Advanced Search
Display options
Filter resources
Text Availability
Article type
Publication date
Species
Language
Sex
Age
Showing 1 to 12 of 30 entries
Sorted by: Best Match Show Resources per page
Neurovascular and Immuno-Imaging: From Mechanisms to Therapies. Proceedings of the Inaugural Symposium.

Frontiers in neuroscience

Akassoglou K, Agalliu D, Chang CJ, Davalos D, Grutzendler J, Hillman EM, Khakh BS, Kleinfeld D, McGavern DB, Nelson SJ, Zlokovic BV.
PMID: 26941593
Front Neurosci. 2016 Feb 22;10:46. doi: 10.3389/fnins.2016.00046. eCollection 2016.

Breakthrough advances in intravital imaging have launched a new era for the study of dynamic interactions at the neurovascular interface in health and disease. The first Neurovascular and Immuno-Imaging Symposium was held at the Gladstone Institutes, University of California,...

Cite
Akassoglou K, Agalliu D, Chang CJ, et al. Neurovascular and Immuno-Imaging: From Mechanisms to Therapies. Proceedings of the Inaugural Symposium. Front Neurosci. 2016;10:46doi: 10.3389/fnins.2016.00046.
Akassoglou, K., Agalliu, D., Chang, C. J., Davalos, D., Grutzendler, J., Hillman, E. M., Khakh, B. S., Kleinfeld, D., McGavern, D. B., Nelson, S. J., & Zlokovic, B. V. (2016). Neurovascular and Immuno-Imaging: From Mechanisms to Therapies. Proceedings of the Inaugural Symposium. Frontiers in neuroscience, 1046. https://doi.org/10.3389/fnins.2016.00046
Akassoglou, Katerina, et al. "Neurovascular and Immuno-Imaging: From Mechanisms to Therapies. Proceedings of the Inaugural Symposium." Frontiers in neuroscience vol. 10 (2016): 46. doi: https://doi.org/10.3389/fnins.2016.00046
Akassoglou K, Agalliu D, Chang CJ, Davalos D, Grutzendler J, Hillman EM, Khakh BS, Kleinfeld D, McGavern DB, Nelson SJ, Zlokovic BV. Neurovascular and Immuno-Imaging: From Mechanisms to Therapies. Proceedings of the Inaugural Symposium. Front Neurosci. 2016 Feb 22;10:46. doi: 10.3389/fnins.2016.00046. eCollection 2016. PMID: 26941593; PMCID: PMC4761864.
Copy Download .nbib Format: NLM
Clearance of amyloid beta-peptide from brain: transport or metabolism?.

Nature medicine

Zlokovic BV, Yamada S, Holtzman D, Ghiso J, Frangione B.
PMID: 10888893
Nat Med. 2000 Jul;6(7):718. doi: 10.1038/77397.

No abstract available.

Cite
Zlokovic BV, Yamada S, Holtzman D, et al. Clearance of amyloid beta-peptide from brain: transport or metabolism?. Nat Med. 2000;6(7):718doi: 10.1038/77397.
Zlokovic, B. V., Yamada, S., Holtzman, D., Ghiso, J., & Frangione, B. (2000). Clearance of amyloid beta-peptide from brain: transport or metabolism?. Nature medicine, 6(7), 718. https://doi.org/10.1038/77397
Zlokovic, et al. "Clearance of amyloid beta-peptide from brain: transport or metabolism?." Nature medicine vol. 6,7 (2000): 718. doi: https://doi.org/10.1038/77397
Zlokovic BV, Yamada S, Holtzman D, Ghiso J, Frangione B. Clearance of amyloid beta-peptide from brain: transport or metabolism?. Nat Med. 2000 Jul;6(7):718. doi: 10.1038/77397. PMID: 10888893.
Copy Download .nbib Format: NLM
Lack of Smad or Notch leads to a fatal game of brain pericyte hopscotch.

Developmental cell

Winkler EA, Bell RD, Zlokovic BV.
PMID: 21397835
Dev Cell. 2011 Mar 15;20(3):279-80. doi: 10.1016/j.devcel.2011.03.002.

