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Prostaglandin E2 inhibits alveolar macrophage phagocytosis through an E-prostanoid 2 receptor-mediated increase in intracellular cyclic AMP

Build and Broaden 3.0 AND Program Evaluation

Aronoff DM, Canetti C.
GSID: dTvz6S0n6-wJ
DM Aronoff, C Canetti… - The Journal of …, 2004 - Am Assoc Immnol

Prostaglandin E 2 is a potent lipid mediator of inflammation that effects changes in cell functions through ligation of four distinct G protein-coupled receptors (E-prostanoid (EP) 1, EP2, EP3, and EP4). During pneumonia, PGE 2 production is enhanced. In...

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Aronoff, D. M., Canetti, C., & Peters-Golden, M. (2004). Prostaglandin E2 inhibits alveolar macrophage phagocytosis through an E-prostanoid 2 receptor-mediated increase in intracellular cyclic AMP. The Journal of Immunology, 173(1), 559-565.
Aronoff, David M., Claudio Canetti, and Marc Peters-Golden. "Prostaglandin E2 inhibits alveolar macrophage phagocytosis through an E-prostanoid 2 receptor-mediated increase in intracellular cyclic AMP." The Journal of Immunology 173.1 (2004): 559-565.
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The Neuroprotective Effect of Lithium in cannabinoid Dependence is Mediated through Modulation of Cyclic AMP, ERK1/2 and GSK-3β Phosphorylation in Cerebellar Granular Neurons of Rat.

Iranian journal of pharmaceutical research : IJPR

Rahimi HR, Ghahremani MH, Dehpour AR, Sharifzadeh M, Ejtemaei-Mehr S, Razmi A, Ostad SN.
PMID: 26664379
Iran J Pharm Res. 2015;14(4):1123-35.

Lithium (Li), a glycogen synthase kinase-3β (GSK-3β) inhibitor, has used to attenuate the cannabinoid-induced dependence/withdrawal signs, but molecular mechanisms related to this are unclear. Recent studies indicate the involvement of upstream extracellular signal kinase1/2 (ERK1/2) and downstream GSK-3β pathways...

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Rahimi HR, Ghahremani MH, Dehpour AR, et al. The Neuroprotective Effect of Lithium in cannabinoid Dependence is Mediated through Modulation of Cyclic AMP, ERK1/2 and GSK-3β Phosphorylation in Cerebellar Granular Neurons of Rat. Iran J Pharm Res. 2015;14(4):1123-35
Rahimi, H. R., Ghahremani, M. H., Dehpour, A. R., Sharifzadeh, M., Ejtemaei-Mehr, S., Razmi, A., & Ostad, S. N. (2015). The Neuroprotective Effect of Lithium in cannabinoid Dependence is Mediated through Modulation of Cyclic AMP, ERK1/2 and GSK-3β Phosphorylation in Cerebellar Granular Neurons of Rat. Iranian journal of pharmaceutical research : IJPR, 14(4), 1123-35.
Rahimi, Hamid Reza, et al. "The Neuroprotective Effect of Lithium in cannabinoid Dependence is Mediated through Modulation of Cyclic AMP, ERK1/2 and GSK-3β Phosphorylation in Cerebellar Granular Neurons of Rat." Iranian journal of pharmaceutical research : IJPR vol. 14,4 (2015): 1123-35.
Rahimi HR, Ghahremani MH, Dehpour AR, Sharifzadeh M, Ejtemaei-Mehr S, Razmi A, Ostad SN. The Neuroprotective Effect of Lithium in cannabinoid Dependence is Mediated through Modulation of Cyclic AMP, ERK1/2 and GSK-3β Phosphorylation in Cerebellar Granular Neurons of Rat. Iran J Pharm Res. 2015;14(4):1123-35. PMID: 26664379; PMCID: PMC4673940.
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HS-142-1, a Novel Non-peptide Antagonist for Atrial Natriuretic Peptide Receptor, Selectively Inhibits Particulate Guanylyl Cyclase and Lowers Cyclic GMP in LLC-PK1 Cells.

Bioscience, biotechnology, and biochemistry

Tanaka T, Ichimura M, Nakajo S, Snajdar RM, Morishita Y, Sano T, Yamada K, Inagami T, Matsuda Y.
PMID: 27286374
Biosci Biotechnol Biochem. 1992 Jan;56(7):1041-5. doi: 10.1271/bbb.56.1041.

HS-142-1, a novel atrial natriuretic peptide (ANP) antagonist isolated from the culture broth of Aureobasidium sp., selectively inhibits ANP-induced cyclic GMP accumulation in porcine kidney epithelial LLC-PK1 cells. At concentrations from 0.1 to 100 μg/ml (= 2.5 × 10(-8)...

