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Analysis of tryptophan, tyrosine and related dipeptides in mouse brain by isocratic high-performance liquid chromatography with switchable wavelength fluorescence detection.

Journal of pharmaceutical and biomedical analysis

Viell B, Weidler B, Krause B, Vestweber KH.
PMID: 16867364
J Pharm Biomed Anal. 1988;6(6):933-8. doi: 10.1016/0731-7085(88)80112-2.

Tryptophan (Trp) and tyrosine (Tyr) are pharmacologically active compounds which, after administration of adequate doses, are increased in level in the brain, and stimulate neurotransmitter synthesis. Trp and Tyr containing dipeptides were tested as possible substitutes with regard to...

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Viell B, Weidler B, Krause B, et al. Analysis of tryptophan, tyrosine and related dipeptides in mouse brain by isocratic high-performance liquid chromatography with switchable wavelength fluorescence detection. J Pharm Biomed Anal. 1988;6(6):933-8doi: 10.1016/0731-7085(88)80112-2.
Viell, B., Weidler, B., Krause, B., & Vestweber, K. H. (1988). Analysis of tryptophan, tyrosine and related dipeptides in mouse brain by isocratic high-performance liquid chromatography with switchable wavelength fluorescence detection. Journal of pharmaceutical and biomedical analysis, 6(6), 933-8. https://doi.org/10.1016/0731-7085(88)80112-2
Viell, B, et al. "Analysis of tryptophan, tyrosine and related dipeptides in mouse brain by isocratic high-performance liquid chromatography with switchable wavelength fluorescence detection." Journal of pharmaceutical and biomedical analysis vol. 6,6 (1988): 933-8. doi: https://doi.org/10.1016/0731-7085(88)80112-2
Viell B, Weidler B, Krause B, Vestweber KH. Analysis of tryptophan, tyrosine and related dipeptides in mouse brain by isocratic high-performance liquid chromatography with switchable wavelength fluorescence detection. J Pharm Biomed Anal. 1988;6(6):933-8. doi: 10.1016/0731-7085(88)80112-2. PMID: 16867364.
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VIPAR, a quantitative approach to 3D histopathology applied to lymphatic malformations.

JCI insight

Hägerling R, Drees D, Scherzinger A, Dierkes C, Martin-Almedina S, Butz S, Gordon K, Schäfers M, Hinrichs K, Ostergaard P, Vestweber D, Goerge T, Mansour S, Jiang X, Mortimer PS, Kiefer F.
PMID: 28814672
JCI Insight. 2017 Aug 17;2(16). doi: 10.1172/jci.insight.93424. eCollection 2017 Aug 17.

BACKGROUND: Lack of investigatory and diagnostic tools has been a major contributing factor to the failure to mechanistically understand lymphedema and other lymphatic disorders in order to develop effective drug and surgical therapies. One difficulty has been understanding the...

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Hägerling R, Drees D, Scherzinger A, et al. VIPAR, a quantitative approach to 3D histopathology applied to lymphatic malformations. JCI Insight. 2017;2(16)doi: 10.1172/jci.insight.93424.
Hägerling, R., Drees, D., Scherzinger, A., Dierkes, C., Martin-Almedina, S., Butz, S., Gordon, K., Schäfers, M., Hinrichs, K., Ostergaard, P., Vestweber, D., Goerge, T., Mansour, S., Jiang, X., Mortimer, P. S., & Kiefer, F. (2017). VIPAR, a quantitative approach to 3D histopathology applied to lymphatic malformations. JCI insight, 2(16), . https://doi.org/10.1172/jci.insight.93424
Hägerling, René, et al. "VIPAR, a quantitative approach to 3D histopathology applied to lymphatic malformations." JCI insight vol. 2,16 (2017). doi: https://doi.org/10.1172/jci.insight.93424
Hägerling R, Drees D, Scherzinger A, Dierkes C, Martin-Almedina S, Butz S, Gordon K, Schäfers M, Hinrichs K, Ostergaard P, Vestweber D, Goerge T, Mansour S, Jiang X, Mortimer PS, Kiefer F. VIPAR, a quantitative approach to 3D histopathology applied to lymphatic malformations. JCI Insight. 2017 Aug 17;2(16). doi: 10.1172/jci.insight.93424. eCollection 2017 Aug 17. PMID: 28814672; PMCID: PMC5621876.
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Extracellular Vesicle Transfer from Endothelial Cells Drives VE-Cadherin Expression in Breast Cancer Cells, Thereby Causing Heterotypic Cell Contacts.

