Erratum: In vivo single-molecule imaging of syntaxin1A reveals polyphosphoinositide- and activity-dependent trapping in presynaptic nanoclusters." Nature communications vol. 8 (2017): 14492. doi: https://doi.org/10.1038/ncomms14492
Bademosi AT, Lauwers E, Padmanabhan P, Odierna L, Chai YJ, Papadopulos A, Goodhill GJ, Verstreken P, van Swinderen B, Meunier FA. Erratum: In vivo single-molecule imaging of syntaxin1A reveals polyphosphoinositide- and activity-dependent trapping in presynaptic nanoclusters. Nat Commun. 2017 Feb 09;8:14492. doi: 10.1038/ncomms14492. PMID: 28181515; PMCID: PMC5309767.
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Synaptic mitochondria in synaptic transmission and organization of vesicle pools in health and disease.

Frontiers in synaptic neuroscience

Vos M, Lauwers E, Verstreken P.
PMID: 21423525
Front Synaptic Neurosci. 2010 Sep 22;2:139. doi: 10.3389/fnsyn.2010.00139. eCollection 2010.

Cell types rich in mitochondria, including neurons, display a high energy demand and a need for calcium buffering. The importance of mitochondria for proper neuronal function is stressed by the occurrence of neurological defects in patients suffering from a...

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Vos M, Lauwers E, Verstreken P. Synaptic mitochondria in synaptic transmission and organization of vesicle pools in health and disease. Front Synaptic Neurosci. 2010;2:139doi: 10.3389/fnsyn.2010.00139.
Vos, M., Lauwers, E., & Verstreken, P. (2010). Synaptic mitochondria in synaptic transmission and organization of vesicle pools in health and disease. Frontiers in synaptic neuroscience, 2139. https://doi.org/10.3389/fnsyn.2010.00139
Vos, Melissa, et al. "Synaptic mitochondria in synaptic transmission and organization of vesicle pools in health and disease." Frontiers in synaptic neuroscience vol. 2 (2010): 139. doi: https://doi.org/10.3389/fnsyn.2010.00139
Vos M, Lauwers E, Verstreken P. Synaptic mitochondria in synaptic transmission and organization of vesicle pools in health and disease. Front Synaptic Neurosci. 2010 Sep 22;2:139. doi: 10.3389/fnsyn.2010.00139. eCollection 2010. PMID: 21423525; PMCID: PMC3059669.
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No ubiquitin anchors and fully RIGged.

Immunity

Verstrepen L, Beyaert R.
PMID: 22749347
Immunity. 2012 Jun 29;36(6):897-9. doi: 10.1016/j.immuni.2012.06.005.

Unanchored chains of ubiquitin have recently been proposed to play a role in signaling. In this issue of Immunity, Jiang et al. (2012) demonstrate that binding of free polyubiquitin to the viral RNA sensors RIG-I and MDA5 leads to...

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Verstrepen L, Beyaert R. No ubiquitin anchors and fully RIGged. Immunity. 2012;36(6):897-9doi: 10.1016/j.immuni.2012.06.005.
Verstrepen, L., & Beyaert, R. (2012). No ubiquitin anchors and fully RIGged. Immunity, 36(6), 897-9. https://doi.org/10.1016/j.immuni.2012.06.005
Verstrepen, Lynn, and Beyaert, Rudi. "No ubiquitin anchors and fully RIGged." Immunity vol. 36,6 (2012): 897-9. doi: https://doi.org/10.1016/j.immuni.2012.06.005
Verstrepen L, Beyaert R. No ubiquitin anchors and fully RIGged. Immunity. 2012 Jun 29;36(6):897-9. doi: 10.1016/j.immuni.2012.06.005. PMID: 22749347.
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Publisher Correction: In vivo single-molecule imaging of syntaxin1A reveals polyphosphoinositide- and activity-dependent trapping in presynaptic nanoclusters.

Nature communications

Bademosi AT, Lauwers E, Padmanabhan P, Odierna L, Chai YJ, Papadopulos A, Goodhill GJ, Verstreken P, van Swinderen B, Meunier FA.
PMID: 29633762
Nat Commun. 2018 Apr 10;9:16191. doi: 10.1038/ncomms16191.

This corrects the article DOI: 10.1038/ncomms13660.

