Advanced Search
Display options
Filter resources
Text Availability
Article type
Publication date
Species
Language
Sex
Age
Showing 1 to 12 of 22 entries
Sorted by: Best Match Show Resources per page
Chitotriosidase in the Pathogenesis of Inflammation, Interstitial Lung Diseases and COPD.

Allergy, asthma & immunology research

Cho SJ, Weiden MD, Lee CG.
PMID: 25553258
Allergy Asthma Immunol Res. 2015 Jan;7(1):14-21. doi: 10.4168/aair.2015.7.1.14. Epub 2014 Nov 05.

As a member of 18 glycosyl hydrolase (GH) family, chitotriosidase (Chitinase 1, CHIT1) is a true chitinase mainly expressed in the differentiated and polarized macrophages. CHIT1 is an innate immune mediator that digests the cell walls of chitin-containing eukaryotic...

Cite
Cho SJ, Weiden MD, Lee CG. Chitotriosidase in the Pathogenesis of Inflammation, Interstitial Lung Diseases and COPD. Allergy Asthma Immunol Res. 2014;7(1):14-21doi: 10.4168/aair.2015.7.1.14.
Cho, S. J., Weiden, M. D., & Lee, C. G. (2015). Chitotriosidase in the Pathogenesis of Inflammation, Interstitial Lung Diseases and COPD. Allergy, asthma & immunology research, 7(1), 14-21. https://doi.org/10.4168/aair.2015.7.1.14
Cho, Soo Jung, et al. "Chitotriosidase in the Pathogenesis of Inflammation, Interstitial Lung Diseases and COPD." Allergy, asthma & immunology research vol. 7,1 (2015): 14-21. doi: https://doi.org/10.4168/aair.2015.7.1.14
Cho SJ, Weiden MD, Lee CG. Chitotriosidase in the Pathogenesis of Inflammation, Interstitial Lung Diseases and COPD. Allergy Asthma Immunol Res. 2015 Jan;7(1):14-21. doi: 10.4168/aair.2015.7.1.14. Epub 2014 Nov 05. PMID: 25553258; PMCID: PMC4274464.
Copy Download .nbib Format: NLM
Estimating gene regulatory networks with pandaR.

Bioinformatics (Oxford, England)

Schlauch D, Paulson JN, Young A, Glass K, Quackenbush J.
PMID: 28334344
Bioinformatics. 2017 Jul 15;33(14):2232-2234. doi: 10.1093/bioinformatics/btx139.

CONTACT: [email protected] or [email protected] AND IMPLEMENTATION: PandaR is provided as a Bioconductor R Package and is available at bioconductor.org/packages/pandaR.

Cite
Schlauch D, Paulson JN, Young A, et al. Estimating gene regulatory networks with pandaR. Bioinformatics. 2017;33(14):2232-2234doi: 10.1093/bioinformatics/btx139.
Schlauch, D., Paulson, J. N., Young, A., Glass, K., & Quackenbush, J. (2017). Estimating gene regulatory networks with pandaR. Bioinformatics (Oxford, England), 33(14), 2232-2234. https://doi.org/10.1093/bioinformatics/btx139
Schlauch, Daniel, et al. "Estimating gene regulatory networks with pandaR." Bioinformatics (Oxford, England) vol. 33,14 (2017): 2232-2234. doi: https://doi.org/10.1093/bioinformatics/btx139
Schlauch D, Paulson JN, Young A, Glass K, Quackenbush J. Estimating gene regulatory networks with pandaR. Bioinformatics. 2017 Jul 15;33(14):2232-2234. doi: 10.1093/bioinformatics/btx139. PMID: 28334344; PMCID: PMC5870629.
Copy Download .nbib Format: NLM
Single-cell RNA-seq reveals ectopic and aberrant lung-resident cell populations in idiopathic pulmonary fibrosis.

Science advances

Adams TS, Schupp JC, Poli S, Ayaub EA, Neumark N, Ahangari F, Chu SG, Raby BA, DeIuliis G, Januszyk M, Duan Q, Arnett HA, Siddiqui A, Washko GR, Homer R, Yan X, Rosas IO, Kaminski N.
PMID: 32832599
Sci Adv. 2020 Jul 08;6(28):eaba1983. doi: 10.1126/sciadv.aba1983. eCollection 2020 Jul.