Cancers

Rezaei M, Martins Cavaco AC, Stehling M, Nottebaum A, Brockhaus K, Caliandro MF, Schelhaas S, Schmalbein F, Vestweber D, Eble JA.
PMID: 32752204
Cancers (Basel). 2020 Aug 01;12(8). doi: 10.3390/cancers12082138.

Cadherins mediate cohesive contacts between isotypic cells by homophilic interaction and prevent contact between heterotypic cells. Breast cancer cells neighboring endothelial cells (ECs) atypically express vascular endothelial (VE)-cadherin. To understand this EC-induced VE-cadherin expression in breast cancer cells, MCF7...

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Rezaei M, Martins Cavaco AC, Stehling M, et al. Extracellular Vesicle Transfer from Endothelial Cells Drives VE-Cadherin Expression in Breast Cancer Cells, Thereby Causing Heterotypic Cell Contacts. Cancers (Basel). 2020;12(8)doi: 10.3390/cancers12082138.
Rezaei, M., Martins Cavaco, A. C., Stehling, M., Nottebaum, A., Brockhaus, K., Caliandro, M. F., Schelhaas, S., Schmalbein, F., Vestweber, D., & Eble, J. A. (2020). Extracellular Vesicle Transfer from Endothelial Cells Drives VE-Cadherin Expression in Breast Cancer Cells, Thereby Causing Heterotypic Cell Contacts. Cancers, 12(8), . https://doi.org/10.3390/cancers12082138
Rezaei, Maryam, et al. "Extracellular Vesicle Transfer from Endothelial Cells Drives VE-Cadherin Expression in Breast Cancer Cells, Thereby Causing Heterotypic Cell Contacts." Cancers vol. 12,8 (2020). doi: https://doi.org/10.3390/cancers12082138
Rezaei M, Martins Cavaco AC, Stehling M, Nottebaum A, Brockhaus K, Caliandro MF, Schelhaas S, Schmalbein F, Vestweber D, Eble JA. Extracellular Vesicle Transfer from Endothelial Cells Drives VE-Cadherin Expression in Breast Cancer Cells, Thereby Causing Heterotypic Cell Contacts. Cancers (Basel). 2020 Aug 01;12(8). doi: 10.3390/cancers12082138. PMID: 32752204; PMCID: PMC7463713.
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Locking endothelial junctions blocks leukocyte extravasation, but not in all tissues.

Tissue barriers

Küppers V, Vestweber D, Schulte D.
PMID: 24665379
Tissue Barriers. 2013 Jan 01;1(1):e23805. doi: 10.4161/tisb.23805.

The passage of leukocytes across the blood vessel wall is a fundamental event in the inflammatory response. During the last decades, there has been significant progress in understanding the molecular mechanisms involved in leukocyte transmigration. However, it is still...

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Küppers V, Vestweber D, Schulte D. Locking endothelial junctions blocks leukocyte extravasation, but not in all tissues. Tissue Barriers. 2013;1(1):e23805doi: 10.4161/tisb.23805.
Küppers, V., Vestweber, D., & Schulte, D. (2013). Locking endothelial junctions blocks leukocyte extravasation, but not in all tissues. Tissue barriers, 1(1), e23805. https://doi.org/10.4161/tisb.23805
Küppers, Verena, et al. "Locking endothelial junctions blocks leukocyte extravasation, but not in all tissues." Tissue barriers vol. 1,1 (2013): e23805. doi: https://doi.org/10.4161/tisb.23805
Küppers V, Vestweber D, Schulte D. Locking endothelial junctions blocks leukocyte extravasation, but not in all tissues. Tissue Barriers. 2013 Jan 01;1(1):e23805. doi: 10.4161/tisb.23805. PMID: 24665379; PMCID: PMC3879176.
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Analysis of 5-OH-indoles in human gut biopsy tissues by reversed-phase high-performance liquid chromatography with fluorimetric detection.

