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Characterization of colloidal structures during intestinal lipolysis using small-angle neutron scattering.

Journal of colloid and interface science

Rezhdo O, Di Maio S, Le P, Littrell KC, Carrier RL, Chen SH.
PMID: 28384537
J Colloid Interface Sci. 2017 Aug 01;499:189-201. doi: 10.1016/j.jcis.2017.03.109. Epub 2017 Mar 30.

HYPOTHESIS: Bile micelles are thought to mediate intestinal absorption, in part by providing a phase into which compounds can partition. Solubilizing capacity of bile micelles is enhanced during the digestion of fat rich food. We hypothesized that the intestinal...

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Rezhdo O, Di Maio S, Le P, et al. Characterization of colloidal structures during intestinal lipolysis using small-angle neutron scattering. J Colloid Interface Sci. 2017;499:189-201doi: 10.1016/j.jcis.2017.03.109.
Rezhdo, O., Di Maio, S., Le, P., Littrell, K. C., Carrier, R. L., & Chen, S. H. (2017). Characterization of colloidal structures during intestinal lipolysis using small-angle neutron scattering. Journal of colloid and interface science, 499189-201. https://doi.org/10.1016/j.jcis.2017.03.109
Rezhdo, Oljora, et al. "Characterization of colloidal structures during intestinal lipolysis using small-angle neutron scattering." Journal of colloid and interface science vol. 499 (2017): 189-201. doi: https://doi.org/10.1016/j.jcis.2017.03.109
Rezhdo O, Di Maio S, Le P, Littrell KC, Carrier RL, Chen SH. Characterization of colloidal structures during intestinal lipolysis using small-angle neutron scattering. J Colloid Interface Sci. 2017 Aug 01;499:189-201. doi: 10.1016/j.jcis.2017.03.109. Epub 2017 Mar 30. PMID: 28384537; PMCID: PMC5627765.
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Toward an Objective Diagnostic Test for Bacterial Cellulitis.

PloS one

Pallin DJ, Bry L, Dwyer RC, Lipworth AD, Leung DY, Camargo CA, Kupper TS, Filbin MR, Murphy GF.
PMID: 27656884
PLoS One. 2016 Sep 22;11(9):e0162947. doi: 10.1371/journal.pone.0162947. eCollection 2016.

BACKGROUND: Prior studies repeatedly showed that cultures of skin lesions diagnosed as "cellulitis" are usually negative. However, lack of a gold standard for diagnosis (against which culture might be judged) and failure to assess the human immune response are...

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Pallin DJ, Bry L, Dwyer RC, et al. Toward an Objective Diagnostic Test for Bacterial Cellulitis. PLoS One. 2016;11(9):e0162947doi: 10.1371/journal.pone.0162947.
Pallin, D. J., Bry, L., Dwyer, R. C., Lipworth, A. D., Leung, D. Y., Camargo, C. A., Kupper, T. S., Filbin, M. R., & Murphy, G. F. (2016). Toward an Objective Diagnostic Test for Bacterial Cellulitis. PloS one, 11(9), e0162947. https://doi.org/10.1371/journal.pone.0162947
Pallin, Daniel J, et al. "Toward an Objective Diagnostic Test for Bacterial Cellulitis." PloS one vol. 11,9 (2016): e0162947. doi: https://doi.org/10.1371/journal.pone.0162947
Pallin DJ, Bry L, Dwyer RC, Lipworth AD, Leung DY, Camargo CA, Kupper TS, Filbin MR, Murphy GF. Toward an Objective Diagnostic Test for Bacterial Cellulitis. PLoS One. 2016 Sep 22;11(9):e0162947. doi: 10.1371/journal.pone.0162947. eCollection 2016. PMID: 27656884; PMCID: PMC5033594.
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Mass Spectrometry-Based (GeLC-MS/MS) Comparative Proteomic Analysis of Endoscopically (ePFT) Collected Pancreatic and Gastroduodenal Fluids.

Clinical and translational gastroenterology

Paulo JA, Kadiyala V, Banks PA, Steen H, Conwell DL.
PMID: 23238231
Clin Transl Gastroenterol. 2012 May 03;3:e14. doi: 10.1038/ctg.2012.7.

OBJECTIVES: The secretin-stimulated endoscopic pancreatic function test (ePFT) allows for the safe collection of gastroduodenal and pancreatic fluid from the duodenum. We test the hypothesis that these endoscopically collected fluids have different proteomes. As such, we aim to show...

