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Proinsulin C-peptide regulates ribosomal RNA expression.

The Journal of biological chemistry

Lindahl E, Nyman U, Zaman F, Palmberg C, Cascante A, Shafqat J, Takigawa M, Sävendahl L, Jörnvall H, Joseph B.
PMID: 28283590
J Biol Chem. 2017 Mar 10;292(10):4382. doi: 10.1074/jbc.A109.053587.

No abstract available.

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Lindahl E, Nyman U, Zaman F, et al. Proinsulin C-peptide regulates ribosomal RNA expression. J Biol Chem. 2017;292(10):4382doi: 10.1074/jbc.A109.053587.
Lindahl, E., Nyman, U., Zaman, F., Palmberg, C., Cascante, A., Shafqat, J., Takigawa, M., Sävendahl, L., Jörnvall, H., & Joseph, B. (2017). Proinsulin C-peptide regulates ribosomal RNA expression. The Journal of biological chemistry, 292(10), 4382. https://doi.org/10.1074/jbc.A109.053587
Lindahl, Emma, et al. "Proinsulin C-peptide regulates ribosomal RNA expression." The Journal of biological chemistry vol. 292,10 (2017): 4382. doi: https://doi.org/10.1074/jbc.A109.053587
Lindahl E, Nyman U, Zaman F, Palmberg C, Cascante A, Shafqat J, Takigawa M, Sävendahl L, Jörnvall H, Joseph B. Proinsulin C-peptide regulates ribosomal RNA expression. J Biol Chem. 2017 Mar 10;292(10):4382. doi: 10.1074/jbc.A109.053587. PMID: 28283590; PMCID: PMC5354506.
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Pancreatic insulinomas: Laparoscopic management.

World journal of gastrointestinal endoscopy

Antonakis PT, Ashrafian H, Martinez-Isla A.
PMID: 26566426
World J Gastrointest Endosc. 2015 Nov 10;7(16):1197-207. doi: 10.4253/wjge.v7.i16.1197.

Insulinomas are rare pancreatic neuroendocrine tumors that are most commonly benign, solitary, and intrapancreatic. Uncontrolled insulin overproduction from the tumor produces neurological and adrenergic symptoms of hypoglycemia. Biochemical diagnosis is confirmed by the presence of Whipple's triad, along with...

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Antonakis PT, Ashrafian H, Martinez-Isla A. Pancreatic insulinomas: Laparoscopic management. World J Gastrointest Endosc. 2015;7(16):1197-207doi: 10.4253/wjge.v7.i16.1197.
Antonakis, P. T., Ashrafian, H., & Martinez-Isla, A. (2015). Pancreatic insulinomas: Laparoscopic management. World journal of gastrointestinal endoscopy, 7(16), 1197-207. https://doi.org/10.4253/wjge.v7.i16.1197
Antonakis, Pantelis T, et al. "Pancreatic insulinomas: Laparoscopic management." World journal of gastrointestinal endoscopy vol. 7,16 (2015): 1197-207. doi: https://doi.org/10.4253/wjge.v7.i16.1197
Antonakis PT, Ashrafian H, Martinez-Isla A. Pancreatic insulinomas: Laparoscopic management. World J Gastrointest Endosc. 2015 Nov 10;7(16):1197-207. doi: 10.4253/wjge.v7.i16.1197. PMID: 26566426; PMCID: PMC4639741.
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Correlation of obstructive sleep apnea hypopnea syndrome with metabolic syndrome in snorers.

Journal of biomedical research

Zhang Q, Zhang X, Zhao M, Huang H, Ding N, Zhang X, Wang H.
PMID: 25013405
J Biomed Res. 2014 May;28(3):222-7. doi: 10.7555/JBR.28.20120120. Epub 2014 Jan 30.

Though obstructive sleep apnea hypopnea syndrome (OSAHS) and metabolic syndrome (MS) are correlated; the contributing factors for the occurrence of MS in Chinese snorers remain largely undefined. We aimed to investigate the associated pathogenesis of coexistence of OSAHS and...