Pericytes surround and stabilize the vast capillary network of the central nervous system. In this issue of Developmental Cell, Li et al. (2011) show that Smad4 and Notch signaling together regulate endothelial expression of an adhesion factor, N-cadherin, which...

Cite
Winkler EA, Bell RD, Zlokovic BV. Lack of Smad or Notch leads to a fatal game of brain pericyte hopscotch. Dev Cell. 2011;20(3):279-80doi: 10.1016/j.devcel.2011.03.002.
Winkler, E. A., Bell, R. D., & Zlokovic, B. V. (2011). Lack of Smad or Notch leads to a fatal game of brain pericyte hopscotch. Developmental cell, 20(3), 279-80. https://doi.org/10.1016/j.devcel.2011.03.002
Winkler, Ethan A, et al. "Lack of Smad or Notch leads to a fatal game of brain pericyte hopscotch." Developmental cell vol. 20,3 (2011): 279-80. doi: https://doi.org/10.1016/j.devcel.2011.03.002
Winkler EA, Bell RD, Zlokovic BV. Lack of Smad or Notch leads to a fatal game of brain pericyte hopscotch. Dev Cell. 2011 Mar 15;20(3):279-80. doi: 10.1016/j.devcel.2011.03.002. PMID: 21397835.
Copy Download .nbib Format: NLM
Pericyte-specific expression of PDGF beta receptor in mouse models with normal and deficient PDGF beta receptor signaling.

Molecular neurodegeneration

Winkler EA, Bell RD, Zlokovic BV.
PMID: 20738866
Mol Neurodegener. 2010 Aug 25;5:32. doi: 10.1186/1750-1326-5-32.

BACKGROUND: Pericytes are integral members of the neurovascular unit. Using mouse models lacking endothelial-secreted platelet derived growth factor-B (PDGF-B) or platelet derived growth factor receptor beta (PDGFRβ) on pericytes, it has been demonstrated that PDGF-B/PDGFRβ interactions mediate pericyte recruitment...

Cite
Winkler EA, Bell RD, Zlokovic BV. Pericyte-specific expression of PDGF beta receptor in mouse models with normal and deficient PDGF beta receptor signaling. Mol Neurodegener. 2010;5:32doi: 10.1186/1750-1326-5-32.
Winkler, E. A., Bell, R. D., & Zlokovic, B. V. (2010). Pericyte-specific expression of PDGF beta receptor in mouse models with normal and deficient PDGF beta receptor signaling. Molecular neurodegeneration, 532. https://doi.org/10.1186/1750-1326-5-32
Winkler, Ethan A, et al. "Pericyte-specific expression of PDGF beta receptor in mouse models with normal and deficient PDGF beta receptor signaling." Molecular neurodegeneration vol. 5 (2010): 32. doi: https://doi.org/10.1186/1750-1326-5-32
Winkler EA, Bell RD, Zlokovic BV. Pericyte-specific expression of PDGF beta receptor in mouse models with normal and deficient PDGF beta receptor signaling. Mol Neurodegener. 2010 Aug 25;5:32. doi: 10.1186/1750-1326-5-32. PMID: 20738866; PMCID: PMC2936891.
Copy Download .nbib Format: NLM
Comparison Between Blood-Brain Barrier Water Exchange Rate and Permeability to Gadolinium-Based Contrast Agent in an Elderly Cohort.

Frontiers in neuroscience

Shao X, Jann K, Ma SJ, Yan L, Montagne A, Ringman JM, Zlokovic BV, Wang DJJ.
PMID: 33328848
Front Neurosci. 2020 Nov 30;14:571480. doi: 10.3389/fnins.2020.571480. eCollection 2020.

No abstract available.

