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Showing 1 to 12 of 71 entries
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Double-blind, Placebo-controlled Study Assessing the Effect of Chocolate Consumption in Subjects with a History of Acne Vulgaris.

The Journal of clinical and aesthetic dermatology

Caperton C, Block S, Viera M, Keri J, Berman B.
PMID: 24847404
J Clin Aesthet Dermatol. 2014 May;7(5):19-23.

OBJECTIVE: To assess the effect of chocolate on acne exacerbation in males between the ages of 18 and 35 with a history of acne vulgaris.DESIGN: Double-blind, placebo-controlled, randomized, controlled trial.SETTING: Single-site, outpatient, research, clinical facility at an academic research...

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Caperton C, Block S, Viera M, et al. Double-blind, Placebo-controlled Study Assessing the Effect of Chocolate Consumption in Subjects with a History of Acne Vulgaris. J Clin Aesthet Dermatol. 2014;7(5):19-23
Caperton, C., Block, S., Viera, M., Keri, J., & Berman, B. (2014). Double-blind, Placebo-controlled Study Assessing the Effect of Chocolate Consumption in Subjects with a History of Acne Vulgaris. The Journal of clinical and aesthetic dermatology, 7(5), 19-23.
Caperton, Caroline, et al. "Double-blind, Placebo-controlled Study Assessing the Effect of Chocolate Consumption in Subjects with a History of Acne Vulgaris." The Journal of clinical and aesthetic dermatology vol. 7,5 (2014): 19-23.
Caperton C, Block S, Viera M, Keri J, Berman B. Double-blind, Placebo-controlled Study Assessing the Effect of Chocolate Consumption in Subjects with a History of Acne Vulgaris. J Clin Aesthet Dermatol. 2014 May;7(5):19-23. PMID: 24847404; PMCID: PMC4025515.
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Vascular Anomalies Caused by Abnormal Signaling within Endothelial Cells: Targets for Novel Therapies.

Seminars in interventional radiology

Nguyen HL, Boon LM, Vikkula M.
PMID: 28955112
Semin Intervent Radiol. 2017 Sep;34(3):233-238. doi: 10.1055/s-0037-1604296. Epub 2017 Sep 11.

Vascular anomalies arise as a consequence of improper development and maintenance of the vasculature. Our knowledge on the pathophysiological bases of vascular anomalies has skyrocketed during the past 5 years. It is becoming clear that common intracellular signaling pathways...

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Nguyen HL, Boon LM, Vikkula M. Vascular Anomalies Caused by Abnormal Signaling within Endothelial Cells: Targets for Novel Therapies. Semin Intervent Radiol. 2017;34(3):233-238doi: 10.1055/s-0037-1604296.
Nguyen, H. L., Boon, L. M., & Vikkula, M. (2017). Vascular Anomalies Caused by Abnormal Signaling within Endothelial Cells: Targets for Novel Therapies. Seminars in interventional radiology, 34(3), 233-238. https://doi.org/10.1055/s-0037-1604296
Nguyen, Ha-Long, et al. "Vascular Anomalies Caused by Abnormal Signaling within Endothelial Cells: Targets for Novel Therapies." Seminars in interventional radiology vol. 34,3 (2017): 233-238. doi: https://doi.org/10.1055/s-0037-1604296
Nguyen HL, Boon LM, Vikkula M. Vascular Anomalies Caused by Abnormal Signaling within Endothelial Cells: Targets for Novel Therapies. Semin Intervent Radiol. 2017 Sep;34(3):233-238. doi: 10.1055/s-0037-1604296. Epub 2017 Sep 11. PMID: 28955112; PMCID: PMC5615384.
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Molecular basis of vascular anomalies.

Trends in cardiovascular medicine

Vikkula M, Boon LM, Mulliken JB, Olsen BR.
PMID: 14987552
Trends Cardiovasc Med. 1998 Oct;8(7):281-92. doi: 10.1016/s1050-1738(98)00024-3.

Vascular anomalies comprise a heterogeneous group of disorders that are divided into tumors (hemangiomas) and malformations. Recent advances in biomedical research provide insights into the molecular basis of these disorders and a deeper understanding of vascular morphogenesis. In the...

