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Co-Administration of Molecular Adjuvants Expressing NF-Kappa B Subunit p65/RelA or Type-1 Transactivator T-bet Enhance Antigen Specific DNA Vaccine-Induced Immunity.

Vaccines

Shedlock DJ, Tingey C, Mahadevan L, Hutnick N, Reuschel EL, Kudchodkar S, Flingai S, Yan J, Kim JJ, Ugen KE, Weiner DB, Muthumani K.
PMID: 26344618
Vaccines (Basel). 2014 Mar 25;2(2):196-215. doi: 10.3390/vaccines2020196.

DNA vaccine-induced immunity can be enhanced by the co-delivery of synthetic gene-encoding molecular adjuvants. Many of these adjuvants have included cytokines, chemokines or co-stimulatory molecules that have been demonstrated to enhance vaccine-induced immunity by increasing the magnitude or type...

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Shedlock DJ, Tingey C, Mahadevan L, et al. Co-Administration of Molecular Adjuvants Expressing NF-Kappa B Subunit p65/RelA or Type-1 Transactivator T-bet Enhance Antigen Specific DNA Vaccine-Induced Immunity. Vaccines (Basel). 2014;2(2):196-215doi: 10.3390/vaccines2020196.
Shedlock, D. J., Tingey, C., Mahadevan, L., Hutnick, N., Reuschel, E. L., Kudchodkar, S., Flingai, S., Yan, J., Kim, J. J., Ugen, K. E., Weiner, D. B., & Muthumani, K. (2014). Co-Administration of Molecular Adjuvants Expressing NF-Kappa B Subunit p65/RelA or Type-1 Transactivator T-bet Enhance Antigen Specific DNA Vaccine-Induced Immunity. Vaccines, 2(2), 196-215. https://doi.org/10.3390/vaccines2020196
Shedlock, Devon J, et al. "Co-Administration of Molecular Adjuvants Expressing NF-Kappa B Subunit p65/RelA or Type-1 Transactivator T-bet Enhance Antigen Specific DNA Vaccine-Induced Immunity." Vaccines vol. 2,2 (2014): 196-215. doi: https://doi.org/10.3390/vaccines2020196
Shedlock DJ, Tingey C, Mahadevan L, Hutnick N, Reuschel EL, Kudchodkar S, Flingai S, Yan J, Kim JJ, Ugen KE, Weiner DB, Muthumani K. Co-Administration of Molecular Adjuvants Expressing NF-Kappa B Subunit p65/RelA or Type-1 Transactivator T-bet Enhance Antigen Specific DNA Vaccine-Induced Immunity. Vaccines (Basel). 2014 Mar 25;2(2):196-215. doi: 10.3390/vaccines2020196. PMID: 26344618; PMCID: PMC4494262.
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PKCθ and HIV-1 Transcriptional Regulator Tat Co-exist at the LTR Promoter in CD4(+) T Cells.

Frontiers in immunology

López-Huertas MR, Li J, Zafar A, Rodríguez-Mora S, García-Domínguez C, Mateos E, Alcamí J, Rao S, Coiras M.
PMID: 26973648
Front Immunol. 2016 Feb 29;7:69. doi: 10.3389/fimmu.2016.00069. eCollection 2016.

PKCθ is essential for the activation of CD4(+) T cells. Upon TCR/CD28 stimulation, PKCθ is phosphorylated and migrates to the immunological synapse, inducing the activation of cellular transcription factors such as NF-κB and kinases as ERK that are critical...

