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Correction to: The Mitochondria-Targeted Methylglyoxal Sequestering Compound, MitoGamide, Is Cardioprotective in the Diabetic Heart.

Cardiovascular drugs and therapy

Tate M, Higgins GC, De Blasio MJ, Lindblom R, Prakoso D, Deo M, Kiriazis H, Park M, Baeza-Garza CD, Caldwell ST, Hartley RC, Krieg T, Murphy MP, Coughlan MT, Ritchie RH.
PMID: 32062792
Cardiovasc Drugs Ther. 2020 Apr;34(2):223. doi: 10.1007/s10557-019-06929-2.

The article "The Mitochondria-Targeted Methylglyoxal Sequestering Compound, MitoGamide, Is Cardioprotective in the Diabetic Heart".

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Tate M, Higgins GC, De Blasio MJ, et al. Correction to: The Mitochondria-Targeted Methylglyoxal Sequestering Compound, MitoGamide, Is Cardioprotective in the Diabetic Heart. Cardiovasc Drugs Ther. 2020;34(2):223doi: 10.1007/s10557-019-06929-2.
Tate, M., Higgins, G. C., De Blasio, M. J., Lindblom, R., Prakoso, D., Deo, M., Kiriazis, H., Park, M., Baeza-Garza, C. D., Caldwell, S. T., Hartley, R. C., Krieg, T., Murphy, M. P., Coughlan, M. T., & Ritchie, R. H. (2020). Correction to: The Mitochondria-Targeted Methylglyoxal Sequestering Compound, MitoGamide, Is Cardioprotective in the Diabetic Heart. Cardiovascular drugs and therapy, 34(2), 223. https://doi.org/10.1007/s10557-019-06929-2
Tate, Mitchel, et al. "Correction to: The Mitochondria-Targeted Methylglyoxal Sequestering Compound, MitoGamide, Is Cardioprotective in the Diabetic Heart." Cardiovascular drugs and therapy vol. 34,2 (2020): 223. doi: https://doi.org/10.1007/s10557-019-06929-2
Tate M, Higgins GC, De Blasio MJ, Lindblom R, Prakoso D, Deo M, Kiriazis H, Park M, Baeza-Garza CD, Caldwell ST, Hartley RC, Krieg T, Murphy MP, Coughlan MT, Ritchie RH. Correction to: The Mitochondria-Targeted Methylglyoxal Sequestering Compound, MitoGamide, Is Cardioprotective in the Diabetic Heart. Cardiovasc Drugs Ther. 2020 Apr;34(2):223. doi: 10.1007/s10557-019-06929-2. PMID: 32062792; PMCID: PMC7645541.
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Enhancing the Mitochondrial Uptake of Phosphonium Cations by Carboxylic Acid Incorporation.

Frontiers in chemistry

Pala L, Senn HM, Caldwell ST, Prime TA, Warrington S, Bright TP, Prag HA, Wilson C, Murphy MP, Hartley RC.
PMID: 33033715
Front Chem. 2020 Sep 09;8:783. doi: 10.3389/fchem.2020.00783. eCollection 2020.

There is considerable interest in developing drugs and probes targeted to mitochondria in order to understand and treat the many pathologies associated with mitochondrial dysfunction. The large membrane potential, negative inside, across the mitochondrial inner membrane enables delivery of...