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Vikkula M, Boon LM, Mulliken JB, et al. Molecular basis of vascular anomalies. Trends Cardiovasc Med. 1998;8(7):281-92doi: 10.1016/s1050-1738(98)00024-3.
Vikkula, M., Boon, L. M., Mulliken, J. B., & Olsen, B. R. (1998). Molecular basis of vascular anomalies. Trends in cardiovascular medicine, 8(7), 281-92. https://doi.org/10.1016/s1050-1738(98)00024-3
Vikkula, M, et al. "Molecular basis of vascular anomalies." Trends in cardiovascular medicine vol. 8,7 (1998): 281-92. doi: https://doi.org/10.1016/s1050-1738(98)00024-3
Vikkula M, Boon LM, Mulliken JB, Olsen BR. Molecular basis of vascular anomalies. Trends Cardiovasc Med. 1998 Oct;8(7):281-92. doi: 10.1016/s1050-1738(98)00024-3. PMID: 14987552.
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A new look at fibroepithelioma of pinkus: features on confocal microscopy.

The Journal of clinical and aesthetic dermatology

Viera M, Amini S, Huo R, Oliviero M, Bassalo S, Rabinovitz H.
PMID: 21103323
J Clin Aesthet Dermatol. 2008 Jul;1(2):42-4.

No abstract available.

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Viera M, Amini S, Huo R, et al. A new look at fibroepithelioma of pinkus: features on confocal microscopy. J Clin Aesthet Dermatol. 2008;1(2):42-4
Viera, M., Amini, S., Huo, R., Oliviero, M., Bassalo, S., & Rabinovitz, H. (2008). A new look at fibroepithelioma of pinkus: features on confocal microscopy. The Journal of clinical and aesthetic dermatology, 1(2), 42-4.
Viera, Martha, et al. "A new look at fibroepithelioma of pinkus: features on confocal microscopy." The Journal of clinical and aesthetic dermatology vol. 1,2 (2008): 42-4.
Viera M, Amini S, Huo R, Oliviero M, Bassalo S, Rabinovitz H. A new look at fibroepithelioma of pinkus: features on confocal microscopy. J Clin Aesthet Dermatol. 2008 Jul;1(2):42-4. PMID: 21103323; PMCID: PMC2989825.
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Mutations in the VEGFR3 signaling pathway explain 36% of familial lymphedema.

Molecular syndromology

Mendola A, Schlögel MJ, Ghalamkarpour A, Irrthum A, Nguyen HL, Fastré E, Bygum A, van der Vleuten C, Fagerberg C, Baselga E, Quere I, Mulliken JB, Boon LM, Brouillard P, Vikkula M.
PMID: 24167460
Mol Syndromol. 2013 Sep;4(6):257-66. doi: 10.1159/000354097. Epub 2013 Aug 21.

Lymphedema is caused by dysfunction of lymphatic vessels, leading to disabling swelling that occurs mostly on the extremities. Lymphedema can be either primary (congenital) or secondary (acquired). Familial primary lymphedema commonly segregates in an autosomal dominant or recessive manner....

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Mendola A, Schlögel MJ, Ghalamkarpour A, et al. Mutations in the VEGFR3 signaling pathway explain 36% of familial lymphedema. Mol Syndromol. 2013;4(6):257-66doi: 10.1159/000354097.
Mendola, A., Schlögel, M. J., Ghalamkarpour, A., Irrthum, A., Nguyen, H. L., Fastré, E., Bygum, A., van der Vleuten, C., Fagerberg, C., Baselga, E., Quere, I., Mulliken, J. B., Boon, L. M., Brouillard, P., Vikkula, M. (2013). Mutations in the VEGFR3 signaling pathway explain 36% of familial lymphedema. Molecular syndromology, 4(6), 257-66. https://doi.org/10.1159/000354097
Mendola, A, et al. "Mutations in the VEGFR3 signaling pathway explain 36% of familial lymphedema." Molecular syndromology vol. 4,6 (2013): 257-66. doi: https://doi.org/10.1159/000354097
Mendola A, Schlögel MJ, Ghalamkarpour A, Irrthum A, Nguyen HL, Fastré E, Bygum A, van der Vleuten C, Fagerberg C, Baselga E, Quere I, Mulliken JB, Boon LM, Brouillard P, Vikkula M. Mutations in the VEGFR3 signaling pathway explain 36% of familial lymphedema. Mol Syndromol. 2013 Sep;4(6):257-66. doi: 10.1159/000354097. Epub 2013 Aug 21. PMID: 24167460; PMCID: PMC3776465.
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Letter: Is Developmental Venous Anomaly an Imaging Biomarker of PIK3CA Mutated Gliomas?.

Neurosurgery

Roux A, Vikkula M, Pallud J.
PMID: 31742343
Neurosurgery. 2020 Jan 01;86(1):E93. doi: 10.1093/neuros/nyz425.