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López-Huertas MR, Li J, Zafar A, et al. PKCθ and HIV-1 Transcriptional Regulator Tat Co-exist at the LTR Promoter in CD4(+) T Cells. Front Immunol. 2016;7:69doi: 10.3389/fimmu.2016.00069.
López-Huertas, M. R., Li, J., Zafar, A., Rodríguez-Mora, S., García-Domínguez, C., Mateos, E., Alcamí, J., Rao, S., & Coiras, M. (2016). PKCθ and HIV-1 Transcriptional Regulator Tat Co-exist at the LTR Promoter in CD4(+) T Cells. Frontiers in immunology, 769. https://doi.org/10.3389/fimmu.2016.00069
López-Huertas, María Rosa, et al. "PKCθ and HIV-1 Transcriptional Regulator Tat Co-exist at the LTR Promoter in CD4(+) T Cells." Frontiers in immunology vol. 7 (2016): 69. doi: https://doi.org/10.3389/fimmu.2016.00069
López-Huertas MR, Li J, Zafar A, Rodríguez-Mora S, García-Domínguez C, Mateos E, Alcamí J, Rao S, Coiras M. PKCθ and HIV-1 Transcriptional Regulator Tat Co-exist at the LTR Promoter in CD4(+) T Cells. Front Immunol. 2016 Feb 29;7:69. doi: 10.3389/fimmu.2016.00069. eCollection 2016. PMID: 26973648; PMCID: PMC4770193.
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Panax Notoginseng flower saponins (PNFS) inhibit LPS-stimulated NO overproduction and iNOS gene overexpression via the suppression of TLR4-mediated MAPK/NF-kappa B signaling pathways in RAW264.7 macrophages.

Chinese medicine

Peng XX, Zhang SH, Wang XL, Ye TJ, Li H, Yan XF, Wei L, Wu ZP, Hu J, Zou CP, Wang YH, Hu XD.
PMID: 26155304
Chin Med. 2015 Jul 01;10:15. doi: 10.1186/s13020-015-0045-x. eCollection 2015.

BACKGROUND: Panax Notoginseng flower saponins (PNFS) are the main active component of Panax notoginseng (Burk) F. H. Chen flower bud (PNF) and possess significant anti-inflammatory efficacy. This study aims to explore the mechanisms underlying PNFS' antiflammatory action in RAW264.7...

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Peng XX, Zhang SH, Wang XL, et al. Panax Notoginseng flower saponins (PNFS) inhibit LPS-stimulated NO overproduction and iNOS gene overexpression via the suppression of TLR4-mediated MAPK/NF-kappa B signaling pathways in RAW264.7 macrophages. Chin Med. 2015;10:15doi: 10.1186/s13020-015-0045-x.
Peng, X. X., Zhang, S. H., Wang, X. L., Ye, T. J., Li, H., Yan, X. F., Wei, L., Wu, Z. P., Hu, J., Zou, C. P., Wang, Y. H., & Hu, X. D. (2015). Panax Notoginseng flower saponins (PNFS) inhibit LPS-stimulated NO overproduction and iNOS gene overexpression via the suppression of TLR4-mediated MAPK/NF-kappa B signaling pathways in RAW264.7 macrophages. Chinese medicine, 1015. https://doi.org/10.1186/s13020-015-0045-x
Peng, Xiao-Xu, et al. "Panax Notoginseng flower saponins (PNFS) inhibit LPS-stimulated NO overproduction and iNOS gene overexpression via the suppression of TLR4-mediated MAPK/NF-kappa B signaling pathways in RAW264.7 macrophages." Chinese medicine vol. 10 (2015): 15. doi: https://doi.org/10.1186/s13020-015-0045-x
Peng XX, Zhang SH, Wang XL, Ye TJ, Li H, Yan XF, Wei L, Wu ZP, Hu J, Zou CP, Wang YH, Hu XD. Panax Notoginseng flower saponins (PNFS) inhibit LPS-stimulated NO overproduction and iNOS gene overexpression via the suppression of TLR4-mediated MAPK/NF-kappa B signaling pathways in RAW264.7 macrophages. Chin Med. 2015 Jul 01;10:15. doi: 10.1186/s13020-015-0045-x. eCollection 2015. PMID: 26155304; PMCID: PMC4493833.
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LL-37 induced cystitis and the receptor for advanced glycation end-products (RAGE) pathway.

Advances in bioscience and biotechnology (Print)

Roundy LM, Jia W, Zhang J, Ye X, Prestwich GD, Oottamasathien S.
PMID: 24883227
Adv Biosci Biotechnol. 2013 Aug 01;4(8):1-8. doi: 10.4236/abb.2013.48a2001.