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Tanaka T, Ichimura M, Nakajo S, et al. HS-142-1, a Novel Non-peptide Antagonist for Atrial Natriuretic Peptide Receptor, Selectively Inhibits Particulate Guanylyl Cyclase and Lowers Cyclic GMP in LLC-PK1 Cells. Biosci Biotechnol Biochem. 1992;56(7):1041-5doi: 10.1271/bbb.56.1041.
Tanaka, T., Ichimura, M., Nakajo, S., Snajdar, R. M., Morishita, Y., Sano, T., Yamada, K., Inagami, T., & Matsuda, Y. (1992). HS-142-1, a Novel Non-peptide Antagonist for Atrial Natriuretic Peptide Receptor, Selectively Inhibits Particulate Guanylyl Cyclase and Lowers Cyclic GMP in LLC-PK1 Cells. Bioscience, biotechnology, and biochemistry, 56(7), 1041-5. https://doi.org/10.1271/bbb.56.1041
Tanaka, T, et al. "HS-142-1, a Novel Non-peptide Antagonist for Atrial Natriuretic Peptide Receptor, Selectively Inhibits Particulate Guanylyl Cyclase and Lowers Cyclic GMP in LLC-PK1 Cells." Bioscience, biotechnology, and biochemistry vol. 56,7 (1992): 1041-5. doi: https://doi.org/10.1271/bbb.56.1041
Tanaka T, Ichimura M, Nakajo S, Snajdar RM, Morishita Y, Sano T, Yamada K, Inagami T, Matsuda Y. HS-142-1, a Novel Non-peptide Antagonist for Atrial Natriuretic Peptide Receptor, Selectively Inhibits Particulate Guanylyl Cyclase and Lowers Cyclic GMP in LLC-PK1 Cells. Biosci Biotechnol Biochem. 1992 Jan;56(7):1041-5. doi: 10.1271/bbb.56.1041. PMID: 27286374.
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Molecular mechanisms of gonadotropin-releasing hormone signaling: integrating cyclic nucleotides into the network.

Frontiers in endocrinology

Perrett RM, McArdle CA.
PMID: 24312080
Front Endocrinol (Lausanne). 2013 Nov 20;4:180. doi: 10.3389/fendo.2013.00180.

Gonadotropin-releasing hormone (GnRH) is the primary regulator of mammalian reproductive function in both males and females. It acts via G-protein coupled receptors on gonadotropes to stimulate synthesis and secretion of the gonadotropin hormones luteinizing hormone and follicle-stimulating hormone. These...

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Perrett RM, McArdle CA. Molecular mechanisms of gonadotropin-releasing hormone signaling: integrating cyclic nucleotides into the network. Front Endocrinol (Lausanne). 2013;4:180doi: 10.3389/fendo.2013.00180.
Perrett, R. M., & McArdle, C. A. (2013). Molecular mechanisms of gonadotropin-releasing hormone signaling: integrating cyclic nucleotides into the network. Frontiers in endocrinology, 4180. https://doi.org/10.3389/fendo.2013.00180
Perrett, Rebecca M, and McArdle, Craig A. "Molecular mechanisms of gonadotropin-releasing hormone signaling: integrating cyclic nucleotides into the network." Frontiers in endocrinology vol. 4 (2013): 180. doi: https://doi.org/10.3389/fendo.2013.00180
Perrett RM, McArdle CA. Molecular mechanisms of gonadotropin-releasing hormone signaling: integrating cyclic nucleotides into the network. Front Endocrinol (Lausanne). 2013 Nov 20;4:180. doi: 10.3389/fendo.2013.00180. PMID: 24312080; PMCID: PMC3834291.
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Different effects of selenium on cyclic AMP metabolism in hepatoma cells and normal liver cells.

Biological trace element research

Yu SY, Wang LM.
PMID: 24263364
Biol Trace Elem Res. 1983 Feb;5(1):9-16. doi: 10.1007/BF02916922.

Sodium selenite administered to normal and hepatoma (HA)-bearing mice sc (2 mg/kg) or ip (1 mg/kg) led to a significant increase in cyclic AMP (cAMP) level and a decrease in cAMP phosphodiesterase (PDE) activity in HA cells. In contrast,...

