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Retraction. Bradykinin production and increased pulmonary endothelial permeability during acute respiratory failure in unanesthetized sheep.

The Journal of clinical investigation

O'Brodovich HM, Stalcup SA, Pang LM, Lipset JS, Mellins RB.
PMID: 3914486
J Clin Invest. 1985 May;75(5):1747.
Free PMC Article

No abstract available.

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O'Brodovich HM, Stalcup SA, Pang LM, et al. Retraction. Bradykinin production and increased pulmonary endothelial permeability during acute respiratory failure in unanesthetized sheep. J Clin Invest. 1985;75(5):1747
O'Brodovich, H. M., Stalcup, S. A., Pang, L. M., Lipset, J. S., & Mellins, R. B. (1985). Retraction. Bradykinin production and increased pulmonary endothelial permeability during acute respiratory failure in unanesthetized sheep. The Journal of clinical investigation, 75(5), 1747.
O'Brodovich, H M, et al. "Retraction. Bradykinin production and increased pulmonary endothelial permeability during acute respiratory failure in unanesthetized sheep." The Journal of clinical investigation vol. 75,5 (1985): 1747.
O'Brodovich HM, Stalcup SA, Pang LM, Lipset JS, Mellins RB. Retraction. Bradykinin production and increased pulmonary endothelial permeability during acute respiratory failure in unanesthetized sheep. J Clin Invest. 1985 May;75(5):1747. PMID: 3914486; PMCID: PMC425522.
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Balanophora spicata and Lupeol Acetate Possess Antinociceptive and Anti-Inflammatory Activities In Vivo and In Vitro.

Evidence-based complementary and alternative medicine : eCAM

Chen YF, Ching C, Wu TS, Wu CR, Hsieh WT, Tsai HY.
PMID: 23243439
Evid Based Complement Alternat Med. 2012;2012:371273. doi: 10.1155/2012/371273. Epub 2012 Nov 11.

Aims of the present study were to investigate effects of Balanophora spicata (BS) on antinociception and anti-inflammation both in vivo and in vitro. Crude extract of BS inhibited vascular permeability induced by histamine, serotonin, bradykinin, and PGE(2), but not...

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Chen YF, Ching C, Wu TS, et al. Balanophora spicata and Lupeol Acetate Possess Antinociceptive and Anti-Inflammatory Activities In Vivo and In Vitro. Evid Based Complement Alternat Med. 2012;2012:371273doi: 10.1155/2012/371273.
Chen, Y. F., Ching, C., Wu, T. S., Wu, C. R., Hsieh, W. T., & Tsai, H. Y. (2012). Balanophora spicata and Lupeol Acetate Possess Antinociceptive and Anti-Inflammatory Activities In Vivo and In Vitro. Evidence-based complementary and alternative medicine : eCAM, 2012371273. https://doi.org/10.1155/2012/371273
Chen, Yuh-Fung, et al. "Balanophora spicata and Lupeol Acetate Possess Antinociceptive and Anti-Inflammatory Activities In Vivo and In Vitro." Evidence-based complementary and alternative medicine : eCAM vol. 2012 (2012): 371273. doi: https://doi.org/10.1155/2012/371273
Chen YF, Ching C, Wu TS, Wu CR, Hsieh WT, Tsai HY. Balanophora spicata and Lupeol Acetate Possess Antinociceptive and Anti-Inflammatory Activities In Vivo and In Vitro. Evid Based Complement Alternat Med. 2012;2012:371273. doi: 10.1155/2012/371273. Epub 2012 Nov 11. PMID: 23243439; PMCID: PMC3518255.
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Correction: Surface- and tip-enhanced Raman scattering of bradykinin onto the colloidal suspended Ag surface.

Physical chemistry chemical physics : PCCP

Swiech D, Ozaki Y, Kim Y, Proniewicz E.
PMID: 26144846
Phys Chem Chem Phys. 2015 Jul 15;17(29):19672. doi: 10.1039/c5cp90117k.

No abstract available.