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Paulo JA, Kadiyala V, Banks PA, et al. Mass Spectrometry-Based (GeLC-MS/MS) Comparative Proteomic Analysis of Endoscopically (ePFT) Collected Pancreatic and Gastroduodenal Fluids. Clin Transl Gastroenterol. 2012;3:e14doi: 10.1038/ctg.2012.7.
Paulo, J. A., Kadiyala, V., Banks, P. A., Steen, H., & Conwell, D. L. (2012). Mass Spectrometry-Based (GeLC-MS/MS) Comparative Proteomic Analysis of Endoscopically (ePFT) Collected Pancreatic and Gastroduodenal Fluids. Clinical and translational gastroenterology, 3e14. https://doi.org/10.1038/ctg.2012.7
Paulo, Joao A, et al. "Mass Spectrometry-Based (GeLC-MS/MS) Comparative Proteomic Analysis of Endoscopically (ePFT) Collected Pancreatic and Gastroduodenal Fluids." Clinical and translational gastroenterology vol. 3 (2012): e14. doi: https://doi.org/10.1038/ctg.2012.7
Paulo JA, Kadiyala V, Banks PA, Steen H, Conwell DL. Mass Spectrometry-Based (GeLC-MS/MS) Comparative Proteomic Analysis of Endoscopically (ePFT) Collected Pancreatic and Gastroduodenal Fluids. Clin Transl Gastroenterol. 2012 May 03;3:e14. doi: 10.1038/ctg.2012.7. PMID: 23238231; PMCID: PMC3367612.
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THE ROLE OF SPECIFIC IgG AND COMPLEMENT IN COMBATING A PRIMARY MUCOSAL INFECTION OF THE GUT EPITHELIUM.

European journal of microbiology & immunology

Belzer C, Liu Q, Carroll MC, Bry L.
PMID: 22485193
Eur J Microbiol Immunol (Bp). 2011 Dec;1(4):311-318. doi: 10.1556/EuJMI.1.2011.4.7. Epub 2011 Dec 23.

The role of complement and complement-fixing IgG isotypes at mucosal surfaces is ill defined. Previous data have demonstrated that survival of an infection with the attaching and effacing pathogen Citrobacter rodentium requires production of systemic and CD4+ T cell-dependent...

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Belzer C, Liu Q, Carroll MC, et al. THE ROLE OF SPECIFIC IgG AND COMPLEMENT IN COMBATING A PRIMARY MUCOSAL INFECTION OF THE GUT EPITHELIUM. Eur J Microbiol Immunol (Bp). 2011;1(4):311-318doi: 10.1556/EuJMI.1.2011.4.7.
Belzer, C., Liu, Q., Carroll, M. C., & Bry, L. (2011). THE ROLE OF SPECIFIC IgG AND COMPLEMENT IN COMBATING A PRIMARY MUCOSAL INFECTION OF THE GUT EPITHELIUM. European journal of microbiology & immunology, 1(4), 311-318. https://doi.org/10.1556/EuJMI.1.2011.4.7
Belzer, C, et al. "THE ROLE OF SPECIFIC IgG AND COMPLEMENT IN COMBATING A PRIMARY MUCOSAL INFECTION OF THE GUT EPITHELIUM." European journal of microbiology & immunology vol. 1,4 (2011): 311-318. doi: https://doi.org/10.1556/EuJMI.1.2011.4.7
Belzer C, Liu Q, Carroll MC, Bry L. THE ROLE OF SPECIFIC IgG AND COMPLEMENT IN COMBATING A PRIMARY MUCOSAL INFECTION OF THE GUT EPITHELIUM. Eur J Microbiol Immunol (Bp). 2011 Dec;1(4):311-318. doi: 10.1556/EuJMI.1.2011.4.7. Epub 2011 Dec 23. PMID: 22485193; PMCID: PMC3319158.
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Interferon (IFN)-λ Takes the Helm: Immunomodulatory Roles of Type III IFNs.

Frontiers in immunology

Zanoni I, Granucci F, Broggi A.
PMID: 29234323
Front Immunol. 2017 Nov 28;8:1661. doi: 10.3389/fimmu.2017.01661. eCollection 2017.

Type III interferons (IFNs) (or IFN-λ) are the latest addition to the IFN family. Even though they share little protein homology with type I IFN, both exhibit remarkable functional similarities: each can be induced in response to viral infections,...

