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Showing 1 to 9 of 9 entries
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Deficiency in outer dense fiber 1 is a marker and potential driver of idiopathic male infertility.

Molecular & cellular proteomics : MCP

Hetherington L, Schneider EK, Scott C, DeKretser D, Muller CH, Hondermarck H, Velkov T, Baker MA.
PMID: 28572402
Mol Cell Proteomics. 2017 Jun;16(6):1172. doi: 10.1074/mcp.A116.060343.

No abstract available.

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Hetherington L, Schneider EK, Scott C, et al. Deficiency in outer dense fiber 1 is a marker and potential driver of idiopathic male infertility. Mol Cell Proteomics. 2017;16(6):1172doi: 10.1074/mcp.A116.060343.
Hetherington, L., Schneider, E. K., Scott, C., DeKretser, D., Muller, C. H., Hondermarck, H., Velkov, T., & Baker, M. A. (2017). Deficiency in outer dense fiber 1 is a marker and potential driver of idiopathic male infertility. Molecular & cellular proteomics : MCP, 16(6), 1172. https://doi.org/10.1074/mcp.A116.060343
Hetherington, Louise, et al. "Deficiency in outer dense fiber 1 is a marker and potential driver of idiopathic male infertility." Molecular & cellular proteomics : MCP vol. 16,6 (2017): 1172. doi: https://doi.org/10.1074/mcp.A116.060343
Hetherington L, Schneider EK, Scott C, DeKretser D, Muller CH, Hondermarck H, Velkov T, Baker MA. Deficiency in outer dense fiber 1 is a marker and potential driver of idiopathic male infertility. Mol Cell Proteomics. 2017 Jun;16(6):1172. doi: 10.1074/mcp.A116.060343. PMID: 28572402; PMCID: PMC5461546.
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Male sexual and reproductive health among the Aboriginal and Torres Strait Islander population.

Rural and remote health

Adams M, de Kretser D, Holden C.
PMID: 15877505
Rural Remote Health. 2003 Jul-Sep;3(2):153. Epub 2003 Aug 21.

No abstract available.

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Adams M, de Kretser D, Holden C. Male sexual and reproductive health among the Aboriginal and Torres Strait Islander population. Rural Remote Health. 2003;3(2):153
Adams, M., de Kretser, D., & Holden, C. (2003). Male sexual and reproductive health among the Aboriginal and Torres Strait Islander population. Rural and remote health, 3(2), 153.
Adams, M, et al. "Male sexual and reproductive health among the Aboriginal and Torres Strait Islander population." Rural and remote health vol. 3,2 (2003): 153.
Adams M, de Kretser D, Holden C. Male sexual and reproductive health among the Aboriginal and Torres Strait Islander population. Rural Remote Health. 2003 Jul-Sep;3(2):153. Epub 2003 Aug 21. PMID: 15877505.
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Hormonal control of spermatogenesis.

Trends in endocrinology and metabolism: TEM

McLachlan RI, Wreford NG, Robertson DM, de Kretser DM.
PMID: 18406689
Trends Endocrinol Metab. 1995 Apr;6(3):95-101. doi: 10.1016/1043-2760(94)00215-p.

FSH and testosterone (T) secretion are essential for the successful completion of spermatogenesis. Because there are no receptors for FSH or testosterone on germ cells, there are intermediate steps in this action, the nature of which are unknown. However,...

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McLachlan RI, Wreford NG, Robertson DM, et al. Hormonal control of spermatogenesis. Trends Endocrinol Metab. 1995;6(3):95-101doi: 10.1016/1043-2760(94)00215-p.
McLachlan, R. I., Wreford, N. G., Robertson, D. M., & de Kretser, D. M. (1995). Hormonal control of spermatogenesis. Trends in endocrinology and metabolism: TEM, 6(3), 95-101. https://doi.org/10.1016/1043-2760(94)00215-p
McLachlan, R I, et al. "Hormonal control of spermatogenesis." Trends in endocrinology and metabolism: TEM vol. 6,3 (1995): 95-101. doi: https://doi.org/10.1016/1043-2760(94)00215-p
McLachlan RI, Wreford NG, Robertson DM, de Kretser DM. Hormonal control of spermatogenesis. Trends Endocrinol Metab. 1995 Apr;6(3):95-101. doi: 10.1016/1043-2760(94)00215-p. PMID: 18406689.
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The Role of Activin A and B and the Benefit of Follistatin Treatment in Renal Ischemia-Reperfusion Injury in Mice.

