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Frontiers in physiology

Dimitrova A, Walko M, Hashemi Shabestari M, Kumar P, Huber M, Kocer A.
PMID: 27708587
Front Physiol. 2016 Sep 21;7:409. doi: 10.3389/fphys.2016.00409. eCollection 2016.

Understanding the functioning of ion channels, as well as utilizing their properties for biochemical applications requires control over channel activity. Herein we report a reversible control over the functioning of a mechanosensitive ion channel by interfering with its interaction...

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Dimitrova A, Walko M, Hashemi Shabestari M, et al. . Front Physiol. 2016;7:409doi: 10.3389/fphys.2016.00409.
Dimitrova, A., Walko, M., Hashemi Shabestari, M., Kumar, P., Huber, M., & Kocer, A. (2016). . Frontiers in physiology, 7409. https://doi.org/10.3389/fphys.2016.00409
Dimitrova, Anna, et al. "." Frontiers in physiology vol. 7 (2016): 409. doi: https://doi.org/10.3389/fphys.2016.00409
Dimitrova A, Walko M, Hashemi Shabestari M, Kumar P, Huber M, Kocer A. . Front Physiol. 2016 Sep 21;7:409. doi: 10.3389/fphys.2016.00409. eCollection 2016. PMID: 27708587; PMCID: PMC5030285.
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Metabolomic signatures associated with disease severity in multiple sclerosis.

Neurology(R) neuroimmunology & neuroinflammation

Villoslada P, Alonso C, Agirrezabal I, Kotelnikova E, Zubizarreta I, Pulido-Valdeolivas I, Saiz A, Comabella M, Montalban X, Villar L, Alvarez-Cermeño JC, Fernández O, Alvarez-Lafuente R, Arroyo R, Castro A.
PMID: 28180139
Neurol Neuroimmunol Neuroinflamm. 2017 Jan 27;4(2):e321. doi: 10.1212/NXI.0000000000000321. eCollection 2017 Mar.

OBJECTIVE: To identify differences in the metabolomic profile in the serum of patients with multiple sclerosis (MS) compared to controls and to identify biomarkers of disease severity.METHODS: We studied 2 cohorts of patients with MS: a retrospective longitudinal cohort...

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Villoslada P, Alonso C, Agirrezabal I, et al. Metabolomic signatures associated with disease severity in multiple sclerosis. Neurol Neuroimmunol Neuroinflamm. 2017;4(2):e321doi: 10.1212/NXI.0000000000000321.
Villoslada, P., Alonso, C., Agirrezabal, I., Kotelnikova, E., Zubizarreta, I., Pulido-Valdeolivas, I., Saiz, A., Comabella, M., Montalban, X., Villar, L., Alvarez-Cermeño, J. C., Fernández, O., Alvarez-Lafuente, R., Arroyo, R., & Castro, A. (2017). Metabolomic signatures associated with disease severity in multiple sclerosis. Neurology(R) neuroimmunology & neuroinflammation, 4(2), e321. https://doi.org/10.1212/NXI.0000000000000321
Villoslada, Pablo, et al. "Metabolomic signatures associated with disease severity in multiple sclerosis." Neurology(R) neuroimmunology & neuroinflammation vol. 4,2 (2017): e321. doi: https://doi.org/10.1212/NXI.0000000000000321
Villoslada P, Alonso C, Agirrezabal I, Kotelnikova E, Zubizarreta I, Pulido-Valdeolivas I, Saiz A, Comabella M, Montalban X, Villar L, Alvarez-Cermeño JC, Fernández O, Alvarez-Lafuente R, Arroyo R, Castro A. Metabolomic signatures associated with disease severity in multiple sclerosis. Neurol Neuroimmunol Neuroinflamm. 2017 Jan 27;4(2):e321. doi: 10.1212/NXI.0000000000000321. eCollection 2017 Mar. PMID: 28180139; PMCID: PMC5278923.
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Comparative metabolomic analysis of HPAC cells following the acquisition of erlotinib resistance.