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Zanoni I, Granucci F, Broggi A. Interferon (IFN)-λ Takes the Helm: Immunomodulatory Roles of Type III IFNs. Front Immunol. 2017;8:1661doi: 10.3389/fimmu.2017.01661.
Zanoni, I., Granucci, F., & Broggi, A. (2017). Interferon (IFN)-λ Takes the Helm: Immunomodulatory Roles of Type III IFNs. Frontiers in immunology, 81661. https://doi.org/10.3389/fimmu.2017.01661
Zanoni, Ivan, et al. "Interferon (IFN)-λ Takes the Helm: Immunomodulatory Roles of Type III IFNs." Frontiers in immunology vol. 8 (2017): 1661. doi: https://doi.org/10.3389/fimmu.2017.01661
Zanoni I, Granucci F, Broggi A. Interferon (IFN)-λ Takes the Helm: Immunomodulatory Roles of Type III IFNs. Front Immunol. 2017 Nov 28;8:1661. doi: 10.3389/fimmu.2017.01661. eCollection 2017. PMID: 29234323; PMCID: PMC5712353.
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Children's rare disease cohorts: an integrative research and clinical genomics initiative.

NPJ genomic medicine

Rockowitz S, LeCompte N, Carmack M, Quitadamo A, Wang L, Park M, Knight D, Sexton E, Smith L, Sheidley B, Field M, Holm IA, Brownstein CA, Agrawal PB, Kornetsky S, Poduri A, Snapper SB, Beggs AH, Yu TW, Williams DA, Sliz P.
PMID: 32655885
NPJ Genom Med. 2020 Jul 06;5:29. doi: 10.1038/s41525-020-0137-0. eCollection 2020.

While genomic data is frequently collected under distinct research protocols and disparate clinical and research regimes, there is a benefit in streamlining sequencing strategies to create harmonized databases, particularly in the area of pediatric rare disease. Research hospitals seeking...

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Rockowitz S, LeCompte N, Carmack M, et al. Children's rare disease cohorts: an integrative research and clinical genomics initiative. NPJ Genom Med. 2020;5:29doi: 10.1038/s41525-020-0137-0.
Rockowitz, S., LeCompte, N., Carmack, M., Quitadamo, A., Wang, L., Park, M., Knight, D., Sexton, E., Smith, L., Sheidley, B., Field, M., Holm, I. A., Brownstein, C. A., Agrawal, P. B., Kornetsky, S., Poduri, A., Snapper, S. B., Beggs, A. H., Yu, T. W., Williams, D. A., & Sliz, P. (2020). Children's rare disease cohorts: an integrative research and clinical genomics initiative. NPJ genomic medicine, 529. https://doi.org/10.1038/s41525-020-0137-0
Rockowitz, Shira, et al. "Children's rare disease cohorts: an integrative research and clinical genomics initiative." NPJ genomic medicine vol. 5 (2020): 29. doi: https://doi.org/10.1038/s41525-020-0137-0
Rockowitz S, LeCompte N, Carmack M, Quitadamo A, Wang L, Park M, Knight D, Sexton E, Smith L, Sheidley B, Field M, Holm IA, Brownstein CA, Agrawal PB, Kornetsky S, Poduri A, Snapper SB, Beggs AH, Yu TW, Williams DA, Sliz P. Children's rare disease cohorts: an integrative research and clinical genomics initiative. NPJ Genom Med. 2020 Jul 06;5:29. doi: 10.1038/s41525-020-0137-0. eCollection 2020. PMID: 32655885; PMCID: PMC7338382.
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Correction: p190 RhoGAP promotes contact inhibition in epithelial cells by repressing YAP activity.

The Journal of cell biology

Frank SR, Köllmann CP, Luong P, Galli GG, Zou L, Bernards A, Getz G, Calogero RA, Frödin M, Hansen SH.
PMID: 30045860
J Cell Biol. 2018 Sep 03;217(9):3313. doi: 10.1083/JCB.20171005807172018c. Epub 2018 Jul 25.

No abstract available.