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Pala L, Senn HM, Caldwell ST, et al. Enhancing the Mitochondrial Uptake of Phosphonium Cations by Carboxylic Acid Incorporation. Front Chem. 2020;8:783doi: 10.3389/fchem.2020.00783.
Pala, L., Senn, H. M., Caldwell, S. T., Prime, T. A., Warrington, S., Bright, T. P., Prag, H. A., Wilson, C., Murphy, M. P., & Hartley, R. C. (2020). Enhancing the Mitochondrial Uptake of Phosphonium Cations by Carboxylic Acid Incorporation. Frontiers in chemistry, 8783. https://doi.org/10.3389/fchem.2020.00783
Pala, Laura, et al. "Enhancing the Mitochondrial Uptake of Phosphonium Cations by Carboxylic Acid Incorporation." Frontiers in chemistry vol. 8 (2020): 783. doi: https://doi.org/10.3389/fchem.2020.00783
Pala L, Senn HM, Caldwell ST, Prime TA, Warrington S, Bright TP, Prag HA, Wilson C, Murphy MP, Hartley RC. Enhancing the Mitochondrial Uptake of Phosphonium Cations by Carboxylic Acid Incorporation. Front Chem. 2020 Sep 09;8:783. doi: 10.3389/fchem.2020.00783. eCollection 2020. PMID: 33033715; PMCID: PMC7509049.
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Translatable mitochondria-targeted protection against programmed cardiovascular dysfunction.

Science advances

Botting KJ, Skeffington KL, Niu Y, Allison BJ, Brain KL, Itani N, Beck C, Logan A, Murray AJ, Murphy MP, Giussani DA.
PMID: 32875110
Sci Adv. 2020 Aug 19;6(34):eabb1929. doi: 10.1126/sciadv.abb1929. eCollection 2020 Aug.

The prenatal origins of heart disease in offspring have been established. However, research in species with developmental milestones comparable to humans is lacking, preventing translation of this knowledge to clinical contexts. Using sheep and chickens, two species with similar...

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Botting KJ, Skeffington KL, Niu Y, et al. Translatable mitochondria-targeted protection against programmed cardiovascular dysfunction. Sci Adv. 2020;6(34):eabb1929doi: 10.1126/sciadv.abb1929.
Botting, K. J., Skeffington, K. L., Niu, Y., Allison, B. J., Brain, K. L., Itani, N., Beck, C., Logan, A., Murray, A. J., Murphy, M. P., & Giussani, D. A. (2020). Translatable mitochondria-targeted protection against programmed cardiovascular dysfunction. Science advances, 6(34), eabb1929. https://doi.org/10.1126/sciadv.abb1929
Botting, K J, et al. "Translatable mitochondria-targeted protection against programmed cardiovascular dysfunction." Science advances vol. 6,34 (2020): eabb1929. doi: https://doi.org/10.1126/sciadv.abb1929
Botting KJ, Skeffington KL, Niu Y, Allison BJ, Brain KL, Itani N, Beck C, Logan A, Murray AJ, Murphy MP, Giussani DA. Translatable mitochondria-targeted protection against programmed cardiovascular dysfunction. Sci Adv. 2020 Aug 19;6(34):eabb1929. doi: 10.1126/sciadv.abb1929. eCollection 2020 Aug. PMID: 32875110; PMCID: PMC7438086.
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Mechanism of succinate efflux upon reperfusion of the ischaemic heart.

Cardiovascular research

Prag HA, Gruszczyk AV, Huang MM, Beach TE, Young T, Tronci L, Nikitopoulou E, Mulvey JF, Ascione R, Hadjihambi A, Shattock MJ, Pellerin L, Saeb-Parsy K, Frezza C, James AM, Krieg T, Murphy MP, Aksentijević D.
PMID: 32766828
Cardiovasc Res. 2021 Mar 21;117(4):1188-1201. doi: 10.1093/cvr/cvaa148.

AIMS: Succinate accumulates several-fold in the ischaemic heart and is then rapidly oxidized upon reperfusion, contributing to reactive oxygen species production by mitochondria. In addition, a significant amount of the accumulated succinate is released from the heart into the...