Transplantation direct

Fang DY, Lu B, Hayward S, de Kretser DM, Cowan PJ, Dwyer KM.
PMID: 27830181
Transplant Direct. 2016 Jun 06;2(7):e87. doi: 10.1097/TXD.0000000000000601. eCollection 2016 Jul.

BACKGROUND: Activins, members of the TGF-β superfamily, are key drivers of inflammation and are thought to play a significant role in ischemia-reperfusion injury (IRI), a process inherent to renal transplantation that negatively impacts early and late allograft function. Follistatin...

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Fang DY, Lu B, Hayward S, et al. The Role of Activin A and B and the Benefit of Follistatin Treatment in Renal Ischemia-Reperfusion Injury in Mice. Transplant Direct. 2016;2(7):e87doi: 10.1097/TXD.0000000000000601.
Fang, D. Y., Lu, B., Hayward, S., de Kretser, D. M., Cowan, P. J., & Dwyer, K. M. (2016). The Role of Activin A and B and the Benefit of Follistatin Treatment in Renal Ischemia-Reperfusion Injury in Mice. Transplantation direct, 2(7), e87. https://doi.org/10.1097/TXD.0000000000000601
Fang, Doreen Y P, et al. "The Role of Activin A and B and the Benefit of Follistatin Treatment in Renal Ischemia-Reperfusion Injury in Mice." Transplantation direct vol. 2,7 (2016): e87. doi: https://doi.org/10.1097/TXD.0000000000000601
Fang DY, Lu B, Hayward S, de Kretser DM, Cowan PJ, Dwyer KM. The Role of Activin A and B and the Benefit of Follistatin Treatment in Renal Ischemia-Reperfusion Injury in Mice. Transplant Direct. 2016 Jun 06;2(7):e87. doi: 10.1097/TXD.0000000000000601. eCollection 2016 Jul. PMID: 27830181; PMCID: PMC5087569.
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Activin Biology After Lung Transplantation.

Transplantation direct

Westall GP, Snell GI, Loskot M, Levvey B, O'Hehir R, Hedger MP, de Kretser DM.
PMID: 28620643
Transplant Direct. 2017 May 11;3(6):e159. doi: 10.1097/TXD.0000000000000676. eCollection 2017 Jun.

BACKGROUND: Activins A and B, members of the TGF-β superfamily, are produced as part of the physiological response to tissue damage and the resulting proinflammatory response. Given that lung allograft reperfusion results in an inflammatory response, it is likely...

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Westall GP, Snell GI, Loskot M, et al. Activin Biology After Lung Transplantation. Transplant Direct. 2017;3(6):e159doi: 10.1097/TXD.0000000000000676.
Westall, G. P., Snell, G. I., Loskot, M., Levvey, B., O'Hehir, R., Hedger, M. P., & de Kretser, D. M. (2017). Activin Biology After Lung Transplantation. Transplantation direct, 3(6), e159. https://doi.org/10.1097/TXD.0000000000000676
Westall, Glen P, et al. "Activin Biology After Lung Transplantation." Transplantation direct vol. 3,6 (2017): e159. doi: https://doi.org/10.1097/TXD.0000000000000676
Westall GP, Snell GI, Loskot M, Levvey B, O'Hehir R, Hedger MP, de Kretser DM. Activin Biology After Lung Transplantation. Transplant Direct. 2017 May 11;3(6):e159. doi: 10.1097/TXD.0000000000000676. eCollection 2017 Jun. PMID: 28620643; PMCID: PMC5464778.
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Rethinking ME/CFS Diagnostic Reference Intervals via Machine Learning, and the Utility of Activin B for Defining Symptom Severity.