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Zhang Q, Zhang X, Zhao M, et al. Correlation of obstructive sleep apnea hypopnea syndrome with metabolic syndrome in snorers. J Biomed Res. 2014;28(3):222-7doi: 10.7555/JBR.28.20120120.
Zhang, Q., Zhang, X., Zhao, M., Huang, H., Ding, N., Zhang, X., & Wang, H. (2014). Correlation of obstructive sleep apnea hypopnea syndrome with metabolic syndrome in snorers. Journal of biomedical research, 28(3), 222-7. https://doi.org/10.7555/JBR.28.20120120
Zhang, Qun, et al. "Correlation of obstructive sleep apnea hypopnea syndrome with metabolic syndrome in snorers." Journal of biomedical research vol. 28,3 (2014): 222-7. doi: https://doi.org/10.7555/JBR.28.20120120
Zhang Q, Zhang X, Zhao M, Huang H, Ding N, Zhang X, Wang H. Correlation of obstructive sleep apnea hypopnea syndrome with metabolic syndrome in snorers. J Biomed Res. 2014 May;28(3):222-7. doi: 10.7555/JBR.28.20120120. Epub 2014 Jan 30. PMID: 25013405; PMCID: PMC4085559.
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Human Insulinomas: Clinical, Cellular, and Molecular Aspects.

Endocrine pathology

Komminoth P, Heitz PU, Roth J.
PMID: 12114763
Endocr Pathol. 1999;10(4):269-281. doi: 10.1007/BF02739769.

Insulinoma is the most frequently encountered functioning endocrine pancreatic tumor in humans. In this overview we summarize morphological and clinical features of insulinomas, report about the proinsulin-insulin conversion in normal and neoplastic B-cells, discuss the new classification, the criteria...

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Komminoth P, Heitz PU, Roth J. Human Insulinomas: Clinical, Cellular, and Molecular Aspects. Endocr Pathol. 1999;10(4):269-281doi: 10.1007/BF02739769.
Komminoth, P., Heitz, P. U., & Roth, J. (1999). Human Insulinomas: Clinical, Cellular, and Molecular Aspects. Endocrine pathology, 10(4), 269-281. https://doi.org/10.1007/BF02739769
Komminoth, Paul, et al. "Human Insulinomas: Clinical, Cellular, and Molecular Aspects." Endocrine pathology vol. 10,4 (1999): 269-281. doi: https://doi.org/10.1007/BF02739769
Komminoth P, Heitz PU, Roth J. Human Insulinomas: Clinical, Cellular, and Molecular Aspects. Endocr Pathol. 1999;10(4):269-281. doi: 10.1007/BF02739769. PMID: 12114763.
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Subtotal pancreatectomy for hypoglycemia due to congenital hyperinsulinism: long-term follow-up of neurodevelopmental and pancreatic function.

Pediatric diabetes

Rother KI, Matsumoto JM, Rasmussen NH, Schwenk WF.
PMID: 15016194
Pediatr Diabetes. 2001 Sep;2(3):115-22. doi: 10.1034/j.1399-5448.2001.002003115.x.

OBJECTIVE: To evaluate neurodevelopmental status as well as endocrine and exocrine pancreatic function in children who have undergone subtotal pancreatectomy for hypoglycemia due to congenital hyperinsulinism.PATIENTS AND METHODS: Out of 15 identified patients, eight children (mean age 12.7 +/-...

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Rother KI, Matsumoto JM, Rasmussen NH, et al. Subtotal pancreatectomy for hypoglycemia due to congenital hyperinsulinism: long-term follow-up of neurodevelopmental and pancreatic function. Pediatr Diabetes. 2001;2(3):115-22doi: 10.1034/j.1399-5448.2001.002003115.x.
Rother, K. I., Matsumoto, J. M., Rasmussen, N. H., & Schwenk, W. F. (2001). Subtotal pancreatectomy for hypoglycemia due to congenital hyperinsulinism: long-term follow-up of neurodevelopmental and pancreatic function. Pediatric diabetes, 2(3), 115-22. https://doi.org/10.1034/j.1399-5448.2001.002003115.x
Rother, K I, et al. "Subtotal pancreatectomy for hypoglycemia due to congenital hyperinsulinism: long-term follow-up of neurodevelopmental and pancreatic function." Pediatric diabetes vol. 2,3 (2001): 115-22. doi: https://doi.org/10.1034/j.1399-5448.2001.002003115.x
Rother KI, Matsumoto JM, Rasmussen NH, Schwenk WF. Subtotal pancreatectomy for hypoglycemia due to congenital hyperinsulinism: long-term follow-up of neurodevelopmental and pancreatic function. Pediatr Diabetes. 2001 Sep;2(3):115-22. doi: 10.1034/j.1399-5448.2001.002003115.x. PMID: 15016194.
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C-Peptide in the vessel wall.

