Advanced Search
Display options
Filter resources
Text Availability
Article type
Publication date
Species
Language
Sex
Age
Showing 1 to 12 of 17 entries
Sorted by: Best Match Show Resources per page
Functional aspects of 5-hydroxytryptamine in early embryogenesis of the sea urchinParacentrotus lividus.

Wilhelm Roux's archives of developmental biology

Toneby M.
PMID: 28305144
Wilehm Roux Arch Dev Biol. 1977 Sep;181(3):247-259. doi: 10.1007/BF00848424.

5-Hydroxytryptamine concentrations have been determined in developing sea urchin embryos. Analysis was performed by thin layer chromatography and by fluorimetry. Good correspondence of the results between the two methods was observed. 5-HT was found in the larvae from 33...

Cite
Toneby M. Functional aspects of 5-hydroxytryptamine in early embryogenesis of the sea urchinParacentrotus lividus. Wilehm Roux Arch Dev Biol. 1977;181(3):247-259doi: 10.1007/BF00848424.
Toneby, M. (1977). Functional aspects of 5-hydroxytryptamine in early embryogenesis of the sea urchinParacentrotus lividus. Wilhelm Roux's archives of developmental biology, 181(3), 247-259. https://doi.org/10.1007/BF00848424
Toneby, Mark. "Functional aspects of 5-hydroxytryptamine in early embryogenesis of the sea urchinParacentrotus lividus." Wilhelm Roux's archives of developmental biology vol. 181,3 (1977): 247-259. doi: https://doi.org/10.1007/BF00848424
Toneby M. Functional aspects of 5-hydroxytryptamine in early embryogenesis of the sea urchinParacentrotus lividus. Wilehm Roux Arch Dev Biol. 1977 Sep;181(3):247-259. doi: 10.1007/BF00848424. PMID: 28305144.
Copy Download .nbib Format: NLM
Polyamines and nucleic acid metabolism in chick embryo. Incorporation of labelled precursors into nucleic acids of subcellular fractions and polyribosomal patterns.

The Biochemical journal

Moruzzi G, Barbiroli B, Caldarera CM.
PMID: 16742581
Biochem J. 1968 May;107(5):609-13. doi: 10.1042/bj1070609.

1. An increase in polyamine concentration, caused by inhibiting the amine oxidase activities with iproniazid, increased the incorporation of [(3)H]orotic acid into chick-embryo RNA and DNA. On the other hand, a decrease in polyamine concentration, obtained by causing an...

Cite
Moruzzi G, Barbiroli B, Caldarera CM. Polyamines and nucleic acid metabolism in chick embryo. Incorporation of labelled precursors into nucleic acids of subcellular fractions and polyribosomal patterns. Biochem J. 1968;107(5):609-13doi: 10.1042/bj1070609.
Moruzzi, G., Barbiroli, B., & Caldarera, C. M. (1968). Polyamines and nucleic acid metabolism in chick embryo. Incorporation of labelled precursors into nucleic acids of subcellular fractions and polyribosomal patterns. The Biochemical journal, 107(5), 609-13. https://doi.org/10.1042/bj1070609
Moruzzi, G, et al. "Polyamines and nucleic acid metabolism in chick embryo. Incorporation of labelled precursors into nucleic acids of subcellular fractions and polyribosomal patterns." The Biochemical journal vol. 107,5 (1968): 609-13. doi: https://doi.org/10.1042/bj1070609
Moruzzi G, Barbiroli B, Caldarera CM. Polyamines and nucleic acid metabolism in chick embryo. Incorporation of labelled precursors into nucleic acids of subcellular fractions and polyribosomal patterns. Biochem J. 1968 May;107(5):609-13. doi: 10.1042/bj1070609. PMID: 16742581; PMCID: PMC1198712.
Copy Download .nbib Format: NLM
Iproniazid in the Treatment of Depressive Syndromes.

Canadian Medical Association journal

Tyndel M.
PMID: 20326181
Can Med Assoc J. 1960 Mar 12;82(11):601.

No abstract available.

