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G protein-coupled Receptor Kinase 2 as a Therapeutic Target for Heart Failure.

Drug discovery today. Therapeutic strategies

Schumacher-Bass SM, Traynham CJ, Koch WJ.
PMID: 24839449
Drug Discov Today Ther Strateg. 2012;9(4):e155-e162. doi: 10.1016/j.ddstr.2014.01.002.

An ever-increasing number of people world-wide are developing and suffering from heart failure, and existing therapies, although improved are not ideal. Therefore, innovative treatment strategies are urgently needed. As our understanding of the underlying dysfunction of the myocardium increases,...

Cite
Schumacher-Bass SM, Traynham CJ, Koch WJ. G protein-coupled Receptor Kinase 2 as a Therapeutic Target for Heart Failure. Drug Discov Today Ther Strateg. 2012;9(4):e155-e162doi: 10.1016/j.ddstr.2014.01.002.
Schumacher-Bass, S. M., Traynham, C. J., & Koch, W. J. (2012). G protein-coupled Receptor Kinase 2 as a Therapeutic Target for Heart Failure. Drug discovery today. Therapeutic strategies, 9(4), e155-e162. https://doi.org/10.1016/j.ddstr.2014.01.002
Schumacher-Bass, Sarah M, et al. "G protein-coupled Receptor Kinase 2 as a Therapeutic Target for Heart Failure." Drug discovery today. Therapeutic strategies vol. 9,4 (2012): e155-e162. doi: https://doi.org/10.1016/j.ddstr.2014.01.002
Schumacher-Bass SM, Traynham CJ, Koch WJ. G protein-coupled Receptor Kinase 2 as a Therapeutic Target for Heart Failure. Drug Discov Today Ther Strateg. 2012;9(4):e155-e162. doi: 10.1016/j.ddstr.2014.01.002. PMID: 24839449; PMCID: PMC4019414.
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Protein Kinase C Inhibition With Ruboxistaurin Increases Contractility and Reduces Heart Size in a Swine Model of Heart Failure With Reduced Ejection Fraction.

JACC. Basic to translational science

Sharp TE, Kubo H, Berretta RM, Starosta T, Wallner M, Schena GJ, Hobby AR, Yu D, Trappanese DM, George JC, Molkentin JD, Houser SR.
PMID: 30062182
JACC Basic Transl Sci. 2017 Dec 25;2(6):669-683. doi: 10.1016/j.jacbts.2017.06.007. eCollection 2017 Dec.

Inotropic support is often required to stabilize the hemodynamics of patients with acute decompensated heart failure; while efficacious, it has a history of leading to lethal arrhythmias and/or exacerbating contractile and energetic insufficiencies. Novel therapeutics that can improve contractility...

Cite
Sharp TE, Kubo H, Berretta RM, et al. Protein Kinase C Inhibition With Ruboxistaurin Increases Contractility and Reduces Heart Size in a Swine Model of Heart Failure With Reduced Ejection Fraction. JACC Basic Transl Sci. 2017;2(6):669-683doi: 10.1016/j.jacbts.2017.06.007.
Sharp, T. E., Kubo, H., Berretta, R. M., Starosta, T., Wallner, M., Schena, G. J., Hobby, A. R., Yu, D., Trappanese, D. M., George, J. C., Molkentin, J. D., & Houser, S. R. (2017). Protein Kinase C Inhibition With Ruboxistaurin Increases Contractility and Reduces Heart Size in a Swine Model of Heart Failure With Reduced Ejection Fraction. JACC. Basic to translational science, 2(6), 669-683. https://doi.org/10.1016/j.jacbts.2017.06.007
Sharp, Thomas E, et al. "Protein Kinase C Inhibition With Ruboxistaurin Increases Contractility and Reduces Heart Size in a Swine Model of Heart Failure With Reduced Ejection Fraction." JACC. Basic to translational science vol. 2,6 (2017): 669-683. doi: https://doi.org/10.1016/j.jacbts.2017.06.007
Sharp TE, Kubo H, Berretta RM, Starosta T, Wallner M, Schena GJ, Hobby AR, Yu D, Trappanese DM, George JC, Molkentin JD, Houser SR. Protein Kinase C Inhibition With Ruboxistaurin Increases Contractility and Reduces Heart Size in a Swine Model of Heart Failure With Reduced Ejection Fraction. JACC Basic Transl Sci. 2017 Dec 25;2(6):669-683. doi: 10.1016/j.jacbts.2017.06.007. eCollection 2017 Dec. PMID: 30062182; PMCID: PMC6058945.
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