We provide a single-cell atlas of idiopathic pulmonary fibrosis (IPF), a fatal interstitial lung disease, by profiling 312,928 cells from 32 IPF, 28 smoker and nonsmoker controls, and 18 chronic obstructive pulmonary disease (COPD) lungs. Among epithelial cells enriched...

Cite
Adams TS, Schupp JC, Poli S, et al. Single-cell RNA-seq reveals ectopic and aberrant lung-resident cell populations in idiopathic pulmonary fibrosis. Sci Adv. 2020;6(28):eaba1983doi: 10.1126/sciadv.aba1983.
Adams, T. S., Schupp, J. C., Poli, S., Ayaub, E. A., Neumark, N., Ahangari, F., Chu, S. G., Raby, B. A., DeIuliis, G., Januszyk, M., Duan, Q., Arnett, H. A., Siddiqui, A., Washko, G. R., Homer, R., Yan, X., Rosas, I. O., & Kaminski, N. (2020). Single-cell RNA-seq reveals ectopic and aberrant lung-resident cell populations in idiopathic pulmonary fibrosis. Science advances, 6(28), eaba1983. https://doi.org/10.1126/sciadv.aba1983
Adams, Taylor S, et al. "Single-cell RNA-seq reveals ectopic and aberrant lung-resident cell populations in idiopathic pulmonary fibrosis." Science advances vol. 6,28 (2020): eaba1983. doi: https://doi.org/10.1126/sciadv.aba1983
Adams TS, Schupp JC, Poli S, Ayaub EA, Neumark N, Ahangari F, Chu SG, Raby BA, DeIuliis G, Januszyk M, Duan Q, Arnett HA, Siddiqui A, Washko GR, Homer R, Yan X, Rosas IO, Kaminski N. Single-cell RNA-seq reveals ectopic and aberrant lung-resident cell populations in idiopathic pulmonary fibrosis. Sci Adv. 2020 Jul 08;6(28):eaba1983. doi: 10.1126/sciadv.aba1983. eCollection 2020 Jul. PMID: 32832599; PMCID: PMC7439502.
Copy Download .nbib Format: NLM
The Association of Multiparity with Lung Function and Chronic Obstructive Pulmonary Disease-Related Phenotypes.

Chronic obstructive pulmonary diseases (Miami, Fla.)

Moll M, Regan EA, Hokanson JE, Lutz SM, Silverman EK, Crapo JD, Make BJ, DeMeo DL.
PMID: 32324980
Chronic Obstr Pulm Dis. 2020 Apr;7(2):86-98. doi: 10.15326/jcopdf.7.2.2019.0166.

BACKGROUND: Apparent increased female susceptibility to chronic obstructive pulmonary disease (COPD) suggests sex hormones modulate disease pathogenesis. Little is known about associations between multiparity and lung function in smokers.RESEARCH QUESTION: We hypothesized that multiparity is associated with lung function...

Cite
Moll M, Regan EA, Hokanson JE, et al. The Association of Multiparity with Lung Function and Chronic Obstructive Pulmonary Disease-Related Phenotypes. Chronic Obstr Pulm Dis. 2020;7(2):86-98doi: 10.15326/jcopdf.7.2.2019.0166.
Moll, M., Regan, E. A., Hokanson, J. E., Lutz, S. M., Silverman, E. K., Crapo, J. D., Make, B. J., & DeMeo, D. L. (2020). The Association of Multiparity with Lung Function and Chronic Obstructive Pulmonary Disease-Related Phenotypes. Chronic obstructive pulmonary diseases (Miami, Fla.), 7(2), 86-98. https://doi.org/10.15326/jcopdf.7.2.2019.0166
Moll, Matthew, et al. "The Association of Multiparity with Lung Function and Chronic Obstructive Pulmonary Disease-Related Phenotypes." Chronic obstructive pulmonary diseases (Miami, Fla.) vol. 7,2 (2020): 86-98. doi: https://doi.org/10.15326/jcopdf.7.2.2019.0166
Moll M, Regan EA, Hokanson JE, Lutz SM, Silverman EK, Crapo JD, Make BJ, DeMeo DL. The Association of Multiparity with Lung Function and Chronic Obstructive Pulmonary Disease-Related Phenotypes. Chronic Obstr Pulm Dis. 2020 Apr;7(2):86-98. doi: 10.15326/jcopdf.7.2.2019.0166. PMID: 32324980; PMCID: PMC7454017.
Copy Download .nbib Format: NLM
Genetic Variation in the Mitochondrial Glycerol-3-Phosphate Acyltransferase Is Associated With Liver Injury.

