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Heme oxygenase induction attenuates TNF-α-induced hypertension in pregnant rodents.

Frontiers in pharmacology

George EM, Stout JM, Stec DE, Granger JP.
PMID: 26347650
Front Pharmacol. 2015 Aug 17;6:165. doi: 10.3389/fphar.2015.00165. eCollection 2015.

Pre-eclampsia is a hypertensive disorder of pregnancy initiated by placental insufficiency and chronic ischemia. In response, several pathways activated in the placenta are responsible for the maternal syndrome, including increased production of the anti-angiogenic protein, sFlt-1, and inflammatory cytokines,...

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George EM, Stout JM, Stec DE, et al. Heme oxygenase induction attenuates TNF-α-induced hypertension in pregnant rodents. Front Pharmacol. 2015;6:165doi: 10.3389/fphar.2015.00165.
George, E. M., Stout, J. M., Stec, D. E., & Granger, J. P. (2015). Heme oxygenase induction attenuates TNF-α-induced hypertension in pregnant rodents. Frontiers in pharmacology, 6165. https://doi.org/10.3389/fphar.2015.00165
George, Eric M, et al. "Heme oxygenase induction attenuates TNF-α-induced hypertension in pregnant rodents." Frontiers in pharmacology vol. 6 (2015): 165. doi: https://doi.org/10.3389/fphar.2015.00165
George EM, Stout JM, Stec DE, Granger JP. Heme oxygenase induction attenuates TNF-α-induced hypertension in pregnant rodents. Front Pharmacol. 2015 Aug 17;6:165. doi: 10.3389/fphar.2015.00165. eCollection 2015. PMID: 26347650; PMCID: PMC4538306.
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Role of fetal programming in the development of hypertension.

Future cardiology

Ojeda NB, Grigore D, Alexander BT.
PMID: 19672482
Future Cardiol. 2008 Mar;4(2):163-74. doi: 10.2217/14796678.4.2.163.

Epidemiological studies have suggested that size at birth contributes to increased cardiovascular disease (CVD) risk in later life. Findings from experimental studies are providing insight into the mechanisms linking impaired fetal growth and the increased risk of CVD and...

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Ojeda NB, Grigore D, Alexander BT. Role of fetal programming in the development of hypertension. Future Cardiol. 2008;4(2):163-74doi: 10.2217/14796678.4.2.163.
Ojeda, N. B., Grigore, D., & Alexander, B. T. (2008). Role of fetal programming in the development of hypertension. Future cardiology, 4(2), 163-74. https://doi.org/10.2217/14796678.4.2.163
Ojeda, Norma B, et al. "Role of fetal programming in the development of hypertension." Future cardiology vol. 4,2 (2008): 163-74. doi: https://doi.org/10.2217/14796678.4.2.163
Ojeda NB, Grigore D, Alexander BT. Role of fetal programming in the development of hypertension. Future Cardiol. 2008 Mar;4(2):163-74. doi: 10.2217/14796678.4.2.163. PMID: 19672482; PMCID: PMC2723856.
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Obesity, hypertension, and chronic kidney disease.

International journal of nephrology and renovascular disease

Hall ME, do Carmo JM, da Silva AA, Juncos LA, Wang Z, Hall JE.
PMID: 24600241
Int J Nephrol Renovasc Dis. 2014 Feb 18;7:75-88. doi: 10.2147/IJNRD.S39739. eCollection 2014.

Obesity is a major risk factor for essential hypertension, diabetes, and other comorbid conditions that contribute to development of chronic kidney disease. Obesity raises blood pressure by increasing renal tubular sodium reabsorption, impairing pressure natriuresis, and causing volume expansion...

