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The costimulatory molecule CD226 signals through VAV1 to amplify TCR signals and promote IL-17 production by CD4.

Science signaling

Gaud G, Roncagalli R, Chaoui K, Bernard I, Familiades J, Colacios C, Kassem S, Monsarrat B, Burlet-Schiltz O, de Peredo AG, Malissen B, Saoudi A.
PMID: 29991650
Sci Signal. 2018 Jul 10;11(538). doi: 10.1126/scisignal.aar3083.

The activation of T cells requires the guanine nucleotide exchange factor VAV1. Using mice in which a tag for affinity purification was attached to endogenous VAV1 molecules, we analyzed by quantitative mass spectrometry the signaling complex that assembles around...

Cite
Gaud G, Roncagalli R, Chaoui K, et al. The costimulatory molecule CD226 signals through VAV1 to amplify TCR signals and promote IL-17 production by CD4. Sci Signal. 2018;11(538)doi: 10.1126/scisignal.aar3083.
Gaud, G., Roncagalli, R., Chaoui, K., Bernard, I., Familiades, J., Colacios, C., Kassem, S., Monsarrat, B., Burlet-Schiltz, O., de Peredo, A. G., Malissen, B., & Saoudi, A. (2018). The costimulatory molecule CD226 signals through VAV1 to amplify TCR signals and promote IL-17 production by CD4. Science signaling, 11(538), . https://doi.org/10.1126/scisignal.aar3083
Gaud, Guillaume, et al. "The costimulatory molecule CD226 signals through VAV1 to amplify TCR signals and promote IL-17 production by CD4." Science signaling vol. 11,538 (2018). doi: https://doi.org/10.1126/scisignal.aar3083
Gaud G, Roncagalli R, Chaoui K, Bernard I, Familiades J, Colacios C, Kassem S, Monsarrat B, Burlet-Schiltz O, de Peredo AG, Malissen B, Saoudi A. The costimulatory molecule CD226 signals through VAV1 to amplify TCR signals and promote IL-17 production by CD4. Sci Signal. 2018 Jul 10;11(538). doi: 10.1126/scisignal.aar3083. PMID: 29991650.
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ProMetIS, deep phenotyping of mouse models by combined proteomics and metabolomics analysis.

Scientific data

Imbert A, Rompais M, Selloum M, Castelli F, Mouton-Barbosa E, Brandolini-Bunlon M, Chu-Van E, Joly C, Hirschler A, Roger P, Burger T, Leblanc S, Sorg T, Ouzia S, Vandenbrouck Y, Médigue C, Junot C, Ferro M, Pujos-Guillot E, de Peredo AG, Fenaille F, Carapito C, Herault Y, Thévenot EA.
PMID: 34862403
Sci Data. 2021 Dec 03;8(1):311. doi: 10.1038/s41597-021-01095-3.

Genes are pleiotropic and getting a better knowledge of their function requires a comprehensive characterization of their mutants. Here, we generated multi-level data combining phenomic, proteomic and metabolomic acquisitions from plasma and liver tissues of two C57BL/6 N mouse...

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Imbert A, Rompais M, Selloum M, et al. ProMetIS, deep phenotyping of mouse models by combined proteomics and metabolomics analysis. Sci Data. 2021;8(1):311doi: 10.1038/s41597-021-01095-3.
Imbert, A., Rompais, M., Selloum, M., Castelli, F., Mouton-Barbosa, E., Brandolini-Bunlon, M., Chu-Van, E., Joly, C., Hirschler, A., Roger, P., Burger, T., Leblanc, S., Sorg, T., Ouzia, S., Vandenbrouck, Y., Médigue, C., Junot, C., Ferro, M., Pujos-Guillot, E., de Peredo, A. G., Fenaille, F., Carapito, C., Herault, Y., & Thévenot, E. A. (2021). ProMetIS, deep phenotyping of mouse models by combined proteomics and metabolomics analysis. Scientific data, 8(1), 311. https://doi.org/10.1038/s41597-021-01095-3
Imbert, Alyssa, et al. "ProMetIS, deep phenotyping of mouse models by combined proteomics and metabolomics analysis." Scientific data vol. 8,1 (2021): 311. doi: https://doi.org/10.1038/s41597-021-01095-3
Imbert A, Rompais M, Selloum M, Castelli F, Mouton-Barbosa E, Brandolini-Bunlon M, Chu-Van E, Joly C, Hirschler A, Roger P, Burger T, Leblanc S, Sorg T, Ouzia S, Vandenbrouck Y, Médigue C, Junot C, Ferro M, Pujos-Guillot E, de Peredo AG, Fenaille F, Carapito C, Herault Y, Thévenot EA. ProMetIS, deep phenotyping of mouse models by combined proteomics and metabolomics analysis. Sci Data. 2021 Dec 03;8(1):311. doi: 10.1038/s41597-021-01095-3. PMID: 34862403.
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Opposing regulatory functions of the TIM3 (HAVCR2) signalosome in primary effector T cells as revealed by quantitative interactomics.

Cellular & molecular immunology

Zhai Y, Celis-Gutierrez J, Voisinne G, Mori D, Girard L, Burlet-Schiltz O, de Peredo AG, Roncagalli R, Malissen B.
PMID: 33139903
Cell Mol Immunol. 2021 Jun;18(6):1581-1583. doi: 10.1038/s41423-020-00575-7. Epub 2020 Nov 02.

No abstract available.

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Zhai Y, Celis-Gutierrez J, Voisinne G, et al. Opposing regulatory functions of the TIM3 (HAVCR2) signalosome in primary effector T cells as revealed by quantitative interactomics. Cell Mol Immunol. 2020;18(6):1581-1583doi: 10.1038/s41423-020-00575-7.
Zhai, Y., Celis-Gutierrez, J., Voisinne, G., Mori, D., Girard, L., Burlet-Schiltz, O., de Peredo, A. G., Roncagalli, R., & Malissen, B. (2021). Opposing regulatory functions of the TIM3 (HAVCR2) signalosome in primary effector T cells as revealed by quantitative interactomics. Cellular & molecular immunology, 18(6), 1581-1583. https://doi.org/10.1038/s41423-020-00575-7
Zhai, Yunhao, et al. "Opposing regulatory functions of the TIM3 (HAVCR2) signalosome in primary effector T cells as revealed by quantitative interactomics." Cellular & molecular immunology vol. 18,6 (2021): 1581-1583. doi: https://doi.org/10.1038/s41423-020-00575-7
Zhai Y, Celis-Gutierrez J, Voisinne G, Mori D, Girard L, Burlet-Schiltz O, de Peredo AG, Roncagalli R, Malissen B. Opposing regulatory functions of the TIM3 (HAVCR2) signalosome in primary effector T cells as revealed by quantitative interactomics. Cell Mol Immunol. 2021 Jun;18(6):1581-1583. doi: 10.1038/s41423-020-00575-7. Epub 2020 Nov 02. PMID: 33139903; PMCID: PMC8167182.
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