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Frank SR, Köllmann CP, Luong P, et al. Correction: p190 RhoGAP promotes contact inhibition in epithelial cells by repressing YAP activity. J Cell Biol. 2018;217(9):3313doi: 10.1083/JCB.20171005807172018c.
Frank, S. R., Köllmann, C. P., Luong, P., Galli, G. G., Zou, L., Bernards, A., Getz, G., Calogero, R. A., Frödin, M., & Hansen, S. H. (2018). Correction: p190 RhoGAP promotes contact inhibition in epithelial cells by repressing YAP activity. The Journal of cell biology, 217(9), 3313. https://doi.org/10.1083/JCB.20171005807172018c
Frank, Scott R, et al. "Correction: p190 RhoGAP promotes contact inhibition in epithelial cells by repressing YAP activity." The Journal of cell biology vol. 217,9 (2018): 3313. doi: https://doi.org/10.1083/JCB.20171005807172018c
Frank SR, Köllmann CP, Luong P, Galli GG, Zou L, Bernards A, Getz G, Calogero RA, Frödin M, Hansen SH. Correction: p190 RhoGAP promotes contact inhibition in epithelial cells by repressing YAP activity. J Cell Biol. 2018 Sep 03;217(9):3313. doi: 10.1083/JCB.20171005807172018c. Epub 2018 Jul 25. PMID: 30045860; PMCID: PMC6122999.
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An integrated clinical program and crowdsourcing strategy for genomic sequencing and Mendelian disease gene discovery.

NPJ genomic medicine

Haghighi A, Krier JB, Toth-Petroczy A, Cassa CA, Frank NY, Carmichael N, Fieg E, Bjonnes A, Mohanty A, Briere LC, Lincoln S, Lucia S, Gupta VA, Söylemez O, Sutti S, Kooshesh K, Qiu H, Fay CJ, Perroni V, Valerius J, Hanna M, Frank A, Ouahed J, Snapper SB, Pantazi A, Chopra SS, Leshchiner I, Stitziel NO, Feldweg A, Mannstadt M, Loscalzo J, Sweetser DA, Liao E, Stoler JM, Nowak CB, Sanchez-Lara PA, Klein OD, Perry H, Patsopoulos NA, Raychaudhuri S, Goessling W, Green RC, Seidman CE, MacRae CA, Sunyaev SR, Maas RL, Vuzman D.
PMID: 30131872
NPJ Genom Med. 2018 Aug 13;3:21. doi: 10.1038/s41525-018-0060-9. eCollection 2018.

Despite major progress in defining the genetic basis of Mendelian disorders, the molecular etiology of many cases remains unknown. Patients with these undiagnosed disorders often have complex presentations and require treatment by multiple health care specialists. Here, we describe...

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Haghighi A, Krier JB, Toth-Petroczy A, et al. An integrated clinical program and crowdsourcing strategy for genomic sequencing and Mendelian disease gene discovery. NPJ Genom Med. 2018;3:21doi: 10.1038/s41525-018-0060-9.
Haghighi, A., Krier, J. B., Toth-Petroczy, A., Cassa, C. A., Frank, N. Y., Carmichael, N., Fieg, E., Bjonnes, A., Mohanty, A., Briere, L. C., Lincoln, S., Lucia, S., Gupta, V. A., Söylemez, O., Sutti, S., Kooshesh, K., Qiu, H., Fay, C. J., Perroni, V., Valerius, J., Hanna, M., Frank, A., Ouahed, J., Snapper, S. B., Pantazi, A., Chopra, S. S., Leshchiner, I., Stitziel, N. O., Feldweg, A., Mannstadt, M., Loscalzo, J., Sweetser, D. A., Liao, E., Stoler, J. M., Nowak, C. B., Sanchez-Lara, P. A., Klein, O. D., Perry, H., Patsopoulos, N. A., Raychaudhuri, S., Goessling, W., Green, R. C., Seidman, C. E., MacRae, C. A., Sunyaev, S. R., Maas, R. L., Vuzman, D. (2018). An integrated clinical program and crowdsourcing strategy for genomic sequencing and Mendelian disease gene discovery. NPJ genomic medicine, 321. https://doi.org/10.1038/s41525-018-0060-9
Haghighi, Alireza, et al. "An integrated clinical program and crowdsourcing strategy for genomic sequencing and Mendelian disease gene discovery." NPJ genomic medicine vol. 3 (2018): 21. doi: https://doi.org/10.1038/s41525-018-0060-9
Haghighi A, Krier JB, Toth-Petroczy A, Cassa CA, Frank NY, Carmichael N, Fieg E, Bjonnes A, Mohanty A, Briere LC, Lincoln S, Lucia S, Gupta VA, Söylemez O, Sutti S, Kooshesh K, Qiu H, Fay CJ, Perroni V, Valerius J, Hanna M, Frank A, Ouahed J, Snapper SB, Pantazi A, Chopra SS, Leshchiner I, Stitziel NO, Feldweg A, Mannstadt M, Loscalzo J, Sweetser DA, Liao E, Stoler JM, Nowak CB, Sanchez-Lara PA, Klein OD, Perry H, Patsopoulos NA, Raychaudhuri S, Goessling W, Green RC, Seidman CE, MacRae CA, Sunyaev SR, Maas RL, Vuzman D. An integrated clinical program and crowdsourcing strategy for genomic sequencing and Mendelian disease gene discovery. NPJ Genom Med. 2018 Aug 13;3:21. doi: 10.1038/s41525-018-0060-9. eCollection 2018. PMID: 30131872; PMCID: PMC6089983.
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Robust differentiation of human enteroendocrine cells from intestinal stem cells.