Journal of pharmaceutical and biomedical analysis

Viell B, Vestweber KH, Krause B.
PMID: 16867365
J Pharm Biomed Anal. 1988;6(6):939-44. doi: 10.1016/0731-7085(88)80113-4.

A method was devised for the rapid simultaneous determination of major indoles in human gut tissues. Analysis with picomol detection limits was done by HPLC on a C(18) reversed-phase column with fluorimetric detection at 276/350 nm. This simple method...

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Viell B, Vestweber KH, Krause B. Analysis of 5-OH-indoles in human gut biopsy tissues by reversed-phase high-performance liquid chromatography with fluorimetric detection. J Pharm Biomed Anal. 1988;6(6):939-44doi: 10.1016/0731-7085(88)80113-4.
Viell, B., Vestweber, K. H., & Krause, B. (1988). Analysis of 5-OH-indoles in human gut biopsy tissues by reversed-phase high-performance liquid chromatography with fluorimetric detection. Journal of pharmaceutical and biomedical analysis, 6(6), 939-44. https://doi.org/10.1016/0731-7085(88)80113-4
Viell, B, et al. "Analysis of 5-OH-indoles in human gut biopsy tissues by reversed-phase high-performance liquid chromatography with fluorimetric detection." Journal of pharmaceutical and biomedical analysis vol. 6,6 (1988): 939-44. doi: https://doi.org/10.1016/0731-7085(88)80113-4
Viell B, Vestweber KH, Krause B. Analysis of 5-OH-indoles in human gut biopsy tissues by reversed-phase high-performance liquid chromatography with fluorimetric detection. J Pharm Biomed Anal. 1988;6(6):939-44. doi: 10.1016/0731-7085(88)80113-4. PMID: 16867365.
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A Novel Cervical Spinal Cord Window Preparation Allows for Two-Photon Imaging of T-Cell Interactions with the Cervical Spinal Cord Microvasculature during Experimental Autoimmune Encephalomyelitis.

Frontiers in immunology

Haghayegh Jahromi N, Tardent H, Enzmann G, Deutsch U, Kawakami N, Bittner S, Vestweber D, Zipp F, Stein JV, Engelhardt B.
PMID: 28443093
Front Immunol. 2017 Apr 11;8:406. doi: 10.3389/fimmu.2017.00406. eCollection 2017.

T-cell migration across the blood-brain barrier (BBB) is a crucial step in the pathogenesis of experimental autoimmune encephalomyelitis (EAE), an animal model of multiple sclerosis (MS). Two-photon intravital microscopy (2P-IVM) has been established as a powerful tool to study...

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Haghayegh Jahromi N, Tardent H, Enzmann G, et al. A Novel Cervical Spinal Cord Window Preparation Allows for Two-Photon Imaging of T-Cell Interactions with the Cervical Spinal Cord Microvasculature during Experimental Autoimmune Encephalomyelitis. Front Immunol. 2017;8:406doi: 10.3389/fimmu.2017.00406.
Haghayegh Jahromi, N., Tardent, H., Enzmann, G., Deutsch, U., Kawakami, N., Bittner, S., Vestweber, D., Zipp, F., Stein, J. V., & Engelhardt, B. (2017). A Novel Cervical Spinal Cord Window Preparation Allows for Two-Photon Imaging of T-Cell Interactions with the Cervical Spinal Cord Microvasculature during Experimental Autoimmune Encephalomyelitis. Frontiers in immunology, 8406. https://doi.org/10.3389/fimmu.2017.00406
Haghayegh Jahromi, Neda, et al. "A Novel Cervical Spinal Cord Window Preparation Allows for Two-Photon Imaging of T-Cell Interactions with the Cervical Spinal Cord Microvasculature during Experimental Autoimmune Encephalomyelitis." Frontiers in immunology vol. 8 (2017): 406. doi: https://doi.org/10.3389/fimmu.2017.00406
Haghayegh Jahromi N, Tardent H, Enzmann G, Deutsch U, Kawakami N, Bittner S, Vestweber D, Zipp F, Stein JV, Engelhardt B. A Novel Cervical Spinal Cord Window Preparation Allows for Two-Photon Imaging of T-Cell Interactions with the Cervical Spinal Cord Microvasculature during Experimental Autoimmune Encephalomyelitis. Front Immunol. 2017 Apr 11;8:406. doi: 10.3389/fimmu.2017.00406. eCollection 2017. PMID: 28443093; PMCID: PMC5387098.
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Force-induced changes of α-catenin conformation stabilize vascular junctions independently of vinculin.