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Swiech D, Ozaki Y, Kim Y, et al. Correction: Surface- and tip-enhanced Raman scattering of bradykinin onto the colloidal suspended Ag surface. Phys Chem Chem Phys. 2015;17(29):19672doi: 10.1039/c5cp90117k.
Swiech, D., Ozaki, Y., Kim, Y., & Proniewicz, E. (2015). Correction: Surface- and tip-enhanced Raman scattering of bradykinin onto the colloidal suspended Ag surface. Physical chemistry chemical physics : PCCP, 17(29), 19672. https://doi.org/10.1039/c5cp90117k
Swiech, D, et al. "Correction: Surface- and tip-enhanced Raman scattering of bradykinin onto the colloidal suspended Ag surface." Physical chemistry chemical physics : PCCP vol. 17,29 (2015): 19672. doi: https://doi.org/10.1039/c5cp90117k
Swiech D, Ozaki Y, Kim Y, Proniewicz E. Correction: Surface- and tip-enhanced Raman scattering of bradykinin onto the colloidal suspended Ag surface. Phys Chem Chem Phys. 2015 Jul 15;17(29):19672. doi: 10.1039/c5cp90117k. PMID: 26144846.
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Angiotensin II Type 1 Receptor Blockade: a Novel Therapeutic Concept.

Blood pressure

Johnston CI.
PMID: 28425795
Blood Press. 2000;9:9-13. doi: 10.1080/080370500439155.

Angiotensin II type 1 (AT 1 ) receptor blockers, such as candesartan, are attractive alternatives to ACE inhibitors in the treatment of hypertension and cardiovascular disease. Although angiotensin-converting enzyme (ACE) inhibitors are able to suppress the renin-angiotensin system (RAS),...

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Johnston CI. Angiotensin II Type 1 Receptor Blockade: a Novel Therapeutic Concept. Blood Press. 2000;9:9-13doi: 10.1080/080370500439155.
Johnston, C. I. (2000). Angiotensin II Type 1 Receptor Blockade: a Novel Therapeutic Concept. Blood pressure, 99-13. https://doi.org/10.1080/080370500439155
Johnston, Colin I. "Angiotensin II Type 1 Receptor Blockade: a Novel Therapeutic Concept." Blood pressure vol. 9 (2000): 9-13. doi: https://doi.org/10.1080/080370500439155
Johnston CI. Angiotensin II Type 1 Receptor Blockade: a Novel Therapeutic Concept. Blood Press. 2000;9:9-13. doi: 10.1080/080370500439155. PMID: 28425795.
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[Spinal serotonergic receptor is involved in descending inhibition of cardiac nociception by the lateral reticular nucleus in rats].

Nan fang yi ke da xue xue bao = Journal of Southern Medical University

Han M, Liu XH, DU JQ.
PMID: 28951360
Nan Fang Yi Ke Da Xue Xue Bao. 2017 Sep 20;37(9):1190-1194.

OBJECTIVE: To investigate the role of the lateral reticular nucleus (LRN) in descending inhibition of cardiac nociception in rats and the involvement of spinal serotonergic receptors in the descending inhibition.METHODS: Male SD rats were randomly divided into 5 groups,...

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Han M, Liu XH, DU JQ. [Spinal serotonergic receptor is involved in descending inhibition of cardiac nociception by the lateral reticular nucleus in rats]. Nan Fang Yi Ke Da Xue Xue Bao. 2017;37(9):1190-1194
Han, M., Liu, X. H., & DU, J. Q. (2017). [Spinal serotonergic receptor is involved in descending inhibition of cardiac nociception by the lateral reticular nucleus in rats]. Nan fang yi ke da xue xue bao = Journal of Southern Medical University, 37(9), 1190-1194.
Han, Man, et al. "[Spinal serotonergic receptor is involved in descending inhibition of cardiac nociception by the lateral reticular nucleus in rats]." Nan fang yi ke da xue xue bao = Journal of Southern Medical University vol. 37,9 (2017): 1190-1194.
Han M, Liu XH, DU JQ. [Spinal serotonergic receptor is involved in descending inhibition of cardiac nociception by the lateral reticular nucleus in rats]. Nan Fang Yi Ke Da Xue Xue Bao. 2017 Sep 20;37(9):1190-1194. Chinese. PMID: 28951360; PMCID: PMC6765497.
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Processing of Factor XII during Inflammatory Reactions.