To elucidate pathways in bladder inflammation, we employed our physiologically relevant LL-37 induced cystitis model. Based on inflammatory studies involving other organ systems implicating the receptor for advanced glycation end-products (RAGE), we first hypothesized that RAGE is critically involved...

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Roundy LM, Jia W, Zhang J, et al. LL-37 induced cystitis and the receptor for advanced glycation end-products (RAGE) pathway. Adv Biosci Biotechnol. 2013;4(8):1-8doi: 10.4236/abb.2013.48a2001.
Roundy, L. M., Jia, W., Zhang, J., Ye, X., Prestwich, G. D., & Oottamasathien, S. (2013). LL-37 induced cystitis and the receptor for advanced glycation end-products (RAGE) pathway. Advances in bioscience and biotechnology (Print), 4(8), 1-8. https://doi.org/10.4236/abb.2013.48a2001
Roundy, Lindsi McCoard, et al. "LL-37 induced cystitis and the receptor for advanced glycation end-products (RAGE) pathway." Advances in bioscience and biotechnology (Print) vol. 4,8 (2013): 1-8. doi: https://doi.org/10.4236/abb.2013.48a2001
Roundy LM, Jia W, Zhang J, Ye X, Prestwich GD, Oottamasathien S. LL-37 induced cystitis and the receptor for advanced glycation end-products (RAGE) pathway. Adv Biosci Biotechnol. 2013 Aug 01;4(8):1-8. doi: 10.4236/abb.2013.48a2001. PMID: 24883227; PMCID: PMC4039633.
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Asterias rubens: Evidence of NF-kappa B genes.

Meta gene

Leclerc M, Kresdorn N, Horres R.
PMID: 27014588
Meta Gene. 2016 Jan 05;8:30-2. doi: 10.1016/j.mgene.2015.12.006. eCollection 2016 Jun.

In the present paper we show a survey of the Asterias rubens sea star genome for genes associated with NF-kappa-B proteins implied in the immune response. The NF-kappa B gene, into 2 subunits, was found in this invertebrate.

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Leclerc M, Kresdorn N, Horres R. Asterias rubens: Evidence of NF-kappa B genes. Meta Gene. 2016;8:30-2doi: 10.1016/j.mgene.2015.12.006.
Leclerc, M., Kresdorn, N., & Horres, R. (2016). Asterias rubens: Evidence of NF-kappa B genes. Meta gene, 830-2. https://doi.org/10.1016/j.mgene.2015.12.006
Leclerc, Michel, et al. "Asterias rubens: Evidence of NF-kappa B genes." Meta gene vol. 8 (2016): 30-2. doi: https://doi.org/10.1016/j.mgene.2015.12.006
Leclerc M, Kresdorn N, Horres R. Asterias rubens: Evidence of NF-kappa B genes. Meta Gene. 2016 Jan 05;8:30-2. doi: 10.1016/j.mgene.2015.12.006. eCollection 2016 Jun. PMID: 27014588; PMCID: PMC4792854.
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Requirement of NF-kappa B Activation in Different Mice Brain Areas during Long-Term Memory Consolidation in Two Contextual One-Trial Tasks with Opposing Valences.

Frontiers in molecular neuroscience

Salles A, Krawczyk MDC, Blake M, Romano A, Boccia MM, Freudenthal R.
PMID: 28439227
Front Mol Neurosci. 2017 Apr 07;10:104. doi: 10.3389/fnmol.2017.00104. eCollection 2017.

NF-kappa B is a transcription factor whose activation has been shown to be necessary for long-term memory consolidation in several species. NF-kappa B is activated and translocates to the nucleus of cells in a specific temporal window during consolidation....

