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Showing 1 to 12 of 54 entries
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Targeting Alzheimer's Disease Neuro-Metabolic Dysfunction with a Small Molecule Nuclear Receptor Agonist (T3D-959) Reverses Disease Pathologies.

Journal of Alzheimer's disease & Parkinsonism

Tong M, Dominguez C, Didsbury J, de la Monte SM.
PMID: 27525190
J Alzheimers Dis Parkinsonism. 2016 Jun;6(3). doi: 10.4172/2161-0460.1000238. Epub 2016 Jun 03.

BACKGROUND: Alzheimer's disease (AD) could be regarded as a brain form of diabetes since insulin resistance and deficiency develop early and progress with severity of neurodegeneration. Preserving insulin's actions in the brain restores function and reduces neurodegeneration. T3D-959 is...

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Tong M, Dominguez C, Didsbury J, et al. Targeting Alzheimer's Disease Neuro-Metabolic Dysfunction with a Small Molecule Nuclear Receptor Agonist (T3D-959) Reverses Disease Pathologies. J Alzheimers Dis Parkinsonism. 2016;6(3)doi: 10.4172/2161-0460.1000238.
Tong, M., Dominguez, C., Didsbury, J., & de la Monte, S. M. (2016). Targeting Alzheimer's Disease Neuro-Metabolic Dysfunction with a Small Molecule Nuclear Receptor Agonist (T3D-959) Reverses Disease Pathologies. Journal of Alzheimer's disease & Parkinsonism, 6(3), . https://doi.org/10.4172/2161-0460.1000238
Tong, Ming, et al. "Targeting Alzheimer's Disease Neuro-Metabolic Dysfunction with a Small Molecule Nuclear Receptor Agonist (T3D-959) Reverses Disease Pathologies." Journal of Alzheimer's disease & Parkinsonism vol. 6,3 (2016). doi: https://doi.org/10.4172/2161-0460.1000238
Tong M, Dominguez C, Didsbury J, de la Monte SM. Targeting Alzheimer's Disease Neuro-Metabolic Dysfunction with a Small Molecule Nuclear Receptor Agonist (T3D-959) Reverses Disease Pathologies. J Alzheimers Dis Parkinsonism. 2016 Jun;6(3). doi: 10.4172/2161-0460.1000238. Epub 2016 Jun 03. PMID: 27525190; PMCID: PMC4979550.
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Regulation of Aspartyl-(Asparaginyl)-β-Hydroxylase Protein Expression and Function by Phosphorylation in Hepatocellular Carcinoma Cells.

Journal of nature and science

Borgas DL, Gao JS, Tong M, Roper N, de la Monte SM.
PMID: 26029741
J Nat Sci. 2015 Apr 01;1(4).

BACKGROUND: Asparaginyl-β-hydroxylase (AAH) promotes cell adhesion, migration, and invasion via Notch activation. AAH's expression is up-regulated by insulin/IGF signaling through PI3K-Akt, but its protein is independently regulated by GSK-3β. The multiple predicted GSK-3β phosphorylation sites suggest post-translational mechanisms may...

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Borgas DL, Gao JS, Tong M, et al. Regulation of Aspartyl-(Asparaginyl)-β-Hydroxylase Protein Expression and Function by Phosphorylation in Hepatocellular Carcinoma Cells. J Nat Sci. 2015;1(4)
Borgas, D. L., Gao, J. S., Tong, M., Roper, N., & de la Monte, S. M. (2015). Regulation of Aspartyl-(Asparaginyl)-β-Hydroxylase Protein Expression and Function by Phosphorylation in Hepatocellular Carcinoma Cells. Journal of nature and science, 1(4), .
Borgas, Diana L, et al. "Regulation of Aspartyl-(Asparaginyl)-β-Hydroxylase Protein Expression and Function by Phosphorylation in Hepatocellular Carcinoma Cells." Journal of nature and science vol. 1,4 (2015).
Borgas DL, Gao JS, Tong M, Roper N, de la Monte SM. Regulation of Aspartyl-(Asparaginyl)-β-Hydroxylase Protein Expression and Function by Phosphorylation in Hepatocellular Carcinoma Cells. J Nat Sci. 2015 Apr 01;1(4). PMID: 26029741; PMCID: PMC4445734.
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Childhood maltreatment and inflammation among pregnant women with gestational diabetes mellitus: A pilot study.

