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The alpha 1-alpha 6 subunits of integrins are characteristically expressed in distinct segments of developing and adult human nephron

adult

Korhonen M.
GSID: Dt4yeN3Y3cQJ
M Korhonen, J Ylänne, L Laitinen… - The Journal of Cell …, 1990 - rupress.org

We studied the distribution of the alpha 1-alpha 6 subunits of beta 1 integrins in developing and adult human kidney using a panel of mAbs in indirect immunofluorescence microscopy. …

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Korhonen, M., Ylänne, J., Laitinen, L., & Virtanen, I. (1990). The alpha 1-alpha 6 subunits of integrins are characteristically expressed in distinct segments of developing and adult human nephron. The Journal of Cell Biology, 111(3), 1245-1254.
Korhonen, Matti, et al. "The alpha 1-alpha 6 subunits of integrins are characteristically expressed in distinct segments of developing and adult human nephron." The Journal of Cell Biology 111.3 (1990): 1245-1254.
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[Nephronophthisis].

Casopis lekaru ceskych

Gostof R, Půr J, Jandová L.
PMID: 5856260
Cas Lek Cesk. 1965 Nov 05;104(44):1199-206.

No abstract available.

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Gostof R, Půr J, Jandová L. Nefronoftiza. [Nephronophthisis]. Cas Lek Cesk. 1965;104(44):1199-206
Gostof, R., Půr, J., & Jandová, L. (1965). Nefronoftiza. [Nephronophthisis]. Casopis lekaru ceskych, 104(44), 1199-206.
Gostof, R, et al. "Nefronoftiza." [Nephronophthisis]. Casopis lekaru ceskych vol. 104,44 (1965): 1199-206.
Gostof R, Půr J, Jandová L. Nefronoftiza. [Nephronophthisis]. Cas Lek Cesk. 1965 Nov 05;104(44):1199-206. Czech. PMID: 5856260.
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Preventive counseling for nursing students.

Nursing outlook

Nehren J, Killen B.
PMID: 5180883
Nurs Outlook. 1967 Jan;15(1):37-9.

No abstract available.

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Nehren J, Killen B. Preventive counseling for nursing students. Nurs Outlook. 1967;15(1):37-9
Nehren, J., & Killen, B. (1967). Preventive counseling for nursing students. Nursing outlook, 15(1), 37-9.
Nehren, J, and Killen, B. "Preventive counseling for nursing students." Nursing outlook vol. 15,1 (1967): 37-9.
Nehren J, Killen B. Preventive counseling for nursing students. Nurs Outlook. 1967 Jan;15(1):37-9. PMID: 5180883.
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The role of neoadjuvant therapy in the management of locally advanced renal cell carcinoma.

Therapeutic advances in urology

Borregales LD, Adibi M, Thomas AZ, Wood CG, Karam JA.
PMID: 27034725
Ther Adv Urol. 2016 Apr;8(2):130-41. doi: 10.1177/1756287215612962. Epub 2015 Nov 20.

In the past decade, the armamentarium of targeted therapy agents for the treatment of metastatic renal cell carcinoma (RCC) has significantly increased. Improvements in response rates and survival, with more manageable side effects compared with interleukin 2/interferon immunotherapy, have...

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Borregales LD, Adibi M, Thomas AZ, et al. The role of neoadjuvant therapy in the management of locally advanced renal cell carcinoma. Ther Adv Urol. 2015;8(2):130-41doi: 10.1177/1756287215612962.
Borregales, L. D., Adibi, M., Thomas, A. Z., Wood, C. G., & Karam, J. A. (2016). The role of neoadjuvant therapy in the management of locally advanced renal cell carcinoma. Therapeutic advances in urology, 8(2), 130-41. https://doi.org/10.1177/1756287215612962
Borregales, Leonardo D, et al. "The role of neoadjuvant therapy in the management of locally advanced renal cell carcinoma." Therapeutic advances in urology vol. 8,2 (2016): 130-41. doi: https://doi.org/10.1177/1756287215612962
Borregales LD, Adibi M, Thomas AZ, Wood CG, Karam JA. The role of neoadjuvant therapy in the management of locally advanced renal cell carcinoma. Ther Adv Urol. 2016 Apr;8(2):130-41. doi: 10.1177/1756287215612962. Epub 2015 Nov 20. PMID: 27034725; PMCID: PMC4772353.
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CFTR and TNR-CFTR expression and function in the kidney.