Hepatology (Baltimore, Md.)

Hakim A, Moll M, Brancale J, Liu J, Lasky-Su JA, Silverman EK, Vilarinho S, Jiang ZG, Pita-Juárez YH, Vlachos IS, Zhang X, Åberg F, Afdhal NH, Hobbs BD, Cho MH.
PMID: 34216018
Hepatology. 2021 Dec;74(6):3394-3408. doi: 10.1002/hep.32038. Epub 2021 Nov 09.

BACKGROUND AND AIMS: Most of the genetic basis of chronic liver disease remains undiscovered.APPROACH AND RESULTS: To identify genetic loci that modulate the risk of liver injury, we performed genome-wide association studies on circulating levels of alanine aminotransferase (ALT),...

Cite
Hakim A, Moll M, Brancale J, et al. Genetic Variation in the Mitochondrial Glycerol-3-Phosphate Acyltransferase Is Associated With Liver Injury. Hepatology. 2021;74(6):3394-3408doi: 10.1002/hep.32038.
Hakim, A., Moll, M., Brancale, J., Liu, J., Lasky-Su, J. A., Silverman, E. K., Vilarinho, S., Jiang, Z. G., Pita-Juárez, Y. H., Vlachos, I. S., Zhang, X., Åberg, F., Afdhal, N. H., Hobbs, B. D., & Cho, M. H. (2021). Genetic Variation in the Mitochondrial Glycerol-3-Phosphate Acyltransferase Is Associated With Liver Injury. Hepatology (Baltimore, Md.), 74(6), 3394-3408. https://doi.org/10.1002/hep.32038
Hakim, Aaron, et al. "Genetic Variation in the Mitochondrial Glycerol-3-Phosphate Acyltransferase Is Associated With Liver Injury." Hepatology (Baltimore, Md.) vol. 74,6 (2021): 3394-3408. doi: https://doi.org/10.1002/hep.32038
Hakim A, Moll M, Brancale J, Liu J, Lasky-Su JA, Silverman EK, Vilarinho S, Jiang ZG, Pita-Juárez YH, Vlachos IS, Zhang X, Åberg F, Afdhal NH, Hobbs BD, Cho MH. Genetic Variation in the Mitochondrial Glycerol-3-Phosphate Acyltransferase Is Associated With Liver Injury. Hepatology. 2021 Dec;74(6):3394-3408. doi: 10.1002/hep.32038. Epub 2021 Nov 09. PMID: 34216018; PMCID: PMC8639615.
Copy Download .nbib Format: NLM
Lung proteomic biomarkers associated with chronic obstructive pulmonary disease.

American journal of physiology. Lung cellular and molecular physiology

Zhang YH, Hoopmann MR, Castaldi PJ, Simonsen KA, Midha MK, Cho MH, Criner GJ, Bueno R, Liu J, Moritz RL, Silverman EK.
PMID: 34668408
Am J Physiol Lung Cell Mol Physiol. 2021 Dec 01;321(6):L1119-L1130. doi: 10.1152/ajplung.00198.2021. Epub 2021 Oct 20.

Identifying protein biomarkers for chronic obstructive pulmonary disease (COPD) has been challenging. Most previous studies have used individual proteins or preselected protein panels measured in blood samples. Mass spectrometry proteomic studies of lung tissue have been based on small...

Cite
Zhang YH, Hoopmann MR, Castaldi PJ, et al. Lung proteomic biomarkers associated with chronic obstructive pulmonary disease. Am J Physiol Lung Cell Mol Physiol. 2021;321(6):L1119-L1130doi: 10.1152/ajplung.00198.2021.
Zhang, Y. H., Hoopmann, M. R., Castaldi, P. J., Simonsen, K. A., Midha, M. K., Cho, M. H., Criner, G. J., Bueno, R., Liu, J., Moritz, R. L., & Silverman, E. K. (2021). Lung proteomic biomarkers associated with chronic obstructive pulmonary disease. American journal of physiology. Lung cellular and molecular physiology, 321(6), L1119-L1130. https://doi.org/10.1152/ajplung.00198.2021
Zhang, Yu-Hang, et al. "Lung proteomic biomarkers associated with chronic obstructive pulmonary disease." American journal of physiology. Lung cellular and molecular physiology vol. 321,6 (2021): L1119-L1130. doi: https://doi.org/10.1152/ajplung.00198.2021
Zhang YH, Hoopmann MR, Castaldi PJ, Simonsen KA, Midha MK, Cho MH, Criner GJ, Bueno R, Liu J, Moritz RL, Silverman EK. Lung proteomic biomarkers associated with chronic obstructive pulmonary disease. Am J Physiol Lung Cell Mol Physiol. 2021 Dec 01;321(6):L1119-L1130. doi: 10.1152/ajplung.00198.2021. Epub 2021 Oct 20. PMID: 34668408.
Copy Download .nbib Format: NLM
Idiopathic pulmonary fibrosis and coronary artery disease.