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Salles A, Krawczyk MDC, Blake M, et al. Requirement of NF-kappa B Activation in Different Mice Brain Areas during Long-Term Memory Consolidation in Two Contextual One-Trial Tasks with Opposing Valences. Front Mol Neurosci. 2017;10:104doi: 10.3389/fnmol.2017.00104.
Salles, A., Krawczyk, M. D. C., Blake, M., Romano, A., Boccia, M. M., & Freudenthal, R. (2017). Requirement of NF-kappa B Activation in Different Mice Brain Areas during Long-Term Memory Consolidation in Two Contextual One-Trial Tasks with Opposing Valences. Frontiers in molecular neuroscience, 10104. https://doi.org/10.3389/fnmol.2017.00104
Salles, Angeles, et al. "Requirement of NF-kappa B Activation in Different Mice Brain Areas during Long-Term Memory Consolidation in Two Contextual One-Trial Tasks with Opposing Valences." Frontiers in molecular neuroscience vol. 10 (2017): 104. doi: https://doi.org/10.3389/fnmol.2017.00104
Salles A, Krawczyk MDC, Blake M, Romano A, Boccia MM, Freudenthal R. Requirement of NF-kappa B Activation in Different Mice Brain Areas during Long-Term Memory Consolidation in Two Contextual One-Trial Tasks with Opposing Valences. Front Mol Neurosci. 2017 Apr 07;10:104. doi: 10.3389/fnmol.2017.00104. eCollection 2017. PMID: 28439227; PMCID: PMC5383659.
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Modulating the Innate Immune Response to Influenza A Virus: Potential Therapeutic Use of Anti-Inflammatory Drugs.

Frontiers in immunology

Ramos I, Fernandez-Sesma A.
PMID: 26257731
Front Immunol. 2015 Jul 20;6:361. doi: 10.3389/fimmu.2015.00361. eCollection 2015.

Infection by influenza A viruses (IAV) is frequently characterized by robust inflammation that is usually more pronounced in the case of avian influenza. It is becoming clearer that the morbidity and pathogenesis caused by IAV are consequences of this...

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Ramos I, Fernandez-Sesma A. Modulating the Innate Immune Response to Influenza A Virus: Potential Therapeutic Use of Anti-Inflammatory Drugs. Front Immunol. 2015;6:361doi: 10.3389/fimmu.2015.00361.
Ramos, I., & Fernandez-Sesma, A. (2015). Modulating the Innate Immune Response to Influenza A Virus: Potential Therapeutic Use of Anti-Inflammatory Drugs. Frontiers in immunology, 6361. https://doi.org/10.3389/fimmu.2015.00361
Ramos, Irene, and Fernandez-Sesma, Ana. "Modulating the Innate Immune Response to Influenza A Virus: Potential Therapeutic Use of Anti-Inflammatory Drugs." Frontiers in immunology vol. 6 (2015): 361. doi: https://doi.org/10.3389/fimmu.2015.00361
Ramos I, Fernandez-Sesma A. Modulating the Innate Immune Response to Influenza A Virus: Potential Therapeutic Use of Anti-Inflammatory Drugs. Front Immunol. 2015 Jul 20;6:361. doi: 10.3389/fimmu.2015.00361. eCollection 2015. PMID: 26257731; PMCID: PMC4507467.
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Contemplating the Importance of Toll-like Receptors I and II Regarding Human Viral Pathogenesis.

Jundishapur journal of microbiology

Saeed U, Mazoor S, Jalal N, Zahid Piracha Z.
PMID: 25763131
Jundishapur J Microbiol. 2014 Dec 06;8(1):e13348. doi: 10.5812/jjm.13348. eCollection 2015 Jan.

BACKGROUND: Toll-like receptors (TLRs) play a major role in innate immunity, since they detect conserved pathogen-associated molecular patterns (PAMPs) on a range of microbes, including viruses, leading to innate immune activation and orchestration of the adaptive immune response.OBJECTIVES: The...

