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Subtyping Chronic Obstructive Pulmonary Disease Using Peripheral Blood Proteomics.

Chronic obstructive pulmonary diseases (Miami, Fla.)

Zarei S, Mirtar A, Morrow JD, Castaldi PJ, Belloni P, Hersh CP.
PMID: 28848918
Chronic Obstr Pulm Dis. 2017 Feb 08;4(2):97-108. doi: 10.15326/jcopdf.4.2.2016.0147.

Chronic obstructive pulmonary disease (COPD) is a heterogeneous disorder. COPD patients may have different clinical features, imaging characteristics and natural history. Multiple studies have investigated heterogeneity using statistical methods such as unsupervised clustering to define different subgroups of COPD...

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Zarei S, Mirtar A, Morrow JD, et al. Subtyping Chronic Obstructive Pulmonary Disease Using Peripheral Blood Proteomics. Chronic Obstr Pulm Dis. 2017;4(2):97-108doi: 10.15326/jcopdf.4.2.2016.0147.
Zarei, S., Mirtar, A., Morrow, J. D., Castaldi, P. J., Belloni, P., & Hersh, C. P. (2017). Subtyping Chronic Obstructive Pulmonary Disease Using Peripheral Blood Proteomics. Chronic obstructive pulmonary diseases (Miami, Fla.), 4(2), 97-108. https://doi.org/10.15326/jcopdf.4.2.2016.0147
Zarei, Sara, et al. "Subtyping Chronic Obstructive Pulmonary Disease Using Peripheral Blood Proteomics." Chronic obstructive pulmonary diseases (Miami, Fla.) vol. 4,2 (2017): 97-108. doi: https://doi.org/10.15326/jcopdf.4.2.2016.0147
Zarei S, Mirtar A, Morrow JD, Castaldi PJ, Belloni P, Hersh CP. Subtyping Chronic Obstructive Pulmonary Disease Using Peripheral Blood Proteomics. Chronic Obstr Pulm Dis. 2017 Feb 08;4(2):97-108. doi: 10.15326/jcopdf.4.2.2016.0147. PMID: 28848918; PMCID: PMC5559108.
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Estimating gene regulatory networks with pandaR.

Bioinformatics (Oxford, England)

Schlauch D, Paulson JN, Young A, Glass K, Quackenbush J.
PMID: 28334344
Bioinformatics. 2017 Jul 15;33(14):2232-2234. doi: 10.1093/bioinformatics/btx139.

CONTACT: [email protected] or [email protected] AND IMPLEMENTATION: PandaR is provided as a Bioconductor R Package and is available at bioconductor.org/packages/pandaR.

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Schlauch D, Paulson JN, Young A, et al. Estimating gene regulatory networks with pandaR. Bioinformatics. 2017;33(14):2232-2234doi: 10.1093/bioinformatics/btx139.
Schlauch, D., Paulson, J. N., Young, A., Glass, K., & Quackenbush, J. (2017). Estimating gene regulatory networks with pandaR. Bioinformatics (Oxford, England), 33(14), 2232-2234. https://doi.org/10.1093/bioinformatics/btx139
Schlauch, Daniel, et al. "Estimating gene regulatory networks with pandaR." Bioinformatics (Oxford, England) vol. 33,14 (2017): 2232-2234. doi: https://doi.org/10.1093/bioinformatics/btx139
Schlauch D, Paulson JN, Young A, Glass K, Quackenbush J. Estimating gene regulatory networks with pandaR. Bioinformatics. 2017 Jul 15;33(14):2232-2234. doi: 10.1093/bioinformatics/btx139. PMID: 28334344; PMCID: PMC5870629.
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Diagnosing alpha-1 antitrypsin deficiency: the first step in precision medicine.

F1000Research

Hersh CP.
PMID: 29225784
F1000Res. 2017 Nov 27;6:2049. doi: 10.12688/f1000research.12399.1. eCollection 2017.

Severe alpha-1 antitrypsin (AAT) deficiency is one of the most common serious genetic diseases in adults of European descent. Individuals with AAT deficiency have a greatly increased risk for emphysema and liver disease. Other manifestations include bronchiectasis, necrotizing panniculitis...