Frontiers in medicine

Jukema BN, de Maat S, Maas C.
PMID: 27867935
Front Med (Lausanne). 2016 Nov 04;3:52. doi: 10.3389/fmed.2016.00052. eCollection 2016.

The contact system was originally identified as an obsolete part of the coagulation system, but it has been repeatedly implicated in inflammatory states, such as infection, as well as in allergic- and chronic inflammatory disease. Under these conditions, there...

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Jukema BN, de Maat S, Maas C. Processing of Factor XII during Inflammatory Reactions. Front Med (Lausanne). 2016;3:52doi: 10.3389/fmed.2016.00052.
Jukema, B. N., de Maat, S., & Maas, C. (2016). Processing of Factor XII during Inflammatory Reactions. Frontiers in medicine, 352. https://doi.org/10.3389/fmed.2016.00052
Jukema, Bernard Nico, et al. "Processing of Factor XII during Inflammatory Reactions." Frontiers in medicine vol. 3 (2016): 52. doi: https://doi.org/10.3389/fmed.2016.00052
Jukema BN, de Maat S, Maas C. Processing of Factor XII during Inflammatory Reactions. Front Med (Lausanne). 2016 Nov 04;3:52. doi: 10.3389/fmed.2016.00052. eCollection 2016. PMID: 27867935; PMCID: PMC5095611.
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QM/MM investigation of the catalytic mechanism of angiotensin-converting enzyme.

Journal of molecular modeling

Mu X, Zhang C, Xu D.
PMID: 27184002
J Mol Model. 2016 Jun;22(6):132. doi: 10.1007/s00894-016-3004-2. Epub 2016 May 16.

Angiotensin-converting enzyme (ACE) converts angiotensin I to angiotensin II and degrades bradykinin and other vasoactive peptides. ACE inhibitors are used to treat diseases such as hypertension and heart failure. It is thus highly desirable to understand the catalytic mechanism...

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Mu X, Zhang C, Xu D. QM/MM investigation of the catalytic mechanism of angiotensin-converting enzyme. J Mol Model. 2016;22(6):132doi: 10.1007/s00894-016-3004-2.
Mu, X., Zhang, C., & Xu, D. (2016). QM/MM investigation of the catalytic mechanism of angiotensin-converting enzyme. Journal of molecular modeling, 22(6), 132. https://doi.org/10.1007/s00894-016-3004-2
Mu, Xia, et al. "QM/MM investigation of the catalytic mechanism of angiotensin-converting enzyme." Journal of molecular modeling vol. 22,6 (2016): 132. doi: https://doi.org/10.1007/s00894-016-3004-2
Mu X, Zhang C, Xu D. QM/MM investigation of the catalytic mechanism of angiotensin-converting enzyme. J Mol Model. 2016 Jun;22(6):132. doi: 10.1007/s00894-016-3004-2. Epub 2016 May 16. PMID: 27184002.
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Isolation and biochemical characterization of bradykinin-potentiating peptides from .

The journal of venomous animals and toxins including tropical diseases

Fucase TM, Sciani JM, Cavalcante I, Viala VL, Chagas BB, Pimenta DC, Spencer PJ.
PMID: 28670326
J Venom Anim Toxins Incl Trop Dis. 2017 Jun 26;23:33. doi: 10.1186/s40409-017-0124-9. eCollection 2017.

BACKGROUND: Venoms represent a still underexplored reservoir of bioactive components that might mitigate or cure diseases in conditions in which conventional therapy is ineffective. The bradykinin-potentiating peptides (BPPs) comprise a class of angiotensin-I converting enzyme (ACE) inhibitors. The BPPs...

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Fucase TM, Sciani JM, Cavalcante I, et al. Isolation and biochemical characterization of bradykinin-potentiating peptides from . J Venom Anim Toxins Incl Trop Dis. 2017;23:33doi: 10.1186/s40409-017-0124-9.
Fucase, T. M., Sciani, J. M., Cavalcante, I., Viala, V. L., Chagas, B. B., Pimenta, D. C., & Spencer, P. J. (2017). Isolation and biochemical characterization of bradykinin-potentiating peptides from . The journal of venomous animals and toxins including tropical diseases, 2333. https://doi.org/10.1186/s40409-017-0124-9
Fucase, Tamara M, et al. "Isolation and biochemical characterization of bradykinin-potentiating peptides from ." The journal of venomous animals and toxins including tropical diseases vol. 23 (2017): 33. doi: https://doi.org/10.1186/s40409-017-0124-9
Fucase TM, Sciani JM, Cavalcante I, Viala VL, Chagas BB, Pimenta DC, Spencer PJ. Isolation and biochemical characterization of bradykinin-potentiating peptides from . J Venom Anim Toxins Incl Trop Dis. 2017 Jun 26;23:33. doi: 10.1186/s40409-017-0124-9. eCollection 2017. PMID: 28670326; PMCID: PMC5485657.
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Antamanide Prevents Bradykinin-lnduced Filamin Translocation by Inhibiting Extracellular Calcium Influx.