The review of diabetic studies : RDS

Walcher D, Marx N.
PMID: 20039007
Rev Diabet Stud. 2009;6(3):180-6. doi: 10.1900/RDS.2009.6.180. Epub 2009 Nov 10.

Patients with insulin resistance and early type 2 diabetes exhibit an increased sensitivity to develop a diffuse and extensive pattern of arteriosclerosis leading to a remarkable increase in vascular complications, including myocardial infarction and stroke. The accelerated atherosclerosis in...

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Walcher D, Marx N. C-Peptide in the vessel wall. Rev Diabet Stud. 2009;6(3):180-6doi: 10.1900/RDS.2009.6.180.
Walcher, D., & Marx, N. (2009). C-Peptide in the vessel wall. The review of diabetic studies : RDS, 6(3), 180-6. https://doi.org/10.1900/RDS.2009.6.180
Walcher, Daniel, and Marx, Nikolaus. "C-Peptide in the vessel wall." The review of diabetic studies : RDS vol. 6,3 (2009): 180-6. doi: https://doi.org/10.1900/RDS.2009.6.180
Walcher D, Marx N. C-Peptide in the vessel wall. Rev Diabet Stud. 2009;6(3):180-6. doi: 10.1900/RDS.2009.6.180. Epub 2009 Nov 10. PMID: 20039007; PMCID: PMC2827270.
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Insulinotropic glucagonlike peptide-I(7-37)/(7-36)amide A new incretin hormone.

Trends in endocrinology and metabolism: TEM

Fehmann HC, Habener JF.
PMID: 18407095
Trends Endocrinol Metab. 1992 Jul;3(5):158-63. doi: 10.1016/1043-2760(92)90165-w.

Intestinal peptide hormones, released into circulation in response to a meal, are important augmentors of the postprandial insulin release stimulated by absorbed nutrients. In this "Incretin concept" glucagon-like peptide I(7-37) plays a major role because it is the most...

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Fehmann HC, Habener JF. Insulinotropic glucagonlike peptide-I(7-37)/(7-36)amide A new incretin hormone. Trends Endocrinol Metab. 1992;3(5):158-63doi: 10.1016/1043-2760(92)90165-w.
Fehmann, H. C., & Habener, J. F. (1992). Insulinotropic glucagonlike peptide-I(7-37)/(7-36)amide A new incretin hormone. Trends in endocrinology and metabolism: TEM, 3(5), 158-63. https://doi.org/10.1016/1043-2760(92)90165-w
Fehmann, H C, and Habener, J F. "Insulinotropic glucagonlike peptide-I(7-37)/(7-36)amide A new incretin hormone." Trends in endocrinology and metabolism: TEM vol. 3,5 (1992): 158-63. doi: https://doi.org/10.1016/1043-2760(92)90165-w
Fehmann HC, Habener JF. Insulinotropic glucagonlike peptide-I(7-37)/(7-36)amide A new incretin hormone. Trends Endocrinol Metab. 1992 Jul;3(5):158-63. doi: 10.1016/1043-2760(92)90165-w. PMID: 18407095.
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Computer-aided process analysis and economic evaluation for biosynthetic human insulin production-A case study.

Biotechnology and bioengineering

Petrides D, Sapidou E, Calandranis J.
PMID: 18623518
Biotechnol Bioeng. 1995 Dec 05;48(5):529-41. doi: 10.1002/bit.260480516.

Human insulin was the first mammalian protein produced in bacteria using recombinant DNA technology. Two technologies were developed; the first based on the separate expression of precursors of chains A and B of insulin, and the second based on...

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Petrides D, Sapidou E, Calandranis J. Computer-aided process analysis and economic evaluation for biosynthetic human insulin production-A case study. Biotechnol Bioeng. 1995;48(5):529-41doi: 10.1002/bit.260480516.
Petrides, D., Sapidou, E., & Calandranis, J. (1995). Computer-aided process analysis and economic evaluation for biosynthetic human insulin production-A case study. Biotechnology and bioengineering, 48(5), 529-41. https://doi.org/10.1002/bit.260480516
Petrides, D, et al. "Computer-aided process analysis and economic evaluation for biosynthetic human insulin production-A case study." Biotechnology and bioengineering vol. 48,5 (1995): 529-41. doi: https://doi.org/10.1002/bit.260480516
Petrides D, Sapidou E, Calandranis J. Computer-aided process analysis and economic evaluation for biosynthetic human insulin production-A case study. Biotechnol Bioeng. 1995 Dec 05;48(5):529-41. doi: 10.1002/bit.260480516. PMID: 18623518.
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Differential expression of vacuolar-type H+-ATPase between normal human pancreatic islet B-cells and insulinoma cells.