Nature communications

Zeve D, Stas E, de Sousa Casal J, Mannam P, Qi W, Yin X, Dubois S, Shah MS, Syverson EP, Hafner S, Karp JM, Carlone DL, Ordovas-Montanes J, Breault DT.
PMID: 35017529
Nat Commun. 2022 Jan 11;13(1):261. doi: 10.1038/s41467-021-27901-5.

Enteroendocrine (EE) cells are the most abundant hormone-producing cells in humans and are critical regulators of energy homeostasis and gastrointestinal function. Challenges in converting human intestinal stem cells (ISCs) into functional EE cells, ex vivo, have limited progress in...

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Zeve D, Stas E, de Sousa Casal J, et al. Robust differentiation of human enteroendocrine cells from intestinal stem cells. Nat Commun. 2022;13(1):261doi: 10.1038/s41467-021-27901-5.
Zeve, D., Stas, E., de Sousa Casal, J., Mannam, P., Qi, W., Yin, X., Dubois, S., Shah, M. S., Syverson, E. P., Hafner, S., Karp, J. M., Carlone, D. L., Ordovas-Montanes, J., & Breault, D. T. (2022). Robust differentiation of human enteroendocrine cells from intestinal stem cells. Nature communications, 13(1), 261. https://doi.org/10.1038/s41467-021-27901-5
Zeve, Daniel, et al. "Robust differentiation of human enteroendocrine cells from intestinal stem cells." Nature communications vol. 13,1 (2022): 261. doi: https://doi.org/10.1038/s41467-021-27901-5
Zeve D, Stas E, de Sousa Casal J, Mannam P, Qi W, Yin X, Dubois S, Shah MS, Syverson EP, Hafner S, Karp JM, Carlone DL, Ordovas-Montanes J, Breault DT. Robust differentiation of human enteroendocrine cells from intestinal stem cells. Nat Commun. 2022 Jan 11;13(1):261. doi: 10.1038/s41467-021-27901-5. PMID: 35017529.
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Cytosolic detection of phagosomal bacteria-Mechanisms underlying PAMP exodus from the phagosome into the cytosol.

Molecular microbiology

Ragland SA, Kagan JC.
PMID: 34738270
Mol Microbiol. 2021 Dec;116(6):1420-1432. doi: 10.1111/mmi.14841. Epub 2021 Nov 22.

The metazoan innate immune system senses bacterial infections by detecting highly conserved bacterial molecules, termed pathogen-associated molecular patterns (PAMPs). PAMPs are detected by a variety of host pattern recognition receptors (PRRs), whose function is to coordinate downstream immune responses....

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Ragland SA, Kagan JC. Cytosolic detection of phagosomal bacteria-Mechanisms underlying PAMP exodus from the phagosome into the cytosol. Mol Microbiol. 2021;116(6):1420-1432doi: 10.1111/mmi.14841.
Ragland, S. A., & Kagan, J. C. (2021). Cytosolic detection of phagosomal bacteria-Mechanisms underlying PAMP exodus from the phagosome into the cytosol. Molecular microbiology, 116(6), 1420-1432. https://doi.org/10.1111/mmi.14841
Ragland, Stephanie A, and Kagan, Jonathan C. "Cytosolic detection of phagosomal bacteria-Mechanisms underlying PAMP exodus from the phagosome into the cytosol." Molecular microbiology vol. 116,6 (2021): 1420-1432. doi: https://doi.org/10.1111/mmi.14841
Ragland SA, Kagan JC. Cytosolic detection of phagosomal bacteria-Mechanisms underlying PAMP exodus from the phagosome into the cytosol. Mol Microbiol. 2021 Dec;116(6):1420-1432. doi: 10.1111/mmi.14841. Epub 2021 Nov 22. PMID: 34738270; PMCID: PMC8688326.
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Yersinia pseudotuberculosis YopE prevents uptake by M cells and instigates M cell extrusion in human ileal enteroid-derived monolayers.