No abstract available.

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Roux A, Vikkula M, Pallud J. Letter: Is Developmental Venous Anomaly an Imaging Biomarker of PIK3CA Mutated Gliomas?. Neurosurgery. 2020;86(1):E93doi: 10.1093/neuros/nyz425.
Roux, A., Vikkula, M., & Pallud, J. (2020). Letter: Is Developmental Venous Anomaly an Imaging Biomarker of PIK3CA Mutated Gliomas?. Neurosurgery, 86(1), E93. https://doi.org/10.1093/neuros/nyz425
Roux, Alexandre, et al. "Letter: Is Developmental Venous Anomaly an Imaging Biomarker of PIK3CA Mutated Gliomas?." Neurosurgery vol. 86,1 (2020): E93. doi: https://doi.org/10.1093/neuros/nyz425
Roux A, Vikkula M, Pallud J. Letter: Is Developmental Venous Anomaly an Imaging Biomarker of PIK3CA Mutated Gliomas?. Neurosurgery. 2020 Jan 01;86(1):E93. doi: 10.1093/neuros/nyz425. PMID: 31742343.
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Hypotrichosis-lymphedema-telangiectasia syndrome: Report of ileal atresia associated with a SOX18 de novo pathogenic variant and review of the phenotypic spectrum.

American journal of medical genetics. Part A

Coulie R, Niyazov DM, Gambello MJ, Fastré E, Brouillard P, Vikkula M.
PMID: 33851505
Am J Med Genet A. 2021 Jul;185(7):2153-2159. doi: 10.1002/ajmg.a.62205. Epub 2021 Apr 14.

Hypotrichosis-lymphedema-telangiectasia syndrome (HLTS) is a rare condition caused by pathogenic variants in the SOX18 gene. SOX18 plays a key role in angio- and lymphangiogenesis due to its expression in venous endothelial cells from which the lymphatic system develops. It...

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Coulie R, Niyazov DM, Gambello MJ, et al. Hypotrichosis-lymphedema-telangiectasia syndrome: Report of ileal atresia associated with a SOX18 de novo pathogenic variant and review of the phenotypic spectrum. Am J Med Genet A. 2021;185(7):2153-2159doi: 10.1002/ajmg.a.62205.
Coulie, R., Niyazov, D. M., Gambello, M. J., Fastré, E., Brouillard, P., & Vikkula, M. (2021). Hypotrichosis-lymphedema-telangiectasia syndrome: Report of ileal atresia associated with a SOX18 de novo pathogenic variant and review of the phenotypic spectrum. American journal of medical genetics. Part A, 185(7), 2153-2159. https://doi.org/10.1002/ajmg.a.62205
Coulie, Richard, et al. "Hypotrichosis-lymphedema-telangiectasia syndrome: Report of ileal atresia associated with a SOX18 de novo pathogenic variant and review of the phenotypic spectrum." American journal of medical genetics. Part A vol. 185,7 (2021): 2153-2159. doi: https://doi.org/10.1002/ajmg.a.62205
Coulie R, Niyazov DM, Gambello MJ, Fastré E, Brouillard P, Vikkula M. Hypotrichosis-lymphedema-telangiectasia syndrome: Report of ileal atresia associated with a SOX18 de novo pathogenic variant and review of the phenotypic spectrum. Am J Med Genet A. 2021 Jul;185(7):2153-2159. doi: 10.1002/ajmg.a.62205. Epub 2021 Apr 14. PMID: 33851505.
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Comprehensive Overview of Vaccination during Pregnancy in Europe.

Journal of personalized medicine

Simionescu AA, Streinu-Cercel A, Popescu FD, Stanescu AMA, Vieru M, Danciu BM, Miron VD, Săndulescu O.
PMID: 34834548
J Pers Med. 2021 Nov 13;11(11). doi: 10.3390/jpm11111196.

Vaccinations during pregnancy can protect the mother from several infections, thus blocking vertical transmission. Furthermore, through passive antibody transfer, the newborn can be protected against some infections in the first months of life until their own vaccination regimen is...