International journal of oncology

Ohta T, Yamamoto M, Numata M, Iseki S, Kitagawa H, Kayahara M, Nagakawa T, Miwa K, Nakagawa A, Morise T, Ohkuma S, Terada T.
PMID: 21528252
Int J Oncol. 1997 Sep;11(3):597-601. doi: 10.3892/ijo.11.3.597.

Recent studies have shown that bafilomycin A(1)-sensitive vacuolar type H+-ATPase (V-ATPase) is responsible for the acidification of intracellular compartments in eukaryotic cells. B-cells of pancreatic islets also are known to include acidifying secretory vesicles which are the major cellular...

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Ohta T, Yamamoto M, Numata M, et al. Differential expression of vacuolar-type H+-ATPase between normal human pancreatic islet B-cells and insulinoma cells. Int J Oncol. 1997;11(3):597-601doi: 10.3892/ijo.11.3.597.
Ohta, T., Yamamoto, M., Numata, M., Iseki, S., Kitagawa, H., Kayahara, M., Nagakawa, T., Miwa, K., Nakagawa, A., Morise, T., Ohkuma, S., & Terada, T. (1997). Differential expression of vacuolar-type H+-ATPase between normal human pancreatic islet B-cells and insulinoma cells. International journal of oncology, 11(3), 597-601. https://doi.org/10.3892/ijo.11.3.597
Ohta, T, et al. "Differential expression of vacuolar-type H+-ATPase between normal human pancreatic islet B-cells and insulinoma cells." International journal of oncology vol. 11,3 (1997): 597-601. doi: https://doi.org/10.3892/ijo.11.3.597
Ohta T, Yamamoto M, Numata M, Iseki S, Kitagawa H, Kayahara M, Nagakawa T, Miwa K, Nakagawa A, Morise T, Ohkuma S, Terada T. Differential expression of vacuolar-type H+-ATPase between normal human pancreatic islet B-cells and insulinoma cells. Int J Oncol. 1997 Sep;11(3):597-601. doi: 10.3892/ijo.11.3.597. PMID: 21528252.
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Mesenchymal stem cells derived from bone marrow of diabetic patients portrait unique markers influenced by the diabetic microenvironment.

The review of diabetic studies : RDS

Phadnis SM, Ghaskadbi SM, Hardikar AA, Bhonde RR.
PMID: 20043038
Rev Diabet Stud. 2009;6(4):260-70. doi: 10.1900/RDS.2009.6.260. Epub 2009 Dec 30.

Cellular microenvironment is known to play a critical role in the maintenance of human bone marrow-derived mesenchymal stem cells (BM-MSCs). It was uncertain whether BM-MSCs obtained from a 'diabetic milieu' (dBM-MSCs) offer the same regenerative potential as those obtained...

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Phadnis SM, Ghaskadbi SM, Hardikar AA, et al. Mesenchymal stem cells derived from bone marrow of diabetic patients portrait unique markers influenced by the diabetic microenvironment. Rev Diabet Stud. 2009;6(4):260-70doi: 10.1900/RDS.2009.6.260.
Phadnis, S. M., Ghaskadbi, S. M., Hardikar, A. A., & Bhonde, R. R. (2009). Mesenchymal stem cells derived from bone marrow of diabetic patients portrait unique markers influenced by the diabetic microenvironment. The review of diabetic studies : RDS, 6(4), 260-70. https://doi.org/10.1900/RDS.2009.6.260
Phadnis, Smruti M, et al. "Mesenchymal stem cells derived from bone marrow of diabetic patients portrait unique markers influenced by the diabetic microenvironment." The review of diabetic studies : RDS vol. 6,4 (2009): 260-70. doi: https://doi.org/10.1900/RDS.2009.6.260
Phadnis SM, Ghaskadbi SM, Hardikar AA, Bhonde RR. Mesenchymal stem cells derived from bone marrow of diabetic patients portrait unique markers influenced by the diabetic microenvironment. Rev Diabet Stud. 2009;6(4):260-70. doi: 10.1900/RDS.2009.6.260. Epub 2009 Dec 30. PMID: 20043038; PMCID: PMC2836197.
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Anti-diabetic treatment regulates pro-fibrotic TGF-β serum levels in type 2 diabetics.