Multidisciplinary respiratory medicine

Cicchitto G, Musella V, Acitorio M, Capuano N, Fiorenzano G, Owen CA, Polverino M, Polverino F.
PMID: 24976970
Multidiscip Respir Med. 2014 May 30;9(1):31. doi: 10.1186/2049-6958-9-31. eCollection 2014.

Idiopathic pulmonary fibrosis (IPF) is defined as a chronic fibrosing interstitial disease of unknown cause, limited to the lungs, and associated with the histopathologic and/or radiologic pattern of usual interstitial pneumonia (UIP); it generally progresses into respiratory failure and...

Cite
Cicchitto G, Musella V, Acitorio M, et al. Idiopathic pulmonary fibrosis and coronary artery disease. Multidiscip Respir Med. 2014;9(1):31doi: 10.1186/2049-6958-9-31.
Cicchitto, G., Musella, V., Acitorio, M., Capuano, N., Fiorenzano, G., Owen, C. A., Polverino, M., & Polverino, F. (2014). Idiopathic pulmonary fibrosis and coronary artery disease. Multidisciplinary respiratory medicine, 9(1), 31. https://doi.org/10.1186/2049-6958-9-31
Cicchitto, Gaetano, et al. "Idiopathic pulmonary fibrosis and coronary artery disease." Multidisciplinary respiratory medicine vol. 9,1 (2014): 31. doi: https://doi.org/10.1186/2049-6958-9-31
Cicchitto G, Musella V, Acitorio M, Capuano N, Fiorenzano G, Owen CA, Polverino M, Polverino F. Idiopathic pulmonary fibrosis and coronary artery disease. Multidiscip Respir Med. 2014 May 30;9(1):31. doi: 10.1186/2049-6958-9-31. eCollection 2014. PMID: 24976970; PMCID: PMC4074111.
Copy Download .nbib Format: NLM
Sex-specific associations with DNA methylation in lung tissue demonstrate smoking interactions.

Epigenetics

Koo HK, Morrow J, Kachroo P, Tantisira K, Weiss ST, Hersh CP, Silverman EK, DeMeo DL.
PMID: 32962511
Epigenetics. 2021 Jun;16(6):692-703. doi: 10.1080/15592294.2020.1819662. Epub 2020 Sep 22.

Cigarette smoking impacts DNA methylation, but the investigation of sex-specific features of lung tissue DNA methylation in smokers has been limited. Women appear more susceptible to cigarette smoke, and often develop more severe lung disease at an earlier age...

Cite
Koo HK, Morrow J, Kachroo P, et al. Sex-specific associations with DNA methylation in lung tissue demonstrate smoking interactions. Epigenetics. 2020;16(6):692-703doi: 10.1080/15592294.2020.1819662.
Koo, H. K., Morrow, J., Kachroo, P., Tantisira, K., Weiss, S. T., Hersh, C. P., Silverman, E. K., & DeMeo, D. L. (2021). Sex-specific associations with DNA methylation in lung tissue demonstrate smoking interactions. Epigenetics, 16(6), 692-703. https://doi.org/10.1080/15592294.2020.1819662
Koo, Hyeon-Kyoung, et al. "Sex-specific associations with DNA methylation in lung tissue demonstrate smoking interactions." Epigenetics vol. 16,6 (2021): 692-703. doi: https://doi.org/10.1080/15592294.2020.1819662
Koo HK, Morrow J, Kachroo P, Tantisira K, Weiss ST, Hersh CP, Silverman EK, DeMeo DL. Sex-specific associations with DNA methylation in lung tissue demonstrate smoking interactions. Epigenetics. 2021 Jun;16(6):692-703. doi: 10.1080/15592294.2020.1819662. Epub 2020 Sep 22. PMID: 32962511; PMCID: PMC8143227.
Copy Download .nbib Format: NLM
Genetics of COPD.