Oncology letters

Lee S, Jang WJ, Choi B, Joo SH, Jeong CH.
PMID: 28529573
Oncol Lett. 2017 May;13(5):3437-3444. doi: 10.3892/ol.2017.5940. Epub 2017 Mar 28.

Pancreatic cancer is one of the most lethal types of cancer, due to difficulty in early detection and the limited efficacy of available treatments. Erlotinib is used to inhibit the epidermal growth factor receptor for the treatment of pancreatic...

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Lee S, Jang WJ, Choi B, et al. Comparative metabolomic analysis of HPAC cells following the acquisition of erlotinib resistance. Oncol Lett. 2017;13(5):3437-3444doi: 10.3892/ol.2017.5940.
Lee, S., Jang, W. J., Choi, B., Joo, S. H., & Jeong, C. H. (2017). Comparative metabolomic analysis of HPAC cells following the acquisition of erlotinib resistance. Oncology letters, 13(5), 3437-3444. https://doi.org/10.3892/ol.2017.5940
Lee, Sooyeun, et al. "Comparative metabolomic analysis of HPAC cells following the acquisition of erlotinib resistance." Oncology letters vol. 13,5 (2017): 3437-3444. doi: https://doi.org/10.3892/ol.2017.5940
Lee S, Jang WJ, Choi B, Joo SH, Jeong CH. Comparative metabolomic analysis of HPAC cells following the acquisition of erlotinib resistance. Oncol Lett. 2017 May;13(5):3437-3444. doi: 10.3892/ol.2017.5940. Epub 2017 Mar 28. PMID: 28529573; PMCID: PMC5431587.
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Advantages of total lipid hydrogenation prior to lipid class determination on chromarods-SIII.

Lipids

Shantha NC, Ackman RG.
PMID: 27520799
Lipids. 1990 Sep;25(9):570-4. doi: 10.1007/BF02537167.

The changes in detector responses of lipid classes for standards and natural lipid samples after hydrogenation were investigated with the TLC/FID Iatroscan system using Chromarods-SIII (Newman-Howells Associates, Ltd., Midwales, U.K.). Samples included lipids of human plasma and erythrocytes, egg...

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Shantha NC, Ackman RG. Advantages of total lipid hydrogenation prior to lipid class determination on chromarods-SIII. Lipids. 1990;25(9):570-4doi: 10.1007/BF02537167.
Shantha, N. C., & Ackman, R. G. (1990). Advantages of total lipid hydrogenation prior to lipid class determination on chromarods-SIII. Lipids, 25(9), 570-4. https://doi.org/10.1007/BF02537167
Shantha, N C, and Ackman, R G. "Advantages of total lipid hydrogenation prior to lipid class determination on chromarods-SIII." Lipids vol. 25,9 (1990): 570-4. doi: https://doi.org/10.1007/BF02537167
Shantha NC, Ackman RG. Advantages of total lipid hydrogenation prior to lipid class determination on chromarods-SIII. Lipids. 1990 Sep;25(9):570-4. doi: 10.1007/BF02537167. PMID: 27520799.
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LC-MS-based metabolomic analysis of serum and livers from red ginseng-fed rats.

Journal of ginseng research

Kim HJ, Cho CW, Hwang JT, Son N, Choi JH, Shim GS, Han CK.
PMID: 24198664
J Ginseng Res. 2013 Jul;37(3):371-8. doi: 10.5142/jgr.2013.37.371.

Serum and liver metabolites in rats fed red ginseng (RG) were analyzed by ultra-performance liquid chromatography-quadrupole time-of-flight mass spectrometry. The mass data were analyzed by partial least squares-discriminant analysis (PLS-DA) to discriminate between control and RG groups and identify...

