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Endothelial Antioxidant-1: a Key Mediator of Copper-dependent Wound Healing in vivo.

Scientific reports

Das A, Sudhahar V, Chen GF, Kim HW, Youn SW, Finney L, Vogt S, Yang J, Kweon J, Surenkhuu B, Ushio-Fukai M, Fukai T.
PMID: 27666810
Sci Rep. 2016 Sep 26;6:33783. doi: 10.1038/srep33783.

Copper (Cu), an essential nutrient, promotes wound healing, however, target of Cu action and underlying mechanisms remain elusive. Cu chaperone Antioxidant-1 (Atox1) in the cytosol supplies Cu to the secretory enzymes such as lysyl oxidase (LOX), while Atox1 in...

Cite
Das A, Sudhahar V, Chen GF, et al. Endothelial Antioxidant-1: a Key Mediator of Copper-dependent Wound Healing in vivo. Sci Rep. 2016;6:33783doi: 10.1038/srep33783.
Das, A., Sudhahar, V., Chen, G. F., Kim, H. W., Youn, S. W., Finney, L., Vogt, S., Yang, J., Kweon, J., Surenkhuu, B., Ushio-Fukai, M., & Fukai, T. (2016). Endothelial Antioxidant-1: a Key Mediator of Copper-dependent Wound Healing in vivo. Scientific reports, 633783. https://doi.org/10.1038/srep33783
Das, Archita, et al. "Endothelial Antioxidant-1: a Key Mediator of Copper-dependent Wound Healing in vivo." Scientific reports vol. 6 (2016): 33783. doi: https://doi.org/10.1038/srep33783
Das A, Sudhahar V, Chen GF, Kim HW, Youn SW, Finney L, Vogt S, Yang J, Kweon J, Surenkhuu B, Ushio-Fukai M, Fukai T. Endothelial Antioxidant-1: a Key Mediator of Copper-dependent Wound Healing in vivo. Sci Rep. 2016 Sep 26;6:33783. doi: 10.1038/srep33783. PMID: 27666810; PMCID: PMC5036036.
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Mouse Specific Cleavage-Resistant RAGE Splice Variant.

PloS one

Fukai T.
PMID: 27653772
PLoS One. 2016 Sep 21;11(9):e0162120. doi: 10.1371/journal.pone.0162120. eCollection 2016.

No abstract available.

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Fukai T. Mouse Specific Cleavage-Resistant RAGE Splice Variant. PLoS One. 2016;11(9):e0162120doi: 10.1371/journal.pone.0162120.
Fukai, T. (2016). Mouse Specific Cleavage-Resistant RAGE Splice Variant. PloS one, 11(9), e0162120. https://doi.org/10.1371/journal.pone.0162120
Fukai, Tohru. "Mouse Specific Cleavage-Resistant RAGE Splice Variant." PloS one vol. 11,9 (2016): e0162120. doi: https://doi.org/10.1371/journal.pone.0162120
Fukai T. Mouse Specific Cleavage-Resistant RAGE Splice Variant. PLoS One. 2016 Sep 21;11(9):e0162120. doi: 10.1371/journal.pone.0162120. eCollection 2016. PMID: 27653772; PMCID: PMC5031468.
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Modification of Cardiac Progenitor Cell-Derived Exosomes by miR-322 Provides Protection against Myocardial Infarction through Nox2-Dependent Angiogenesis.

Antioxidants (Basel, Switzerland)

Youn SW, Li Y, Kim YM, Sudhahar V, Abdelsaid K, Kim HW, Liu Y, Fulton DJR, Ashraf M, Tang Y, Fukai T, Ushio-Fukai M.
PMID: 30634641
Antioxidants (Basel). 2019 Jan 10;8(1). doi: 10.3390/antiox8010018.

Myocardial infarction (MI) is the primary cause of cardiovascular mortality, and therapeutic strategies to prevent or mitigate the consequences of MI are a high priority. Cardiac progenitor cells (CPCs) have been used to treat cardiac injury post-MI, and despite...

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Youn SW, Li Y, Kim YM, et al. Modification of Cardiac Progenitor Cell-Derived Exosomes by miR-322 Provides Protection against Myocardial Infarction through Nox2-Dependent Angiogenesis. Antioxidants (Basel). 2019;8(1)doi: 10.3390/antiox8010018.
Youn, S. W., Li, Y., Kim, Y. M., Sudhahar, V., Abdelsaid, K., Kim, H. W., Liu, Y., Fulton, D. J. R., Ashraf, M., Tang, Y., Fukai, T., & Ushio-Fukai, M. (2019). Modification of Cardiac Progenitor Cell-Derived Exosomes by miR-322 Provides Protection against Myocardial Infarction through Nox2-Dependent Angiogenesis. Antioxidants (Basel, Switzerland), 8(1), . https://doi.org/10.3390/antiox8010018
Youn, Seock-Won, et al. "Modification of Cardiac Progenitor Cell-Derived Exosomes by miR-322 Provides Protection against Myocardial Infarction through Nox2-Dependent Angiogenesis." Antioxidants (Basel, Switzerland) vol. 8,1 (2019). doi: https://doi.org/10.3390/antiox8010018
Youn SW, Li Y, Kim YM, Sudhahar V, Abdelsaid K, Kim HW, Liu Y, Fulton DJR, Ashraf M, Tang Y, Fukai T, Ushio-Fukai M. Modification of Cardiac Progenitor Cell-Derived Exosomes by miR-322 Provides Protection against Myocardial Infarction through Nox2-Dependent Angiogenesis. Antioxidants (Basel). 2019 Jan 10;8(1). doi: 10.3390/antiox8010018. PMID: 30634641; PMCID: PMC6356993.
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Interplay Between Reactive Oxygen/Reactive Nitrogen Species and Metabolism in Vascular Biology and Disease.

Antioxidants & redox signaling

Ushio-Fukai M, Ash D, Nagarkoti S, Belin de Chantemèle EJ, Fulton DJR, Fukai T.
PMID: 33899493
Antioxid Redox Signal. 2021 Jun 01;34(16):1319-1354. doi: 10.1089/ars.2020.8161.

Reactive oxygen species (ROS;

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Ushio-Fukai M, Ash D, Nagarkoti S, et al. Interplay Between Reactive Oxygen/Reactive Nitrogen Species and Metabolism in Vascular Biology and Disease. Antioxid Redox Signal. 2021;34(16):1319-1354doi: 10.1089/ars.2020.8161.
Ushio-Fukai, M., Ash, D., Nagarkoti, S., Belin de Chantemèle, E. J., Fulton, D. J. R., & Fukai, T. (2021). Interplay Between Reactive Oxygen/Reactive Nitrogen Species and Metabolism in Vascular Biology and Disease. Antioxidants & redox signaling, 34(16), 1319-1354. https://doi.org/10.1089/ars.2020.8161
Ushio-Fukai, Masuko, et al. "Interplay Between Reactive Oxygen/Reactive Nitrogen Species and Metabolism in Vascular Biology and Disease." Antioxidants & redox signaling vol. 34,16 (2021): 1319-1354. doi: https://doi.org/10.1089/ars.2020.8161
Ushio-Fukai M, Ash D, Nagarkoti S, Belin de Chantemèle EJ, Fulton DJR, Fukai T. Interplay Between Reactive Oxygen/Reactive Nitrogen Species and Metabolism in Vascular Biology and Disease. Antioxid Redox Signal. 2021 Jun 01;34(16):1319-1354. doi: 10.1089/ars.2020.8161. PMID: 33899493; PMCID: PMC8418449.
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Showing 1 to 4 of 4 entries