Diabetology & metabolic syndrome

Pscherer S, Freude T, Forst T, Nussler AK, Braun KF, Ehnert S.
PMID: 24004910
Diabetol Metab Syndr. 2013 Aug 31;5(1):48. doi: 10.1186/1758-5996-5-48.

BACKGROUND: The single-center, open-label, four-arm, exploratory study investigates the relation of different anti-diabetics to serum levels of active TGF-β, a known pro-fibrotic stimulus, before and after a defined test meal.FINDINGS: We investigated sera of patients with type 2 diabetes...

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Pscherer S, Freude T, Forst T, et al. Anti-diabetic treatment regulates pro-fibrotic TGF-β serum levels in type 2 diabetics. Diabetol Metab Syndr. 2013;5(1):48doi: 10.1186/1758-5996-5-48.
Pscherer, S., Freude, T., Forst, T., Nussler, A. K., Braun, K. F., & Ehnert, S. (2013). Anti-diabetic treatment regulates pro-fibrotic TGF-β serum levels in type 2 diabetics. Diabetology & metabolic syndrome, 5(1), 48. https://doi.org/10.1186/1758-5996-5-48
Pscherer, Stefan, et al. "Anti-diabetic treatment regulates pro-fibrotic TGF-β serum levels in type 2 diabetics." Diabetology & metabolic syndrome vol. 5,1 (2013): 48. doi: https://doi.org/10.1186/1758-5996-5-48
Pscherer S, Freude T, Forst T, Nussler AK, Braun KF, Ehnert S. Anti-diabetic treatment regulates pro-fibrotic TGF-β serum levels in type 2 diabetics. Diabetol Metab Syndr. 2013 Aug 31;5(1):48. doi: 10.1186/1758-5996-5-48. PMID: 24004910; PMCID: PMC3847073.
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Insulin sensitivity and β-cell function in normoglycemic offspring of individuals with type 2 diabetes mellitus: Impact of line of inheritance.

Indian journal of endocrinology and metabolism

Praveen EP, Sahoo J, Khurana ML, Kulshreshtha B, Khadgawat R, Gupta N, Dwivedi SN, Kumar G, Prabhakaran D, Ammini AC.
PMID: 22276260
Indian J Endocrinol Metab. 2012 Jan;16(1):105-11. doi: 10.4103/2230-8210.91204.

AIMS: The aim was to study the effect of family history of type 2 diabetes mellitus (T2DM) on insulin sensitivity and β-cell function in normoglycemic offspring.MATERIAL AND METHODS: Offspring of T2DM patients (cases) and individuals without family history of...

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Praveen EP, Sahoo J, Khurana ML, et al. Insulin sensitivity and β-cell function in normoglycemic offspring of individuals with type 2 diabetes mellitus: Impact of line of inheritance. Indian J Endocrinol Metab. 2012;16(1):105-11doi: 10.4103/2230-8210.91204.
Praveen, E. P., Sahoo, J., Khurana, M. L., Kulshreshtha, B., Khadgawat, R., Gupta, N., Dwivedi, S. N., Kumar, G., Prabhakaran, D., & Ammini, A. C. (2012). Insulin sensitivity and β-cell function in normoglycemic offspring of individuals with type 2 diabetes mellitus: Impact of line of inheritance. Indian journal of endocrinology and metabolism, 16(1), 105-11. https://doi.org/10.4103/2230-8210.91204
Praveen, Edavan P, et al. "Insulin sensitivity and β-cell function in normoglycemic offspring of individuals with type 2 diabetes mellitus: Impact of line of inheritance." Indian journal of endocrinology and metabolism vol. 16,1 (2012): 105-11. doi: https://doi.org/10.4103/2230-8210.91204
Praveen EP, Sahoo J, Khurana ML, Kulshreshtha B, Khadgawat R, Gupta N, Dwivedi SN, Kumar G, Prabhakaran D, Ammini AC. Insulin sensitivity and β-cell function in normoglycemic offspring of individuals with type 2 diabetes mellitus: Impact of line of inheritance. Indian J Endocrinol Metab. 2012 Jan;16(1):105-11. doi: 10.4103/2230-8210.91204. PMID: 22276260; PMCID: PMC3263177.
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Showing 1 to 12 of 175 entries