Drug delivery

Patton WF, Wang Q, Chiang ET, Hechtman HB, Shepro D.
PMID: 26582443
Drug Deliv. 1997;4(4):247-54. doi: 10.3109/10717549709052010.

Bradykinin-induced paracellular gap formation in cultured endothelial cells (ECs) is preceded by cytoskeletal rearrangement. Actin binding proteins, such as nonmuscle filamin, perform a pivotal role in modulating actin organization. In response to bradykinin treatment, EC filamin translocates from the...

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Patton WF, Wang Q, Chiang ET, et al. Antamanide Prevents Bradykinin-lnduced Filamin Translocation by Inhibiting Extracellular Calcium Influx. Drug Deliv. 1997;4(4):247-54doi: 10.3109/10717549709052010.
Patton, W. F., Wang, Q., Chiang, E. T., Hechtman, H. B., & Shepro, D. (1997). Antamanide Prevents Bradykinin-lnduced Filamin Translocation by Inhibiting Extracellular Calcium Influx. Drug delivery, 4(4), 247-54. https://doi.org/10.3109/10717549709052010
Patton, W F, et al. "Antamanide Prevents Bradykinin-lnduced Filamin Translocation by Inhibiting Extracellular Calcium Influx." Drug delivery vol. 4,4 (1997): 247-54. doi: https://doi.org/10.3109/10717549709052010
Patton WF, Wang Q, Chiang ET, Hechtman HB, Shepro D. Antamanide Prevents Bradykinin-lnduced Filamin Translocation by Inhibiting Extracellular Calcium Influx. Drug Deliv. 1997;4(4):247-54. doi: 10.3109/10717549709052010. PMID: 26582443.
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Ex Vivo Smooth Muscle Pharmacological Effects of a Novel Bradykinin-Related Peptide, and Its Analogue, from Chinese Large Odorous Frog, Odorrana livida Skin Secretions.

Toxins

Xiang J, Wang H, Ma C, Zhou M, Wu Y, Wang L, Guo S, Chen T, Shaw C.
PMID: 27690099
Toxins (Basel). 2016 Sep 27;8(10). doi: 10.3390/toxins8100283.

Bradykinin-related peptides (BRPs) are one of the most extensively studied frog secretions-derived peptide families identified from many amphibian species. The diverse primary structures of BRPs have been proven essential for providing valuable information in understanding basic mechanisms associated with...

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Xiang J, Wang H, Ma C, et al. Ex Vivo Smooth Muscle Pharmacological Effects of a Novel Bradykinin-Related Peptide, and Its Analogue, from Chinese Large Odorous Frog, Odorrana livida Skin Secretions. Toxins (Basel). 2016;8(10)doi: 10.3390/toxins8100283.
Xiang, J., Wang, H., Ma, C., Zhou, M., Wu, Y., Wang, L., Guo, S., Chen, T., & Shaw, C. (2016). Ex Vivo Smooth Muscle Pharmacological Effects of a Novel Bradykinin-Related Peptide, and Its Analogue, from Chinese Large Odorous Frog, Odorrana livida Skin Secretions. Toxins, 8(10), . https://doi.org/10.3390/toxins8100283
Xiang, Jie, et al. "Ex Vivo Smooth Muscle Pharmacological Effects of a Novel Bradykinin-Related Peptide, and Its Analogue, from Chinese Large Odorous Frog, Odorrana livida Skin Secretions." Toxins vol. 8,10 (2016). doi: https://doi.org/10.3390/toxins8100283
Xiang J, Wang H, Ma C, Zhou M, Wu Y, Wang L, Guo S, Chen T, Shaw C. Ex Vivo Smooth Muscle Pharmacological Effects of a Novel Bradykinin-Related Peptide, and Its Analogue, from Chinese Large Odorous Frog, Odorrana livida Skin Secretions. Toxins (Basel). 2016 Sep 27;8(10). doi: 10.3390/toxins8100283. PMID: 27690099; PMCID: PMC5086643.
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Transplantation of bradykinin-preconditioned human endothelial progenitor cells improves cardiac function via enhanced Akt/eNOS phosphorylation and angiogenesis.