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Kim HJ, Cho CW, Hwang JT, et al. LC-MS-based metabolomic analysis of serum and livers from red ginseng-fed rats. J Ginseng Res. 2013;37(3):371-8doi: 10.5142/jgr.2013.37.371.
Kim, H. J., Cho, C. W., Hwang, J. T., Son, N., Choi, J. H., Shim, G. S., & Han, C. K. (2013). LC-MS-based metabolomic analysis of serum and livers from red ginseng-fed rats. Journal of ginseng research, 37(3), 371-8. https://doi.org/10.5142/jgr.2013.37.371
Kim, Hyun-Jin, et al. "LC-MS-based metabolomic analysis of serum and livers from red ginseng-fed rats." Journal of ginseng research vol. 37,3 (2013): 371-8. doi: https://doi.org/10.5142/jgr.2013.37.371
Kim HJ, Cho CW, Hwang JT, Son N, Choi JH, Shim GS, Han CK. LC-MS-based metabolomic analysis of serum and livers from red ginseng-fed rats. J Ginseng Res. 2013 Jul;37(3):371-8. doi: 10.5142/jgr.2013.37.371. PMID: 24198664; PMCID: PMC3818965.
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In vivo isotopically labeled atherosclerotic aorta plaques in ApoE KO mice and molecular profiling by matrix-assisted laser desorption/ionization mass spectrometric imaging.

Rapid communications in mass spectrometry : RCM

Castro-Perez J, Hatcher N, Kofi Karikari N, Wang SP, Mendoza V, Shion H, Millar A, Shockcor J, Towers M, McLaren D, Shah V, Previs S, Akinsanya K, Cleary M, Roddy TP, Johns DG.
PMID: 25303476
Rapid Commun Mass Spectrom. 2014 Nov 30;28(22):2471-9. doi: 10.1002/rcm.7039.

RATIONALE: The ability to quantify rates of formation, regression and/or remodeling of atherosclerotic plaque should facilitate a better understanding of the pathogenesis and management of cardiovascular disease. In the current study, we coupled a stable isotope labeled tracer protocol...

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Castro-Perez J, Hatcher N, Kofi Karikari N, et al. In vivo isotopically labeled atherosclerotic aorta plaques in ApoE KO mice and molecular profiling by matrix-assisted laser desorption/ionization mass spectrometric imaging. Rapid Commun Mass Spectrom. 2014;28(22):2471-9doi: 10.1002/rcm.7039.
Castro-Perez, J., Hatcher, N., Kofi Karikari, N., Wang, S. P., Mendoza, V., Shion, H., Millar, A., Shockcor, J., Towers, M., McLaren, D., Shah, V., Previs, S., Akinsanya, K., Cleary, M., Roddy, T. P., & Johns, D. G. (2014). In vivo isotopically labeled atherosclerotic aorta plaques in ApoE KO mice and molecular profiling by matrix-assisted laser desorption/ionization mass spectrometric imaging. Rapid communications in mass spectrometry : RCM, 28(22), 2471-9. https://doi.org/10.1002/rcm.7039
Castro-Perez, Jose, et al. "In vivo isotopically labeled atherosclerotic aorta plaques in ApoE KO mice and molecular profiling by matrix-assisted laser desorption/ionization mass spectrometric imaging." Rapid communications in mass spectrometry : RCM vol. 28,22 (2014): 2471-9. doi: https://doi.org/10.1002/rcm.7039
Castro-Perez J, Hatcher N, Kofi Karikari N, Wang SP, Mendoza V, Shion H, Millar A, Shockcor J, Towers M, McLaren D, Shah V, Previs S, Akinsanya K, Cleary M, Roddy TP, Johns DG. In vivo isotopically labeled atherosclerotic aorta plaques in ApoE KO mice and molecular profiling by matrix-assisted laser desorption/ionization mass spectrometric imaging. Rapid Commun Mass Spectrom. 2014 Nov 30;28(22):2471-9. doi: 10.1002/rcm.7039. PMID: 25303476.
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UPLC-QTOF/MS-Based Lipidomic Profiling of Liver Qi-Stagnation and Spleen-Deficiency Syndrome in Patients with Hyperlipidemia.

Evidence-based complementary and alternative medicine : eCAM

Shenghua P, Shuyu T, Kunping L, Huixia Z, Xue X, Jiao G.
PMID: 30245731
Evid Based Complement Alternat Med. 2018 Aug 30;2018:4530849. doi: 10.1155/2018/4530849. eCollection 2018.