Cite
Shao X, Jann K, Ma SJ, et al. Comparison Between Blood-Brain Barrier Water Exchange Rate and Permeability to Gadolinium-Based Contrast Agent in an Elderly Cohort. Front Neurosci. 2020;14:571480doi: 10.3389/fnins.2020.571480.
Shao, X., Jann, K., Ma, S. J., Yan, L., Montagne, A., Ringman, J. M., Zlokovic, B. V., & Wang, D. J. J. (2020). Comparison Between Blood-Brain Barrier Water Exchange Rate and Permeability to Gadolinium-Based Contrast Agent in an Elderly Cohort. Frontiers in neuroscience, 14571480. https://doi.org/10.3389/fnins.2020.571480
Shao, Xingfeng, et al. "Comparison Between Blood-Brain Barrier Water Exchange Rate and Permeability to Gadolinium-Based Contrast Agent in an Elderly Cohort." Frontiers in neuroscience vol. 14 (2020): 571480. doi: https://doi.org/10.3389/fnins.2020.571480
Shao X, Jann K, Ma SJ, Yan L, Montagne A, Ringman JM, Zlokovic BV, Wang DJJ. Comparison Between Blood-Brain Barrier Water Exchange Rate and Permeability to Gadolinium-Based Contrast Agent in an Elderly Cohort. Front Neurosci. 2020 Nov 30;14:571480. doi: 10.3389/fnins.2020.571480. eCollection 2020. PMID: 33328848; PMCID: PMC7733970.
Copy Download .nbib Format: NLM
Prion Protein Antagonists Rescue Alzheimer's Amyloid-β-Related Cognitive Deficits.

Trends in molecular medicine

Sagare AP, Sweeney MD, Nelson AR, Zhao Z, Zlokovic BV.
PMID: 30661727
Trends Mol Med. 2019 Feb;25(2):74-76. doi: 10.1016/j.molmed.2019.01.001. Epub 2019 Jan 17.

Recent studies revealed that cellular prion protein on neurons bind Alzheimer's amyloid-β oligomers, causing neurotoxic effects. A new article in Cell Reports by Gunther and colleagues (Cell Rep. 2019; 26:145-158) shows that an orally administered cellular prion protein antagonist...

Cite
Sagare AP, Sweeney MD, Nelson AR, et al. Prion Protein Antagonists Rescue Alzheimer's Amyloid-β-Related Cognitive Deficits. Trends Mol Med. 2019;25(2):74-76doi: 10.1016/j.molmed.2019.01.001.
Sagare, A. P., Sweeney, M. D., Nelson, A. R., Zhao, Z., & Zlokovic, B. V. (2019). Prion Protein Antagonists Rescue Alzheimer's Amyloid-β-Related Cognitive Deficits. Trends in molecular medicine, 25(2), 74-76. https://doi.org/10.1016/j.molmed.2019.01.001
Sagare, Abhay P, et al. "Prion Protein Antagonists Rescue Alzheimer's Amyloid-β-Related Cognitive Deficits." Trends in molecular medicine vol. 25,2 (2019): 74-76. doi: https://doi.org/10.1016/j.molmed.2019.01.001
Sagare AP, Sweeney MD, Nelson AR, Zhao Z, Zlokovic BV. Prion Protein Antagonists Rescue Alzheimer's Amyloid-β-Related Cognitive Deficits. Trends Mol Med. 2019 Feb;25(2):74-76. doi: 10.1016/j.molmed.2019.01.001. Epub 2019 Jan 17. PMID: 30661727; PMCID: PMC6377285.
Copy Download .nbib Format: NLM
Channelrhodopsin Excitation Contracts Brain Pericytes and Reduces Blood Flow in the Aging Mouse Brain .

Frontiers in aging neuroscience

Nelson AR, Sagare MA, Wang Y, Kisler K, Zhao Z, Zlokovic BV.
PMID: 32410982
Front Aging Neurosci. 2020 Apr 29;12:108. doi: 10.3389/fnagi.2020.00108. eCollection 2020.

Brains depend on blood flow for the delivery of oxygen and nutrients essential for proper neuronal and synaptic functioning. French physiologist Rouget was the first to describe pericytes in 1873 as regularly arranged longitudinal amoeboid cells on capillaries that...