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Prag HA, Gruszczyk AV, Huang MM, et al. Mechanism of succinate efflux upon reperfusion of the ischaemic heart. Cardiovasc Res. 2021;117(4):1188-1201doi: 10.1093/cvr/cvaa148.
Prag, H. A., Gruszczyk, A. V., Huang, M. M., Beach, T. E., Young, T., Tronci, L., Nikitopoulou, E., Mulvey, J. F., Ascione, R., Hadjihambi, A., Shattock, M. J., Pellerin, L., Saeb-Parsy, K., Frezza, C., James, A. M., Krieg, T., Murphy, M. P., & Aksentijević, D. (2021). Mechanism of succinate efflux upon reperfusion of the ischaemic heart. Cardiovascular research, 117(4), 1188-1201. https://doi.org/10.1093/cvr/cvaa148
Prag, Hiran A, et al. "Mechanism of succinate efflux upon reperfusion of the ischaemic heart." Cardiovascular research vol. 117,4 (2021): 1188-1201. doi: https://doi.org/10.1093/cvr/cvaa148
Prag HA, Gruszczyk AV, Huang MM, Beach TE, Young T, Tronci L, Nikitopoulou E, Mulvey JF, Ascione R, Hadjihambi A, Shattock MJ, Pellerin L, Saeb-Parsy K, Frezza C, James AM, Krieg T, Murphy MP, Aksentijević D. Mechanism of succinate efflux upon reperfusion of the ischaemic heart. Cardiovasc Res. 2021 Mar 21;117(4):1188-1201. doi: 10.1093/cvr/cvaa148. PMID: 32766828; PMCID: PMC7983001.
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Impact of the mitochondria-targeted antioxidant MitoQ on hypoxia-induced pulmonary hypertension.

The European respiratory journal

Pak O, Scheibe S, Esfandiary A, Gierhardt M, Sydykov A, Logan A, Fysikopoulos A, Veit F, Hecker M, Kroschel F, Quanz K, Erb A, Schäfer K, Fassbinder M, Alebrahimdehkordi N, Ghofrani HA, Schermuly RT, Brandes RP, Seeger W, Murphy MP, Weissmann N, Sommer N.
PMID: 29419444
Eur Respir J. 2018 Feb 01; doi: 10.1183/13993003.01024-2017. Epub 2018 Feb 01.

Increased mitochondrial reactive oxygen species (ROS), particularly superoxide have been suggested to mediate hypoxic pulmonary vasoconstriction (HPV), chronic hypoxia-induced pulmonary hypertension (PH) and right ventricular (RV) remodelling.We determined ROS in acute, chronic hypoxia and investigated the effect of the...

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Pak O, Scheibe S, Esfandiary A, et al. Impact of the mitochondria-targeted antioxidant MitoQ on hypoxia-induced pulmonary hypertension. Eur Respir J. 2018;doi: 10.1183/13993003.01024-2017.
Pak, O., Scheibe, S., Esfandiary, A., Gierhardt, M., Sydykov, A., Logan, A., Fysikopoulos, A., Veit, F., Hecker, M., Kroschel, F., Quanz, K., Erb, A., Schäfer, K., Fassbinder, M., Alebrahimdehkordi, N., Ghofrani, H. A., Schermuly, R. T., Brandes, R. P., Seeger, W., Murphy, M. P., Weissmann, N., & Sommer, N. (2018). Impact of the mitochondria-targeted antioxidant MitoQ on hypoxia-induced pulmonary hypertension. The European respiratory journal, . https://doi.org/10.1183/13993003.01024-2017
Pak, Oleg, et al. "Impact of the mitochondria-targeted antioxidant MitoQ on hypoxia-induced pulmonary hypertension." The European respiratory journal vol. (2018). doi: https://doi.org/10.1183/13993003.01024-2017
Pak O, Scheibe S, Esfandiary A, Gierhardt M, Sydykov A, Logan A, Fysikopoulos A, Veit F, Hecker M, Kroschel F, Quanz K, Erb A, Schäfer K, Fassbinder M, Alebrahimdehkordi N, Ghofrani HA, Schermuly RT, Brandes RP, Seeger W, Murphy MP, Weissmann N, Sommer N. Impact of the mitochondria-targeted antioxidant MitoQ on hypoxia-induced pulmonary hypertension. Eur Respir J. 2018 Feb 01; doi: 10.1183/13993003.01024-2017. Epub 2018 Feb 01. PMID: 29419444.
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Showing 1 to 5 of 5 entries