Hyperlipidemia is a common disease caused by abnormal plasma lipid metabolism. Lipidomics is a powerful and efficient technology to study the integration of disease and syndrome of Chinese medicine. This study investigated specific changes in lipid metabolites from hyperlipidemia...

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Shenghua P, Shuyu T, Kunping L, et al. UPLC-QTOF/MS-Based Lipidomic Profiling of Liver Qi-Stagnation and Spleen-Deficiency Syndrome in Patients with Hyperlipidemia. Evid Based Complement Alternat Med. 2018;2018:4530849doi: 10.1155/2018/4530849.
Shenghua, P., Shuyu, T., Kunping, L., Huixia, Z., Xue, X., & Jiao, G. (2018). UPLC-QTOF/MS-Based Lipidomic Profiling of Liver Qi-Stagnation and Spleen-Deficiency Syndrome in Patients with Hyperlipidemia. Evidence-based complementary and alternative medicine : eCAM, 20184530849. https://doi.org/10.1155/2018/4530849
Shenghua, Piao, et al. "UPLC-QTOF/MS-Based Lipidomic Profiling of Liver Qi-Stagnation and Spleen-Deficiency Syndrome in Patients with Hyperlipidemia." Evidence-based complementary and alternative medicine : eCAM vol. 2018 (2018): 4530849. doi: https://doi.org/10.1155/2018/4530849
Shenghua P, Shuyu T, Kunping L, Huixia Z, Xue X, Jiao G. UPLC-QTOF/MS-Based Lipidomic Profiling of Liver Qi-Stagnation and Spleen-Deficiency Syndrome in Patients with Hyperlipidemia. Evid Based Complement Alternat Med. 2018 Aug 30;2018:4530849. doi: 10.1155/2018/4530849. eCollection 2018. PMID: 30245731; PMCID: PMC6136559.
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The Systemic Alterations of Lipids, Alanine-Glucose Cycle and Inter-Organ Amino Acid Metabolism in Swine Model Confirms the Role of Liver in Early Phase of Septic Shock.

Frontiers in physiology

Ferrario M, Brunelli L, Su F, Herpain A, Pastorelli R.
PMID: 30745875
Front Physiol. 2019 Jan 28;10:11. doi: 10.3389/fphys.2019.00011. eCollection 2019.

Septic shock is a medical emergency and is one of the main causes of mortality in critically ill patients. Given the pathophysiological complexity of sepsis spectrum and progression in clinical settings, animal models become essential tools to improve patient...

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Ferrario M, Brunelli L, Su F, et al. The Systemic Alterations of Lipids, Alanine-Glucose Cycle and Inter-Organ Amino Acid Metabolism in Swine Model Confirms the Role of Liver in Early Phase of Septic Shock. Front Physiol. 2019;10:11doi: 10.3389/fphys.2019.00011.
Ferrario, M., Brunelli, L., Su, F., Herpain, A., & Pastorelli, R. (2019). The Systemic Alterations of Lipids, Alanine-Glucose Cycle and Inter-Organ Amino Acid Metabolism in Swine Model Confirms the Role of Liver in Early Phase of Septic Shock. Frontiers in physiology, 1011. https://doi.org/10.3389/fphys.2019.00011
Ferrario, Manuela, et al. "The Systemic Alterations of Lipids, Alanine-Glucose Cycle and Inter-Organ Amino Acid Metabolism in Swine Model Confirms the Role of Liver in Early Phase of Septic Shock." Frontiers in physiology vol. 10 (2019): 11. doi: https://doi.org/10.3389/fphys.2019.00011
Ferrario M, Brunelli L, Su F, Herpain A, Pastorelli R. The Systemic Alterations of Lipids, Alanine-Glucose Cycle and Inter-Organ Amino Acid Metabolism in Swine Model Confirms the Role of Liver in Early Phase of Septic Shock. Front Physiol. 2019 Jan 28;10:11. doi: 10.3389/fphys.2019.00011. eCollection 2019. PMID: 30745875; PMCID: PMC6360162.
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Changes in Circulating Metabolites During Weight Loss are Associated with Adiposity Improvement, and Body Weight and Adiposity Regain During Weight Loss Maintenance: The SATIN Study.