Journal of cell science

Duong CN, Brückner R, Schmitt M, Nottebaum AF, Braun LJ, Meyer Zu Brickwedde M, Ipe U, Vom Bruch H, Schöler HR, Trapani G, Trappmann B, Ebrahimkutty MP, Huveneers S, de Rooij J, Ishiyama N, Ikura M, Vestweber D.
PMID: 34851405
J Cell Sci. 2021 Dec 15;134(24). doi: 10.1242/jcs.259012. Epub 2021 Dec 22.

Cadherin-mediated cell adhesion requires anchoring via the β-catenin-α-catenin complex to the actin cytoskeleton, yet, α-catenin only binds F-actin weakly. A covalent fusion of VE-cadherin to α-catenin enhances actin anchorage in endothelial cells and strongly stabilizes endothelial junctions in vivo,...

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Duong CN, Brückner R, Schmitt M, et al. Force-induced changes of α-catenin conformation stabilize vascular junctions independently of vinculin. J Cell Sci. 2021;134(24)doi: 10.1242/jcs.259012.
Duong, C. N., Brückner, R., Schmitt, M., Nottebaum, A. F., Braun, L. J., Meyer Zu Brickwedde, M., Ipe, U., Vom Bruch, H., Schöler, H. R., Trapani, G., Trappmann, B., Ebrahimkutty, M. P., Huveneers, S., de Rooij, J., Ishiyama, N., Ikura, M., & Vestweber, D. (2021). Force-induced changes of α-catenin conformation stabilize vascular junctions independently of vinculin. Journal of cell science, 134(24), . https://doi.org/10.1242/jcs.259012
Duong, Cao Nguyen, et al. "Force-induced changes of α-catenin conformation stabilize vascular junctions independently of vinculin." Journal of cell science vol. 134,24 (2021). doi: https://doi.org/10.1242/jcs.259012
Duong CN, Brückner R, Schmitt M, Nottebaum AF, Braun LJ, Meyer Zu Brickwedde M, Ipe U, Vom Bruch H, Schöler HR, Trapani G, Trappmann B, Ebrahimkutty MP, Huveneers S, de Rooij J, Ishiyama N, Ikura M, Vestweber D. Force-induced changes of α-catenin conformation stabilize vascular junctions independently of vinculin. J Cell Sci. 2021 Dec 15;134(24). doi: 10.1242/jcs.259012. Epub 2021 Dec 22. PMID: 34851405.
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Conventional mesh repair of a giant iatrogenic bilateral diaphragmatic hernia with an enterothorax.

International medical case reports journal

Lingohr P, Galetin T, Vestweber B, Matthaei H, Kalff JC, Vestweber KH.
PMID: 24600251
Int Med Case Rep J. 2014 Feb 12;7:23-5. doi: 10.2147/IMCRJ.S54361. eCollection 2014.

PURPOSE: Diaphragmatic hernias (DHs) are divided into congenital and acquired hernias, most of which are congenital. Among acquired DHs, up to 80% are left-sided, only a few iatrogenic DHs have been reported, and bilateral hernias are extremely rare. For...