Gut microbes

Fasciano AC, Dasanayake GS, Estes MK, Zachos NC, Breault DT, Isberg RR, Tan S, Mecsas J.
PMID: 34793276
Gut Microbes. 2021 Jan-Dec;13(1):1988390. doi: 10.1080/19490976.2021.1988390.

Many pathogens use M cells to access the underlying Peyer's patches and spread to systemic sites via the lymph as demonstrated by ligated loop murine intestinal models. However, the study of interactions between M cells and microbial pathogens has...

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Fasciano AC, Dasanayake GS, Estes MK, et al. Yersinia pseudotuberculosis YopE prevents uptake by M cells and instigates M cell extrusion in human ileal enteroid-derived monolayers. Gut Microbes. 2021;13(1):1988390doi: 10.1080/19490976.2021.1988390.
Fasciano, A. C., Dasanayake, G. S., Estes, M. K., Zachos, N. C., Breault, D. T., Isberg, R. R., Tan, S., & Mecsas, J. (2021). Yersinia pseudotuberculosis YopE prevents uptake by M cells and instigates M cell extrusion in human ileal enteroid-derived monolayers. Gut microbes, 13(1), 1988390. https://doi.org/10.1080/19490976.2021.1988390
Fasciano, Alyssa C, et al. "Yersinia pseudotuberculosis YopE prevents uptake by M cells and instigates M cell extrusion in human ileal enteroid-derived monolayers." Gut microbes vol. 13,1 (2021): 1988390. doi: https://doi.org/10.1080/19490976.2021.1988390
Fasciano AC, Dasanayake GS, Estes MK, Zachos NC, Breault DT, Isberg RR, Tan S, Mecsas J. Yersinia pseudotuberculosis YopE prevents uptake by M cells and instigates M cell extrusion in human ileal enteroid-derived monolayers. Gut Microbes. 2021 Jan-Dec;13(1):1988390. doi: 10.1080/19490976.2021.1988390. PMID: 34793276; PMCID: PMC8604394.
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Microbiota-neuroimmune cross talk in stress-induced visceral hypersensitivity of the bowel.

American journal of physiology. Gastrointestinal and liver physiology

van Thiel IAM, de Jonge WJ, Chiu IM, van den Wijngaard RM.
PMID: 32308040
Am J Physiol Gastrointest Liver Physiol. 2020 Jun 01;318(6):G1034-G1041. doi: 10.1152/ajpgi.00196.2019. Epub 2020 Apr 20.

Visceral hypersensitivity of the lower gastrointestinal tract, defined as an increased response to colorectal distension, frequently prompts episodes of debilitating abdominal pain in irritable bowel syndrome (IBS). Although the pathophysiology of IBS is not yet fully elucidated, it is...

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van Thiel IAM, de Jonge WJ, Chiu IM, et al. Microbiota-neuroimmune cross talk in stress-induced visceral hypersensitivity of the bowel. Am J Physiol Gastrointest Liver Physiol. 2020;318(6):G1034-G1041doi: 10.1152/ajpgi.00196.2019.
van Thiel, I. A. M., de Jonge, W. J., Chiu, I. M., & van den Wijngaard, R. M. (2020). Microbiota-neuroimmune cross talk in stress-induced visceral hypersensitivity of the bowel. American journal of physiology. Gastrointestinal and liver physiology, 318(6), G1034-G1041. https://doi.org/10.1152/ajpgi.00196.2019
van Thiel, Isabelle A M, et al. "Microbiota-neuroimmune cross talk in stress-induced visceral hypersensitivity of the bowel." American journal of physiology. Gastrointestinal and liver physiology vol. 318,6 (2020): G1034-G1041. doi: https://doi.org/10.1152/ajpgi.00196.2019
van Thiel IAM, de Jonge WJ, Chiu IM, van den Wijngaard RM. Microbiota-neuroimmune cross talk in stress-induced visceral hypersensitivity of the bowel. Am J Physiol Gastrointest Liver Physiol. 2020 Jun 01;318(6):G1034-G1041. doi: 10.1152/ajpgi.00196.2019. Epub 2020 Apr 20. PMID: 32308040; PMCID: PMC7642838.
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Showing 1 to 12 of 98 entries