Biophysical reviews

Souza-Menezes J, da Silva Feltran G, Morales MM.
PMID: 28510183
Biophys Rev. 2014 Jun;6(2):227-236. doi: 10.1007/s12551-014-0140-8. Epub 2014 May 07.

The cystic fibrosis transmembrane conductance regulator (CFTR) is abundantly expressed in the kidney. CFTR mRNA is detected in all nephron segments of rats and humans and its expression is higher in the renal cortex and outer medulla than in...

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Souza-Menezes J, da Silva Feltran G, Morales MM. CFTR and TNR-CFTR expression and function in the kidney. Biophys Rev. 2014;6(2):227-236doi: 10.1007/s12551-014-0140-8.
Souza-Menezes, J., da Silva Feltran, G., & Morales, M. M. (2014). CFTR and TNR-CFTR expression and function in the kidney. Biophysical reviews, 6(2), 227-236. https://doi.org/10.1007/s12551-014-0140-8
Souza-Menezes, Jackson, et al. "CFTR and TNR-CFTR expression and function in the kidney." Biophysical reviews vol. 6,2 (2014): 227-236. doi: https://doi.org/10.1007/s12551-014-0140-8
Souza-Menezes J, da Silva Feltran G, Morales MM. CFTR and TNR-CFTR expression and function in the kidney. Biophys Rev. 2014 Jun;6(2):227-236. doi: 10.1007/s12551-014-0140-8. Epub 2014 May 07. PMID: 28510183; PMCID: PMC5425698.
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Potential Reparative Role of Resident Adult Renal Stem/Progenitor Cells in Acute Kidney Injury.

BioResearch open access

Sallustio F, Serino G, Schena FP.
PMID: 26309808
Biores Open Access. 2015 Jul 01;4(1):326-33. doi: 10.1089/biores.2015.0011. eCollection 2015.

Human kidney is particularly susceptible to ischemia and toxins with consequential tubular necrosis and activation of inflammatory processes. This process can lead to the acute renal injury, and even if the kidney has a great capacity for regeneration after...

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Sallustio F, Serino G, Schena FP. Potential Reparative Role of Resident Adult Renal Stem/Progenitor Cells in Acute Kidney Injury. Biores Open Access. 2015;4(1):326-33doi: 10.1089/biores.2015.0011.
Sallustio, F., Serino, G., & Schena, F. P. (2015). Potential Reparative Role of Resident Adult Renal Stem/Progenitor Cells in Acute Kidney Injury. BioResearch open access, 4(1), 326-33. https://doi.org/10.1089/biores.2015.0011
Sallustio, Fabio, et al. "Potential Reparative Role of Resident Adult Renal Stem/Progenitor Cells in Acute Kidney Injury." BioResearch open access vol. 4,1 (2015): 326-33. doi: https://doi.org/10.1089/biores.2015.0011
Sallustio F, Serino G, Schena FP. Potential Reparative Role of Resident Adult Renal Stem/Progenitor Cells in Acute Kidney Injury. Biores Open Access. 2015 Jul 01;4(1):326-33. doi: 10.1089/biores.2015.0011. eCollection 2015. PMID: 26309808; PMCID: PMC4509615.
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Innovations in the management of Wilms' tumor.

Therapeutic advances in urology

Gleason JM, Lorenzo AJ, Bowlin PR, Koyle MA.
PMID: 25083165
Ther Adv Urol. 2014 Aug;6(4):165-76. doi: 10.1177/1756287214528023.

Advances in the management of Wilms' tumor have been dramatic over the past half century, not in small part due to the institution of multimodal therapy and the formation of collaborative study groups. While different opinions exist in the...