Annual review of physiology

Silverman EK.
PMID: 31730394
Annu Rev Physiol. 2020 Feb 10;82:413-431. doi: 10.1146/annurev-physiol-021317-121224. Epub 2019 Nov 15.

Although chronic obstructive pulmonary disease (COPD) risk is strongly influenced by cigarette smoking, genetic factors are also important determinants of COPD. In addition to Mendelian syndromes such as alpha-1 antitrypsin deficiency, many genomic regions that influence COPD susceptibility have...

Cite
Silverman EK. Genetics of COPD. Annu Rev Physiol. 2019;82:413-431doi: 10.1146/annurev-physiol-021317-121224.
Silverman, E. K. (2020). Genetics of COPD. Annual review of physiology, 82413-431. https://doi.org/10.1146/annurev-physiol-021317-121224
Silverman, Edwin K. "Genetics of COPD." Annual review of physiology vol. 82 (2020): 413-431. doi: https://doi.org/10.1146/annurev-physiol-021317-121224
Silverman EK. Genetics of COPD. Annu Rev Physiol. 2020 Feb 10;82:413-431. doi: 10.1146/annurev-physiol-021317-121224. Epub 2019 Nov 15. PMID: 31730394; PMCID: PMC7193187.
Copy Download .nbib Format: NLM
Lung proteomic biomarkers associated with chronic obstructive pulmonary disease.

American journal of physiology. Lung cellular and molecular physiology

Zhang YH, Hoopmann MR, Castaldi PJ, Simonsen KA, Midha MK, Cho MH, Criner GJ, Bueno R, Liu J, Moritz RL, Silverman EK.
PMID: 34668408
Am J Physiol Lung Cell Mol Physiol. 2021 Dec 01;321(6):L1119-L1130. doi: 10.1152/ajplung.00198.2021. Epub 2021 Oct 20.

Identifying protein biomarkers for chronic obstructive pulmonary disease (COPD) has been challenging. Most previous studies have used individual proteins or preselected protein panels measured in blood samples. Mass spectrometry proteomic studies of lung tissue have been based on small...

Cite
Zhang YH, Hoopmann MR, Castaldi PJ, et al. Lung proteomic biomarkers associated with chronic obstructive pulmonary disease. Am J Physiol Lung Cell Mol Physiol. 2021;321(6):L1119-L1130doi: 10.1152/ajplung.00198.2021.
Zhang, Y. H., Hoopmann, M. R., Castaldi, P. J., Simonsen, K. A., Midha, M. K., Cho, M. H., Criner, G. J., Bueno, R., Liu, J., Moritz, R. L., & Silverman, E. K. (2021). Lung proteomic biomarkers associated with chronic obstructive pulmonary disease. American journal of physiology. Lung cellular and molecular physiology, 321(6), L1119-L1130. https://doi.org/10.1152/ajplung.00198.2021
Zhang, Yu-Hang, et al. "Lung proteomic biomarkers associated with chronic obstructive pulmonary disease." American journal of physiology. Lung cellular and molecular physiology vol. 321,6 (2021): L1119-L1130. doi: https://doi.org/10.1152/ajplung.00198.2021
Zhang YH, Hoopmann MR, Castaldi PJ, Simonsen KA, Midha MK, Cho MH, Criner GJ, Bueno R, Liu J, Moritz RL, Silverman EK. Lung proteomic biomarkers associated with chronic obstructive pulmonary disease. Am J Physiol Lung Cell Mol Physiol. 2021 Dec 01;321(6):L1119-L1130. doi: 10.1152/ajplung.00198.2021. Epub 2021 Oct 20. PMID: 34668408.
Copy Download .nbib Format: NLM
CHI3L1 regulates PD-L1 and anti-CHI3L1-PD-1 antibody elicits synergistic antitumor responses.

The Journal of clinical investigation

Ma B, Akosman B, Kamle S, Lee CM, He CH, Koo JS, Lee CG, Elias JA.
PMID: 34720089
J Clin Invest. 2021 Nov 01;131(21). doi: 10.1172/JCI137750.