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Bademosi AT, Lauwers E, Padmanabhan P, et al. Publisher Correction: In vivo single-molecule imaging of syntaxin1A reveals polyphosphoinositide- and activity-dependent trapping in presynaptic nanoclusters. Nat Commun. 2018;9:16191doi: 10.1038/ncomms16191.
Bademosi, A. T., Lauwers, E., Padmanabhan, P., Odierna, L., Chai, Y. J., Papadopulos, A., Goodhill, G. J., Verstreken, P., van Swinderen, B., & Meunier, F. A. (2018). Publisher Correction: In vivo single-molecule imaging of syntaxin1A reveals polyphosphoinositide- and activity-dependent trapping in presynaptic nanoclusters. Nature communications, 916191. https://doi.org/10.1038/ncomms16191
Bademosi, Adekunle T, et al. "Publisher Correction: In vivo single-molecule imaging of syntaxin1A reveals polyphosphoinositide- and activity-dependent trapping in presynaptic nanoclusters." Nature communications vol. 9 (2018): 16191. doi: https://doi.org/10.1038/ncomms16191
Bademosi AT, Lauwers E, Padmanabhan P, Odierna L, Chai YJ, Papadopulos A, Goodhill GJ, Verstreken P, van Swinderen B, Meunier FA. Publisher Correction: In vivo single-molecule imaging of syntaxin1A reveals polyphosphoinositide- and activity-dependent trapping in presynaptic nanoclusters. Nat Commun. 2018 Apr 10;9:16191. doi: 10.1038/ncomms16191. PMID: 29633762; PMCID: PMC5898461.
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Percutaneous, Transtrapezial Fixation without Bone Graft Leads to Consolidation in Selected Cases of Delayed Union of the Scaphoid Waist.

Journal of wrist surgery

Vanhees M, van Riet RRP, van Haver A, Kebrle R, Meermans G, Verstreken F.
PMID: 28725498
J Wrist Surg. 2017 Aug;6(3):183-187. doi: 10.1055/s-0036-1597681. Epub 2016 Dec 28.

No abstract available.

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Vanhees M, van Riet RRP, van Haver A, et al. Percutaneous, Transtrapezial Fixation without Bone Graft Leads to Consolidation in Selected Cases of Delayed Union of the Scaphoid Waist. J Wrist Surg. 2016;6(3):183-187doi: 10.1055/s-0036-1597681.
Vanhees, M., van Riet, R. R. P., van Haver, A., Kebrle, R., Meermans, G., & Verstreken, F. (2017). Percutaneous, Transtrapezial Fixation without Bone Graft Leads to Consolidation in Selected Cases of Delayed Union of the Scaphoid Waist. Journal of wrist surgery, 6(3), 183-187. https://doi.org/10.1055/s-0036-1597681
Vanhees, Matthias, et al. "Percutaneous, Transtrapezial Fixation without Bone Graft Leads to Consolidation in Selected Cases of Delayed Union of the Scaphoid Waist." Journal of wrist surgery vol. 6,3 (2017): 183-187. doi: https://doi.org/10.1055/s-0036-1597681
Vanhees M, van Riet RRP, van Haver A, Kebrle R, Meermans G, Verstreken F. Percutaneous, Transtrapezial Fixation without Bone Graft Leads to Consolidation in Selected Cases of Delayed Union of the Scaphoid Waist. J Wrist Surg. 2017 Aug;6(3):183-187. doi: 10.1055/s-0036-1597681. Epub 2016 Dec 28. PMID: 28725498; PMCID: PMC5515604.
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Image-Based Profiling of Synaptic Connectivity in Primary Neuronal Cell Culture.

Frontiers in neuroscience

Verstraelen P, Van Dyck M, Verschuuren M, Kashikar ND, Nuydens R, Timmermans JP, De Vos WH.
PMID: 29997468
Front Neurosci. 2018 Jun 26;12:389. doi: 10.3389/fnins.2018.00389. eCollection 2018.

Neurological disorders display a broad spectrum of clinical manifestations. Yet, at the cellular level, virtually all these diseases converge into a common phenotype of dysregulated synaptic connectivity. In dementia, synapse dysfunction precedes neurodegeneration and cognitive impairment by several years,...