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Hersh CP. Diagnosing alpha-1 antitrypsin deficiency: the first step in precision medicine. F1000Res. 2017;6:2049doi: 10.12688/f1000research.12399.1.
Hersh, C. P. (2017). Diagnosing alpha-1 antitrypsin deficiency: the first step in precision medicine. F1000Research, 62049. https://doi.org/10.12688/f1000research.12399.1
Hersh, Craig P. "Diagnosing alpha-1 antitrypsin deficiency: the first step in precision medicine." F1000Research vol. 6 (2017): 2049. doi: https://doi.org/10.12688/f1000research.12399.1
Hersh CP. Diagnosing alpha-1 antitrypsin deficiency: the first step in precision medicine. F1000Res. 2017 Nov 27;6:2049. doi: 10.12688/f1000research.12399.1. eCollection 2017. PMID: 29225784; PMCID: PMC5710307.
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Sex-specific associations with DNA methylation in lung tissue demonstrate smoking interactions.

Epigenetics

Koo HK, Morrow J, Kachroo P, Tantisira K, Weiss ST, Hersh CP, Silverman EK, DeMeo DL.
PMID: 32962511
Epigenetics. 2021 Jun;16(6):692-703. doi: 10.1080/15592294.2020.1819662. Epub 2020 Sep 22.

Cigarette smoking impacts DNA methylation, but the investigation of sex-specific features of lung tissue DNA methylation in smokers has been limited. Women appear more susceptible to cigarette smoke, and often develop more severe lung disease at an earlier age...

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Koo HK, Morrow J, Kachroo P, et al. Sex-specific associations with DNA methylation in lung tissue demonstrate smoking interactions. Epigenetics. 2020;16(6):692-703doi: 10.1080/15592294.2020.1819662.
Koo, H. K., Morrow, J., Kachroo, P., Tantisira, K., Weiss, S. T., Hersh, C. P., Silverman, E. K., & DeMeo, D. L. (2021). Sex-specific associations with DNA methylation in lung tissue demonstrate smoking interactions. Epigenetics, 16(6), 692-703. https://doi.org/10.1080/15592294.2020.1819662
Koo, Hyeon-Kyoung, et al. "Sex-specific associations with DNA methylation in lung tissue demonstrate smoking interactions." Epigenetics vol. 16,6 (2021): 692-703. doi: https://doi.org/10.1080/15592294.2020.1819662
Koo HK, Morrow J, Kachroo P, Tantisira K, Weiss ST, Hersh CP, Silverman EK, DeMeo DL. Sex-specific associations with DNA methylation in lung tissue demonstrate smoking interactions. Epigenetics. 2021 Jun;16(6):692-703. doi: 10.1080/15592294.2020.1819662. Epub 2020 Sep 22. PMID: 32962511; PMCID: PMC8143227.
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Controllability in an islet specific regulatory network identifies the transcriptional factor NFATC4, which regulates Type 2 Diabetes associated genes.

NPJ systems biology and applications

Sharma A, Halu A, Decano JL, Padi M, Liu YY, Prasad RB, Fadista J, Santolini M, Menche J, Weiss ST, Vidal M, Silverman EK, Aikawa M, Barabási AL, Groop L, Loscalzo J.
PMID: 29977601
NPJ Syst Biol Appl. 2018 Jul 03;4:25. doi: 10.1038/s41540-018-0057-0. eCollection 2018.

Probing the dynamic control features of biological networks represents a new frontier in capturing the dysregulated pathways in complex diseases. Here, using patient samples obtained from a pancreatic islet transplantation program, we constructed a tissue-specific gene regulatory network and...

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Sharma A, Halu A, Decano JL, et al. Controllability in an islet specific regulatory network identifies the transcriptional factor NFATC4, which regulates Type 2 Diabetes associated genes. NPJ Syst Biol Appl. 2018;4:25doi: 10.1038/s41540-018-0057-0.
Sharma, A., Halu, A., Decano, J. L., Padi, M., Liu, Y. Y., Prasad, R. B., Fadista, J., Santolini, M., Menche, J., Weiss, S. T., Vidal, M., Silverman, E. K., Aikawa, M., Barabási, A. L., Groop, L., & Loscalzo, J. (2018). Controllability in an islet specific regulatory network identifies the transcriptional factor NFATC4, which regulates Type 2 Diabetes associated genes. NPJ systems biology and applications, 425. https://doi.org/10.1038/s41540-018-0057-0
Sharma, Amitabh, et al. "Controllability in an islet specific regulatory network identifies the transcriptional factor NFATC4, which regulates Type 2 Diabetes associated genes." NPJ systems biology and applications vol. 4 (2018): 25. doi: https://doi.org/10.1038/s41540-018-0057-0
Sharma A, Halu A, Decano JL, Padi M, Liu YY, Prasad RB, Fadista J, Santolini M, Menche J, Weiss ST, Vidal M, Silverman EK, Aikawa M, Barabási AL, Groop L, Loscalzo J. Controllability in an islet specific regulatory network identifies the transcriptional factor NFATC4, which regulates Type 2 Diabetes associated genes. NPJ Syst Biol Appl. 2018 Jul 03;4:25. doi: 10.1038/s41540-018-0057-0. eCollection 2018. PMID: 29977601; PMCID: PMC6028434.
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A Disintegrin and A Metalloproteinase-9 (ADAM9): A Novel Proteinase Culprit with Multifarious Contributions to COPD.