Cite
Tyndel M. Iproniazid in the Treatment of Depressive Syndromes. Can Med Assoc J. 1960;82(11):601
Tyndel, M. (1960). Iproniazid in the Treatment of Depressive Syndromes. Canadian Medical Association journal, 82(11), 601.
Tyndel, M. "Iproniazid in the Treatment of Depressive Syndromes." Canadian Medical Association journal vol. 82,11 (1960): 601.
Tyndel M. Iproniazid in the Treatment of Depressive Syndromes. Can Med Assoc J. 1960 Mar 12;82(11):601. PMID: 20326181; PMCID: PMC1937901.
Copy Download .nbib Format: NLM
Paroxetine Administration Affects Microbiota and Bile Acid Levels in Mice.

Frontiers in psychiatry

Dethloff F, Vargas F, Elijah E, Quinn R, Park DI, Herzog DP, Müller MB, Gentry EC, Knight R, Gonzalez A, Dorrestein PC, Turck CW.
PMID: 32581888
Front Psychiatry. 2020 Jun 04;11:518. doi: 10.3389/fpsyt.2020.00518. eCollection 2020.

Recent interest in the role of microbiota in health and disease has implicated gut microbiota dysbiosis in psychiatric disorders including major depressive disorder. Several antidepressant drugs that belong to the class of selective serotonin reuptake inhibitors have been found...

Cite
Dethloff F, Vargas F, Elijah E, et al. Paroxetine Administration Affects Microbiota and Bile Acid Levels in Mice. Front Psychiatry. 2020;11:518doi: 10.3389/fpsyt.2020.00518.
Dethloff, F., Vargas, F., Elijah, E., Quinn, R., Park, D. I., Herzog, D. P., Müller, M. B., Gentry, E. C., Knight, R., Gonzalez, A., Dorrestein, P. C., & Turck, C. W. (2020). Paroxetine Administration Affects Microbiota and Bile Acid Levels in Mice. Frontiers in psychiatry, 11518. https://doi.org/10.3389/fpsyt.2020.00518
Dethloff, Frederik, et al. "Paroxetine Administration Affects Microbiota and Bile Acid Levels in Mice." Frontiers in psychiatry vol. 11 (2020): 518. doi: https://doi.org/10.3389/fpsyt.2020.00518
Dethloff F, Vargas F, Elijah E, Quinn R, Park DI, Herzog DP, Müller MB, Gentry EC, Knight R, Gonzalez A, Dorrestein PC, Turck CW. Paroxetine Administration Affects Microbiota and Bile Acid Levels in Mice. Front Psychiatry. 2020 Jun 04;11:518. doi: 10.3389/fpsyt.2020.00518. eCollection 2020. PMID: 32581888; PMCID: PMC7287167.
Copy Download .nbib Format: NLM
A Proteotranscriptomic-Based Computational Drug-Repositioning Method for Alzheimer's Disease.

Frontiers in pharmacology

Lee SY, Song MY, Kim D, Park C, Park DK, Kim DG, Yoo JS, Kim YH.
PMID: 32063857
Front Pharmacol. 2020 Jan 29;10:1653. doi: 10.3389/fphar.2019.01653. eCollection 2019.

Numerous clinical trials of drug candidates for Alzheimer's disease (AD) have failed, and computational drug repositioning approaches using omics data have been proposed as effective alternative approaches to the discovery of drug candidates. However, little multi-omics data is available...

Cite
Lee SY, Song MY, Kim D, et al. A Proteotranscriptomic-Based Computational Drug-Repositioning Method for Alzheimer's Disease. Front Pharmacol. 2020;10:1653doi: 10.3389/fphar.2019.01653.
Lee, S. Y., Song, M. Y., Kim, D., Park, C., Park, D. K., Kim, D. G., Yoo, J. S., & Kim, Y. H. (2019). A Proteotranscriptomic-Based Computational Drug-Repositioning Method for Alzheimer's Disease. Frontiers in pharmacology, 101653. https://doi.org/10.3389/fphar.2019.01653
Lee, Soo Youn, et al. "A Proteotranscriptomic-Based Computational Drug-Repositioning Method for Alzheimer's Disease." Frontiers in pharmacology vol. 10 (2019): 1653. doi: https://doi.org/10.3389/fphar.2019.01653
Lee SY, Song MY, Kim D, Park C, Park DK, Kim DG, Yoo JS, Kim YH. A Proteotranscriptomic-Based Computational Drug-Repositioning Method for Alzheimer's Disease. Front Pharmacol. 2020 Jan 29;10:1653. doi: 10.3389/fphar.2019.01653. eCollection 2019. PMID: 32063857; PMCID: PMC7000455.
Copy Download .nbib Format: NLM
Minimizing Polymorphic Risk through Cooperative Computational and Experimental Exploration.