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Hall ME, do Carmo JM, da Silva AA, et al. Obesity, hypertension, and chronic kidney disease. Int J Nephrol Renovasc Dis. 2014;7:75-88doi: 10.2147/IJNRD.S39739.
Hall, M. E., do Carmo, J. M., da Silva, A. A., Juncos, L. A., Wang, Z., & Hall, J. E. (2014). Obesity, hypertension, and chronic kidney disease. International journal of nephrology and renovascular disease, 775-88. https://doi.org/10.2147/IJNRD.S39739
Hall, Michael E, et al. "Obesity, hypertension, and chronic kidney disease." International journal of nephrology and renovascular disease vol. 7 (2014): 75-88. doi: https://doi.org/10.2147/IJNRD.S39739
Hall ME, do Carmo JM, da Silva AA, Juncos LA, Wang Z, Hall JE. Obesity, hypertension, and chronic kidney disease. Int J Nephrol Renovasc Dis. 2014 Feb 18;7:75-88. doi: 10.2147/IJNRD.S39739. eCollection 2014. PMID: 24600241; PMCID: PMC3933708.
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Baroreflex stimulation: A novel treatment option for resistant hypertension.

Journal of the American Society of Hypertension : JASH

Navaneethan SD, Lohmeier TE, Bisognano JD.
PMID: 20409946
J Am Soc Hypertens. 2009 Jan-Feb;3(1):69-74. doi: 10.1016/j.jash.2008.09.001. Epub 2008 Dec 02.

Hypertension is a major public health problem in both developing and developed countries. Despite the increasing awareness of hypertension and its implications among patients and the treating physicians, the prevalence of resistant hypertension remains high and is expected to...

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Navaneethan SD, Lohmeier TE, Bisognano JD. Baroreflex stimulation: A novel treatment option for resistant hypertension. J Am Soc Hypertens. 2008;3(1):69-74doi: 10.1016/j.jash.2008.09.001.
Navaneethan, S. D., Lohmeier, T. E., & Bisognano, J. D. (2009). Baroreflex stimulation: A novel treatment option for resistant hypertension. Journal of the American Society of Hypertension : JASH, 3(1), 69-74. https://doi.org/10.1016/j.jash.2008.09.001
Navaneethan, Sankar D, et al. "Baroreflex stimulation: A novel treatment option for resistant hypertension." Journal of the American Society of Hypertension : JASH vol. 3,1 (2009): 69-74. doi: https://doi.org/10.1016/j.jash.2008.09.001
Navaneethan SD, Lohmeier TE, Bisognano JD. Baroreflex stimulation: A novel treatment option for resistant hypertension. J Am Soc Hypertens. 2009 Jan-Feb;3(1):69-74. doi: 10.1016/j.jash.2008.09.001. Epub 2008 Dec 02. PMID: 20409946; PMCID: PMC3814230.
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Systems biology and integrative physiological modelling.

The Journal of physiology

Hester RL, Iliescu R, Summers R, Coleman TG.
PMID: 21135044
J Physiol. 2011 Mar 01;589:1053-60. doi: 10.1113/jphysiol.2010.201558. Epub 2010 Dec 06.

Over the last 10 years, 'Systems Biology' has focused on the integration of biology and medicine with information technology and computation. The current challenge is to use the discoveries of the last 20 years, such as genomics and proteomics,...

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Hester RL, Iliescu R, Summers R, et al. Systems biology and integrative physiological modelling. J Physiol. 2010;589:1053-60doi: 10.1113/jphysiol.2010.201558.
Hester, R. L., Iliescu, R., Summers, R., & Coleman, T. G. (2011). Systems biology and integrative physiological modelling. The Journal of physiology, 5891053-60. https://doi.org/10.1113/jphysiol.2010.201558
Hester, Robert L, et al. "Systems biology and integrative physiological modelling." The Journal of physiology vol. 589 (2011): 1053-60. doi: https://doi.org/10.1113/jphysiol.2010.201558
Hester RL, Iliescu R, Summers R, Coleman TG. Systems biology and integrative physiological modelling. J Physiol. 2011 Mar 01;589:1053-60. doi: 10.1113/jphysiol.2010.201558. Epub 2010 Dec 06. PMID: 21135044; PMCID: PMC3060586.
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Obese melanocortin-4 receptor-deficient rats exhibit augmented angiogenic balance and vasorelaxation during pregnancy.