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Yu SY, Wang LM. Different effects of selenium on cyclic AMP metabolism in hepatoma cells and normal liver cells. Biol Trace Elem Res. 1983;5(1):9-16doi: 10.1007/BF02916922.
Yu, S. Y., & Wang, L. M. (1983). Different effects of selenium on cyclic AMP metabolism in hepatoma cells and normal liver cells. Biological trace element research, 5(1), 9-16. https://doi.org/10.1007/BF02916922
Yu, S Y, and Wang, L M. "Different effects of selenium on cyclic AMP metabolism in hepatoma cells and normal liver cells." Biological trace element research vol. 5,1 (1983): 9-16. doi: https://doi.org/10.1007/BF02916922
Yu SY, Wang LM. Different effects of selenium on cyclic AMP metabolism in hepatoma cells and normal liver cells. Biol Trace Elem Res. 1983 Feb;5(1):9-16. doi: 10.1007/BF02916922. PMID: 24263364.
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Intercellular adhesion : I. A quantitative assay for measuring the rate of adhesion.

The Journal of membrane biology

Orr CW, Roseman S.
PMID: 24174045
J Membr Biol. 1969 Dec;1(1):109-24. doi: 10.1007/BF01869777.

A quantitative procedure for determining the early kinetics of cell aggregation (adhesion) is described. The cells used for this study were obtained by dissociation of 8-day-old embryonic chicken neural retina with crude trypsin. The method is based on determining...

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Orr CW, Roseman S. Intercellular adhesion : I. A quantitative assay for measuring the rate of adhesion. J Membr Biol. 1969;1(1):109-24doi: 10.1007/BF01869777.
Orr, C. W., & Roseman, S. (1969). Intercellular adhesion : I. A quantitative assay for measuring the rate of adhesion. The Journal of membrane biology, 1(1), 109-24. https://doi.org/10.1007/BF01869777
Orr, C W, and Roseman, S. "Intercellular adhesion : I. A quantitative assay for measuring the rate of adhesion." The Journal of membrane biology vol. 1,1 (1969): 109-24. doi: https://doi.org/10.1007/BF01869777
Orr CW, Roseman S. Intercellular adhesion : I. A quantitative assay for measuring the rate of adhesion. J Membr Biol. 1969 Dec;1(1):109-24. doi: 10.1007/BF01869777. PMID: 24174045.
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Isorhynchophylline Protects PC12 Cells Against Beta-Amyloid-Induced Apoptosis via PI3K/Akt Signaling Pathway.

Evidence-based complementary and alternative medicine : eCAM

Xian YF, Lin ZX, Mao QQ, Chen JN, Su ZR, Lai XP, Ip PS.
PMID: 24319473
Evid Based Complement Alternat Med. 2013;2013:163057. doi: 10.1155/2013/163057. Epub 2013 Nov 06.

The neurotoxicity of amyloid- β (A β ) has been implicated as a critical cause of Alzheimer's disease. Isorhynchophylline (IRN), an oxindole alkaloid isolated from Uncaria rhynchophylla, exerts neuroprotective effect against Aβ 25-35-induced neurotoxicity in vitro. However, the exact...

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Xian YF, Lin ZX, Mao QQ, et al. Isorhynchophylline Protects PC12 Cells Against Beta-Amyloid-Induced Apoptosis via PI3K/Akt Signaling Pathway. Evid Based Complement Alternat Med. 2013;2013:163057doi: 10.1155/2013/163057.
Xian, Y. F., Lin, Z. X., Mao, Q. Q., Chen, J. N., Su, Z. R., Lai, X. P., & Ip, P. S. (2013). Isorhynchophylline Protects PC12 Cells Against Beta-Amyloid-Induced Apoptosis via PI3K/Akt Signaling Pathway. Evidence-based complementary and alternative medicine : eCAM, 2013163057. https://doi.org/10.1155/2013/163057
Xian, Yan-Fang, et al. "Isorhynchophylline Protects PC12 Cells Against Beta-Amyloid-Induced Apoptosis via PI3K/Akt Signaling Pathway." Evidence-based complementary and alternative medicine : eCAM vol. 2013 (2013): 163057. doi: https://doi.org/10.1155/2013/163057
Xian YF, Lin ZX, Mao QQ, Chen JN, Su ZR, Lai XP, Ip PS. Isorhynchophylline Protects PC12 Cells Against Beta-Amyloid-Induced Apoptosis via PI3K/Akt Signaling Pathway. Evid Based Complement Alternat Med. 2013;2013:163057. doi: 10.1155/2013/163057. Epub 2013 Nov 06. PMID: 24319473; PMCID: PMC3836301.
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PRKACA: the catalytic subunit of protein kinase A and adrenocortical tumors.

Frontiers in cell and developmental biology

Berthon AS, Szarek E, Stratakis CA.
PMID: 26042218
Front Cell Dev Biol. 2015 May 20;3:26. doi: 10.3389/fcell.2015.00026. eCollection 2015.