Journal of the American Chemical Society

Taylor CR, Mulvee MT, Perenyi DS, Probert MR, Day GM, Steed JW.
PMID: 32897065
J Am Chem Soc. 2020 Sep 30;142(39):16668-16680. doi: 10.1021/jacs.0c06749. Epub 2020 Sep 17.

We combine state-of-the-art computational crystal structure prediction (CSP) techniques with a wide range of experimental crystallization methods to understand and explore crystal structure in pharmaceuticals and minimize the risk of unanticipated late-appearing polymorphs. Initially, we demonstrate the power of...

Cite
Taylor CR, Mulvee MT, Perenyi DS, et al. Minimizing Polymorphic Risk through Cooperative Computational and Experimental Exploration. J Am Chem Soc. 2020;142(39):16668-16680doi: 10.1021/jacs.0c06749.
Taylor, C. R., Mulvee, M. T., Perenyi, D. S., Probert, M. R., Day, G. M., & Steed, J. W. (2020). Minimizing Polymorphic Risk through Cooperative Computational and Experimental Exploration. Journal of the American Chemical Society, 142(39), 16668-16680. https://doi.org/10.1021/jacs.0c06749
Taylor, Christopher R, et al. "Minimizing Polymorphic Risk through Cooperative Computational and Experimental Exploration." Journal of the American Chemical Society vol. 142,39 (2020): 16668-16680. doi: https://doi.org/10.1021/jacs.0c06749
Taylor CR, Mulvee MT, Perenyi DS, Probert MR, Day GM, Steed JW. Minimizing Polymorphic Risk through Cooperative Computational and Experimental Exploration. J Am Chem Soc. 2020 Sep 30;142(39):16668-16680. doi: 10.1021/jacs.0c06749. Epub 2020 Sep 17. PMID: 32897065; PMCID: PMC7586337.
Copy Download .nbib Format: NLM
The 5-hydroxytryptamine content of mouse brain and whole mice after treatment with some drugs affecting the central nervous system.

British journal of pharmacology and chemotherapy

Bartlet AL.
PMID: 19108131
Br J Pharmacol Chemother. 1960 Mar;15(1):140-6. doi: 10.1111/j.1476-5381.1960.tb01222.x.

A number of drugs were examined for their ability to change the concentration of 5-hydroxytryptamine in mouse brain and in whole mice treated with 5-hydroxytryptophan. After beta-phenylisopropylhydrazine or iproniazid, two inhibitors of monoamine oxidase, the brain 5-hydroxytryptamine rose to...

Cite
Bartlet AL. The 5-hydroxytryptamine content of mouse brain and whole mice after treatment with some drugs affecting the central nervous system. Br J Pharmacol Chemother. 1960;15(1):140-6doi: 10.1111/j.1476-5381.1960.tb01222.x.
Bartlet, A. L. (1960). The 5-hydroxytryptamine content of mouse brain and whole mice after treatment with some drugs affecting the central nervous system. British journal of pharmacology and chemotherapy, 15(1), 140-6. https://doi.org/10.1111/j.1476-5381.1960.tb01222.x
Bartlet, A L. "The 5-hydroxytryptamine content of mouse brain and whole mice after treatment with some drugs affecting the central nervous system." British journal of pharmacology and chemotherapy vol. 15,1 (1960): 140-6. doi: https://doi.org/10.1111/j.1476-5381.1960.tb01222.x
Bartlet AL. The 5-hydroxytryptamine content of mouse brain and whole mice after treatment with some drugs affecting the central nervous system. Br J Pharmacol Chemother. 1960 Mar;15(1):140-6. doi: 10.1111/j.1476-5381.1960.tb01222.x. PMID: 19108131; PMCID: PMC1481981.
Copy Download .nbib Format: NLM
Xanthoangelol and 4-Hydroxyderricin Are the Major Active Principles of the Inhibitory Activities against Monoamine Oxidases on Angelica keiskei K.