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Gleason JM, Lorenzo AJ, Bowlin PR, et al. Innovations in the management of Wilms' tumor. Ther Adv Urol. 2014;6(4):165-76doi: 10.1177/1756287214528023.
Gleason, J. M., Lorenzo, A. J., Bowlin, P. R., & Koyle, M. A. (2014). Innovations in the management of Wilms' tumor. Therapeutic advances in urology, 6(4), 165-76. https://doi.org/10.1177/1756287214528023
Gleason, Joseph M, et al. "Innovations in the management of Wilms' tumor." Therapeutic advances in urology vol. 6,4 (2014): 165-76. doi: https://doi.org/10.1177/1756287214528023
Gleason JM, Lorenzo AJ, Bowlin PR, Koyle MA. Innovations in the management of Wilms' tumor. Ther Adv Urol. 2014 Aug;6(4):165-76. doi: 10.1177/1756287214528023. PMID: 25083165; PMCID: PMC4054506.
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Complement activation in progressive renal disease.

World journal of nephrology

Fearn A, Sheerin NS.
PMID: 25664245
World J Nephrol. 2015 Feb 06;4(1):31-40. doi: 10.5527/wjn.v4.i1.31.

Chronic kidney disease (CKD) is common and the cause of significant morbidity and mortality. The replacement of functioning nephrons by fibrosis is characteristic of progressive disease. The pathways that lead to fibrosis are not fully understood, although chronic non-resolving...

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Fearn A, Sheerin NS. Complement activation in progressive renal disease. World J Nephrol. 2015;4(1):31-40doi: 10.5527/wjn.v4.i1.31.
Fearn, A., & Sheerin, N. S. (2015). Complement activation in progressive renal disease. World journal of nephrology, 4(1), 31-40. https://doi.org/10.5527/wjn.v4.i1.31
Fearn, Amy, and Sheerin, Neil Stephen. "Complement activation in progressive renal disease." World journal of nephrology vol. 4,1 (2015): 31-40. doi: https://doi.org/10.5527/wjn.v4.i1.31
Fearn A, Sheerin NS. Complement activation in progressive renal disease. World J Nephrol. 2015 Feb 06;4(1):31-40. doi: 10.5527/wjn.v4.i1.31. PMID: 25664245; PMCID: PMC4317626.
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'Lemonade Legs': Why do Some Patients Get Profound Hypomagnesaemia on Proton-Pump Inhibitors?.

Intestinal research

Atkinson NS, Reynolds DJ, Travis SP.
PMID: 26130997
Intest Res. 2015 Jul;13(3):227-32. doi: 10.5217/ir.2015.13.3.227. Epub 2015 Jun 09.

Proton pump inhibitors (PPIs) are widely used though an association with hypomagnesaemia and hypocalcaemia has only been described since 2006. Patients typically present after years of stable dosing with musculoskeletal, neurological or cardiac arrhythmic symptoms, but it is likely...

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Atkinson NS, Reynolds DJ, Travis SP. 'Lemonade Legs': Why do Some Patients Get Profound Hypomagnesaemia on Proton-Pump Inhibitors?. Intest Res. 2015;13(3):227-32doi: 10.5217/ir.2015.13.3.227.
Atkinson, N. S., Reynolds, D. J., & Travis, S. P. (2015). 'Lemonade Legs': Why do Some Patients Get Profound Hypomagnesaemia on Proton-Pump Inhibitors?. Intestinal research, 13(3), 227-32. https://doi.org/10.5217/ir.2015.13.3.227
Atkinson, Nathan S S, et al. "'Lemonade Legs': Why do Some Patients Get Profound Hypomagnesaemia on Proton-Pump Inhibitors?." Intestinal research vol. 13,3 (2015): 227-32. doi: https://doi.org/10.5217/ir.2015.13.3.227
Atkinson NS, Reynolds DJ, Travis SP. 'Lemonade Legs': Why do Some Patients Get Profound Hypomagnesaemia on Proton-Pump Inhibitors?. Intest Res. 2015 Jul;13(3):227-32. doi: 10.5217/ir.2015.13.3.227. Epub 2015 Jun 09. PMID: 26130997; PMCID: PMC4479737.
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Erratum.