Physiological reports

Spradley FT, Palei AC, Granger JP.
PMID: 24159378
Physiol Rep. 2013 Sep 01;1(4):e00081. doi: 10.1002/phy2.81.

While obesity is a major risk factor for preeclampsia, the mechanisms linking obesity and hypertension during preeclampsia remain unclear. Hypertension in preeclampsia is associated with placental ischemia-induced release of anti-angiogenic soluble fms-like tyrosine kinase (sFlt-1) into the maternal circulation,...

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Spradley FT, Palei AC, Granger JP. Obese melanocortin-4 receptor-deficient rats exhibit augmented angiogenic balance and vasorelaxation during pregnancy. Physiol Rep. 2013;1(4):e00081doi: 10.1002/phy2.81.
Spradley, F. T., Palei, A. C., & Granger, J. P. (2013). Obese melanocortin-4 receptor-deficient rats exhibit augmented angiogenic balance and vasorelaxation during pregnancy. Physiological reports, 1(4), e00081. https://doi.org/10.1002/phy2.81
Spradley, Frank T, et al. "Obese melanocortin-4 receptor-deficient rats exhibit augmented angiogenic balance and vasorelaxation during pregnancy." Physiological reports vol. 1,4 (2013): e00081. doi: https://doi.org/10.1002/phy2.81
Spradley FT, Palei AC, Granger JP. Obese melanocortin-4 receptor-deficient rats exhibit augmented angiogenic balance and vasorelaxation during pregnancy. Physiol Rep. 2013 Sep 01;1(4):e00081. doi: 10.1002/phy2.81. PMID: 24159378; PMCID: PMC3804345.
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Reduced Renal Mass, Salt-Sensitive Hypertension Is Resistant to Renal Denervation.

Frontiers in physiology

Tudorancea I, Lohmeier TE, Alexander BT, Pieptu D, Serban DN, Iliescu R.
PMID: 29760664
Front Physiol. 2018 Apr 30;9:455. doi: 10.3389/fphys.2018.00455. eCollection 2018.

No abstract available.

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Tudorancea I, Lohmeier TE, Alexander BT, et al. Reduced Renal Mass, Salt-Sensitive Hypertension Is Resistant to Renal Denervation. Front Physiol. 2018;9:455doi: 10.3389/fphys.2018.00455.
Tudorancea, I., Lohmeier, T. E., Alexander, B. T., Pieptu, D., Serban, D. N., & Iliescu, R. (2018). Reduced Renal Mass, Salt-Sensitive Hypertension Is Resistant to Renal Denervation. Frontiers in physiology, 9455. https://doi.org/10.3389/fphys.2018.00455
Tudorancea, Ionut, et al. "Reduced Renal Mass, Salt-Sensitive Hypertension Is Resistant to Renal Denervation." Frontiers in physiology vol. 9 (2018): 455. doi: https://doi.org/10.3389/fphys.2018.00455
Tudorancea I, Lohmeier TE, Alexander BT, Pieptu D, Serban DN, Iliescu R. Reduced Renal Mass, Salt-Sensitive Hypertension Is Resistant to Renal Denervation. Front Physiol. 2018 Apr 30;9:455. doi: 10.3389/fphys.2018.00455. eCollection 2018. PMID: 29760664; PMCID: PMC5936777.
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Simulating a virtual population's sensitivity to salt and uninephrectomy.

Interface focus

Clemmer JS, Hester RL, Pruett WA.
PMID: 29285341
Interface Focus. 2018 Feb 06;8(1):20160134. doi: 10.1098/rsfs.2016.0134. Epub 2017 Dec 15.

Salt sensitivity, with or without concomitant hypertension, is associated with increased mortality. Reduced functional renal mass plays an important role in causing salt-sensitive hypertension for many individuals. Factors that are important in the condition of decreased renal mass and...