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Saeed U, Mazoor S, Jalal N, et al. Contemplating the Importance of Toll-like Receptors I and II Regarding Human Viral Pathogenesis. Jundishapur J Microbiol. 2014;8(1):e13348doi: 10.5812/jjm.13348.
Saeed, U., Mazoor, S., Jalal, N., & Zahid Piracha, Z. (2015). Contemplating the Importance of Toll-like Receptors I and II Regarding Human Viral Pathogenesis. Jundishapur journal of microbiology, 8(1), e13348. https://doi.org/10.5812/jjm.13348
Saeed, Umar, et al. "Contemplating the Importance of Toll-like Receptors I and II Regarding Human Viral Pathogenesis." Jundishapur journal of microbiology vol. 8,1 (2015): e13348. doi: https://doi.org/10.5812/jjm.13348
Saeed U, Mazoor S, Jalal N, Zahid Piracha Z. Contemplating the Importance of Toll-like Receptors I and II Regarding Human Viral Pathogenesis. Jundishapur J Microbiol. 2014 Dec 06;8(1):e13348. doi: 10.5812/jjm.13348. eCollection 2015 Jan. PMID: 25763131; PMCID: PMC4344769.
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Inhibitory Effects of Antioxidants on NF-KappaB Activation in Bovine Carotid Artery Endothelial Cells.

Sheng wu hua xue yu sheng wu wu li xue bao Acta biochimica et biophysica Sinica

Zhao F, Murota SI, Morita I, Yao XS.
PMID: 12075419
Sheng Wu Hua Xue Yu Sheng Wu Wu Li Xue Bao (Shanghai). 2000;32(4):342-346.

NF-Kappa B is one of important transcription factor, which plays an important role in many immune and inflammatory responses. A large variety of stimulus can induce the degradation of IKappa B-alpha, an inhibitor of NF-KappaB, leading to the activation...

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Zhao F, Murota SI, Morita I, et al. Inhibitory Effects of Antioxidants on NF-KappaB Activation in Bovine Carotid Artery Endothelial Cells. Sheng Wu Hua Xue Yu Sheng Wu Wu Li Xue Bao (Shanghai). 2000;32(4):342-346
Zhao, F., Murota, S. I., Morita, I., & Yao, X. S. (2000). Inhibitory Effects of Antioxidants on NF-KappaB Activation in Bovine Carotid Artery Endothelial Cells. Sheng wu hua xue yu sheng wu wu li xue bao Acta biochimica et biophysica Sinica, 32(4), 342-346.
Zhao, Feng, et al. "Inhibitory Effects of Antioxidants on NF-KappaB Activation in Bovine Carotid Artery Endothelial Cells." Sheng wu hua xue yu sheng wu wu li xue bao Acta biochimica et biophysica Sinica vol. 32,4 (2000): 342-346.
Zhao F, Murota SI, Morita I, Yao XS. Inhibitory Effects of Antioxidants on NF-KappaB Activation in Bovine Carotid Artery Endothelial Cells. Sheng Wu Hua Xue Yu Sheng Wu Wu Li Xue Bao (Shanghai). 2000;32(4):342-346. PMID: 12075419.
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Expression of transcription factor c-Rel and apoptosis occurrence in polydactylous and syndactylous limb buds of the talpid3 mutant chick embryo.

Apoptosis : an international journal on programmed cell death

Huguet C, Morrice DR, Bouali F, Vandenbunder B, Burt DW, Abbadie C.
PMID: 14634293
Apoptosis. 1999 Feb;4(1):31-8. doi: 10.1023/a:1009678015249.

The chicken proto-oncogene c-rel encodes a transcription factor of the Rel/NF-kappa B family. We have previously shown that c-rel mRNAs accumulate in different types of apoptotic cells of the chick embryo, especially in mesenchymal cells within the four cell...