Cite
Nelson AR, Sagare MA, Wang Y, et al. Channelrhodopsin Excitation Contracts Brain Pericytes and Reduces Blood Flow in the Aging Mouse Brain . Front Aging Neurosci. 2020;12:108doi: 10.3389/fnagi.2020.00108.
Nelson, A. R., Sagare, M. A., Wang, Y., Kisler, K., Zhao, Z., & Zlokovic, B. V. (2020). Channelrhodopsin Excitation Contracts Brain Pericytes and Reduces Blood Flow in the Aging Mouse Brain . Frontiers in aging neuroscience, 12108. https://doi.org/10.3389/fnagi.2020.00108
Nelson, Amy R, et al. "Channelrhodopsin Excitation Contracts Brain Pericytes and Reduces Blood Flow in the Aging Mouse Brain ." Frontiers in aging neuroscience vol. 12 (2020): 108. doi: https://doi.org/10.3389/fnagi.2020.00108
Nelson AR, Sagare MA, Wang Y, Kisler K, Zhao Z, Zlokovic BV. Channelrhodopsin Excitation Contracts Brain Pericytes and Reduces Blood Flow in the Aging Mouse Brain . Front Aging Neurosci. 2020 Apr 29;12:108. doi: 10.3389/fnagi.2020.00108. eCollection 2020. PMID: 32410982; PMCID: PMC7201096.
Copy Download .nbib Format: NLM
Protection of ischemic white matter and oligodendrocytes in mice by 3K3A-activated protein C.

The Journal of experimental medicine

Huuskonen MT, Wang Y, Nikolakopoulou AM, Montagne A, Dai Z, Lazic D, Sagare AP, Zhao Z, Fernandez JA, Griffin JH, Zlokovic BV.
PMID: 34846535
J Exp Med. 2022 Jan 03;219(1). doi: 10.1084/jem.20211372. Epub 2021 Nov 30.

Subcortical white matter (WM) stroke accounts for 25% of all strokes and is the second leading cause of dementia. Despite such clinical importance, we still do not have an effective treatment for ischemic WM stroke, and the mechanisms of...

Cite
Huuskonen MT, Wang Y, Nikolakopoulou AM, et al. Protection of ischemic white matter and oligodendrocytes in mice by 3K3A-activated protein C. J Exp Med. 2021;219(1)doi: 10.1084/jem.20211372.
Huuskonen, M. T., Wang, Y., Nikolakopoulou, A. M., Montagne, A., Dai, Z., Lazic, D., Sagare, A. P., Zhao, Z., Fernandez, J. A., Griffin, J. H., & Zlokovic, B. V. (2022). Protection of ischemic white matter and oligodendrocytes in mice by 3K3A-activated protein C. The Journal of experimental medicine, 219(1), . https://doi.org/10.1084/jem.20211372
Huuskonen, Mikko T, et al. "Protection of ischemic white matter and oligodendrocytes in mice by 3K3A-activated protein C." The Journal of experimental medicine vol. 219,1 (2022). doi: https://doi.org/10.1084/jem.20211372
Huuskonen MT, Wang Y, Nikolakopoulou AM, Montagne A, Dai Z, Lazic D, Sagare AP, Zhao Z, Fernandez JA, Griffin JH, Zlokovic BV. Protection of ischemic white matter and oligodendrocytes in mice by 3K3A-activated protein C. J Exp Med. 2022 Jan 03;219(1). doi: 10.1084/jem.20211372. Epub 2021 Nov 30. PMID: 34846535; PMCID: PMC8635278.
Copy Download .nbib Format: NLM
Air Pollution Particulate Matter Exposure and Chronic Cerebral Hypoperfusion and Measures of White Matter Injury in a Murine Model.

Environmental health perspectives

Liu Q, Shkirkova K, Lamorie-Foote K, Connor M, Patel A, Babadjouni R, Huuskonen M, Montagne A, Baertsch H, Zhang H, Chen JC, Mack WJ, Walcott BP, Zlokovic BV, Sioutas C, Morgan TE, Finch CE, Mack WJ.
PMID: 34424052
Environ Health Perspect. 2021 Aug;129(8):87006. doi: 10.1289/EHP8792. Epub 2021 Aug 23.