American journal of respiratory and critical care medicine

Wang X, Polverino F, Rojas-Quintero J, Zhang D, Sánchez J, Yambayev I, Lindqvist E, Virtala R, Djukanovic R, Davies DE, Wilson S, O'Donnell R, Cunoosamy D, Hazon P, Higham A, Singh D, Olsson H, Owen CA.
PMID: 29864380
Am J Respir Crit Care Med. 2018 Jun 04; doi: 10.1164/rccm.201711-2300OC. Epub 2018 Jun 04.

INTRODUCTION: Proteinases with a disintegrin and a metalloproteinase domain (ADAMs) have not been well studied in COPD. We investigated whether ADAM9 is linked to COPD in humans and mice.METHODS: ADAM9 blood and lung levels were measured in COPD patients...

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Wang X, Polverino F, Rojas-Quintero J, et al. A Disintegrin and A Metalloproteinase-9 (ADAM9): A Novel Proteinase Culprit with Multifarious Contributions to COPD. Am J Respir Crit Care Med. 2018;doi: 10.1164/rccm.201711-2300OC.
Wang, X., Polverino, F., Rojas-Quintero, J., Zhang, D., Sánchez, J., Yambayev, I., Lindqvist, E., Virtala, R., Djukanovic, R., Davies, D. E., Wilson, S., O'Donnell, R., Cunoosamy, D., Hazon, P., Higham, A., Singh, D., Olsson, H., & Owen, C. A. (2018). A Disintegrin and A Metalloproteinase-9 (ADAM9): A Novel Proteinase Culprit with Multifarious Contributions to COPD. American journal of respiratory and critical care medicine, . https://doi.org/10.1164/rccm.201711-2300OC
Wang, Xiaoyun, et al. "A Disintegrin and A Metalloproteinase-9 (ADAM9): A Novel Proteinase Culprit with Multifarious Contributions to COPD." American journal of respiratory and critical care medicine vol. (2018). doi: https://doi.org/10.1164/rccm.201711-2300OC
Wang X, Polverino F, Rojas-Quintero J, Zhang D, Sánchez J, Yambayev I, Lindqvist E, Virtala R, Djukanovic R, Davies DE, Wilson S, O'Donnell R, Cunoosamy D, Hazon P, Higham A, Singh D, Olsson H, Owen CA. A Disintegrin and A Metalloproteinase-9 (ADAM9): A Novel Proteinase Culprit with Multifarious Contributions to COPD. Am J Respir Crit Care Med. 2018 Jun 04; doi: 10.1164/rccm.201711-2300OC. Epub 2018 Jun 04. PMID: 29864380; PMCID: PMC6298633.
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Chronic Obstructive Pulmonary Disease Genetics: A Review of the Past and a Look Into the Future.

Chronic obstructive pulmonary diseases (Miami, Fla.)

Hardin M, Silverman EK.
PMID: 28848809
Chronic Obstr Pulm Dis. 2014 May 06;1(1):33-46. doi: 10.15326/jcopdf.1.1.2014.0120.

Chronic obstructive pulmonary disease (COPD) affects over 10 million Americans.