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Simionescu AA, Streinu-Cercel A, Popescu FD, et al. Comprehensive Overview of Vaccination during Pregnancy in Europe. J Pers Med. 2021;11(11)doi: 10.3390/jpm11111196.
Simionescu, A. A., Streinu-Cercel, A., Popescu, F. D., Stanescu, A. M. A., Vieru, M., Danciu, B. M., Miron, V. D., & Săndulescu, O. (2021). Comprehensive Overview of Vaccination during Pregnancy in Europe. Journal of personalized medicine, 11(11), . https://doi.org/10.3390/jpm11111196
Simionescu, Anca Angela, et al. "Comprehensive Overview of Vaccination during Pregnancy in Europe." Journal of personalized medicine vol. 11,11 (2021). doi: https://doi.org/10.3390/jpm11111196
Simionescu AA, Streinu-Cercel A, Popescu FD, Stanescu AMA, Vieru M, Danciu BM, Miron VD, Săndulescu O. Comprehensive Overview of Vaccination during Pregnancy in Europe. J Pers Med. 2021 Nov 13;11(11). doi: 10.3390/jpm11111196. PMID: 34834548; PMCID: PMC8623700.
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We Do Not Talk to Patients About Their Prognosis, But Is Any of This Surprising?.

Journal of cardiac failure

Straw S, McGinlay M, Gierula J, Witte KK.
PMID: 34324925
J Card Fail. 2021 Dec;27(12):1479-1480. doi: 10.1016/j.cardfail.2021.06.024. Epub 2021 Jul 26.

No abstract available.

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Straw S, McGinlay M, Gierula J, et al. We Do Not Talk to Patients About Their Prognosis, But Is Any of This Surprising?. J Card Fail. 2021;27(12):1479-1480doi: 10.1016/j.cardfail.2021.06.024.
Straw, S., McGinlay, M., Gierula, J., & Witte, K. K. (2021). We Do Not Talk to Patients About Their Prognosis, But Is Any of This Surprising?. Journal of cardiac failure, 27(12), 1479-1480. https://doi.org/10.1016/j.cardfail.2021.06.024
Straw, Sam, et al. "We Do Not Talk to Patients About Their Prognosis, But Is Any of This Surprising?." Journal of cardiac failure vol. 27,12 (2021): 1479-1480. doi: https://doi.org/10.1016/j.cardfail.2021.06.024
Straw S, McGinlay M, Gierula J, Witte KK. We Do Not Talk to Patients About Their Prognosis, But Is Any of This Surprising?. J Card Fail. 2021 Dec;27(12):1479-1480. doi: 10.1016/j.cardfail.2021.06.024. Epub 2021 Jul 26. PMID: 34324925.
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Chronic Presence of Oligomeric Aβ Differentially Modulates Spine Parameters in the Hippocampus and Cortex of Mice With Low APP Transgene Expression.

Frontiers in synaptic neuroscience

Hrynchak MV, Rierola M, Golovyashkina N, Penazzi L, Pump WC, David B, Sündermann F, Brandt R, Bakota L.
PMID: 32390822
Front Synaptic Neurosci. 2020 Apr 24;12:16. doi: 10.3389/fnsyn.2020.00016. eCollection 2020.

Alzheimer's disease is regarded as a synaptopathy with a long presymptomatic phase. Soluble, oligomeric amyloid-β (Aβ) is thought to play a causative role in this disease, which eventually leads to cognitive decline. However, most animal studies have employed mice...

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Hrynchak MV, Rierola M, Golovyashkina N, et al. Chronic Presence of Oligomeric Aβ Differentially Modulates Spine Parameters in the Hippocampus and Cortex of Mice With Low APP Transgene Expression. Front Synaptic Neurosci. 2020;12:16doi: 10.3389/fnsyn.2020.00016.
Hrynchak, M. V., Rierola, M., Golovyashkina, N., Penazzi, L., Pump, W. C., David, B., Sündermann, F., Brandt, R., & Bakota, L. (2020). Chronic Presence of Oligomeric Aβ Differentially Modulates Spine Parameters in the Hippocampus and Cortex of Mice With Low APP Transgene Expression. Frontiers in synaptic neuroscience, 1216. https://doi.org/10.3389/fnsyn.2020.00016
Hrynchak, Mariya V, et al. "Chronic Presence of Oligomeric Aβ Differentially Modulates Spine Parameters in the Hippocampus and Cortex of Mice With Low APP Transgene Expression." Frontiers in synaptic neuroscience vol. 12 (2020): 16. doi: https://doi.org/10.3389/fnsyn.2020.00016
Hrynchak MV, Rierola M, Golovyashkina N, Penazzi L, Pump WC, David B, Sündermann F, Brandt R, Bakota L. Chronic Presence of Oligomeric Aβ Differentially Modulates Spine Parameters in the Hippocampus and Cortex of Mice With Low APP Transgene Expression. Front Synaptic Neurosci. 2020 Apr 24;12:16. doi: 10.3389/fnsyn.2020.00016. eCollection 2020. PMID: 32390822; PMCID: PMC7194154.
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Personalised reprogramming to prevent progressive pacemaker-related left ventricular dysfunction: A phase II randomised, controlled clinical trial.