Diagnostics (Basel, Switzerland)

Lidbury BA, Kita B, Richardson AM, Lewis DP, Privitera E, Hayward S, de Kretser D, Hedger M.
PMID: 31331036
Diagnostics (Basel). 2019 Jul 19;9(3). doi: 10.3390/diagnostics9030079.

Biomarker discovery applied to myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS), a disabling disease of inconclusive aetiology, has identified several cytokines to potentially fulfil a role as a quantitative blood/serum marker for laboratory diagnosis, with activin B a recent addition. We...

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Lidbury BA, Kita B, Richardson AM, et al. Rethinking ME/CFS Diagnostic Reference Intervals via Machine Learning, and the Utility of Activin B for Defining Symptom Severity. Diagnostics (Basel). 2019;9(3)doi: 10.3390/diagnostics9030079.
Lidbury, B. A., Kita, B., Richardson, A. M., Lewis, D. P., Privitera, E., Hayward, S., de Kretser, D., & Hedger, M. (2019). Rethinking ME/CFS Diagnostic Reference Intervals via Machine Learning, and the Utility of Activin B for Defining Symptom Severity. Diagnostics (Basel, Switzerland), 9(3), . https://doi.org/10.3390/diagnostics9030079
Lidbury, Brett A, et al. "Rethinking ME/CFS Diagnostic Reference Intervals via Machine Learning, and the Utility of Activin B for Defining Symptom Severity." Diagnostics (Basel, Switzerland) vol. 9,3 (2019). doi: https://doi.org/10.3390/diagnostics9030079
Lidbury BA, Kita B, Richardson AM, Lewis DP, Privitera E, Hayward S, de Kretser D, Hedger M. Rethinking ME/CFS Diagnostic Reference Intervals via Machine Learning, and the Utility of Activin B for Defining Symptom Severity. Diagnostics (Basel). 2019 Jul 19;9(3). doi: 10.3390/diagnostics9030079. PMID: 31331036; PMCID: PMC6787626.
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Experimental Cryptorchidism Causes Chronic Inflammation and a Progressive Decline in Sertoli Cell and Leydig Cell Function in the Adult Rat Testis.

Reproductive sciences (Thousand Oaks, Calif.)

Aldahhan RA, Stanton PG, Ludlow H, de Kretser DM, Hedger MP.
PMID: 34008157
Reprod Sci. 2021 Oct;28(10):2916-2928. doi: 10.1007/s43032-021-00616-0. Epub 2021 May 18.

Cryptorchidism causes spermatogenic failure and reduced serum androgen levels, as well as testicular oedema and fibrosis, which are hallmarks of inflammation. However, the role of inflammation and the effects of cryptorchidism on Sertoli cell and Leydig cell function at...

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Aldahhan RA, Stanton PG, Ludlow H, et al. Experimental Cryptorchidism Causes Chronic Inflammation and a Progressive Decline in Sertoli Cell and Leydig Cell Function in the Adult Rat Testis. Reprod Sci. 2021;28(10):2916-2928doi: 10.1007/s43032-021-00616-0.
Aldahhan, R. A., Stanton, P. G., Ludlow, H., de Kretser, D. M., & Hedger, M. P. (2021). Experimental Cryptorchidism Causes Chronic Inflammation and a Progressive Decline in Sertoli Cell and Leydig Cell Function in the Adult Rat Testis. Reproductive sciences (Thousand Oaks, Calif.), 28(10), 2916-2928. https://doi.org/10.1007/s43032-021-00616-0
Aldahhan, Rashid A, et al. "Experimental Cryptorchidism Causes Chronic Inflammation and a Progressive Decline in Sertoli Cell and Leydig Cell Function in the Adult Rat Testis." Reproductive sciences (Thousand Oaks, Calif.) vol. 28,10 (2021): 2916-2928. doi: https://doi.org/10.1007/s43032-021-00616-0
Aldahhan RA, Stanton PG, Ludlow H, de Kretser DM, Hedger MP. Experimental Cryptorchidism Causes Chronic Inflammation and a Progressive Decline in Sertoli Cell and Leydig Cell Function in the Adult Rat Testis. Reprod Sci. 2021 Oct;28(10):2916-2928. doi: 10.1007/s43032-021-00616-0. Epub 2021 May 18. PMID: 34008157.
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Potential treatment of keloid pathogenesis with follistatin 288 by blocking the activin molecular pathway.