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Hardin M, Silverman EK. Chronic Obstructive Pulmonary Disease Genetics: A Review of the Past and a Look Into the Future. Chronic Obstr Pulm Dis. 2014;1(1):33-46doi: 10.15326/jcopdf.1.1.2014.0120.
Hardin, M., & Silverman, E. K. (2014). Chronic Obstructive Pulmonary Disease Genetics: A Review of the Past and a Look Into the Future. Chronic obstructive pulmonary diseases (Miami, Fla.), 1(1), 33-46. https://doi.org/10.15326/jcopdf.1.1.2014.0120
Hardin, Megan, and Silverman, Edwin K. "Chronic Obstructive Pulmonary Disease Genetics: A Review of the Past and a Look Into the Future." Chronic obstructive pulmonary diseases (Miami, Fla.) vol. 1,1 (2014): 33-46. doi: https://doi.org/10.15326/jcopdf.1.1.2014.0120
Hardin M, Silverman EK. Chronic Obstructive Pulmonary Disease Genetics: A Review of the Past and a Look Into the Future. Chronic Obstr Pulm Dis. 2014 May 06;1(1):33-46. doi: 10.15326/jcopdf.1.1.2014.0120. PMID: 28848809; PMCID: PMC5559139.
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Folliculin regulates cell-cell adhesion, AMPK, and mTORC1 in a cell-type-specific manner in lung-derived cells.

Physiological reports

Khabibullin D, Medvetz DA, Pinilla M, Hariharan V, Li C, Hergrueter A, Laucho Contreras M, Zhang E, Parkhitko A, Yu JJ, Owen CA, Huang H, Baron RM, Henske EP.
PMID: 25121506
Physiol Rep. 2014 Aug 12;2(8). doi: 10.14814/phy2.12107. Print 2014 Aug 01.

Germline loss-of-function BHD mutations cause cystic lung disease and hereditary pneumothorax, yet little is known about the impact of BHD mutations in the lung. Folliculin (FLCN), the product of the Birt-Hogg-Dube (BHD) gene, has been linked to altered cell-cell...

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Khabibullin D, Medvetz DA, Pinilla M, et al. Folliculin regulates cell-cell adhesion, AMPK, and mTORC1 in a cell-type-specific manner in lung-derived cells. Physiol Rep. 2014;2(8)doi: 10.14814/phy2.12107.
Khabibullin, D., Medvetz, D. A., Pinilla, M., Hariharan, V., Li, C., Hergrueter, A., Laucho Contreras, M., Zhang, E., Parkhitko, A., Yu, J. J., Owen, C. A., Huang, H., Baron, R. M., & Henske, E. P. (2014). Folliculin regulates cell-cell adhesion, AMPK, and mTORC1 in a cell-type-specific manner in lung-derived cells. Physiological reports, 2(8), . https://doi.org/10.14814/phy2.12107
Khabibullin, Damir, et al. "Folliculin regulates cell-cell adhesion, AMPK, and mTORC1 in a cell-type-specific manner in lung-derived cells." Physiological reports vol. 2,8 (2014). doi: https://doi.org/10.14814/phy2.12107
Khabibullin D, Medvetz DA, Pinilla M, Hariharan V, Li C, Hergrueter A, Laucho Contreras M, Zhang E, Parkhitko A, Yu JJ, Owen CA, Huang H, Baron RM, Henske EP. Folliculin regulates cell-cell adhesion, AMPK, and mTORC1 in a cell-type-specific manner in lung-derived cells. Physiol Rep. 2014 Aug 12;2(8). doi: 10.14814/phy2.12107. Print 2014 Aug 01. PMID: 25121506; PMCID: PMC4246594.
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Haploinsufficiency of Hedgehog interacting protein causes increased emphysema induced by cigarette smoke through network rewiring.

Genome medicine

Lao T, Glass K, Qiu W, Polverino F, Gupta K, Morrow J, Mancini JD, Vuong L, Perrella MA, Hersh CP, Owen CA, Quackenbush J, Yuan GC, Silverman EK, Zhou X.
PMID: 25763110
Genome Med. 2015 Feb 14;7(1):12. doi: 10.1186/s13073-015-0137-3. eCollection 2015.

BACKGROUND: The HHIP gene, encoding Hedgehog interacting protein, has been implicated in chronic obstructive pulmonary disease (COPD) by genome-wide association studies (GWAS), and our subsequent studies identified a functional upstream genetic variant that decreased HHIP transcription. However, little is...