BACKGROUND: Exposure to ambient air pollution particulate matter (PM) is associated with increased risk of dementia and accelerated cognitive loss. Vascular contributions to cognitive impairment are well recognized. Chronic cerebral hypoperfusion (CCH) promotes neuroinflammation and blood-brain barrier weakening, which...

Cite
Liu Q, Shkirkova K, Lamorie-Foote K, et al. Air Pollution Particulate Matter Exposure and Chronic Cerebral Hypoperfusion and Measures of White Matter Injury in a Murine Model. Environ Health Perspect. 2021;129(8):87006doi: 10.1289/EHP8792.
Liu, Q., Shkirkova, K., Lamorie-Foote, K., Connor, M., Patel, A., Babadjouni, R., Huuskonen, M., Montagne, A., Baertsch, H., Zhang, H., Chen, J. C., Mack, W. J., Walcott, B. P., Zlokovic, B. V., Sioutas, C., Morgan, T. E., Finch, C. E., & Mack, W. J. (2021). Air Pollution Particulate Matter Exposure and Chronic Cerebral Hypoperfusion and Measures of White Matter Injury in a Murine Model. Environmental health perspectives, 129(8), 87006. https://doi.org/10.1289/EHP8792
Liu, Qinghai, et al. "Air Pollution Particulate Matter Exposure and Chronic Cerebral Hypoperfusion and Measures of White Matter Injury in a Murine Model." Environmental health perspectives vol. 129,8 (2021): 87006. doi: https://doi.org/10.1289/EHP8792
Liu Q, Shkirkova K, Lamorie-Foote K, Connor M, Patel A, Babadjouni R, Huuskonen M, Montagne A, Baertsch H, Zhang H, Chen JC, Mack WJ, Walcott BP, Zlokovic BV, Sioutas C, Morgan TE, Finch CE, Mack WJ. Air Pollution Particulate Matter Exposure and Chronic Cerebral Hypoperfusion and Measures of White Matter Injury in a Murine Model. Environ Health Perspect. 2021 Aug;129(8):87006. doi: 10.1289/EHP8792. Epub 2021 Aug 23. PMID: 34424052; PMCID: PMC8382048.
Copy Download .nbib Format: NLM
Protection of ischemic white matter and oligodendrocytes in mice by 3K3A-activated protein C.

The Journal of experimental medicine

Huuskonen MT, Wang Y, Nikolakopoulou AM, Montagne A, Dai Z, Lazic D, Sagare AP, Zhao Z, Fernandez JA, Griffin JH, Zlokovic BV.
PMID: 34846535
J Exp Med. 2022 Jan 03;219(1). doi: 10.1084/jem.20211372. Epub 2021 Nov 30.

Subcortical white matter (WM) stroke accounts for 25% of all strokes and is the second leading cause of dementia. Despite such clinical importance, we still do not have an effective treatment for ischemic WM stroke, and the mechanisms of...

Cite
Huuskonen MT, Wang Y, Nikolakopoulou AM, et al. Protection of ischemic white matter and oligodendrocytes in mice by 3K3A-activated protein C. J Exp Med. 2021;219(1)doi: 10.1084/jem.20211372.
Huuskonen, M. T., Wang, Y., Nikolakopoulou, A. M., Montagne, A., Dai, Z., Lazic, D., Sagare, A. P., Zhao, Z., Fernandez, J. A., Griffin, J. H., & Zlokovic, B. V. (2022). Protection of ischemic white matter and oligodendrocytes in mice by 3K3A-activated protein C. The Journal of experimental medicine, 219(1), . https://doi.org/10.1084/jem.20211372
Huuskonen, Mikko T, et al. "Protection of ischemic white matter and oligodendrocytes in mice by 3K3A-activated protein C." The Journal of experimental medicine vol. 219,1 (2022). doi: https://doi.org/10.1084/jem.20211372
Huuskonen MT, Wang Y, Nikolakopoulou AM, Montagne A, Dai Z, Lazic D, Sagare AP, Zhao Z, Fernandez JA, Griffin JH, Zlokovic BV. Protection of ischemic white matter and oligodendrocytes in mice by 3K3A-activated protein C. J Exp Med. 2022 Jan 03;219(1). doi: 10.1084/jem.20211372. Epub 2021 Nov 30. PMID: 34846535; PMCID: PMC8635278.
Copy Download .nbib Format: NLM
Clearance of interstitial fluid (ISF) and CSF (CLIC) group-part of Vascular Professional Interest Area (PIA): Cerebrovascular disease and the failure of elimination of Amyloid-β from the brain and retina with age and Alzheimer's disease-Opportunities for Therapy.