Cyclic-AMP (cAMP)-dependent protein kinase (PKA) is the main effector of cAMP signaling in all tissues. Inactivating mutations of the PRKAR1A gene, coding for the type 1A regulatory subunit of PKA, are responsible for Carney complex and primary pigmented nodular...

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Berthon AS, Szarek E, Stratakis CA. PRKACA: the catalytic subunit of protein kinase A and adrenocortical tumors. Front Cell Dev Biol. 2015;3:26doi: 10.3389/fcell.2015.00026.
Berthon, A. S., Szarek, E., & Stratakis, C. A. (2015). PRKACA: the catalytic subunit of protein kinase A and adrenocortical tumors. Frontiers in cell and developmental biology, 326. https://doi.org/10.3389/fcell.2015.00026
Berthon, Annabel S, et al. "PRKACA: the catalytic subunit of protein kinase A and adrenocortical tumors." Frontiers in cell and developmental biology vol. 3 (2015): 26. doi: https://doi.org/10.3389/fcell.2015.00026
Berthon AS, Szarek E, Stratakis CA. PRKACA: the catalytic subunit of protein kinase A and adrenocortical tumors. Front Cell Dev Biol. 2015 May 20;3:26. doi: 10.3389/fcell.2015.00026. eCollection 2015. PMID: 26042218; PMCID: PMC4438593.
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Modification of taurine and hypotaurine uptake systems in cultured primary astrocytes by serum-free medium and dibutyryl cyclic AMP treatment.

International journal of developmental neuroscience : the official journal of the International Society for Developmental Neuroscience

Holopainen I.
PMID: 24874393
Int J Dev Neurosci. 1984;2(6):529-34. doi: 10.1016/0736-5748(84)90030-3.

The characteristics of taurine and hypotaurine uptake were studied during astrocyte maturation in cultures grown in normal, serum-containing medium or in serum-free medium in the presence of 0.1 mmol/l dibutyryl cyclic AMP (dBcAMP). The uptake of both amino acids...

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Holopainen I. Modification of taurine and hypotaurine uptake systems in cultured primary astrocytes by serum-free medium and dibutyryl cyclic AMP treatment. Int J Dev Neurosci. 1984;2(6):529-34doi: 10.1016/0736-5748(84)90030-3.
Holopainen, I. (1984). Modification of taurine and hypotaurine uptake systems in cultured primary astrocytes by serum-free medium and dibutyryl cyclic AMP treatment. International journal of developmental neuroscience : the official journal of the International Society for Developmental Neuroscience, 2(6), 529-34. https://doi.org/10.1016/0736-5748(84)90030-3
Holopainen, I. "Modification of taurine and hypotaurine uptake systems in cultured primary astrocytes by serum-free medium and dibutyryl cyclic AMP treatment." International journal of developmental neuroscience : the official journal of the International Society for Developmental Neuroscience vol. 2,6 (1984): 529-34. doi: https://doi.org/10.1016/0736-5748(84)90030-3
Holopainen I. Modification of taurine and hypotaurine uptake systems in cultured primary astrocytes by serum-free medium and dibutyryl cyclic AMP treatment. Int J Dev Neurosci. 1984;2(6):529-34. doi: 10.1016/0736-5748(84)90030-3. PMID: 24874393.
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MSK1 regulates transcriptional induction of Arc/Arg3.1 in response to neurotrophins.

FEBS open bio

Hunter CJ, Remenyi J, Correa SA, Privitera L, Reyskens KMSE, Martin KJ, Toth R, Frenguelli BG, Arthur JSC.
PMID: 28593137
FEBS Open Bio. 2017 May 11;7(6):821-834. doi: 10.1002/2211-5463.12232. eCollection 2017 Jun.

The immediate early gene activity-regulated cytoskeletal protein (Arc)/Arg3.1 and the neurotrophin brain-derived neurotrophic factor (BDNF) play important roles in synaptic plasticity and learning and memory in the mammalian brain. However, the mechanisms by which BDNF regulates the expression of...