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Verstraelen P, Van Dyck M, Verschuuren M, et al. Image-Based Profiling of Synaptic Connectivity in Primary Neuronal Cell Culture. Front Neurosci. 2018;12:389doi: 10.3389/fnins.2018.00389.
Verstraelen, P., Van Dyck, M., Verschuuren, M., Kashikar, N. D., Nuydens, R., Timmermans, J. P., & De Vos, W. H. (2018). Image-Based Profiling of Synaptic Connectivity in Primary Neuronal Cell Culture. Frontiers in neuroscience, 12389. https://doi.org/10.3389/fnins.2018.00389
Verstraelen, Peter, et al. "Image-Based Profiling of Synaptic Connectivity in Primary Neuronal Cell Culture." Frontiers in neuroscience vol. 12 (2018): 389. doi: https://doi.org/10.3389/fnins.2018.00389
Verstraelen P, Van Dyck M, Verschuuren M, Kashikar ND, Nuydens R, Timmermans JP, De Vos WH. Image-Based Profiling of Synaptic Connectivity in Primary Neuronal Cell Culture. Front Neurosci. 2018 Jun 26;12:389. doi: 10.3389/fnins.2018.00389. eCollection 2018. PMID: 29997468; PMCID: PMC6028601.
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Endophilin-B regulates autophagy during synapse development and neurodegeneration.

Neurobiology of disease

Hernandez-Diaz S, Ghimire S, Sanchez-Mirasierra I, Montecinos-Oliva C, Swerts J, Kuenen S, Verstreken P, Soukup SF.
PMID: 34933093
Neurobiol Dis. 2021 Dec 20;163:105595. doi: 10.1016/j.nbd.2021.105595. Epub 2021 Dec 20.

Synapses are critical for neuronal communication and brain function. To maintain neuronal homeostasis, synapses rely on autophagy. Autophagic alterations cause neurodegeneration and synaptic dysfunction is a feature in neurodegenerative diseases. In Parkinson's disease (PD), where the loss of synapses...

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Hernandez-Diaz S, Ghimire S, Sanchez-Mirasierra I, et al. Endophilin-B regulates autophagy during synapse development and neurodegeneration. Neurobiol Dis. 2021;163:105595doi: 10.1016/j.nbd.2021.105595.
Hernandez-Diaz, S., Ghimire, S., Sanchez-Mirasierra, I., Montecinos-Oliva, C., Swerts, J., Kuenen, S., Verstreken, P., & Soukup, S. F. (2021). Endophilin-B regulates autophagy during synapse development and neurodegeneration. Neurobiology of disease, 163105595. https://doi.org/10.1016/j.nbd.2021.105595
Hernandez-Diaz, Sergio, et al. "Endophilin-B regulates autophagy during synapse development and neurodegeneration." Neurobiology of disease vol. 163 (2021): 105595. doi: https://doi.org/10.1016/j.nbd.2021.105595
Hernandez-Diaz S, Ghimire S, Sanchez-Mirasierra I, Montecinos-Oliva C, Swerts J, Kuenen S, Verstreken P, Soukup SF. Endophilin-B regulates autophagy during synapse development and neurodegeneration. Neurobiol Dis. 2021 Dec 20;163:105595. doi: 10.1016/j.nbd.2021.105595. Epub 2021 Dec 20. PMID: 34933093.
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Synaptic proteostasis in Parkinson's disease.

Current opinion in neurobiology

Nachman E, Verstreken P.
PMID: 34653835
Curr Opin Neurobiol. 2021 Oct 12;72:72-79. doi: 10.1016/j.conb.2021.09.001. Epub 2021 Oct 12.

There are over 7 million people worldwide suffering from Parkinson's disease, and this number will double in the next decade. Causative mutations and risk variants in >20 genes that predominantly act at synapses have been linked to Parkinson's disease....