Obstetric medicine

Bublitz M, De La Monte S, Martin S, Larson L, Bourjeily G.
PMID: 29051779
Obstet Med. 2017 Sep;10(3):120-124. doi: 10.1177/1753495X17701320. Epub 2017 May 10.

BACKGROUND: Women with childhood maltreatment histories are at increased risk for adverse birth outcomes. Mechanisms explaining this link are poorly understood. Past research is limited by sampling pregnant women at low risk for adverse maternal and neonatal outcomes.METHODS: This...

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Bublitz M, De La Monte S, Martin S, et al. Childhood maltreatment and inflammation among pregnant women with gestational diabetes mellitus: A pilot study. Obstet Med. 2017;10(3):120-124doi: 10.1177/1753495X17701320.
Bublitz, M., De La Monte, S., Martin, S., Larson, L., & Bourjeily, G. (2017). Childhood maltreatment and inflammation among pregnant women with gestational diabetes mellitus: A pilot study. Obstetric medicine, 10(3), 120-124. https://doi.org/10.1177/1753495X17701320
Bublitz, Margaret, et al. "Childhood maltreatment and inflammation among pregnant women with gestational diabetes mellitus: A pilot study." Obstetric medicine vol. 10,3 (2017): 120-124. doi: https://doi.org/10.1177/1753495X17701320
Bublitz M, De La Monte S, Martin S, Larson L, Bourjeily G. Childhood maltreatment and inflammation among pregnant women with gestational diabetes mellitus: A pilot study. Obstet Med. 2017 Sep;10(3):120-124. doi: 10.1177/1753495X17701320. Epub 2017 May 10. PMID: 29051779; PMCID: PMC5637996.
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Rationale for Embedded Musculoskeletal Care in Air Force Training and Operational Units.

Journal of athletic training

Nye NS, de la Motte SJ.
PMID: 27685161
J Athl Train. 2016 Nov;51(11):846-848. doi: 10.4085/1062-6050.51.5.10. Epub 2016 Sep 29.

No abstract available.

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Nye NS, de la Motte SJ. Rationale for Embedded Musculoskeletal Care in Air Force Training and Operational Units. J Athl Train. 2016;51(11):846-848doi: 10.4085/1062-6050.51.5.10.
Nye, N. S., & de la Motte, S. J. (2016). Rationale for Embedded Musculoskeletal Care in Air Force Training and Operational Units. Journal of athletic training, 51(11), 846-848. https://doi.org/10.4085/1062-6050.51.5.10
Nye, Nathaniel S, and de la Motte, Sarah J. "Rationale for Embedded Musculoskeletal Care in Air Force Training and Operational Units." Journal of athletic training vol. 51,11 (2016): 846-848. doi: https://doi.org/10.4085/1062-6050.51.5.10
Nye NS, de la Motte SJ. Rationale for Embedded Musculoskeletal Care in Air Force Training and Operational Units. J Athl Train. 2016 Nov;51(11):846-848. doi: 10.4085/1062-6050.51.5.10. Epub 2016 Sep 29. PMID: 27685161; PMCID: PMC5224724.
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Early intranasal insulin therapy halts progression of neurodegeneration: progress in Alzheimer's disease therapeutics.

Aging health

de la Monte SM.
PMID: 26855666
Aging health. 2012;8(1):61-64. doi: 10.2217/ahe.11.89.

No abstract available.

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de la Monte SM. Early intranasal insulin therapy halts progression of neurodegeneration: progress in Alzheimer's disease therapeutics. Aging health. 2012;8(1):61-64doi: 10.2217/ahe.11.89.
de la Monte, S. M. (2012). Early intranasal insulin therapy halts progression of neurodegeneration: progress in Alzheimer's disease therapeutics. Aging health, 8(1), 61-64. https://doi.org/10.2217/ahe.11.89
de la Monte, Suzanne M. "Early intranasal insulin therapy halts progression of neurodegeneration: progress in Alzheimer's disease therapeutics." Aging health vol. 8,1 (2012): 61-64. doi: https://doi.org/10.2217/ahe.11.89
de la Monte SM. Early intranasal insulin therapy halts progression of neurodegeneration: progress in Alzheimer's disease therapeutics. Aging health. 2012;8(1):61-64. doi: 10.2217/ahe.11.89. PMID: 26855666; PMCID: PMC4743662.
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Early limited nitrosamine exposures exacerbate high fat diet-mediated type 2 diabetes and neurodegeneration.