Molecular nutrition & food research

Papandreou C, García-Gavilán J, Camacho-Barcia L, Toft Hansen T, Harrold JA, Sjödin A, Halford JCG, Bulló M.
PMID: 34184401
Mol Nutr Food Res. 2021 Sep;65(17):e2001154. doi: 10.1002/mnfr.202001154. Epub 2021 Jul 21.

SCOPE: To examine the relationship between changes in circulating metabolites during diet-induced weight loss and changes of adiposity. This study also investigates changes in these metabolites in relation to body weight and adiposity regain during a weight loss maintenance...

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Papandreou C, García-Gavilán J, Camacho-Barcia L, et al. Changes in Circulating Metabolites During Weight Loss are Associated with Adiposity Improvement, and Body Weight and Adiposity Regain During Weight Loss Maintenance: The SATIN Study. Mol Nutr Food Res. 2021;65(17):e2001154doi: 10.1002/mnfr.202001154.
Papandreou, C., García-Gavilán, J., Camacho-Barcia, L., Toft Hansen, T., Harrold, J. A., Sjödin, A., Halford, J. C. G., & Bulló, M. (2021). Changes in Circulating Metabolites During Weight Loss are Associated with Adiposity Improvement, and Body Weight and Adiposity Regain During Weight Loss Maintenance: The SATIN Study. Molecular nutrition & food research, 65(17), e2001154. https://doi.org/10.1002/mnfr.202001154
Papandreou, Christopher, et al. "Changes in Circulating Metabolites During Weight Loss are Associated with Adiposity Improvement, and Body Weight and Adiposity Regain During Weight Loss Maintenance: The SATIN Study." Molecular nutrition & food research vol. 65,17 (2021): e2001154. doi: https://doi.org/10.1002/mnfr.202001154
Papandreou C, García-Gavilán J, Camacho-Barcia L, Toft Hansen T, Harrold JA, Sjödin A, Halford JCG, Bulló M. Changes in Circulating Metabolites During Weight Loss are Associated with Adiposity Improvement, and Body Weight and Adiposity Regain During Weight Loss Maintenance: The SATIN Study. Mol Nutr Food Res. 2021 Sep;65(17):e2001154. doi: 10.1002/mnfr.202001154. Epub 2021 Jul 21. PMID: 34184401.
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Pharmacometabolomic profiles in type 2 diabetic subjects treated with liraglutide or glimepiride.

Cardiovascular diabetology

Jendle J, Hyötyläinen T, Orešič M, Nyström T.
PMID: 34920733
Cardiovasc Diabetol. 2021 Dec 17;20(1):237. doi: 10.1186/s12933-021-01431-2.

BACKGROUND: Treatment with glucagon-like peptide-1 receptor agonists (GLP-1 RAs) leads to multiple metabolic changes, reduction in glucose levels and body weight are well established. In people with type 2 diabetes, GLP-1 RAs reduce the risk of cardiovascular (CV) disease...

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Jendle J, Hyötyläinen T, Orešič M, et al. Pharmacometabolomic profiles in type 2 diabetic subjects treated with liraglutide or glimepiride. Cardiovasc Diabetol. 2021;20(1):237doi: 10.1186/s12933-021-01431-2.
Jendle, J., Hyötyläinen, T., Orešič, M., & Nyström, T. (2021). Pharmacometabolomic profiles in type 2 diabetic subjects treated with liraglutide or glimepiride. Cardiovascular diabetology, 20(1), 237. https://doi.org/10.1186/s12933-021-01431-2
Jendle, J, et al. "Pharmacometabolomic profiles in type 2 diabetic subjects treated with liraglutide or glimepiride." Cardiovascular diabetology vol. 20,1 (2021): 237. doi: https://doi.org/10.1186/s12933-021-01431-2
Jendle J, Hyötyläinen T, Orešič M, Nyström T. Pharmacometabolomic profiles in type 2 diabetic subjects treated with liraglutide or glimepiride. Cardiovasc Diabetol. 2021 Dec 17;20(1):237. doi: 10.1186/s12933-021-01431-2. PMID: 34920733; PMCID: PMC8684205.
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Age and sex dependent changes of free circulating blood metabolite and lipid abundances, correlations and ratios.