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Lingohr P, Galetin T, Vestweber B, et al. Conventional mesh repair of a giant iatrogenic bilateral diaphragmatic hernia with an enterothorax. Int Med Case Rep J. 2014;7:23-5doi: 10.2147/IMCRJ.S54361.
Lingohr, P., Galetin, T., Vestweber, B., Matthaei, H., Kalff, J. C., & Vestweber, K. H. (2014). Conventional mesh repair of a giant iatrogenic bilateral diaphragmatic hernia with an enterothorax. International medical case reports journal, 723-5. https://doi.org/10.2147/IMCRJ.S54361
Lingohr, Philipp, et al. "Conventional mesh repair of a giant iatrogenic bilateral diaphragmatic hernia with an enterothorax." International medical case reports journal vol. 7 (2014): 23-5. doi: https://doi.org/10.2147/IMCRJ.S54361
Lingohr P, Galetin T, Vestweber B, Matthaei H, Kalff JC, Vestweber KH. Conventional mesh repair of a giant iatrogenic bilateral diaphragmatic hernia with an enterothorax. Int Med Case Rep J. 2014 Feb 12;7:23-5. doi: 10.2147/IMCRJ.S54361. eCollection 2014. PMID: 24600251; PMCID: PMC3928060.
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Cortactin regulates the activity of small GTPases and ICAM-1 clustering in endothelium: Implications for the formation of docking structures.

Tissue barriers

Vestweber D, Zeuschner D, Rottner K, Schnoor M.
PMID: 24665381
Tissue Barriers. 2013 Jan 01;1(1):e23862. doi: 10.4161/tisb.23862.

Cortactin is an actin-binding molecule that regulates various cellular processes requiring actin dynamics. We recently described cortactin-deficient mice and despite its pivotal role for actin remodeling in vitro, these mice are surprisingly healthy. Analyzing cortactin functions in endothelium under...

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Vestweber D, Zeuschner D, Rottner K, et al. Cortactin regulates the activity of small GTPases and ICAM-1 clustering in endothelium: Implications for the formation of docking structures. Tissue Barriers. 2013;1(1):e23862doi: 10.4161/tisb.23862.
Vestweber, D., Zeuschner, D., Rottner, K., & Schnoor, M. (2013). Cortactin regulates the activity of small GTPases and ICAM-1 clustering in endothelium: Implications for the formation of docking structures. Tissue barriers, 1(1), e23862. https://doi.org/10.4161/tisb.23862
Vestweber, Dietmar, et al. "Cortactin regulates the activity of small GTPases and ICAM-1 clustering in endothelium: Implications for the formation of docking structures." Tissue barriers vol. 1,1 (2013): e23862. doi: https://doi.org/10.4161/tisb.23862
Vestweber D, Zeuschner D, Rottner K, Schnoor M. Cortactin regulates the activity of small GTPases and ICAM-1 clustering in endothelium: Implications for the formation of docking structures. Tissue Barriers. 2013 Jan 01;1(1):e23862. doi: 10.4161/tisb.23862. PMID: 24665381; PMCID: PMC3879184.
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VE-Cadherin Phosphorylation Regulates Endothelial Fluid Shear Stress Responses through the Polarity Protein LGN.

Current biology : CB

Conway DE, Coon BG, Budatha M, Arsenovic PT, Orsenigo F, Wessel F, Zhang J, Zhuang Z, Dejana E, Vestweber D, Schwartz MA.
PMID: 28898639
Curr Biol. 2017 Sep 11;27(17):2727. doi: 10.1016/j.cub.2017.08.064.

No abstract available.

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Conway DE, Coon BG, Budatha M, et al. VE-Cadherin Phosphorylation Regulates Endothelial Fluid Shear Stress Responses through the Polarity Protein LGN. Curr Biol. 2017;27(17):2727doi: 10.1016/j.cub.2017.08.064.
Conway, D. E., Coon, B. G., Budatha, M., Arsenovic, P. T., Orsenigo, F., Wessel, F., Zhang, J., Zhuang, Z., Dejana, E., Vestweber, D., & Schwartz, M. A. (2017). VE-Cadherin Phosphorylation Regulates Endothelial Fluid Shear Stress Responses through the Polarity Protein LGN. Current biology : CB, 27(17), 2727. https://doi.org/10.1016/j.cub.2017.08.064
Conway, Daniel E, et al. "VE-Cadherin Phosphorylation Regulates Endothelial Fluid Shear Stress Responses through the Polarity Protein LGN." Current biology : CB vol. 27,17 (2017): 2727. doi: https://doi.org/10.1016/j.cub.2017.08.064
Conway DE, Coon BG, Budatha M, Arsenovic PT, Orsenigo F, Wessel F, Zhang J, Zhuang Z, Dejana E, Vestweber D, Schwartz MA. VE-Cadherin Phosphorylation Regulates Endothelial Fluid Shear Stress Responses through the Polarity Protein LGN. Curr Biol. 2017 Sep 11;27(17):2727. doi: 10.1016/j.cub.2017.08.064. PMID: 28898639; PMCID: PMC5752114.
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Mechanisms Ensuring Endothelial Junction Integrity Beyond VE-Cadherin.