BMC endocrine disorders

Tong M, Longato L, de la Monte SM.
PMID: 20302640
BMC Endocr Disord. 2010 Mar 19;10:4. doi: 10.1186/1472-6823-10-4.

BACKGROUND: Type 2 diabetes mellitus (T2DM) and several types of neurodegeneration, including Alzheimer's, are linked to insulin-resistance, and chronic high dietary fat intake causes T2DM with mild neurodegeneration. Intra-cerebral Streptozotocin, a nitrosamine-related compound, causes neurodegeneration, whereas peripheral treatment causes...

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Tong M, Longato L, de la Monte SM. Early limited nitrosamine exposures exacerbate high fat diet-mediated type 2 diabetes and neurodegeneration. BMC Endocr Disord. 2010;10:4doi: 10.1186/1472-6823-10-4.
Tong, M., Longato, L., & de la Monte, S. M. (2010). Early limited nitrosamine exposures exacerbate high fat diet-mediated type 2 diabetes and neurodegeneration. BMC endocrine disorders, 104. https://doi.org/10.1186/1472-6823-10-4
Tong, Ming, et al. "Early limited nitrosamine exposures exacerbate high fat diet-mediated type 2 diabetes and neurodegeneration." BMC endocrine disorders vol. 10 (2010): 4. doi: https://doi.org/10.1186/1472-6823-10-4
Tong M, Longato L, de la Monte SM. Early limited nitrosamine exposures exacerbate high fat diet-mediated type 2 diabetes and neurodegeneration. BMC Endocr Disord. 2010 Mar 19;10:4. doi: 10.1186/1472-6823-10-4. PMID: 20302640; PMCID: PMC3161394.
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Nitrosamine exposure exacerbates high fat diet-mediated type 2 diabetes mellitus, non-alcoholic steatohepatitis, and neurodegeneration with cognitive impairment.

Molecular neurodegeneration

de la Monte SM, Tong M, Lawton M, Longato L.
PMID: 20034403
Mol Neurodegener. 2009 Dec 24;4:54. doi: 10.1186/1750-1326-4-54.

BACKGROUND: The current epidemics of type 2 diabetes mellitus (T2DM), non-alcoholic steatohepatitis (NASH), and Alzheimer's disease (AD) all represent insulin-resistance diseases. Previous studies linked insulin resistance diseases to high fat diets or exposure to streptozotocin, a nitrosamine-related compound that...

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de la Monte SM, Tong M, Lawton M, et al. Nitrosamine exposure exacerbates high fat diet-mediated type 2 diabetes mellitus, non-alcoholic steatohepatitis, and neurodegeneration with cognitive impairment. Mol Neurodegener. 2009;4:54doi: 10.1186/1750-1326-4-54.
de la Monte, S. M., Tong, M., Lawton, M., & Longato, L. (2009). Nitrosamine exposure exacerbates high fat diet-mediated type 2 diabetes mellitus, non-alcoholic steatohepatitis, and neurodegeneration with cognitive impairment. Molecular neurodegeneration, 454. https://doi.org/10.1186/1750-1326-4-54
de la Monte, Suzanne M, et al. "Nitrosamine exposure exacerbates high fat diet-mediated type 2 diabetes mellitus, non-alcoholic steatohepatitis, and neurodegeneration with cognitive impairment." Molecular neurodegeneration vol. 4 (2009): 54. doi: https://doi.org/10.1186/1750-1326-4-54
de la Monte SM, Tong M, Lawton M, Longato L. Nitrosamine exposure exacerbates high fat diet-mediated type 2 diabetes mellitus, non-alcoholic steatohepatitis, and neurodegeneration with cognitive impairment. Mol Neurodegener. 2009 Dec 24;4:54. doi: 10.1186/1750-1326-4-54. PMID: 20034403; PMCID: PMC2803782.
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Enzymatic Responses to Alcohol and Tobacco Nicotine-Derived Nitrosamine Ketone Exposures in Long Evans Rat Livers.

Austin liver

Yalcin EB, Tong M, de la Monte SM.
PMID: 29658012
Austin Liver. 2016;1(1). Epub 2016 Dec 01.

BACKGROUND: Chronic feeding plus binge administration of ethanol causes very high blood alcohol concentrations. However, its co-administration with tobacco Nicotine-Derived Nitrosamine Ketone (NNK) results in somewhat lower blood alcohol levels, suggesting that NNK and therefore smoking, alters alcohol metabolism...