Alzheimer's & dementia (Amsterdam, Netherlands)

Carare RO, Aldea R, Agarwal N, Bacskai BJ, Bechman I, Boche D, Bu G, Bulters D, Clemens A, Counts SE, de Leon M, Eide PK, Fossati S, Greenberg SM, Hamel E, Hawkes CA, Koronyo-Hamaoui M, Hainsworth AH, Holtzman D, Ihara M, Jefferson A, Kalaria RN, Kipps CM, Kanninen KM, Leinonen V, McLaurin J, Miners S, Malm T, Nicoll JAR, Piazza F, Paul G, Rich SM, Saito S, Shih A, Scholtzova H, Snyder H, Snyder P, Thormodsson FR, van Veluw SJ, Weller RO, Werring DJ, Wilcock D, Wilson MR, Zlokovic BV, Verma A.
PMID: 32775596
Alzheimers Dement (Amst). 2020 Aug 03;12(1):e12053. doi: 10.1002/dad2.12053. eCollection 2020.

Two of the key functions of arteries in the brain are (1) the well-recognized supply of blood via the vascular lumen and (2) the emerging role for the arterial walls as routes for the elimination of interstitial fluid (ISF)...

Cite
Carare RO, Aldea R, Agarwal N, et al. Clearance of interstitial fluid (ISF) and CSF (CLIC) group-part of Vascular Professional Interest Area (PIA): Cerebrovascular disease and the failure of elimination of Amyloid-β from the brain and retina with age and Alzheimer's disease-Opportunities for Therapy. Alzheimers Dement (Amst). 2020;12(1):e12053doi: 10.1002/dad2.12053.
Carare, R. O., Aldea, R., Agarwal, N., Bacskai, B. J., Bechman, I., Boche, D., Bu, G., Bulters, D., Clemens, A., Counts, S. E., de Leon, M., Eide, P. K., Fossati, S., Greenberg, S. M., Hamel, E., Hawkes, C. A., Koronyo-Hamaoui, M., Hainsworth, A. H., Holtzman, D., Ihara, M., Jefferson, A., Kalaria, R. N., Kipps, C. M., Kanninen, K. M., Leinonen, V., McLaurin, J., Miners, S., Malm, T., Nicoll, J. A. R., Piazza, F., Paul, G., Rich, S. M., Saito, S., Shih, A., Scholtzova, H., Snyder, H., Snyder, P., Thormodsson, F. R., van Veluw, S. J., Weller, R. O., Werring, D. J., Wilcock, D., Wilson, M. R., Zlokovic, B. V., & Verma, A. (2020). Clearance of interstitial fluid (ISF) and CSF (CLIC) group-part of Vascular Professional Interest Area (PIA): Cerebrovascular disease and the failure of elimination of Amyloid-β from the brain and retina with age and Alzheimer's disease-Opportunities for Therapy. Alzheimer's & dementia (Amsterdam, Netherlands), 12(1), e12053. https://doi.org/10.1002/dad2.12053
Carare, Roxana O, et al. "Clearance of interstitial fluid (ISF) and CSF (CLIC) group-part of Vascular Professional Interest Area (PIA): Cerebrovascular disease and the failure of elimination of Amyloid-β from the brain and retina with age and Alzheimer's disease-Opportunities for Therapy." Alzheimer's & dementia (Amsterdam, Netherlands) vol. 12,1 (2020): e12053. doi: https://doi.org/10.1002/dad2.12053
Carare RO, Aldea R, Agarwal N, Bacskai BJ, Bechman I, Boche D, Bu G, Bulters D, Clemens A, Counts SE, de Leon M, Eide PK, Fossati S, Greenberg SM, Hamel E, Hawkes CA, Koronyo-Hamaoui M, Hainsworth AH, Holtzman D, Ihara M, Jefferson A, Kalaria RN, Kipps CM, Kanninen KM, Leinonen V, McLaurin J, Miners S, Malm T, Nicoll JAR, Piazza F, Paul G, Rich SM, Saito S, Shih A, Scholtzova H, Snyder H, Snyder P, Thormodsson FR, van Veluw SJ, Weller RO, Werring DJ, Wilcock D, Wilson MR, Zlokovic BV, Verma A. Clearance of interstitial fluid (ISF) and CSF (CLIC) group-part of Vascular Professional Interest Area (PIA): Cerebrovascular disease and the failure of elimination of Amyloid-β from the brain and retina with age and Alzheimer's disease-Opportunities for Therapy. Alzheimers Dement (Amst). 2020 Aug 03;12(1):e12053. doi: 10.1002/dad2.12053. eCollection 2020. PMID: 32775596; PMCID: PMC7396859.
Copy Download .nbib Format: NLM
Air Pollution Particulate Matter Exposure and Chronic Cerebral Hypoperfusion and Measures of White Matter Injury in a Murine Model.