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Lao T, Glass K, Qiu W, et al. Haploinsufficiency of Hedgehog interacting protein causes increased emphysema induced by cigarette smoke through network rewiring. Genome Med. 2015;7(1):12doi: 10.1186/s13073-015-0137-3.
Lao, T., Glass, K., Qiu, W., Polverino, F., Gupta, K., Morrow, J., Mancini, J. D., Vuong, L., Perrella, M. A., Hersh, C. P., Owen, C. A., Quackenbush, J., Yuan, G. C., Silverman, E. K., & Zhou, X. (2015). Haploinsufficiency of Hedgehog interacting protein causes increased emphysema induced by cigarette smoke through network rewiring. Genome medicine, 7(1), 12. https://doi.org/10.1186/s13073-015-0137-3
Lao, Taotao, et al. "Haploinsufficiency of Hedgehog interacting protein causes increased emphysema induced by cigarette smoke through network rewiring." Genome medicine vol. 7,1 (2015): 12. doi: https://doi.org/10.1186/s13073-015-0137-3
Lao T, Glass K, Qiu W, Polverino F, Gupta K, Morrow J, Mancini JD, Vuong L, Perrella MA, Hersh CP, Owen CA, Quackenbush J, Yuan GC, Silverman EK, Zhou X. Haploinsufficiency of Hedgehog interacting protein causes increased emphysema induced by cigarette smoke through network rewiring. Genome Med. 2015 Feb 14;7(1):12. doi: 10.1186/s13073-015-0137-3. eCollection 2015. PMID: 25763110; PMCID: PMC4355149.
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Risk loci for chronic obstructive pulmonary disease: a genome-wide association study and meta-analysis.

The Lancet. Respiratory medicine

Cho MH, McDonald ML, Zhou X, Mattheisen M, Castaldi PJ, Hersh CP, Demeo DL, Sylvia JS, Ziniti J, Laird NM, Lange C, Litonjua AA, Sparrow D, Casaburi R, Barr RG, Regan EA, Make BJ, Hokanson JE, Lutz S, Dudenkov TM, Farzadegan H, Hetmanski JB, Tal-Singer R, Lomas DA, Bakke P, Gulsvik A, Crapo JD, Silverman EK, Beaty TH.
PMID: 24621683
Lancet Respir Med. 2014 Mar;2(3):214-25. doi: 10.1016/S2213-2600(14)70002-5. Epub 2014 Feb 07.

BACKGROUND: The genetic risk factors for susceptibility to chronic obstructive pulmonary disease (COPD) are still largely unknown. Additional genetic variants are likely to be identified by genome-wide association studies in larger cohorts or specific subgroups. We sought to identify...

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Cho MH, McDonald ML, Zhou X, et al. Risk loci for chronic obstructive pulmonary disease: a genome-wide association study and meta-analysis. Lancet Respir Med. 2014;2(3):214-25doi: 10.1016/S2213-2600(14)70002-5.
Cho, M. H., McDonald, M. L., Zhou, X., Mattheisen, M., Castaldi, P. J., Hersh, C. P., Demeo, D. L., Sylvia, J. S., Ziniti, J., Laird, N. M., Lange, C., Litonjua, A. A., Sparrow, D., Casaburi, R., Barr, R. G., Regan, E. A., Make, B. J., Hokanson, J. E., Lutz, S., Dudenkov, T. M., Farzadegan, H., Hetmanski, J. B., Tal-Singer, R., Lomas, D. A., Bakke, P., Gulsvik, A., Crapo, J. D., Silverman, E. K., Beaty, T. H. (2014). Risk loci for chronic obstructive pulmonary disease: a genome-wide association study and meta-analysis. The Lancet. Respiratory medicine, 2(3), 214-25. https://doi.org/10.1016/S2213-2600(14)70002-5
Cho, Michael H, et al. "Risk loci for chronic obstructive pulmonary disease: a genome-wide association study and meta-analysis." The Lancet. Respiratory medicine vol. 2,3 (2014): 214-25. doi: https://doi.org/10.1016/S2213-2600(14)70002-5
Cho MH, McDonald ML, Zhou X, Mattheisen M, Castaldi PJ, Hersh CP, Demeo DL, Sylvia JS, Ziniti J, Laird NM, Lange C, Litonjua AA, Sparrow D, Casaburi R, Barr RG, Regan EA, Make BJ, Hokanson JE, Lutz S, Dudenkov TM, Farzadegan H, Hetmanski JB, Tal-Singer R, Lomas DA, Bakke P, Gulsvik A, Crapo JD, Silverman EK, Beaty TH. Risk loci for chronic obstructive pulmonary disease: a genome-wide association study and meta-analysis. Lancet Respir Med. 2014 Mar;2(3):214-25. doi: 10.1016/S2213-2600(14)70002-5. Epub 2014 Feb 07. PMID: 24621683; PMCID: PMC4176924.
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Increased expression of A Proliferation-inducing Ligand (APRIL) in lung leukocytes and alveolar epithelial cells in COPD patients with non small cell lung cancer: a possible link between COPD and lung cancer?.