The journals of gerontology. Series A, Biological sciences and medical sciences

Di Cesare F, Luchinat C, Tenori L, Saccenti E.
PMID: 34748631
J Gerontol A Biol Sci Med Sci. 2021 Nov 08; doi: 10.1093/gerona/glab335. Epub 2021 Nov 08.

In this study we investigated how the concentrations, pairwise correlations, and ratios of 202 free circulating blood metabolites and lipids vary with age in a panel of n=1882 subjects ranging from 48 to 94 years. We report a statistically...

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Di Cesare F, Luchinat C, Tenori L, et al. Age and sex dependent changes of free circulating blood metabolite and lipid abundances, correlations and ratios. J Gerontol A Biol Sci Med Sci. 2021;doi: 10.1093/gerona/glab335.
Di Cesare, F., Luchinat, C., Tenori, L., & Saccenti, E. (2021). Age and sex dependent changes of free circulating blood metabolite and lipid abundances, correlations and ratios. The journals of gerontology. Series A, Biological sciences and medical sciences, . https://doi.org/10.1093/gerona/glab335
Di Cesare, Francesca, et al. "Age and sex dependent changes of free circulating blood metabolite and lipid abundances, correlations and ratios." The journals of gerontology. Series A, Biological sciences and medical sciences vol. (2021). doi: https://doi.org/10.1093/gerona/glab335
Di Cesare F, Luchinat C, Tenori L, Saccenti E. Age and sex dependent changes of free circulating blood metabolite and lipid abundances, correlations and ratios. J Gerontol A Biol Sci Med Sci. 2021 Nov 08; doi: 10.1093/gerona/glab335. Epub 2021 Nov 08. PMID: 34748631.
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Epigenomics and Lipidomics Integration in Alzheimer Disease: Pathways Involved in Early Stages.

Biomedicines

Peña-Bautista C, Álvarez-Sánchez L, Cañada-Martínez AJ, Baquero M, Cháfer-Pericás C.
PMID: 34944628
Biomedicines. 2021 Dec 02;9(12). doi: 10.3390/biomedicines9121812.

BACKGROUND: Alzheimer Disease (AD) is the most prevalent dementia. However, the physiopathological mechanisms involved in its development are unclear. In this sense, a multi-omics approach could provide some progress.METHODS: Epigenomic and lipidomic analysis were carried out in plasma samples...

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Peña-Bautista C, Álvarez-Sánchez L, Cañada-Martínez AJ, et al. Epigenomics and Lipidomics Integration in Alzheimer Disease: Pathways Involved in Early Stages. Biomedicines. 2021;9(12)doi: 10.3390/biomedicines9121812.
Peña-Bautista, C., Álvarez-Sánchez, L., Cañada-Martínez, A. J., Baquero, M., & Cháfer-Pericás, C. (2021). Epigenomics and Lipidomics Integration in Alzheimer Disease: Pathways Involved in Early Stages. Biomedicines, 9(12), . https://doi.org/10.3390/biomedicines9121812
Peña-Bautista, Carmen, et al. "Epigenomics and Lipidomics Integration in Alzheimer Disease: Pathways Involved in Early Stages." Biomedicines vol. 9,12 (2021). doi: https://doi.org/10.3390/biomedicines9121812
Peña-Bautista C, Álvarez-Sánchez L, Cañada-Martínez AJ, Baquero M, Cháfer-Pericás C. Epigenomics and Lipidomics Integration in Alzheimer Disease: Pathways Involved in Early Stages. Biomedicines. 2021 Dec 02;9(12). doi: 10.3390/biomedicines9121812. PMID: 34944628; PMCID: PMC8698767.
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