PloS one

Paton MF, Gierula J, Lowry JE, Cairns DA, Bose Rosling K, Cole CA, McGinlay M, Straw S, Byrom R, Cubbon RM, Kearney MT, Witte KK.
PMID: 34898655
PLoS One. 2021 Dec 13;16(12):e0259450. doi: 10.1371/journal.pone.0259450. eCollection 2021.

BACKGROUND: Pacemakers are widely utilised to treat bradycardia, but right ventricular (RV) pacing is associated with heightened risk of left ventricular (LV) systolic dysfunction and heart failure. We aimed to compare personalised pacemaker reprogramming to avoid RV pacing with...

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Paton MF, Gierula J, Lowry JE, et al. Personalised reprogramming to prevent progressive pacemaker-related left ventricular dysfunction: A phase II randomised, controlled clinical trial. PLoS One. 2021;16(12):e0259450doi: 10.1371/journal.pone.0259450.
Paton, M. F., Gierula, J., Lowry, J. E., Cairns, D. A., Bose Rosling, K., Cole, C. A., McGinlay, M., Straw, S., Byrom, R., Cubbon, R. M., Kearney, M. T., & Witte, K. K. (2021). Personalised reprogramming to prevent progressive pacemaker-related left ventricular dysfunction: A phase II randomised, controlled clinical trial. PloS one, 16(12), e0259450. https://doi.org/10.1371/journal.pone.0259450
Paton, Maria F, et al. "Personalised reprogramming to prevent progressive pacemaker-related left ventricular dysfunction: A phase II randomised, controlled clinical trial." PloS one vol. 16,12 (2021): e0259450. doi: https://doi.org/10.1371/journal.pone.0259450
Paton MF, Gierula J, Lowry JE, Cairns DA, Bose Rosling K, Cole CA, McGinlay M, Straw S, Byrom R, Cubbon RM, Kearney MT, Witte KK. Personalised reprogramming to prevent progressive pacemaker-related left ventricular dysfunction: A phase II randomised, controlled clinical trial. PLoS One. 2021 Dec 13;16(12):e0259450. doi: 10.1371/journal.pone.0259450. eCollection 2021. PMID: 34898655.
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Patient Factors and Their Association with Nonmelanoma Skin Cancer Morbidity and the Performance of Self-skin Exams: A Cross-Sectional Study.

The Journal of clinical and aesthetic dermatology

Amber KT, Bloom R, Abyaneh MY, Falto-Aizpurua LA, Viera M, Zaiac MN, Nouri K, Hu S.
PMID: 27878058
J Clin Aesthet Dermatol. 2016 Sep;9(9):16-22. Epub 2016 Sep 01.

No abstract available.

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Amber KT, Bloom R, Abyaneh MY, et al. Patient Factors and Their Association with Nonmelanoma Skin Cancer Morbidity and the Performance of Self-skin Exams: A Cross-Sectional Study. J Clin Aesthet Dermatol. 2016;9(9):16-22
Amber, K. T., Bloom, R., Abyaneh, M. Y., Falto-Aizpurua, L. A., Viera, M., Zaiac, M. N., Nouri, K., & Hu, S. (2016). Patient Factors and Their Association with Nonmelanoma Skin Cancer Morbidity and the Performance of Self-skin Exams: A Cross-Sectional Study. The Journal of clinical and aesthetic dermatology, 9(9), 16-22.
Amber, Kyle T, et al. "Patient Factors and Their Association with Nonmelanoma Skin Cancer Morbidity and the Performance of Self-skin Exams: A Cross-Sectional Study." The Journal of clinical and aesthetic dermatology vol. 9,9 (2016): 16-22.
Amber KT, Bloom R, Abyaneh MY, Falto-Aizpurua LA, Viera M, Zaiac MN, Nouri K, Hu S. Patient Factors and Their Association with Nonmelanoma Skin Cancer Morbidity and the Performance of Self-skin Exams: A Cross-Sectional Study. J Clin Aesthet Dermatol. 2016 Sep;9(9):16-22. Epub 2016 Sep 01. PMID: 27878058; PMCID: PMC5110325.
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Showing 1 to 12 of 71 entries