Biomolecules & therapeutics

Kim JH, Son YK, Kim GH, Hwang KH.
PMID: 24265870
Biomol Ther (Seoul). 2013 May 30;21(3):234-40. doi: 10.4062/biomolther.2012.100.

Monoamine oxidase inhibitors (MAOI) have been widely used as antidepressants. Recently, there has been renewed interest in MAO inhibitors. The activity-guided fractionation of extracts from Angelica keiskei Koidzumi (A. keiskei K.) led to the isolation of two prenylated chalcones,...

Cite
Kim JH, Son YK, Kim GH, et al. Xanthoangelol and 4-Hydroxyderricin Are the Major Active Principles of the Inhibitory Activities against Monoamine Oxidases on Angelica keiskei K. Biomol Ther (Seoul). 2013;21(3):234-40doi: 10.4062/biomolther.2012.100.
Kim, J. H., Son, Y. K., Kim, G. H., & Hwang, K. H. (2013). Xanthoangelol and 4-Hydroxyderricin Are the Major Active Principles of the Inhibitory Activities against Monoamine Oxidases on Angelica keiskei K. Biomolecules & therapeutics, 21(3), 234-40. https://doi.org/10.4062/biomolther.2012.100
Kim, Ji Ho, et al. "Xanthoangelol and 4-Hydroxyderricin Are the Major Active Principles of the Inhibitory Activities against Monoamine Oxidases on Angelica keiskei K." Biomolecules & therapeutics vol. 21,3 (2013): 234-40. doi: https://doi.org/10.4062/biomolther.2012.100
Kim JH, Son YK, Kim GH, Hwang KH. Xanthoangelol and 4-Hydroxyderricin Are the Major Active Principles of the Inhibitory Activities against Monoamine Oxidases on Angelica keiskei K. Biomol Ther (Seoul). 2013 May 30;21(3):234-40. doi: 10.4062/biomolther.2012.100. PMID: 24265870; PMCID: PMC3830123.
Copy Download .nbib Format: NLM
New antidepressant drugs for new depressions.

Psychiatria Danubina

Juli A, Juli L.
PMID: 25413524
Psychiatr Danub. 2014 Nov;26:115-6.

Although depressive disorders have been known for centuries, drug treatment is relatively recent. It was only in the 1950s, when studyof a drug for tuberculosis, Iproniazid, that its antidepressant propertieswere identified. After 50 years more selective and specific drugs...

Cite
Juli A, Juli L. New antidepressant drugs for new depressions. Psychiatr Danub. 2014;26:115-6
Juli, A., & Juli, L. (2014). New antidepressant drugs for new depressions. Psychiatria Danubina, 26115-6.
Juli, Alfredo, and Juli, Luigi. "New antidepressant drugs for new depressions." Psychiatria Danubina vol. 26 (2014): 115-6.
Juli A, Juli L. New antidepressant drugs for new depressions. Psychiatr Danub. 2014 Nov;26:115-6. PMID: 25413524.
Copy Download .nbib Format: NLM
Hepatic Necrosis Due to Marsilid.

Gut

McCarthy CF, Sheridan MR.
PMID: 18668732
Gut. 1960 Jun;1(2):169-70. doi: 10.1136/gut.1.2.169.

In the patient reported here severe jaundice and death resulted from treatment with iproniazide ("marsilid") given for only three days.