Environmental health perspectives

Liu Q, Shkirkova K, Lamorie-Foote K, Connor M, Patel A, Babadjouni R, Huuskonen M, Montagne A, Baertsch H, Zhang H, Chen JC, Mack WJ, Walcott BP, Zlokovic BV, Sioutas C, Morgan TE, Finch CE, Mack WJ.
PMID: 34424052
Environ Health Perspect. 2021 Aug;129(8):87006. doi: 10.1289/EHP8792. Epub 2021 Aug 23.

BACKGROUND: Exposure to ambient air pollution particulate matter (PM) is associated with increased risk of dementia and accelerated cognitive loss. Vascular contributions to cognitive impairment are well recognized. Chronic cerebral hypoperfusion (CCH) promotes neuroinflammation and blood-brain barrier weakening, which...

Cite
Liu Q, Shkirkova K, Lamorie-Foote K, et al. Air Pollution Particulate Matter Exposure and Chronic Cerebral Hypoperfusion and Measures of White Matter Injury in a Murine Model. Environ Health Perspect. 2021;129(8):87006doi: 10.1289/EHP8792.
Liu, Q., Shkirkova, K., Lamorie-Foote, K., Connor, M., Patel, A., Babadjouni, R., Huuskonen, M., Montagne, A., Baertsch, H., Zhang, H., Chen, J. C., Mack, W. J., Walcott, B. P., Zlokovic, B. V., Sioutas, C., Morgan, T. E., Finch, C. E., & Mack, W. J. (2021). Air Pollution Particulate Matter Exposure and Chronic Cerebral Hypoperfusion and Measures of White Matter Injury in a Murine Model. Environmental health perspectives, 129(8), 87006. https://doi.org/10.1289/EHP8792
Liu, Qinghai, et al. "Air Pollution Particulate Matter Exposure and Chronic Cerebral Hypoperfusion and Measures of White Matter Injury in a Murine Model." Environmental health perspectives vol. 129,8 (2021): 87006. doi: https://doi.org/10.1289/EHP8792
Liu Q, Shkirkova K, Lamorie-Foote K, Connor M, Patel A, Babadjouni R, Huuskonen M, Montagne A, Baertsch H, Zhang H, Chen JC, Mack WJ, Walcott BP, Zlokovic BV, Sioutas C, Morgan TE, Finch CE, Mack WJ. Air Pollution Particulate Matter Exposure and Chronic Cerebral Hypoperfusion and Measures of White Matter Injury in a Murine Model. Environ Health Perspect. 2021 Aug;129(8):87006. doi: 10.1289/EHP8792. Epub 2021 Aug 23. PMID: 34424052; PMCID: PMC8382048.
Copy Download .nbib Format: NLM
Showing 1 to 12 of 30 entries