Cite
Nachman E, Verstreken P. Synaptic proteostasis in Parkinson's disease. Curr Opin Neurobiol. 2021;72:72-79doi: 10.1016/j.conb.2021.09.001.
Nachman, E., & Verstreken, P. (2021). Synaptic proteostasis in Parkinson's disease. Current opinion in neurobiology, 7272-79. https://doi.org/10.1016/j.conb.2021.09.001
Nachman, Eliana, and Verstreken, Patrik. "Synaptic proteostasis in Parkinson's disease." Current opinion in neurobiology vol. 72 (2021): 72-79. doi: https://doi.org/10.1016/j.conb.2021.09.001
Nachman E, Verstreken P. Synaptic proteostasis in Parkinson's disease. Curr Opin Neurobiol. 2021 Oct 12;72:72-79. doi: 10.1016/j.conb.2021.09.001. Epub 2021 Oct 12. PMID: 34653835.
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Endophilin-B regulates autophagy during synapse development and neurodegeneration.

Neurobiology of disease

Hernandez-Diaz S, Ghimire S, Sanchez-Mirasierra I, Montecinos-Oliva C, Swerts J, Kuenen S, Verstreken P, Soukup SF.
PMID: 34933093
Neurobiol Dis. 2021 Dec 20;163:105595. doi: 10.1016/j.nbd.2021.105595. Epub 2021 Dec 20.

Synapses are critical for neuronal communication and brain function. To maintain neuronal homeostasis, synapses rely on autophagy. Autophagic alterations cause neurodegeneration and synaptic dysfunction is a feature in neurodegenerative diseases. In Parkinson's disease (PD), where the loss of synapses...

Cite
Hernandez-Diaz S, Ghimire S, Sanchez-Mirasierra I, et al. Endophilin-B regulates autophagy during synapse development and neurodegeneration. Neurobiol Dis. 2021;163:105595doi: 10.1016/j.nbd.2021.105595.
Hernandez-Diaz, S., Ghimire, S., Sanchez-Mirasierra, I., Montecinos-Oliva, C., Swerts, J., Kuenen, S., Verstreken, P., & Soukup, S. F. (2021). Endophilin-B regulates autophagy during synapse development and neurodegeneration. Neurobiology of disease, 163105595. https://doi.org/10.1016/j.nbd.2021.105595
Hernandez-Diaz, Sergio, et al. "Endophilin-B regulates autophagy during synapse development and neurodegeneration." Neurobiology of disease vol. 163 (2021): 105595. doi: https://doi.org/10.1016/j.nbd.2021.105595
Hernandez-Diaz S, Ghimire S, Sanchez-Mirasierra I, Montecinos-Oliva C, Swerts J, Kuenen S, Verstreken P, Soukup SF. Endophilin-B regulates autophagy during synapse development and neurodegeneration. Neurobiol Dis. 2021 Dec 20;163:105595. doi: 10.1016/j.nbd.2021.105595. Epub 2021 Dec 20. PMID: 34933093.
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MAPRE2 mutations result in altered human cranial neural crest migration, underlying craniofacial malformations in CSC-KT syndrome.

Scientific reports

Thues C, Valadas JS, Deaulmerie L, Geens A, Chouhan AK, Duran-Romaña R, Schymkowitz J, Rousseau F, Bartusel M, Rehimi R, Rada-Iglesias A, Verstreken P, Van Esch H.
PMID: 33654163
Sci Rep. 2021 Mar 02;11(1):4976. doi: 10.1038/s41598-021-83771-3.

Circumferential skin creases (CSC-KT) is a rare polymalformative syndrome characterised by intellectual disability associated with skin creases on the limbs, and very characteristic craniofacial malformations. Previously, heterozygous and homozygous mutations in MAPRE2 were found to be causal for this...

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Thues C, Valadas JS, Deaulmerie L, et al. MAPRE2 mutations result in altered human cranial neural crest migration, underlying craniofacial malformations in CSC-KT syndrome. Sci Rep. 2021;11(1):4976doi: 10.1038/s41598-021-83771-3.
Thues, C., Valadas, J. S., Deaulmerie, L., Geens, A., Chouhan, A. K., Duran-Romaña, R., Schymkowitz, J., Rousseau, F., Bartusel, M., Rehimi, R., Rada-Iglesias, A., Verstreken, P., & Van Esch, H. (2021). MAPRE2 mutations result in altered human cranial neural crest migration, underlying craniofacial malformations in CSC-KT syndrome. Scientific reports, 11(1), 4976. https://doi.org/10.1038/s41598-021-83771-3
Thues, Cedric, et al. "MAPRE2 mutations result in altered human cranial neural crest migration, underlying craniofacial malformations in CSC-KT syndrome." Scientific reports vol. 11,1 (2021): 4976. doi: https://doi.org/10.1038/s41598-021-83771-3
Thues C, Valadas JS, Deaulmerie L, Geens A, Chouhan AK, Duran-Romaña R, Schymkowitz J, Rousseau F, Bartusel M, Rehimi R, Rada-Iglesias A, Verstreken P, Van Esch H. MAPRE2 mutations result in altered human cranial neural crest migration, underlying craniofacial malformations in CSC-KT syndrome. Sci Rep. 2021 Mar 02;11(1):4976. doi: 10.1038/s41598-021-83771-3. PMID: 33654163; PMCID: PMC7925611.
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Circulating SERPINA3 improves prognostic stratification in patients with a de novo or worsened heart failure.