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Clemmer JS, Hester RL, Pruett WA. Simulating a virtual population's sensitivity to salt and uninephrectomy. Interface Focus. 2017;8(1):20160134doi: 10.1098/rsfs.2016.0134.
Clemmer, J. S., Hester, R. L., & Pruett, W. A. (2018). Simulating a virtual population's sensitivity to salt and uninephrectomy. Interface focus, 8(1), 20160134. https://doi.org/10.1098/rsfs.2016.0134
Clemmer, John S, et al. "Simulating a virtual population's sensitivity to salt and uninephrectomy." Interface focus vol. 8,1 (2018): 20160134. doi: https://doi.org/10.1098/rsfs.2016.0134
Clemmer JS, Hester RL, Pruett WA. Simulating a virtual population's sensitivity to salt and uninephrectomy. Interface Focus. 2018 Feb 06;8(1):20160134. doi: 10.1098/rsfs.2016.0134. Epub 2017 Dec 15. PMID: 29285341; PMCID: PMC5740217.
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Hypertension in response to IL-6 during pregnancy: role of AT1-receptor activation.

International journal of interferon, cytokine and mediator research

Lamarca B, Speed J, Ray LF, Cockrell K, Wallukat G, Dechend R, Granger J.
PMID: 23002372
Int J Interferon Cytokine Mediat Res. 2011 Nov;2011(3):65-70. doi: 10.2147/IJICMR.S22329.

BACKGROUND: Increases in interleukin 6 (IL-6) and agonistic autoantibodies to the angiotensin II type 1 receptor (AT1-AA) are proposed to be important links between placental ischemia and hypertension in preeclampsia. METHODS: The purpose of this study was to determine...

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Lamarca B, Speed J, Ray LF, et al. Hypertension in response to IL-6 during pregnancy: role of AT1-receptor activation. Int J Interferon Cytokine Mediat Res. 2011;2011(3):65-70doi: 10.2147/IJICMR.S22329.
Lamarca, B., Speed, J., Ray, L. F., Cockrell, K., Wallukat, G., Dechend, R., & Granger, J. (2011). Hypertension in response to IL-6 during pregnancy: role of AT1-receptor activation. International journal of interferon, cytokine and mediator research, 2011(3), 65-70. https://doi.org/10.2147/IJICMR.S22329
Lamarca, Babbette, et al. "Hypertension in response to IL-6 during pregnancy: role of AT1-receptor activation." International journal of interferon, cytokine and mediator research vol. 2011,3 (2011): 65-70. doi: https://doi.org/10.2147/IJICMR.S22329
Lamarca B, Speed J, Ray LF, Cockrell K, Wallukat G, Dechend R, Granger J. Hypertension in response to IL-6 during pregnancy: role of AT1-receptor activation. Int J Interferon Cytokine Mediat Res. 2011 Nov;2011(3):65-70. doi: 10.2147/IJICMR.S22329. PMID: 23002372; PMCID: PMC3446210.
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Elastin-Like Polypeptide: VEGF-B Fusion Protein for Treatment of Preeclampsia.

Hypertension (Dallas, Tex. : 1979)

Waller JP, Howell JA, Peterson H, George EM, Bidwell GL.
PMID: 34719237
Hypertension. 2021 Dec;78(6):1888-1901. doi: 10.1161/HYPERTENSIONAHA.121.17713. Epub 2021 Nov 01.

[Figure: see text].

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Waller JP, Howell JA, Peterson H, et al. Elastin-Like Polypeptide: VEGF-B Fusion Protein for Treatment of Preeclampsia. Hypertension. 2021;78(6):1888-1901doi: 10.1161/HYPERTENSIONAHA.121.17713.
Waller, J. P., Howell, J. A., Peterson, H., George, E. M., & Bidwell, G. L. (2021). Elastin-Like Polypeptide: VEGF-B Fusion Protein for Treatment of Preeclampsia. Hypertension (Dallas, Tex. : 1979), 78(6), 1888-1901. https://doi.org/10.1161/HYPERTENSIONAHA.121.17713
Waller, Jamarius P, et al. "Elastin-Like Polypeptide: VEGF-B Fusion Protein for Treatment of Preeclampsia." Hypertension (Dallas, Tex. : 1979) vol. 78,6 (2021): 1888-1901. doi: https://doi.org/10.1161/HYPERTENSIONAHA.121.17713
Waller JP, Howell JA, Peterson H, George EM, Bidwell GL. Elastin-Like Polypeptide: VEGF-B Fusion Protein for Treatment of Preeclampsia. Hypertension. 2021 Dec;78(6):1888-1901. doi: 10.1161/HYPERTENSIONAHA.121.17713. Epub 2021 Nov 01. PMID: 34719237; PMCID: PMC8585700.
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Targeting Heme Oxygenase-1 in Cardiovascular and Kidney Disease.