American journal of translational research

Sheng ZL, Yao YY, Li YF, Fu C, Ma GS.
PMID: 26279749
Am J Transl Res. 2015 Jun 15;7(6):1045-57. eCollection 2015.

This study determines whether preconditioning (PC) of human endothelial progenitor cells (hEPCs) with bradykinin promotes infarcted myocardium repair via enhanced activation of B2 receptor (B2R)-dependent Akt/eNOS and increased angiogenesis. hEPCs with or without bradykinin preconditioning (BK-PC) were transplanted into...

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Sheng ZL, Yao YY, Li YF, et al. Transplantation of bradykinin-preconditioned human endothelial progenitor cells improves cardiac function via enhanced Akt/eNOS phosphorylation and angiogenesis. Am J Transl Res. 2015;7(6):1045-57
Sheng, Z. L., Yao, Y. Y., Li, Y. F., Fu, C., & Ma, G. S. (2015). Transplantation of bradykinin-preconditioned human endothelial progenitor cells improves cardiac function via enhanced Akt/eNOS phosphorylation and angiogenesis. American journal of translational research, 7(6), 1045-57.
Sheng, Zu-Long, et al. "Transplantation of bradykinin-preconditioned human endothelial progenitor cells improves cardiac function via enhanced Akt/eNOS phosphorylation and angiogenesis." American journal of translational research vol. 7,6 (2015): 1045-57.
Sheng ZL, Yao YY, Li YF, Fu C, Ma GS. Transplantation of bradykinin-preconditioned human endothelial progenitor cells improves cardiac function via enhanced Akt/eNOS phosphorylation and angiogenesis. Am J Transl Res. 2015 Jun 15;7(6):1045-57. eCollection 2015. PMID: 26279749; PMCID: PMC4532738.
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In Vivo Effects of Bradykinin B2 Receptor Agonists with Varying Susceptibility to Peptidases.

Frontiers in pharmacology

Jean M, Gera L, Charest-Morin X, Marceau F, Bachelard H.
PMID: 26793104
Front Pharmacol. 2016 Jan 12;6:306. doi: 10.3389/fphar.2015.00306. eCollection 2015.

We reported evidence of bradykinin (BK) regeneration from C-terminal extended BK sequences that behave as peptidase-activated B2 receptor (B2R) agonists. Further to these in vitro studies, we carried out in vivo experiments to verify hemodynamic effects of BK analogs...

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Jean M, Gera L, Charest-Morin X, et al. In Vivo Effects of Bradykinin B2 Receptor Agonists with Varying Susceptibility to Peptidases. Front Pharmacol. 2016;6:306doi: 10.3389/fphar.2015.00306.
Jean, M., Gera, L., Charest-Morin, X., Marceau, F., & Bachelard, H. (2015). In Vivo Effects of Bradykinin B2 Receptor Agonists with Varying Susceptibility to Peptidases. Frontiers in pharmacology, 6306. https://doi.org/10.3389/fphar.2015.00306
Jean, Mélissa, et al. "In Vivo Effects of Bradykinin B2 Receptor Agonists with Varying Susceptibility to Peptidases." Frontiers in pharmacology vol. 6 (2015): 306. doi: https://doi.org/10.3389/fphar.2015.00306
Jean M, Gera L, Charest-Morin X, Marceau F, Bachelard H. In Vivo Effects of Bradykinin B2 Receptor Agonists with Varying Susceptibility to Peptidases. Front Pharmacol. 2016 Jan 12;6:306. doi: 10.3389/fphar.2015.00306. eCollection 2015. PMID: 26793104; PMCID: PMC4709452.
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Showing 1 to 12 of 648 entries