Experimental dermatology

Ham S, Harrison C, de Kretser D, Wallace EM, Southwick G, Temple-Smith P.
PMID: 33119185
Exp Dermatol. 2021 Mar;30(3):402-408. doi: 10.1111/exd.14223. Epub 2020 Nov 16.

Keloids are benign tumours caused by abnormal wound healing driven by increased expression of cytokines, including activin A. This study compared effects of activins on normal and keloid-derived human dermal fibroblasts and investigated a novel treatment for keloids using...

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Ham S, Harrison C, de Kretser D, et al. Potential treatment of keloid pathogenesis with follistatin 288 by blocking the activin molecular pathway. Exp Dermatol. 2020;30(3):402-408doi: 10.1111/exd.14223.
Ham, S., Harrison, C., de Kretser, D., Wallace, E. M., Southwick, G., & Temple-Smith, P. (2021). Potential treatment of keloid pathogenesis with follistatin 288 by blocking the activin molecular pathway. Experimental dermatology, 30(3), 402-408. https://doi.org/10.1111/exd.14223
Ham, Seungmin, et al. "Potential treatment of keloid pathogenesis with follistatin 288 by blocking the activin molecular pathway." Experimental dermatology vol. 30,3 (2021): 402-408. doi: https://doi.org/10.1111/exd.14223
Ham S, Harrison C, de Kretser D, Wallace EM, Southwick G, Temple-Smith P. Potential treatment of keloid pathogenesis with follistatin 288 by blocking the activin molecular pathway. Exp Dermatol. 2021 Mar;30(3):402-408. doi: 10.1111/exd.14223. Epub 2020 Nov 16. PMID: 33119185.
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Follistatin and the Breast Implant Capsule.

Plastic and reconstructive surgery. Global open

Frenkiel BA, Temple-Smith P, de Kretser D, Southwick GJ.
PMID: 28458972
Plast Reconstr Surg Glob Open. 2017 Mar 01;5(3):e1258. doi: 10.1097/GOX.0000000000001258. eCollection 2017 Mar.

BACKGROUND: Breast capsular contracture remains an elusive problem faced by plastic surgeons and is the leading long-term complication after breast implantation. Follistatin (Fst) is a protein with known anti-inflammatory and antifibrotic properties and has the potential to limit the...

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Frenkiel BA, Temple-Smith P, de Kretser D, et al. Follistatin and the Breast Implant Capsule. Plast Reconstr Surg Glob Open. 2017;5(3):e1258doi: 10.1097/GOX.0000000000001258.
Frenkiel, B. A., Temple-Smith, P., de Kretser, D., & Southwick, G. J. (2017). Follistatin and the Breast Implant Capsule. Plastic and reconstructive surgery. Global open, 5(3), e1258. https://doi.org/10.1097/GOX.0000000000001258
Frenkiel, Brett A, et al. "Follistatin and the Breast Implant Capsule." Plastic and reconstructive surgery. Global open vol. 5,3 (2017): e1258. doi: https://doi.org/10.1097/GOX.0000000000001258
Frenkiel BA, Temple-Smith P, de Kretser D, Southwick GJ. Follistatin and the Breast Implant Capsule. Plast Reconstr Surg Glob Open. 2017 Mar 01;5(3):e1258. doi: 10.1097/GOX.0000000000001258. eCollection 2017 Mar. PMID: 28458972; PMCID: PMC5404443.
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Showing 1 to 9 of 9 entries