Antioxidants (Basel, Switzerland)

Drummond HA, Mitchell ZL, Abraham NG, Stec DE.
PMID: 31216709
Antioxidants (Basel). 2019 Jun 18;8(6). doi: 10.3390/antiox8060181.

Heme oxygenase (HO) plays an important role in the cardiovascular system. It is involved in many physiological and pathophysiological processes in all organs of the cardiovascular system. From the regulation of blood pressure and blood flow to the adaptive...

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Drummond HA, Mitchell ZL, Abraham NG, et al. Targeting Heme Oxygenase-1 in Cardiovascular and Kidney Disease. Antioxidants (Basel). 2019;8(6)doi: 10.3390/antiox8060181.
Drummond, H. A., Mitchell, Z. L., Abraham, N. G., & Stec, D. E. (2019). Targeting Heme Oxygenase-1 in Cardiovascular and Kidney Disease. Antioxidants (Basel, Switzerland), 8(6), . https://doi.org/10.3390/antiox8060181
Drummond, Heather A, et al. "Targeting Heme Oxygenase-1 in Cardiovascular and Kidney Disease." Antioxidants (Basel, Switzerland) vol. 8,6 (2019). doi: https://doi.org/10.3390/antiox8060181
Drummond HA, Mitchell ZL, Abraham NG, Stec DE. Targeting Heme Oxygenase-1 in Cardiovascular and Kidney Disease. Antioxidants (Basel). 2019 Jun 18;8(6). doi: 10.3390/antiox8060181. PMID: 31216709; PMCID: PMC6617021.
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Magnesium Sulfate Prevents Placental Ischemia-Induced Increases in Brain Water Content and Cerebrospinal Fluid Cytokines in Pregnant Rats.

Frontiers in neuroscience

Zhang LW, Warrington JP.
PMID: 28008305
Front Neurosci. 2016 Dec 08;10:561. doi: 10.3389/fnins.2016.00561. eCollection 2016.

Magnesium sulfate (MgSO

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Zhang LW, Warrington JP. Magnesium Sulfate Prevents Placental Ischemia-Induced Increases in Brain Water Content and Cerebrospinal Fluid Cytokines in Pregnant Rats. Front Neurosci. 2016;10:561doi: 10.3389/fnins.2016.00561.
Zhang, L. W., & Warrington, J. P. (2016). Magnesium Sulfate Prevents Placental Ischemia-Induced Increases in Brain Water Content and Cerebrospinal Fluid Cytokines in Pregnant Rats. Frontiers in neuroscience, 10561. https://doi.org/10.3389/fnins.2016.00561
Zhang, Linda W, and Warrington, Junie P. "Magnesium Sulfate Prevents Placental Ischemia-Induced Increases in Brain Water Content and Cerebrospinal Fluid Cytokines in Pregnant Rats." Frontiers in neuroscience vol. 10 (2016): 561. doi: https://doi.org/10.3389/fnins.2016.00561
Zhang LW, Warrington JP. Magnesium Sulfate Prevents Placental Ischemia-Induced Increases in Brain Water Content and Cerebrospinal Fluid Cytokines in Pregnant Rats. Front Neurosci. 2016 Dec 08;10:561. doi: 10.3389/fnins.2016.00561. eCollection 2016. PMID: 28008305; PMCID: PMC5143678.
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Showing 1 to 12 of 64 entries