Evasion of the immune response is a hallmark of cancer, and programmed cell death 1 (PD-1) and PD-1 ligand 1 (PD-L1) are major mediators of this immunosuppression. Chitinase 3-like 1 (CHI3L1) is induced in many cancers, where it portends...

Cite
Ma B, Akosman B, Kamle S, et al. CHI3L1 regulates PD-L1 and anti-CHI3L1-PD-1 antibody elicits synergistic antitumor responses. J Clin Invest. 2021;131(21)doi: 10.1172/JCI137750.
Ma, B., Akosman, B., Kamle, S., Lee, C. M., He, C. H., Koo, J. S., Lee, C. G., & Elias, J. A. (2021). CHI3L1 regulates PD-L1 and anti-CHI3L1-PD-1 antibody elicits synergistic antitumor responses. The Journal of clinical investigation, 131(21), . https://doi.org/10.1172/JCI137750
Ma, Bing, et al. "CHI3L1 regulates PD-L1 and anti-CHI3L1-PD-1 antibody elicits synergistic antitumor responses." The Journal of clinical investigation vol. 131,21 (2021). doi: https://doi.org/10.1172/JCI137750
Ma B, Akosman B, Kamle S, Lee CM, He CH, Koo JS, Lee CG, Elias JA. CHI3L1 regulates PD-L1 and anti-CHI3L1-PD-1 antibody elicits synergistic antitumor responses. J Clin Invest. 2021 Nov 01;131(21). doi: 10.1172/JCI137750. PMID: 34720089; PMCID: PMC8553560.
Copy Download .nbib Format: NLM
A Disintegrin and A Metalloproteinase-9 (ADAM9): A Novel Proteinase Culprit with Multifarious Contributions to COPD.

American journal of respiratory and critical care medicine

Wang X, Polverino F, Rojas-Quintero J, Zhang D, Sánchez J, Yambayev I, Lindqvist E, Virtala R, Djukanovic R, Davies DE, Wilson S, O'Donnell R, Cunoosamy D, Hazon P, Higham A, Singh D, Olsson H, Owen CA.
PMID: 29864380
Am J Respir Crit Care Med. 2018 Jun 04; doi: 10.1164/rccm.201711-2300OC. Epub 2018 Jun 04.

INTRODUCTION: Proteinases with a disintegrin and a metalloproteinase domain (ADAMs) have not been well studied in COPD. We investigated whether ADAM9 is linked to COPD in humans and mice.METHODS: ADAM9 blood and lung levels were measured in COPD patients...

Cite
Wang X, Polverino F, Rojas-Quintero J, et al. A Disintegrin and A Metalloproteinase-9 (ADAM9): A Novel Proteinase Culprit with Multifarious Contributions to COPD. Am J Respir Crit Care Med. 2018;doi: 10.1164/rccm.201711-2300OC.
Wang, X., Polverino, F., Rojas-Quintero, J., Zhang, D., Sánchez, J., Yambayev, I., Lindqvist, E., Virtala, R., Djukanovic, R., Davies, D. E., Wilson, S., O'Donnell, R., Cunoosamy, D., Hazon, P., Higham, A., Singh, D., Olsson, H., & Owen, C. A. (2018). A Disintegrin and A Metalloproteinase-9 (ADAM9): A Novel Proteinase Culprit with Multifarious Contributions to COPD. American journal of respiratory and critical care medicine, . https://doi.org/10.1164/rccm.201711-2300OC
Wang, Xiaoyun, et al. "A Disintegrin and A Metalloproteinase-9 (ADAM9): A Novel Proteinase Culprit with Multifarious Contributions to COPD." American journal of respiratory and critical care medicine vol. (2018). doi: https://doi.org/10.1164/rccm.201711-2300OC
Wang X, Polverino F, Rojas-Quintero J, Zhang D, Sánchez J, Yambayev I, Lindqvist E, Virtala R, Djukanovic R, Davies DE, Wilson S, O'Donnell R, Cunoosamy D, Hazon P, Higham A, Singh D, Olsson H, Owen CA. A Disintegrin and A Metalloproteinase-9 (ADAM9): A Novel Proteinase Culprit with Multifarious Contributions to COPD. Am J Respir Crit Care Med. 2018 Jun 04; doi: 10.1164/rccm.201711-2300OC. Epub 2018 Jun 04. PMID: 29864380; PMCID: PMC6298633.
Copy Download .nbib Format: NLM
Showing 1 to 12 of 22 entries