Nephron

[No authors listed]
PMID: 27286735
Nephron. 2016;133(1):80. doi: 10.1159/000445989. Epub 2016 Jun 07.

No abstract available.

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Erratum. Nephron. 2016;133(1):80doi: 10.1159/000445989.
(2016). Erratum. Nephron, 133(1), 80. https://doi.org/10.1159/000445989
"Erratum." Nephron vol. 133,1 (2016): 80. doi: https://doi.org/10.1159/000445989
Erratum. Nephron. 2016;133(1):80. doi: 10.1159/000445989. Epub 2016 Jun 07. PMID: 27286735.
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Bartter and Gitelman syndromes: Spectrum of clinical manifestations caused by different mutations.

World journal of methodology

Al Shibli A, Narchi H.
PMID: 26140272
World J Methodol. 2015 Jun 26;5(2):55-61. doi: 10.5662/wjm.v5.i2.55. eCollection 2015 Jun 26.

Bartter and Gitelman syndromes (BS and GS) are inherited disorders resulting in defects in renal tubular handling of sodium, potassium and chloride. Previously considered as genotypic and phenotypic heterogeneous diseases, recent evidence suggests that they constitute a spectrum of...

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Al Shibli A, Narchi H. Bartter and Gitelman syndromes: Spectrum of clinical manifestations caused by different mutations. World J Methodol. 2015;5(2):55-61doi: 10.5662/wjm.v5.i2.55.
Al Shibli, A., & Narchi, H. (2015). Bartter and Gitelman syndromes: Spectrum of clinical manifestations caused by different mutations. World journal of methodology, 5(2), 55-61. https://doi.org/10.5662/wjm.v5.i2.55
Al Shibli, Amar, and Narchi, Hassib. "Bartter and Gitelman syndromes: Spectrum of clinical manifestations caused by different mutations." World journal of methodology vol. 5,2 (2015): 55-61. doi: https://doi.org/10.5662/wjm.v5.i2.55
Al Shibli A, Narchi H. Bartter and Gitelman syndromes: Spectrum of clinical manifestations caused by different mutations. World J Methodol. 2015 Jun 26;5(2):55-61. doi: 10.5662/wjm.v5.i2.55. eCollection 2015 Jun 26. PMID: 26140272; PMCID: PMC4482822.
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Management of the cardiorenal syndrome in decompensated heart failure.

Cardiorenal medicine

Verbrugge FH, Grieten L, Mullens W.
PMID: 25737682
Cardiorenal Med. 2014 Dec;4(3):176-88. doi: 10.1159/000366168.

BACKGROUND: The management of the cardiorenal syndrome (CRS) in decompensated heart failure (HF) is challenging, with high-quality evidence lacking.SUMMARY: The pathophysiology of CRS in decompensated HF is complex, with glomerular filtration rate (GFR) and urine output representing different aspects...

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Verbrugge FH, Grieten L, Mullens W. Management of the cardiorenal syndrome in decompensated heart failure. Cardiorenal Med. 2014;4(3):176-88doi: 10.1159/000366168.
Verbrugge, F. H., Grieten, L., & Mullens, W. (2014). Management of the cardiorenal syndrome in decompensated heart failure. Cardiorenal medicine, 4(3), 176-88. https://doi.org/10.1159/000366168
Verbrugge, Frederik Hendrik, et al. "Management of the cardiorenal syndrome in decompensated heart failure." Cardiorenal medicine vol. 4,3 (2014): 176-88. doi: https://doi.org/10.1159/000366168
Verbrugge FH, Grieten L, Mullens W. Management of the cardiorenal syndrome in decompensated heart failure. Cardiorenal Med. 2014 Dec;4(3):176-88. doi: 10.1159/000366168. PMID: 25737682; PMCID: PMC4299260.
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Showing 1 to 12 of 1785 entries