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Yalcin EB, Tong M, de la Monte SM. Enzymatic Responses to Alcohol and Tobacco Nicotine-Derived Nitrosamine Ketone Exposures in Long Evans Rat Livers. Austin Liver. 2016;1(1)
Yalcin, E. B., Tong, M., & de la Monte, S. M. (2016). Enzymatic Responses to Alcohol and Tobacco Nicotine-Derived Nitrosamine Ketone Exposures in Long Evans Rat Livers. Austin liver, 1(1), .
Yalcin, E B, et al. "Enzymatic Responses to Alcohol and Tobacco Nicotine-Derived Nitrosamine Ketone Exposures in Long Evans Rat Livers." Austin liver vol. 1,1 (2016).
Yalcin EB, Tong M, de la Monte SM. Enzymatic Responses to Alcohol and Tobacco Nicotine-Derived Nitrosamine Ketone Exposures in Long Evans Rat Livers. Austin Liver. 2016;1(1). Epub 2016 Dec 01. PMID: 29658012; PMCID: PMC5898820.
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Tobacco Smoke-Induced Hepatic Injury with Steatosis, Inflammation, and Impairments in Insulin and Insulin-Like Growth Factor Signaling.

Journal of clinical & experimental pathology

de la Monte SM, Tong M, Agarwal AR, Cadenas E.
PMID: 27525191
J Clin Exp Pathol. 2016 Apr;6(2). doi: 10.4172/2161-0681.1000269. Epub 2016 Apr 17.

BACKGROUND: Alcoholic liver disease (ALD) is associated with impairments in hepatic insulin and insulin-like growth factor (IGF) signaling through cell growth, survival, and metabolic pathways. Since not all heavy drinkers develop ALD, co-factors may be important. Epidemiologic data indicate...

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de la Monte SM, Tong M, Agarwal AR, et al. Tobacco Smoke-Induced Hepatic Injury with Steatosis, Inflammation, and Impairments in Insulin and Insulin-Like Growth Factor Signaling. J Clin Exp Pathol. 2016;6(2)doi: 10.4172/2161-0681.1000269.
de la Monte, S. M., Tong, M., Agarwal, A. R., & Cadenas, E. (2016). Tobacco Smoke-Induced Hepatic Injury with Steatosis, Inflammation, and Impairments in Insulin and Insulin-Like Growth Factor Signaling. Journal of clinical & experimental pathology, 6(2), . https://doi.org/10.4172/2161-0681.1000269
de la Monte, Suzanne M, et al. "Tobacco Smoke-Induced Hepatic Injury with Steatosis, Inflammation, and Impairments in Insulin and Insulin-Like Growth Factor Signaling." Journal of clinical & experimental pathology vol. 6,2 (2016). doi: https://doi.org/10.4172/2161-0681.1000269
de la Monte SM, Tong M, Agarwal AR, Cadenas E. Tobacco Smoke-Induced Hepatic Injury with Steatosis, Inflammation, and Impairments in Insulin and Insulin-Like Growth Factor Signaling. J Clin Exp Pathol. 2016 Apr;6(2). doi: 10.4172/2161-0681.1000269. Epub 2016 Apr 17. PMID: 27525191; PMCID: PMC4979551.
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Sustained Impairments in Brain Insulin/IGF Signaling in Adolescent Rats Subjected to Binge Alcohol Exposures during Development.

Journal of clinical & experimental pathology

Ewenczyk A, Ziplow J, Tong M, Le T, de la Monte SM.
PMID: 26322248
J Clin Exp Pathol. 2012 Mar;2(2). doi: 10.4172/2161-0681.1000106.

BACKGROUND: Chronic or binge ethanol exposures during development can cause fetal alcohol spectrum disorder (FASD) which consists of an array of neurobehavioral deficits, together with structural, molecular, biochemical, and neurotransmitter abnormalities in the brain. Previous studies showed that perinatal...