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Huguet C, Morrice DR, Bouali F, et al. Expression of transcription factor c-Rel and apoptosis occurrence in polydactylous and syndactylous limb buds of the talpid3 mutant chick embryo. Apoptosis. 1999;4(1):31-8doi: 10.1023/a:1009678015249.
Huguet, C., Morrice, D. R., Bouali, F., Vandenbunder, B., Burt, D. W., & Abbadie, C. (1999). Expression of transcription factor c-Rel and apoptosis occurrence in polydactylous and syndactylous limb buds of the talpid3 mutant chick embryo. Apoptosis : an international journal on programmed cell death, 4(1), 31-8. https://doi.org/10.1023/a:1009678015249
Huguet, C, et al. "Expression of transcription factor c-Rel and apoptosis occurrence in polydactylous and syndactylous limb buds of the talpid3 mutant chick embryo." Apoptosis : an international journal on programmed cell death vol. 4,1 (1999): 31-8. doi: https://doi.org/10.1023/a:1009678015249
Huguet C, Morrice DR, Bouali F, Vandenbunder B, Burt DW, Abbadie C. Expression of transcription factor c-Rel and apoptosis occurrence in polydactylous and syndactylous limb buds of the talpid3 mutant chick embryo. Apoptosis. 1999 Feb;4(1):31-8. doi: 10.1023/a:1009678015249. PMID: 14634293.
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NF- kappa B and chemoresistance: potentiation of cancer drugs via inhibition of NF- kappa B.

Drug resistance updates : reviews and commentaries in antimicrobial and anticancer chemotherapy

Cusack JC, Liu R, Baldwin AS.
PMID: 11504498
Drug Resist Updat. 1999 Aug;2(4):271-273. doi: 10.1054/drup.1999.0094.

No abstract available.

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Cusack JC, Liu R, Baldwin AS. NF- kappa B and chemoresistance: potentiation of cancer drugs via inhibition of NF- kappa B. Drug Resist Updat. 1999;2(4):271-273doi: 10.1054/drup.1999.0094.
Cusack, J. C., Liu, R., & Baldwin, A. S. (1999). NF- kappa B and chemoresistance: potentiation of cancer drugs via inhibition of NF- kappa B. Drug resistance updates : reviews and commentaries in antimicrobial and anticancer chemotherapy, 2(4), 271-273. https://doi.org/10.1054/drup.1999.0094
Cusack, James C., et al. "NF- kappa B and chemoresistance: potentiation of cancer drugs via inhibition of NF- kappa B." Drug resistance updates : reviews and commentaries in antimicrobial and anticancer chemotherapy vol. 2,4 (1999): 271-273. doi: https://doi.org/10.1054/drup.1999.0094
Cusack JC, Liu R, Baldwin AS. NF- kappa B and chemoresistance: potentiation of cancer drugs via inhibition of NF- kappa B. Drug Resist Updat. 1999 Aug;2(4):271-273. doi: 10.1054/drup.1999.0094. PMID: 11504498.
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Proteins controlling the nuclear uptake of NF-kappa B, Rel and dorsal.

Trends in cell biology

Schmitz ML, Henkel T, Baeuerle PA.
PMID: 14731544
Trends Cell Biol. 1991 Nov;1(5):130-7. doi: 10.1016/0962-8924(91)90118-s.

The two DNA-binding subunits of the transcription factor NF-kappa B, the products of the rel oncogene family and the product of the developmental control gene dorsal of Drosophila are homologous within a 300 amino acid region. This sequence represents...

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Schmitz ML, Henkel T, Baeuerle PA. Proteins controlling the nuclear uptake of NF-kappa B, Rel and dorsal. Trends Cell Biol. 1991;1(5):130-7doi: 10.1016/0962-8924(91)90118-s.
Schmitz, M. L., Henkel, T., & Baeuerle, P. A. (1991). Proteins controlling the nuclear uptake of NF-kappa B, Rel and dorsal. Trends in cell biology, 1(5), 130-7. https://doi.org/10.1016/0962-8924(91)90118-s
Schmitz, M L, et al. "Proteins controlling the nuclear uptake of NF-kappa B, Rel and dorsal." Trends in cell biology vol. 1,5 (1991): 130-7. doi: https://doi.org/10.1016/0962-8924(91)90118-s
Schmitz ML, Henkel T, Baeuerle PA. Proteins controlling the nuclear uptake of NF-kappa B, Rel and dorsal. Trends Cell Biol. 1991 Nov;1(5):130-7. doi: 10.1016/0962-8924(91)90118-s. PMID: 14731544.
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