Multidisciplinary respiratory medicine

Polverino F, Laucho-Contreras M, Rojas Quintero J, Divo M, Pinto-Plata V, Sholl L, de-Torres JP, Celli BR, Owen CA.
PMID: 27047662
Multidiscip Respir Med. 2016 Apr 04;11:17. doi: 10.1186/s40248-016-0051-6. eCollection 2016.

BACKGROUND: Chronic Obstructive Pulmonary Disease (COPD) is characterized by an excessive activation of the adaptive immune system and, in particular, uncontrolled expansion of the B-cell pool. One of the key promoters of B cell expansion is A PRoliferation-Inducing Ligand...

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Polverino F, Laucho-Contreras M, Rojas Quintero J, et al. Increased expression of A Proliferation-inducing Ligand (APRIL) in lung leukocytes and alveolar epithelial cells in COPD patients with non small cell lung cancer: a possible link between COPD and lung cancer?. Multidiscip Respir Med. 2016;11:17doi: 10.1186/s40248-016-0051-6.
Polverino, F., Laucho-Contreras, M., Rojas Quintero, J., Divo, M., Pinto-Plata, V., Sholl, L., de-Torres, J. P., Celli, B. R., & Owen, C. A. (2016). Increased expression of A Proliferation-inducing Ligand (APRIL) in lung leukocytes and alveolar epithelial cells in COPD patients with non small cell lung cancer: a possible link between COPD and lung cancer?. Multidisciplinary respiratory medicine, 1117. https://doi.org/10.1186/s40248-016-0051-6
Polverino, Francesca, et al. "Increased expression of A Proliferation-inducing Ligand (APRIL) in lung leukocytes and alveolar epithelial cells in COPD patients with non small cell lung cancer: a possible link between COPD and lung cancer?." Multidisciplinary respiratory medicine vol. 11 (2016): 17. doi: https://doi.org/10.1186/s40248-016-0051-6
Polverino F, Laucho-Contreras M, Rojas Quintero J, Divo M, Pinto-Plata V, Sholl L, de-Torres JP, Celli BR, Owen CA. Increased expression of A Proliferation-inducing Ligand (APRIL) in lung leukocytes and alveolar epithelial cells in COPD patients with non small cell lung cancer: a possible link between COPD and lung cancer?. Multidiscip Respir Med. 2016 Apr 04;11:17. doi: 10.1186/s40248-016-0051-6. eCollection 2016. PMID: 27047662; PMCID: PMC4819280.
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Sex-specific associations with DNA methylation in lung tissue demonstrate smoking interactions.

Epigenetics

Koo HK, Morrow J, Kachroo P, Tantisira K, Weiss ST, Hersh CP, Silverman EK, DeMeo DL.
PMID: 32962511
Epigenetics. 2021 Jun;16(6):692-703. doi: 10.1080/15592294.2020.1819662. Epub 2020 Sep 22.

Cigarette smoking impacts DNA methylation, but the investigation of sex-specific features of lung tissue DNA methylation in smokers has been limited. Women appear more susceptible to cigarette smoke, and often develop more severe lung disease at an earlier age...

Cite
Koo HK, Morrow J, Kachroo P, et al. Sex-specific associations with DNA methylation in lung tissue demonstrate smoking interactions. Epigenetics. 2020;16(6):692-703doi: 10.1080/15592294.2020.1819662.
Koo, H. K., Morrow, J., Kachroo, P., Tantisira, K., Weiss, S. T., Hersh, C. P., Silverman, E. K., & DeMeo, D. L. (2021). Sex-specific associations with DNA methylation in lung tissue demonstrate smoking interactions. Epigenetics, 16(6), 692-703. https://doi.org/10.1080/15592294.2020.1819662
Koo, Hyeon-Kyoung, et al. "Sex-specific associations with DNA methylation in lung tissue demonstrate smoking interactions." Epigenetics vol. 16,6 (2021): 692-703. doi: https://doi.org/10.1080/15592294.2020.1819662
Koo HK, Morrow J, Kachroo P, Tantisira K, Weiss ST, Hersh CP, Silverman EK, DeMeo DL. Sex-specific associations with DNA methylation in lung tissue demonstrate smoking interactions. Epigenetics. 2021 Jun;16(6):692-703. doi: 10.1080/15592294.2020.1819662. Epub 2020 Sep 22. PMID: 32962511; PMCID: PMC8143227.
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