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Ewenczyk A, Ziplow J, Tong M, et al. Sustained Impairments in Brain Insulin/IGF Signaling in Adolescent Rats Subjected to Binge Alcohol Exposures during Development. J Clin Exp Pathol. 2012;2(2)doi: 10.4172/2161-0681.1000106.
Ewenczyk, A., Ziplow, J., Tong, M., Le, T., & de la Monte, S. M. (2012). Sustained Impairments in Brain Insulin/IGF Signaling in Adolescent Rats Subjected to Binge Alcohol Exposures during Development. Journal of clinical & experimental pathology, 2(2), . https://doi.org/10.4172/2161-0681.1000106
Ewenczyk, Alexandra, et al. "Sustained Impairments in Brain Insulin/IGF Signaling in Adolescent Rats Subjected to Binge Alcohol Exposures during Development." Journal of clinical & experimental pathology vol. 2,2 (2012). doi: https://doi.org/10.4172/2161-0681.1000106
Ewenczyk A, Ziplow J, Tong M, Le T, de la Monte SM. Sustained Impairments in Brain Insulin/IGF Signaling in Adolescent Rats Subjected to Binge Alcohol Exposures during Development. J Clin Exp Pathol. 2012 Mar;2(2). doi: 10.4172/2161-0681.1000106. PMID: 26322248; PMCID: PMC4550301.
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A Case of Congenital Brainstem Oligodendroglioma: Pathology Findings and Review of the Literature.

Case reports in neurological medicine

Kostadinov S, de la Monte S.
PMID: 28815096
Case Rep Neurol Med. 2017;2017:2465681. doi: 10.1155/2017/2465681. Epub 2017 Jul 26.

Congenital and perinatal primary brain neoplasms are extremely rare. Brainstem neoplasms in the perinatal and neonatal period are typically of high-grade nature and have poor prognoses with survival rates of less than 2 years from diagnosis. Herein, we report...

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Kostadinov S, de la Monte S. A Case of Congenital Brainstem Oligodendroglioma: Pathology Findings and Review of the Literature. Case Rep Neurol Med. 2017;2017:2465681doi: 10.1155/2017/2465681.
Kostadinov, S., & de la Monte, S. (2017). A Case of Congenital Brainstem Oligodendroglioma: Pathology Findings and Review of the Literature. Case reports in neurological medicine, 20172465681. https://doi.org/10.1155/2017/2465681
Kostadinov, Stefan, and de la Monte, Suzanne. "A Case of Congenital Brainstem Oligodendroglioma: Pathology Findings and Review of the Literature." Case reports in neurological medicine vol. 2017 (2017): 2465681. doi: https://doi.org/10.1155/2017/2465681
Kostadinov S, de la Monte S. A Case of Congenital Brainstem Oligodendroglioma: Pathology Findings and Review of the Literature. Case Rep Neurol Med. 2017;2017:2465681. doi: 10.1155/2017/2465681. Epub 2017 Jul 26. PMID: 28815096; PMCID: PMC5549497.
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The role of inflammation in the association between gestational diabetes and obstructive sleep apnea: A pilot study.

Obstetric medicine

Bublitz MH, Carpenter M, Amin S, Okun ML, Millman R, De La Monte SM, Bourjeily G.
PMID: 30574181
Obstet Med. 2018 Dec;11(4):186-191. doi: 10.1177/1753495X18780095. Epub 2018 Jul 24.

BACKGROUND: Obstructive sleep apnea is associated with pregnancy complications including gestational diabetes. Mechanisms underlying the association between obstructive sleep apnea and gestational diabetes remain to be elucidated.METHODS: Twenty-three participants with gestational diabetes underwent home sleep apnea testing. Obstructive sleep...

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Bublitz MH, Carpenter M, Amin S, et al. The role of inflammation in the association between gestational diabetes and obstructive sleep apnea: A pilot study. Obstet Med. 2018;11(4):186-191doi: 10.1177/1753495X18780095.
Bublitz, M. H., Carpenter, M., Amin, S., Okun, M. L., Millman, R., De La Monte, S. M., & Bourjeily, G. (2018). The role of inflammation in the association between gestational diabetes and obstructive sleep apnea: A pilot study. Obstetric medicine, 11(4), 186-191. https://doi.org/10.1177/1753495X18780095
Bublitz, M H, et al. "The role of inflammation in the association between gestational diabetes and obstructive sleep apnea: A pilot study." Obstetric medicine vol. 11,4 (2018): 186-191. doi: https://doi.org/10.1177/1753495X18780095
Bublitz MH, Carpenter M, Amin S, Okun ML, Millman R, De La Monte SM, Bourjeily G. The role of inflammation in the association between gestational diabetes and obstructive sleep apnea: A pilot study. Obstet Med. 2018 Dec;11(4):186-191. doi: 10.1177/1753495X18780095. Epub 2018 Jul 24. PMID: 30574181; PMCID: PMC6295763.
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Showing 1 to 12 of 54 entries