ESC heart failure

Delrue L, Vanderheyden M, Beles M, Paolisso P, Di Gioia G, Dierckx R, Verstreken S, Goethals M, Heggermont W, Bartunek J.
PMID: 34725968
ESC Heart Fail. 2021 Dec;8(6):4780-4790. doi: 10.1002/ehf2.13659. Epub 2021 Nov 01.

AIMS: We investigated the prognostic relevance of serpin peptidase inhibitor, clade A member 3 (SERPINA3) in patients admitted with a de novo or worsened heart failure (HF).METHODS AND RESULTS: In the first stage, 83 HF-related left ventricular (LV) transcripts...

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Delrue L, Vanderheyden M, Beles M, et al. Circulating SERPINA3 improves prognostic stratification in patients with a de novo or worsened heart failure. ESC Heart Fail. 2021;8(6):4780-4790doi: 10.1002/ehf2.13659.
Delrue, L., Vanderheyden, M., Beles, M., Paolisso, P., Di Gioia, G., Dierckx, R., Verstreken, S., Goethals, M., Heggermont, W., & Bartunek, J. (2021). Circulating SERPINA3 improves prognostic stratification in patients with a de novo or worsened heart failure. ESC heart failure, 8(6), 4780-4790. https://doi.org/10.1002/ehf2.13659
Delrue, Leen, et al. "Circulating SERPINA3 improves prognostic stratification in patients with a de novo or worsened heart failure." ESC heart failure vol. 8,6 (2021): 4780-4790. doi: https://doi.org/10.1002/ehf2.13659
Delrue L, Vanderheyden M, Beles M, Paolisso P, Di Gioia G, Dierckx R, Verstreken S, Goethals M, Heggermont W, Bartunek J. Circulating SERPINA3 improves prognostic stratification in patients with a de novo or worsened heart failure. ESC Heart Fail. 2021 Dec;8(6):4780-4790. doi: 10.1002/ehf2.13659. Epub 2021 Nov 01. PMID: 34725968.
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Synaptic proteostasis in Parkinson's disease.

Current opinion in neurobiology

Nachman E, Verstreken P.
PMID: 34653835
Curr Opin Neurobiol. 2021 Oct 12;72:72-79. doi: 10.1016/j.conb.2021.09.001. Epub 2021 Oct 12.

There are over 7 million people worldwide suffering from Parkinson's disease, and this number will double in the next decade. Causative mutations and risk variants in >20 genes that predominantly act at synapses have been linked to Parkinson's disease....

Cite
Nachman E, Verstreken P. Synaptic proteostasis in Parkinson's disease. Curr Opin Neurobiol. 2021;72:72-79doi: 10.1016/j.conb.2021.09.001.
Nachman, E., & Verstreken, P. (2021). Synaptic proteostasis in Parkinson's disease. Current opinion in neurobiology, 7272-79. https://doi.org/10.1016/j.conb.2021.09.001
Nachman, Eliana, and Verstreken, Patrik. "Synaptic proteostasis in Parkinson's disease." Current opinion in neurobiology vol. 72 (2021): 72-79. doi: https://doi.org/10.1016/j.conb.2021.09.001
Nachman E, Verstreken P. Synaptic proteostasis in Parkinson's disease. Curr Opin Neurobiol. 2021 Oct 12;72:72-79. doi: 10.1016/j.conb.2021.09.001. Epub 2021 Oct 12. PMID: 34653835.
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Showing 1 to 12 of 66 entries