Cite
McCarthy CF, Sheridan MR. Hepatic Necrosis Due to Marsilid. Gut. 1960;1(2):169-70doi: 10.1136/gut.1.2.169.
McCarthy, C. F., & Sheridan, M. R. (1960). Hepatic Necrosis Due to Marsilid. Gut, 1(2), 169-70. https://doi.org/10.1136/gut.1.2.169
McCarthy, C F, and Sheridan, M R. "Hepatic Necrosis Due to Marsilid." Gut vol. 1,2 (1960): 169-70. doi: https://doi.org/10.1136/gut.1.2.169
McCarthy CF, Sheridan MR. Hepatic Necrosis Due to Marsilid. Gut. 1960 Jun;1(2):169-70. doi: 10.1136/gut.1.2.169. PMID: 18668732; PMCID: PMC1413210.
Copy Download .nbib Format: NLM
Pioneers in Psychopharmacology.

The international journal of neuropsychopharmacology

Healy D.
PMID: 11281963
Int J Neuropsychopharmacol. 1998 Dec;1(2):191-194. doi: 10.1017/S146114579800114X.

In pursuing the history of any field, even one in which many of the main exponents are still alive, it can be very difficult to establish facts and priorities. Detailed scrutiny of the events leading to the recognition of...

Cite
Healy D. Pioneers in Psychopharmacology. Int J Neuropsychopharmacol. 1998;1(2):191-194doi: 10.1017/S146114579800114X.
Healy, D. (1998). Pioneers in Psychopharmacology. The international journal of neuropsychopharmacology, 1(2), 191-194. https://doi.org/10.1017/S146114579800114X
Healy, David. "Pioneers in Psychopharmacology." The international journal of neuropsychopharmacology vol. 1,2 (1998): 191-194. doi: https://doi.org/10.1017/S146114579800114X
Healy D. Pioneers in Psychopharmacology. Int J Neuropsychopharmacol. 1998 Dec;1(2):191-194. doi: 10.1017/S146114579800114X. PMID: 11281963.
Copy Download .nbib Format: NLM
The effect of antidepressive drugs and some related compounds on the levels of adenine nucleotides, inorganic phosphate and phosphocreatine in the rat brain.

British journal of pharmacology and chemotherapy

Lewis JJ, Van Petten GR.
PMID: 19108178
Br J Pharmacol Chemother. 1963 Jun;20(3):462-70. doi: 10.1111/j.1476-5381.1963.tb01483.x.

The effects upon levels of adenine nucleotides, phosphocreatine and inorganic phosphate of iproniazid, isoniazid, phenelzine, pheniprazine, tranylcypromine, harmine, imipramine, amitriptyline, orphenadrine, diphenhydramine and cocaine have been studied. With the exception of harmine and diphenhydramine, each of these compounds increased...

Cite
Lewis JJ, Van Petten GR. The effect of antidepressive drugs and some related compounds on the levels of adenine nucleotides, inorganic phosphate and phosphocreatine in the rat brain. Br J Pharmacol Chemother. 1963;20(3):462-70doi: 10.1111/j.1476-5381.1963.tb01483.x.
Lewis, J. J., & Van Petten, G. R. (1963). The effect of antidepressive drugs and some related compounds on the levels of adenine nucleotides, inorganic phosphate and phosphocreatine in the rat brain. British journal of pharmacology and chemotherapy, 20(3), 462-70. https://doi.org/10.1111/j.1476-5381.1963.tb01483.x
Lewis, J J, and Van Petten, G R. "The effect of antidepressive drugs and some related compounds on the levels of adenine nucleotides, inorganic phosphate and phosphocreatine in the rat brain." British journal of pharmacology and chemotherapy vol. 20,3 (1963): 462-70. doi: https://doi.org/10.1111/j.1476-5381.1963.tb01483.x
Lewis JJ, Van Petten GR. The effect of antidepressive drugs and some related compounds on the levels of adenine nucleotides, inorganic phosphate and phosphocreatine in the rat brain. Br J Pharmacol Chemother. 1963 Jun;20(3):462-70. doi: 10.1111/j.1476-5381.1963.tb01483.x. PMID: 19108178; PMCID: PMC1703802.
Copy Download .nbib Format: NLM
Showing 1 to 12 of 17 entries