Frontiers in physiology

Duong CN, Vestweber D.
PMID: 32670077
Front Physiol. 2020 May 21;11:519. doi: 10.3389/fphys.2020.00519. eCollection 2020.

Endothelial junctions provide blood and lymph vessel integrity and are essential for the formation of a vascular system. They control the extravasation of solutes, leukocytes and metastatic cells from blood vessels and the uptake of fluid and leukocytes into...

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Duong CN, Vestweber D. Mechanisms Ensuring Endothelial Junction Integrity Beyond VE-Cadherin. Front Physiol. 2020;11:519doi: 10.3389/fphys.2020.00519.
Duong, C. N., & Vestweber, D. (2020). Mechanisms Ensuring Endothelial Junction Integrity Beyond VE-Cadherin. Frontiers in physiology, 11519. https://doi.org/10.3389/fphys.2020.00519
Duong, Cao Nguyen, and Vestweber, Dietmar. "Mechanisms Ensuring Endothelial Junction Integrity Beyond VE-Cadherin." Frontiers in physiology vol. 11 (2020): 519. doi: https://doi.org/10.3389/fphys.2020.00519
Duong CN, Vestweber D. Mechanisms Ensuring Endothelial Junction Integrity Beyond VE-Cadherin. Front Physiol. 2020 May 21;11:519. doi: 10.3389/fphys.2020.00519. eCollection 2020. PMID: 32670077; PMCID: PMC7326147.
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Synthetic Inhibitors of Cell Adhesion: A Glycopeptide from E-Selectin Ligand 1 (ESL-1) with the Arabino Sialyl Lewis(x) Structure This work was supported by the Deutsche Forschungsgemeinschaft and by the Fonds der Chemischen Industrie.

Angewandte Chemie (International ed. in English)

Rösch M, Herzner H, Dippold W, Wild M, Vestweber D, Kunz H.
PMID: 11668549
Angew Chem Int Ed Engl. 2001 Oct 15;40(20):3836-3839.

No abstract available.

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Rösch M, Herzner H, Dippold W, et al. Synthetic Inhibitors of Cell Adhesion: A Glycopeptide from E-Selectin Ligand 1 (ESL-1) with the Arabino Sialyl Lewis(x) Structure This work was supported by the Deutsche Forschungsgemeinschaft and by the Fonds der Chemischen Industrie. Angew Chem Int Ed Engl. 2001;40(20):3836-3839
Rösch, M., Herzner, H., Dippold, W., Wild, M., Vestweber, D., & Kunz, H. (2001). Synthetic Inhibitors of Cell Adhesion: A Glycopeptide from E-Selectin Ligand 1 (ESL-1) with the Arabino Sialyl Lewis(x) Structure This work was supported by the Deutsche Forschungsgemeinschaft and by the Fonds der Chemischen Industrie. Angewandte Chemie (International ed. in English), 40(20), 3836-3839.
Rösch, Markus, et al. "Synthetic Inhibitors of Cell Adhesion: A Glycopeptide from E-Selectin Ligand 1 (ESL-1) with the Arabino Sialyl Lewis(x) Structure This work was supported by the Deutsche Forschungsgemeinschaft and by the Fonds der Chemischen Industrie." Angewandte Chemie (International ed. in English) vol. 40,20 (2001): 3836-3839.
Rösch M, Herzner H, Dippold W, Wild M, Vestweber D, Kunz H. Synthetic Inhibitors of Cell Adhesion: A Glycopeptide from E-Selectin Ligand 1 (ESL-1) with the Arabino Sialyl Lewis(x) Structure This work was supported by the Deutsche Forschungsgemeinschaft and by the Fonds der Chemischen Industrie. Angew Chem Int Ed Engl. 2001 Oct 15;40(20):3836-3839. PMID: 11668549.
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