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Hunter CJ, Remenyi J, Correa SA, et al. MSK1 regulates transcriptional induction of Arc/Arg3.1 in response to neurotrophins. FEBS Open Bio. 2017;7(6):821-834doi: 10.1002/2211-5463.12232.
Hunter, C. J., Remenyi, J., Correa, S. A., Privitera, L., Reyskens, K. M. S. E., Martin, K. J., Toth, R., Frenguelli, B. G., & Arthur, J. S. C. (2017). MSK1 regulates transcriptional induction of Arc/Arg3.1 in response to neurotrophins. FEBS open bio, 7(6), 821-834. https://doi.org/10.1002/2211-5463.12232
Hunter, Chris J, et al. "MSK1 regulates transcriptional induction of Arc/Arg3.1 in response to neurotrophins." FEBS open bio vol. 7,6 (2017): 821-834. doi: https://doi.org/10.1002/2211-5463.12232
Hunter CJ, Remenyi J, Correa SA, Privitera L, Reyskens KMSE, Martin KJ, Toth R, Frenguelli BG, Arthur JSC. MSK1 regulates transcriptional induction of Arc/Arg3.1 in response to neurotrophins. FEBS Open Bio. 2017 May 11;7(6):821-834. doi: 10.1002/2211-5463.12232. eCollection 2017 Jun. PMID: 28593137; PMCID: PMC5458472.
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Maternal effects of general and regional specificity on embryos of Drosophila melanogaster caused by dunce and rutabaga mutant combinations.

Roux's archives of developmental biology : the official organ of the EDBO

Bellen HJ, Kiger JA.
PMID: 28305785
Rouxs Arch Dev Biol. 1988 Aug;197(5):258-268. doi: 10.1007/BF00380019.

The developmental patterns of embryos produced by female germ line cells homozygous for null-enzyme mutations of dunce and for dunce in combination with each of three different rutabaga mutations are compared with the normal pattern. At least three discrete...

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Bellen HJ, Kiger JA. Maternal effects of general and regional specificity on embryos of Drosophila melanogaster caused by dunce and rutabaga mutant combinations. Rouxs Arch Dev Biol. 1988;197(5):258-268doi: 10.1007/BF00380019.
Bellen, H. J., & Kiger, J. A. (1988). Maternal effects of general and regional specificity on embryos of Drosophila melanogaster caused by dunce and rutabaga mutant combinations. Roux's archives of developmental biology : the official organ of the EDBO, 197(5), 258-268. https://doi.org/10.1007/BF00380019
Bellen, Hugo Jozef, and Kiger, John Andrew. "Maternal effects of general and regional specificity on embryos of Drosophila melanogaster caused by dunce and rutabaga mutant combinations." Roux's archives of developmental biology : the official organ of the EDBO vol. 197,5 (1988): 258-268. doi: https://doi.org/10.1007/BF00380019
Bellen HJ, Kiger JA. Maternal effects of general and regional specificity on embryos of Drosophila melanogaster caused by dunce and rutabaga mutant combinations. Rouxs Arch Dev Biol. 1988 Aug;197(5):258-268. doi: 10.1007/BF00380019. PMID: 28305785.
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Regulatory T-Cell-Mediated Suppression of Conventional T-Cells and Dendritic Cells by Different cAMP Intracellular Pathways.

Frontiers in immunology

Rueda CM, Jackson CM, Chougnet CA.
PMID: 27313580
Front Immunol. 2016 Jun 02;7:216. doi: 10.3389/fimmu.2016.00216. eCollection 2016.

Regulatory T-cells (Tregs) mediate their suppressive action by acting directly on conventional T-cells (Tcons) or dendritic cells (DCs). One mechanism of Treg suppression is the increase of cyclic adenosine 3',5'-monophosphate (cAMP) levels in target cells. Tregs utilize cAMP to...

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Rueda CM, Jackson CM, Chougnet CA. Regulatory T-Cell-Mediated Suppression of Conventional T-Cells and Dendritic Cells by Different cAMP Intracellular Pathways. Front Immunol. 2016;7:216doi: 10.3389/fimmu.2016.00216.
Rueda, C. M., Jackson, C. M., & Chougnet, C. A. (2016). Regulatory T-Cell-Mediated Suppression of Conventional T-Cells and Dendritic Cells by Different cAMP Intracellular Pathways. Frontiers in immunology, 7216. https://doi.org/10.3389/fimmu.2016.00216
Rueda, Cesar M, et al. "Regulatory T-Cell-Mediated Suppression of Conventional T-Cells and Dendritic Cells by Different cAMP Intracellular Pathways." Frontiers in immunology vol. 7 (2016): 216. doi: https://doi.org/10.3389/fimmu.2016.00216
Rueda CM, Jackson CM, Chougnet CA. Regulatory T-Cell-Mediated Suppression of Conventional T-Cells and Dendritic Cells by Different cAMP Intracellular Pathways. Front Immunol. 2016 Jun 02;7:216. doi: 10.3389/fimmu.2016.00216. eCollection 2016. PMID: 27313580; PMCID: PMC4889573.
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